Relevant bibliographies by topics / Lupinosis

Academic literature on the topic 'Lupinosis'

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Published: 10 December 2022
Last updated: 29 January 2023

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Journal articles on the topic "Lupinosis"

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MACKIE, JT, RS RAHALEY, and R. BENNETT. "Lupinosis in yearling cattle." Australian Veterinary Journal 69, no. 7 (July 1992): 172–73. http://dx.doi.org/10.1111/j.1751-0813.1992.tb07507.x.

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White, CL, DG Masters, DI Paynter, JMC Howell, SP Roe, MJ Barnes, and JG Allen. "The effects of supplementary copper and a mineral mix on the development of lupinosis in sheep." Australian Journal of Agricultural Research 45, no. 2 (1994): 279. http://dx.doi.org/10.1071/ar9940279.

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Lupinosis in grazing sheep is often associated with a potentially deleterious increase in the concentration of copper in the liver. Siromin�, a mineral mix developed by CSIRO for sheep on dry herbage and containing Cu at 116 mg/kg, was tested for its suitability for use with sheep consuming toxic lupin stubble, taking particular regard for dangers of Cu toxicity. There were three dietary treatments applied at two levels (either present or absent) to 40 Merino wethers for 8 weeks. Treatments consisted of adding Cu (10 mg Cu as CuS04/kg), minerals (25 g/kg as Siromin�) and toxic lupin stubble (50 g/kg) to a basal diet consisting of oaten hay and lupin seed and containing 3 mg/kg of Cu. Toxic lupin stubble decreased feed intake and growth, increased plasma activities of liver enzymes and increased plasma concentrations of protein, globulin, bilirubin, Cu and Zn. It also decreased concentrations of Zn and Fe in liver, but had no effect on Cu. The mineral mix had no adverse effects on any signs of lupinosis, and it did not increase liver Cu concentration at either level of dietary Cu. The mineral mix prevented the inappetence caused by lupinosis in sheep fed the low Cu basal diet. Cu treatment resulted in increased concentrations of Cu and Fe in liver, and exacerbated some signs of lupinosis. The results show that the mineral mix is safe to feed to sheep grazing toxic lupin stubble, and it may provide a practical means of supplying additional Zn. The exacerbation of signs of lupinosis by the addition of only 10 mg Cu/kg as CuS04 suggests that under field conditions the supply of Cu in the absence of suitable amounts of molybdenum and sulfur should be kept to a minimum.
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Peterson, Carl. "Development of a lupinosis vaccine." Australian Veterinary Journal 77, no. 1 (January 1999): 47. http://dx.doi.org/10.1111/j.1751-0813.1999.tb12429.x.

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Barnes, A. L., K. P. Croker, J. G. Allen, and N. D. Costa. "Lupinosis and reproduction reduce the wool growth of Merino ewes." Australian Journal of Experimental Agriculture 37, no. 5 (1997): 515. http://dx.doi.org/10.1071/ea97004.

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Summary. Lupinosis was induced in Merino ewes by subcutaneous injections of phomopsins. Liver damage and impairment of liver function was measured by increases in plasma activities of glutamate dehydrogenase and gamma glutamyl transferase, plasma concentrations of bilirubin, and plasma clearance of bromosulfthalein. The wool growth of the ewes during and after exposure to phomopsins at different periods relative to mating was measured, and the impact of lupinosis on annual wool production assessed. Phomopsin administration decreased the length of staple grown during, and for at least 6 weeks after, exposure to phomopsins. Mean fibre diameter of wool grown during this time was also reduced. Annual wool production of the ewes was affected by exposure to phomopsins, with effects noted on fleece weight, yield, fibre diameter, strength and position of break. These effects were minor and varied between experiments. The adverse effects of reproduction on annual wool production were more significant than those of phomopsins.
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PEET, RL, TM ELLIS, and KG ELLIOT. "Adenoviral intranuclear inclusions in lupinosis-damaged sheep livers." Australian Veterinary Journal 67, no. 5 (May 1990): 195. http://dx.doi.org/10.1111/j.1751-0813.1990.tb07756.x.

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Barnes, AL, KP Croker, JG Allen, and ND Costa. "Lupinosis of ewes around the time of mating reduces reproductive performance." Australian Journal of Agricultural Research 47, no. 8 (1996): 1305. http://dx.doi.org/10.1071/ar9961305.

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Lupinosis was induced in Merino ewes by subcutaneous injections of phomopsin toxins, given for 2 weeks before and up to 2 weeks after the start of joining, and the reproductive performance of the injected ewes was compared with untreated control ewes. Two experiments were conducted in consecutive years, with similar results. The liver damage and impairment of liver function due to lupinosis were measured by increases in plasma activities of glutamate dehydrogenase and gamma glutamyl transferase, plasma concentration of bilirubin, and plasma clearance of bromosulfthalein. Ovulation rate was depressed by phomopsin administration (1.11 v. 1.33 for the combined phomopsin-treated and control ewes, P < 0.001), and fewer phomopsin-treated ewes were pregnant (70.4% for the combined phomopsin-treated ewes v. 93.8% for the combined control ewes, P < 0.01). Thus, fewer lambs were born to the phomopsin-treated ewes compared with the control ewes (66.0 v. 109.3% for the combined years, P < 0.01). These results indicate that there may be severe detrimental effects of phomopsin intoxication on reproductive performance of ewes.
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Morcombe, PW, KP Croker, and JG Allen. "Liver damage and the liveweight changes in Merino wether weaners grazing mixed crops of oats and sweet narrow-leafed lupins." Australian Journal of Experimental Agriculture 27, no. 1 (1987): 19. http://dx.doi.org/10.1071/ea9870019.

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Merino wether weaners were grazed on mixed crops of oats and sweet lupins at 3 stocking rates (15, 30 and 60 sheep/ha), pure lupins (60 sheep/ha) or pastures (7.5 sheep/ha) with and without an ad libitum ration of an oat-lupin grain mix in 2 experiments. Changes in liveweights and liver damage due to lupinosis were measured. In experiment 1, when grazing commenced in late January 1982, the weaners grazed on the mixed crops at 15, 30 and 60 sheep/ha gained 4.8 kg over 91 days, 2.9 kg over 43 days and 1.2 kg over 43 days, respectively, while those on the pure lupins gained 0.7 kg over 14 days. These liveweight gains were all less (P<0.05) than the 10 kg increase over 70 days by the weaners on pasture which received the ad libitum supplement of oat-lupin grain (about 2.2% N). Liver damage developed at a slower rate in the sheep on the mixed crops. In experiment 2, when grazing commenced in late November 1983, the weaners grazed on the mixed crops at 15,30 and 60 sheep/ha gained 1 1.3 kg over 70 days, 6.2 kg over 42 days and 3.5 kg over 28 days, respectively, while those on the pure lupins gained 3.4 kg over 28 days. These liveweight gains compared with a 12.5 kg gain over 84 days by the weaners on pasture which received the ad libitum supplement of oat-lupin grain (about 2.60%N). There was less Phomopsis leptostromiformis on the lupin stems in this experiment and the sheep livers were not as badly damaged as in the previous experiment. The development of liver damage was again delayed in the weaners grazed on the mixed crops. It was concluded that Merino wether weaners can increase liveweights when grazed on mixed crops of oats and sweet lupins. The amount of increase depends on the grain available to the sheep and the degree of lupinosis-associated liver damage suffered by the sheep. Grazing mixed crops reduces the development of lupinosis.
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Morcombe, PW, WJ Ryan, and JG Allen. "Sandplain lupins (Lupinus cosentinii) as a summer feed for yearling steers." Australian Journal of Experimental Agriculture 26, no. 1 (1986): 13. http://dx.doi.org/10.1071/ea9860013.

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The response of 9-month-old Angus steers, grazing sandplain lupins during summer and autumn and fed oat and oat plus urea supplements, or grazing hay made from the lupins and fed no supplement, was studied in two experiments. In the first experiment there were 3.3 t DM/ha present at the start of grazing. Average daily weight changes over 84 days were -0.548, -0.274 and 0.333 kg/day (P<0.05) for steers fed 0, 1 and 2 kg oats/steer.day respectively. The steers grazing lupin hay averaged -0.417 kg/day. There were 8.4 t DM/ha present at the start of the second experiment and the daily weight changes by steers fed 0, 2 and 6 kg oats plus urea/head.day were - 0.012, 0.524 and 0.952 kg/day (P<0.05), respectively. The steers grazing lupin hay gained 0.215 kg/day. No obvious clinical signs of lupinosis were observed in either experiment. However, in the first experiment the lupins were toxic, liver damage occurred and lupinosis may have contributed to the lower growth rates. In both experiments steers not fed a supplement made compensatory weight gains during winter and spring, when all steers were run together on mixed clover-grass pasture.
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Williamson, P. M., A. S. Highet, W. Gams, K. Sivasithamparam, and W. A. Cowling. "Diaporthe toxica sp. nov., the cause of lupinosis in sheep." Mycological Research 98, no. 12 (December 1994): 1364–68. http://dx.doi.org/10.1016/s0953-7562(09)81064-2.

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CROKER, KP, and JG ALLEN. "Treating lupin stubbles with alkali is unlikely to prevent lupinosis." Australian Veterinary Journal 67, no. 6 (June 1990): 230–32. http://dx.doi.org/10.1111/j.1751-0813.1990.tb07770.x.

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Dissertations / Theses on the topic "Lupinosis"

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Allen, Jeremy George. "Studies of the pathogenesis, toxicology and pathology of lupinosis and associated conditions." Thesis, Allen, Jeremy George (1989) Studies of the pathogenesis, toxicology and pathology of lupinosis and associated conditions. PhD thesis, Murdoch University, 1989. https://researchrepository.murdoch.edu.au/id/eprint/53693/.

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Lupinosis is caused by phomopsins, which are toxic metabolites of the fungus Phomopsis leptostromiformis. This thesis records a series of studies of aspects of the pathogenesis, toxicology and pathology of lupinosis and associated conditions. A sheep bioassay developed for the estimation of phomopsins-associated toxicity in feed samples infected with Phomopsis leptostromiformis is described. Toxicity ratings provided by this bioassay were compared with results provided by a nursling rat bioassay and a chemical assay for phomopsin A. There was a poor correlation between the results from the two bioassays, but results from the sheep bioassay correlated well with those from the chemical assay. The procedure for scoring microscopic liver damage in the sheep bioassay was modified to enable assessment of the severity of lupinosis in sheep grazing toxic lupin stubbles for various periods. Individual sheep were awarded liver injury scores as a measure of the severity of their lupinosis. The microscopic changes assessed had a dynamic interrelationship with each other, and with other parameters including the daily intake of phomopsins and the period over which they were consumed. For this reason liver injury scores could only be used to compare the severity of lupinosis in sheep in individual experiments, and at the same times during experiments. Nevertheless, liver injury scores proved to be a valuable experimental tool and were used in several of the investigations reported in this thesis. The roles of copper and the hepatic microsomal enzymes in the pathogenesis of lupinosis were examined. It was established that copper accumulates in the liver of sheep with subacute and chronic lupinosis. The concentration of copper in the liver was positively correlated with the degree of liver injury. It was also found that the degree of liver injury was correlated positively with the concentration of selenium and negatively with the concentration of zinc. Induction of the hepatic microsomal enzymes with DDT and phenobarbitone, and inhibition with SKF 525A and carbon disulphide, did not alter the toxicity of phomopsin A in rats. However, the toxicity in both males and females was significantly increased when hepatic microsomal enzymes were inhibited by adrenalectomy, and was significantly increased in males only when they were inhibited by cobalt chloride. Toxicological studies established 50% lethal doses for phomopsin A in sheep and pigs following a single subcutaneous administration, and in sheep following a single oral administration and daily oral administrations. Sheep were approximately 40 times more susceptible than pigs to phomopsin A, and in sheep phomopsin A was approximately 65 times more toxic following a single subcutaneous injection rather than a single oral administration. It was demonstrated that only the Biotype A strains of P. leptostromiformis were toxicogenic when grown on autoclaved wheat. The different strains of this biotype had varying potentials to produce phomopsin A. Evidence for a toxic phomopsin additional to phomopsins A and B is presented, and it was referred to as phomopsin C. Aspects of the pathology, clinical chemistry and haematology of experimental phomopsins toxicosis in sheep and rats, which complemented or differed from previous descriptions, are reported. Special attention was given to lupinosis-associated spongy degeneration of the brain and lupinosis-associated myopathy in sheep. Specific experiments conducted to study the pathogenesis and pathology of these conditions are documented.
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Barnes, Anne Louise. "The effect of lupinosis on reproduction and wool growth of the merino ewe." Thesis, Barnes, Anne Louise ORCID: 0000-0002-7227-230X (1993) The effect of lupinosis on reproduction and wool growth of the merino ewe. PhD thesis, Murdoch University, 1993. https://researchrepository.murdoch.edu.au/id/eprint/41883/.

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Lupinosis, a mycotoxicosis caused by phomopsins produced on lupin plants (Lupinus spp.) by Phomopsis leptostromiformis, is a disease of economic importance in W estem Australia. Preliminary studies and anecdotal reports indicated detrimental effects of lupinosis on reproduction and wool growth of ewes. The research described in this thesis was conducted to confirm experimentally the existence and magnitude of these effects on production, and to determine the stages of the reproductive cycle sensitive to phomopsin intoxication. Mice were used to assess the effects of phomopsins on ovulation rate and on the early preimplantation embryo. Administration of phomopsins to female mice before ovulation reduced the number of mice mating and the number of embryos recovered. Culture of two-cell mouse embryos in medium containing phomopsins reduced the number developing to the blastocyst stage, with the embryos most sensitive to the effects of phomopsins before compaction and formation of the blastocoele. Ovulation rate was depressed and wastage of ova was increased by the administration of phomopsins to ewes for 14 days before and 14 days after oestrus, resulting in 39 less lambs produced per 100 ewes than in the control groups. The survival and growth of embryos/foetuses was reduced in ewes injected with phomopsins from approximately day 8 to 58 of pregnancy. Lamb marking rates were 10-20% less from phomopsin-treated ewes than from control ewes. The wool production of ewes was significantly depressed by lupinosis. Length growth rate and mean fibre diameter were reduced after administration of phomopsins, and took up to 69 days to recover. The annual wool growth of the ewes was variable, and pregnancy and lactation reduced the quality and quantity of wool grown. There were reductions in clean fleece weight, yield, mean fibre diameter and staple strength, and changes in position of break attributable to administration of phomopsins. Phomopsin intoxication significantly depressed the reproductive performances of ewes at all stages of the reproductive cycle examined, and reduced the quantity and quality of wool grown by the ewes.
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Rosalino, Nuno Ricardo Tavares Fernandes. "Labirintos Lupinos: Wolfe Traduzido em Contos." Master's thesis, 2017. https://repositorio-aberto.up.pt/handle/10216/108961.

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Rosalino, Nuno Ricardo Tavares Fernandes. "Labirintos Lupinos: Wolfe Traduzido em Contos." Dissertação, 2017. https://repositorio-aberto.up.pt/handle/10216/108961.

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Book chapters on the topic "Lupinosis"

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Aust, Steven D. "Occurrence and clinical manifestations of lupinosis and slaframine toxicosis." In Diagnosis of Mycotoxicoses, 81–90. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4235-6_8.

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Hastie, Amelie. "Lupino’s Familial Milieu." In The Bigamist, 11–15. London: British Film Institute, 2009. http://dx.doi.org/10.1007/978-1-84457-552-7_1.

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"Lupinoside PA1." In Natural Compounds, 517–18. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-0541-2_711.

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"Lupinoside PA2." In Natural Compounds, 518. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-0541-2_712.

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"Lupinoside PA4." In Natural Compounds, 524–25. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-0541-2_723.

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"Lupinoside PA5." In Natural Compounds, 525. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-0541-2_724.

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Hastie, Amelie. "Lupino’s Familial Milieu." In The Bigamist, 11–15. British Film Institute, 2009. http://dx.doi.org/10.5040/9781838713294.ch-001.

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Donnelly, Ashley M. "Ida Lupino’s Moral Filmmaking." In Ida Lupino, Filmmaker. Bloomsbury Academic, 2021. http://dx.doi.org/10.5040/9781501352119.0014.

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Lipscomb, Valerie Barnes. "Ida Lupino’s Manipulation of Age Conventions." In Ida Lupino, Filmmaker. Bloomsbury Academic, 2021. http://dx.doi.org/10.5040/9781501352119.0015.

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Pagnoni Berns, Fernando Gabriel. "Ida Lupino’s Thrillers: The Terror of the “Lethal Woman”." In Ida Lupino, Filmmaker. Bloomsbury Academic, 2021. http://dx.doi.org/10.5040/9781501352119.0022.

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