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Journal articles on the topic 'Lactate'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 July 2024

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1

Opdahl, Lee J., Ricky W. Lewis, Lee A. Kalcsits, Tarah S. Sullivan, and Karen A. Sanguinet. "Plant Uptake of Lactate-Bound Metals: A Sustainable Alternative to Metal Chlorides." Biomolecules 11, no. 8 (July 23, 2021): 1085. http://dx.doi.org/10.3390/biom11081085.

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Global agricultural intensification has prompted investigations into biostimulants to enhance plant nutrition and soil ecosystem processes. Metal lactates are an understudied class of organic micronutrient supplement that provide both a labile carbon source and mineral nutrition for plant and microbial growth. To gain a fundamental understanding of plant responses to metal lactates, we employed a series of sterile culture-vessel experiments to compare the uptake and toxicity of five metals (Zn, Mn, Cu, Ni, and Co) supplied in lactate and chloride salt form. Additionally, primary root growth in plate-grown Arabidopsis thaliana seedlings was used to determine optimal concentrations of each metal lactate. Our results suggest that uptake and utilization of metals in wheat (Triticum aestivum L.) when supplied in lactate form is comparable to that of metal chlorides. Metal lactates also have promotional growth effects on A. thaliana seedlings with optimal concentrations identified for Zn (0.5–1.0 µM), Mn (0.5–1.0 µM), Cu (0.5 µM), Ni (1.0 µM), and Co (0.5 µM) lactate. These findings present foundational evidence to support the use of metal lactates as potential crop biostimulants due to their ability to both supply nutrients and stimulate plant growth.

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Nikseresht, Asghar, Iman Yabande, Karamatollah Rahmanian, and Abdolreza Sotoodeh Jahromi. "Blood lactate level in Elite boy swimmers after lactate tolerance exercise test." Biomedical Research and Therapy 4, no. 05 (May 22, 2017): 1318. http://dx.doi.org/10.15419/bmrat.v4i05.170.

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Introduction: To avoid injuries during high-intensity sports training, it is important to recognize conditions of bodily consumption and production of adequate energy; exercise increases the concentration of the blood lactate. This paper is an attempt to compare pre and post lactate tolerance exercise test - blood lactate concentrations - of elite boy swimmers. Methods: Blood lactates are measured by an enzymatic method on 12 subjects 30 minutes before and adjust and 24 hours after the test. Results: The mean lactate concentration of 30.35±12.16 mg/dl is observed in swimmers 30 minutes before the test. Swimmers adjust after the test show mean blood lactate concentration of 108.52±18.17 mg/dl that is significantly higher than 30 minutes before the test (p<0.001). Then blood lactate level decreases below baseline level at 24 hours after the test. Conclusion: Blood lactate increases with the test and decreases below baseline within 24 hours after the test.

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3

Zitek, Tony, Zachary D. Skaggs, Aryan Rahbar, Jayme Patel, and Memona Khan. "Does Intravenous Lactated Ringer’s Solution Raise Serum Lactate?" Journal of Emergency Medicine 55, no. 3 (September 2018): 313–18. http://dx.doi.org/10.1016/j.jemermed.2018.05.031.

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4

Shephard, R. J. "Evaluation of three portable blood lactate analysers: Lactate Pro, Lactate Scout and Lactate Plus." Yearbook of Sports Medicine 2011 (January 2011): 153–54. http://dx.doi.org/10.1016/s0162-0908(10)79751-8.

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5

Tanner, Rebecca K., Kate L. Fuller, and Megan L. R. Ross. "Evaluation of three portable blood lactate analysers: Lactate Pro, Lactate Scout and Lactate Plus." European Journal of Applied Physiology 109, no. 3 (February 10, 2010): 551–59. http://dx.doi.org/10.1007/s00421-010-1379-9.

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6

Balakrishnan, Vamsi, John Wilson, Brent Taggart, James Cipolla, and Rebecca Jeanmonod. "Impact of Phlebotomy Tourniquet Use on Blood Lactate Levels in Acutely Ill Patients." CJEM 18, no. 5 (February 11, 2016): 358–62. http://dx.doi.org/10.1017/cem.2016.6.

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AbstractObjectiveLactate levels are increasingly used to guide resuscitation efforts. Some surgical literature suggests that tourniquet use during phlebotomy falsely elevates results, although studies in healthy volunteers have not demonstrated this. The purpose of this study was to determine in clinical practice whether tourniquet use during the drawing of a lactate results in significantly altered levels compared to the result of a level drawn without a tourniquet.MethodsA prospective cohort study was carried out on emergency department patients whose clinical presentation led a physician to order a lactate level. Written informed consent was obtained from patients or their proxies. Study lactates were obtained using a tourniquet during the draw sequence of other laboratory studies. Lactate levels for clinical use were drawn per hospital protocol with no tourniquet. The time of lactate measurements and patient demographic information were recorded. Lactate levels for each patient were compared with the Wilcoxon Rank-Sum Test.Results40 patients were consented and enrolled. The median clinical lactate level was 1.9 (interquartile range 1.5-2.6), and the median study lactate level was 1.9 (interquartile range 1.4-2.7). There was no difference between paired lactate values (p=0.95).ConclusionsTourniquet use appears to have no impact on measured lactate levels. Our findings suggest that current practices at many institutions regarding lactate collection are likely too stringent and should be changed.

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7

Wu, Yuhan, Hui Hu, Weiwei Liu, Yun Zhao, Fang Xie, Zhaowei Sun, Ling Zhang, Huafeng Dong, Xue Wang, and Lingjia Qian. "Hippocampal Lactate-Infusion Enhances Spatial Memory Correlated with Monocarboxylate Transporter 2 and Lactylation." Brain Sciences 14, no. 4 (March 28, 2024): 327. http://dx.doi.org/10.3390/brainsci14040327.

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Lactate has emerged as a key player in regulating neural functions and cognitive processes. Beyond its function as an energy substrate and signal molecule, recent research has revealed lactate to serve as an epigenetic regulator in the brain. However, the molecular mechanisms by which lactate regulates spatial memory and its role in the prevention of cognitive disorders remain unclear. Herein, we injected L-lactate (10 μmol/kg/d for 6 d) into the mouse’s hippocampus, followed by the Morris water maze (MWM) test and molecular analyses. Improved spatial memory performances were observed in mice injected with lactate. Besides, lactate upregulated the expression of synaptic proteins post-synaptic density 95 (PSD95), synaptophysin (SYP), and growth associated protein 43 (GAP43) in hippocampal tissues and HT22 cells, suggesting a potential role in synaptic transmission and memory formation. The facilitative role of monocarboxylate transporter 2 (MCT2), a neuron-specific lactate transporter, in this process was confirmed, as MCT2 antagonists attenuated the lactate-induced upregulation of synaptic proteins. Moreover, lactate induced protein lactylation, a post-translational modification, which could be suppressed by MCT2 inhibition. RNA sequencing of lactated-injected hippocampal tissues revealed a comprehensive gene expression profile influenced by lactate, with significant changes in genes associated with transcriptional progress. These data demonstrate that hippocampal lactate injection enhances spatial memory in mice, potentially through the upregulation of synaptic proteins and induction of protein lactylation, with MCT2 playing a crucial role in these processes. Our findings shed light on the multi-faceted role of lactate in neural function and memory regulation, opening new avenues for therapeutic interventions targeting cognitive disorders.

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8

Brooks, George A. "Lactate." Sports Medicine 37, no. 4 (2007): 341–43. http://dx.doi.org/10.2165/00007256-200737040-00017.

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9

Gollnick, Philip D. "LACTATE." Medicine & Science in Sports & Exercise 17, no. 2 (April 1985): 258. http://dx.doi.org/10.1249/00005768-198504000-00342.

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10

Tomis, Claire, and Anne Vassault. "Lactate." EMC - Biologie Médicale 1, no. 1 (January 2006): 1–4. http://dx.doi.org/10.1016/s2211-9698(06)76089-7.

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11

Bakker, Jan, Radu Postelnicu, and Vikramjit Mukherjee. "Lactate." Critical Care Clinics 36, no. 1 (January 2020): 115–24. http://dx.doi.org/10.1016/j.ccc.2019.08.009.

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12

Walker, Craig A., David M. Griffith, Alasdair J. Gray, Deepankar Datta, and Alasdair W. Hay. "“Lactate Shift,” Rather Than “Lactate Clearance,” for Serial Blood Lactate Monitoring?" Critical Care Medicine 43, no. 12 (December 2015): e596. http://dx.doi.org/10.1097/ccm.0000000000001315.

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13

Skaggs, Z. D., T. Zitek, A. Rahbar, J. Patel, and M. Khan. "59 Does Intravenous Lactated Ringer's Solution Raise Serum Lactate?" Annals of Emergency Medicine 70, no. 4 (October 2017): S24. http://dx.doi.org/10.1016/j.annemergmed.2017.07.084.

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14

Chavarria, Víctor, Emma Ortiz-Islas, Alelí Salazar, Verónica Pérez-de la Cruz, Alejandra Espinosa-Bonilla, Rubén Figueroa, Alma Ortíz-Plata, Julio Sotelo, Francisco Javier Sánchez-García, and Benjamín Pineda. "Lactate-Loaded Nanoparticles Induce Glioma Cytotoxicity and Increase the Survival of Rats Bearing Malignant Glioma Brain Tumor." Pharmaceutics 14, no. 2 (January 29, 2022): 327. http://dx.doi.org/10.3390/pharmaceutics14020327.

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A glioblastoma is an aggressive form of a malignant glial-derived tumor with a poor prognosis despite multimodal therapy approaches. Lactate has a preponderant role in the tumor microenvironment, playing an immunoregulatory role as well as being a carbon source for tumor growth. Lactate homeostasis depends on the proper functioning of intracellular lactate regulation systems, such as transporters and enzymes involved in its synthesis and degradation, with evidence that an intracellular lactate overload generates metabolic stress on tumor cells and tumor cell death. We propose that the delivery of a lactate overload carried in nanoparticles, allowing the intracellular release of lactate, would compromise the survival of tumor cells. We synthesized and characterized silica and titania nanoparticles loaded with lactate to evaluate the cellular uptake, metabolic activity, pH modification, and cytotoxicity on C6 cells under normoxia and chemical hypoxia, and, finally, determined the survival of an orthotopic malignant glioma model after in situ administration. A dose-dependent reduction in metabolic activity of treated cells under normoxia was found, but not under hypoxia, independent of glucose concentration. Lactated-loaded silica nanoparticles were highly cytotoxic (58.1% of dead cells) and generated significant supernatant acidification. In vivo, lactate-loaded silica nanoparticles significantly increased the median survival time of malignant glioma-bearing rats (p = 0.005) when administered in situ. These findings indicate that lactate-loaded silica nanoparticles are cytotoxic on glioma cells in vitro and in vivo.

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15

Villar, Julian, Jack H. Short, and Geoffrey Lighthall. "Lactate Predicts Both Short- and Long-Term Mortality in Patients With and Without Sepsis." Infectious Diseases: Research and Treatment 12 (January 2019): 117863371986277. http://dx.doi.org/10.1177/1178633719862776.

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Objective: To measure the relationship between lactate and mortality in hospital inpatients. Main outcomes of interest were 3-day, 30-day, and 1-year all-cause mortality. Design: Retrospective cohort study, October 2011 to September 2013. Setting: University-affiliated US Veterans Affairs Hospital. Patients: All inpatients with lactate level measured during the study period. Measurements: Analysis of peak lactate level (mmol/L) during the most recent admission for patients who died, and peak lactate level during an admission for surviving patients. Covariates including sepsis, ICU admission, code blue and rapid response calls, medical vs surgical ward, liver disease, kidney disease, and hospice status were recorded. Results: In total, 3325 inpatients were included; 564 patients had sepsis. Median lactate 1.7 mmol/L (interquartile range [IQR] 1.2-2.6). The 3-day, 30-day, and 1-year mortality were 2.5%, 10%, and 24%, respectively. A lactate level cutoff of ⩾4 mmol/L had best test characteristics (sensitivity 52.4%, specificity 91.4%) to predict increased 3-day mortality. Unadjusted risk ratio of death in 3 days for lactate ⩾4 was 10.3 (95% confidence interval [CI] 6.8-15.7). Patients with sepsis had a consistently higher risk of death compared with patients without sepsis for any given level of lactate. Adjusted odds ratio (OR) of 3-day mortality for lactate ⩾4 was 7.6 (95% CI 4.6-12.5); 30-day mortality was 2.6 (95% CI 1.9-3.6); and 1-year mortality was 1.8 (95% CI 1.4-2.6). Lactates in the normal range (<1.7) were also independently associated with 30-day and 1-year mortality. Conclusions: Lactate predicts risk of death in all patients, although patients with sepsis have a higher mortality for any given lactate level. We report the novel finding that serum lactate, including normal values, is associated with long-term mortality.

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16

SHELEF, LEORA A. "Antimicrobial Effects of Lactates: A Review." Journal of Food Protection 57, no. 5 (May 1, 1994): 445–50. http://dx.doi.org/10.4315/0362-028x-57.5.445.

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Sodium lactate is used as humectant and flavor enhancer in meat and poultry products, and there is growing evidence of antimicrobial properties of the salt. Potassium and calcium lactate are equally effective in controlling growth of aerobes and anaerobes in meats, and antibotulinal and antilisterial activities of the lactate anion have been established. The specific action of lactate on the microbial cell is not well understood. No intracellular pH lowering effect could be demonstrated, and the reported small decreases in water activity appear insufficient to explain the effect. Other explanations have been proposed but not yet confirmed. Although lactates appear to be bacteriostatic, their ability to control spoilage and pathogenic bacteria in fresh and processed meat favors their use, particularly in refrigerated meat products in combination with other microbial inhibitors.

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17

Grayck, Eva Nozik, Jon N. Meliones, Frank H. Kern, Doug R. Hansell, Ross M. Ungerleider, and William J. Greeley. "Elevated Serum Lactate Correlates With Intracranial Hemorrhage in Neonates Treated With Extracorporeal Life Support." Pediatrics 96, no. 5 (November 1, 1995): 914–17. http://dx.doi.org/10.1542/peds.96.5.914.

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Objectives. To correlate the initial and maximal lactate levels with the occurrence of intracranial hemorrhage (ICH) and survival in patients treated with extracorporeal life support (ECLS). Design. Retrospective chart review. Setting. Pediatric intensive care unit. Patients. Eighty-two neonatal patients placed on ECLS for respiratory failure due to sepsis, meconium aspiration, or persistent pulmonary hypertension of the newborn. Measurements. The initial lactate level measured within 6 hours of initiating ECLS and the maximal lactate level measured throughout the ECLS course were collected. Lactate levels were described as mean lactate ± SE (mM). Head ultrasound reports and survival were reviewed. Platelet counts and activated clotting times (ACTs) were examined. Results. The mean initial and maximal lactate levels were higher in ECLS patients who developed ICH (initial: 10 ± 1.7 mM vs 6.4 ± 0.8 mM, p = .05 and maximal: 12.4 ± 2.5 mM vs 7.9 ± 0.8 mM, p = .04). Initial and maximal lactate levels were also elevated in nonsurvivors (initial: 11.7 ± 3 mM vs 6.4 ± 0.7 mM, p = .01 and maximal: 14.8 ± 3.3 mM vs 7.8 ± 0.8 mM, P < .01). Platelet counts and ACT did not differ in patients with and without ICH. Conclusions. Lactate is a useful marker for the development of ICH in ECLS patients. In addition, elevated lactates during ECLS identify a subgroup of patients with poor outcome. Prospective studies are needed to determine whether the incorporation of this information into pre-ECLS and ECLS management will decrease the occurrence of ICH and improve survival.

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18

Kubo, Hitoshi, Yoshindo Kawaguchi, Hiroshi Fujimaki, Kenji Kasai, Ryou Shouji, Souichi Sakai, Yousuke Ogura, and Tadashi Miyahara. "CAPD and lactate metabolism." Journal of Japanese Society for Dialysis Therapy 18, no. 6 (1985): 625–30. http://dx.doi.org/10.4009/jsdt1985.18.625.

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19

Lamberti, Fabio M., Luis A. Román-Ramírez, Paul Mckeown, Matthew D. Jones, and Joseph Wood. "Kinetics of Alkyl Lactate Formation from the Alcoholysis of Poly(Lactic Acid)." Processes 8, no. 6 (June 24, 2020): 738. http://dx.doi.org/10.3390/pr8060738.

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Alkyl lactates are green solvents that are successfully employed in several industries such as pharmaceutical, food and agricultural. They are considered prospective renewable substitutes for petroleum-derived solvents and the opportunity exists to obtain these valuable chemicals from the chemical recycling of waste poly(lactic acid). Alkyl lactates (ethyl lactate, propyl lactate and butyl lactate) were obtained from the catalysed alcoholysis reaction of poly(lactic acid) with the corresponding linear alcohol. Reactions were catalysed by a Zn complex synthesised from an ethylenediamine Schiff base. The reactions were studied in the 50–130 °C range depending on the alcohol, at autogenous pressure. Arrhenius temperature-dependent parameters (activation energies and pre-exponential factors) were estimated for the formation of the lactates. The activation energies (Ea1, Ea2 and Ea−2) for alcoholysis in ethanol were 62.58, 55.61 and 54.11 kJ/mol, respectively. Alcoholysis proceeded fastest in ethanol in comparison to propanol and butanol and reasonable rates can be achieved in temperatures as low as 50 °C. This is a promising reaction that could be used to recycle end-of-life poly(lactic acid) and could help create a circular production economy.

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20

Bazzano, Carmelo, Lee N. Cunningham, Giovanni Cama, and Tony Falconio. "The Relationship of Lactate to 1-Mile Walk Performance in Women Aged 60 to 70 Years." Journal of Aging and Physical Activity 6, no. 3 (July 1998): 285–89. http://dx.doi.org/10.1123/japa.6.3.285.

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The purpose of this study was to examine the relationship between selected physiological variables and lactate accumulation at the end of a l-mile walk test (MWT) in older women (mean ± SD: 64.6 ± 3.1 years). Seventeen women with a peak (ml · kg-1 · min-1) of 21.1 ± 4.2 volunteered to participate. Physiological data were obtained via a COSMED K2 miniaturized O2 analyzer with telemetric capabilities during a maximal treadmill (TM) test and MWT. Blood samples were obtained from the ear lobe for lactale analysis immediately before and after the treadmill test and MWT. Subjects performed the MWT in 15.4 ± 1.4 min at an intensity of 76% of peak and 86% of HRmax. The blood lactate accumulated at the end of the MWT was 2.61 ± 1.47 mmol/L. Peak lactate following the maximal treadmill test was 3.8 ± 1.42 mmol/L. HR during the test was significantly related with blood lactate (r= .65, p< .01). The lactate values observed during the lest suggest that the I-mile walk test is a suitable field testing procedure for older women.

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21

Finsterer, Josef. "Lactate Stress Testing by Bedside Lactate Determination." Metabolic Brain Disease 18, no. 4 (December 2003): 265–72. http://dx.doi.org/10.1023/b:mebr.0000020188.57786.0e.

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22

Fawcett, J. Paul, David J. Woods, Bhadra Munasiri, and Gordon Becket. "Compatibility of cyclizine lactate and haloperidol lactate." American Journal of Health-System Pharmacy 51, no. 18 (September 15, 1994): 2292. http://dx.doi.org/10.1093/ajhp/51.18.2292.

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23

VELUGOTI, PADMANABHA REDDY, LALIT K. BOHRA, VIJAY K. JUNEJA, and HARSHAVARDHAN THIPPAREDDI. "Inhibition of Germination and Outgrowth of Clostridium perfringens Spores by Lactic Acid Salts during Cooling of Injected Turkey†." Journal of Food Protection 70, no. 4 (April 1, 2007): 923–29. http://dx.doi.org/10.4315/0362-028x-70.4.923.

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Inhibition of Clostridium perfringens spore germination and outgrowth by lactic acid salts (calcium, potassium, and sodium) during exponential cooling of injected turkey product was evaluated. Injected turkey samples containing calcium lactate, potassium lactate, or sodium lactate (1.0, 2.0, 3.0, or 4.8% [w/w]), along with a control (product without lactate), were inoculated with a three-strain cocktail of C. perfringens spores to achieve a final spore population of 2.5 to 3.0 log CFU/g. The inoculated product was heat treated and exponentially cooled from 54.5 to 7.2°C within 21, 18, 15, 12, 9, or 6.5 h. Cooling of injected turkey (containing no antimicrobials) resulted in C. perfringens germination and an outgrowth of 0.5, 2.4, 3.4, 5.1, 5.8, and 5.8 log CFU/g when exponentially cooled from 54.4 to 7.2°C in 6.5, 12, 15, 18, and 21 h, respectively. The incorporation of antimicrobials (lactates), regardless of the type (Ca, Na, or K salts), inhibited the germination and outgrowth of C. perfringens spores at all the concentrations evaluated (1.0, 2.0, 3.0, and 4.8%) compared to the injected turkey without acetate (control). Increasing the concentrations of the antimicrobials resulted in a greater inhibition of the spore germination and outgrowth in the products. In general, calcium lactate was more effective in inhibiting the germination and outgrowth of C. perfringens spores at ≥1.0% concentration than were sodium and potassium lactates. Incorporation of these antimicrobials in cooked, ready-to-eat turkey products can provide additional protection in controlling the germination and outgrowth of C. perfringens spores during cooling (stabilization).

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Horváth, Péter, András Gergely, and Béla Noszál. "Characterization of lactate–guanidinium and lactate–lactate interactions in aqueous solution by spectropolarimetry." J. Chem. Soc., Perkin Trans. 2, no. 7 (1996): 1419–22. http://dx.doi.org/10.1039/p29960001419.

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25

Brooks, G. A., H. Dubouchaud, M. Brown, J. P. Sicurello, and C. E. Butz. "Role of mitochondrial lactate dehydrogenase and lactate oxidation in the intracellular lactate shuttle." Proceedings of the National Academy of Sciences 96, no. 3 (February 2, 1999): 1129–34. http://dx.doi.org/10.1073/pnas.96.3.1129.

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26

Del Río Valdivia, Jose E., Ciria Margarita Salazar, Julio Cuevas Romo, Adriana Isabel Andrade Sánchez, Pedro Julian Flores Moreno, Lenin Tlamatini Barajas Pineda, and Isela Guadalupe Ramos Carranza. "Diferencias en el OBLA en jugadoras de fútbol en relación a su posición en el campo de juego (Differences in OBLA in football players based on their field position)." Retos, no. 32 (December 28, 2016): 58–61. http://dx.doi.org/10.47197/retos.v0i32.48937.

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La posición en el campo de juego en el fútbol, no obedece solo a un interés personal, sino que podría depender de variables fisiológicas individuales. El presente estudio determina el OBLA en las diferentes posiciones. La población del estudio fue integrada por 16 jugadoras universitarias de la modalidad soccer (4 por cada posición) evaluadas en una banda sin fin, con una prueba de velocidades crecientes. En cada velocidad utilizada se midió la concentración de lactato sanguíneo con un analizador portátil Accuntrend Lactate Plus. Para el análisis de los datos se propuso una fórmula matemática de interpolación entre las variables velocidad y concentración de lactato. Se encontró, una variación significativa del OBLA, en la muestra estudiada, dependiendo de la posición de juego (porteras, defensas, medio-campistas y delanteras). Utilizando la prueba de Kruskal-Wallis, con un nivel de significancia (α) del 5 %, en búsqueda de la relación entre el OBLA y la posición de las jugadoras, se obtuvo un p-valor de 0.004, por lo que no es posible aceptar la igualdad entre las poblaciones, por lo tanto, se puede afirmar que existe una diferencia estadística significativa cuando se compara el comienzo de acumulación de lactato en sangre (OBLA) con la posición en la que juegan las futbolistas. Los resultados demuestran que existen diferencias estadísticamente significativas entre los grupos de jugadoras de acuerdo a la posición que desempeñan en el campo de juego y a la concentración de lactato en sangre.Abstract. Choosing a field position in football may not depend only on personal interest, but also on individual physiological variables. The aim of the study is to determine the Onset of Blood Lactate Accumulation (OBLA) in the different field positions of football. The study sample was composed by 16 female players (four per position) evaluated using an increased speed test carried out on treadmill. At each speed level, blood lactate concentrations were measured with the portable analyzer Accuntrend Lactate Plus. A mathematical formula interpolating speed and lactate concentration was proposed for the analysis of the data obtained. A significant variation of OBLA was found in the participants, associated with playing position (goalkeepers, defenders, midfielders, and strikers). Setting the significance level (α) at 5%, a p-value of 0.004 was obtained using the Kruskal-Wallis test in order to study the relation between OBLA and players’ field position. This demonstrates that there is no equivalence among positions, at the same time as a statistically significant difference is shown when comparing OBLA with players’ field position. Results show that there are statistically significant differences between the groups of players based on their field position and on blood lactate concentration.

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27

Anurag Prasad Tapas, Albee. "Lactate Albumin Ratio in Comparison with Lactate to Predict Outcomes in Sepsis and Septic Shock." International Journal of Science and Research (IJSR) 12, no. 3 (March 5, 2023): 851–53. http://dx.doi.org/10.21275/mr23315193436.

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28

&NA;. "Ringer's lactate." Reactions Weekly &NA;, no. 1360 (July 2011): 33. http://dx.doi.org/10.2165/00128415-201113600-00115.

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&NA;. "Ringer's lactate." Reactions Weekly &NA;, no. 1365 (August 2011): 40. http://dx.doi.org/10.2165/00128415-201113650-00153.

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30

&NA;. "Calcium lactate." Reactions Weekly &NA;, no. 554 (June 1995): 4. http://dx.doi.org/10.2165/00128415-199505540-00008.

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31

Jacobs, Ira. "Blood Lactate." Sports Medicine 3, no. 1 (1986): 10–25. http://dx.doi.org/10.2165/00007256-198603010-00003.

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&NA;. "Sodium lactate." Reactions Weekly &NA;, no. 509 (July 1994): 10. http://dx.doi.org/10.2165/00128415-199405090-00048.

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33

Baheerathan, Aravindhan, Robert DS Pitceathly, Carmel Curtis, and Nicholas WS Davies. "CSF lactate." Practical Neurology 20, no. 4 (May 13, 2020): 320–23. http://dx.doi.org/10.1136/practneurol-2019-002191.

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Lactate is produced from anaerobic glycolysis, which occurs in most tissues in the human body. Blood lactate is tested in most physiologically unwell patients in the Emergency Department and helps to guide treatment and prognosis. Cerebrospinal fluid (CSF) lactate, however, is not often measured. Various central nervous system (CNS) conditions lead to a rise in CSF lactate, including acute neurological infection, stroke, seizures and mitochondrial pathologies. This article discusses the utility and limitations of CSF lactate, highlighting specific clinical situations where it can help in the diagnosis of CNS infections and unexplained encephalopathy.

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34

Yates, Darran. "Lactate mobilization." Nature Reviews Neuroscience 13, no. 11 (October 17, 2012): 739. http://dx.doi.org/10.1038/nrn3374.

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35

Kruse, James A., and Richard W. Carlson. "Lactate Metabolism." Critical Care Clinics 3, no. 4 (October 1987): 725–46. http://dx.doi.org/10.1016/s0749-0704(18)30516-5.

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Morales, Carlos, Johana Ascuntar, Jessica María Londoño, César Daniel Niño, Jimmy Paul León, Elisa Bernal, Cesar Vargas, and Fabián Jaimes. "Lactate clearance." Colombian Journal of Anesthesiology 47, no. 1 (January 2019): 41–48. http://dx.doi.org/10.1097/cj9.0000000000000084.

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37

Anand, Usha. "Lactate Dehydrogenase." Clinical Chemistry 59, no. 3 (March 1, 2013): 585. http://dx.doi.org/10.1373/clinchem.2011.178541.

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38

Rigor, &NA;, A. Schurr, R. S. Payne, and J. J. Miller. "A709 LACTATE." Anesthesiology 87, Supplement (September 1997): 709A. http://dx.doi.org/10.1097/00000542-199709001-00709.

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39

Shirey, Terry L. "POC Lactate." Point of Care: The Journal of Near-Patient Testing & Technology 6, no. 3 (September 2007): 192–200. http://dx.doi.org/10.1097/poc.0b013e3181271545.

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40

Quintela-Fandino, M., and R. Colomer. "Dovitinib lactate." Drugs of the Future 38, no. 2 (2013): 81. http://dx.doi.org/10.1358/dof.2013.038.02.1918242.

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Bakker, Jan, and Glenn Hernandez. "Lactate Measurements." Chest 154, no. 6 (December 2018): 1461. http://dx.doi.org/10.1016/j.chest.2018.07.041.

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42

Quintela-Fandino, M., and R. Colomer. "Dovitinib lactate." Drugs of the Future 38, no. 2 (2013): 81. http://dx.doi.org/10.1358/dof.2013.38.2.1918242.

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43

Bonen, Arend, Steven K. Baker, and Hideo Hatta. "Lactate Transport and Lactate Transporters in Skeletal Muscle." Canadian Journal of Applied Physiology 22, no. 6 (November 1, 1997): 531–52. http://dx.doi.org/10.1139/h97-034.

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Abstract:

The study of lactate transport in skeletal muscle had until recently been hampered by the lack of suitable sarcolemmal vesicle preparations. Researchers are now at the threshold of developing some very new understandings about the movement of lactate into and out of skeletal muscle with (a) evidence for a lactate transport system in skeletal muscle, (b) the very recent cloning of several monocarboxylate transporter genes, (c) the expression of at least one monocarboxylate transporter protein that facilitates the transport of lactate in heart and skeletal muscle, and (d) the realization that lactate transport can be altered with changes in chronic muscle activity. The MCT1 expression patterns in metabolically heterogeneous skeletal suggest that a primary role of this lactate transporter is to take up lactate into the oxidative muscle fibers where it may be used as a fuel in mitochondrial oxidation. Increments in both MCT1 and lactate transport with training support this role. Key words: vesicles, MCT1, diffusion

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Pyne, David B., Tanya Boston, David T. Martin, and Andrew Logan. "Evaluation of the Lactate Pro blood lactate analyser." European Journal of Applied Physiology 82, no. 1-2 (May 15, 2000): 112–16. http://dx.doi.org/10.1007/s004210050659.

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Tilton, W. M., C. Seaman, and S. Piomelli. "Measurement of Radiolabeled Lactate Production Using Lactate Monooxygenase." Analytical Biochemistry 208, no. 2 (February 1993): 400–402. http://dx.doi.org/10.1006/abio.1993.1069.

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46

Apelblat, Alexander, Emanuel Manzurola, Jan van Krieken, and Gert L. Nanninga. "Solubilities and vapour pressures of water over saturated solutions of magnesium-l-lactate, calcium-l-lactate, zinc-l-lactate, ferrous-l-lactate and aluminum-l-lactate." Fluid Phase Equilibria 236, no. 1-2 (September 2005): 162–68. http://dx.doi.org/10.1016/j.fluid.2005.06.018.

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47

Mineshima, Michio, Naomi Watanabe, Mieko Takemoto, Masami Hasuo, Toshiaki Suzuki, and Kazuo Ota. "Lactate kinetics in CAPD." Journal of Japanese Society for Dialysis Therapy 18, no. 2 (1985): 111–14. http://dx.doi.org/10.4009/jsdt1968.18.111.

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48

Pedro Manuel Canales Lara, Luisa María Charco Roca, Jose María Jiménez Vizuete, and Carlos Martínez Villar. "¿Hay factores pronósticos de mortalidad en el paciente séptico en la UCI? El valor de la tasa de eliminación de lactato sérico." Revista Electrónica AnestesiaR 12, no. 4 (May 4, 2020): 2. http://dx.doi.org/10.30445/rear.v12i4.809.

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La sepsis es la primera causa de muerte por infección en el mundo y el pronóstico es tiempo dependiente de un tratamiento eficaz. Existen descritos numerosos factores pronósticos en la bibliografía sin embargo hay pocos estudios que los comparen entre ellos para valorar si hay alguno con mayor peso frente a otros. El presente artículo, con una muestra de 82 pacientes, analiza diferentes factores pronósticos en las primeras 24 horas de ingreso como indicadores de supervivencia a los 28 días. Estos son: cifras de lactato, su tasa de eliminación, la saturación venosa central y la diferencia entre la saturación de oxígeno arterio-venosa central. Los resultados muestras que las cifras de lactato a las 24 horas son los que presentan una sensibilidad-especificadad mayores (punto de corte de 2,15 mmol/dl), seguido por la cifra de lactato a las 12 y a las 6 horas. ABSTRACT Are there prognostic factors for mortality in the septic patient in the ICU? The value of the serum lactate elimination rate Sepsis is the first cause of death due to infection in the world and the prognosis is time dependent on effective treatment. There are numerous prognostic factors described in the literature, however there are just a few studies comparing them among them to assess if there is one with greater weight compared to others. The present article, with a sample of 82 patients, analyzes different prognostic factors in the first 24 hours of admission as survival indicators at 28 days. These are: lactate numbers, their elimination rate, central venous saturation and the difference between central arteriovenous oxygen saturation. The results show that the lactate levels at 24 hours are the ones with higher sensitivity-specificity (cut-off point of 2.15 mmol / dl), followed by the lactate levels at 12 and at 6 hours.

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Więcek, Sabina, Jerzy Chudek, Halina Woś, Maria Bożentowicz-Wikarek, Bożena Kordys-Darmolinska, and Urszula Grzybowska-Chlebowczyk. "Serum Level of D-Lactate in Patients with Cystic Fibrosis: Preliminary Data." Disease Markers 2018 (July 12, 2018): 1–5. http://dx.doi.org/10.1155/2018/5940893.

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D-Lactate is produced by the intestinal biota and later absorbed into circulation. Some patients with cystic fibrosis (CF) develop exocrine pancreatic insufficiency that may disturb the gut microbiome and enhance the production of D-lactate. However, this concept has not been studied yet. The aim of the study was to assess D-lactate concentration in relation to the occurrence of clinical features, activity of CF, and diet composition in paediatric patients. Patients and Method. Serum concentrations of D-lactate were measured in 38 CF patients (19 girls and 19 boys) from 6 months to 18 years of age. The analysis included age, sex, clinical symptoms, diet (the variety and calorie needs), the laboratory tests for pancreatic efficiency (serum levels of albumin and glucose, faecal elastase activity, and faecal fat index) and faecal calprotectin (the marker of intestinal inflammation), and parameters of liver damage and of cholestasis (the activity of aminotransferases, γ-glutamyltransferase, level of bilirubin, and international normalized ratio). Results. The median level of D-lactate was 0.86 μg/ml (1Q–3Q: 0.48–2.03) and correlated with the CF severity in the Schwachman-Kulczycki score, parameters of pancreatic insufficiency, and the presence of intestinal inflammation. An increased level of D-lactate was observed in the subgroup with pancreas insufficiency (1.05 versus 0.73; p<0.05), parallel with an elevated level of calprotectin (0.948 versus 0.755; p=0.08). There was no relationship between energy consumption and diet composition and serum D-lactates. Conclusion. Serum D-lactate concentration in CF patients is a promising new marker of exocrine pancreatic insufficiency probably related to intestinal flora dysbiosis/overgrowth.

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von Duvillard, Serge P., Rochus Pokan, Peter Hofmann, Manfred Wonisch, Gerhard Smekal, Ahmad Alkhatib, Ralph Beneke, and Renate Leithauser. "Comparing Blood Lactate Values Of Three Different Handheld Lactate Analyzers To YSI 1500 Lactate Analyzer." Medicine & Science in Sports & Exercise 37, Supplement (May 2005): S25. http://dx.doi.org/10.1249/00005768-200505001-00153.

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