Relevant bibliographies by topics / Langerhans

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Langerhans'

Author: Grafiati
Published: 4 June 2021
Last updated: 19 February 2022

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Journal articles on the topic "Langerhans"

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FUKAGAWA, Shuji, Yuichi YOSHIDA, Kazunori URABE, Tetsuya KOGA, Masutaka FURUE, Aiko SUMINOE, and Akinobu MATSUZAKI. "Langerhans Cell Histiocytosis." Nishi Nihon Hifuka 65, no. 3 (2003): 211–12. http://dx.doi.org/10.2336/nishinihonhifu.65.211.

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Haubrich, William S. "Langerhans of the Islets of Langerhans." Gastroenterology 129, no. 3 (September 2005): 783. http://dx.doi.org/10.1053/j.gastro.2005.07.037.

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Raica, Marius, and Anca Maria Cimpean. "Paul Langerhans." Acta medico-historica Adriatica 15, no. 1 (2017): 139–46. http://dx.doi.org/10.31952/amha.15.1.7.

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Devetnaesto stoljeće bilo je vrijeme prave revolucije u znanosti i medicini. U tom je stoljeću došlo do mnogih otkrića u medicini i biologiji, a mnoga od njih su se podudarala s primjenom mikroskopa (Hermann van Deijl) i uvođenjem standardne histološke tehnike (Paul Ehrlich). Glavne vrste tkiva i pojedinačne stanice karakterizirane su i izvorno klasificirane prije više od stotinu godina, iako je manje pozornosti bilo posvećeno njihovim osnovnim funkcijama. To je uglavnom bilo zbog modaliteta obrade uzoraka tkiva koja je omogućavala posebno detaljne deskriptivne studije. Ipak, primjećuju se neki pokušaji povezivanja strukture s funkcijom. Njemački znanstvenik Paul Langerhans, poznat po otkriću Langerhansovih otočića gušterače i Langerhansovih stanica u epidermisu, pokušao je promijeniti konvencionalnu sudbinu morfoloških istraživanja uvodeći već od početka svoje znanstvene karijere u svoja istraživanja funkcionalnu hipotezu koja se temelji na tradicionalnim mikroskopskim promatranjima. Paul Langerhans bio je složena ličnost druge polovice XIX. stoljeća ne samo u medicini nego i u drugim područjima biologije. U ovom radu predstavljen je njegov život i njegova istraživačka djelatnost ne samo zbog važnosti njegovih otkrića već i zbog gledišta koja su ta otkrića potaknula u neočekivanim područjima medicine i biologije.
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Corts, Magga. "Langerhans-Inseln." Deutsche Heilpraktiker-Zeitschrift 12, no. 01 (February 21, 2017): 61. http://dx.doi.org/10.1055/s-0037-1599814.

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Jolles, S. "Paul Langerhans." Journal of Clinical Pathology 55, no. 4 (April 1, 2002): 243. http://dx.doi.org/10.1136/jcp.55.4.243.

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Emile, Jean-François, Sylvie Fraitag, Michèle Leborgne, Yves de Prost, and Nicole Brousse. "Langerhans' cell histiocytosis cells are activated langerhans' cells." Journal of Pathology 174, no. 2 (October 1994): 71–76. http://dx.doi.org/10.1002/path.1711740202.

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Geissmann, Frederic, Yves Lepelletier, Sylvie Fraitag, Jenny Valladeau, Christine Bodemer, Marianne Debré, Michelle Leborgne, Sem Saeland, and Nicole Brousse. "Differentiation of Langerhans cells in Langerhans cell histiocytosis." Blood 97, no. 5 (March 1, 2001): 1241–48. http://dx.doi.org/10.1182/blood.v97.5.1241.

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Langerhans cell histiocytosis (LCH) consists of lesions composed of cells with a dendritic Langerhans cell (LC) phenotype. The clinical course of LCH ranges from spontaneous resolution to a chronic and sometimes lethal disease. We studied 25 patients with various clinical forms of the disease. In bone and chronic lesions, LCH cells had immature phenotype and function. They coexpressed LC antigens CD1a and Langerin together with monocyte antigens CD68 and CD14. Class II antigens were intracellular and LCH cells almost never expressed CD83 or CD86 or dendritic cell (DC)–Lamp, despite their CD40 expression. Consistently, LCH cells sorted from bone lesions (eosinophilic granuloma) poorly stimulated allogeneic T-cell proliferation in vitro. Strikingly, however, in vitro treatment with CD40L induced the expression of membrane class II and CD86 and strongly increased LCH cell allostimulatory activity to a level similar to that of mature DCs. Numerous interleukin-10–positive (IL-10+), Langerin−, and CD68+ macrophages were found within bone and lymph node lesions. In patients with self-healing and/or isolated cutaneous disease, LCH cells had a more mature phenotype. LCH cells were frequently CD14− and CD86+, and macrophages were rare or absent, as were IL-10–expressing cells. We conclude that LCH cells in the bone and/or chronic forms of the disease accumulate within the tissues in an immature state and that most probably result from extrinsic signals and may be induced to differentiate toward mature DCs after CD40 triggering. Drugs that enhance the in vivo maturation of these immature DCs, or that induce their death, may be of therapeutic benefit.
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THIERY M. "Paul Langerhans (1847-1888) en de eilandjes van Langerhans." Tijdschrift voor Geneeskunde 63, no. 5 (January 1, 2007): 211–13. http://dx.doi.org/10.2143/tvg.63.05.2000040.

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Yi, Wu, Wan-yuan Chen, Tian-xin Yang, Jian-ping Lan, and Wen-na Liang. "Langerhans cell sarcoma arising from antecedent langerhans cell histiocytosis." Medicine 98, no. 10 (March 2019): e14531. http://dx.doi.org/10.1097/md.0000000000014531.

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Yu, R. C., A. C. Chu, C. Chu, and L. Buluwela. "Clonal proliferation of Langerhans cells in Langerhans cell histiocytosis." Lancet 343, no. 8900 (March 1994): 767–68. http://dx.doi.org/10.1016/s0140-6736(94)91842-2.

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Dissertations / Theses on the topic "Langerhans"

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Chorro, Laurent. "Inside Langerhans cells." Paris 5, 2010. http://www.theses.fr/2010PA05T050.

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Les cellules de Langerhans (CLs) sont des cellules myéloïdes de l’épiderme, actuellement considérées en Immunologie comme des cellules dendritiques (CDs) originaires de la moelle osseuse. Contrairement aux autres cellules myéloïdes, comme les monocytes ou les CDs conventionnelles qui dérivent et se renouvellent à partir de cellules souches hématopoïétiques (SCHs), l’origine et les mécanismes de renouvellement des CLs restent encore aujourd’hui mal connus. Mon travail de thèse a pour but (i) de déterminer l’origine des CLs et (ii) de comprendre les mécanismes contrôlant leur homéostasie chez l’adulte en conditions normales ou inflammatoires. Mes résultats montrent que les CLs se développent à partir de précurseurs extra embryonnaires originaires du sac vitellin. Peu avant la naissance, ces précurseurs colonisent l’épiderme avant de se différentier en CLs et de proliférer. Chez l’adulte et en conditions normales, les CLs se maintiennent indépendamment des SCHs. Elles sont leurs propres précurseurs et s’auto renouvellent dans l’épiderme pour maintenir leur nombre. Dans un modèle murin de Dermatite atopique, le nombre des CLs est dramatiquement augmenté suite à la multiplication des ces cellules dans l’épiderme. Nous trouvons que la prolifération des CLs est contrôlée par un signal moléculaire produit par les kératinocytes de l’épiderme mais qui restent encore à identifier. Associés à de récents travaux sur la microglie du cerveau, mes résultats suggèrent que certaines populations de cellules myéloïdes, situées dans des tissues où le recrutement de leucocytes est limité ou délétère, suivent un modèle différent de développement chez l’embryon et d’homéostasie chez l’adulte
Langerhans Cells (LCs) are myeloid cells of the epidermis, currently featured in immunology textbooks as bone marrow-derived dendritic cells (DC). Unlike other myeloid cells such as monocytes and classical DCs that derive and continuously renew from haematopoietic stem cell (HSC) precursors, the exact origin and homeostasis of LCs remained unclear. My work aimed at (i) determining the origin of LCs and (ii) defining the mechanisms of LC homeostasis in adult during steady and inflammatory conditions. My results showed that LCs developed from an extra-embryonic precursor originating from the yolk sac, and not from HSC precursors, that colonize the epidermis during late embryogenesis and further acquire features of adult LCs during the neonatal period. In adult and under steady state conditions, the LC network is maintained independently from HSC precursors. Differentiated LCs are their own precursors and can self-renew in the epidermis to maintain their number in vivo. In a model of skin Atopic Dermatitis, LCs can increase their number by local proliferation and not by the recruitment of HSC precursors. We also found that proliferation of LCs is controlled by a yet unknown molecular signal derived from neighbouring keratinocytes. My results on LCs, together with recent works on brain microglia, suggest that selected populations of myeloid cells, located in tissues where leukocyte recruitment may be limited or deleterious, follow a different pathway of development in embryos and homeostasis in adult, where extra-embryonic precursors are recruited during development and maintained by self-renewal via molecular cues from surrounding cells
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Misery, Laurent. "Précurseurs des cellules de Langerhans." Lyon 1, 1995. http://www.theses.fr/1995LYO1T020.

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Alam, Sam Mohammad Kutubul. "Pathogenesis of Langerhans Cell Histiocytosis." Thesis, Imperial College London, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487289.

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The Langerhans cell (LC) is a histiocytic cell that represents a resident immigrant population in the epidermis and functions as a professional antigen presenting cell (APC). Langerhans cell histiocytosis (LCH) is a rare disorder that can affect both adults and children and can involve various organs. Clinically the disease ranges from a solitary lytic bone lesion, which may resolve following curettage, to a disseminated leukaemia-like disease with multiple organ involvement, which can be fatal. Despite considerable advances in our understanding of the disease, the aetiology ofLCH remains obscure. The aim of this study is to identify differentially expressed genes in LCH cell as compared to normal LC to gain novel insight into potential pathologic mechanisms involved in this disease. This has been carried out by using immunoaffinity cell purification against the LC marker CDla, a technique developed in our laboratory, together with a suppressive subtractive cDNA hybridisation (SSH) technique. Because of the limited LCH RNA material, an initial study was carried out to optimise conditions for SSH using HeLa cells and YCI cells, a derivative of HeLa in which CDla is constitutively expressed. By doing these experiments, it was found that the inclusion of an additional step to the subtraction protocol, namely size fractionation and purification of subtracted cDNA, allowed efficient recovery ofcDNA clones for the differentially expressed CDla gene. Using this optimised protocol, gene expression between normal LC and LCH cells obtained from bronchial alveolar lavage from one patient with LCH have been compared. A total of 291 differentially expressed clones were identified by differential screening and sequenced. The resulting sequences were used to search DNA sequence databases and the results used to classify sequences into three groups: (i) sequences that matched known human gene sequences, (ii) sequences that were found to originate from the pathogen Mycoplasma hyorhinis (M hyorhinis) and (iii) gene sequences of unknown origin. Using RT-PCR I have evaluated the expression of these in other LCH samples and have demonstrated one of the known human genes, argininosuccinate synthetase to be differentially expressed exclusively in 18 LCH-involved tissues from 17 LCH involved patients examined to date. Further, using RT-PCR, M hyorhinis sequences were also confirmed in six out of six additional LCH samples derived from bronchial alveolar lavage and two additional LCH samples involving gum. Taken together, these findings suggest that a discrete set of genes are differentially expressed in LCH, which may be important in the pathogenesis of this disease. Further, my data suggest the possible involvement of a novel infectious agent in the disease.
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Reis, e. Sousa Caetano Maria Pacheco Pais dos. "Phagocytosis of antigens by Langerhans cells." Thesis, University of Oxford, 1992. http://ora.ox.ac.uk/objects/uuid:1f69e0f0-1d08-4c2f-aa02-216231100f14.

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Mature dendritic cells (DC) isolated from lymphoid tissues initiate antigen-specific T-dependent responses even though they are non-phagocytic and weakly pinocytic, whereas Langerhans cells (LC; immature DC) can process protein antigens but are poorly immunostimulatory. Thus antigens may be acquired by cells of this lineage at an immature stage but, to our knowledge, there have been no studies on the phagocytic capacity of these cells in vitro. Using a newly-developed flow cytometric assay to measure the association between fluorescent markers and LC in epidermal cell cultures, and light and electron microscopy, we have observed phagocytosis of a variety of particles by freshly-isolated LC. The cells readily phagocytosed zymosan, heat-killed S. cerevisiae, bacteria (S. aureus and C. parvum) and fluorescent latex beads, but were unable to take up IgG- or complement-coated sheep erythrocytes, as opposed to MØ. Similarly, many freshly-isolated splenic DC had some phagocytic activity. However, the capacity of both LC and splenic DC to phagocytose zymosan, bacteria and fluorescent latex beads was markedly decreased after maturation in culture, consistently with the fact that mature DC are poorly phagocytic. Zymosan binding and uptake were much greater in fresh LC from C57BL/6 compared to BALB/c mice, and the loss of phagocytic capacity for zymosan during maturation followed different kinetics in the two strains. Two receptors mediating uptake of zymosan in LC were identified based on the effect of different inhibitors. Both of these receptors, recognising mannose and β-glucan residues, appear to be differentially regulated in the two mouse strains and during culture of LC. Our findings support the notion that DC are capable of acquiring particulate antigens for presentation at an immature stage, through recognition units for carbohydrate determinants common to a variety of potentially pathogenic organisms.
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Calming, Ulrika. "Langerhans cell histiocytosis : detection, monitoring and pathophysiology /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-180-2.

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Vedel, Jean. "Cellules de Langerhans humaines et molécules d'adhésion." Bordeaux 2, 1990. http://www.theses.fr/1990BOR23108.

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Ar'Rajab, Aamer. "Islet transplantation in the treatment of diabetes number of islets, functional regulation and metabolic control /." Lund : Dept. of Surgery, Lund University, 1991. http://catalog.hathitrust.org/api/volumes/oclc/38187937.html.

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Westerlund, Johanna. "Pulsatile insulin release from single islets of Langerhans." Doctoral thesis, Uppsala University, Department of Medical Cell Biology, 2000. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-494.

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Insulin release from single islets of Langerhans is pulsatile. The secretory activities of the islets in the pancreas are coordinated resulting in plasma insulin oscillations. Nutrients amplitude-regulate the insulin pulses without influencing their frequency. Diabetic patients show an abnormal plasma insulin pattern, but the cause of the disturbance remains to be elucidated. Ithe present thesis the influence of the cytoplasmic calcium concentratio([Ca2+]i) and cell metabolism on pulsatile insulin release was examined in single islets of Langerhans from ob/ob-mice. Glucose stimulation of insulin release involves closure of ATP-sensitive K+ channels (KATP channels), depolarization, and Ca2+ influx in β-cells. In the presence of 11 mM glucose, pulsatile insulin secretion occurs in synchrony with oscillations i[Ca2+]i. When [Ca2+]i is low and stable, e.g. under basal conditions, low amplitude insulin pulses are still observed. When [Ca2+]i is elevated and non-oscillating, e.g. when the β-cells are depolarized by potassium, high amplitude insulin pulses are observed. The frequency of the insulin pulses under these conditions is similar to that observed when [Ca2+]i oscillations are present. By permanently opening or closing the KATP channels with diazoxide or tolbutamide, respectively, it was investigated if glucose can modulate pulsatile insulin secretion when it does not influence the channel activity. Under these conditions, [Ca2+]i remained stable whereas the amplitude of the insulin pulses increased with sugar stimulation without change in the frequency. Metabolic inhibition blunted but did not prevent the insulin pulses. The results indicate that oscillations in metabolism can generate pulsatile insulin release when [Ca2+]i is stable. However, under physiological conditions, pulsatile secretion is driven by oscillations in metabolism and [Ca2+]i, acting in synergy.

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Bernstrand, Cecilia. "Langerhans cell histiocytosis : a clinical and immunological study /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-468-2/.

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Dandapat, Shera. "Studies of maturation in human epidermal Langerhans cells." Thesis, Imperial College London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.429394.

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Books on the topic "Langerhans"

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Workshop on Langerhans Cells. (2nd 1988 Lyon, France). The Langerhans cell =: La Cellule de Langerhans. London: J. Libby Eurotext, 1988.

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service), SpringerLink (Online, ed. The Islets of Langerhans. Dordrecht: Springer Science+Business Media B.V., 2010.

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Islam, Md Shahidul, ed. The Islets of Langerhans. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-3271-3.

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Islam, Md Shahidul, ed. Islets of Langerhans, 2. ed. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6884-0.

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Workshop on Langerhans Cells (2nd 1988 Lyon, France). The Langerhans cell =: La cellule de Langerhans : proceedings of the Second Workshop on Langerhans Cells, held in Lyon (France), April 21-22, 1988. Paris, France: Editions INSERM, 1988.

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Moll, Heidrun. The Immune Functions of Epidermal Langerhans Cells. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22497-7.

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Soria, Bernat, ed. Physiology and Pathophysiology of the Islets of Langerhans. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1819-2.

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Smith, Paul A. Electrophysiology of beta-cells from the Islets of Langerhans. Norwich: University of EastAnglia, 1988.

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Kiranadi, Bambang. Electrophysiology of B-cells from the islets of Langerhans. Norwich: University of East Anglia, 1990.

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Becker, Yechiel, ed. Skin Langerhans (Dendritic) Cells in Virus Infections and AIDS. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3942-1.

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Book chapters on the topic "Langerhans"

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Hien, Peter. "Langerhans-Zellhistiozytose." In Praktische Pneumologie, 611–13. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-10209-7_75.

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De Bartolo, Loredana, and Antonietta Messina. "Langerhans Islet." In Encyclopedia of Membranes, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-40872-4_713-1.

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De Bartolo, Loredana, and Antonietta Messina. "Langerhans Islet." In Encyclopedia of Membranes, 1087–89. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-44324-8_713.

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Bährle-Rapp, Marina. "Langerhans-Inseln." In Springer Lexikon Kosmetik und Körperpflege, 310. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_5803.

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Bährle-Rapp, Marina. "Langerhans-Zellen." In Springer Lexikon Kosmetik und Körperpflege, 310. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_5804.

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Kalil, Ricardo K. "Langerhans Cell Histiocytosis." In Tumors and Tumor-Like Lesions of Bone, 805–13. London: Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-6578-1_58.

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Yen, Michael T., and Scott Kelly. "Langerhans Cell Histiocytosis." In Encyclopedia of Ophthalmology, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-35951-4_202-3.

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Ruggieri, Pietro. "Langerhans Cell Histiocytosis." In Atlas of Musculoskeletal Tumors and Tumorlike Lesions, 45–50. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-01748-8_11.

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Morimoto, Akira. "Langerhans Cell Histiocytosis." In Encyclopedia of Cancer, 1975–78. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_3273.

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Leung, Alexander K. C., William Lane M. Robson, Carsten Büning, Johann Ockenga, Janine Büttner, Hartmut Schmidt, Antonio V. Delgado-Escueta, et al. "Langerhans Cell Granulomatosis." In Encyclopedia of Molecular Mechanisms of Disease, 1140. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6027.

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Conference papers on the topic "Langerhans"

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Švihlík, Jan, Jan Kybic, and David Habart. "Automated separation of merged Langerhans islets." In SPIE Medical Imaging, edited by Martin A. Styner and Elsa D. Angelini. SPIE, 2016. http://dx.doi.org/10.1117/12.2216798.

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Radzikowska, E., K. Blasinska-Przerwa, M. Jeśkiewicz-Kopij, R. Langfort, M. Szołkowska, K. Modrzewska, I. Bestry, et al. "Pulmonary Langerhans Cell Histiocytosis in Adults." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1423.

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Dimitropoulos, Charalampos, Ioannis Vamvakaris, Dimitris Vassos, George Hamalakis, Aggeliki Kokkini, Pandelis Giamboudakis, Konstantina Frangia, and Ioannis Ntanos. "Pulmonary Langerhans Cell Histiocytosis Or Miliary Tuberculosis?" In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2929.

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Cimen, Ozlem, Nagehan Emiralioğlu, Mina Gharibzadehhızal, Yagmur Bayindir, Sanem Eryilmazpolat, Dilber Ademhan, Berna Oguz, et al. "Severe Langerhans Cell Histiocytosis with Pulmonary Involvement." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa1064.

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Švihlík, Jan, Jan Kybic, David Habart, Zuzana Berková, Peter Girman, Jan Kříž, and Klára Zacharovová. "Classification of microscopy images of Langerhans islets." In SPIE Medical Imaging, edited by Sebastien Ourselin and Martin A. Styner. SPIE, 2014. http://dx.doi.org/10.1117/12.2043621.

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Siraj, K. M., and E. Klein. "Pulmonary Langerhans Cell Histiocytosis and IgA Nephropathy." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6300.

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Radzikowska, Elżbieta, Katarzyna Błasińska-Przerwa, Elżbieta Wiatr, Renata Langfort, and Kazimierz Roszkowski-Śliż. "Pneumothorax in patients with Langerhans cell histiocytosis." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa4747.

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Jouenne, Fanélie, Gwenael Lorillon, Carine Laurent-Issartel, Aurélie Sadoux, Véronique Meignin, Cristina Leschi, Emmanuelle Bugnet, Samia Mourah, and Abdellatif Tazi. "Genetic landscape of pulmonary langerhans cell histiocytosis." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.oa3781.

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Le Guen, Pierre, Sylvie Chevret, Emmanuelle Bugnet, Constance De Margerie-Mellon, Fanélie Jouenne, Gwenaël Lorillon, Agathe Seguin-Givelet, et al. "Pneumothorax in pulmonary langerhans cell histiocytosis (PLCH)." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2241.

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10

Rosse, C., B. Avirneni, and K. Lanka. "An Atypical Presentation of Pulmonary Langerhans Cell Histiocytosis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5024.

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Reports on the topic "Langerhans"

1

Larrick, James W., Vera Morhenn, Yawen L. Chiang, and Tim Shi. Activated Langerhans Cells Release Tumor Necrosis Factor. Fort Belvoir, VA: Defense Technical Information Center, January 1988. http://dx.doi.org/10.21236/ada206646.

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Guess, Jennifer C., and Dennis J. McCance. Decreased Migration of Langerhans Precursor-Like Cells in Response to Human Keratinocytes Expressing HPV-16 E6/E7 is Related to Reduced Macrophage Inflammatory Protein-3Alpha Production. Fort Belvoir, VA: Defense Technical Information Center, January 2005. http://dx.doi.org/10.21236/ada435872.

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You might also be interested in the extended bibliographies on the topic 'Langerhans' for particular source types:
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