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Books on the topic 'Latent inhibition'

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Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Lubow, R. E., and Ina Weiner, eds. Latent Inhibition. Cambridge: Cambridge University Press, 2009. http://dx.doi.org/10.1017/cbo9780511730184.

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2

Latent inhibition and conditioned attention theory. Cambridge [England]: Cambridge University Press, 1989.

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3

Schmajuk, Nestor A. Latent inhibition and its neural substrates. Boston: Kluwer Academic Pub., 2002.

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4

Latent inhibition and its neural substrates. Boston: Kluwer Academic Pub., 2002.

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5

Schmajuk, Nestor A. Latent Inhibition and Its Neural Substrates. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0841-0.

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6

Weiner, Ina, and Robert E. Lubow. Latent inhibition: Cognition, neuroscience, and applications to schizophrenia. Cambridge: Cambridge University Press, 2010.

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7

Wellman, N. A. Latent inhibition, negative priming and neuroleptics in schizophrenic patients and matched controls. [Guildford]: University of Surrey, 1994.

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8

Schmajuk, Nestor. Latent Inhibition and Its Neural Substrates. Springer, 2012.

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9

Schmajuk, Nestor. Latent Inhibition and Its Neural Substrates. Springer, 2002.

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10

Lubow, R. E. Latent Inhibition and Conditioned Attention Theory. Cambridge University Press, 2011.

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11

Schmajuk, Nestor. Latent Inhibition and Its Neural Substrates. Springer London, Limited, 2012.

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12

Lubow, R. E. Latent Inhibition and Conditioned Attention Theory. Cambridge University Press, 2009.

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13

Lubow, R. E. Latent Inhibition and Conditioned Attention Theory. Cambridge University Press, 2009.

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14

E, Lubow Robert, and Weiner Ina 1949-, eds. Latent inhibition: Cognition, neuroscience, and applications to schizophrenia. Cambridge: Cambridge University Press, 2010.

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15

Weiner, Ina, and Robert Lubow. Latent Inhibition: Cognition, Neuroscience and Applications to Schizophrenia. Cambridge University Press, 2010.

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16

Weiner, Ina, and Robert Lubow. Latent Inhibition: Cognition, Neuroscience and Applications to Schizophrenia. Cambridge University Press, 2010.

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17

Weiner, Ina, and Robert Lubow. Latent Inhibition: Cognition, Neuroscience and Applications to Schizophrenia. Cambridge University Press, 2010.

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18

Borgnis, Ramie Lynn. Latent inhibition of multiple unit activity and EEG in rabbit hippocampus. 1993.

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19

Brown, Rick D. Latent inhibition goes social: Reducing the expression of the attractiveness stereotype. 2004.

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20

Daskalakis, Zafiris J., and Robert Chen. Evaluating the interaction between cortical inhibitory and excitatory circuits measured by TMS. Edited by Charles M. Epstein, Eric M. Wassermann, and Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0012.

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Transcranial magnetic stimulation was first introduced in the late 1980s. Numerous studies have used TMS as an investigational tool to elucidate cortical physiology and to probe cognitive processes. This article introduces TMS paradigms and presents information gathered on cortical neuronal connectivity. TMS paradigms that demonstrate intracortical inhibition include short-interval cortical inhibition (SICI), cortical silence period (cSP) and long interval cortical inhibition (LICI). There are two types of cortical inhibitions from the stimulation of other brain areas, interhemispheric inhibition and cerebellum inhibition. The inhibition of the motor cortex can also be induced through the stimulation of peripheral nerves. This article talks about studies that describe interaction between inhibitory and facilitatory paradigms, the results of which are discussed in terms of cortical physiology and connectivity. The study of the interactions among cortical inhibitory and excitatory circuits may help to elucidate pathophysiology of neurological and psychiatric diseases.

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21

Atta-ur-Rahman and M. Iqbal Choudhary, eds. Frontiers in Cardiovascular Drug Discovery: Volume 4. BENTHAM SCIENCE PUBLISHERS, 2019. http://dx.doi.org/10.2174/97816810839951180401.

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Frontiers in Cardiovascular Drug Discovery is an eBook series devoted to publishing the latest advances in cardiovascular drug design and discovery. Each volume brings reviews on the biochemistry, in-silico drug design, combinatorial chemistry, high-throughput screening, drug targets, recent important patents, and structure-activity relationships of molecules used in cardiovascular therapy. The eBook series should prove to be of great interest to all medicinal chemists and pharmaceutical scientists involved in preclinical and clinical research in cardiology. The fourth volume of the series covers the following topics: -Aspirin administration -Adenosine receptor targeting for cardiovascular therapy -Drug treatment of patients with coronary stenting -Immunosuppressive drugs in heart transplantation -PCSK9 inhibition for lowering LDL-C levels.

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22

Hammond, Christopher J., Marc N. Potenza, and Linda C. Mayes. Development of Impulse Control, Inhibition, and Self-Regulatory Behaviors in Normative Populations across the Lifespan. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0082.

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Impulsivity represents a complex multidimensional construct that may change across the lifespan and is associated with numerous neuropsychiatric disorders including substance use disorders, conduct disorder/antisocial personality disorder, and traumatic brain injury. Multiple psychological theories have considered impulsivity and the development of impulse control, inhibition, and self-regulatory behaviors during childhood. Some psychoanalytic theorists have viewed impulse control and self-regulatory behaviors as developing ego functions emerging in the context of id-based impulses and inhibitory pressures from the superego. Object relationists added to this framework but placed more emphasis on mother–child dyadic relationships and the process of separation and individuation within the infant. Cognitive and developmental theorists have viewed impulse control and self-regulation as a series of additive cognitive functions emerging at different temporal points during childhood and with an emphasis on attentional systems and the ability to inhibit a prepotent response. Commonalities exist across all of these developmental theories, and they all are consistent with the idea that the development of impulse control appears cumulative and emergent in early life, with the age range of 24–36 months being a formative period. Impulsivity is part of normal development in the healthy child, and emerging empirical data on normative populations (as measured by neuropsychological testing batteries, self-report measures, and behavioral observation) suggest that impulse control, self-regulation, and other impulsivity-related phenomena may follow different temporal trajectories, with impulsivity decreasing linearly over time and sensation seeking and reward responsiveness following an inverted U-shaped trajectory across the lifespan. These different trajectories coincide with developmental brain changes, including early maturation of subcortical regions in relation to the later maturation of the frontal lobes, and may underlie the frequent risk-taking behavior often observed during adolescence.

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23

Mukherji, Deborah, Aurelius Omlin, Carmel Pezaro, and Johann De Bono. Novel therapies and emerging strategies for the treatment of patients with castration-resistant prostate cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0069.

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Castration-resistant prostate cancer (CRPC) represents a final stage of this malignancy for many men and is defined as the progression of prostate cancer despite castrate levels of testosterone. CRPC may present as a rising PSA, the development of new metastases, or worsening of known metastases. Recent advances have resulted in five new treatments for CRPC: the immunotherapy sipuleucel-T; the cytotoxic cabazitaxel; the androgen biosynthesis inhibitor abiraterone acetate; the radioisotope radium-223; and the antiandrogen enzalutamide. These have all improved overall survival in randomized phase III studies for patients with metastatic CRPC. Furthermore, multiple agents and combinations are currently in late-stage clinical testing. Men with advanced prostate cancer represent an important population for clinical and translational research and clinical trial participation should be considered as part of standard care.

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24

Aminoff, Michael J. Sir Charles Bell. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190614966.001.0001.

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Charles Bell (1774–1842) was a Scottish anatomist–surgeon whose original ideas on the nervous system have been equated with those of William Harvey on the circulation. He suggested that the anterior and posterior nerve roots have different functions, and based on their connectivity he showed that different parts of the brain have different functions. He noted that individual peripheral nerves actually contain nerve fibers with different functions, that nerves conduct only in one direction, that sense organs are specialized to receive only one form of sensory stimulus, and that there is a sixth (muscle) sense. In addition to the facial palsy and its associated features named after him, he provided the first clinical descriptions of several neurological disorders and important insights into referred pain and reciprocal inhibition. Bell helped to change the way art students are taught, described the anatomical basis of facial expressions, initiated the scientific study of the physical expression of emotions, and stimulated the later work of Charles Darwin on facial expressions. His teachings influenced British and European art. Bell was a renowned medical teacher who founded his own medical school, subsequently took over the famous Hunterian school, and eventually helped establish the University of London and the Middlesex Hospital Medical School in London. However, his belief in intelligent design caused him to be left behind by the evolutionist thought that developed in the nineteenth century. He was a brilliant but flawed human being who contributed much to the advance of knowledge.

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