Academic literature on the topic 'Lupus érythémateus aigu disséminé'
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Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Lupus érythémateus aigu disséminé.'
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Journal articles on the topic "Lupus érythémateus aigu disséminé"
Agoumi, S., B. Himdi, K. Abidi, A. Zeggwagh, and R. Abouqal. "Pancréatite aiguë révélatrice d'un lupus érythémateux aigu disséminé." La Revue de Médecine Interne 27, no. 10 (October 2006): 799–802. http://dx.doi.org/10.1016/j.revmed.2006.05.012.
Full textRouty, J. P., F. Zarrouk, B. Taillan, and A. P. Blanc. "L'hypertriglycéridémie du lupus érythémateux aigu disséminé." La Revue de Médecine Interne 7, no. 2 (March 1986): 174–76. http://dx.doi.org/10.1016/s0248-8663(86)80110-2.
Full textFaure, I., ME Lafon, JF Viallard, P. Mercié, JL Pellegrin, and B. Leng. "Epstein-Barr virus et lupus érythémateux aigu disséminé." La Revue de Médecine Interne 19 (June 1998): 131S. http://dx.doi.org/10.1016/s0248-8663(98)80213-0.
Full textEn−niya, F., J. Hind, D. Jean Pierre, M. Elbelrhiti, T. Bouattar, L. Benamar, N. Ouzeddoune, and R. Bayahia. "Amylose rénale : complication rare du lupus érythémateux aigu disséminé." Néphrologie & Thérapeutique 13, no. 5 (September 2017): 347. http://dx.doi.org/10.1016/j.nephro.2017.08.186.
Full textLe Loet, X., F. Pouyol, O. Lees, T. Ducastelle, M. Godin, and P. Deshayes. "Lupus érythémateux aigu disséminé survenu après thymectomie pour myasthénie." La Revue de Médecine Interne 7, no. 4 (September 1986): 425–32. http://dx.doi.org/10.1016/s0248-8663(86)80134-5.
Full textCoutrey, L., N. Magontier, S. Cantagrel, A. Chergui, P. Neville, M. C. Vaillant, F. Despert, and A. Chantepie. "Tamponade cardiaque révélant un lupus érythémateux aigu disséminé chez l'enfant." Archives de Pédiatrie 6 (January 1999): S562. http://dx.doi.org/10.1016/s0929-693x(99)81721-7.
Full textBenjelloun, O., L. Aitmhammed, K. Bouayed, and N. Mikou. "P136 - Lupus aigu érythémateux disséminé pédiatrique : à propos de 17 cas." Archives de Pédiatrie 17, no. 6 (June 2010): 84. http://dx.doi.org/10.1016/s0929-693x(10)70536-4.
Full textCastanet, J., B. Taillan, G. Garnier, J. G. Fuzibet, H. Vinti, and P. Dujardin. "Syndrome de sécrétion inappropriée d'A.D.H. au cours d'un lupus érythémateux aigu disséminé." La Revue de Médecine Interne 13, no. 7 (December 1992): S433. http://dx.doi.org/10.1016/s0248-8663(05)81012-4.
Full textGauthier-Chelle, K., P. Morlat, P. Mercié, M. Beylot-Bany, MS Doutre, A. Baquey, N. Bernard, et al. "Encéphalopathie rapidement fatale inaugurale d'un lupus érythémateux aigu disséminé avec anticorps antiribosomes et antigangliosides." La Revue de Médecine Interne 19 (June 1998): 129S. http://dx.doi.org/10.1016/s0248-8663(98)80208-7.
Full textBlanco, P., A. K. Palucka, V. Pascual, and J. Banchereau. "Rôle de l'interféron-alpha et des cellules dendritiques dans le lupus érythémateux aigu disséminé." La Revue de Médecine Interne 22 (December 2001): 464s. http://dx.doi.org/10.1016/s0248-8663(01)80095-3.
Full textDissertations / Theses on the topic "Lupus érythémateus aigu disséminé"
Jouan, Dominique. "Les manifestations ophtalmologiques du lupus érhytémateux aigu disséminé." Caen, 1990. http://www.theses.fr/1990CAEN3075.
Full textTrebeden-Negre, Hélène. "Modulation de la réponse auto-immune et anti-tumorale par l'injection de cellules apoptotiques." Paris 5, 2002. http://www.theses.fr/2002PA05N079.
Full textWe investigated whether the increased rate of lymphocyte apoptosis in systemic lupus erythematosus can trigger the onset of the disease. Only injection of apoptotic B cells accelerates the onset of the lupus. We observe an increase in the Th2 response directed against the histone H2A after injection of apoptotic B cells. This phenomenon can be explained by a preferential expression of the histone H2A by B cells and the histone H3 by T cells. We also analysed tumor cells apoptosis implication in the anti-tumoral immune response. Injection of two million apoptotic cells to BALB / c mice induces a Th2 antiinflammatory response through complement dependent cytotoxicity
Nathou, Marie-Odile. "Le lupus érythemateux disséminé en Guadeloupe : étude rétrospective à propos de 81 cas." Bordeaux 2, 1995. http://www.theses.fr/1995BOR2M030.
Full textGRE, JEROME. "Le lupus erythemateux dissemine familial : a partir de deux observations cliniques." Bordeaux 2, 1988. http://www.theses.fr/1988BOR25151.
Full textChazal, Jean-Louis. "Manisfestations neuro-psychiques du lupus érythémateux disséminé : à propos d' une observation." Clermont-Ferrand 1, 1987. http://www.theses.fr/1987CLF11032.
Full textJanah, Taoufik. "Sur quelques modes de réponses psychologiques et psychopathologiques à la maladie organique chronique chez l'adulte : cas du lupus érythémateux disséminé systémique (la maladie lupique) : esquisse d'une approche somatopsychologique." Paris 13, 1993. http://www.theses.fr/1993PA131032.
Full textMeillon, Paul. "La myélofibrose au cours du lupus érythémateux aigu disséminé : à propos d'un cas." Bordeaux 2, 1996. http://www.theses.fr/1996BOR2M018.
Full textBoisvert, Jean. "Lupus et pancréatites : à propos de trois observations." Bordeaux 2, 1995. http://www.theses.fr/1995BOR2M078.
Full textPlantin-Carrenard, Emmanuelle. "Thalidomide et lupus érythémateux cutanés." Paris 5, 1996. http://www.theses.fr/1996PA05P113.
Full textSchickel, Jean-Nicolas. "Lupus érythémateux disséminé et sous-expression de Carabin et Sh3kbp1 : étude par génomique fonctionnelle." Strasbourg, 2011. https://publication-theses.unistra.fr/public/theses_doctorat/2011/SCHICKEL_Jean-Nicolas_2011_ED414.pdf.
Full text(Carabin) and SH3KBP1. The aim of this thesis project was to precise the consequences of Carabin and Sh3kbp1 underexpression in B cell function and in the development of autoimmunity. To address those issues, we produced: 1/ Carabin and Sh3kbp1 knock-down (KD) B cells and studied their phenotype; 2) a knock-out (KO) and conditional KO of Carabin in B cells or in mature B or T cells. Our results show that Carabin deficiency leads to an increase in T and B cell activation after TCR and BCR stimulation, respectively. Moreover Carabin KO mice and B cell conditional KO mice shows an accelerated T-dependant and T-independent antigen-specific B cell response in vivo. Finally, Carabin KO mice develop signs of autoimmunity after CpG treatment characterized by sustained production of anti-DNA IgG as well as an important deposition of IgG in renal glomeruli. Altogether these results define a new role for Carabin as a negative regulator of B cell signaling that points out a new defective biological pathway in autoimmunity. For Sh3kbp1, we show an acceleration of Erk and Akt phosphorylation in Sh3kbp1 KD B cells after BCR engagement, showing a role for Sh3kbp1 as a negative regulator of B cell receptor signaling. In conclusion, we have identified two genes that are deregulated in B cells during SLE. A deficiency in one of these two genes speed up the B cells response and predispose for the development of autoimmunity in vivo. Further experiments could potentially identify these two genes as new susceptibility genes in SLE