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Relevant bibliographies by topics / Lambert-Eaton

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Academic literature on the topic 'Lambert-Eaton'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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Journal articles on the topic "Lambert-Eaton"

1

Goodrick, Steven. "Lambert-Eaton myasthenia." Lancet Neurology 14, no. 4 (April 2015): 357. http://dx.doi.org/10.1016/s1474-4422(15)70057-8.

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Pascuzzi, Robert, and Yong Kim. "Lambert-Eaton Syndrome." Seminars in Neurology 10, no. 01 (March 1990): 35–41. http://dx.doi.org/10.1055/s-2008-1041251.

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Sanders, Donald B. "Lambert-Eaton myasthenia?" Muscle & Nerve 53, no. 3 (January 23, 2016): 495. http://dx.doi.org/10.1002/mus.25001.

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Kesner, Vita G., Shin J. Oh, Mazen M. Dimachkie, and Richard J. Barohn. "Lambert-Eaton Myasthenic Syndrome." Neurologic Clinics 36, no. 2 (May 2018): 379–94. http://dx.doi.org/10.1016/j.ncl.2018.01.008.

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El Salem, K. "Lambert-Eaton myasthenic syndromes." Journal of the Neurological Sciences 405 (October 2019): 28–29. http://dx.doi.org/10.1016/j.jns.2019.10.078.

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Katirji, Basfhar. "Lambert-Eaton Myasthenic Syndrome." Journal of Clinical Neuromuscular Disease 1, no. 3 (March 2000): 134–36. http://dx.doi.org/10.1097/00131402-200003000-00004.

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Young, Jeffery D., and Jacqueline A. Leavitt. "Lambert–Eaton Myasthenic Syndrome." Journal of Neuro-Ophthalmology 36, no. 1 (March 2016): 20–22. http://dx.doi.org/10.1097/wno.0000000000000258.

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D'Amour, M., G. Gariepy, and J. Braidy. "Lambert-Eaton myasthenic syndrome." Canadian Medical Association Journal 176, no. 1 (December 13, 2006): 37. http://dx.doi.org/10.1503/cmaj.060767.

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Mareska, Michael, and Laurie Gutmann. "Lambert-Eaton Myasthenic Syndrome." Seminars in Neurology 24, no. 02 (July 15, 2004): 149–53. http://dx.doi.org/10.1055/s-2004-830900.

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Newsom-Davis, J. "Lambert-Eaton Myasthenic Syndrome." Revue Neurologique 160, no. 2 (February 2004): 177–80. http://dx.doi.org/10.1016/s0035-3787(04)70888-7.

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Dissertations / Theses on the topic "Lambert-Eaton"

1

Armbruster, Lena. "Lambert-Eaton Myasthenie Syndrom." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-112592.

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2

Muller, Lucien. "Le syndrome de Lambert Eaton : étude à propos de quatre cas." Université Louis Pasteur (Strasbourg) (1971-2008), 1989. http://www.theses.fr/1989STR1M083.

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Giovannini, Federica. "Voltage-dependent calcium channel subtypes at the mouse neuromuscular junction : evidence for the role of a resistant component." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365445.

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Pinto, Ashwin. "Specificity of autoantibodies in Lambert-Eaton myasthenic syndrome for neuronal calcium channels." Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342539.

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David, Pascale. "Préparation et caractérisation d'anticorps anti-canaux calcium neuronaux et caractérisation d'auto-anticorps dans le syndrome myasthénique de Lambert-Eaton." Aix-Marseille 2, 1993. http://www.theses.fr/1993AIX22043.

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Dans le systeme nerveux central, les canaux calcium dependant du potentiel assurent des fonctions essentielles comme, par exemple, le couplage excitation-secretion. Ils sont classes en quatre sous-types n, l, p et t, d'apres leurs proprietes electrophysiologiques et pharmacologiques. Le sous-type l ou recepteur dihydropyridine (dhp), qui est le plus abondant dans le muscle squelettique, est le mieux caracterise structuralement. Le sous-type n, ou recepteur de l'omega-conotoxine, est represente presque exclusivement dans le systeme nerveux central et sa caracterisation structurale fait l'objet de nombreux travaux actuellement. L'implication respective des trois sous-types n, l et p dans la liberation de neurotransmetteurs a ete demontree dans differents modeles neuronaux ou neuroendocrines. Le syndrome myasthenique de lambert-eaton (lems) est une perturbation de la transmission neuromusculaire, d'origine auto-immune, associee a une diminution de la liberation d'acetylcholine. Le cancer du poumon a petites cellules (sclc) est une tumeur frequemment associee au lems. Les immunoglobulines g (igg) des serums des patients lems induisent une diminution du nombre de canaux calcium fonctionnels et immunoprecipitent specifiquement le recepteur de l'omegaconotoxine. Les resultats les plus importants, presentes ici, peuvent se resumer en deux points. D'une part, nous avons demontre la presence d'autoanticorps, dans le serum des patients lems, diriges contre la synaptotagmine, une proteine des vesicules synaptiques. Nous avons egalement demontre, a l'aide d'un anticorps monoclonal anti-synaptotagmine, que la synaptotagmine est associee au recepteur de l'omegaconotoxine. Cette interaction doit etre importante pour l'amarrage des vesicules synaptiques a proximite de leur site de liberation dans les terminaisons presynaptiques. Nous proposons que l'interaction des auto-anticorps des patients lems avec la synaptotagmine soit impliquee dans l'etiologie du lems. D'autre part, nous avons demontre l'expression concomitante du recepteur de l'omegaconotoxine, de la synaptotagmine et de la syntaxine, une proteine specifique des terminaisons nerveuses, dans un certain nombre de lignees sclc. Nous faisons l'hypothese que l'expression de la synaptotagmine dans ce tissu neoplasique est a l'origine de la reponse autoimmune

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Chesnel, Sylvie. "Le syndrome de Lambert-Eaton : présentation d'une observation et revue des conceptions récentes." Caen, 1990. http://www.theses.fr/1990CAEN3063.

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RIFLE, SYLVIE. "Association syndrome de lambert-eaton et carcinome bronchique a petites cellules : revue de la litterature recente a propos de 2 cas." Clermont-Ferrand 1, 1990. http://www.theses.fr/1990CLF13021.

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Maddison, Paul. "A quantitative study of the immune-mediated neuromuscular disorders of acquired neuromyotonia and Lambert-Eaton myasthenic syndrome." Thesis, University of Newcastle Upon Tyne, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285378.

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Goulay-Dufaÿ, Sophie. "Contribution au développement analytique et bioanalytique d'une substance active utilisée dans le traitement d'une maladie rare : application au développement de la 3,4-Diaminopyridine." Paris 5, 2009. http://www.theses.fr/2009PA05P605.

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LANDES, PAUL. "Syndrome de gougerot-sjogren, cancer bronchopulmonaire a petites cellules, syndrome de lambert-eaton chez un meme malade : cette association est-elle fortuite ?" Toulouse 3, 1988. http://www.theses.fr/1988TOU31221.

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Books on the topic "Lambert-Eaton"

1

Jones, Dominic. Antibodies against the voltage-gated calcium channel in Lambert-Eaton myasthenic syndrome. Oxford: Oxford Brookes University, 2000.

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2

Miller, Aaron E., and Teresa M. DeAngelis. Lambert-Eaton Myasthenia Syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0023.

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Lambert-Eaton myasthenia syndrome (LEMS) is an autoimmune-mediated disorder of the neuromuscular junction, which involves autoantibodies to voltage-gated calcium channels on the presynaptic membrane. In this chapter, we discuss the characteristic clinical features of LEMS, its electrophysiological distinction from MG, as well as its paraneoplastic presentation. We also review the immunotherapeutic management options in cases with severe weakness and those refractory to tumor removal.

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3

P, Lisak Robert, ed. Handbook of myasthenia gravis and myasthenic syndromes. New York: M. Dekker, 1994.

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4

Katirji, Bashar. Case 21. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0025.

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Lambert-Eaton myasthenic syndrome is a rare yet very important neuromuscular disorder that may be difficult to confirm if not considered in the differential diagnosis. It is often misdiagnosed as myasthenic gravis or other nonspecific neuromuscular disorder. The electrodiagnostic findings in Lambert-Eaton myasthenic syndrome continue to be the cornerstone of the diagnosis. This case outlines the clinical and electrodiagnostic features of a patient with this syndrome and highlights the findings on repetitive nerve stimulation. It also discusses the practical approach in the search for occult malignancy. The distinguishing features among the various neuromuscular junction disorders are emphasized.

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Kaplan, Tamara, and Tracey Milligan. Neuromuscular Junction Disease (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190650261.003.0015.

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The video in this chapter explores neuromuscular junction disease. It outlines the steps in neuromuscular transmission, as well as the symptoms, causes, and treatments for Myasthenia Gravis, and Lambert Eaton Syndrome

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Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 46-Year-Old Man with Double Vision and Proximal Leg Weakness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0033.

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Lambert-Eaton myasthenic syndrome (LEMS) can be a difficult condition to diagnose. In this chapter, the clinical picture and characteristics are discussed. The pathophysiology is also reviewed. Features to distinguish LEMS from more common neuromuscular junction conditions such as myasthenia gravis are reviewed. We review features that distinguish between idiopathic and paraneoplastic forms, and we discuss the importance of tumor surveillance. Antibody testing and the correct electrodiagnostic strategy are presented. Treatment of LEMS is outlined.Lambert-Eaton syndrome (LES) is a difficult condition to diagnose. The unique clinical picture and its characteristics are discussed. This is especially true in the patient without a diagnois of cancer. Antibody testing and the correct electrodiagnostic strategy are presented. Treatment of LES is outlined.

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Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Disorders of muscle and neuromuscular junction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0008.

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This chapter discusses the clinical features and evidence base for the pharmacological treatment of muscular disorders (inflammatory myopathies: polymyositis, dermatomyositis, and inclusion body myositis), mitochondrial myopathies, Duchenne muscular dystrophy (DMD), myotonic dystrophy, inherited neuromuscular channelopathies, non-dystrophic myotonias (myotonia congenita, paramyotonia congenita), periodic paralyses, acquired neuromyotonia (Isaac syndrome and Morvan syndrome), stiff person syndrome, and disorders of the neuromuscular junction (myasthenia gravis (MG), myasthenic crisis, and Lambert–Eaton myasthenic syndrome (LEMS).

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Katirji, Bashar. Electrodiagnostic Findings in Neuromuscular Disorders. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0004.

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Neuromuscular disorders are often classified into four major categories: anterior horn cell disorders, peripheral neuropathies, neuromuscular junction disorders and myopathies. This chapter discusses the electrodiagnostic and clinical EMG findings in these various neuromuscular disorders. Peripheral neuropathies are subdivided into focal mononeuropathies, radiculopathies, plexopathies and generalized peripheral polyneuropathies. Focal peripheral nerve lesions and generalized peripheral polyneuropathies may be axonal or demyelinating, and manifest quite distinctly on nerve conduction studies. Neuromuscular junction disorders may be presynaptic, as seen with the Lambert-Eaton myasthenic syndrome, or postsynaptic, as seen with myasthenia gravis.

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Murray, E. Lee, and Veda V. Vedanarayanan. Neuromuscular Disorders. Edited by Karl E. Misulis and E. Lee Murray. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190259419.003.0021.

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The hospital neurologist may encounter neuromuscular disorders as known chronic conditions that are exacerbated by a hospital stay, be the principal reason for admission, or develop during a prolonged hospitalization. This chapter details the presentation, diagnosis, and management of conditions affecting the peripheral nerves and neuromuscular junction, such as myasthenia gravis, Lambert-Eaton (myasthenic) syndrome, botulism, and tick paralysis; as well as muscular weakness from various causes such as rhabdomyolysis, critical illness neuromyopathy, inflammatory myopathies, muscular dystrophies, periodic paralysis, and metabolic and endocrine myopathies. Also discussed are motoneuron degeneration, including amyotrophic lateral sclerosis and progressive muscle atrophy, and neuromuscular respiratory failure.

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Katirji, Bashar. Case 24. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0028.

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Botulism is an extremely rare neuromuscular disorder, caused by botulinum toxin which is produced by the anaerobic bacteria Clostridium botulinum. It has several forms: classic foodborne, infantile, wound, intestinal, and iatrogenic forms. The presentation is often acute and severe but may be occasionally subacute and moderate. The diagnosis may be difficult and requires a high index of suspicion. This case presents an adult with classic foodborne botulism and highlights the clinical and electrodiagnostic findings that distinguish this disorder from other neuromuscular junction disorders including myasthenia gravis and Lambert-Eaton myasthenic syndrome. Specifically, the findings on repetitive nerve stimulation are discussed and distinguished from the results seen in other neuromuscular junction disorders.

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Book chapters on the topic "Lambert-Eaton"

1

Harper, C. Michel, and Vanda A. Lennon. "Lambert-Eaton Syndrome." In Myasthenia Gravis and Related Disorders, 269–91. Totowa, NJ: Humana Press, 2003. http://dx.doi.org/10.1007/978-1-59259-341-5_12.

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Jordan, Berit, and Stephan Zierz. "Lambert-Eaton-Myastheniesyndrom." In Klinische Neurologie, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-44768-0_42-1.

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Jordan, Berit, and Stephan Zierz. "Lambert-Eaton-Myastheniesyndrom." In Klinische Neurologie, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-662-44768-0_42-2.

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Harper, C. Michel, and Vanda A. Lennon. "Lambert-Eaton Syndrome." In Myasthenia Gravis and Related Disorders, 221–37. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73585-6_14.

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Harper, Charles M., and Vanda A. Lennon. "Lambert-Eaton Syndrome." In Myasthenia Gravis and Related Disorders, 209–25. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-156-7_13.

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6

Aarli, Johan A. "Lambert-Eaton Syndrome." In International Neurology, 442–44. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444317008.ch115.

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Lang, Bethan. "Lambert-Eaton Myasthenic Syndrome." In Diagnostic Criteria in Autoimmune Diseases, 413–16. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-285-8_75.

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Pourmand, Rahman. "Lambert-Eaton Myasthenic Syndrome." In Frontiers of Neurology and Neuroscience, 120–25. Basel: KARGER, 2009. http://dx.doi.org/10.1159/000212373.

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9

Jordan, Berit, and Stephan Zierz. "Lambert-Eaton-Myastheniesyndrom (LEMS)." In Klinische Neurologie, 431–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-662-60676-6_42.

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Vagts, Dierk A., Heike Kaltofen, Uta Emmig, and Peter Biro. "Lambert-Eaton-Rooke-Syndrom." In Anästhesie bei seltenen Erkrankungen, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-44368-2_121-1.

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Conference papers on the topic "Lambert-Eaton"

1

Ashhurst, Jasmine, Rami Haddad, and Rob Zielinski. "062 Pembrolizumab induced lambert-eaton myasthenic syndrome." In ANZAN Annual Scientific Meeting 2021 Abstracts. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/bmjno-2021-anzan.62.

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2

Stockwell, Joseph B., Leon S. Edwards, Daniel Wardman, and Neil Griffith. "102 Lambert eaton myaesthenic syndrome in the absence of malignancy." In ANZAN Annual Scientific Meeting 2021 Abstracts. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/bmjno-2021-anzan.102.

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3

Rizzo, A. N., A. Gupta, P. Hume, and T. M. Bull. "Lambert Eaton Myasthenic Syndrome Presenting as Hypoventilation-Induced Right Heart Dysfunction." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3768.

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