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Books on the topic 'LDL'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 May 2024

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1

Cholesterol metabolism, LDL, and the LDL receptor. San Diego: Academic Press, 1990.

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2

Moga, Carmen. Low density lipoprotein apheresis for the treatment of familial hypercholesterolemia. Edmonton: Alberta Heritage Foundation for Medical Research, 2004.

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3

Makino, Naoko, and Tamio Teramoto. LDL koresuterōru, chūsei shibō o sageru! reshipi. Tōkyō: Nihon Hōsō Shuppan Kyōkai, 2008.

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4

Dayal, Surendra. LDL online 1998: Computer assisted legal research. Sydney: Butterworths, 1998.

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5

Shalom, Tsur, ed. A logical language for data and knowledge bases. New York: Computer Science Press, 1989.

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6

S, Pagano Irwin, and Strait Nathan B, eds. HDL and LDL cholesterol physiology and clinical significance. Hauppauge, NY: Nova Science Publishers, 2009.

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7

Modified lipoproteins in the pathogenesis of atherosclerosis. Austin: R.G. Landes Co., 1994.

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8

Naqvi, Shamim. A logical language for data and knowledge bases. New York: Computer Science Press, 1989.

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9

Yang, Chih-Chin. The interaction of bilirubin with human low density lipoprotein (LDL). Ottawa: National Library of Canada, 1995.

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10

Lewińska, Dorota. Możliwoʹsci wykorzystania pektyny jako materiału sorpcyjnego do usuwania LDL-cholesterolu. Warszawa: Instytut Biocybernetyki i Inżynierii Biomedycznej, 2000.

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11

F, Keaney John, ed. Oxidative stress and vascular disease. Boston: Kluwer Academic Publishers, 1999.

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12

Alfredo, Cantafora, ed. Metodi per l'analisi di mutazione nel gene del recettore LDL ricorrenti in Italia. Roma: Istituto superiore di sanità, 1998.

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13

Turun yliopisto. Cardiorespiratory Research Unit. and Turun yliopisto. Dept. of Medicine., eds. Factors influencing tracking of serum lipid values in children and young adults: A 9-year follow-up study. Turku: Turun Yliopisto, 1991.

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14

Gotō, Yūichirō. Global perspectives in lipid management: Regulating LDL-cholesterol, HDL-cholesterol, and triglycerides : their role in primary prevention of CHD. London: Royal Society of Medicine Press, 1998.

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15

N, Holmqvist Gunnar, ed. Statins: Indications and uses, safety and modes of action. New York: Nova Science Publishers, 2009.

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16

Johnston, Philip. A study regarding the stability of peroxynitrite and its ability to oxidatively modify the protein moities of HDL and LDL cholesterol. [S.l: The Author], 1996.

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17

N, Holmqvist Gunnar, ed. Statins: Indications and uses, safety and modes of action. New York: Nova Science Publishers, 2009.

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18

N, Holmqvist Gunnar, ed. Statins. New York: Nova Science Publishers, 2008.

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19

Jubran, Khalil Jubran. Al-nabi tarjamah muwaziyah ah lil lil-nassayin lal-injilizi wa-al-'arabi. Cairo: Dar al-Shuruq, 2000.

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20

M, Gotto Antonio, and Gotō Yūichirō 1922-, eds. Global perspectives in lipid management: Regulating LDL-cholesterol, HDL-cholesterol and triglycerides : their role in the primary prevention of CHD : proceedings of a symposium sponsored by Parke-Davis Pharmaceuticals held in London, UK, 9-10 October 1997. London: Royal Society of Medicine Press, 1998.

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21

1924-, Scanu Angelo M., ed. Lipoprotein (a). San Diego: Academic Press, 1990.

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22

Ḥūrr, Nizār. Lil-waṭan ... lil-muqāwamah. Bayrūt: N. al-Ḥurr, 2001.

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23

Lester, Packer, and Ong Augustine S. H, eds. Biological oxidants and antioxidants: Molecular mechanisms and health effects. Champaign, Ill: AOCS Press, 1998.

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24

LDK. New York: Kodansha America, Incorporated, 2017.

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25

Sara, Linsley, and Dashiell Christine 1984 translator, eds. LDK. New York: Kodansha America, Incorporated, 2016.

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26

Ayu, Watanabe. LDK. New York: Kodansha America, Incorporated, 2016.

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27

LDK. New York: Kodansha America, Incorporated, 2016.

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28

Cholesterol Metabolism, Ldl, and the Ldl Receptor. Elsevier, 1990. http://dx.doi.org/10.1016/b978-0-12-512300-6.x5001-7.

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29

Myant, N. Cholesterol Metabolism, LDL, and the LDL Receptor. Elsevier Science & Technology Books, 2012.

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30

The LDL Sicilian. Thinkers' Press, 1987.

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31

Abuhah. Closed Road/Ldl 556. Intl Book Centre, 1992.

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32

Jibran. Tear & Smile/Ldl 562. Midsea Books, 1992.

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33

Wilson, Kay. Arab Horses/Ldl 4819. International Book Centre, Inc., 1986.

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34

Tarascon. Tarascon HTN/LDL Quick Reference Card 2007 (Tarascon, HTN/LDL Quick Reference Card). Tarascon Publishing, 2007.

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35

Christie, Agatha. Adventures of Poirot/Ldl 637. Intl Book Centre, 1992.

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36

Disney, Walt. Un Paseo En Bicicleta - LDL -. Grupo Editorial Norma, 1992.

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37

Disney, Walt. Huerto de Donald, El - LDL -. Grupo Editorial Norma, 1992.

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38

Woods, Dan. Three Short Stories/Ldl 4959. Intl Book Centre, 1990.

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39

Jubair. Rahlat Ibn Jubair/Ldl 8234. Midsea Books, 1992.

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40

Wiklund, Olov, and Jan Borén. Pathogenesis of atherosclerosis: lipid metabolism. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0011.

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Abstract:

Lipids are carried in plasma as microparticles, lipoproteins, composed of a core of hydrophobic lipids and a surface of amphipathic lipids. In addition, the particles carry proteins (i.e. apolipoproteins). The proteins have key functions in the metabolism as receptor ligands, enzymes or activators. Lipoproteins are classified based on density into: chylomicrons, VLDL, IDL, LDL, and HDL. Retention of apoB-containing lipoproteins (LDL, IDL, and VLDL) in the arterial intima is the initiating event in the development of atherosclerosis. Retention is mediated by binding of apoB to structural proteoglycans in the intima. Increased plasma concentration of apoB-containing lipoproteins is the main risk factor for atherosclerotic cardiovascular disease (CVD) and the causative role of LDL has been demonstrated in several studies. Lp(a) is a subclass of LDL and elevated Lp(a) is an independent risk-factor, primarily genetically mediated. Genetic data support that high Lp(a) causes atherosclerosis. Elevated triglycerides in plasma are associated with increased risk for CVD. Whether triglycerides directly induce atherogenesis is still unclear, but current data strongly support that remnant particles from triglyceride-rich lipoproteins are causal. HDL are lipoproteins that have been considered to be important for reversed cholesterol transport. Low HDL is a strong risk-factor for CVD. However, the causative role of HDL is debated and intervention studies to raise HDL have not been successful. Reduction of LDL is the main target for prevention and treatment, using drugs that inhibit the enzyme HMG-CoA reductase, i.e. statins. Other drugs for LDL reduction and to modify other lipoproteins may further reduce risk, and new therapeutic targets are explored.

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41

Clutterbuck, Mary. Man Who Never Laughed/Ldl 276. International Book Centre, Inc., 1987.

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42

Heap, J. Joha & the Three Merchants/Ldl 4827. Intl Book Centre, 1991.

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43

Christie, Agatha. Murder on the Orient Express/Ldl 601. Intl Book Centre, 1992.

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44

Leighton, Jessica. Predicators of total and LDL cholesterol response to dietary intervention. 1990.

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45

Grzega, Joachim. LdL für Pepe, Pfeiffer und die Pauker (German Edition). epubli GmbH, 2012.

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46

Tarascon. Tarascon Pocket Pharmacopoeia Quick Reference Card, 2006: HTN/LDL. 2nd ed. Tarascon Publishing, 2005.

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47

Pub, Tarascon. Tarascon HTN / LDL Quick Reference Card 2008 (Tarascon Pocket Pharmacopoeia). Not Avail, 2007.

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48

Jr, John F. Keaney. Oxidative Stress and Vascular Disease (Developments in Cardiovascular Medicine). Springer, 2000.

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49

Rebstock, Martin Jörg. Anwendung eines LDL-Apheresesystems mit zwei Dextransulfatadsorptionssäulen in Verbindung mit einer Blutzellzentrifuge. 1995.

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50

Brill, Janet. Cholesterol Down: Ten Simple Steps to Lower Your Cholesterol in Four Weeks--Without Prescription Drugs. Three Rivers Press, 2006.

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