Relevant bibliographies by topics / Labyrinth (ear)

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Labyrinth (ear)'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Labyrinth (ear)"

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Cerio, Donald G., and Lawrence M. Witmer. "Intraspecific variation and symmetry of the inner-ear labyrinth in a population of wild turkeys: implications for paleontological reconstructions." PeerJ 7 (July 23, 2019): e7355. http://dx.doi.org/10.7717/peerj.7355.

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The cochlea and semicircular canals (SCCs) of the inner ear are vital neurosensory devices. There are associations between the anatomy of these sensorineural structures, their function, and the function of related biological systems, for example, hearing ability, gaze stabilization, locomotor agility, and posture. The endosseous labyrinth is frequently used as a proxy to infer the performance of the hearing and vestibular systems, locomotor abilities, and ecology of extinct species. Such fossil inferences are often based on single specimens or even a single ear, representing an entire species. To address whether a single ear is representative of a population, we used geometric morphometrics to quantitatively assess the variation in shape and symmetry in a sample of endosseous labyrinths of wild turkeys Meleagris gallopavo of southern Ohio. We predicted that ears would be symmetrical both within individuals and across the sample; that labyrinth shape and size would covary; that labyrinth shape would vary with the size of the brain, measured as width of the endocranium at the cerebellum; and that labyrinths would be morphologically integrated. To test these predictions, we microCT-scanned the heads of 26 cadaveric turkeys, digitally segmented their endosseous labyrinths in Avizo, and assigned 15 manual landmarks and 20 sliding semilandmarks to each digital model. Following Procrustes alignment, we conducted an analysis of bilateral symmetry, a Procrustes regression analysis for allometry and other covariates including side and replicate, and analyses of global integration and modularity. Based on Procrustes distances, no individual’s left and right ears were clearly different from each other. When comparing the ears of different specimens, statistically clear differences in shape were found in only 66 of more than 1,300 contrasts. Moreover, effects of both directional and fluctuating asymmetry were very small—generally, two orders of magnitude smaller than the variance explained by individual variation. Statistical tests disagreed on whether these asymmetric effects crossed the threshold of significance, possibly due to non-isotropic variation among landmarks. Regardless, labyrinths appeared to primarily vary in shape symmetrically. Neither labyrinth size nor endocranial width was correlated with labyrinth shape, contrary to our expectations. Finally, labyrinths were found to be moderately integrated in a global sense, but four weakly separated modules—the three SCCs and cochlea—were recovered using a maximum-likelihood analysis. The results show that both fluctuating and directional asymmetry play a larger role in shape variation than expected—but nonetheless, endosseous labyrinths are symmetrical within individuals and at the level of the population, and their shape varies symmetrically. Thus, inferences about populations, and very possibly species, may be confidently made when only a single specimen, or even a single ear, is available for study.
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Hu, Fangyuan, Xiaofei Ye, Yinghong Zhai, Jinfang Xu, Xiaojing Guo, Zhijian Guo, Xiang Zhou, Yiming Ruan, YongLong Zhuang, and Jia He. "Ear and labyrinth toxicities induced by immune checkpoint inhibitors: a disproportionality analysis from 2014 to 2019." Immunotherapy 12, no. 7 (May 2020): 531–40. http://dx.doi.org/10.2217/imt-2019-0120.

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Aim: We aimed to systematically characterize ear and labyrinth toxicities after immune checkpoint inhibitors (ICIs) initiation. Materials & methods: Data were extracted from the US FDA Adverse Event Reporting System database. Disproportionality analysis including information component and reporting odds ratio (ROR) was performed to access potential signals. Results: In FDA Adverse Event Reporting System database, 284 records for ICIs-associated ear/labyrinth adverse events (AEs) were involved. In general, there was no significant association between total ICIs use and total ear and labyrinth AEs (ROR025: 0.576). However, in ICIs monotherapy and polytherapy groups, signals were detected in several specific ear and labyrinth AEs. Conclusion: Total ear and labyrinth toxicities were not significantly reported with ICI immunotherapy, while class-specific ear toxicities were detected in some strategies.
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Šikolová, Soňa, Milan Urík, Jana Jančíková, Dagmar Hošnová, and Rami Katra. "Congenital malformations of the inner ear." Otorinolaryngologie a foniatrie 70, no. 3 (September 27, 2021): 167–73. http://dx.doi.org/10.48095/ccorl2021167.

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Summary Congenital malformations of the inner ear consist of many different anomalies of the labyrinth. They often cause hearing loss, mostly of the sensorineural type. Eighty percent of hearing loss is caused by an anomaly of the membranous labyrinth, and 20% by an anomaly of the bone labyrinth. The role in pathogenesis is played by hereditary factors and influence of the environment. The treatment depends on the severity of the hearing loss, abnormalities of the external and middle ear, associated defects, and presence and function of the auditory nerve. We have modern hearing aids or implantable systems. Another options include a sign language and mouth-reading. The article includes a retrospective analysis of patients with congenital inner ear malformations at our tertiary center in 2010–2020. In conclusion, our patients clearly prove that even children with profound hearing loss are successfully implanted and restored hearing can be achieved in most of them. Key words congenital ear malformation – inner ear – hearing loss – anomalies of bony labyrinth – anomalies of membranous labyrinth – rehabilitation
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Schröder, Dirk, Gloria Grupe, Grit Rademacher, Sven Mutze, Arneborg Ernst, Rainer Seidl, and Philipp Mittmann. "Magnetic Resonance Imaging Artifacts and Cochlear Implant Positioning at 1.5 T In Vivo." BioMed Research International 2018 (November 8, 2018): 1–4. http://dx.doi.org/10.1155/2018/9163285.

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Objective. Cerebral magnetic resonance imaging with the magnet of the cochlear implant receiver/stimulator in place causes artifacts and hinders evaluation of intracerebral structures. The aim of this study was to evaluate the internal auditory canal and the labyrinth in a 1.5T MRI with the magnet in place. Study Design. Observational study. Setting. Tertiary referral center. Subjects and Methods. The receiver/stimulator unit was placed and fixed onto the head of three volunteers at three different angles to the nasion–outer ear canal (90°–160°) and at three different distances from the outer ear canal (5–9 cm). T1 and T2 weighted sequences were conducted for each position. Results. Excellent visibility of the internal auditory canal and the labyrinth was seen in the T2 weighted sequences with 9 cm between the magnet and the outer ear canal at every nasion–outer ear canal angle. T1 sequences showed poorer visibility of the internal auditory canal and the labyrinth. Conclusion. Aftercare and visibility of intracerebral structures after cochlear implantation is becoming more important as cochlear implant indications are widened worldwide. With a distance of at least 9 cm from the outer ear canal the artifact induced by the magnet allows evaluation of the labyrinth and the internal auditory canal.
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Nyberg, Sophie, N. Joan Abbott, Xiaorui Shi, Peter S. Steyger, and Alain Dabdoub. "Delivery of therapeutics to the inner ear: The challenge of the blood-labyrinth barrier." Science Translational Medicine 11, no. 482 (March 6, 2019): eaao0935. http://dx.doi.org/10.1126/scitranslmed.aao0935.

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Permanent hearing loss affects more than 5% of the world’s population, yet there are no nondevice therapies that can protect or restore hearing. Delivery of therapeutics to the cochlea and vestibular system of the inner ear is complicated by their inaccessible location. Drug delivery to the inner ear via the vasculature is an attractive noninvasive strategy, yet the blood-labyrinth barrier at the luminal surface of inner ear capillaries restricts entry of most blood-borne compounds into inner ear tissues. Here, we compare the blood-labyrinth barrier to the blood-brain barrier, discuss invasive intratympanic and intracochlear drug delivery methods, and evaluate noninvasive strategies for drug delivery to the inner ear.
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YANG, Juan-mei, Fang-lu CHI, Zhao HAN, Yi-bo HUANG, and Yi-ke LI. "Clinical characteristics of patients with labyrinthine fistulae caused by middle ear cholesteatoma." Chinese Medical Journal 126, no. 11 (June 5, 2013): 2116–19. http://dx.doi.org/10.3760/cma.j.issn.0366-6999.20123026.

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Background Labyrinthine fistula (LF) is a very common clinical complication mainly caused by middle ear cholesteatoma. Whether the presence of different degree LF caused by middle ear cholesteatoma aggravates neurosensory hearing loss (NSHL) and what is the degree of the hearing loss caused by LF were still under controversial. This study aimed to investigate whether the LF degree is correlative with the age distribution, disease duration and hearing loss degree for cholesteatomatous patients. Methods The files of 143 patients with middle ear cholesteatoma were selected and reviewed in a retrospective study. Seventy-eight patients with LF were divided into three types according to the degree of destruction of labyrinth. Sixty-five patients without LF were randomly chosen for control. Then, we compared the clinical characteristics of patients with or without labyrinthine fistulae caused by middle ear cholesteatoma. Results According to the study, cholesteatomatous patients with LF were older and suffered longer disease duration than those without LF. Hearing loss is severe with high frequencies both in patients with and without LF. Moreover, inner ear impairment is correlative with the degree of destruction in labyrinth, and more severe destruction in labyrinth follow the more severe symptoms correlative with inner ear impairment. Conclusion Surgical intervention should be performed as early as possible for these cholesteatomatous patients.
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S, Allen Counter, Peter Damberg, Sahar Nikkhou Aski, Kálmán Nagy, Cecilia Engmér Berglin, and Göran Laurell. "Experimental Fusion of Contrast Enhanced High-Field Magnetic Resonance Imaging and High-Resolution Micro-Computed Tomography in Imaging the Mouse Inner Ear." Open Neuroimaging Journal 9, no. 1 (July 31, 2015): 7–12. http://dx.doi.org/10.2174/1874440001509010007.

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Objective: Imaging cochlear, vestibular, and 8th cranial nerve abnormalities remains a challenge. In this study, the membranous and osseous labyrinths of the wild type mouse inner ear were examined using volumetric data from ultra high-field magnetic resonance imaging (MRI) with gadolinium contrast at 9.4 Tesla and high-resolution micro-computed tomography (µCT) to visualize the scalae and vestibular apparatus, and to establish imaging protocols and parameters for comparative analysis of the normal and mutant mouse inner ear. Methods: For in vivo MRI acquisition, animals were placed in a Milleped coil situated in the isocenter of a horizontal 9.4 T Varian magnet. For µCT examination, cone beam scans were performed ex vivo following MRI using the µCT component of a nanoScan PET/CT in vivo scanner. Results: The fusion of Gd enhanced high field MRI and high-resolution µCT scans revealed the dynamic membranous labyrinth of the perilymphatic fluid filled scala tympani and scala vestibule of the cochlea, and semicircular canals of the vestibular apparatus, within the µCT visualized contours of the contiguous osseous labyrinth. The ex vivo µCT segmentation revealed the surface contours and structural morphology of each cochlea turn and the semicircular canals in 3 planes. Conclusions: The fusion of ultra high-field MRI and high-resolution µCT imaging techniques were complementary, and provided high-resolution dynamic and static visualization of the complex morphological features of the normal mouse inner ear structures, which may offer a valuable approach for the investigation of cochlear and vestibular abnormalities that are associated with birth defects related to genetic inner ear disorders in humans.
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Terao, Kyoichi, Sebahattin Cureoglu, Schachern Patricia, Michael M. Paparella, Norimasa Morita, Nomiya Rie, and Kiyotaka Murata. "R447 – Temporal Bone Histopathology in Acute Lymphocytic Leukemia." Otolaryngology–Head and Neck Surgery 139, no. 2_suppl (August 2008): P194. http://dx.doi.org/10.1016/j.otohns.2008.05.603.

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Problem There are reports of hearing loss, tinnitus, and/or vertigo in patients with leukemia. However, there is no human temporal bone study of a large number of cases specific to acute lymphocytic leukemia. We studied the correlation between clinical otologic complaints and temporal bone histopathology in patients with this disease. Methods Clinical otologic complaints and histologic findings were evaluated in 13 patients (25 temporal bones) with acute lymphocytic leukemia. Results Nine patients had a history of clinical otologic complaints including: hearing loss in 5 patients; otalgia in 3; otorrhea in 3; and dizziness in 2. Hemorrhage was seen most commonly in the middle ear in 10 patients, but was also evident in the cochlea in 5 and the vestibular labyrinth in 2. Leukemic infiltration was observed in the petrous apex in 12 patients, in the middle ear in 6, the cochlea in 5, the vestibular labyrinth in 2 and the internal auditory canal in 3. Inflammatory cell infiltration was also seen in the cochlear labyrinth in 4 patient, the vestibular labyrinth in 5 and the modiolus in 1. Otitis media with hyperplasia of subepithelial fibrous tissue was seen in 10 patients. Three patients had granulation tissue extending into the middle ear or mastoid. Conclusion Ear involvement is a common finding in patients with acute lymphocytic leukemia. Significance With prolonged survival due to new chemotherapeutics, the diagnosis and treatment of non-hematopoietic system complications such as ear problems due to acute lymphocytic leukemia have become more important. Support International Hearing Foundation, Hubbard Foundation, Starkey Foundation.
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Golz, Avishay, S. Thomas Westerman, Liane M. Gilbert, Henry Z. Joachims, and Aviram Netzer. "Effect of middle ear effusion on the vestibular labyrinth." Journal of Laryngology & Otology 105, no. 12 (December 1991): 987–89. http://dx.doi.org/10.1017/s002221510011802x.

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AbstractAlthough middle ear effusion was once described as the most common cause of vestibular disturbance in children, the association between glue ear and symptoms of dysequilibrium has never been quantified objectively.In this study the effect of middle ear effusion on the vestibular system of the inner ear was studied in a select group of children suffering from long lasting effusion in the middle ear with no evidence of infection at least one year prior to the study. The results were compared with results obtained from otitis free children, as well as from examinations of children after the insertion of ventilating tubes.The results of this study confirm the assumption that middle ear effusion has an adverse effect on the vestibular system, which can be resolved following the insertion of ventilation tubes.This effect may also contribute to the adverse effect that otitis media has on a child's development.
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Le Maître, A. "Role of Spatial Integration in the Morphology of the Bony Labyrinth in Modern Humans." Bulletins et Mémoires de la Société d'Anthropologie de Paris 31, no. 1-2 (January 30, 2019): 34–42. http://dx.doi.org/10.3166/bmsap-2018-0039.

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The bony labyrinth corresponds to the osseous wall of the inner ear, the hearing and balance organ located in the petrous pyramids, in the base of the cranium. The morphology of the labyrinth reflects phylogenetic and ecological signals. In hominoid primates, it is also influenced by its anatomical environment. The aim of this study is to determine whether, in the species Homo sapiens, the morphological relationships between the labyrinth and the skull result from geometrical constraints linked to equilibrioception, or from spatial constraints due to the inclusion of the inner ear in the petrous bone. Based on CT-scans of the skulls of adult individuals (n=30), two sets of 22 landmarks each were placed on the labyrinth and on the basicranium. The covariations between these two sets were investigated using twoblock partial least squares (2B-PLS) analyses. The shape of the labyrinth is significantly correlated with the cranial base, but not with the isolated temporal bone. This indicates that the morphology of the labyrinth mainly results from functional constraints. However, several observations suggest that spatial constraints also have an influence, especially on the cochlea. The associated changes in shape are consistent with the ontogenetic trend, but differ slightly from the phylogenetic trend. These covariations caution against exclusively ecological interpretations of the morphology of the labyrinth.
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Dissertations / Theses on the topic "Labyrinth (ear)"

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Adamska, Maja. "Nkx5 genes in inner ear development and genome evolution." [S.l. : s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=96230459X.

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Li, Junchang, and 李俊畅. "Sox2 and inner ear development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hdl.handle.net/10722/206990.

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Sox2, a HMG box transcription factor, is well known for its role in stem cell maintenance, iPS (induced pluripotent stem cell) induction, and development of neural tissues such as central nervous system and sensory organs. Sox2 has been demonstrated to be essential for the development of inner ear sensory patches. It has been shown that Sox2 is under the regulation of multiple regulatory elements to obtain a tissue specific manner. Two allelic mouse mutants, yellow submarine (Ysb) and Light coat and circling (Lcc) show hearing and balance impairments with different severity. They were made by random insertions of a transgene (pAA2) and X-ray irradiation respectively. Ysb and Lcc are both localized to chromosome 3 and involve complex chromosomal rearrangements. The Sox2 coding region is intact in the mutants, while the Sox2 expression in the otocyst is greatly reduced in Ysb and totally lost in Lcc, which indicates the tissue specific reduction of Sox2 may be due to the rearrangement of Sox2 regulatory element(s). Since Sox2 null mutants die before implantation, the two Sox2 inner ear mutants are valuable models for studying Sox2 knock down (Ysb) and Sox2 knock out (Lcc) condition in the inner ear. To understand the molecular basis behind Sox2 regulation in the inner ear, this project aims to identify the Sox2 otic regulatory elements, and potential Sox2 downstream targets involved in the development of inner ear. Previous work has indicated that Nop1 and Nop2 are the otic specific regulatory elements of Sox2 in chicken ear. In this project, transgenic mice were generated using Nop1-Nop2, and the result showed Nop1-Nop2 could drive Sox2 expression to the dorsal side of the otiv vesicle, which is different from the endogenous Sox2 expression pattern. Therefore, Nop1 and Nop2 may require other regulatory element(s) to gain a correct regulatory pattern. BAC(RP23-335P23), which contained the DNA sequences close to Ysb integration site 1 was also been tested in transgenic mice. Interestingly, the result showed that BAC(RP23-335P23) could drive Sox2 expression to the ventral side of the otic vesicle, indicating that this BAC may contain the Sox2 otic regulatory element(s). In this project, the binding relationship between Sox2 protein and Math1 enhancer has also been identified using chromatin immunoprecipitation (Ch-IP). Results showed that Sox2 could bind to Math1 enhancer A in the inner ear cochlea. So Sox2 may regulate Math1 through binding to Math1 enhancer A in inner ear development. Using a bioinformatics approach, potential Sox2 target genes in inner ear development have been identified from public microarray data on E9 to E15 inner ear tissue by the presence of conserved Sox2 binding sites. Among these potential targets, 4 genes (Itga6, Erbb3, Sox10 and Mycn) have been selected based on their known functions. Their expression patterns in the cochlea of wild type, Ysb and Lcc were verified. The identification of Sox2 downstream target genes using a bioinformatics approach will help us to understand the molecular basis of Sox2 regulation, and also understand the role of Sox2 in the inner ear development.
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Biochemistry
Master
Master of Philosophy
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Hu, Zhengqing. "Investigating a cell replacement therapy in the inner ear /." Stockholm, 2004. http://diss.kib.ki.se/2005/91-7140-170-9/.

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Zelenskaya, Alexandra. "Atomic force microscopic studies of inner ear structure and mechanics /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-021-4/.

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Tang, Shiu-ping Anna. "Molecular developmental genetics of the inner ear mutant, yellow submarine (Ysb)." Click to view the E-thesis via HKUTO, 2004. http://sunzi.lib.hku.hk/hkuto/record/B43895712.

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Mak, Chi-yan Angel, and 麥志昕. "Bioinformatic and functional approaches to identify potential SOX9 target genes in inner ear development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hdl.handle.net/10722/193405.

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Liang, Guihua. "K⁺ channels in the inner ear : electrophysiological and molecular studies /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7349-971-4/.

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Duncan, Robert Keith. "Finite-element analysis of inner ear hair bundles : a parameter study of bundle mechanics /." Thesis, This resource online, 1993. http://scholar.lib.vt.edu/theses/available/etd-09292009-020226/.

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Liu, Yuchen, and 刘雨辰. "The roles of Irx3 and Irx5 genes in mammalian inner ear development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hdl.handle.net/10722/207900.

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Iroquois genes encode a family of highly conserved TALE homeodomain transcription factors that are involved in multiple developmental processes. Physiological tests indicated that Irx3 and Irx5 mutant mice displayed hearing impairment. However, the functions of these two genes during inner ear development are not known. The aim of this study is to characterize the roles of Irx3 and Irx5 during mammalian inner ear development using mouse models, in order to reveal the underlying mechanism for the hearing abnormality in the mutants. Two mouse mutants, Irx3tauLacZ and Irx3flox5EGFP with β-gal and EGFP reporters, were analyzed to examine the expression of these two genes in the otic vesicle and cochlear epithelium. In the otocyst, both Irx3 and Irx5 were expressed in the ventral-medial region. Irx5 expression was restricted to the non-sensory domain of the cochlear epithelia, while Irx3 was widely expressed, including the auditory sensory organ, the organ of Corti. The overlapping expression patterns of Irx3 and Irx5 suggest that they may share redundant functions. To investigate the roles of Irx3 and Irx5 during inner ear development, phenotypic analysis was performed on Irx3-/-, Irx5-/- and Irx3/5-/- mutant embryos. As shown by paint-filling analysis, Irx3/5-/- displayed shortened cochlear duct, enlarged cochlear lumen with fused sensory organ. Whole-mount phalloidin staining of hair cell bundles showed that Irx3-/- displayed occasional ectopic inner hair cells. Moreover, only supernumerary vestibular hair cell-like cells were developed in Irx3/5-/- mutant. These results suggest that Irx3 and Irx5 are required for inner ear morphogenesis and the formation of organ of Corti. To understand the effect of Irx3 and Irx5 in the cellular patterning of the cochlea, mutant cochleae were analyzed with markers for different regions of the cochlear epithelia. Altered expression domain of MyoVIIa, Sox2 and Gata2 in Irx3/5-/- cochlea revealed that the boundary between the Kolliker’s organ and the organ of Corti was lost and the location of sensory and non-sensory region was shifted. These results imply that Irx3 and Irx5 function in the establishment of the sensory/non-sensory boundary. It is known that p27kip1 regulates the wave of cell cycle exit in the developing organ of Corti and Sox2 takes part in prosensory specification. To explore the underlying reason for the patterning defects in Irx3/5-/- mutant, cochlear duct from prosensory stages were analyzed. Irx3/5-/- showed altered Sox2 and p27kip1 expression, with expanded prosensory domain and disrupted cell cycle exit. Ectopic prosensory proliferation was detected in the middle turn of the cochlear duct at E13.5 by BrdU incorporation assay. Therefore, Irx3 and Irx5 may participate in the subdivision of sensory territory in developing cochlea by controlling prosensory proliferation. In summary, this study demonstrates that Irx3 and Irx5 cooperate in multiple aspects of inner ear development: an early role to regulate prosensory proliferation and cell cycle exit; a second role to regulate cellular patterning of the cochlear duct by controlling the setting of sensory/non-sensory boundaries in the cochlea; a later role to regulate inner ear morphogenesis. This study supports the idea that Irx3 and Irx5 act as patterning genes during vertebrate evolution.
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Biochemistry
Master
Master of Philosophy
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Tang, Shiu-ping Anna, and 鄧紹平. "Molecular developmental genetics of the inner ear mutant, yellow submarine (Ysb)." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B43895712.

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Books on the topic "Labyrinth (ear)"

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1932-, Ciges M., and Campos A. 1951-, eds. Inner ear pathobiology. Basel: Karger, 1990.

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D, Felix, and Oestreicher E, eds. Rational pharmacotherapy of the inner ear. Basel: Karger, 2002.

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L, Leverenz Ellen, and Bialek William S, eds. The vertebrate inner ear. Boca Raton, Fla: CRC Press, 1985.

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Robert, Marchbanks, Ernst Arne 1958-, Reid Andrew 1954-, and International Conference on Intracranial and Inner Ear Fluids (2nd : 1997 : Bath, England), eds. Intracranial and inner ear physiology and pathophysiology. London: Whurr, 1998.

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Dileo, Michael D. Trauma to the middle and inner ear. Alexandria, VA: American Academy of Otolaryngology--Head and Neck Surgery Foundation, 1997.

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Harada, Yasuo. The vestibular organs: S.E.M. atlas of the inner ear. Niigata, Japan: Nishimura, 1988.

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Iurato, Salvatore. New and conventional techniques in human auditory and vestibular pathology. Stockholm, Sweden: Distributed by Almqvist & Wiksell International, 1990.

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P. R. J. W. Ruding. Experimental endolymphatic hydrops: A histophysiological study. The Hague: SPB Academic Publishing, 1987.

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International Cochlea-Symposion. (8th 1987 Halle an der Saale, Germany). VIII International Cochlea-Symposion: Halle (Saale), May 28.-31., 1987. Edited by Loebe Lutz-Peter and Lotz Peter. Halle (Saale): Martin-Luther-Universität Halle-Wittenberg, 1988.

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National Institute on Deafness and Other Communication Disorders (U.S.). Balance disorders. 2nd ed. Bethesda, MD: U.S. Department of Health & Human Services, National Institutes of Health, National Institute on Deafness and Other Communication Disorders, 2009.

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Book chapters on the topic "Labyrinth (ear)"

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Schrott-Fischer, A., K. Kammen-Jolly, A. W. Scholtz, W. j. Kong, and M. Eybalin. "Neurotransmission in the Human Labyrinth." In Rational Pharmacotherapy of the Inner Ear, 11–17. Basel: KARGER, 2002. http://dx.doi.org/10.1159/000059237.

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Manfré, L., R. Lagalla, and A. E. Cardinale. "MRI of inner ear fluids using modified GRASS sequences: a useful tool in the assessment of the normal and pathological labyrinth." In Proceedings of the XV Symposium Neuroradiologicum, 397–99. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79434-6_189.

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"The Labyrinth." In Middle Ear and Mastoid Microsurgery, edited by Mario Sanna, Hiroshi Sunose, Fernando Mancini, Alessandra Russo, Abdelkader Taibah, and Maurizio Falcioni. Stuttgart: Georg Thieme Verlag, 2012. http://dx.doi.org/10.1055/b-0034-74919.

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"The Labyrinth." In Middle Ear and Mastoid Microsurgery, edited by Mario Sanna, Hiroshi Sunose, Fernando Mancini, Alessandra Russo, and Abdelkader Taibah. Stuttgart: Georg Thieme Verlag, 2003. http://dx.doi.org/10.1055/b-0034-52318.

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van Gijn, Daniel R., and Jonathan Dunne. "The ear." In Oxford Handbook of Head and Neck Anatomy, edited by Susan Standring and Simon Eccles, 497–518. Oxford University Press, 2022. http://dx.doi.org/10.1093/med/9780198767831.003.0014.

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The delicate yet definitive deflections of the pinna (wing/fin) of the external ear contribute to the collection of sound. The external acoustic meatus is responsible for the transmission of sounds to the tympanic membrane, which in turn separates the external ear from the middle ear. The middle ear is an air filled (from the nasopharynx via the eustachian tube), mucous membrane lined space in the petrous temporal bone. It is separated from the inner ear by the medial wall of the tympanic cavity – bridged by the trio of ossicles. The inner ear refers to the bony and membranous labyrinth and their respective contents. The osseus labyrinth lies within the petrous temporal bone. It consists of the cochlea anteriorly, semicircular canals posterosuperiorly and intervening vestibule – the entrance hall to the inner ear whose lateral wall bears the oval window occupied by the stapes footplate.
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"THE LABYRINTH AND EIGHTH NERVE." In Diseases of the Nose, Throat and Ear, 411–35. Elsevier, 2013. http://dx.doi.org/10.1016/b978-1-4831-6685-8.50048-2.

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MCNALLY, W. J., and E. A. STUART. "FUNCTIONS OF THE LABYRINTH." In Modern Trends in Diseases of the Ear, Nose and Throat, 1–12. Elsevier, 2013. http://dx.doi.org/10.1016/b978-1-4831-6732-9.50006-3.

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Fisch, Adam. "Vestibular System." In Neuroanatomy : Draw It to Know It, 234–45. Oxford University PressNew York, NY, 2009. http://dx.doi.org/10.1093/oso/9780195369946.003.0022.

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Abstract In This Chapter, we will draw the peripheral vestibulocochlear system and central vestibular system. The peripheral vestibulocochlear region comprises the outer ear; the external ear canal, which extends through the mastoid process; the middle ear, which encompasses the ossicles in the mastoid process; and the inner ear, which is the bony labyrinth in the temporal bone. The auditory component functions in sound perception and the vestibular component keeps us balanced despite constant motion and also helps us fixate our vision. Move your head about while reading this text. If your vestibular system is intact, you won’t become nauseated or have much drop in visual accuity—just a one line drop on a Snellen visual chart (e.g., from 20/20 to 20/25). When the vestibular system is damaged, on the other hand, even slight movements are dizzying.
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Haddad, Joseph. "The Inner Ear and Diseases of the Bony Labyrinth." In Nelson Textbook of Pediatrics, 2213–2213. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4377-0755-7.00633-3.

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Burrington, Debra D., and Robin Throne. "Navigating the Doctoral Labyrinth." In Practice-Based and Practice-Led Research for Dissertation Development, 64–86. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-6664-0.ch004.

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The journey through a practitioner doctorate program to earn the terminal degree has been likened to a labyrinth with its complex maze or a mountain climb with its inherent obstacles and challenges the doctoral scholar must surpass to be successful. Reflexive positionality is a concomitant process which can be used throughout the practitioner doctorate to facilitate the iterative and recursive journey of the scholar-practitioner. Reflexivity facilitates an introspective process to elevate professional practitioners to scholar-practitioners and incorporate a reflexive view to transcend doctoral hurdles, attain the terminal degree, and return to practice to solve societal problems and problems within the discipline. Practitioner doctorate leadership may also be well served to incorporate reflexivity in the pedagogical redesign of the practitioner doctorate to strengthen the development and preparation of these scholar-practitioners.
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Conference papers on the topic "Labyrinth (ear)"

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Wu, Caiqin, Keqiang Wang, Lin Yang, and Peidong Dai. "Three-dimensional models of the membranous vestibular labyrinth in the guinea pig inner ear." In 2011 4th International Conference on Biomedical Engineering and Informatics. IEEE, 2011. http://dx.doi.org/10.1109/bmei.2011.6098245.

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