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Books on the topic 'Lipid Mediators of Inflammation (LMI)'

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Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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1

G, Bazán Nicholás, ed. Lipid mediators in ischemic brain damage and experimental epilepsy. Basel: Karger, 1990.

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2

International Conference on Advances in Prostaglandin, Leukotriene, and Other Bioactive Lipid Research (12th 2002 Istanbul, Turkey). Advances in prostaglandin, leukotriene, and other bioactive lipid research: Basic science and clinical applications. New York: Kluwer Academic/Plenum Publishers, 2003.

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3

International, Conference on Advances in Prostaglandin Leukotriene and Other Bioactive Lipid Research (12th 2002 Istanbul Turkey). Advances in prostaglandin, leukotriene, and other bioactive lipid research: Basic science and clinical applications. New York: Kluwer Academic/Plenum Publishers, 2003.

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4

Zeliha, Yazıcı, ed. Advances in prostaglandin, leukotriene, and other bioactive lipid research: Basic science and clinical applications. New York: Kluwer Academic/Plenum Publishers, 2003.

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5

V, Honn Kenneth, and International Conference on Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation, and Radiation Injury (4th : 1995 : Hong Kong), eds. Eicosanoids and other bioactive lipids in cancer, inflammation, and radiation injury 3. New York: Plenum Press, 1997.

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6

K, Nigam S., ed. Eicosanoids and other bioactive lipids in cancer, inflammation, and radiation injury: Proceedings of the 2nd international conference, September 17-21, 1991, Berlin, FRG. Boston: Kluwer Academic Publishers, 1993.

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7

G, Bazán Nicolás, ed. Lipid mediators in eye inflammation. Basel: Karger, 1990.

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8

De Sanctis, Juan Bautista, Martin Giera, and Danuta Radzioch, eds. Quo Vadis Lipid Mediators – Lipid Mediators Implication in Inflammation and Chronic Inflammatory Diseases. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88966-989-9.

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9

1958-, Robinson Clive, ed. Lipid mediators in allergic diseases of the respiratory tract. Boca Raton, Fla: CRC Press, 1994.

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10

Ray, Abhijit, and Punit Kumar Srivastava. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2016.

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11

Ray, Abhijit. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2011.

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12

Ray, Abhijit, and Punit Kumar Srivastava. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2016.

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13

Ray, Abhijit, and Punit Kumar Srivastava. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2018.

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14

Raeburn, D. Ed. Airways Smooth Muscle: Neurotransmitters, Amines, Lipid Mediators & Signal Transduction (Exs). Birkhauser, 1996.

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15

1953, Raeburn D., and Giembycz M. A. 1961-, eds. Airways smooth muscle: Neurotransmitters, amines, lipid mediators, and signal transduction. Basel: Birkhauser Verlag, 1995.

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16

Airways Smooth Muscle: Neurotransmitters, Amines, Lipid Mediators and Signal Transduction (Respiratory Pharmacology and Pharmacotherapy). Birkhauser Boston, 1995.

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17

L, Jones Robert, Lawrence J. Marnett, Kenneth V. Honn, Santosh Nigam, and Patrick Y.-K. Wong. Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation, and Radiation Injury 3. Springer London, Limited, 2013.

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18

Honn, Kenneth V. Eicosanoids and other Bioactive Lipids in Cancer, Inflammation, and Radiation Injury 3. Springer, 2013.

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19

(Editor), Santosh Nigam, Kenneth V. Honn (Editor), Lawrence J. Marnett (Editor), and Thomas Walden Jr (Editor), eds. Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation and Radiation Injury (Developments in Oncology). Springer, 1992.

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20

Advances in Prostaglandin, Leukotriene and Other Bioactive Lipid Research: Basic Science and Clinical Applications (Advances in Experimental Medicine and Biology). Springer, 2003.

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21

Parlato, Marianna, and Jean-Marc Cavaillon. Innate immunity and the inflammatory cascade. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0299.

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Inflammation results from a complex interaction between a large number of mediators able to induce each other and to favour the generation of other inflammatory molecules (e.g. free radicals, lipid mediators, and proteases). The perpetuation of inflammation by these cascades of mediators is favoured by their ability to induce coagulation, leukocyte recruitment, and cell and tissue alteration (apoptosis, necrosis, and barrier disruption). Other cascades of mediators occur to generate anti-inflammatory mediators favouring the healing process. A neuroendocrine loop and neuromediators from central and peripheral nervous system are also involved in the process, allowing a return to homeostasis.
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22

Monaco, Claudia, and Giuseppina Caligiuri. Molecular mechanisms. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0014.

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The development of the atherosclerotic plaque relies on specific cognate interactions between ligands and receptors with the ability to regulate cell recruitment, inflammatory signalling, and the production of powerful inflammatory and bioactive lipid mediators. This chapter describes how signalling is engaged by cell-cell surface interactions when the endothelium interacts with platelets and leukocytes enhancing leukocyte recruitment during atherogenesis. It also exemplifies intracellular signalling pathways induced by the activation of innate immune receptors, the most potent activators of inflammation in physiology and disease. Differences are highlighted in innate signalling pathways in metabolic diseases such as atherosclerosis compared to canonical immunological responses. Finally, the key lipid mediators whose production can affect endothelial function, inflammation, and atherosclerosis development are summarized. This Chapter will take you through these fundamental steps in the development of the atherosclerotic plaque by summarizing very recent knowledge in the field and highlighting recent or ongoing clinical trials that may enrich our ability to target cardiovascular disease in the future.
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23

Iversen, Leslie. Endocannabinoids. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190846848.003.0004.

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The endocannabinoids are part of a large family of lipid signaling molecules derived from arachidonic acid, including the prostaglandins and leukotrienes, which are important mediators of inflammation. Far less is known about the newer members of the endocannabinoid group, and it remains unclear whether they all play important functional roles. This chapter reviews the multiple members of this family and their biosynthesis and inactivation. Physiological functions, including retrograde synaptic signaling, control of energy metabolism, regulation of pain sensitivity, and cardiovascular control, are discussed. In addition, the chapter reports the synthesis of novel agonists, antagonists, and compounds inhibiting endocannabinoid inactivation as novel medicines.
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