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Books on the topic 'LEMS'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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1

Gräfrath, Bernd. Lems Golem: Parerga und Paralipomena. Frankfurt: Suhrkamp, 1996.

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2

Jarzębski, Jerzy. Zufall und Ordnung: Zum Werk Stanisław Lems. Frankfurt am Main: Suhrkamp, 1986.

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3

Weltprothesen und Prothesenwelten: Zu den technischen Prognosen Arno Schmidts und Stanislaw Lems. Frankfurt am Main: P. Lang, 1991.

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4

Stanisa︡w Lems Prognose des Epochenendes: Die Bedrohung der menschlichen Kultur durch Wissenschaft, Technologie und Dogmatismus. Darmstadt: Wissenschaftliche Buchgesellschaft, 2000.

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5

Tęcza, Zygmunt. Das Wortspiel in der Übersetzung: Stanisław Lems Spiele mit dem Wort als Gegenstand interlingualen Transfers. Tübingen: Max Niemeyer Verlag, 1997.

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6

Ein zerebraler Schriftsteller und Philosoph namens Lem: Zur Rekonstruktion von Stanisław Lems Autoren- und Werkbild im deutschen Sprachraum anhand von Fallbeispielen. Wrocław: Wydawn. Uniwersytetu Wrocławskiego, 2003.

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7

1983-, Salmieri Daniel, ed. Around the world on eighty legs (more or less). New York: Scholastic Press, 2010.

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8

Nahma, Sandrow, Engquist Richard, and Goldfaden Abraham 1840-1908, eds. Kuni-Leml: Based on Avrom Goldfadn's "The Two Kuni-Lemls". New York: S. French, 1986.

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9

Krueger, Carol. Legs, legs, legs. Boulton Road, Stevenage: Badger, 1993.

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10

V, Alatalo Rauno, ed. Leks. Princeton, N.J: Princeton University Press, 1995.

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11

Höglund, Jacob. Leks. Princeton, NJ: Princeton University Press, 1995.

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12

Genardière, Philippe de La. Legs. [Paris]: Stock, 1991.

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13

Peter, Reynolds. Leos. Barcelona: RBA, 2006.

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14

Legs. Auckland, N.Z: Shortland Publications, 1989.

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15

William, Kennedy. Legs. Thorndike, Me: G.K. Hall, 2000.

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16

Leake, Diyan. Legs. Chicago, IL: Heinemann Library, 2007.

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17

Genardière, Philippe de La. Legs. [Paris]: Editions Stock, 1991.

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18

Grow more with less: [sustainable garden methods : less water, less work, less money]. Minneapolis, Minn: Cool Springs Press, 2013.

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19

Christie, Judy Pace. Hurry less, worry less at work. Nashville: Abingdon Press, 2009.

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20

Lesser, Marc. Less: Accomplishing more by doing less. Novato, Calif: New World Library, 2009.

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21

Lesser, Marc. Less: Accomplishing more by doing less. Novato, Calif: New World Library, 2009.

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22

Hurry less, worry less for moms. Nashville: Abingdon Press, 2011.

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23

Contact lens optics and lens design. 3rd ed. Edinburgh: Elsevier Butterworth-Heinemann, 2006.

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24

Christie, Judy Pace. Hurry less, worry less at work. Nashville, TN: Abingdon Press, 2009.

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25

Contact lens optics and lens design. 2nd ed. Oxford: Butterworth-Heinemann, 1995.

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26

Christie, Judy Pace. Hurry less, worry less for families. Nashville: Abingdon Press, 2010.

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27

Hurry less, worry less for families. Nashville: Abingdon Press, 2010.

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28

Hurry less, worry less at work. Nashville: Abingdon Press, 2009.

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29

Johnson, D. B. Four legs bad, two legs good! Boston: Houghton Mifflin, 2007.

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30

O'Brien, Jean. Lovely legs. Cliffs of Moher, County Clare, Ireland: Salmon Poetry, 2009.

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31

Sea legs. Tallahassee, FL: Bella Books, 2009.

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32

Paul, Lyons. Table legs. New York: New Amsterdam Books, 1989.

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33

Janette, Ahrens, ed. Less Vegas. New York, NY: Ahrens Editions, 2011.

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34

illustrator, Meadows Graham, ed. These legs. Bothell, WA: The Wright Group, 1997.

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35

Johnson, Susan. Hot legs. New York: Berkley Books, 2004.

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36

Arndt, Holger. Stanislaw Lems Prognose des Epochenendes. Wissenschaftliche Buchgesellschaft, 2001.

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37

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 46-Year-Old Man with Double Vision and Proximal Leg Weakness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0033.

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Lambert-Eaton myasthenic syndrome (LEMS) can be a difficult condition to diagnose. In this chapter, the clinical picture and characteristics are discussed. The pathophysiology is also reviewed. Features to distinguish LEMS from more common neuromuscular junction conditions such as myasthenia gravis are reviewed. We review features that distinguish between idiopathic and paraneoplastic forms, and we discuss the importance of tumor surveillance. Antibody testing and the correct electrodiagnostic strategy are presented. Treatment of LEMS is outlined.Lambert-Eaton syndrome (LES) is a difficult condition to diagnose. The unique clinical picture and its characteristics are discussed. This is especially true in the patient without a diagnois of cancer. Antibody testing and the correct electrodiagnostic strategy are presented. Treatment of LES is outlined.

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38

Halliday, David. Fundamentals of Physics 6e Part 1-5 Enchanced Prob Lems Set. John Wiley & Sons Inc, 2002.

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39

Sanders, Donald B. Clinical aspects of neuromuscular junction disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0023.

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Disorders that primarily impair neuromuscular transmission (NMT) produce weakness that characteristically affects certain muscle groups and varies with activity. Acquired, autoimmune myasthenia gravis (MG) is the most common of these disorders. Much less common are genetic abnormalities of the neuromuscular junction (NMJ), the Lambert–Eaton myasthenic syndrome (LEMS), and toxic effects of various biological and chemical agents. The diagnosis of MG or LEMS is suspected from the history and clinical findings, and is confirmed in most patients by the presence of specific auto-antibodies. The precise diagnosis of most genetic myasthenic syndromes may require sophisticated DNA analysis. Impaired NMT can be confirmed in all of these conditions by repetitive nerve stimulation (RNS) testing and measuring the neuromuscular jitter. Treatment of MG requires selecting among several therapeutic options, taking into consideration the clinical characteristics of the individual patient. Treatment of LEMS and genetic myasthenic syndromes is more limited.

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40

Miller, Aaron E., and Teresa M. DeAngelis. Lambert-Eaton Myasthenia Syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0023.

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Lambert-Eaton myasthenia syndrome (LEMS) is an autoimmune-mediated disorder of the neuromuscular junction, which involves autoantibodies to voltage-gated calcium channels on the presynaptic membrane. In this chapter, we discuss the characteristic clinical features of LEMS, its electrophysiological distinction from MG, as well as its paraneoplastic presentation. We also review the immunotherapeutic management options in cases with severe weakness and those refractory to tumor removal.

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41

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 72-Year-Old Female with Facial Weakness and Droopy Eyelids. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0030.

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Fatigable weakness is the hallmark of myasthenia gravis (MG). It may present with false localizing signs leading to an itinal incorrect diagnosis of a brainstem stroke. MRI scanning of the brain with specific sequences can rule out the diagnosis of stroke. Differential diagnosis of MG may also include also motor neuron disease. Electromyography is very helpful in confirming the diagnosis of motor neuron disease. The two major diseases of the neuromuscular junction are MG and Lambert-Eaton syndrome (LEMS). A table presents the differing characteristics of each. LEMS can be associated with malignancy and MG with thyoma. Laboratory examinations have greatly assisted in differentiating these two conditions. There is specific antibody testing for each condition. Repetitive stimulation and single fiber electromyography also improve diagnostic acumen.

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42

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Disorders of muscle and neuromuscular junction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0008.

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This chapter discusses the clinical features and evidence base for the pharmacological treatment of muscular disorders (inflammatory myopathies: polymyositis, dermatomyositis, and inclusion body myositis), mitochondrial myopathies, Duchenne muscular dystrophy (DMD), myotonic dystrophy, inherited neuromuscular channelopathies, non-dystrophic myotonias (myotonia congenita, paramyotonia congenita), periodic paralyses, acquired neuromyotonia (Isaac syndrome and Morvan syndrome), stiff person syndrome, and disorders of the neuromuscular junction (myasthenia gravis (MG), myasthenic crisis, and Lambert–Eaton myasthenic syndrome (LEMS).

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43

Laboratory for Engineering Man/Machine Systems, LEMS, system identification, model reduction and deconvolution filtering using Fourier based modulating signals and higher order statistics. Providence, R.I: Brown University, Division of Engineering, 1992.

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44

Spike Lees America PalsPolity America Through the Lens. Polity Press, 2013.

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45

Spike Lees America Palspolity America Through The Lens Series. Polity Press, 2013.

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46

Reddy, Ugan, and Nicholas Hirsch. Diagnosis, assessment, and management of myasthenia gravis and paramyasthenic syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0244.

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Diseases that affect the neuromuscular junction (NMJ) interfere with normal nerve transmission and cause weakness of voluntary muscles. The two most commonly encountered are acquired myasthenia gravis (MG) and the Lambert–Eaton myasthenic syndrome (LEMS). Acquired MG is an autoimmune disease in which antibodies are directed towards receptors at the NMJ. In 85% of patients, IgG antibodies against the postsynaptic acetylcholine receptor (AChR) are found (seropositive MG). The thymus gland appears to be involved in the production of these which cause an increase rate of degradation of AChR resulting in a decreased receptor density resulting in a reduced postsynaptic end-plate potential following motor nerve stimulation and leading to muscle weakness. Although all voluntary muscles can be affected, ocular, bulbar, respiratory, and proximal limb weakness predominates. In the majority of seronegative patients, an antibody directed towards a NMJ protein called muscle specific tyrosine kinase (MUSK) is found. Anti-MUSK MG is characterized by severe bulbar and respiratory muscle weakness. Diagnosis of MG requires a high degree of clinical suspicion coupled with pharmacological and electrophysiological testing, and detection of the various causative antibodies. Treatment of MG involves enhancing neuromuscular transmission with long-acting anticholinesterase agents and immunosuppression. Acute exacerbations are treated with either plasma exchange or intravenous immunoglobulin. Myasthenic crisis is associated with severe muscle weakness that necessitates tracheal intubation and mechanical ventilation. LEMS is an autoimmune disease in which IgG antibodies are directed towards the pre-synaptic voltage-gated calcium channels at the NMJ. It is often associated with malignant disease (usually small cell carcinoma of the lung). Autonomic dysfunction is prominent and patients show abnormal responses to neuromuscular blocking drugs.

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47

Rosenfeld, Myrna R., Maarten J. Titulaer, and Josep Dalmau. Overview. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0142.

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The chapter reviews a diversity of neurologic syndromes that are either proven or putatively autoimmune. The disorders affect all levels of the nervous system from neuromuscular junction, autonomic nervous system, spinal cord, brain, to retina. The pathologic substrates underlying the neurologic dysfunction are varied and include vasculitis (Behçets, Susac’s), encephalitis (NMDA receptor encephalitis), channelopathies (myasthenia, LEMS) and even an inflammatory granulomatous disease (neuro-sarcoid). The resulting syndromes cover myriad aspects of clinical neurology and neuropsychiatry. The understanding of and continued identification of autoimmune neurologic disorders is a very active area. With time some of these disorders may be moved to other sections while new disorders will join the autoimmune list. In this chapter we briefly review the emergence of the field of autoimmunity as it relates to the nervous system and make note of some of the open questions that remain.

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48

Legs, legs, legs (Wonder world I). The Wright Group, 1993.

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49

Less Safe Less Free. New Press, 2009.

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50

Farah, Toufee. The Future Commons: State Less Class Less Cash Less Tax Less. Ken & Scatter Publications, 1994.

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