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Relevant bibliographies by topics / Leukemia

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Academic literature on the topic 'Leukemia'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "Leukemia"

1

Yan, Ying, Peter Steinherz, Xiuqin Guan, Ann Jakubowski, Joesph P. McGuirk, and Richard J. O’Reilly. "Growth Potential of Human Leukemia Blast Cells in SCID Mice and Association with Prognosis of Human Acute Leukemias." Blood 104, no. 11 (2004): 1900. http://dx.doi.org/10.1182/blood.v104.11.1900.1900.

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Abstract We have described a severe combined immunodeficiency (SCID) mouse model that permits the subcutaneous growth of primary human acute leukemia blast cells into a measurable subcutaneous nodule which may be followed by the development of disseminated disease. Utilizing the SCID mouse model, we examined the ability of patient-derived leukemic blasts to generate leukemic growth in the animals after subcutaneous inoculation without conditioning treatment. Leukemia blasts derived from 133 patients with acute leukemias, (67 acute lymphoblastic leukemia (ALL) and 66 acute myeloid leukemia (AML

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Gilliland, D. Gary, Craig T. Jordan, and Carolyn A. Felix. "The Molecular Basis of Leukemia." Hematology 2004, no. 1 (2004): 80–97. http://dx.doi.org/10.1182/asheducation-2004.1.80.

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Abstract Major strides have been made in our understanding of the molecular basis of adult and pediatric leukemias. More than one hundred disease alleles have been identified and characterized in cell culture and murine models of leukemia. In some instances, molecularly targeted therapies have been developed based on these insights that are currently in clinical trials, such as small molecule inhibitors of FLT3. In addition, it has recently been appreciated that, as with normal hematopoiesis, there is a hierarchical organization among leukemic cells that includes a rare population of leukemic

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Horton, Sarah J., Vanessa Walf-Vorderwülbecke, Steve J. Chatters, Neil J. Sebire, Jasper de Boer, and Owen Williams. "Acute myeloid leukemia induced by MLL-ENL is cured by oncogene ablation despite acquisition of complex genetic abnormalities." Blood 113, no. 20 (2009): 4922–29. http://dx.doi.org/10.1182/blood-2008-07-170480.

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Abstract Chromosomal translocations involving 11q23 are frequent in infant acute leukemia and give rise to the formation of MLL fusion genes. The mechanism of leukemic transformation by these fusions has been the subject of numerous investigations. However, the dependence of acute leukemia on MLL fusion activity in vivo and the efficacy of targeting this activity to eliminate disease have not been established. We have developed a model for conditional expression of MLL-ENL in hematopoietic progenitor cells, in which expression of the fusion oncogene is turned off by doxycycline. Conditionally

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Abdel-Wahab, Omar, and Ross L. Levine. "Metabolism and the leukemic stem cell." Journal of Experimental Medicine 207, no. 4 (2010): 677–80. http://dx.doi.org/10.1084/jem.20100523.

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Acute leukemias are clonal disorders of hematopoiesis wherein a leukemic stem cell (LSC) acquires mutations that confer the capacity for unlimited self-renewal, impaired hematopoietic differentiation, and enhanced proliferation to the leukemic clone. Many recent advances in understanding the biology of leukemia have come from studies defining specific genetic and epigenetic abnormalities in leukemic cells. Three recent articles, however, further our understanding of leukemia biology by elucidating specific abnormalities in metabolic pathways in leukemic hematopoiesis. These studies potentially

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Jones, RJ, SJ Sharkis, CB Miller, EK Rowinsky, PJ Burke, and WS May. "Bryostatin 1, a unique biologic response modifier: anti-leukemic activity in vitro." Blood 75, no. 6 (1990): 1319–23. http://dx.doi.org/10.1182/blood.v75.6.1319.1319.

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Abstract Bryostatin 1, a macrocyclic lactone isolated from the marine bryozoan Bugula neritina, has demonstrated both antineoplastic activity against the murine P388 leukemia line in vivo and stimulatory activity against mouse and human hematopoietic progenitors. We studied the effects of bryostatin 1 on the growth of human leukemias in vitro. Bryostatin 1 inhibited 1 to 4 logs of clonogenic leukemia cell growth from three of four leukemia cell lines. Bryostatin 1 also inhibited, by at least 1 log, the proliferation of clonogenic acute nonlymphocytic leukemia (ANLL) cells from 10 to 12 patient

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Jones, RJ, SJ Sharkis, CB Miller, EK Rowinsky, PJ Burke, and WS May. "Bryostatin 1, a unique biologic response modifier: anti-leukemic activity in vitro." Blood 75, no. 6 (1990): 1319–23. http://dx.doi.org/10.1182/blood.v75.6.1319.bloodjournal7561319.

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Bryostatin 1, a macrocyclic lactone isolated from the marine bryozoan Bugula neritina, has demonstrated both antineoplastic activity against the murine P388 leukemia line in vivo and stimulatory activity against mouse and human hematopoietic progenitors. We studied the effects of bryostatin 1 on the growth of human leukemias in vitro. Bryostatin 1 inhibited 1 to 4 logs of clonogenic leukemia cell growth from three of four leukemia cell lines. Bryostatin 1 also inhibited, by at least 1 log, the proliferation of clonogenic acute nonlymphocytic leukemia (ANLL) cells from 10 to 12 patients with ne

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Dias, Sergio, Margaret Choy, Kari Alitalo, and Shahin Rafii. "Vascular endothelial growth factor (VEGF)–C signaling through FLT-4 (VEGFR-3) mediates leukemic cell proliferation, survival, and resistance to chemotherapy." Blood 99, no. 6 (2002): 2179–84. http://dx.doi.org/10.1182/blood.v99.6.2179.

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Abstract Similar to solid tumors, growth of leukemias may also be angiogenesis dependent. Furthermore, tyrosine kinase receptors specific to endothelial cells are expressed on certain subsets of leukemias. We have previously demonstrated the existence of a VEGF/VEGFR-2 autocrine loop on leukemic cells that supports their growth and migration. Here, we demonstrate that in response to leukemia-derived proangiogenic and proinflammatory cytokines such as basic fibroblast growth factor and IL-1, endothelial cells release increasing amounts of another vascular endothelial growth factor (VEGF) family

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Adamaki, Maria, Spiros Vlahopoulos, George I. Lambrou, Athanasios G. Papavassiliou, and Maria Moschovi. "Aberrant AML1 gene expression in the diagnosis of childhood leukemias not characterized by AML1-involved cytogenetic abnormalities." Tumor Biology 39, no. 3 (2017): 101042831769430. http://dx.doi.org/10.1177/1010428317694308.

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The AML1 ( acute myeloid leukemia 1) gene, a necessary prerequisite of embryonic hematopoiesis and a critical regulator of normal hematopoietic development, is one of the most frequently mutated genes in human leukemia, involving over 50 chromosome translocations and over 20 partner genes. In the few existing studies investigating AML1 gene expression in childhood leukemias, aberrant upregulation seems to specifically associate with AML1 translocations and amplifications. The aim of this study was to determine whether overexpression also extends to other leukemic subtypes than the ones karyoty

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Kogan, Scott C., Diane E. Brown, David B. Shultz та ін. "Bcl-2 Cooperates with Promyelocytic Leukemia Retinoic Acid Receptor α Chimeric Protein (Pmlrarα) to Block Neutrophil Differentiation and Initiate Acute Leukemia". Journal of Experimental Medicine 193, № 4 (2001): 531–44. http://dx.doi.org/10.1084/jem.193.4.531.

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The promyelocytic leukemia retinoic acid receptor α (PMLRARα) chimeric protein is associated with acute promyelocytic leukemia (APL). PMLRARα transgenic mice develop leukemia only after several months, suggesting that PMLRARα does not by itself confer a fully malignant phenotype. Suppression of apoptosis can have a central role in tumorigenesis; therefore, we assessed whether BCL-2 influenced the ability of PMLRARα to initiate leukemia. Evaluation of preleukemic animals showed that whereas PMLRARα alone modestly altered neutrophil maturation, the combination of PMLRARα and BCL-2 caused a marke

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Wang, Wei, Magdalena Czader, and Sa A. Wang. "Blood- and bone marrow–based mature T-cell and natural killer cell leukemias and lymphomas: a summary in the series of the 2023 SH/EAHP Workshop." American Journal of Clinical Pathology 164, no. 1 (2025): 7–25. https://doi.org/10.1093/ajcp/aqaf009.

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Abstract This session included 51 cases submitted to the workshop “Progress in T- and NK-cell Lymphomas and Leukemias” by the Society for Hematopathology and European Association for Haematopathology under “Blood/Bone Marrow–Based Mature T- and NK-Cell Leukemias/Lymphomas” or “T/NK-cell neoplasms with a Leukemic Presentation.” Entities encompassed T-cell prolymphocytic leukemia, T-cell large granular lymphocytic leukemia (LGLL), natural killer (NK)-LGLL/chronic lymphoproliferative disorder of NK cells, adult T-cell leukemia/lymphoma, aggressive NK-cell leukemia, and their mimics. Submitted cas

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Dissertations / Theses on the topic "Leukemia"

1

Cheung, Man-sze, and 張敏思. "Characterization of Leukemic stem cells in acute myeloid Leukemia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40687582.

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Cheung, Man-sze. "Characterization of Leukemic stem cells in acute myeloid Leukemia." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B40687582.

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Cornforth, Terri Victoria. "Characterising the cell biology of leukemic stem cells in acute myeloid leukemia." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:654b2176-fd50-427e-86f2-74e928054bef.

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Acute Myeloid leukemia (AML) is an aggressive haematological malignancy that mainly affects the elderly. Relapse is common and is thought to be due to the presence of chemotherapy resistant leukemic stem cells (LSC). Within the CD34+ disease (>5% of the blast cells expressing CD34) , two subtypes have been identified; an LMPP/GMPlike expanded type and a MPP/CMP-like expanded type, the former is the most common, accounting for around 80% of CD34+ AML. Both the GMP-like and LMPPlike expanded populations show LSC activity. To improve our understanding of the disease and gain better insight in to

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Dutta, Sayantanee. "CALM/AF10 leukemia." Diss., Ludwig-Maximilians-Universität München, 2014. http://nbn-resolving.de/urn:nbn:de:bvb:19-174268.

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Bagacean, Cristina. "Epigenetics in leukemia." Thesis, Brest, 2018. http://www.theses.fr/2018BRES0012.

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Les dérivés de la cytosine sont d’importantes modifications épigénétiques dont le rôle dans l’évolution de la leucémie lymphoïde chronique (LLC) n’est pas totalement exploré. Dans ce contexte, notre première étude vise à examiner le niveau global de la 5-methylcytosine (5-mCyt), 5-hydroxymethylcytosine (5-hmCyt), 5-carboxylcytosine (5-CaCyt) et 5-hydroxymethyluridine (5-hmU) dans des lymphocytes B purifiés de patients LLC (n=56) et d’individus sains (n=17). Les principaux acteurs de la régulation épigénétique (DNMT1/3A/3B, MBD2/4, TET1/2/3, SAT1) ont été évalués par PCR quantitative en temps r

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Papayannidis, Cristina <1980&gt. "Pre-clinical and clinical development of leukemia stem cell inhibitors in acute leukemias." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2013. http://amsdottorato.unibo.it/5596/1/TesiPapayannidisDottorato2013_Copy.pdf.

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In Leukemias, recent developments have demonstrated that the Hedgehog pathway plays a key-role in the peculiar ability of self renewal of leukemia stem cells. The aim of this research activity was to investigate, through a first in man, Phase I, open label, clinical trial, the role and the impact, mainly in terms of safety profile, adverse events and pharmacokinetics, of a Sonic Hedgehog inhibitor compound on a population of heavely pretreated patients affected by AML, CML, MF, or MDS, resistant or refractory to standard chemotherapy. Thirty-five patients have been enrolled. The drug was admin

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Papayannidis, Cristina <1980&gt. "Pre-clinical and clinical development of leukemia stem cell inhibitors in acute leukemias." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2013. http://amsdottorato.unibo.it/5596/.

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In Leukemias, recent developments have demonstrated that the Hedgehog pathway plays a key-role in the peculiar ability of self renewal of leukemia stem cells. The aim of this research activity was to investigate, through a first in man, Phase I, open label, clinical trial, the role and the impact, mainly in terms of safety profile, adverse events and pharmacokinetics, of a Sonic Hedgehog inhibitor compound on a population of heavely pretreated patients affected by AML, CML, MF, or MDS, resistant or refractory to standard chemotherapy. Thirty-five patients have been enrolled. The drug was admin

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Zhang, Lu [Verfasser]. "Immunogenicity of leukemia stem cells in acute myeloid leukemia / Lu Zhang." Ulm : Universität Ulm. Medizinische Fakultät, 2012. http://d-nb.info/1020022574/34.

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Xue, Liting. "Oncogene Function in Pre-Leukemia Stage of INV(16) Acute Myeloid Leukemia: A Dissertation." eScholarship@UMMS, 2014. https://escholarship.umassmed.edu/gsbs_diss/740.

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The CBFbeta-SMMHC fusion protein is expressed in acute myeloid leukemia (AML) samples with the chromosome inversion inv(16)(p13;q22). This fusion protein binds the transcription factor RUNX with higher affinity than its physiological partner CBFbeta and disrupts the core binding factor (CBF) activity in hematopoietic stem and progenitor cells. Studies in the Castilla laboratory have shown that CBFbeta-SMMHC expression blocks differentiation of hematopoietic progenitors, creating a pre-leukemic progenitor that progresses to AML in cooperation with other mutations. However, the combined function

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Xue, Liting. "Oncogene Function in Pre-Leukemia Stage of INV(16) Acute Myeloid Leukemia: A Dissertation." eScholarship@UMMS, 2010. http://escholarship.umassmed.edu/gsbs_diss/740.

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The CBFbeta-SMMHC fusion protein is expressed in acute myeloid leukemia (AML) samples with the chromosome inversion inv(16)(p13;q22). This fusion protein binds the transcription factor RUNX with higher affinity than its physiological partner CBFbeta and disrupts the core binding factor (CBF) activity in hematopoietic stem and progenitor cells. Studies in the Castilla laboratory have shown that CBFbeta-SMMHC expression blocks differentiation of hematopoietic progenitors, creating a pre-leukemic progenitor that progresses to AML in cooperation with other mutations. However, the combined function

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Books on the topic "Leukemia"

1

William, Dameshek, Gunz Frederick, and Henderson Edward S, eds. Leukemia. 6th ed. Saunders, 1996.

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Peacock, Judith. Leukemia. LifeMatters, 2000.

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Wilmoth, Lerner Adrienne, ed. Leukemia. Greenhaven Press, 2009.

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Eric So, Chi Wai, ed. Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6.

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Weissman, I. L., ed. Leukemia. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-69722-7.

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Goldsmith, Connie. Leukemia. Twenty-First Century Books, 2012.

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Parks, Peggy J. Leukemia. Daniel A. Leone, Publisher, 2009.

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B, Silverstein Virginia, and Nunn Laura Silverstein, eds. Leukemia. Enslow Publishers, 2000.

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National Institutes of Health (U.S.), ed. Leukemia. National Institutes of Health, 1985.

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Chamberlain, Joan. Leukemia. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Cancer Institute, 1987.

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Book chapters on the topic "Leukemia"

1

So, Chi Wai Eric. "An Overview: From Discovery of Candidate Mutations to Disease Modeling and Transformation Mechanisms of Acute Leukemia." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_1.

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Zeisig, Bernd B., and Chi Wai Eric So. "Retroviral/Lentiviral Transduction and Transformation Assay." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_10.

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Krivtsov, Andrei V., Yingzi Wang, Zhaohui Feng, and Scott A. Armstrong. "Gene Expression Profiling of Leukemia Stem Cells." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_11.

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Bonnet, Dominique. "Humanized Model to Study Leukemic Stem Cells." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_12.

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Wunderlich, Mark, and James C. Mulloy. "Model Systems for Examining Effects of Leukemia Associated Oncogenes in Primary Human CD34+ Cells via Retroviral Transduction." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_13.

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Schuringa, Jan Jacob, and Hein Schepers. "Ex Vivo Assays to Study Self-Renewal and Long-Term Expansion of Genetically Modified Primary Human Acute Myeloid Leukemia Stem Cells." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_14.

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Yeung, Jenny, and and Chi Wai Eric So. "Identification and Characterization of Hematopoietic Stem and Progenitor Cell Populations in Mouse Bone Marrow by Flow Cytometry." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_15.

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Sroczynska, Patrycja, Christophe Lancrin, Stella Pearson, Valerie Kouskoff, and Georges Lacaud. "In Vitro Differentiation of Embryonic Stem Cells as a Model of Early Hematopoietic Development." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_16.

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Ottersbach, Katrin, and Elaine Dzierzak. "Analysis of the Mouse Placenta as a Hematopoietic Stem Cell Niche." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_17.

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Garcia-Cuellar, Maria-Paz, Deniz Mederer, and Robert K. Slany. "Identification of Protein Interaction Partners by the Yeast Two-Hybrid System." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_18.

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Conference papers on the topic "Leukemia"

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Kundu, Srijit, Diptayan Jash, Rudrajit Dutta, Deeba Kannan, K. C. Prabu Shankar, and Fitri Yakub. "Leukemia Classification using Transfer Learning Models." In 2024 Second International Conference on Advances in Information Technology (ICAIT). IEEE, 2024. http://dx.doi.org/10.1109/icait61638.2024.10690484.

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S, Tharanipriya, Ajith Kumar R, Akash K, Aswin Babu S, and Sunilkumar S. "Leukemia Disease Detection Using Machine Learning Algorithm." In 2024 10th International Conference on Advanced Computing and Communication Systems (ICACCS). IEEE, 2024. http://dx.doi.org/10.1109/icaccs60874.2024.10716967.

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Bindhusha, Boda, Thati Swathi, Kothareddy Gowthami, Allu JayaPrakash, S. P. Velmurugan, and Jenyfal Sampson. "Leukemia Blood Cancer Detection Using Mobile Net." In 2025 3rd International Conference on Intelligent Data Communication Technologies and Internet of Things (IDCIoT). IEEE, 2025. https://doi.org/10.1109/idciot64235.2025.10914829.

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Kundu, Sulekha, Arpita Dutta, and Krishna Kumar Jha. "Analysis and Identification of Leukemia Using YOLOv8." In 2024 4th International Conference on Computer, Communication, Control & Information Technology (C3IT). IEEE, 2024. https://doi.org/10.1109/c3it60531.2024.10829424.

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Ahmed, Rayan, Laraib Nadeem, Maryam Shafique, Zameera Saleem, Ayesha Iqbal, and Hafsa Iqbal. "Potential of Vision Transformer in Leukemia Detection." In 2025 3rd International Conference on Intelligent Systems, Advanced Computing and Communication (ISACC). IEEE, 2025. https://doi.org/10.1109/isacc65211.2025.10969351.

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Sharma, Shweta, Shalli Rani, and R. Sujitha. "Leukemia Classification Using CNN and VGG19 Architecture." In 2025 International Conference on Intelligent Control, Computing and Communications (IC3). IEEE, 2025. https://doi.org/10.1109/ic363308.2025.10957277.

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Ahmed Mustafa, Srwa, and Gullanar M Hadi. "Automated Leukemia Detection using K-means Clustering for Feature Extraction." In 5TH INTERNATIONAL CONFERENCE ON COMMUNICATION ENGINEERING AND COMPUTER SCIENCE (CIC-COCOS'24). Cihan University-Erbil, 2024. http://dx.doi.org/10.24086/cocos2024/paper.1529.

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Leukemia is a kind of blood cancer that may cause significant damage to a person's general health. It is characterized by the production of an excessive number of white blood cells. To address leukemia quickly and effectively, it is important to have a diagnosis that is both correct and quick. Not too long ago, experts started using AI methods to help find cancer much earlier. One of the hardest parts of making a method to find leukemia is separating the nuclei from the rest of the picture. When medical staff use quick and accurate division methods, they can find patients faster and treat them

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DeMarco, B., M. O. Al-Qadi, S. S. Carson, and S. Ghosh. "Leukemic Pleural Effusion in Acute Myeloid Leukemia." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4863.

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Tanaka, H., N. Narahara, H. Sadakata, K. Andoh, N. Kobayashi, and T. Maekawa. "ANALYSIS OF LEUKEMIA PELT. TISSUE FACTOR BY WESTERN BLOTTING TECHNIQUE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643285.

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It has been reported that the tissue factor(TF) of monocytes and leukemic leukocytes is one of the trigger substances of disseminated intravascular coagulation(DIC) in leukemia patients. To assess the properties of TF of leukemia cells, their TF was analyzed by the method of Western blotting. Placenta TF was purified using Concanavalin-A affinity chromatography. Briefly, human placenta TF was extracted from placenta acetone powder using Triton X-100 extraction and purified by Concanavalin-A affinity chromatography and SDS-preparative PAGE. Hie final product of the purified placenta TF-apoprote

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Falang, A., G. M. Alessio, M. Donati, and T. A. Barbui. "DISSEMINATED INTRAVASCULAR COAGULATION (DIC) AND ACUTE LEUKEMIA:IDENTIFICATION OF A NEW CELLULAR PROCOAGULANT." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643661.

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There is an enhanced incidence (>50%) of severe coagulopathy in association with several types of acute leukemias. Cell associated procoagulants are considered important in this context. So far only a Tissue Factor (TF)-type procoagulant has been described in leukemic cells. We have set up here the experimentalconditions to identify other possible cellular procoagulants in leukemia. We have tested blast cell extracts from 21 patients with 5 different cytological subtypes (from Ml to M5 of acute non lymphoid leukemia (ANLL), according to theFAB classification, in order to assay whether they

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Reports on the topic "Leukemia"

1

Shlomchik, Warren D. Mechanisms of Graft-vs.-Leukemia Against a Novel Murine Model of Chronic Myelogenous Leukemia. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada443726.

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Shlomchik, Warren D. Mechanisms of Graft-vs. -Leukemia Against a Novel Murine Model of Chronic Myelogenous Leukemia. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada426337.

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Shlomchik, Warren D. Mechanisms of Graft-vs.-Leukemia against a Novel Murine Model of Chronic Myelogenous Leukemia. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada464064.

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Jangid, Ajay, Anurag Mishra, Rachit Raj, Sumit Kumar, Priyanka Munjal, and Neha Pandey. Chronic Myeloid Leukemia (CML) as Surgical Emergency. Science Repository, 2024. http://dx.doi.org/10.31487/j.ajscr.2024.01.02.

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Ileal perforation peritonitis is a critical surgical emergency often encountered in developing countries, commonly associated with typhoid fever, tuberculosis, trauma, and non-specific enteritis. This case report presents a unique instance of nonspecific enteritis associated with chronic myeloid leukemia (CML). A 16-year-old girl with a history of pulmonary tuberculosis presented with symptoms, leading to the diagnosis of ileal perforations and CML. Surgical intervention involved ileal resection and double barrel ileostomy. The postoperative course included complications and chemotherapy with

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Untaaveesup, Suvijak, Sasinipa Trithiphen, Kamolchanok Kulchutisin, et al. Genetic Alterations in Extramedullary Leukemia among Acute Myeloid Leukemia Patients: Insights from a Cohort Study and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.8.0091.

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Davis, Jalin, Summer Hood, Alex Miller, Sam Stein, and Kaylee Stem. Effectiveness of Aerobic Exercise for Adults with Leukemia. University of Tennessee Health Science Center, 2020. http://dx.doi.org/10.21007/chp.mot2.2020.0001.

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Shannon, Kevin M. Molecular Analysis of Preleukemic and Leukemic Bone Marrow from Children with Monosomy V Syndrome and Juvenile Chronic Myelogenous Leukemia. Defense Technical Information Center, 1996. http://dx.doi.org/10.21236/ada324280.

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Wang, Jean Y. Pathogenic Mechanism of Malignant Progression in Chronic Myelogenous Leukemia. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada446373.

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Haas, M. Radiation-induced leukemia: Comparative studies in mouse and man. Office of Scientific and Technical Information (OSTI), 1991. http://dx.doi.org/10.2172/6053623.

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Hingley, Sally. Psycho-social Aspects of Acute Lymphocytic Leukemia in Children. Portland State University Library, 2000. http://dx.doi.org/10.15760/etd.1616.

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