Books on the topic 'Macrophage'

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1

Bernard, Burke, and Lewis Claire E, eds. The macrophage. 2nd ed. Oxford: Oxford University Press, 2002.

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2

E, Lewis Claire, and McGee J. O'D, eds. The Macrophage. Oxford: IRL Press at Oxford University Press, 1992.

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3

The human macrophage system: Activity and functional morphology. Basel: Karger, 1988.

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4

Röszer, Tamás. The M2 Macrophage. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50480-9.

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5

Clemens, Sorg, and Immunologische Tage. (1987 : Münster, West Germany), eds. The Alveolar macrophage. Münster: Regensberg & Biermann Wissenschaftliche Verlagsgellschaft, 1988.

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6

1943-, Zwilling Bruce S., and Eisenstein Toby K, eds. Macrophage-pathogen interactions. New York: M. Dekker, 1994.

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7

Gupta, Swati, and Yashwant V. Pathak, eds. Macrophage Targeted Delivery Systems. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-84164-5.

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8

Russell, Stephen W., and Siamon Gordon, eds. Macrophage Biology and Activation. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77377-8.

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9

Harris, James, and Eric F. Morand, eds. Macrophage Migration Inhibitory Factor. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-4939-9936-1.

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10

W, Russell Stephen, and Gordon Siamon, eds. Macrophage biology and activation. Berlin: Springer-Verlag, 1992.

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11

1929-, Staub Norman C., ed. The Pulmonary intravascular macrophage. Mount Kisco, NY: Futura Pub. Co., 1989.

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12

Herbein, Georges. HIV and the macrophage. Kerala, India: Transworld Research Network, 2007.

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13

Gordon, Siamon, ed. The Macrophage as Therapeutic Target. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55742-2.

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14

Clemens, Sorg, ed. Macrophage-derived cell regulatory factors. Basel: Karger, 1989.

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15

Rossi, Bartira Consini. Macrophage function in African trypanosomiasis. Uxbridge: Brunel University, 1986.

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16

Damoiseaux, Jan G. M. C., ed. Macrophage heterogeneity established by immunocytochemistry. Stuttgart: Gustav Fischer, 1993.

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17

Nicholls, S. E. Control of granulocyte / macrophage progenitor cells bygrowthfactors. Manchester: UMIST, 1993.

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18

Voronov, Irina. Macrophage phagocytosis of polyethylene particulate in vitro. Ottawa: National Library of Canada, 1997.

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19

Paladi, Magdalena. A genetic analysis of Drosophila embryonic macrophage migration. Ottawa: National Library of Canada, 2003.

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20

Murphy, Colin J. The importance of glutamine metabolism to macrophage function. Dublin: University College Dublin, 1997.

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21

Bottalico, Lori A. Regulation and consequences of macrophage foam cell formation. [New York]: [Columbia University], 1993.

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22

Flora-Rehal, Pauline Kaur. Mechanisms involved in macrophage recognition of apoptotic B cells. Birmingham: University of Birmingham, 1995.

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23

Kucey, Daryl Stanton. Modulation of macrophage procoagulant activity by platelet activating factor. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1992.

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24

Wilson, Christopher James. Macrophage mediated drug delivery to tumour cells in vitro. Leicester: DeMontfort University, 1992.

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25

Richard, Bucala, ed. MIF: Most interesting factor. Singapore: World Scientific, 2007.

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26

Adipocytes Secrete Lipid-Laden Exosomes and Influence Local Macrophage Behavior. [New York, N.Y.?]: [publisher not identified], 2020.

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27

Xing, Shaomo. The effect of chemically altered polyethylene on human macrophage function. Ottawa: National Library of Canada, 2000.

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28

EULEP, Symposium (1990 Oxford United Kingdom). Role of the alveolar macrophage in the clearance of inhaled particles. Research Triangle Park, N.C: National Institute of Environmental Health Sciences, 1992.

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29

Simpson, Jeremy Andrew. Pseudomonas aeruginosa and the evasion of macrophage function in cystic fibrosis. Uxbridge: Brunel University, 1988.

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30

Roles of Macrophage Mitochondrial Oxidative Stress and Mitochondrial Fission in Atherosclerosis. [New York, N.Y.?]: [publisher not identified], 2014.

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31

1955-, Suttles Jill, ed. T-cell signaling of macrophage activation: Cell contact-dependent and cytokine signals. Austin: R.G. Landes, 1995.

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32

M, Marty, ed. Manual of GM-CSF. Oxford: Blackwell Science, 1996.

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33

Eason, Stephen J. Studies of the inhibition of macrophage respiratory burst by tumor cell derived product(s). [s.l: The Author], 1992.

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34

Hingley-Wilson, Suzanne Maria. The detection and identification of macrophage genes induced by intracellular infection with mycobacterium tuberculosis. Birmingham: University of Birmingham, 1998.

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35

Hildebrand, Dominic. The 'inhibitor of NF-kappa B zeta' is a pleiotropic key regulator of macrophage function. [S.l: s.n.], 2013.

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36

Cynn, Esther. Investigation of Novel lincRNAs SIMALR and RP11-184M15.1 Functions in Inflammatory and Resolving Human Macrophage. [New York, N.Y.?]: [publisher not identified], 2022.

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37

Stober, Carmel Bernadette. P2 purinoceptor involvement in ATP-mediated macrophage death and killing of intracellular Mycobacterium bovis bacille calmette-guérin. Birmingham: University of Birmingham, 2000.

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38

Kloc, Malgorzata, ed. Macrophages. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-54090-0.

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39

Adams, Dolph O. Macrophage Activation. Springer, 2013.

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40

ZHU, LI MEI. macrophage Love. Academia Press, 1991.

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41

Adams, Dolph O. Macrophage Activation. Springer, 2013.

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42

Röszer, Tamás. M2 Macrophage. Springer International Publishing AG, 2021.

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43

(Editor), Bernard Burke, and Claire E. Lewis (Editor), eds. The Macrophage. 2nd ed. Oxford University Press, USA, 2002.

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44

Grom, Alexei A., and Athimalaipet V. Ramanan. Macrophage activation syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0168.

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Macrophage activation syndrome (MAS) is a life-threatening condition caused by excessive activation and proliferation of T lymphocytes and haemophagocytic macrophages. Although MAS has been reported in association with almost any rheumatic disease, it is by far most common in systemic juvenile idiopathic arthritis. Flares of the underlying disease or infection are most common triggers of MAS. The pathognomonic feature of MAS is typically found in bone marrow: numerous, well-differentiated macrophagic histiocytes phagocytosing normal haematopoietic elements. The expansion of these histiocytes leads to a massive systemic inflammatory reaction associated with three cardinal clinical features: severe cytopenias, liver dysfunction, and coagulopathy consistent with disseminated intravascular coagulation. Clinically, MAS is strikingly similar to the autosomal recessive disorders collectively known as familial haemophagocytic lymphohistiocytosis (FHLH). FHLH has been associated with various genetic defects affecting the cytolytic pathway. Cytolytic function is profoundly depressed in MAS patients as well, and this abnormality is caused by both genetic and acquired factors. Studies in animals suggest that uncontrolled expansion of activated CD8+ T lymphocytes secreting cytokines that activate macrophages is central to the pathophysiology of haemophagocytic syndromes. Consistent with this view, the combination of steroids and ciclosporin, an immunosuppressant that preferentially inhibits T lymphocytes, is an effective treatment for the majority of MAS patients. Patients in whom MAS remains active despite this treatment present a serious challenge and require more aggressive immunosuppression. However, in MAS triggered by infection, the optimal level of immunosuppression is difficult to determine. As a result, reported mortality rates reach 20%.
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45

Bondarenko, Olesja, and Fernando Torres Andón, eds. Nanoparticle-Macrophage Interactions. MDPI, 2022. http://dx.doi.org/10.3390/books978-3-0365-4600-1.

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46

REICHARD, S. Reichard Macrophage Biology. John Wiley & Sons Inc, 1986.

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47

Röszer, Tamás. The M2 Macrophage. Springer, 2020.

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48

Fink, Mary. Macrophage in Neoplasia. Elsevier Science & Technology Books, 2012.

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49

Nelson, David S. Immunobiology of the Macrophage. Elsevier Science & Technology Books, 2014.

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50

Gordon, Siamon, and Stephen W. Russell. Macrophage Biology and Activation. Springer London, Limited, 2011.

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