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Relevant bibliographies by topics / Lung development

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Academic literature on the topic 'Lung development'

Author: Grafiati

Published: 4 June 2021

Last updated: 9 March 2023

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Journal articles on the topic "Lung development"

1

Pronych, Scott, and Richard Wassersug. "Lung use and development in Xenopus laevis tadpoles." Canadian Journal of Zoology 72, no. 4 (April 1, 1994): 738–43. http://dx.doi.org/10.1139/z94-099.

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Shortly after hatching, Xenopus laevis tadpoles fill their lungs with air. We examined the role played by early lung use in these organisms, since they are able to respire with both their lungs and their gills. We investigated the effect on X. laevis development when the larvae were prevented from inflating their lungs, and whether early lung use influenced the size of the lungs or the tadpole's ability to metamorphose. Tadpoles that were denied access to air had lungs one-half the size of those of controls. This difference in lung size was too large to be explained merely by a stretching of the lung due to inflation. The longer tadpoles were denied access to air, the longer they took to metamorphose, and their probability of completing metamorphosis diminished. One tadpole raised throughout its larval life without access to air successfully metamorphosed but had abnormal, solidified lungs and an enlarged heart. Collectively, these experiments demonstrate that early lung use in tadpoles is important in determining both ultimate lung size and the probability of successfully metamorphosing. Lung use during early larval development in X. laevis is not absolutely necessary for survival through metamorphosis, but its absence severely handicaps growth.

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Mullassery, Dhanya, and Nicola P. Smith. "Lung development." Seminars in Pediatric Surgery 24, no. 4 (August 2015): 152–55. http://dx.doi.org/10.1053/j.sempedsurg.2015.01.011.

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Chen, Ling, and Graeme R. Zosky. "Lung development." Photochemical & Photobiological Sciences 16, no. 3 (2017): 339–46. http://dx.doi.org/10.1039/c6pp00278a.

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Recent equivocal results in high profile randomised controlled trials suggest that the impact of vitamin D deficiency on lung development is complex. In this narrative review we summarise our current understanding of the link between UV exposure, vitamin D and lung development.

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Massaro, Donald, and Gloria DeCarlo Massaro. "Lung Development, Lung Function, and Retinoids." New England Journal of Medicine 362, no. 19 (May 13, 2010): 1829–31. http://dx.doi.org/10.1056/nejme1002366.

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Shi, Wei, Saverio Bellusci, and David Warburton. "Lung Development and Adult Lung Diseases." Chest 132, no. 2 (August 2007): 651–56. http://dx.doi.org/10.1378/chest.06-2663.

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Copland, Ian, and Martin Post. "Lung development and fetal lung growth." Paediatric Respiratory Reviews 5 (January 2004): S259—S264. http://dx.doi.org/10.1016/s1526-0542(04)90049-8.

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Stenmark, Kurt R., and Sarah A. Gebb. "Lung Vascular Development." American Journal of Respiratory Cell and Molecular Biology 28, no. 2 (February 2003): 133–37. http://dx.doi.org/10.1165/rcmb.f259.

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Stovin, P. G. "Early lung development." Thorax 40, no. 6 (June 1, 1985): 401–4. http://dx.doi.org/10.1136/thx.40.6.401.

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Shibuya, Soichi, Jessica Allen-Hyttinen, Paolo De Coppi, and Federica Michielin. "In vitro models of fetal lung development to enhance research into congenital lung diseases." Pediatric Surgery International 37, no. 5 (March 31, 2021): 561–68. http://dx.doi.org/10.1007/s00383-021-04864-8.

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AbstractPurposeThis paper aims to build upon previous work to definitively establish in vitro models of murine pseudoglandular stage lung development. These can be easily translated to human fetal lung samples to allow the investigation of lung development in physiologic and pathologic conditions.MethodsLungs were harvested from mouse embryos at E12.5 and cultured in three different settings, i.e., whole lung culture, mesenchyme-free epithelium culture, and organoid culture. For the whole lung culture, extracted lungs were embedded in Matrigel and incubated on permeable filters. Separately, distal epithelial tips were isolated by firstly removing mesothelial and mesenchymal cells, and then severing the tips from the airway tubes. These were then cultured either in branch-promoting or self-renewing conditions.ResultsCultured whole lungs underwent branching morphogenesis similarly to native lungs. Real-time qPCR analysis demonstrated expression of key genes essential for lung bud formation. The culture condition for epithelial tips was optimized by testing different concentrations of FGF10 and CHIR99021 and evaluating branching formation. The epithelial rudiments in self-renewing conditions formed spherical 3D structures with homogeneous Sox9 expression.ConclusionWe report efficient protocols for ex vivo culture systems of pseudoglandular stage mouse embryonic lungs. These models can be applied to human samples and could be useful to paediatric surgeons to investigate normal lung development, understand the pathogenesis of congenital lung diseases, and explore novel therapeutic strategies.

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Wanczyk, Heather, Todd Jensen, Daniel J. Weiss, and Christine Finck. "Advanced single-cell technologies to guide the development of bioengineered lungs." American Journal of Physiology-Lung Cellular and Molecular Physiology 320, no. 6 (June 1, 2021): L1101—L1117. http://dx.doi.org/10.1152/ajplung.00089.2021.

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Lung transplantation remains the only viable option for individuals suffering from end-stage lung failure. However, a number of current limitations exist including a continuing shortage of suitable donor lungs and immune rejection following transplantation. To address these concerns, engineering a decellularized biocompatible lung scaffold from cadavers reseeded with autologous lung cells to promote tissue regeneration is being explored. Proof-of-concept transplantation of these bioengineered lungs into animal models has been accomplished. However, these lungs were incompletely recellularized with resulting epithelial and endothelial leakage and insufficient basement membrane integrity. Failure to repopulate lung scaffolds with all of the distinct cell populations necessary for proper function remains a significant hurdle for the progression of current engineering approaches and precludes clinical translation. Advancements in 3D bioprinting, lung organoid models, and microfluidic device and bioreactor development have enhanced our knowledge of pulmonary lung development, as well as important cell-cell and cell-matrix interactions, all of which will help in the path to a bioengineered transplantable lung. However, a significant gap in knowledge of the spatiotemporal interactions between cell populations as well as relative quantities and localization within each compartment of the lung necessary for its proper growth and function remains. This review will provide an update on cells currently used for reseeding decellularized scaffolds with outcomes of recent lung engineering attempts. Focus will then be on how data obtained from advanced single-cell analyses, coupled with multiomics approaches and high-resolution 3D imaging, can guide current lung bioengineering efforts for the development of fully functional, transplantable lungs.

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Dissertations / Theses on the topic "Lung development"

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Grier, D. G. "Homeobox genes in lung development." Thesis, Queen's University Belfast, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426729.

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Cherukupalli, Kamala. "Studies on the normal and abnormal lung growth in the human and in the rat with emphasis on the connective tissue fibers of the lung." Thesis, University of British Columbia, 1989. http://hdl.handle.net/2429/30607.

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Infants with bronchopulmonary dysplasia (BPD), showed impaired body growth when compared to control infants. In terms of changes in the biochemical composition of the lung, BPD infants had higher DNA, soluble protein, collagen and desmosine contents as well as increased concentrations of DNA, collagen and desmosine in their lungs when compared to the growth patterns obtained for the lungs of control infants. Pathologically BPD was classified into 4 grades. Grade I BPD, was a phase of acute lung injury, grades II and III were proliferative phases. In grade IV BPD, lung structure returned towards normal. Evidence of fibrosis was seen by a significant increase in collagen concentration in grades II and III while desmosine concentration was seen to increase in grades III and IV suggesting that the increase in collagen and desmosine contents in the lungs of BPD infants may be controlled by two different mechanisms. Collagen type I/III ratio was seen to decrease progressively from grade II to grade IV BPD in comparison to age matched controls, indicating a higher proportion of type III collagen in the lungs of infants with BPD. From the clinical analysis and the results obtained from discriminant analysis procedure, it was seen that there was a high degree of correlation between the continuation of the disease and collagen accumulation in the lungs suggesting that pulmonary fibrosis with excessive collagen accumulation is an integral part of BPD. This fibrotic process seemed to correlate significantly with assisted ventilation and high oxygen supplementation received by the infants, but it was difficult to assess the individual contribution of the two treatments in the pathogenesis of BPD. Other variables such as severity of the initial disease and the length of survival of the infants, made the assessment of individual contribution much more difficult.
Medicine, Faculty of
Pathology and Laboratory Medicine, Department of
Graduate

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Gao, Weichen. "Development of human lung query atlas." Thesis, University of Iowa, 2010. https://ir.uiowa.edu/etd/806.

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This thesis reports on our initial work constructing a human lung query atlas which provides clinically relevant population statistics for normal and abnormal individuals. The atlas incorporates front-end interfaces with back-end database. The interfaces were developed using Microsoft Access 2007 and the database was implemented using MySQL. ODBC was used to import database into Access and provide connection for database and interfaces. VBA is used to write SQL queries and realized the interaction with interfaces. SQL queries is written to extract the data which researchers may interest in. The atlas provides measurements of the human airway tree and lung volumes from a population of individuals and also provides a population statistics based on age, race, ethnicity, gender and other information. It also provides functionality for comparing airway measurements between populations, individuals to a population, and individuals to individuals. Statistical significance, such as p-value, is provided to analyze two individuals or populations.

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Tiozzo, Caterina. "Role of Pten in lung development." Doctoral thesis, Università degli studi di Padova, 2009. http://hdl.handle.net/11577/3426411.

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Rationale: Pten is a tumor-suppressor gene, involved in stem cell homeostasis and tumorigenesis. In mouse, Pten expression is ubiquitous and begins as early as 7 days of gestation. Pten-/- mouse embryos die early during gestation indicating a critical role for Pten in embryonic development. Objective: To test the role of Pten in lung development and injury, we conditionally deleted Pten throughout the lung epithelium by crossing Ptenflox/flox with Nkx2.1-cre driver mice and throughout the lung mesenchyme by crossing Ptenflox/flox with Nkx2.1-cre driver or Dermo1-cre driver. The resulting PtenNkx2.1-cre mutants were analyzed for lung defects and response to injury. Results: PtenNkx2.1-cre embryonic lungs showed airway epithelial hyperplasia with no branching abnormalities. In vitro culture of mutant lungs also showed an altered responsed to TGF-? when in vivo In adult mice, PtenNkx2.1-cre lungs exhibit increased progenitor cell pools comprised of basal cells in the trachea, CGRP/CC10 double-positive neuroendocrine cells in the bronchi and CC10/SpC double positive cells in the bronchioalveolar duct junction (BADJ). Pten deletion impacted differentiation of various lung epithelial cell lineages, with decreased number of terminally differentiated cells. Over time, PtenNxk2.1-cre epithelial cells residing in the BADJ underwent proliferation, and formed uniform masses, supporting the concept that the cells residing in this distal niche may also be the source of pro-carcinogenic “stem” cells. Finally, increased progenitor cells in all the lung compartments conferred an overall selective advantage to naphthalene injury compared to wild type control mice. PtenDermo1cre embryonic lungs, moreover, showed normal lung development but increased collagen1 and extracellular matrix production. Conclusions: Pten has a pivotal role in lung stem cell homeostasis, cell differentiation and consequently resistance to lung injury in the epithelium, Further studies are necessary to clarify the real role of Pten in lung mesenchyme.
Rationale: Pten e’ un gene coinvolto nell’omeostasi delle cellule staminali e nella formazione di tumori. Nei topi, Pten inizia ad essere presente 7 giorni dopo il concepimento. Pten ha un ruolo critico nello sviluppo embrionale: gli embrioni di topo Pten-/-, infatti, muoiono molto presto durante la gestazione. Scopo dello studio: Studiare il ruolo di Pten nello sviluppo polmonare, eliminando Pten nell’epitelio polmonare, incrociando Ptenflox/flox con topi portatori di Nkx2.1-cre; Pten e’ stato anche eliminato dal mesenchima polmonare incrociando Ptenflox/flox con topi Dermo1-cre. I risultanti PtenNkx2.1cre sono stati analizzati alla ricerca di difetti nello sviluppo polmonare. Risultati: I polmoni PtenNkx2.1-cre hanno evidenziato in vitro una alterata risposta al TGF-?. In vivo non presentavano nessuna alterazione nel branching bensi una iperplasia polmonare nelle vie aerre. Nei topi adulti, I polmoni PtenNkx2.1-cre presentavano un aumentato pool di cellule progenitori in tutti i distretti: nella trachea, le cellule basali, nei bronchi le cellule neuroepiteliali, positive per CGRP/CC10 ed infine, nella giunzione tra gli alveoli e I bronchi terminali (BADJ), le cellule positive per Spc/CC10. L’assenza di Pten ha un impatto nella differenziazione cellulare, con un diminuito aumento delle cellule all’ultimo stadio di differenziazione. Nel tempo, le cellule epiteliali PtenNxk2.1-cre residenti a livello del BADJ proliferano e formano delle masse di tipo tumorale; questi dati supportano l’idea che le cellule presenti in questa niche possano essere l’origine delle cosidette “pro-carcinogenic stem cells”. L’aumento delle cellule progenitrici, inoltre, conferisce un selettivo vantaggio dopo danno polmonare. I topi con Pten eliminato nell’epitelio, invece, non evidenziavano ne uno sviluppo polmonare alterato ne una alterata differenziazione delle cellule mesenchimali; tuttavia, dimostravano un aumentata deposizione di collagene1 e di matrice extracellulare. Conclusioni: Pten ha un ruolo importante nell’omeostasi delle cellule progenitori del polmone, nella differenziazione epiteliale polmonare e nella resistenza dopo danno. Ulteriori studi sono necessary per chiarire l’esatto ruolo di Pten nel mesenchima polmonare.

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Mattes, Charlott, and Ulrich H. Thome. "Rapid elevation of sodium transport through insulin is mediated by AKT in alveolar cells." Universitätsbibliothek Leipzig, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-142643.

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Alveolar fluid clearance is driven by vectorial Na+ transport and promotes postnatal lung adaptation. The effect of insulin on alveolar epithelial Na+ transport was studied in isolated alveolar cells from 18–19-day gestational age rat fetuses. Equivalent short-circuit currents (ISC) were measured in Ussing chambers and different kinase inhibitors were used to determine the pathway of insulin stimulation. In Western Blot measurements the activation of mediators stimulated by insulin was analyzed. The ISC showed a fast dose-dependent increase by insulin, which could be attributed to an increased ENaC (epithelial Na+ channel) activity in experiments with permeabilized apical or basolateral membrane. 5-(N-Ethyl-N-isopropyl)amiloride inhibition of ISC was not affected, however, benzamil-sensitive ISC was increased in insulin-stimulated monolayers. The application of LY-294002 and Akti1/2 both completely blocked the stimulating effect of insulin on ISC. PP242 partly blocked the effect of insulin, whereas Rapamycin evoked no inhibition. Western Blot measurements revealed an increased phosphorylation of AKT after insulin stimulation. SGK1 activity was also increased by insulin as shown by Western Blot of pNDRG1. However, in Ussing chamber measurements, GSK650394, an inhibitor of SGK1 did not prevent the increase in ISC induced by insulin. The application of IGF-1 mimicked the effect of insulin and increased the ENaC activity. In addition, an increased autophosphorylation of the IGF-1R/IR was observed after insulin stimulation. We conclude that insulin rapidly increases epithelial Na+ transport by enhancing the activity of endogenous ENaC through activation of PI3K/AKT in alveolar cells.

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Salvati, Valentina. "Development of effective lung cancer therapies based on lung cancer stem cella targeting." Doctoral thesis, Università di Catania, 2015. http://hdl.handle.net/10761/4035.

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Il carcinoma polmonare non a piccole cellule (NSCLC) rappresenta circa l 80% di tutti i tumori al polmone ed è il cancro più comune e più mortale al mondo. Il trattamento convenzionale per il NSCLC in stadio avanzato è stato basato per molto tempo sull uso della chemioterapia, ma con basso impatto sulla sopravvivenza . Una migliore comprensione dei meccanismi molecolari coinvolti nel processo di tumorigenesi e una maggiore capacità nell identificazione di specifiche alterazioni genetiche come bersagli terapeutici, hanno portato ad un significativo avanzamento verso lo sviluppo di terapie più efficaci. Il recettore del fattore di crescita dell epidermide (EGFR) è spesso over-espresso nel NSCLC ed è considerato un promettente bersaglio terapeutico per il trattamento di questo tumore. La presenza di mutazioni nel gene EGFR sono un importante predittore di risposta agli inibitori dell EGFR. Sebbene gli inibitori dell EGFR di prima generazione hanno mostrato incoraggianti risposte cliniche nei tumori al polmone, quasi tutti i pazienti sviluppano resistenza al trattamento nel corso del tempo. La resistenza ai trattamenti potrebbe dipendere anche dalla presenza delle cellule staminali tumorali (CSCs), una sottopopolazione di cellule intrinsecamente resistenti. Così, lo studio delle cellule staminali tumorali del polmone, potrebbe essere uno strumento efficace per l identificazione e validazione di bersagli terapeutici innovativi contribuendo all'introduzione di importanti miglioramenti nell ambito dell oncologia clinica. Pertanto, la terapia mirata verso l EGFR continua ad evolvere in seguito alla scoperta della sensibilità agli inibitori tirosin-chinasici da parte di pazienti caratterizzati da mutazioni attivanti del gene EGFR. Tuttavia, circa il 10-20% dei pazienti privi della mutazione dell EGFR, beneficiano anch essi del trattamento con gli inibitori TKIs, suggerendo che potrebbero esistere altri determinanti di risposta al trattamento, indipendenti dalla mutazione del recettore. Questo progetto, quindi, è stato focalizzato sull analisi della via di segnale dell EGFR e sullo studio della sensibilità delle cellule staminali tumorali di polmone e di modelli murini da esse derivati, agli inibitori dell EGFR, al fine di identificare possibili biomarcatori predittivi di risposta agli TKIs, in cellule prive della mutazione dell EGFR. Questo studio ha portato all identificazione della fosforilazione dell EGFR al residuo tirosina 1068, ma non 1173, come potenziale marcatore di risposta all Erlotinib nelle cellule staminali tumorali di polmone e negli xenografts da esse derivati. Inoltre, anche linee cellulari commerciali di polmone sensibili all Erlotinib, esprimevano pEGFR-tyr-1068 indipendentemente dalla mutazione dell EGFR, così, l espressione di pEGFR-tyr1068 nelle cellule staminali tumorali di polmone è risultata essere associata ad una risposta positiva al trattamento con l Erlotinib. La valutazione, mediante immunoistochimica, dello stato di fosforilazione dell EGFR in pazienti con mutazione e senza mutazione del recettore, ha portato a correlare solo pEGFR-tyr1068 e non pEGFR-tyr1173, con la mutazione dell EGFR. In base a questi dati, quindi, è possibile ipotizzare che l identificazione del livello di fosforilazione dell EGFR al residuo tirosina 1068 nei tumori dei pazienti, permetterebbe di individuare tumori con e senza mutazione dell EGFR ma caratterizzati da attivazione del recettore, in grado probabilmente di rispondere in modo positivo al trattamento con l Erlotinib. Questi studi potrebbero avere importanti implicazioni terapeutiche per il trattamento dei tumori al polmone e potrebbero permettere ai pazienti con NSCLC di essere selezionati per terapie più efficaci e meno tossiche.

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Wu, Mau-Ching. "Mouse models of human lung cancer development." Thesis, University of Cambridge, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.624711.

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Nickel, Jacob. "Development of an electronic lung airway atlas." Thesis, University of Iowa, 2008. http://ir.uiowa.edu/etd/14.

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Bonner, Allison E. "Organ development and tumorigenesis: a molecular link." The Ohio State University, 2004. http://rave.ohiolink.edu/etdc/view?acc_num=osu1073936508.

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Bloor, Claire Alexandra. "Insulin-like Growth Factor-1 (IGF-1) axis : role in development of lung fibrosis." Thesis, Keele University, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323715.

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Books on the topic "Lung development"

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C, Gaultier, Bourbon Jacques R, Post Martin 1952-, and American Physiological Society (1887- ), eds. Lung development. New York: Published for the American Physiological Society by Oxford University Press, 1999.

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Gaultier, Claude, Jacques R. Bourbon, and Martin Post, eds. Lung Development. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4614-7537-8.

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1943-, McDonald John A., ed. Lung growth and development. New York: M. Dekker, 1997.

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1932-, Massaro Donald, Massaro Gloria DeCarlo, and Chambon Pierre, eds. Lung development and regeneration. New York: Marcel Dekker, 2004.

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Jobe, Alan, Jeffrey Whitsett, and Steven Abman, eds. Fetal and Neonatal Lung Development. Cambridge: Cambridge University Press, 2016. http://dx.doi.org/10.1017/cbo9781139680349.

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Pinto, M. L. Vitamin A and lung development. Hauppauge, N.Y: Nova Science, 2010.

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Dr, Harding Richard, Pinkerton Kent Ed 1950-, and Plopper Charles G, eds. The lung: Development, aging and the environment. London, UK: Elsevier Academic Press, 2004.

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Soldt, Benjamin Jonathan van. Proximal-distal patterning of the lung: Molecular determinants in lung development and evolution. [New York, N.Y.?]: [publisher not identified], 2020.

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Nikolić, Marko Z., and Brigid L. M. Hogan, eds. Lung Stem Cells in Development, Health and Disease. Sheffield, United Kingdom: European Respiratory Society, 2021. http://dx.doi.org/10.1183/2312508x.erm9121.

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Eduardo, Bancalari, and Polin Richard A. 1945-, eds. The newborn lung. Philadelphia: Saunders/Elsevier, 2008.

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Book chapters on the topic "Lung development"

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Ballard, Philip L. "Lung Development." In Hormones and Lung Maturation, 1–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-82483-8_1.

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Bush, Douglas, Steve H. Abman, and Csaba Galambos. "Lung Development." In Pediatric and Congenital Cardiology, Cardiac Surgery and Intensive Care, 1–16. London: Springer London, 2022. http://dx.doi.org/10.1007/978-1-4471-4999-6_294-1.

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Allen-Hyttinen, Jessica, Henry Yung, and Marko Z. Nikolić. "Lung development." In Lung Stem Cells in Development, Health and Disease, 1–16. Sheffield, United Kingdom: European Respiratory Society, 2021. http://dx.doi.org/10.1183/2312508x.10008720.

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Keÿzer, Richard, and Martin Post. "Lung Branching Morphogenesis: Role of Growth Factors and Extracellular Matrix." In Lung Development, 1–27. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4614-7537-8_1.

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Clement, Annick, and Jerome S. Brody. "Mechanisms of Cell Growth and Tissue Repair." In Lung Development, 282–303. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4614-7537-8_10.

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Sunday, Mary E. "Bioactive Peptides and Lung Development." In Lung Development, 304–26. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4614-7537-8_11.

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Korfhagen, Thomas R., and Jeffrey A. Whitsett. "Transgenic Models of Lung Development and Disease." In Lung Development, 327–46. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4614-7537-8_12.

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Kalenga, Masendu, Claude Gaultier, and Peter H. Burri. "Nutritional Aspects of Lung Development." In Lung Development, 347–63. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4614-7537-8_13.

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Harding, Richard, and Cheryl Albuquerque. "Pulmonary Hypoplasia: Role of Mechanical Factors in Prenatal Lung Growth." In Lung Development, 364–94. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4614-7537-8_14.

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Morton, Ronald L., and Carl W. White. "Pulmonary Antioxidant Defense Mechanisms." In Lung Development, 395–424. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-1-4614-7537-8_15.

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Conference papers on the topic "Lung development"

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Richardson, Emily, Hailey Sze, Tomasz Kostrzewski, and David Hughes. "Development of novel alveolar and bronchial microphysiological systems for use in disease research and drug development." In ERS Lung Science Conference 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/23120541.lsc-2022.149.

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Kordi, Haya, Amena Al-Sadi, Fatiha Benslimane, and Huseyin Cagatay Yalcin. "Development and in Vitro Testing of a Nitric Oxide Nanoparticle Carrier for Acute Lung Injury." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0193.

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Abstract:

Acute respiratory distress syndrome (ARDS) is an infectious clinical condition in which gas exchange inside the airways and alveoli are disturbed. Fluid filled lungs need to be mechanically ventilated for airway reopening.Ventilation might further damage delicate lung tissue and lead to edema,a phenomenon known as ventilator-induced lung injury VILI is a result of propagation of small air bubbles in gas exchange sites, injuring epithelial cells due to shear stress. Potential rescue of epithelial cells (EPCs) under injurious stresses is possible by altering their mechanical properties and hence deformation amount under stress (decreased stiffness, decreased deformation). This is possible by altering the cytoskeleton. Nitric oxide (NO) inhalation therapy for ARDS enhances oxygenation. In addition, NO secretion was shown to decrease stiffness in various tissue types which can aid as a treatment of conditions like ARDS.One issue with using NO is that the life-time is too short so the treatment is not very effective. We have used nanoparticles, which secretes NO in aqueous environment. We hypothesize that Administration of NO through releasing polymers will soften lung cells and suppress inflammatory markers which enhance survival of lung cells against shear stress.

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Gillard, Alison, Agnès Noël, and Didier Cataldo. "Prenatal exposure to ozone triggers development of lung diseases in offspring." In ERS Lung Science Conference 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/23120541.lsc-2022.181.

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Talaei Pashiri, M., L. Portas, N. Aung, M. Hind, C. Dean, C. Minelli, and S. O. Shaheen. "Lung development genes, adult lung function and cardiovascular comorbidities." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2326.

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Dmitrieva, Anna, Anne Hilgendorff, and Heiko Adler. "The impact of virus infection on the development of Bronchopulmonary Dysplasia (BPD)." In ERS Lung Science Conference 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/23120541.lsc-2022.167.

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Mujahid, S., HC Nielsen, and MV Volpe. "Connecting Lung Development and Lung Cancer through microRNA Expression Profiling in Fetal Lung." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5018.

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Zhou, D., Y. Chen, and J. Wang. "Development and Validation of a Novel Prognostic Nomogram for COPD patients with Pulmonary Hypertension." In ERS Lung Science Conference 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.lsc-2021.46.

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Fung, Genevieve Po Gee, Renee Wan Yi Chan, Yuen Yi Kathy Chan, and Hugh Simon Hung San Lam. "A study to evaluate nasal mucosal IgA development in neonates using a non-invasive sampling method." In ERS Lung Science Conference 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/23120541.lsc-2022.37.

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9

Isabekov, N. R. "METHOD OF RESPIRATORY SUPPORT FOR ACUTE CHEMICAL LUNG LESIONS OF PROFESSIONAL GENESIS." In The 4th «OCCUPATION and HEALTH» International Youth Forum (OHIYF-2022). FSBSI «IRIOH», 2022. http://dx.doi.org/10.31089/978-5-6042929-6-9-2022-1-87-90.

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Abstract:

Introduction: Currently, the problem of chemical defeats at work continues to be relevant. According to the Federal Center for Hygiene and Epidemiology of Rospotrebnadzor, diseases (intoxication) caused by chemical factors exposure averaged 5.74–6.39% of occupational diseases per year all registered cases. As result of toxic substances inhalation, direct damage to lungs occurs with the further development of acute respiratory distress syndrome (ARDS). According to the national intensive care guide in the treatment of ARDS, the following main areas are distinguished - respiratory therapy and drug treatment. The latter includes infusion therapy, the use of glucocorticoids, surfactant preparations, narcotic analgesics and diuretics. Respiratory support consists in the use of mechanical ventilation (MV), and if the latter is not effective, extracorporeal membrane oxygenation (ECMO). Methods: literature data analyzes of acute chemical lung lesions treatment, as well as data on types, methods of liquid ventilation lung use. Results: It was found that the use of lungs both partial and total liquid ventilation using perfluorocarbon (PFC) compounds not only improved gas exchange in the lungs, but reduced the severity of the inflammatory response also. It was shown that the method of lung liquid ventilation (LVL) is promising. In turn, the increase in oxygenation we received, the increase in partial blood pressure during the alveolar phase of oedema, shows the advantage of liquid ventilation over existing ventilation regimes MV, thereby reflecting the potential of LAVL as a new method of respiratory support in acute lung lesions.

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Iriondo, C., K. Skarp, M. Veltman, M. Buscop-Van Kempen, A. Boerema-De Munck, G. Edel, B. Scholte, R. Wijnen, and R. Rottier. "Development of a 3D in vitro co-culture model of the airway mucosa using human primary cells." In ERS Lung Science Conference 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.lsc-2021.30.

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Reports on the topic "Lung development"

1

Asgharian, Bahman. Development of Mouse Lung Deposition Models. Fort Belvoir, VA: Defense Technical Information Center, July 2015. http://dx.doi.org/10.21236/ada621585.

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Asgharian, Bahman. Development of a Guinea Pig Lung Deposition Model. Fort Belvoir, VA: Defense Technical Information Center, January 2016. http://dx.doi.org/10.21236/ad1002742.

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Wang, Xiao H., Walter F. Good, Carl R. Fuhrman, Howard E. Rockett, and David Gur. Development and Evaluation of Sterographic Display for Lung Cancer Screening. Fort Belvoir, VA: Defense Technical Information Center, December 2008. http://dx.doi.org/10.21236/ada495768.

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Wong, David. Salivary Proteomic and microRNA Biomarkers Development for Lung Cancer Detection. Fort Belvoir, VA: Defense Technical Information Center, August 2014. http://dx.doi.org/10.21236/ada613286.

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Wong, David. Salivary Proteomic and microRNA Biomarkers Development for Lung Cancer Detection. Fort Belvoir, VA: Defense Technical Information Center, August 2013. http://dx.doi.org/10.21236/ada593384.

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Wang, Xiao H., Walter F. Good, Carl R. Fuhrman, Howard E. Rockett, and David Gur. Development and Evaluation of Stereographic Display for Lung Cancer Screening. Fort Belvoir, VA: Defense Technical Information Center, December 2006. http://dx.doi.org/10.21236/ada462882.

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Sage, Julien. Development of Antidepressants as Novel Agents To Treat Small Cell Lung Cancer. Fort Belvoir, VA: Defense Technical Information Center, August 2014. http://dx.doi.org/10.21236/ada613784.

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Yuan, Chongli, Julie Liu, Seong-Eun Kim, Charny-Yun Lin, Agnes Mendonca, and Renay Su. Development of a Diagnostic Tool to Detect DNA Methylation Biomarkers for Early-Stage Lung Cancer. Fort Belvoir, VA: Defense Technical Information Center, February 2015. http://dx.doi.org/10.21236/ada615421.

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Cancio, Leopoldo C., Brack Hattler, and Andriy I. Batchinsky. Development and Evaluation of New Products for the Far-Forward Care of Combat Casualties with Acute Lung Injury. Fort Belvoir, VA: Defense Technical Information Center, February 2006. http://dx.doi.org/10.21236/ada458205.

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Cancio, Leopoldo C., Brack Hattler, and Andriy I. Batchinsky. Development and Evaluation of New Products for the Far-Forward Care of Combat Casualities With Acute Lung Injury. Fort Belvoir, VA: Defense Technical Information Center, February 2007. http://dx.doi.org/10.21236/ada472924.

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