Relevant bibliographies by topics / Lymphoblasts

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Lymphoblasts'

Author: Grafiati
Published: 4 June 2021
Last updated: 3 February 2022

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Journal articles on the topic "Lymphoblasts"

1

Whitehead, VM, DS Rosenblatt, MJ Vuchich, JJ Shuster, A. Witte, and D. Beaulieu. "Accumulation of methotrexate and methotrexate polyglutamates in lymphoblasts at diagnosis of childhood acute lymphoblastic leukemia: a pilot prognostic factor analysis." Blood 76, no. 1 (1990): 44–49. http://dx.doi.org/10.1182/blood.v76.1.44.44.

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Abstract Lymphoblasts in bone marrow samples, obtained from 43 children with acute lymphoblastic leukemia at diagnosis, were incubated with 1.0 mumols/L [3H] methotrexate for 24 hours in vitro. Nonexchangeable methotrexate and methotrexate polyglutamates were separated and quantitated. Event-free survival at 5 years was 38% +/- 9% for all 43 patients (27 failures), and 44% +/- 10% for the 35 with non-T, non-B- cell acute lymphoblastic leukemia (20 failures). Of these 35 children, those whose lymphoblasts accumulated more than 100 pmol methotrexate and 500 pmol methotrexate polyglutamates per b
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Whitehead, VM, DS Rosenblatt, MJ Vuchich, JJ Shuster, A. Witte, and D. Beaulieu. "Accumulation of methotrexate and methotrexate polyglutamates in lymphoblasts at diagnosis of childhood acute lymphoblastic leukemia: a pilot prognostic factor analysis." Blood 76, no. 1 (1990): 44–49. http://dx.doi.org/10.1182/blood.v76.1.44.bloodjournal76144.

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Lymphoblasts in bone marrow samples, obtained from 43 children with acute lymphoblastic leukemia at diagnosis, were incubated with 1.0 mumols/L [3H] methotrexate for 24 hours in vitro. Nonexchangeable methotrexate and methotrexate polyglutamates were separated and quantitated. Event-free survival at 5 years was 38% +/- 9% for all 43 patients (27 failures), and 44% +/- 10% for the 35 with non-T, non-B- cell acute lymphoblastic leukemia (20 failures). Of these 35 children, those whose lymphoblasts accumulated more than 100 pmol methotrexate and 500 pmol methotrexate polyglutamates per billion ce
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Little, L., M. Alcouloumre, A. M. Drotar, et al. "Properties of N-acetylglucosamine 1-phosphotransferase from human lymphoblasts." Biochemical Journal 248, no. 1 (1987): 151–59. http://dx.doi.org/10.1042/bj2480151.

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Human lymphoblast and fibroblast cell lines from a patient with I-cell disease and normal individuals were characterized with respect to certain properties of UDP-N-acetylglucosamine:lysosomal enzyme precursor N-acetylglucosamine phosphotransferase. The enzyme isolated from normal lymphoblast and fibroblast cell lines expressed similar kinetic properties, substrate specificities and subcellular localizations. Coincident with the severe reduction of N-acetylglucosamine phosphotransferase activity in both I-cell fibroblast and lymphoblast cell lines, there was an increased secretion of several l
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Prokop, Aram, Banu Bagci, Guenaelle Lingfeld, et al. "Sensitivity of Childhood Acute Lymphoblastic Leukemia (ALL) to Idarubicin Is Significantly Higher Than to Daunorubicin Treatment Based on Ex Vivo Apoptosis Induction." Blood 104, no. 11 (2004): 4495. http://dx.doi.org/10.1182/blood.v104.11.4495.4495.

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Abstract Anthracyclines, especially daunorubicin, play a very important role in the treatment of acute lymphoblastic leukemia (ALL) and the relapsed ALL in childhood. In the present study, primary lymphoblasts isolated from 65 children with de novo ALL (median: 5.8 years; range: 1.9 – 16.9 years) and relapsed ALL (median: 12.7 years; range: 1.3 – 17.9 years) were treated with daunorubicin (10 mmol/l) or idarubicin (2 mmol/l) in vitro. We could show that both anthracylines induce apoptosis, as evidenced by measurement of genomic DNA fragmentation. Interestingly, daunorubicin only induced modest
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Katik, Banu, Anja Selig, Janna Velder, et al. "New Pinostilbene Analogues Overcome Anthracycline Resistance in Acute Lymphoplastic Leukemia Ex Vivo." Blood 108, no. 11 (2006): 4403. http://dx.doi.org/10.1182/blood.v108.11.4403.4403.

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Abstract Anthracylines play a very important role in the treatment of acute lymphoblastic leukemia (ALL) and relapsed ALL in childhood, however resistance to anthracyclines leads to a poor prognosis. In the present study, we have synthesized two new pinostilbene analogues, i.e. trans-3,4′-dihydroxy-5-methoxystilbene and (E)-resveratrol 4′-O-ß-D-glucopyranoside, which are able to overcome anthracycline resistance in childhood acute lymphoblastic leukemia (ALL) ex vivo and induce apoptosis in established leukemia cell lines via mitochondrial pathway. Apoptosis induction has been investigated by
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Maslak, P. "Lymphoblasts." ASH Image Bank 2004, no. 0202 (2004): 100992. http://dx.doi.org/10.1182/ashimagebank-2004-100992.

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Whitehead, VM, MJ Vuchich, SJ Lauer, et al. "Accumulation of high levels of methotrexate polyglutamates in lymphoblasts from children with hyperdiploid (greater than 50 chromosomes) B-lineage acute lymphoblastic leukemia: a Pediatric Oncology Group study." Blood 80, no. 5 (1992): 1316–23. http://dx.doi.org/10.1182/blood.v80.5.1316.bloodjournal8051316.

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Hyperdiploidy (greater than 50 chromosomes, or a DNA index greater than 1.16) confers a favorable prognosis in B-lineage acute lymphoblastic leukemia of childhood. Children with B-lineage acute lymphoblastic leukemia whose lymphoblasts at diagnosis accumulate high levels of methotrexate (MTX) and MTX polyglutamates (MTXPGs) in vitro experience a better event-free survival than those whose lymphoblasts do not (Blood 76:44, 1990). Lymphoblasts from 13 children with hyperdiploidy (greater than 50 chromosomes) accumulated high levels of MTX-PGs (1,095 and 571 to 2,346 pmol/10(9) cells [median and
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Schwartz, C. L., C. P. Minniti, P. Harwood, et al. "Elimination of clonogenic malignant human T cells using monoclonal antibodies in combination with 2'-deoxycoformycin." Journal of Clinical Oncology 5, no. 12 (1987): 1900–1911. http://dx.doi.org/10.1200/jco.1987.5.12.1900.

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2'Deoxycoformycin (dCF) specifically inhibits adenosine deaminase (ADA) and causes selective cytotoxicity of normal and malignant T cells. In clinical trials, dCF caused rapid lysis of malignant T lymphoblasts. Although dCF has been associated with dose-limiting nonhematopoietic toxicities, myelosuppression has not been observed. Since dCF is relatively nontoxic to hematopoietic stem cells, we tested dCF for utility in the ex vivo purging of malignant T lymphoblasts from remission leukemic bone marrow for autologous bone marrow transplantation. We found that T lymphoblast cell lines were sensi
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Sandlund, John T., Patricia L. Harrison, Gaston Rivera, et al. "Persistence of lymphoblasts in bone marrow on day 15 and days 22 to 25 of remission induction predicts a dismal treatment outcome in children with acute lymphoblastic leukemia." Blood 100, no. 1 (2002): 43–47. http://dx.doi.org/10.1182/blood.v100.1.43.

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Abstract We determined the prognostic importance of morphologically identifiable persistent disease at day 15 and days 22 to 25 of remission induction in childhood acute lymphoblastic leukemia (ALL). Among 546 patients entered on 2 consecutive protocols, 397 patients had evaluable bone marrow (BM) examinations on day 15 (± 1 day) and 218 on days 22 to 25 (± 1 day). Fifty-seven patients (14%) had persistent lymphoblasts (≥ 1%) in the BM on day 15 and 27 patients (5.5%) had persistent lymphoblasts on days 22 to 25. The 5-year event-free survival (EFS) was significantly worse for patients with ly
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Homans, AC, EN Forman, and BE Barker. "Use of monoclonal antibodies to identify cerebrospinal fluid lymphoblasts in children with acute lymphoblastic leukemia." Blood 66, no. 6 (1985): 1321–25. http://dx.doi.org/10.1182/blood.v66.6.1321.1321.

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Abstract The identification of small numbers of leukemic cells in the cerebrospinal fluid (CSF) presents a diagnostic problem in the treatment of children with acute lymphoblastic leukemia (ALL). We adapted a latex sphere rosetting technique to allow us to identify simultaneously cell surface markers and cell morphology in 199 CSF samples from 34 patients and 14 control subjects. In patients without leukemic meningitis, the majority of CSF lymphocytes (69%) were found to be mature T cells positive for OKT11. A much smaller number of cells (8%) were found to be B cells positive for la. In these
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Dissertations / Theses on the topic "Lymphoblasts"

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Murton, R. A. "Mechanisms associated with ethacrynic acid-induced Na'+, K'+ pump upregulation in Epstein-Barr virus-transformed lymphocytes." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244775.

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Wright, Adrienne M. P. "A role for nucleoside transport processes in the cytotoxicity of 2-chlorodeoxyadenosine in leukemic lymphoblasts from children with acute lymphoblastic leukemia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq23091.pdf.

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Antia, Irina J. "The turnover of sodium/potassium pumps in human lymphocytes during upregulation in response to lithium." Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297074.

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Teyssou, Elisa. "Analyses génétiques et fonctionnelles de nouveaux gènes incriminés dans la Sclérose Latérale Amyotrophique (SLA) Genetic analysis of matrin 3 gene in French amyotrophic lateral sclerosis patients and frontotemporal lobar degeneration with amyotrophic lateral sclerosis patients Genetic analysis of CHCHD10 in French familial amyotrophic lateral sclerosis patients." Thesis, Paris 6, 2017. http://www.theses.fr/2017PA066738.

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La Sclérose Latérale Amyotrophique (SLA) est une maladie neurodégénérative fatale caractérisée par la dégénérescence des motoneurones centraux et périphériques. Elle est le plus souvent sporadique (SALS, 90% des cas), tandis que les formes familiales (FALS) représentent 10% des patients. Une vingtaine de gènes liés à la SLA ont été identifiés et sont responsables de 70% des FALS et 10% des SALS. Le but de ce projet était d’étudier la contribution de 6 gènes rares dans une large cohorte de patients français atteints de SLA et d’étudier les conséquences fonctionnelles de certains variants identi
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Zborovszky, Camila Carolina. "Mitochondrial complex I functionality and protein oxidative damage in lymphoblasts from lithium responsive patients with bipolar disorder, affected and unaffected relatives." Thesis, University of British Columbia, 2011. http://hdl.handle.net/2429/33274.

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Background: Evidence has demonstrated that mitochondria underline the neurobiology of bipolar disorder (BD). Investigating complex I functionality in transformed lymphoblasts from lithium responsive patients with BD, its relation with increased oxidative damage, and the response to stress induced by low glucose treatment, will aid in the search for peripheral biomarkers for this debilitating disorder. Objective: Confirm the alterations in complex I activity as well as levels of its subunit, NDUFS7, in addition to oxidative damage to mitochondrial proteins, by increased levels of protein carb
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Kuchinskaya, Ekaterina. "Genetic studies of acute lymphoblastic leukemia /." Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-337-5/.

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Pettit, Andrew I. "Oxygen radical generation by lymphoblast NADPH oxidase in hypertension." Thesis, University of Leicester, 2006. http://hdl.handle.net/2381/29519.

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Objectives. The aim of this thesis was to investigate increased reactive oxygen species production originating from NADPH oxidase in lymphoblasts from hypertensive subjects. Results. Combined intra and extracellular stimulated reactive oxygen species production was increased in hypertensive cell lines. The ROS production was abolished by Diphenyleneiodonium chloride but not by rotenone, indicating that a non-mitochondrial flavoprotein, such as NADPH oxidase, was involved. Analysis of NADPH oxidase subcomponents revealed an increase in p22phox in lymphoblasts from hypertensive subjects, account
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Cartwright, Cher Suzanne. "Thiopurine Metabolism in Childhood Acute Lymphoblastic Leukaemia." Thesis, University of Sheffield, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.500442.

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Lo, Tony Chung Tung. "Inactivation of SHIP1 in acute lymphoblastic leukemia." Diss., [La Jolla] : University of California, San Diego, 2009. http://wwwlib.umi.com/cr/ucsd/fullcit?p1465620.

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Thesis (M.S.)--University of California, San Diego, 2009.<br>Title from first page of PDF file (viewed July 22, 2009). Available via ProQuest Digital Dissertations. Includes bibliographical references (p. 63-69).
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Kay, Gillian. "Characterisation of the lymphoblast β-adrenergic receptor in bipolar depression". Thesis, Imperial College London, 1991. http://hdl.handle.net/10044/1/46859.

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Books on the topic "Lymphoblasts"

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Vora, Ajay, ed. Childhood Acute Lymphoblastic Leukemia. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-39708-5.

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Kato, Motohiro, ed. Pediatric Acute Lymphoblastic Leukemia. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-0548-5.

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Pullarkat, Vinod. Contemporary management of acute lymphoblastic leukemia. Jaypee Brothers Medical Publishers (P) Ltd, 2014.

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Vecchione, Severo. Acute lymphoblastic leukemia: Etiology, pathogenesis, and treatments. Nova Science Publishers, 2011.

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Secker-Walker, Lorna M. Chromosomes and genes in acute lymphoblastic leukemia. Springer, 1997.

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Saha, Vaskar, and Pamela Kearns. New agents for the treatment of acute lymphoblastic leukemia. Springer, 2011.

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Saha, Vaskar, and Pamela Kearns, eds. New Agents for the Treatment of Acute Lymphoblastic Leukemia. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-8459-3.

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Raybould, Simon. Spatial analysis of acute lymphoblastic leukaemia in Tyne and Wear. University of Newcastle upon Tyne Centre for Urban and Regional Development Studies, 1987.

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Raybould, Simon. Spatial analysis of acute lymphoblastic leukaemia in Tyne and Wear. University of Newcastle upon Tyne Centre for Urban and Regional Development Studies, 1987.

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Peter, Gale Robert, and Hoelzer Dieter, eds. Acute lymphoblastic leukemia: Proceedings of a Wyeth-Ayerst-UCLA Western Workshop--Acute Lymphoblastic Leukemia, held at Tapatio Springs, Texas, November 29-December 2, 1988. Wiley-Liss, 1990.

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Book chapters on the topic "Lymphoblasts"

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Carson, Dennis A., Carlos J. Carrera, Masaru Kubota, D. Bruce Wasson, and Taizo Iizasa. "Genetic Analysis of Deoxyadenosine Toxicity in Dividing Human Lymphoblasts." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-1248-2_31.

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Nuki, G., K. Astrini, D. Brenton, M. Cruikshank, J. Lever, and J. E. Seegmiller. "Purine and Pyrimidine Nucleotides in Some Mutant Human Lymphoblasts." In Ciba Foundation Symposium 48 - Purine and Pyrimidine Metabolism. John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720301.ch9.

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Schmitt-Gräff, Annette, and Giulio Gabbiani. "Cytoskeletal Organization of Normal and Leukemic Lymphocytes and Lymphoblasts." In Blood Cell Biochemistry. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3796-0_3.

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Yamada, Yasukazu, Haruko Goto, and Nobuaki Ogasawara. "Phosphorylation of Purine Deoxynucleosides in Human T- and B-Lymphoblasts." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5104-7_89.

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Vorwerk, P., J. Elsner, K. Mohnike, Y. Oh, R. G. Rosenfeld, and U. Mittler. "Expression of IGFBP-rP2 / CTGF is Specific for Malignant Lymphoblasts of Children with Acute Lymphoblastic Leucemia (ALL)." In Haematology and Blood Transfusion / Hämatologie und Bluttransfusion. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-18156-6_15.

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Boss, Gerry R. "Cobalamin Inactivation Decreases Purine and Methionine Synthesis in Cultured Human Lymphoblasts." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-1248-2_99.

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Barankiewicz, J., R. Jimenez, J. Uyesaka, and H. E. Gruber. "Regulation of Adenosine Concentrations by Acadesine (Aica-Riboside) in Human B-Lymphoblasts." In Advances in Experimental Medicine and Biology. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4899-2638-8_62.

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Zhao, Ke-Wei, and Arnold L. Miller. "Purification of the N-Acetylglucosamine-1-Phosphodiester α-N-Acetylglucosamindase from Human Lymphoblasts." In Molecular Mechanisms of Membrane Traffic. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-02928-2_76.

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Edwards, N. Lawrence, Annette M. Zaytoun, and Gail A. Renard. "Separate Mechanisms for Cellular uptake of Purine Nucleotides by B- and T-Lymphoblasts." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-1248-2_72.

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Pilz, Renate B., and Gerry R. Boss. "The Synthesis of Phosphoribosylpyrophosphate from Glucose Decreases During Amino Acid Starvation of Human Lymphoblasts." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-1248-2_95.

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Conference papers on the topic "Lymphoblasts"

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Gupta, Lalit, Sanjay Jayavanth, and Aruna Ramaiah. "Identification of different types of lymphoblasts in acute lymphoblastic leukemia using relevance vector machines." In 2009 31st Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5334016.

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Gabler, C., T. Hieronymus, N. Blank, et al. "OP0073 Histone release into the cytoplasm of human pbmcs and preapoptotic lymphoblasts." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.1237.

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Rocchi, Emmanuelle, Jean Vigo, Pierre M. Viallet, and Jean-Marie Salmon. "Multiparametric investigations for studying the time-dependence of the effect of Adriamycin on some human leukemic lymphoblasts using multiwavelength videomicrofluorometry." In BiOS Europe '96, edited by Irving J. Bigio, Warren S. Grundfest, Herbert Schneckenburger, Katarina Svanberg, and Pierre M. Viallet. SPIE, 1996. http://dx.doi.org/10.1117/12.260827.

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Leclerc, Gilles M., Guy J. Leclerc, Jeffim N. Kuznetsov, and Julio C. Barredo. "Abstract 2736: PIM2 is up-regulated in response to metformin-induced cell death triggered by ER stress/UPR in ALL lymphoblasts." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2736.

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Safuan, Syadia Nabilah Mohd, Mohd Razali Md Tomari, Wan Nurshazwani Wan Zakaria, Nurmiza Othman, and Nor Surayahani Suriani. "Computer Aided System (CAS) Of Lymphoblast Classification For Acute Lymphoblastic Leukemia (ALL) Detection Using Various Pre-Trained Models." In 2020 IEEE Student Conference on Research and Development (SCOReD). IEEE, 2020. http://dx.doi.org/10.1109/scored50371.2020.9251000.

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Tsomartova, Dibakhan Aslanbekovna, Nataliya Valentinovna Yaglova, and Sergey Stanislavovich Obernikhin. "ALTERED PROLIFERATION OF THYMIC LYMPHOCYTES IN PUBERTAL RATS EXPOSED TO LOW DOSES OF DDT." In International conference New technologies in medicine, biology, pharmacology and ecology (NT +M&Ec ' 2020). Institute of information technology, 2020. http://dx.doi.org/10.47501/978-5-6044060-0-7.14.

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Low-dose developmental exposure to DDT alters proliferative activity of thymic lymphocytes of rats. Higher proliferation rate and low differentiated lymphoblast content are found in rat thymus during puberty.
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Suapang, Piyamas, Kathtaliya Pumjaroen, and Prasong Tosranon. "Segmentation of Acute Lymphoblastic Leukemia." In International Conference on Industrial Application Engineering 2020. The Institute of Industrial Applications Engineers, 2020. http://dx.doi.org/10.12792/iciae2020.021.

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Raasch, S., A. Bronsema, J. Müller, et al. "FOXM1 haploinsufficiency in acute lymphoblastic leukemia." In 30. Jahrestagung der Kind-Philipp-Stiftung für pädiatrisch-onkologische Forschung. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1602194.

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Kassani, Sara Hosseinzadeh, Peyman Hosseinzadeh Kassani, Michal J. Wesolowski, Kevin A. Schneider, and Ralph Deters. "A Hybrid Deep Learning Architecture for Leukemic B-lymphoblast Classification." In 2019 International Conference on Information and Communication Technology Convergence (ICTC). IEEE, 2019. http://dx.doi.org/10.1109/ictc46691.2019.8939959.

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Thomas, Blessy, R. Harshitha, and A. Rafega Beham. "A Novel Approach to Detect Acute Lymphoblastic Leukemia." In 2018 3rd IEEE International Conference on Recent Trends in Electronics, Information & Communication Technology (RTEICT). IEEE, 2018. http://dx.doi.org/10.1109/rteict42901.2018.9012461.

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Reports on the topic "Lymphoblasts"

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Shaw, Collin. Increased Glutathione Metabolic Defense Capabilities in Cultured Alzheimer's Diseased Lymphoblast Cell Lines. Portland State University Library, 2000. http://dx.doi.org/10.15760/etd.1702.

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Ferrando, Adolfo A. Targeting Class I PI3Ks in the Treatment of T-cell Acute Lymphoblastic Leukemia. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada591435.

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Brinker, C. Jeffrey, and Mekensey Buley. Identification and display of CRLF2 ligands for targeted nanoparticle delivery to acute lymphoblastic leukemia. Office of Scientific and Technical Information (OSTI), 2012. http://dx.doi.org/10.2172/1051698.

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Xiang, Qi, and Yuping Zhang. The Influence of pegaspase on coagulation function and remission rate in adult patients with acute lymphoblastic leukemia. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.7.0016.

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Ponvilawan, Ben, Pongthep Vittayawacharin, Pattaraporn Tunsing, and Weerapat Owattanapanich. Efficacy of Targeted Immunotherapy as Induction or Salvage Therapy in Acute Lymphoblastic Leukemia: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.7.0011.

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