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Dissertations / Theses on the topic 'Lipoprotein A'

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Published: 4 June 2021
Last updated: 25 July 2025

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1

Kohn, Meifania Monica. "Lipoprotein ontology: a formal representation of Lipoproteins." Thesis, Curtin University, 2013. http://hdl.handle.net/20.500.11937/1827.

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Lipoproteins serve as a mode of transport for the uptake, storage and metabolism of lipids. Dysregulation in lipoprotein metabolism, known as dyslipidaemia, is strongly correlated to various diseases such as cardiovascular disease. Lipoprotein Ontology provides a formal representation of lipoprotein concepts and relationships that can be used to support the intelligent retrieval of information, faciliate collaboration between research groups, and provide the basis for the development of tools for the diagnosis and treatment of dyslipidaemia.
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2

Soran, Handrean. "Glycation of Lipoproteins and the Role High Density Lipoprotein and Paraoxonase -1." Thesis, University of Manchester, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.532197.

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3

Espinosa, Garcia Irma Leticia. "Differential density lipoprotein profiling for the characterization of Lipoprotein(a)." Texas A&M University, 2006. http://hdl.handle.net/1969.1/4359.

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Lipoprotein(a) (Lp(a)) has been described as an emerging risk factor for cardiovascular disease. The complexity of the Lp(a) molecule sets a challenge for the determination of the risk it represents for the cardiovascular system. The objective of the present study was to develop a rapid method for the separation, purification, density measurement, and characterization of Lp(a) from serum using a procedure that is isoform independent. The objective was met by linking ultracentrifugation with affinity separations for the specific separation of Lp(a) from other lipoproteins. The mean density dist
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4

Hacquebard, Mirjam Rebecca. "Alpha-tocopherol acquisition by plasma lipoproteins and changes in lipoprotein profile after cardiac surgery." Doctoral thesis, Universite Libre de Bruxelles, 2008. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/216586.

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Alpha-tocopherol, the most abundant form of vitamin E in man, is transported in the circulation by plasma lipoproteins. It plays important roles, not only in preventing lipid peroxidation, but also in modulating several cell functions such as cell signaling and gene expression. While chylomicrons transport dietary alpha-tocopherol after intestinal absorption, LDL and HDL are the major carriers of alpha-tocopherol in fasting plasma and largely contribute to its delivery to cells and tissues. Exchanges of alpha-tocopherol occur between plasma lipoproteins. In addition, alpha-tocopherol transfers
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5

Ma, Feng. "Clinical Assessment of Anti-Atherogenic Function of HighDensity Lipoprotein (HDL)." Thesis, Sorbonne université, 2018. http://www.theses.fr/2018SORUS583.

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Il a été bien établi chez l’Homme qu'il existe une forte association entre les faibles concentrations plasmatiques de cholestérol associé aux lipoprotéines de haute densité (HDL-C) et le risque accru de maladie cardiovasculaire (MCV). Augmenter le taux de HDL-C a donc été proposé comme stratégie thérapeutique visant à réduire le risque de MCV. En effet, les HDL présentent de multiples fonctions athéroprotectrices, notamment la capacité d’efflux du cholestérol, ainsi que des activités antioxydantes, anti-inflammatoires, vasodilatatrices, cytoprotectrices, anti-infectieuses et anti- thrombotique
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6

Ooi, Esther M. M. "Regulation of lipoprotein transport in the metabolic syndrome : impact of statin therapy." University of Western Australia. School of Medicine and Pharmacology, 2007. http://theses.library.uwa.edu.au/adt-WU2007.0125.

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[Truncated abstract] The metabolic syndrome is characterized by cardiovascular risk factors including dyslipidemia, insulin resistance, visceral obesity, hypertension and diabetes. The dyslipidemia of the metabolic syndrome includes elevated plasma triglyceride and apolipoprotein (apo) B levels, accumulation of small, dense low-density lipoprotein (LDL) particles and low high-density lipoprotein (HDL) cholesterol concentration. However, the precise mechanisms for this dyslipoproteinemia, specifically low plasma HDL cholesterol, are not well understood. This thesis therefore, focuses on HDL, it
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7

Toledo, Júnior Alceu de Oliveira. "MARCADORES BIOQUÍMICOS NAS DISLIPIDEMIAS E NO RISCO CARDIOVASCULAR: ANÁLISE COMPARATIVA À FÓRMULA DE MARTIN." Universidade Estadual de Ponta Grossa, 2018. http://tede2.uepg.br/jspui/handle/prefix/2712.

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Submitted by Angela Maria de Oliveira (amolivei@uepg.br) on 2018-12-19T13:31:11Z No. of bitstreams: 2 license_rdf: 811 bytes, checksum: e39d27027a6cc9cb039ad269a5db8e34 (MD5) Alceu de Oliveira Toledo Junior.pdf: 3822777 bytes, checksum: 8621dbda843628c32379a4d6c54e0ac2 (MD5)<br>Made available in DSpace on 2018-12-19T13:31:11Z (GMT). No. of bitstreams: 2 license_rdf: 811 bytes, checksum: e39d27027a6cc9cb039ad269a5db8e34 (MD5) Alceu de Oliveira Toledo Junior.pdf: 3822777 bytes, checksum: 8621dbda843628c32379a4d6c54e0ac2 (MD5) Previous issue date: 2018-12-17<br>Este estudo tem como objetivo ava
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8

Gordon, Scott M. "The role of high density lipoprotein compositional and functional heterogeneity in metabolic disease." University of Cincinnati / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1353100684.

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9

Sledziecka, Anna Katarzyna. "Covalent lipoprotein(a) assembly : characterization of oxidase activity responsible for catalyzing covalent lipoprotein(a)." Kingston, Ont. : [s.n.], 2008. http://hdl.handle.net/1974/1596.

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10

Palm, Wilhelm, Marta M. Swierczynska, Veena Kumari, Monika Ehrhart-Bornstein, Stefan R. Bornstein, and Suzanne Eaton. "Secretion and Signaling Activities of Lipoprotein-Associated Hedgehog and Non-Sterol-Modified Hedgehog in Flies and Mammals." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-180911.

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Hedgehog (Hh) proteins control animal development and tissue homeostasis. They activate gene expression by regulating processing, stability, and activation of Gli/Cubitus interruptus (Ci) transcription factors. Hh proteins are secreted and spread through tissue, despite becoming covalently linked to sterol during processing. Multiple mechanisms have been proposed to release Hh proteins in distinct forms; in Drosophila, lipoproteins facilitate long-range Hh mobilization but also contain lipids that repress the pathway. Here, we show that mammalian lipoproteins have conserved roles in Sonic Hedg
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11

Gabel, Brent R. "Analysis of lipoprotein(a) assembly." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ35960.pdf.

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12

Rip, Jacob. "Lipoprotein lipase, hypertriglyceridemia and atherosclerosis." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2006. http://dare.uva.nl/document/28665.

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13

Zhang, Liyan. "Lipoprotein lipase - unstable on purpose? /." Doctoral thesis, Umeå : Department of Medical Biosciences, Physiological Chemistry, University of Umeå, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1058.

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14

Watson, Timothy David George. "Lipoprotein metabolism in the horse." Thesis, University of Glasgow, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335859.

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15

Gaw, Allan. "Molecular genetics of lipoprotein(a)." Thesis, University of Glasgow, 1995. http://theses.gla.ac.uk/4015/.

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The work presented in this thesis aims to extend our understanding of controlling mechanisms that influence the plasma concentration of lipoprotein(a) [LP(a)]. The techniques of apo(a) genotyping and apo(a) phenotyping are used to study Lp(a) in different ethnic and genetic groups to achieve this end. In chapter 3 the effective use of a commercially available monoclonal antibody is demonstrated. Two primary antibodies were compared and apo(a) isoform analysis was performed using an immunoblotting method. With both monoclonals it was possible to detect 0.05mg. dL-1 Lp(a). Distributions of plasm
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16

Dionne, Carole. "La déficience en lipoprotéine lipase chez les canadiens français : étude spatiale, génétique et généalogique /." Thèse, Québec : Université Laval, École des gradués, 1991. http://theses.uqac.ca.

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Mémoire (M.Sc.)-- Université du Québec à Chicoutimi, 1991.<br>"Mémoire présenté pour l'obtention du grade de maître en sciences (M.Sc.)" Ce mémoire a été réalisé à l'UQAC dans le cadre du programme de maîtrise en médecine expérimentale (volet génétique) extensionné de l'Un. Laval à l'UQAC. CaQCU CaQCU Bibliogr.: f. 82-86. Document électronique également accessible en format PDF. CaQCU
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17

Koska, Juraj, Hussein Yassine, Olgica Trenchevska, et al. "Disialylated apolipoprotein C-III proteoform is associated with improved lipids in prediabetes and type 2 diabetes." AMER SOC BIOCHEMISTRY MOLECULAR BIOLOGY INC, 2016. http://hdl.handle.net/10150/614755.

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The apoC-III proteoform containing two sialic acid residues (apoC-III2) has different in vitro effects on lipid metabolism compared with asialylated (apoC-III0) or the most abundant monosialylated (apoC-III1) proteoforms. Cross-sectional and longitudinal associations between plasma apoC-III proteoforms (by mass spectrometric immunoassay) and plasma lipids were tested in two randomized clinical trials: ACT NOW, a study of pioglitazone in subjects with impaired glucose tolerance (n = 531), and RACED (n = 296), a study of intensive glycemic control and atherosclerosis in type 2 diabetes patients.
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18

Wilson, Heather Marion. "High density lipoprotein subspecies and the control of lipoprotein metabolism in relation to coronary heart disease." Thesis, University of Aberdeen, 1990. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU031961.

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The concept that high plasma concentrations of high density lipoproteins (HDL) offers protection against coronary heart disease has been challenged on epidemiological grounds. This dispute may arise as a result of the heterogeneous nature of HDL in which only some subspecies exert a protective function while the major portion has a neutral role. In order to test this hypothesis, the distribution of HDL subfractions in 100 myocardial infarction (MI) survivors was examined within 12 hours of the onset of chest pain and before post-MI changes in lipoproteins occurred. These patients were shown to
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19

Ngqaneka, Thobile. "The impact of Niacin on PCSK9 levels in vervet monkeys (Chlorocebus aethiops)." University of Western Cape, 2020. http://hdl.handle.net/11394/7931.

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Magister Pharmaceuticae - MPharm<br>Cardiovascular diseases (CVDs) such as ischaemic heart diseases, heart failure and stroke remain a major cause of death globally. Various deep-rooted factors influence CVD development; these include but are not limited to elevated blood lipids, high blood pressure, obesity and diabetes. A considerable number of proteins are involved directly and indirectly in the transport, maintenance and elimination of plasma lipids, including high and low-density lipoprotein cholesterol (HDL-C and LDL-C). There are several mechanisms involved in the removal of LDL p
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20

Lester, Sandy Marie. "Lipoprotein subclass analysis by immunospecific density." [College Station, Tex. : Texas A&M University, 2008. http://hdl.handle.net/1969.1/ETD-TAMU-3123.

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21

Neuger, Lucyna. "Aspects on lipoprotein lipase and atherosclerosis." Doctoral thesis, Umeå : Univ, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-564.

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22

Potts, Jennifer Lucy. "Lipoprotein metabolism in human adipose tissue." Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334875.

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23

Loughrey, Clodagh Maria. "Lipoprotein oxidation in chronic renal failure." Thesis, Queen's University Belfast, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318949.

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24

Sani, Halimah Abdullah. "Mechanisms of control of lipoprotein lipase." Thesis, University of Newcastle Upon Tyne, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.386912.

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25

Hogg, Neil. "Oxidative modification of low density lipoprotein." Thesis, University of Essex, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.316228.

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26

Le, Riche Mia. "Lipoprotein X : biochemical predictors and detection by non-denaturing polyacrylamide gradient gel electrophoresis." Thesis, Stellenbosch : Stellenbosch University, 2007. http://hdl.handle.net/10019.1/18218.

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Assignment (MMed)--University of Stellenbosch, 2007.<br>ENGLISH ABSTRACT: Lipoprotein X (LpX) is an abnormal cholesterol-containing particle that may be present in the serum of subjects with cholestasis, lecithin:cholesterol acyltransferase (LCAT) deficiency and parenteral nutrition. The biochemistry, metabolism, clinical significance and laboratory analysis of LpX is discussed in this study. This laboratory-based project investigated icteric samples received at the Chemical Pathology laboratory, Tygerberg Hospital, for serum predictors of LpX and the use of a modified non-denaturing polyacryl
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27

Humphries, William Henry IV. "Intracellular degradation of low-density lipoprotein probed with two-color fluorescence microscopy." Diss., Georgia Institute of Technology, 2011. http://hdl.handle.net/1853/42832.

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The vesicle-mediated degradation of low-density lipoprotein (LDL) is an essential cellular function due to its role in cellular biosynthesis of membranes and steroids. Using multi-color single particle tracking fluorescence microscopy, the intracellular degradation of LDL was probed in live, intact cells. Unique to these experiments is the direct observation of LDL degradation using an LDL-based probe that increases fluorescence intensity upon degradation. Specifically, individual LDL particles were labeled with multiple fluorophores resulting in a quenched fluorescent signal. The characterist
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28

Huang, Annong. "Effects of Oxidized Low Density Lipoprotein on Nitric Oxide Production in Macrophages." Digital Commons @ East Tennessee State University, 1997. https://dc.etsu.edu/etd/2924.

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The effects of oxidatively modified low density lipoprotein (oxLDL) on atherogenesis may be partly mediated by alterations in nitric oxide (NO) production by macrophages. A major goal of this study was to identify the lipid components in oxLDL modulating NO production. The effect of a water soluble antioxidants (N-acetylcysteine) and lipid soluble antioxidant (alpha-tocopherol) on NO production in macrophages was also determined. A second goal was to determine if the effects of oxLDL occurred at the transcriptional level. Human LDL was oxidized using an azo-initiator 2,2$\sp\pr
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29

Hassel, Craig Alan. "LIPOPROTEIN RECEPTORS IN COPPER-DEFICIENT RATS: IN VITRO BINDING OF HIGH DENSITY LIPOPROTEIN SUBFRACTIONS TO LIVER MEMBRANES." Diss., The University of Arizona, 1986. http://hdl.handle.net/10150/183955.

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Three studies were conducted to determine whether the elevated plasma and HDL cholesterol levels observed in copper-deficient rats could be explained by the interaction of ¹²⁵I-HDL subfractions with liver membrane preparations in vitro. Rats from all studies were randomly divided into two dietary treatments, copper-deficient and adequate (0.7 mg and 8.0 mg Cu/kg diet, respectively). Deionized water and diet were provided ad libitum. After eight weeks, rats were exsanguinated, membranes prepared from livers, and plasma high density lipoproteins (HDL) isolated by ultracentrifugation and agarose
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30

Mohiman, Niloofar. "Étude de la biogénèse des lipoprotéines chez Corynebacterium glutamicum." Thesis, Paris 11, 2012. http://www.theses.fr/2012PA114865.

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En raison de leur contribution à la virulence bactérienne, les lipoprotéines et les membres de la voie de biogenèse des lipoprotéines représentent des cibles prometteuses pour la recherche de nouveaux antibiotiques. À la suite de translocation à travers la membrane interne la future lipoprotéine ancrée dans la membrane par l’intermédiaire de son peptide signal, va subir en premier lieu l’addition de sn-1,2-diacylglyceryle sur la fonction sulfhydryle de la future cystéine N-terminale de la lipoprotéine mature. Cette modification est catalysée par Lgt (prolipoprotéin diacylglycérol transférase)
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31

Ramsay, Amanda. "The role of high density lipoprotein subspecies in the control of lipoprotein metabolism in relation to clinical disorders." Thesis, University of Aberdeen, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338398.

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In order to examine the role of HDL in the control of lipoprotein metabolism, the levels of HDL subspecies, other lipoproteins, apolipoproteins and fatty acids were measured in four groups of subjects presenting clinical disorders which were possibly related to their lipid profiles. (1) A monoclonal antibody was raised against apolipoprotein A-I allowing the development of an ELISA. (2) In a comparison of 43 angina patients and 61 healthy control subjects, the levels of HDL<sub>2</sub>, HDL<sub>2b</sub> and HDL<sub>2a</sub> were lower in the angina group and this was compensated for by an incr
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32

Niu, Youguo. "Cardiac Lipoprotein Metabolism in Health and Diabetes." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491543.

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Triacylglycerols (TAG)-rich lipoproteins (TGRLPs), i.e. very-low-density lipoprotein (VLDL) and chylomicrons (CM), are believed to be important substrates for heart to produce ATP and to incorporate constituents of cardiomyocyte membranes. However, cardiac utilisation of TGRLPs has not been fully understood. Therefore, the aims of the study were to investigate metabolism of TGRLPs by the isolated 'working' rat heart under physiological and diabetic conditions. Radiolabelled VLDL and CM particles were prepared by an extended liver perfusion and a thoracic duct cannulation technique respectively
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33

Breznan, Dalibor. "High-density lipoprotein metabolism in the kidney." Thesis, University of Ottawa (Canada), 2002. http://hdl.handle.net/10393/6120.

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The kidney is believed to play a major role in the clearance and re-absorption of high density lipoprotein (HDL) particles from the blood. Experiments were undertaken to explore the specific sites of renal HDL metabolism in vivo and to investigate in vitro the factors that regulate the renal re-absorption of HDL by HKC-8 human proximal tubule (PT) cells. Perfusion of a rabbit renal artery with [3H]cholesteryl ester (CE) and 125I-protein labeled HDL particles showed that the kidneys are capable of filtering both apolipoprotein A-I (apoA-I) and whole HDL. A fluorescent microscopic study with th
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34

Herd, Sara L. "Exercise, postprandial lipaemia and lipoprotein lipase activity." Thesis, Loughborough University, 1997. https://dspace.lboro.ac.uk/2134/28431.

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Impaired clearance of triacylglycerol-rich lipoproteins contributes to atherogenesis. It can be argued that exercise may decrease the risk of atherosclerotic diseases through its potential to improve the metabolic capacity for triacylgycerol and hence, clearance of triacylglycerol-rich lipoproteins. The investigations described in this thesis focused on the influence of exercise on postprandial lipid and lipoprotein metabolism.
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35

Puckey, Loretto Helena. "The genetic regulation of lipoprotein (a) concentration." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289825.

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36

Gopalraj, Rangaraj K. "LOW DENSITY LIPOPROTEIN RECEPTOR AND ALZHEIMERS DISEASE." UKnowledge, 2009. http://uknowledge.uky.edu/gradschool_diss/697.

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Since apoE allele status is the predominant Alzheimers disease (AD) genetic risk factor, functional single nucleotide polymorphisms (SNPs) in brain apoE receptors represent excellent candidates for association with AD. Therefore, three low density lipoprotein receptor (LDLR) SNPs were evaluated by TaqMan allelic discrimination assays for association with AD and I found that certain haplotypes alter the odds of AD. A SNP within LDLR exon 12, rs688, was identified in silico as neutralizing a putative exon splicing enhancer (ESE). Since LDLR is a major apoE receptor in the brain, I hypothesized t
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37

朱瑞中 and Sui-chung Chu. "Regulation of lipoprotein uptake in mammalian cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2000. http://hub.hku.hk/bib/B31969707.

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38

McDowell, Andrew. "Modifications of low density lipoprotein and atherosclerosis." Thesis, Queen's University Belfast, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.314171.

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39

Oliver, Jason David. "Aspects of control of lipoprotein lipase expression." Thesis, University of Newcastle Upon Tyne, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335174.

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40

Fielding, Barbara Ann. "Studies of the action of lipoprotein lipase." Thesis, Manchester Metropolitan University, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361176.

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Chu, Sui-chung. "Regulation of lipoprotein uptake in mammalian cells." Hong Kong : University of Hong Kong, 2000. http://sunzi.lib.hku.hk/hkuto/record.jsp?B22088982.

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42

Karnas, Kimberly Joy. "Lipoprotein biosynthesis: Examination of the lipidation process." Diss., The University of Arizona, 2001. http://hdl.handle.net/10150/279787.

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Lipoproteins, protein-lipid complexes that have a polar exterior and a non-polar interior, have been found in many vertebrate and insect species, and their basic structure and function appear to be conserved. They facilitate the intercellular transport of hydrophobic lipids through aqueous media. Their synthesis requires an unusual process referred to as lipidation, whereby lipids are added to the protein component of the lipoprotein. Lipidation is thought to occur during or immediately following translation of these proteins, but how this process occurs is unknown. Of particular interest is t
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George, Ian M. R. "Immunolocalization and quantitation of avian lipoprotein lipase." Thesis, University of Edinburgh, 1993. http://hdl.handle.net/1842/28092.

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The overall aim of the project was to investigate the pools of LPL activity that exist within chicken tissues in vivo. Polyclonal and monoclonal anti-chicken LPL antibodies were produced using highly purified chicken adipose tissue LPL as an immunogen. An attempt was made to measure LPL at the luminal surface of the capillary endothelium of individual tissues using an iodinated anti?chicken LPL monoclonal antibody, injected intravenously, in vivo. However, the technique was not successful because high levels of specific antibody binding were not achieved. Heparin-release studies using the isol
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44

Blain, Jean-François. "Modulation of lipoprotein metabolism in response to brain inury and Alzheimer's disease : roles for apolipoprotein E4 and lipoprotein lipase." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=85886.

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From the association of the epsilon4 allele of apolipoprotein (apo) E with Alzheimer's disease (AD) to the more recent reports of reduced risks of developing the disease when taking cholesterol lowering agents, links seem to point for a central role for lipoprotein metabolism in the AD brain. While the association with apoE4 is the strongest and most reproduced risk factor for AD, roles for other proteins involved in lipoprotein metabolism in the periphery have not been thoroughly investigated.<br>Using the entorhinal cortex lesion (ECL) paradigm in human apoE-targeted replacement mice
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45

Khan, Tina. "Clinical outcomes, perfusion and vascular function in patients with refractory angina and raised lipoprotein (a), treated with lipoprotein apheresis." Thesis, Imperial College London, 2016. http://hdl.handle.net/10044/1/51551.

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Background: Angina which is refractory to conventional medical therapy and revascularisation is challenging to manage and novel therapeutic options are needed. Raised lipoprotein(a) is common in refractory angina and is an independent cardiovascular risk factor that can be reduced by lipoprotein apheresis. To date there is no randomised controlled data assessing the clinical benefit of lipoprotein apheresis in patients with refractory angina and raised lipoprotein(a). Methods: We conducted a randomised controlled trial in 20 patients with refractory angina and raised lipoprotein(a), with three
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Cavalcante, Marcela Frota. "Efeito da imunização passiva com fragmentos variáveis de cadeia única anti-LDL eletronegativa na aterosclerose experimental." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/9/9136/tde-07032013-162526/.

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A aterosclerose é uma doença crônico-inflamatória multifatorial com o envolvimento do sistema imunológico, sendo o resultado da interação de diferentes elementos celulares. A lipoproteína de baixa densidade eletronegativa [LDL(-)], capaz de induzir o acúmulo de ésteres de colesterol em macrófagos e a subsequente formação de células espumosas, desempenha um papel-chave na doença. Anticorpos recombinantes têm sido gerados nas últimas décadas, como o scFv (single chain fragment variable), com o intuito de serem utilizados como uma novas alternativas de prevenção para o surgimento da lesão. Diante
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47

Barrett, P. Hugh R. "The kinetic analysis and computer modelling of lipoprotein metabolism in man." Title page, table of contents and abstract only, 1989. http://web4.library.adelaide.edu.au/theses/09PH/09phb274.pdf.

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48

Henriquez, Ronald Rene. "Fluorometric sedimentation equilibrium for lipoprotein sub-class analysis." Thesis, [College Station, Tex. : Texas A&M University, 2007. http://hdl.handle.net/1969.1/ETD-TAMU-2027.

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49

del, Bas Prior Josep Maria. "Modulation of hepatic lipoprotein metabolism by dietary procyanidins." Doctoral thesis, Universitat Rovira i Virgili, 2007. http://hdl.handle.net/10803/8662.

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INTRODUCTION<br/>During the past decade, Nutrition research has been subjected to a shift of focus, from epidemiology and physiology to the comprensión of the molecular basis of nutrients actions.<br/>Thus, the new "-omics" disciplines transcriptomics, proteomics or metabolomics, provide the tools to understand the molecular mechanisms involved in the modulation of gene expression by nutrients. The study of the beneficial properties of wine procyanidins has not avoided this shift of focus. Thus, from the initial studies which defined the "French paradox", to nowadays, a wide array of studies h
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Erqou, Sebhat. "Lipoprotein(a) and the risk of vascular disease." Thesis, University of Cambridge, 2010. https://www.repository.cam.ac.uk/handle/1810/225182.

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Abstract:
Background: Lipoprotein(a) [Lp(a)] is composed of a low density-lipoprotein (LDL) particle and a glycoprotein molecule known as apolipoprotein(a) [apo(a)]. Apo(a) exists in several differently-sized isoforms and is responsible for the unique properties of Lp(a). Although Lp(a) has been known for the past 40 years its relationship with coronary heart disease (CHD) has not been characterized in sufficient detail. Whether Lp(a) causes CHD is not clear. Furthermore, the role of apo(a) isoform variation and other sources of Lp(a) heterogeneity (e.g., level of oxidized phospholipids) in Lp(a)-diseas
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