Journal articles on the topic 'Lodewycks'
To see the other types of publications on this topic, follow the link: Lodewycks.
Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles
Consult the top 18 journal articles for your research on the topic 'Lodewycks.'
Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.
You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.
Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.
1
Kilpatrick, James. "Axel Lodewycks and the Baillieu Library 1959–73." Australian Academic & Research Libraries 29, no. 1 (January 1998): 51–56. http://dx.doi.org/10.1080/00048623.1998.10755038.
Full text
2
Zaaijman, John DuToit. "Lodewyk Adrian Vosloo." South African Medical Journal 100, no. 7 (July 2, 2010): 426. http://dx.doi.org/10.7196/samj.4299.
Full text
3
Morgan, N. "Van Tempeltronk tot katedraal: die kruisweg van Lodewyk XVII." Literator 28, no. 1 (July 30, 2007): 47–70. http://dx.doi.org/10.4102/lit.v28i1.150.
Full textAbstract:
From the Temple to the cathedral: the calvary of Louis XVII In 2004, more than 200 years after his death in the Temple prison, the heart of Louis XVII, the successor to France’s last king of the Ancien Régime, Louis XVI, was buried in the royal necropolis of Saint-Denis. Despite numerous publications on the destiny of the Little Prince, the chronology of his short life was not determined by historians and biographers, but by scientists who, in 2000, performed DNA tests on the petrified organ, which was miraculously preserved. Before this date, the biographies of the many pretenders to Louis XVII’s throne (that of Naundorff in particular) were better-known than the lifehistory of Marie Antoinette’s youngest son. Since then, various publications have changed this state of affairs, including an historical novel by one of France’s most knowledgeable authors on the monarchy of the 17th and 18th centuries and a biographical novel by a member of the Bourbon family. Antonia Fraser’s (2001) biography on Marie Antoinette and Sofia Coppola’s (2006) film on her life have rekindled interest in the events of the French revolution. The story of Louis XVII, who was used as a pawn by the revolutionaries, is one the most tragic of that period in the country’s history. This article provides an overview of key events gleaned from various sources, translated into Afrikaans for the first time.
4
Katritzky, M. A., and Kathleen Marguerite Lea. "Lodewyk Toeput: some pictures related to the commedia dell'arte." Renaissance Studies 1, no. 1 (March 1987): 71–125. http://dx.doi.org/10.1111/j.1477-4658.1987.tb00123.x.
Full text
5
Katritzky, M. A. "Lodewyk Toeput: Some Pictures Related To The Commedia Dellarte." Renaissance Studies 1, no. 1 (March 1987): 71–125. http://dx.doi.org/10.1111/1477-4658.00039.
Full text
6
Sheng, Ying, Qingqing Dong, Qiang Ren, and Mingyang Wang. "Prediction for the Adsorption of Low-Concentration Toluene by Activated Carbon." Sustainability 15, no. 2 (January 13, 2023): 1555. http://dx.doi.org/10.3390/su15021555.
Full textAbstract:
Activated carbon filters are widely used to remove gaseous pollutants in order to guarantee a healthy living environment. The standard method for evaluating the adsorption performance of filters is conducted at ~100 ppm. Although this accelerates the test and avoids the high requirements of the test device, it is still far from the contaminant concentration in the indoor environment, and adsorbents in practical application may show different capabilities. Therefore, this study compared several methods for predicting the adsorption performance of activated carbon and recommended a procedure based on the Wheeler–Jonas model to estimate the breakthrough curve at low concentrations using experimental data at high concentrations. The results showed that the Langmuir model and Wood–Lodewyckx correlation were the most suitable for obtaining the equilibrium adsorption capacity and mass transfer coefficient, which are critical parameters in the Wheeler–Jonas model. The predicted service life was derived from the breakthrough curve. A modification method based on a relationship with inlet gas concentration was proposed to reduce the prediction deviation of the service life. After modification, the maximum deviation was within two hours and the relative deviation was no more than 7%.
7
Anderson, Robert H., Mark S. Bleiweis, F. J. Fricker, Arwa Saidi, Arun Chandran, James C. Fudge, Dipankar Gupta, Giles J. Peek, Diane E. Spicer, and Jeffrey P. Jacobs. "Lodewyk H.S. van Mierop (March 31, 1927–October 17, 2021): a true giant." Cardiology in the Young 32, no. 4 (April 2022): 514–24. http://dx.doi.org/10.1017/s1047951121005266.
Full textAbstract:
AbstractWe honour a great man and a true giant. Lodewyk H.S. van Mierop (March 31, 1927 – October 17, 2021), known as Bob, was not only a Paediatric Cardiologist but also a dedicated Scientist. He made many significant and ground-breaking contributions to the fields of cardiac anatomy and embryology. He was devoted as a teacher, spending many hours with medical students, Residents, and Fellows, all of whom appreciated his regularly scheduled educational sessions. Those of us who were fortunate to know and spend time with him will always remember his great mind, his willingness to share his knowledge, and his ability to encourage spirited and fruitful discussions. His life was most productive, and he will long be remembered by many through his awesome and exemplary scientific contributions.His legacy continues to influence the current and future generations of surgeons and all providers of paediatric and congenital cardiac care through the invaluable archive he established at University of Florida in Gainesville: The University of Florida van Mierop Heart Archive. Undoubtedly, with these extraordinary contributions to the fields of cardiac anatomy and embryology, which were way ahead of his time, Professor van Mierop was a true giant in Paediatric Cardiology. The invaluable archive he established at University of Florida in Gainesville, The University of Florida van Mierop Heart Archive, has been instrumental in teaching medical students, Residents, Medical Fellows, and Surgical Fellows. Only a handful of similar archives exist across the globe, and these archives are the true legacy of giants such as Dr. van Mierop. We have an important obligation to leave no stone unturned to continue to preserve these archives for the future generations of surgeons, physicians, all providers of paediatric and congenital cardiac care, and, most importantly, our patients.
8
du Bruyn, Derek. "Die Platberg van Harrismith: ’n GeskiedenisMARAISJOHANN LODEWYK, Die Platberg van Harrismith: ’n Geskiedenis, Imprimatur, 2021, 146 pp, illustrasies, eindnotas en bronnelys, ISBN: 978-0-620-93882-2." South African Journal of Cultural History 36, no. 2 (December 2022): 191–92. http://dx.doi.org/10.54272/sach.2022.v36n2a10.
Full text
9
Vis, Daniel, Sander Palit, Marie Corradi, Martijn Lolkema, Niven Mehra, Edwin Cuppen, Lodewyk FA Wessels, et al. "Abstract 972: MMR-deficiency is the most prominent genetic feature of prostate cancer metastases organotropism." Cancer Research 82, no. 12_Supplement (June 15, 2022): 972. http://dx.doi.org/10.1158/1538-7445.am2022-972.
Full textAbstract:
Abstract Prostate cancer (PCa) is the most prevalent, non-cutaneous, cancer in men in the Western world. Disease confined to the prostate can be cured, but metastatic disease cannot. PCa metastases are found in bone, lymph-nodes, liver and other visceral organs (visceral), in decreasing order of frequency. After an initial response to androgen receptor directed therapy, metastatic disease will inevitably recur as metastatic castration resistant prostate cancer (mCRPC), which is hallmarked by high morbidity and mortality. Since it is previously established, that the site of metastasis correlates with prostate cancer survival, we need a better understanding of metastatic organotropism. Therefore, we took advantage of the largest Whole Genome Sequencing (HiSeq X Ten system using the paired-end sequencing protocol) data set of mCRPC metastases to date. Molecular profiling of 326 metastases (Bone: 105, Lymph-node: 149, Liver: 49, Visceral: 23) revealed genetic determinants associated with organ-specific metastasis. First, we assessed differential occurrence of mutations in genes associated with targetable pathways. RB1 alteration were enriched in liver (35%) and in visceral metastases (30%), while lower rates were found in bone (10%) and lymph-nodes (13%)(Fisher exact test, p: 0.012). Analysis of aggregated pathway alteration data revealed a trend for increased frequency of alterations in the DNA repair and PI3K pathways in lymph node vs. bone metastases (p: 0.066 and 0.066, respectively). Next, we explored differential tumor mutational burden (TMB) between the sites. A higher TMB was observed in liver and visceral metastases compared to bone and lymph node metastases, while there was no difference between liver and visceral. The increased TMB in liver and visceral samples was associated with an MMR-deficiency mutational signature. Alterations in MSH6, MLH1 and POLD3 characterized a significant proportion of high TMB liver metastases, whereas high TMB visceral metastases predominantly showed MSH2 and POLD1 alterations.In conclusion: Our findings implicate high TMB/MMR-deficiency is a characteristic feature of liver and visceral PCa metastases, potentially impacting disease progression. Moreover, since response to immune check-point inhibitors is associated with high TMB, our findings might direct choice of therapy. Citation Format: Daniel Vis, Sander Palit, Marie Corradi, Martijn Lolkema, Niven Mehra, Edwin Cuppen, Lodewyk FA Wessels, Rene Bernards, Wilbert Zwart, Michiel S. van der Heijden, Andries M. Bergman. MMR-deficiency is the most prominent genetic feature of prostate cancer metastases organotropism [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2022; 2022 Apr 8-13. Philadelphia (PA): AACR; Cancer Res 2022;82(12_Suppl):Abstract nr 972.
10
Bismeijer, Tycho, Ahmed A. Ahmed, Michael Sheinman, Maria Roman-Escorza, Vandna Shah, Jeffrey R. Marks, Lorraine M. King, et al. "Abstract P1-22-05: Identifying predictors of invasive recurrence based on molecular profiles of DCIS lesions." Cancer Research 82, no. 4_Supplement (February 15, 2022): P1–22–05—P1–22–05. http://dx.doi.org/10.1158/1538-7445.sabcs21-p1-22-05.
Full textAbstract:
Abstract Introduction Ductal carcinoma in situ (DCIS) is a non-obligate precursor of invasive breast cancer. Patients with DCIS are routinely treated by breast-conserving surgery often supplemented by radiotherapy, although many will never develop invasive disease. To date, no robust predictors of invasive breast cancer recurrence following DCIS have been identified. In our efforts to find such predictors, we performed gene expression, copy number and mutation analysis on two large DCIS cohorts with long-term follow-up. Methods Two nested case control series were analyzed, where cases are defined as DCIS with a subsequent invasive breast cancer and controls remained disease free during follow up. Cases and controls were matched on age and on follow up duration and were derived from two nation-wide cohort studies. The Sloane cohort is a prospective breast screening cohort from the UK, median follow up is 6 years (range 1-10). The Dutch cohort is population-based and had a median follow up of 13 years (range 2-23). We performed copy number analysis using CytoSNP array or low pass whole genome sequencing (lpWGS) on 310 controls and 196 cases, and RNA-seq on 295 controls and 206 cases. Results First analyses on the copy number data suggest that cases are genetically more aberrant with multiple regions of amplification compared to controls (p < 0.05). RNA-seq was used to classify DCIS into the PAM50 subtypes which did not appear to be predictive of recurrence. Initial RNA-seq analysis did not show consistent gene expression differences between cases and controls in the Sloane or Dutch cohorts, possibly explained by differences in clinical characteristics of the cohorts. A new computational method has been developed accounting for the differences in follow-up times, results will be presented at SABCS. Targeted sequencing revealed that the most common mutations were in PIK3CA and TP53, but there was no association with recurrence. Conclusion Only small molecular differences were identified between DCIS that recurs as invasive breast cancer and DCIS that remains disease-free. Currently, we are seeking to identify reproducible differences by a combined analysis of two population-based cohorts in a time dependent fashion. These will be presented at the SABCS. This work was supported by Cancer Research UK and by KWF Dutch Cancer Society (ref.C38317/A24043) Citation Format: Tycho Bismeijer, Ahmed A Ahmed, Michael Sheinman, Maria Roman-Escorza, Vandna Shah, Jeffrey R Marks, Lorraine M King, Anargyros Megalios, Lindy L Visser, Marlous Hoogstraat, Helen R Davies, Tapsi Kumar, Deborah Collyar, Hilary Stobart, Nicholas N Navin, Andrew Futreal, Serena Nik-Zainal, Shelley Hwang, Esther H Lips, Alastair Thompson, Lodewyk FA Wessels, Elinor J Sawyer, Jelle Wesseling, Grand Challenge PRECISION Consortium. Identifying predictors of invasive recurrence based on molecular profiles of DCIS lesions [abstract]. In: Proceedings of the 2021 San Antonio Breast Cancer Symposium; 2021 Dec 7-10; San Antonio, TX. Philadelphia (PA): AACR; Cancer Res 2022;82(4 Suppl):Abstract nr P1-22-05.
11
Ahmed, Ahmed A., Maria Roman-Escorza, Tycho Bismeijer, Michael Sheinman, Vandna Shah, Rana Shami, Jeffrey R. Marks, et al. "Abstract 5108: Copy number analysis of pure DCIS and association with recurrence." Cancer Research 82, no. 12_Supplement (June 15, 2022): 5108. http://dx.doi.org/10.1158/1538-7445.am2022-5108.
Full textAbstract:
Abstract With the widespread adoption of breast cancer screening the incidence of pure ductal carcinoma in situ (DCIS) has increased. As DCIS is considered a non-obligate precursor of invasive ductal carcinoma most women with pure DCIS are treated with breast conserving surgery (BCS) +/- radiotherapy. However, for many this is likely to be overtreatment as only a minority will develop a subsequent ipsilateral recurrence. Studies also show that only ~60% of these ipsilateral recurrences are invasive disease with the remainder being pure DCIS. To predict which women are most likely to benefit from interventions, there is a need to identify biomarkers that are associated with invasive recurrence. Our aim was to assess whether copy number aberrations (CNAs) could be used to identify DCIS that was likely to recur as invasive disease or remain recurrence-free during long-time follow up. We performed somatic copy number profiling on 309 pure DCIS samples that had not developed an ipsilateral event (controls), 198 that had developed subsequent ipsilateral invasive disease (INV-cases) and 58 that had developed subsequent ipsilateral pure DCIS (DCIS-cases). The samples were obtained from two large nation-wide cohorts: the Sloane cohort, a prospective breast screening cohort from the UK with a median follow up of 12.5 years and a Dutch population based cohort, with a median follow up of 13 years. CNAs were assessed using the CytoSNP array or low pass whole genome sequencing and analyzed using GISTIC. Integrative cluster (IntClust) subtyping revealed that only 5 subtypes were well represented in DCIS compared to 10 in invasive disease and the distribution of clusters between INV-cases and controls was similar with the exception of IntClust 4, which was significantly more common in controls (P= 0.025, Fishers exact test). IntClust 4 is characterized to have low levels of genomic instability and a CNA-devoid. INV-cases were globally more aberrant than controls (P = 0.006, Wilcoxon test) as assessed by the chromosomal instability index (CIN) score. GISTIC identified 17 recurrent amplifications, 21 recurrent gains and 22 recurrent losses in the whole cohort. Six of these regions were more common in INV-cases compared to controls: amplifications at 17q24.1 and 8p11.23, losses at 1p36.13 and 11q23.2 and gains at 17q21.33 and 16p (Nominal P < 0.05 and FDR < 0.1, Fishers exact test). Subgroup analysis of ER+, Her2- INV-cases versus controls revealed an additional differential CNA, amplification at 11q13.3 more common in cases. DCIS-cases had similar CNAs to INV-cases and were more aberrant than controls in terms of CIN score (P < 0.037, Wilcoxon test) but not as aberrant as INV-cases. In conclusion, we have identified potential CNAs that are associated with invasive recurrence. Further analysis will integrate gene expression with copy number data to identify which genes are being targeted by these CNAs in order to identify pathways important in progression of DCIS. Citation Format: Ahmed A. Ahmed, Maria Roman-Escorza, Tycho Bismeijer, Michael Sheinman, Vandna Shah, Rana Shami, Jeffrey R. Marks, Lorraine M. King, Anargyros Megalios, Lindy L. Visser, Marlous Hoogstraat, Helen R. Davies, Tapsi Kumar, Deborah Collyar, Hilary Stobart, Sarah Pinder, Nicholas N. Navin, Andrew Futreal, Serena Nik-Zainal, E. Shelley Hwang, Lodewyk F. Wessels, Esther H. Lips, Alastair Thompson, Jelle Wesseling, Elinor J. Sawyer. Copy number analysis of pure DCIS and association with recurrence [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2022; 2022 Apr 8-13. Philadelphia (PA): AACR; Cancer Res 2022;82(12_Suppl):Abstract nr 5108.
12
Escorza, Maria Roman, Michael Sheinman, Tycho Bismeijer, Ahmed A. Ahmed, Vandna Shah, Jeffrey R. Marks, Lorraine M. King, et al. "Abstract PR002: Genomic predictor can discriminate between high- and low-risk DCIS." Cancer Prevention Research 15, no. 12_Supplement_1 (December 1, 2022): PR002. http://dx.doi.org/10.1158/1940-6215.dcis22-pr002.
Full textAbstract:
Abstract Introduction: Ductal carcinoma in situ (DCIS) is considered a non-obligate precursor of invasive ductal carcinoma. With the aim of preventing a subsequent invasive cancer, all DCIS lesions are currently treated with surgical excision often supplemented with radiotherapy (RT). To prevent DCIS over- or undertreatment, a reliable marker of DCIS invasiveness risk is urgently needed. Methods: We studied two large DCIS cohorts: the Sloane cohort, a prospective breast screening cohort from the UK (median follow-up of 11 years), and a Dutch population-based cohort (NKI, median follow-up of 13 years). FFPE tissue specimens from patients with pure primary DCIS after breast-conserving surgery (BCS) +/- RT that did develop a subsequent ipsilateral event (DCIS or invasive) were considered as cases, whereas patients that did not develop any form of recurrence up to the last follow-up or death were considered as controls. We performed copy number analysis (CNA) and RNAseq analysis on 229 cases (80 DCIS only recurrences) and 344 controls. Results: DCIS was classified into the PAM50 subtypes using RNAseq data which revealed an enrichment of luminal A phenotype in DCIS that did not recur (P = 0.01, Fisher Exact test). No single copy number aberration was more common in cases compared to controls. RNAseq data did not reveal any genes significantly over/under-expressed in cases versus controls after FDR correction. However, by limiting the analysis to samples that had not had RT and excluding pure DCIS recurrences, we could develop a penalized Cox model from RNAseq data. The model was trained on weighted samples (to correct for the biased sampling of the case-control dataset) from the NKI series with double loop cross-validation. The genes were selected using the Elastic net framework of penalization. Using this predicted hazard ratio, the samples were split into high, medium, and low-risk quantiles, with a recurrence risk of 23%, 7% and 2%, respectively at 5 years (p = 10-10, Wald test). The NKI-trained predictor was independently validated in the Sloane No RT no DCIS recurrence cohort (p = 0.02, Wald test). GSEA analysis revealed proliferation hallmarks enriched in the recurrence predictor (FDR = 0.058). The RNAseq predictor was more predictive of recurrence than PAM50, clinical features (Grade, Her2 and ER) and the 12-gene Oncotype DCIS score (p < 0.001, permutation test using the Wald statistic) in both the NKI and Sloane series. Conclusion: Genomic profiling of two independent series of DCIS with outcome data did not reveal any clear associations with recurrence until analysis was limited to a set of samples who had not had radiotherapy and DCIS recurrences were excluded. We then identified an RNAseq-based classifier that could differentiate primary DCIS in low-, medium-, and high-risk groups, and validated it in an independent cohort. This classifier, if validated in other datasets, will allow us to identify women who do not need intensive treatment for their DCIS. Citation Format: Maria Roman Escorza, Michael Sheinman, Tycho Bismeijer, Ahmed A. Ahmed, Vandna Shah, Jeffrey R. Marks, Lorraine M. King, Anargyros Megalios, Lindy L. Visser, Marlous Hoogstrat, Helen R. Davies, Tapsi Kumar, Deborah Collyar, Hilary Stobart, Sarah Pinder, Nicholas N. Navin, Andrew Futreal, Serena Nik-Zainal, E. Shelley Hwang, Esther H. Lips, Alastair Thompson, Lodewyk F.A. Wessels, Jelle Wesseling, Elinor J. Sawyer. Genomic predictor can discriminate between high- and low-risk DCIS [abstract]. In: Proceedings of the AACR Special Conference on Rethinking DCIS: An Opportunity for Prevention?; 2022 Sep 8-11; Philadelphia, PA. Philadelphia (PA): AACR; Can Prev Res 2022;15(12 Suppl_1): Abstract nr PR002.
13
Hutten, Stefan J., Roebi de Bruijn, Catrin Lutz, Madelon Badoux, Timo Eijkman, Xue Chao, Marta Ciwinska, et al. "Abstract PR006: A living biobank of patient-derived ductal carcinoma in situ (DCIS) Mouse-INtraDuctal (MIND) xenografts identifies multiple risk factors of invasive progression." Cancer Prevention Research 15, no. 12_Supplement_1 (December 1, 2022): PR006. http://dx.doi.org/10.1158/1940-6215.dcis22-pr006.
Full textAbstract:
Abstract Ductal Carcinoma in Situ (DCIS) is a non-invasive non-obligate precursor of invasive breast cancer (IBC). DCIS is usually treated by surgery combined with radiotherapy, which can have a large impact on the life of patients. However, many of these DCIS lesions would never progress into IBC. To reduce the overtreatment of DCIS, but assure proper treatment for high risk DCIS, it is crucial to understand the biology underlying DCIS. To study the biology of DCIS we established Mouse INtraDuctal (MIND) patient-derived xenograft (PDX) models by intraductally injecting patient DCIS material into the mammary ducts of female immunocompromised mice. We engrafted 130 samples, which have been incubated in vivo for a period of 12 months. We obtain a take rate of 88% with 46% of our models showing invasive progression. Histology and molecular subtyping by PAM50 classification are well preserved in the MIND models compared to the primary counterpart, ensuring that our MIND models represent the patient disease well. For 102 primary samples we obtained RNAseq profiles as well as for 64 matched MIND-PDX models. In addition whole exome-/panel sequencing data is generated from the same primary DCIS samples together with 12 matched MIND-PDX WES profiles as well as 60 matched Copy Number Variation (CNV) MIND-PDX profiles. Together these data revealed multiple biomarkers related to invasive progression, including factors such as high grade, solid growth, a high copy number aberrations burden, HER2, PTK6 & MYC amplifications and a high Ki67. On top of this we used whole mount imaging of the injected mammary glands extracted from our MIND-PDX models, showing two distinct growth patterns correlated with invasion. And as this is all done in the context of the PRECISION consortium this allows us to confirm and validate our findings in larger sequencing and imaging efforts of human samples. We have also successfully passaged 42 MIND-PDX models which showed minimal changes in pheno- and genotype over time indicating invasive behavior is an intrinsic phenotype of DCIS with minimal evolution, supporting a multiclonal evolution model. Moreover, this provided a collection of 19 stable sequentially transplantable DCIS MIND models including Luminal A, Luminal B, ER+/HER2+ and ER-/HER2+ models. Ultimately these models can be used to validate the biomarkers found to be related to invasive progression, as an example we proved the direct role of HER2 overexpression in invasive progression by inhibiting the HER2 receptor or by overexpressing HER2. In conclusion all this data together enabled us to create a well-characterized biobank of DCIS models with the unique opportunity to follow the natural progression, sequentially transplant 42 models, find genomic and transcriptomic profiles related to high risk DCIS and manipulate gene expression to validate the role of genes in DCIS progression. Citation Format: Stefan J. Hutten, Roebi de Bruijn, Catrin Lutz, Madelon Badoux, Timo Eijkman, Xue Chao, Marta Ciwinska, Andrea Herencia-Ropero, Petra Kristel, Lennart Mulder, Joyce Sanders, Mathilde Almekinders, Alba Llop-Gueverra, Helen R. Davies, Fariba Behbod, Serena Nik-Zainal, Violeta Serra, Jacco van Rheenen, Esther H. Lips, Lodewyk F.A. Wessels, Jelle Wesseling, Colinda Scheele, Jos Jonkers. A living biobank of patient-derived ductal carcinoma in situ (DCIS) Mouse-INtraDuctal (MIND) xenografts identifies multiple risk factors of invasive progression [abstract]. In: Proceedings of the AACR Special Conference on Rethinking DCIS: An Opportunity for Prevention?; 2022 Sep 8-11; Philadelphia, PA. Philadelphia (PA): AACR; Can Prev Res 2022;15(12 Suppl_1): Abstract nr PR006.
14
"Postface par Herman Lodewyckx." L’enseignement philosophique 66e Année, no. 3 (March 1, 2016): 77–78. http://dx.doi.org/10.3917/eph.663.0077.
Full text
15
Fauzan, Moh Ariq, and Endang Suparsetyani. "Analisi Yuridis Terhadap Pembagian Harta Warisan Almarhum Mouritis Alexander Lodewyik Thalele Kepada Ahli Warisnya Menurut Kitab Undang-Undang Hukum Perdata (Studi Kasus Putusan Nomor: 685/Pdt.G/2013/PN. Dps.)." Reformasi Hukum Trisakti 1, no. 1 (June 30, 2019). http://dx.doi.org/10.25105/refor.v1i1.10534.
Full textAbstract:
Hukum Waris adalah kumpulan kaidah-kaidah hukum yang mengatur tentang peralihan hak dan kewajiban pewaris kepada ahli warisnya Di dalam Putusan Nomor: 685/Pdt.G/2013/PN. Dps. Yang menjadi pokok permasalahan dalam penelitian adalah: (1) Bagaimana pembagian harta warisan Almarhum Alexander Lodewyk Thalele kepada ahli warisnya menurut Kitab Undang-undang Hukum Perdata? (2) Apakah Isi Amar putusan pengadilan Nomor: 685/PDT.G/2013/PN. DPS tentang Penetapan Ahli Waris Almarhum Alexander Lodewyk Thalele sudah sesuai atau tidak menurut ketentuan Kitab Undang-undang Hukum Perdata? Untuk menjawab pokok permasalahan tersebut dalam penelitian ini menggunakan tipe penelitian yuridis normatif, sifat penelitian deskriptif analisis, data yang digunakan data sekunder, cara pengumpulan data menggunakan metode studi kepustakaan dan data dianalisis secara kualitatif serta menarik kesimpulan dengan cara metode deduktif. Kesimpulan dari penelitian ini adalah : (1) Berdasarkan Pasal 830, 832, 836, 852, dan 957 Kitab Undang-undang Hukum Perdata Pembagian harta warisan Almarhum Alexander Lodewyk Thalele adalah seluruhnya untuk istrinya dalam hal ini Silvina Lika Thalele (2) Amar Putusan pengadilan Nomor: 685/PDT.G/2013/PN. DPS tentang Penetapan Ahli Waris Dari Almarhum Alexander Lodewyk Thalele tidak sesuai dengan Kitab Undang-undnag Hukum Perdata karena Kitab Undang-undang Hukum Perdata tidak mengenal anak angkat
16
Lemmer, E. "Review: "Diorama: gedigte" (Johann Lodewyk Marais)." Tydskrif vir letterkunde 49, no. 1 (February 9, 2012). http://dx.doi.org/10.4314/tvl.v49i1.20.
Full text
17
Baldewijns, Jeannine. "Een verloren gewaand schilderij van de Gentse schilder Johan Baptist Lodewyk Maes (Gent 1794 - Rome 1856)." Van Mensen en Dingen: tijdschrift voor volkscultuur in Vlaanderen 3, no. 4 (November 11, 2005). http://dx.doi.org/10.21825/vmend.v3i4.5310.
Full textAbstract:
In 200 kon het Bijlokemuseum / STA°M een merkwaardig schilderij van Johan Baptist Lodewyk Maes, beter bekend als Maes-Canini, met een vrij ongewoon thema, aankopen bij het veilinghuis Haboldt en Co. te Parijs. Het doek staat bekend als 'De koepokkenzaal' en dateert van 1819.
18
Huigen, Siegfried. "De eerste etnografische monografie: De Kaffers aan de Zuidkust van Afrika (1810) van Lodewyk Alberti." Tydskrif vir letterkunde 43, no. 1 (September 8, 2006). http://dx.doi.org/10.4314/tvl.v43i1.29719.
Full text