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Books on the topic 'Lymphomes T'

Author: Grafiati

Published: 1 March 2025

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1

Foss, Francine, ed. T-Cell Lymphomas. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-170-7.

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2

Querfeld, Christiane, Jasmine Zain, and Steven T. Rosen, eds. T-Cell and NK-Cell Lymphomas. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-99716-2.

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3

Watanabe, Toshiki, and Takuya Fukushima, eds. Adult T-cell Leukemia/Lymphoma. Tokyo: Springer Japan, 2017. http://dx.doi.org/10.1007/978-4-431-56523-9.

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4

Clark, Lambert W., Giannotti B, Vloten W. A. van, North Atlantic Treaty Organization. Scientific Affairs Division., and NATO Advanced Research Workshop on Basic Mechanisms of Physiologic and Aberrant Lymphoproliferation in the Skin (1991 : San Miniato, Italy), eds. Basic mechanisms of physiologic and aberrant lymphoproliferation in the skin. New York: Plenum Press, 1994.

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5

Symposium, Takamatsu no Miya Hi Gan Kenkyū Kikin International. Retroviruses in human lymphoma/leukemia: Proceedings of the 15th International Symposium of the Princess Takamatsu Cancer Research Fund, Tokyo, 1984. Tokyo: Japan Scientific Societies Press, 1985.

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6

Hall, John C., and Brian J. Hall. Cutaneous lymphoma: Diagnosis and treatment. Shelton, Conn: People's Medical Pub. House-USA, 2012.

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7

Kenton, Charlotte. Human T-cell leukemia/lymphoma virus (HTLV), January 1982 through September 1984, 234 citations. [Bethesda, Md.]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1985.

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8

1952-, Rosen Steven T., ed. Cutaneous t-cell lymphoma: A guide for the community oncologist. Manhasset, NY: CMP Medica, 2006.

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9

S, Zackheim Herschel, ed. Cutaneous T-cell lymphoma: Mycosis fungoides and Sézary syndrome. Boca Raton, Fla: CRC Press, 2005.

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10

Kelleher, Mittie Elizabeth. Relationship of BE2 surface antigen expression to T-cell clonality in cutaneous T-cell lymphoma and scleroderma. [New Haven: s.n.], 1990.

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11

1932-, Vogt P. K., ed. Human T-cell leukemia virus. Berlin: Springer, 1985.

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12

Brennan, Laurie Anne. Low expression of the LSP1 gene in early mouse T-lymphomas induced by N-methyl-N-nitrosourea. Ottawa: National Library of Canada, 1996.

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13

Kim, Christine Sheila. The FYN-TRAF3IP2 gene fusion drives oncogenic NF-κB signaling in peripheral T cell lymphoma. [New York, N.Y.?]: [publisher not identified], 2020.

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14

Shad, Yasmine. Clinical diagnosis of the t (14, 18) chromosomal translocation characteristic of follicular lymphoma using southern blot hybridization. Sudbury, Ont: Laurentian University, 1992.

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15

Vesprini, Danny. Illegitimate V(D)J rearrangement in gcs-irradiation induced T cell lymphoma in newborn scid mice. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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16

Schütt, Silvia. Molekulargenetische Untersuchungen der Immunglobulin- und T-Zellrezeptor-Genarrangements bei akuten lymphoblastischen Leukämien, Non-Hodgkin-Lymphomen und akuten myeloischen Leukämien im Kindesalter. [s.l.]: [s.n.], 1991.

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17

Kay, Lyndsey Sara. Anti-B-cell lymphoma activity mediated by CD3+CD4-CD8- T cells activated in vitro or in vivo. Ottawa: National Library of Canada, 2003.

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18

Constandinou, Christothea Maria. Clinical phenotypic aspects of T-cell lymphomas and Hodgkin's disease,its association with the Epstein-Barr virus, and its influence on the immune response. Wolverhampton: University of Wolverhampton, 2002.

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19

Collins, Graham, and Chris Bunch. Lymphoma. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0289.

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Lymphoma is a cancerous disorder characterized by a clonal proliferation of lymphocytes. There are two broad categories: Hodgkin’s lymphoma, and non-Hodgkin’s lymphoma, with Hodgkin’s lymphoma defined by the presence of Reed–Sternberg cells on histological examination of affected tissue. Within the non-Hodgkin’s lymphomas, there are the much more common B-cell lymphomas and the uncommon T-cell lymphomas. Within the B-cell non-Hodgkin lymphomas, there are clinically aggressive (high-grade) forms and much more indolent (low-grade) forms. This chapter addresses the causes, diagnosis, and management of Hodgkin’s lymphoma and non-Hodgkin’s lymphoma.

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20

Foss, Francine. T-Cell Lymphomas. Humana Press, 2012.

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21

Foss, Francine. T-Cell Lymphomas. Humana, 2016.

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22

Foss, Francine. T-Cell Lymphomas. Humana Press, 2012.

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23

Batchelor, Tracy T., Oussama Abla, Zhong-ping Chen, Dennis C. Shrieve, and Samar Issa. Tumours of the haematopoietic system. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0013.

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‘Tumours of the haematopoietic system’ examines the epidemiology, the pathogenesis, and the clinical features of adult and childhood primary central nervous system lymphomas (PCNSLs), extranodal forms of non-Hodgkin lymphoma, as well as the histiocytoses included in the World Health Organization (WHO) classification of central nervous system (CNS) tumours. It reviews these features in the most common PCNSL, primary central nervous system diffuse large B-cell lymphoma, as well as the other rare histopathological PCNSL variants including lymphomatoid granulomatosis, T-cell lymphoma, anaplastic large T-cell lymphoma, natural killer/T-cell lymphoma, low-grade lymphoma, mucosa-associated lymphoid tissue (MALT) of the dura, and Hodgkin lymphoma. The chapter also discusses clinical and anatomical PCNSL variants including vitreoretinal lymphoma, leptomeningeal lymphoma, intramedullary spinal cord lymphoma, intravascular lymphoma, and PCNSL in the immunocompromised host. It also reviews the CNS presentations of Langerhans cell histiocytosis and the following non-Langerhans cell histiocytoses: Erdheim–Chester disease, Rosai–Dorfman disease, juvenile xanthogranuloma, and histiocytic sarcoma. It is written for specialists and non-specialists managing these various conditions.

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24

Tcell Lymphomas. Humana Press, 2012.

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25

Carton, James. Haematopathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0015.

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This chapter discusses haematopathology, including iron deficiency anaemia, anaemia of chronic disease, megaloblastic anaemias, hereditary spherocytosis, glucose-6-phosphate dehydrogenase deficiency, thalassaemias, sickle-cell disorders, idiopathic thrombocytopenic purpura (ITP), thrombotic thrombocytopenic purpura (TTP), von Willebrand disease, haemophilia, thrombophilia, acute B-lymphoblastic leukaemia, acute myeloid leukaemias, chronic lymphocytic leukaemia (CLL), chronic myelogenous leukaemia, polycythaemia vera (PV), essential thrombocythaemia (ET), primary myelofibrosis (PMF), myelodysplastic syndromes (MDS), follicular lymphoma, diffuse large B-cell lymphoma, Burkitt’s lymphoma (BL), extranodal marginal zone lymphoma of mucosa-associated lymphoid tissue (MALT lymphoma), mantle cell lymphoma, classical Hodgkin’s lymphoma (cHL), lymphoplasmacytic lymphoma (LPL), plasma cell myeloma, primary amyloidosis, and mature T-cell non-Hodgkin’s lymphomas.

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26

William, Basem M., and Catherine Grace Chung, eds. Cutaneous T-cell Lymphomas. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-589-1.

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27

Paolo Piccaluga, Pier, ed. Peripheral T-cell Lymphomas. IntechOpen, 2019. http://dx.doi.org/10.5772/intechopen.73947.

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28

Piccaluga, Pier Paolo. Peripheral T-Cell Lymphomas. IntechOpen, 2019.

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29

O′Connor, Owen A., Won Seog Kim, and Pier L. Zinzani. Peripheral T-Cell Lymphomas. Wiley & Sons, Limited, John, 2020.

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30

O'Connor, Owen A., Won Seog Kim, and Pier L. Zinzani. Peripheral T-Cell Lymphomas. Wiley & Sons, Limited, John, 2021.

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31

O'Connor, Owen A., Won Seog Kim, and Pier L. Zinzani. Peripheral T-Cell Lymphomas. Wiley & Sons, Incorporated, John, 2020.

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32

O'Connor, Owen A., Won Seog Kim, and Pier L. Zinzani. Peripheral T-Cell Lymphomas. Wiley & Sons, Incorporated, John, 2020.

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33

O'Connor, Owen A., Won Seog Kim, and Pier Luigi Zinzani, eds. The Peripheral T‐Cell Lymphomas. Wiley, 2021. http://dx.doi.org/10.1002/9781119671336.

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34

Haun, Paul L., and Julia J. Scarisbrick. Diagnosing Cutaneous T-Cell Lymphoma. Karger AG, S., 2016.

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35

LINDA. Fuck Cancer Lymphoma Awareness Lymphoma Awareness T. Independently Published, 2022.

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36

Zackheim, Herschel S., ed. Cutaneous T-Cell Lymphoma. CRC Press, 2004. http://dx.doi.org/10.1201/b14250.

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37

Zackheim, Herschel S. Cutaneous T-Cell Lymphoma. Taylor & Francis Group, 2019.

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38

Watanabe, Toshiki, and Takuya Fukushima. Adult T-cell Leukemia/Lymphoma. Springer, 2018.

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39

Fernandez-flores, Angel. New Research on Cutaneous Lymphomas. Nova Biomedical Books, 2007.

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40

(Adapter), B. Fruet, P. Colling (Adapter), S. Michaelis (Editor), G. Burg (Editor), J. Feit (Editor), and W. Kempf (Editor), eds. Cutaneous Lymphomas: Unusual Cases 2. Steinkopff-Verlag Darmstadt, 2006.

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41

Adult T-cell Leukemia/Lymphoma. Springer, 2017.

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42

Kiel, Universität, ed. Immunphänotyp und Proliferationsverhalten kindlicher T-Zell-Lymphome. 2005.

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43

Cerhan, James R., Claire M. Vajdic, and John J. Spinelli. The Non-Hodgkin Lymphomas. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0040.

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The non-Hodgkin lymphomas (NHL) are a heterogeneous group of over forty lymphoid neoplasms that have undergone a major redefinition over the last twenty-five years, in part due to advances in immunology and genetics as well as implementation of the WHO classification system. NHLs are considered clonal tumors of B-cells, T-cells, or natural killer (NK) cells arrested at various stages of differentiation, regardless of whether they present in the blood (lymphoid leukemia) or lymphoid tissues (lymphoma). In the United States, the age-standardized NHL incidence rate (per 100,000) doubled from 1973 (10.2) to 2004 (21.4) and then stabilized, while five-year relative survival rates improved from 42% in 1973 to 70% in 2004. Established risk factors for NHL or specific NHL subtypes include infectious agents (HTLV-1, HIV, EBV, HHV8, HCV, H. pylori), immune dysregulation (primary immunodeficiency, transplantation, autoimmunity, and immunosuppressive drugs), family history of lymphoma, and common genetic variants identified by genome-wide association studies.

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44

Zhang, Yanming. Interphasezytogenetik bei Patienten mit peripheren T-Zell-Lymphomen. 1993.

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45

Glass, Bertram. Komponentenanalyse präzipitierbarer Immunkomplexe bei peripheren T-Zell-Lymphomen. 1991.

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46

Hjalgrim, Henrik, Ellen T. Chang, and Sally L. Glaser. Hodgkin Lymphoma. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0039.

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Hodgkin lymphoma (HL) is a malignant neoplasm of the lymphatic system. The malignant cell clone derives from germinal center B lymphocytes in ~98% of cases, the rest being of T-lymphocyte origin. Each year, HL is diagnosed in roughly 66,000 individuals worldwide. HL is curable with modern therapy in the vast majority of patients, with five-year survival rates exceeding 90% for early-stage disease. However, so far this excellent prognosis has been achieved at the expense of a high incidence of severe long-term treatment complications such as secondary malignancies, and endocrine and cardiovascular diseases. In affluent Western countries, HL occurrence follows a distinctive and unusual bimodal age distribution, with one incidence peak among adolescents and younger adults and another in older adults. In socioeconomically less affluent populations, in contrast, the adolescent and younger-adult incidence peak is less pronounced, whereas incidence of HL in young boys may be higher than in affluent populations.

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47

Bierwolf, Stephan. Zytomorphologische, Immunhistochemische und Molekulargenetische Untersuchungen von T-Zell-Reichen B-Zell-Lymphomen und Composite Lymphomen. 1997.

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48

Liebsch, Magitta. Untersuchungen zur chromosomalen Instabilität bei peripheren T-Zell-Lymphomen. 1994.

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49

Geha, Raif, and FRED Rosen. Case Studies in Immunology: T-Cell Lymphoma. W.W. Norton & Company, 2010. http://dx.doi.org/10.4324/9780203853610.

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50

Smedby, Karin Ekström, Mads Melbye, and Hans-Olov Adami. Non-Hodgkin Lymphoma. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0027.

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Non-Hodgkin lymphomas (NHL) are a heterogeneous group of malignancies originating from B- or T-lymphocytes and engaging lymphoid tissue. Clinically, NHL subtypes range from chronic indolent to aggressive life-threatening diseases. The incidence of NHL overall increased dramatically worldwide during the latter half of the twentieth century but has now leveled off in many countries. Although some etiologic factors have been identified, most newly diagnosed cases of NHL as well as the previous rise in incidence remain largely unexplained. Well-established risk factors include severe immune suppression following HIV/AIDS and organ transplantation, autoimmune and inflammatory disorders, some infectious agents, and family history. More recently, lifestyle factors have also been linked with certain subtypes of NHL. Through the work of the international InterLymph consortium, several subtype-specific genetic susceptibility variants have also been revealed, promising to shed further light on mechanisms of lymphomagenesis.

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