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Relevant bibliographies by topics / Macrophages M2-Like

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Academic literature on the topic 'Macrophages M2-Like'

Author: Grafiati

Published: 20 April 2024

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Journal articles on the topic "Macrophages M2-Like"

1

Wen, Zhifa, Hongxiang Liu, Meng Zhou, and Li-xin Wang. "Tumor released autophagosomes regulate M2-like macrophage polarization (TUM6P.974)." Journal of Immunology 194, no. 1_Supplement (2015): 141.22. http://dx.doi.org/10.4049/jimmunol.194.supp.141.22.

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Abstract Tumor cells secrete mediators that modulate macrophage activation and polarization into M2 type tumor associated macrophages (TAMs), which contribute to tumor progression. However, the mechanisms that regulate the polarization are poorly defined. We have previously reported that the enrichment of autophagosomes in tumor cell conditioned medium and malignant ascites from patients. The present study investigated the hypothesis that tumor released autophagosomes promoted M2 polarization. Here we isolated autophagosomes released from murine liver cancer cells using affinity purification.

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2

Draijer, Christina, Patricia Robbe, Carian E. Boorsma, Machteld N. Hylkema, and Barbro N. Melgert. "Characterization of Macrophage Phenotypes in Three Murine Models of House-Dust-Mite-Induced Asthma." Mediators of Inflammation 2013 (2013): 1–10. http://dx.doi.org/10.1155/2013/632049.

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In asthma, an important role for innate immunity is increasingly being recognized. Key innate immune cells in the lungs are macrophages. Depending on the signals they receive, macrophages can at least have an M1, M2, or M2-like phenotype. It is unknown how these macrophage phenotypes behave with regard to (the severity of) asthma. We have quantified the phenotypes in three models of house dust mite (HDM-)induced asthma (14, 21, and 24 days). M1, M2, and M2-like phenotypes were identified by interferon regulatory factor 5 (IRF5), YM1, and IL-10, respectively. We found higher percentages of eosi

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3

Lalor, Richard, та Sandra O’Neill. "Bovine κ-Casein Fragment Induces Hypo-Responsive M2-Like Macrophage Phenotype". Nutrients 11, № 7 (2019): 1688. http://dx.doi.org/10.3390/nu11071688.

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Immunomodulatory nutraceuticals have garnered special attention due to their therapeutic potential for the amelioration of many chronic inflammatory conditions. Macrophages are key players in the induction, propagation and resolution of inflammation, actively contributing to the pathogenesis and resolution of inflammatory disorders. As such, this study aimed to investigate the possible therapeutic effects bovine casein derived nutraceuticals exert on macrophage immunological function. Initial studies demonstrated that sodium caseinate induced a M2-like macrophage phenotype that was attributed

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4

Lyu, Qingkang, Edwin J. A. Veldhuizen, Irene S. Ludwig, et al. "Characterization of polarization states of canine monocyte derived macrophages." PLOS ONE 18, no. 11 (2023): e0292757. http://dx.doi.org/10.1371/journal.pone.0292757.

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Macrophages can reversibly polarize into multiple functional subsets depending on their micro-environment. Identification and understanding the functionality of these subsets is relevant for the study of immune‑related diseases. However, knowledge about canine macrophage polarization is still in its infancy. In this study, we polarized canine monocytes using GM-CSF/IFN- γ and LPS towards M1 macrophages or M-CSF and IL-4 towards M2 macrophages and compared them to undifferentiated monocytes (M0). Polarized M1 and M2 macrophages were thoroughly characterized for morphology, surface marker featur

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Sánchez-Reyes, Karina, Alejandro Bravo-Cuellar, Georgina Hernández-Flores, et al. "Cervical Cancer Cell Supernatants Induce a Phenotypic Switch from U937-Derived Macrophage-Activated M1 State into M2-Like Suppressor Phenotype with Change in Toll-Like Receptor Profile." BioMed Research International 2014 (2014): 1–11. http://dx.doi.org/10.1155/2014/683068.

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Cervical cancer (CC) is the second most common cancer among women worldwide. Infection with human papillomavirus (HPV) is the main risk factor for developing CC. Macrophages are important immune effector cells; they can be differentiated into two phenotypes, identified as M1 (classically activated) and M2 (alternatively activated). Macrophage polarization exerts profound effects on the Toll-like receptor (TLR) profile. In this study, we evaluated whether the supernatant of human CC cells HeLa, SiHa, and C-33A induces a shift of M1 macrophage toward M2 macrophage in U937-derived macrophages.Res

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6

Zhu, Wenya, Qianqian Chen, Yi Li, Jun Wan, Jia Li та Shuai Tang. "HIF-1α-Overexpressing Mesenchymal Stem Cells Attenuate Colitis by Regulating M1-like Macrophages Polarization toward M2-like Macrophages". Biomedicines 11, № 3 (2023): 825. http://dx.doi.org/10.3390/biomedicines11030825.

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A modified mesenchymal stem cell (MSC) transplantation is a highly effective and precise treatment for inflammatory bowel disease (IBD), with a significant curative effect. Thus, we aim to examine the efficacy of hypoxia-inducible factor (HIF)–1α-overexpressing MSC (HIF-MSC) transplantation in experimental colitis and investigate the immunity regulation mechanisms of HIF-MSC through macrophages. A chronic experimental colitis mouse model was established using 2,4,6-trinitrobenzene sulfonic acid. HIF-MSC transplantation significantly attenuated colitis in weight loss rate, disease activity inde

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7

Strizova, Zuzana, Iva Benesova, Robin Bartolini, et al. "M1/M2 macrophages and their overlaps – myth or reality?" Clinical Science 137, no. 15 (2023): 1067–93. http://dx.doi.org/10.1042/cs20220531.

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Abstract Macrophages represent heterogeneous cell population with important roles in defence mechanisms and in homoeostasis. Tissue macrophages from diverse anatomical locations adopt distinct activation states. M1 and M2 macrophages are two polarized forms of mononuclear phagocyte in vitro differentiation with distinct phenotypic patterns and functional properties, but in vivo, there is a wide range of different macrophage phenotypes in between depending on the microenvironment and natural signals they receive. In human infections, pathogens use different strategies to combat macrophages and

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8

Li, Dezhi, Min Yan, Fengfei Sun, et al. "miR-498 inhibits autophagy and M2-like polarization of tumor-associated macrophages in esophageal cancer via MDM2/ATF3." Epigenomics 13, no. 13 (2021): 1013–30. http://dx.doi.org/10.2217/epi-2020-0341.

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Structured abstract Aim: To elucidate the effect of miRNA (miR)-498 on autophagy and M2-like macrophage polarization in esophageal cancer. Methods: Autophagy was evaluated in esophageal cancer. Macrophage markers specific for M1- or M2-like phenotype were determined. The binding relationships between miR-498 and MDM2, MDM2 and ATF3 were analyzed. Results: miR-498 was downregulated in esophageal cancer and was associated with disease-free and overall patient survival. Enhanced miR-498 reduced LC3I conversion to LC3II and increased p62 accumulation in KYSE-150 cells, and increased macrophage pol

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9

Ronaghan, Natalie J., Mandy Soo, Uriel Pena, et al. "M1-like, but not M0- or M2-like, macrophages, reduce RSV infection of primary bronchial epithelial cells in a media-dependent fashion." PLOS ONE 17, no. 10 (2022): e0276013. http://dx.doi.org/10.1371/journal.pone.0276013.

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Respiratory syncytial virus (RSV) is a common childhood infection that in young infants can progress into severe bronchiolitis and pneumonia. Disease pathogenesis results from both viral mediated and host immune processes of which alveolar macrophages play an important part. Here, we investigated the role of different types of alveolar macrophages on RSV infection using an in vitro co-culture model involving primary tissue-derived human bronchial epithelial cells (HBECs) and human blood monocyte-derived M0-like, M1-like, or M2-like macrophages. It was hypothesized that the in vitro model would

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10

Di Martile, Marta, Valentina Farini, Francesca Maria Consonni, et al. "Melanoma-specific bcl-2 promotes a protumoral M2-like phenotype by tumor-associated macrophages." Journal for ImmunoTherapy of Cancer 8, no. 1 (2020): e000489. http://dx.doi.org/10.1136/jitc-2019-000489.

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BackgroundA bidirectional crosstalk between tumor cells and the surrounding microenvironment contributes to tumor progression and response to therapy. Our previous studies have demonstrated that bcl-2 affects melanoma progression and regulates the tumor microenvironment. The aim of this study was to evaluate whether bcl-2 expression in melanoma cells could influence tumor-promoting functions of tumor-associated macrophages, a major constituent of the tumor microenvironment that affects anticancer immunity favoring tumor progression.MethodsTHP-1 monocytic cells, monocyte-derived macrophages and

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