Relevant bibliographies by topics / Liver fat

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Liver fat'

Author: Grafiati
Published: 4 June 2021
Last updated: 21 February 2023

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Journal articles on the topic "Liver fat"

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Koeckerling, David, Jeremy W. Tomlinson, and Jeremy F. Cobbold. "Fighting liver fat." Endocrine Connections 9, no. 7 (July 2020): R173—R186. http://dx.doi.org/10.1530/ec-20-0174.

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Non-alcoholic fatty liver disease is a chronic liver disease which is closely associated with components of the metabolic syndrome. Its high clinical burden results from the growing prevalence, inherent cardiometabolic risk and potential of progressing to cirrhosis. Patients with non-alcoholic fatty liver disease show variable rates of disease progression through a histological spectrum ranging from steatosis to steatohepatitis with or without fibrosis. The presence and severity of fibrosis are the most important prognostic factors in non-alcoholic fatty liver disease. This necessitates risk stratification of patients by fibrosis stage using combinations of non-invasive methods, such as composite scoring systems and/or transient elastography. A multidisciplinary approach to treatment is advised, centred on amelioration of cardiometabolic risk through lifestyle and pharmacological interventions. Despite the current lack of licensed, liver-targeted pharmacotherapy, several promising agents are undergoing late-phase clinical trials to complement standard management in patients with advanced disease. This review summarises the current concepts in diagnosis and disease progression of non-alcoholic liver disease, focusing on pragmatic approaches to risk assessment and management in both primary and secondary care settings.
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Rider, Oliver J., Rajarshi Banerjee, Jennifer J. Rayner, Ravi Shah, Venkatesh L. Murthy, Matthew D. Robson, and Stefan Neubauer. "Investigating a Liver Fat." Arteriosclerosis, Thrombosis, and Vascular Biology 36, no. 1 (January 2016): 198–203. http://dx.doi.org/10.1161/atvbaha.115.306561.

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Robinson, P. J. A. "Fat and the liver." Imaging 16, no. 4 (September 2004): 364–74. http://dx.doi.org/10.1259/imaging/26666175.

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Crunkhorn, Sarah. "Liver enzyme inflames fat." Nature Reviews Drug Discovery 17, no. 5 (April 20, 2018): 315. http://dx.doi.org/10.1038/nrd.2018.59.

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Kersten, Sander, and Michael Müller. "Dropping liver fat droplets." Hepatology 50, no. 2 (July 29, 2009): 645–47. http://dx.doi.org/10.1002/hep.23142.

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Bronson, Steven M., Brian Westwood, Katherine L. Cook, Nancy J. Emenaker, Mark C. Chappell, David D. Roberts, and David R. Soto-Pantoja. "Discrete Correlation Summation Clustering Reveals Differential Regulation of Liver Metabolism by Thrombospondin-1 in Low-Fat and High-Fat Diet-Fed Mice." Metabolites 12, no. 11 (October 28, 2022): 1036. http://dx.doi.org/10.3390/metabo12111036.

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Thrombospondin-1 (TSP1) is a matricellular protein with many important roles in mediating carcinogenesis, fibrosis, leukocyte recruitment, and metabolism. We have previously shown a role of diet in the absence of TSP1 in liver metabolism in the context of a colorectal cancer model. However, the metabolic implications of TSP1 regulation by diet in the liver metabolism are currently understudied. Therefore Discrete correlation summation (DCS) was used to re-interrogate data and determine the metabolic alterations of TSP1 deficiency in the liver, providing new insights into the role of TSP1 in liver injury and the progression of liver pathologies such as nonalcoholic fatty liver disease (NAFLD). DCS analysis provides a straightforward approach to rank covariance and data clustering when analyzing complex data sets. Using this approach, our previous liver metabolite data was re-analyzed by comparing wild-type (WT) and Thrombospondin-1 null (Thbs1−/−) mice, identifying changes driven by genotype and diet. Principal component analysis showed clustering of animals by genotype regardless of diet, indicating that TSP1 deficiency alters metabolite handling in the liver. High-fat diet consumption significantly altered over 150 metabolites in the Thbs1−/− livers versus approximately 90 in the wild-type livers, most involved in amino acid metabolism. The absence of Thbs1 differentially regulated tryptophan and tricarboxylic acid cycle metabolites implicated in the progression of NAFLD. Overall, the lack of Thbs1 caused a significant shift in liver metabolism with potential implications for liver injury and the progression of NAFLD.
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Zídková, J., J. Sajdok, K. Kontrová, A. Kotrbová-Kozak, T. Hanis, V. Zídek, and A. Fuíková. "Effects of oxidised dietary cod liver oil on the reproductive functions of Wistar rat." Czech Journal of Food Sciences 22, No. 3 (November 16, 2011): 108–20. http://dx.doi.org/10.17221/3414-cjfs.

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Weanling Wistar rats, males and females, were fed for 185 days with diets containing 15% of dietary fat in the form of a mixture of lard and partially oxidised cod liver oil. The proportion of cod liver oil in the dietary fat ranged from 0 to 100%, and the content of malonaldehyde from 0.3 to 19.6 mg/kg of the fat used. Animals fed with diets containing higher proportions of oxidised cod liver oil had higher concentrations of malonaldehyde in their livers. Serum lipid levels were lower in animals fed with higher proportions of cod liver oil than in animals fed control diets (milk fat or lard). The lowest concentration of serum lipid was found in the rats fed the diet containing half of its fat as fish oil. Increased intakes of cod liver oil resulted in lower body weight gains, weights of livers, kidneys, and weights of the reproductive organs. The relative weights of livers and kidneys/body weight were higher in the groups with higher intakes of cod liver oil. High intakes of cod liver oil led to a drastically impaired fertility of females, a decreased litter size, a higher postnatal mortality, and an increased incidence of morphologically abnormal spermatozoa in males.  
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Alanen, A., M. Komu, R. Leino, and S. Toikkanen. "MR and magnetisation transfer imaging in cirrhotic and fatty livers." Acta Radiologica 39, no. 4 (July 1998): 434–39. http://dx.doi.org/10.1080/02841859809172459.

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Purpose: To determine whether low-field MR fat/water separation and magnetisation transfer (MT) techniques are useful in studying the livers of patients with parenchymal liver diseases in vivo. Material and Methods: MR and MT imaging of the liver in 33 patients (14 with primary biliary cirrhosis, 15 with alcohol-induced liver disease, and 4 with fatty liver) was performed by means of the fat/water separation technique at 0.1 T. The relaxation time T1 and the MT contrast (MTC) parameter of liver and spleen tissue were measured, and the relative proton density fat content N(%) and MTC of the liver were calculated from the separate fat and water images. The value of N(%) was also compared with the percentage of fatty hepatocytes at histology. Results: The relaxation rate R1 of liver measured from the magnitude image, and the difference in the value of MTC measured from the water image compared with the one measured from the fat and water magnitude image, both depended linearly on the value of N(%). The value of N(%) correlated significantly with the percentage of the fatty hepatocytes. In in vivo fatty tissue, fat infiltration increased both the observed relaxation rate R1 and the measured magnetisation ratio (the steady state magnetisation Ms divided by the equilibrium magnetisation Mo, Ms/Mo) and consequently decreased the MT efficiency measured in a magnitude MR image. The amount of liver fibrosis did not correlate with the value of MTC measured after fat separation. Conclusion: Our results in studying fatty livers with MR imaging and the MT method show that the fat/water separation gives more reliable parametric results. Characterisation of liver cirrhosis by means of the MTC parameter is not reliable, even after fat separation.
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Green, Charlotte, and Leanne Hodson. "The Influence of Dietary Fat on Liver Fat Accumulation." Nutrients 6, no. 11 (November 10, 2014): 5018–33. http://dx.doi.org/10.3390/nu6115018.

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Yamada, Akiko, Kyoko K. Sato, Shigeki Kinuhata, Shinichiro Uehara, Ginji Endo, Yonezo Hikita, Wilfred Y. Fujimoto, Edward J. Boyko, and Tomoshige Hayashi. "Association of Visceral Fat and Liver Fat With Hyperuricemia." Arthritis Care & Research 68, no. 4 (March 24, 2016): 553–61. http://dx.doi.org/10.1002/acr.22729.

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Dissertations / Theses on the topic "Liver fat"

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Migid-Hamzza, Jeffery A. "Fat Metabolism in Smooth Dogfish." University of Akron / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=akron1132414091.

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Lam, Shi. "The significance of hepatic stellate cell activation in small-for-size fatty liver graft injury /." View the Table of Contents & Abstract, 2007. http://sunzi.lib.hku.hk/hkuto/record/B3829686X.

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Grundström, Tobias. "Automated Measurements of Liver Fat Using Machine Learning." Thesis, Linköpings universitet, Datorseende, 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-151286.

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The purpose of the thesis was to investigate the possibility of using machine learn-ing for automation of liver fat measurements in fat-water magnetic resonancei maging (MRI). The thesis presents methods for texture based liver classificationand Proton Density Fat Fraction (PDFF) regression using multi-layer perceptrons utilizing 2D and 3D textural image features. The first proposed method was a data classification method with the goal to distinguish between suitable andunsuitable regions to measure PDFF in. The second proposed method was a combined classification and regression method where the classification distinguishes between liver and non-liver tissue. The goal of the regression model was to predict the difference d = pdff mean − pdff ROI between the manual ground truth mean and the fat fraction of the active Region of Interest (ROI).Tests were performed on varying sizes of Image Feature Regions (froi) and combinations of image features on both of the proposed methods. The tests showed that 3D measurements using image features from discrete wavelet transforms produced measurements similar to the manual fat measurements. The first method resulted in lower relative errors while the second method had a higher method agreement compared to manual measurements.
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Banerjee, Rajarshi. "The effects of excess body weight on the heart and liver." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:652e90cd-1f11-4fb6-8a4b-4ce649f72ee5.

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Obesity in adults and children is associated with increased cardiovascular mortality and morbidity. This is forecast to increase markedly in the next decade as childhood obesity is a burgeoning epidemic. Excess weight is clearly associated with insulin resistance, increased circulating triglycerides, and hypertension, all of these are related to progressive heart and liver disease. Ectopic fat deposition within organs is reported to cause lipotoxicity, which may lead to dysfunction and disease, but there have been few human studies to confirm this. This doctoral thesis set out to study the early pathophysiology of obesity in adults and children using in vivo magnetic resonance (MR) imaging and spectroscopy to assess the composition and function of the heart and liver in lean and obese individuals. The central tenet of this project was to establish and validate a clinically viable method for measuring the fat content of viscera safely and accurately, and to determine a normal range for the triglyceride content of the heart and liver. The initial study demonstrated that the heart remodels in response to weight loss, with over 20% reduction in LV mass, confirming that excess weight is genuinely a modifiable risk factor. Then, using spectroscopy, it was established that the healthy myocardium has a median triglyceride content of 0.37% (IQR 0.24% - 0.47%), which increases linearly in overweight and obese adults. Obesity, in the absence of any confounders, was also associated with a 10% reduction in cardiac contractile function. In comparison, healthy liver median lipid content was 0.67% (IQR 0.44% – 0.88%), which increased in obese adults to 2.9% (IQR 1.6% - 7.6%). There was a graded association between ectopic fat deposition in the liver and dyslipidaemia in adults, characterised by increased circulating triglycerides and reduced high-density lipoprotein. This dyslipidaemia may impair reverse cholesterol transport, and thus could be expected to exacerbate weight gain. Among obese and overweight subjects, there were some with severe steatosis and evidence of coexistent hepatic inflammation and fibrosis. To verify the accuracy of these spectroscopic measures for ectopic fat, a blinded, prospective comparison of non-invasive assessment of unselected liver disease in liver biopsy patients was completed. Liver disease presents with one or more of steatosis, fibrosis and haemosiderosis, all of which are associated with adverse cardiovascular outcomes. Fifty patients were recruited, and interobserver variability among pathologists was measured for histological reference standards for fat, fibrosis and iron deposition. MR measures of each of these metrics predicted the fibrosis, steatosis and haemosiderosis scores accurately. This enabled precise tissue characterisation of all forms of liver disease, including steatohepatitis, with one non-invasive test, to allow the diagnosis and monitoring of hepatic conditions. Lastly, all these new biomarkers of early cardiac and liver disease associated with excess weight were applied to obese and lean children, to understand whether ectopic fat played a substantial role in early life. Obese children had increased ectopic fat in their hearts and livers, as well as impaired strain, evidence of dyslipidaemia, and in some cases evidence of active steatohepatitis, comparable to adults with severe disease. The thesis therefore demonstrates that in vivo magnetic resonance techniques can be used for accurate measurement of visceral lipid content. Furthermore, there is evidence of significant ectopic fat deposition in both adults and children, with evidence of organ dysfunction, which raises the possibility that cardiovascular magnetic resonance may be of value to risk stratify obese individuals based on organ involvement. Finally, the developed methods may have broader applicability and offer a promising new method for the non-invasive diagnosis of chronic liver disease in other clinical settings.
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Lewandowski, Paul, and mikewood@deakin edu au. "Liver fat metabolism, obesity and diabetes in Psammomys Obesus." Deakin University. School of Health Sciences, 1999. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20050825.111432.

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Defects in fat metabolism are central to the aetiology and pathogenesis of obesity and type II diabetes. The liver plays a central role in these disease states via its regulation of glucose and fat metabolism. In addition, accumulation of fat within the liver has been associated with changes in key pathways of carbohydrate and fat metabolism. However a number of questions remain. It is hypothesised that fat accumulation within the liver is a primary defect in the aetiology and pathogenesis of obesity and type II diabetes. Fat accumulating in the liver is the result of changes in the gene expression of key enzymes and proteins involved with fat uptake, fat transport, fat oxidation, fat re-esterification or storage and export of fat from the liver and these changes are regulated by key lipid responsive transcription factors. To study these questions Psammomys obesus was utilised. This polygenic rodent model of obesity and type II diabetes develops obesity and diabetes in a similar pattern to susceptible human populations. In addition dietary and environmental changes to Psammomys obesus were employed to create different states of energy balance, which allowed the regulation of liver fat gene expression to be examined. These investigations include: 1) Measurement of fat accumulation and fatty acid binding proteins in lean, obese and diabetic Psammomys obesus. 2) Characterisation of hepatic lipid enzymes, transport protein and lipid responsive transcription factor gene expression in lean, obese and diabetic Paammomys obesus. 3) The effect of acute and chronic energy restriction on hepatic lipid metabolism in Psammomys obesus. 4) The effect of sucrose feeding on the development of obesity and type II diabetes in Psammomys obesus. 5) The effect of nicotine treatment in lean and obese Psammomys obesus, 6) The effect of high dose leptin administration on hepatic fat metabolism in Psammomys obesus. The results of these studies demonstrated that fat accumulation within the liver was not a primary defect in the aetiology and pathogenesis of obesity and type II diabetes. Fat accumulating in the liver was not the result of changes in the gene expression of key enzymes and proteins involved in hepatic fat metabolism. However changes in the mRNA level of the transcription factors PPAR∝ and SREBP-1C was associated with the development of diabetes and the gene expression of these two transcription factors was associated with changes in diabetic status.
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Gubík, Ladislav. "Automatic quantification of fat in liver using Computer Vision /." Leeds : University of Leeds, School of Computer Studies, 2008. http://www.comp.leeds.ac.uk/fyproj/reports/0708/Gubik.pdf.

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Crabtree, Christopher David. "Effects of Controlled Hypocaloric Ketogenic and Low-Fat Diets on Liver Fat in Overweight/Obese Adults." The Ohio State University, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1586780375128754.

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Shoebotham, Karen. "The relationship between liver fat content and unenhanced computed tomography." [New Haven, Conn. : s.n.], 2008. http://ymtdl.med.yale.edu/theses/available/etd-12092008-161843/.

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Lam, Shi, and 林璽. "The significance of hepatic stellate cell activation in small-for-sizefatty liver graft injury." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B45012933.

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Lee, Sang Jun. "CEACAM1 : a common regulator of fat metabolism and cell proliferation." Connect to full text in OhioLINK ETD Center, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=mco1218146004.

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Dissertation (Ph.D.)--University of Toledo, 2008.
"In partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biomedical Sciences." Title from title page of PDF document. Bibliography: p. 74-82, 116-124, 146-192.
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Books on the topic "Liver fat"

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Day, C. P., A. W. Lohse, P. R. Galle, and S. S. Thorgeirsson, eds. Liver under Constant Attack — From Fat to Viruses. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2759-7.

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C, Surrenti, ed. Fat-storing cells and liver fibrosis: Proceedings of the 71st Falk Symposium held in Florence, Italy, July 1-3, 1993. Dordrecht [The Netherlands]: Kluwer, 1994.

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Ewan, McVicar, ed. Cod liver oil and the orange juice: Reminiscences of a fat folk singer. Edinburgh: Mainstream, 1992.

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Huang, Zhaosheng. Fei pang bing zhi fang gan yu gao zhi xue zheng Zhong Xi yi zhen liao yu tiao yang =: Obesity fat liver and high-lipidemia. Xianggang: Tian heng wen hua chu ban gong si, 2001.

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La cuisine santé de Gabrielle. [Montréal]: Primeur/Sand, 1987.

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ill, Alphona Adrian, and Norton Mike ill, eds. Runaways: Live fast. New York, NY: Marvel Worldwide, Incorporated, 2017.

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Eleuthera: Find your fate and live it, live your fate and die. Los Angeles: One Hand Publishing, 2011.

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Vicious: Too fast to live. [London]: Glitter Books, 2010.

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Vicki, Park, and Park Vicki, eds. The complete live! don't diet! Birmingham, Ala: Sweetwater Press, 1998.

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The hormone diet: Lose fat, gain strength, live younger longer. [Toronto]: Random House Canada, 2009.

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Book chapters on the topic "Liver fat"

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Negro, F. "Fat and viral liver disease." In Liver under Constant Attack — From Fat to Viruses, 41–49. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2759-7_6.

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O’Connor, Sean, and Paul Cohen. "In Vitro Approaches to Model and Study Communication Between Adipose Tissue and the Liver." In Thermogenic Fat, 151–58. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6820-6_15.

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Altun, Ersan, Mohamed El-Azzazi, and Richard C. Semelka. "Hepatic fat and iron deposition." In Liver imaging: MRI with CT correlation, 241–54. Hoboken, NJ, USA: John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781118484852.ch12.

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Szabo, G. "Lipopolysaccharide in liver disease." In Liver under Constant Attack — From Fat to Viruses, 107–20. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2759-7_12.

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Bongers, Malte Niklas. "Gastrointestinal Imaging: Liver Fat and Iron Quantification." In Spectral Imaging, 235–44. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96285-2_15.

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Rosenberg, W., and I. N. Guha. "Non-invasive serum markers of liver fibrosis." In Liver under Constant Attack — From Fat to Viruses, 157–62. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2759-7_16.

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Denzer, U. W. "Assessment of liver fibrosis: gold standard biopsy?" In Liver under Constant Attack — From Fat to Viruses, 163–73. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2759-7_17.

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Thorgeirsson, S. S. "Molecular pathogenesis of liver cancer: early lesions." In Liver under Constant Attack — From Fat to Viruses, 53–57. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2759-7_7.

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Shetty, S., Y. Oo, and D. H. Adams. "Lymphocyte recruitment to the liver; the role of chemokines." In Liver under Constant Attack — From Fat to Viruses, 97–106. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2759-7_11.

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Takamura, Toshinari, Hirofumi Misu, and Shuichi Kaneko. "Ectopic Fat Accumulation in the Liver and Glucose Homeostasis." In Musculoskeletal Disease Associated with Diabetes Mellitus, 185–99. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-55720-3_13.

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Conference papers on the topic "Liver fat"

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Zheng, Yijiang, Qiyue Wang, and James K. Hahn. "Liver Fat Assessment with Body Shape." In 2022 44th Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2022. http://dx.doi.org/10.1109/embc48229.2022.9871321.

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Sumitpaibul, Pawesuda, Anurak Damrongphithakkul, and Ukrit Watchareeruetai. "Fat detection algorithm for liver biopsy images." In 2014 International Electrical Engineering Congress (iEECON). IEEE, 2014. http://dx.doi.org/10.1109/ieecon.2014.6925850.

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Verbrugghe, Adronie, and Alexandra Rankovic. "Dietary choline in feline nutrition and its role in obesity prevention and liver health." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/gyun6061.

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In North America, 60% of cats is estimated to be overweight or obese, predisposing cats to obesity-related health consequences, which reduce quality and quantity of life. Weight loss is recommended, yet current protocols are often unsuccessful. Moreover, drastic energy restriction can put overweight and obese cats at risk of developing feline hepatic lipidosis which can be fatal if left untreated.Choline, an essential nutrient, can be found naturally in organ meats, eggs, soybean and wheat germ. Commercial pet foods contain a supplemental source of choline to meet the recommendations set by the Association of American Feed Control Officials. For cats, 2400 mg/kg diet on a dry matter basis or 600 mg/1000 kcal metabolizable energy is recommended for growth and adult maintenance. Choline is a methyl group donor involved in multiple metabolic pathways and plays an important role in fat metabolism and mobilization, particularly in the liver. Choline is a precursor for phosphatidylcholine, an essential component of very-low-density lipoproteins, crucial for exporting triglycerides and cholesterol out of the liver and into circulation. Research in many animal species, including cats, has found that a diet deficient in choline results in hepatic fat accumulation. Similarly in rats, high-fat diet-induced fatty livers were reversed with choline. Choline supplementation studies in the animal nutrition field have largely focused on growth and weight gain in livestock. This research uncovered decreased fat deposition and increased lean carcass composition with supplementation of choline or its derivative betaine. This session will summarize recent choline research in cats. The findings suggest that supplementing choline, above an animal’s recommended allowance, may help to reduce body fat gain in growing kittens after neutering, and help mobilize fats from the liver in overweight and obese cats, proposing a novel nutritional strategy for obesity prevention and liver health in domestic cats.
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Kriston, Andras, Paulo Mendonça, Alvin Silva, Robert G. Paden, William Pavlicek, Dushyant Sahani, Benedek Janos Kis, Laszlo Rusko, Darin Okerlund, and Rahul Bhotika. "Liver fat quantification using fast kVp-switching dual energy CT." In SPIE Medical Imaging, edited by Benoit M. Dawant and David R. Haynor. SPIE, 2011. http://dx.doi.org/10.1117/12.878206.

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Wu, Yashuo, Leonardo Lopez, Michael P. Andre, Rohit Loomba, Claude B. Sirlin, Mark A. Valasek, Matthew A. Wallig, William D. O'Brien, and Aiguo Han. "Liver Fat Droplet Dependency on Ultrasound Backscatter Coefficient in Nonalcoholic Fatty Liver." In 2020 IEEE International Ultrasonics Symposium (IUS). IEEE, 2020. http://dx.doi.org/10.1109/ius46767.2020.9251748.

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Sehestedt, C., M. Heidary Dastjerdi, O. Dirsch, U. Dahmen, and J. Himmel. "Measuring the fat content of liver tissue - Experimental setup -." In 2009 6th International Multi-Conference on Systems, Signals and Devices (SSD). IEEE, 2009. http://dx.doi.org/10.1109/ssd.2009.4956727.

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Yan, Zhennan, Chaowei Tan, Shaoting Zhang, Yan Zhou, Boubakeur Belaroussi, Hui Jing Yu, Colin Miller, and Dimitris N. Metaxas. "Automatic Liver Segmentation and Hepatic Fat Fraction Assessment in MRI." In 2014 22nd International Conference on Pattern Recognition (ICPR). IEEE, 2014. http://dx.doi.org/10.1109/icpr.2014.565.

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Aggarwal, Kunal, and Sandeep Panwar Jogi. "Automatic Liver and Subcutaneous Fat Segmentation from MRI-PDFF Images." In 2020 International Conference on Computational Performance Evaluation (ComPE). IEEE, 2020. http://dx.doi.org/10.1109/compe49325.2020.9200196.

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Arjmand, Alexandros, Alexandros T. Tzallas, Markos G. Tsipouras, Roberta Forlano, Pinelopi Manousou, Nikolaos Katertsidis, and Nikolaos Giannakeas. "Fat Droplets Identification in Liver Biopsies using Supervised Learning Techniques." In PETRA '18: The 11th PErvasive Technologies Related to Assistive Environments Conference. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3197768.3201554.

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Kahl, S., S. Gancheva, K. Straßburger, C. Herder, J. Machann, H. Katsuyama, S. Kabisch, et al. "Empagliflozin effectively reduces liver fat content in type 2 diabetes." In Diabetes Kongress 2019 – 54. Jahrestagung der DDG. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1688168.

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Reports on the topic "Liver fat"

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Ismaiel, Abdulrahman, Ayman Jaaouani, Daniel-Corneliu Leucuta, Stefan-Lucian Popa, and Dan-Lucian Dumitrascu. The Visceral Adiposity Index in Non-Alcoholic Fatty Liver Disease and Liver Fibrosis — Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2021. http://dx.doi.org/10.37766/inplasy2021.12.0056.

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Review question / Objective: The objective of the study was to compare the mean difference and AUROC of Visceral Adiposity Index (VAI) in NAFLD/NASH/liver fibrosis patients and controls in observational studies. Condition being studied: Nonalcoholic fatty liver disease (NAFLD) is a multi-system disease, being mainly a liver pathology involving excessive hepatic fat accumulation unrelated to alcohol consumption or other secondary causes of hepatic steatosis. It is an emerging cause of concern and increasing clinical burden, imposing a public health challenge. NAFLD is the most common chronic liver disease and is predicted to be the most common indication for a liver transplant in Western countries by 2030, owing to a prevalence of 25% worldwide. The visceral adiposity index (VAI) is a scoring system based on body mass index, triglycerides, high-density lipoproteins (HDLs), and waist circumferences (WCs).
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Moskalenko, O. L., O. V. Smirnova, E. V. Kasparov, and I. E. Kasparova. STRUCTURE OF PSYCHOLOGICAL DISORDERS IN PATIENTS WITH METABOLIC SYNDROME AND NON-ALCOHOLIC FAT LIVER DISEASE. Science and Innovation Center Publishing House, 2021. http://dx.doi.org/10.12731/2658-4034-2021-12-4-2-340-348.

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The article is devoted to the study of the psychological characteristics of the behavior of patients with non-alcoholic fatty liver disease (NAFLD). The manifestations of NAFLD are a powerful frustrating factor for patients, negatively affect the quality of life, hinder psychosocial adaptation and serve as the basis for the formation of chronic stress from the disease, which blocks the actual needs of the individual. Psychological factors are an important component in the clinical assessment of patients in connection with the individualization of the treatment process and secondary psychoprophylaxis, including methods of somato-centered and personality-centered psychotherapy.
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Yu, Mei, Becky Geiger, Nader Deeb, and Max F. Rothschild. Association of Liver X Receptor Alpha and Beta Genes with Carcass Lean and Fat Content in Pigs. Ames (Iowa): Iowa State University, January 2006. http://dx.doi.org/10.31274/ans_air-180814-812.

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Jin, Dachuan, Gao Peng, Shunqin Jin, Tao Zhou, Baoqiang Guo, and Guangming Li. Comparison of therapeutic effects of anti-diabetic drugs on non-alcoholic fatty liver disease patients without diabetes: A network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0014.

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Review question / Objective: To evaluate the efficacy of different anti-diabetic drugs in the treatment of non-diabetic non-alcoholic disease by network meta-analysis, and find the best intervention. Condition being studied: Non-alcoholic fatty liver disease (NAFLD) refers to the disease in which the liver fat content exceeds 5%, and excludes the secondary causes of alcohol, infection, drugs or other specific metabolic diseases. As a spectrum of disorders, it includes hepatocyte steatosis and steatohepatitis at the initial stage, liver fibrosis at the later stage, cirrhosis at the final stage, and even liver cancer. Nowadays Non-alcoholic fatty liver disease (NAFLD) has become the most common chronic liver disease in the world with an incidence rate as high as 25% which has been rising steadily worldwide in the past 30 years. Currently there are still no approved specific therapeutic agents and global treatment guidelines for NAFLD. For non-diabetic NAFLD, there is far from a consensus, too.
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Butler, Walter R., Uzi Moallem, Amichai Arieli, Robert O. Gilbert, and David Sklan. Peripartum dietary supplementation to enhance fertility in high yielding dairy cows. United States Department of Agriculture, April 2007. http://dx.doi.org/10.32747/2007.7587723.bard.

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Objectives of the project: To evaluate the effects of a glucogenic supplement during the peripartum transition period on insulin, hepatic triglyceride accumulation, interval to first ovulation, and progesterone profile in dairy cows. To compare benefits of supplemental fats differing in fatty acid composition and fed prepartum on hepatic triglyceride accumulation, interval to first ovulation, progesterone profile, and uterine prostaglandin production in lactating dairy cows. To assess the differential and carry-over effects of glucogenic and fat supplements fed to peripartum dairy cows on steroidogenesis and fatty acids in ovarian follicles. To determine the carry-over effects of peripartum glucogenic or fat supplements on fertility in high producing dairy cows (modified in year 3 to Israel only). Added during year 3 of project: To assess the activity of genes related to hepatic lipid oxidation and gluconeogenesis following dietary supplementation (USA only). Background: High milk yields in dairy cattle are generally associated with poor reproductive performance. Low fertility results from negative energy balance (NEBAL) of early lactation that delays resumption of ovarian cycles and exerts other carryover effects. During NEBAL, ovulation of ovarian follicles is compromised by low availability of insulin and insulin-like growth factor-I (IGF-I), but fatty acid mobilization from body stores is augmented. Liver function during NEBAL is linked to the resumption of ovulation and fertility: 1) Accumulation of fatty acids by the liver and ketone production are associated with delayed first ovulation; 2) The liver is the main source of IGF-I. NEBAL will continue as a consequence of high milk yield, but dietary supplements are currently available to circumvent the effects on liver function. For this project, supplementation was begun prepartum prior to NEBAL in an effort to reduce detrimental effects on liver and ovarian function. Fats either high or low in unsaturated fatty acids were compared for their ability to reduce liver triglyceride accumulation. Secondarily, feeding specific fats during a period of high lipid turnover caused by NEBAL provides a novel approach for manipulating phospholipid pools in tissues including ovary and uterus. Increased insulin from propylene glycol (glucogenic) was anticipated to reduce lipolysis and increase IGF-I. The same supplements were utilized in both the USA and Israel, to compare effects across different diets and environments. Conclusions: High milk production and very good postpartum health was achieved by dietary supplementation. Peripartum PGLY supplementation had no significant effects on reproductive variables. Prepartum fat supplementation either did not improve metabolic profile and ovarian and uterine responses in early lactation (USA) or decreased intake when added to dry cow diets (Israel). Steroid production in ovarian follicles was greater in lactating dairy cows receiving supplemental fat (unsaturated), although in a field trail fertility to insemination was not improved.
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Ismaiel, Abdulrahman, Oana Ciobanu, Mohamed Ismaiel, Daniel-Corneliu Leucuta, Stefan-Lucian Popa, Liliana David, Dilara Ensar, Nahlah Al Srouji, and Dan L. Dumitrascu. Atherogenic Index of Plasma in Non-Alcoholic Fatty Liver Disease: Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0043.

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Review question / Objective: P - Non-alcoholic fatty liver disease (NAFLD) I - Atherogenic index of plasma (AIP) C - Imaging and histopathology O - Mean difference and Area Under the Curve S - Observational studies. Condition being studied: Non-alcoholic fatty liver disease (NAFLD), is a common liver disease characterized by the presence of excessive fat build up within hepatocytes, in the absence of other conditions that result in hepatic steatosis and with little to no alcohol consumption. It refers to a broad range of conditions including steatosis, non-alcoholic steatohepatitis (NASH) and cirrhosis.
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Lung, Andrea. Live To Fly Fan Edition. Ames: Iowa State University, Digital Repository, 2013. http://dx.doi.org/10.31274/itaa_proceedings-180814-710.

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Trew, Noel J., John D. Camping, Robert A. Lee, and Corey R. Bean. Putting Lives on the Line: The Fast Rope Glove Challenge. Fort Belvoir, VA: Defense Technical Information Center, June 2012. http://dx.doi.org/10.21236/ada563981.

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Schulte, Kyle J., Thomas J. Baas, and Doyle E. Wilson. An Evaluation of Equipment and Procedures for the Prediction of Intramuscular Fat in Live Swine. Ames (Iowa): Iowa State University, January 2011. http://dx.doi.org/10.31274/ans_air-180814-123.

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Schwab, Clint R., and Thomas J. Baas. Development of a Model to Predict Intramuscular Fat in Live Pigs Using Real-Time Ultrasound. Ames (Iowa): Iowa State University, January 2006. http://dx.doi.org/10.31274/ans_air-180814-810.

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