Relevant bibliographies by topics / Keratitis

Academic literature on the topic 'Keratitis'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2024

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Journal articles on the topic "Keratitis"

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Manafli, Leyla, and Saulius Galgauskas. "CONTACT LENS RELATED MICROBIAL KERATITIS." Health Sciences 34, no. 3 (May 1, 2024): 187–90. http://dx.doi.org/10.35988/sm-hs.2024.132.

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Microbial keratitis is frequently encountered condition in otherwise healthy individuals using contact lenses. Hygiene factors and usage of inappropriate cleansing solutions are the leading causes of contact lens related microbial keratitis in healthy population. The aim of this paper is to review the risk factors predisposing to this condition, most common causative agents and their pat­hologic mechanisms, diagnostic methods and treatment options. Raktžodžiai: Mikrobinis keratitas, kontaktiniai lęšiai, su kontaktiniais lęšiais susijęs mikrobinis keratitas, infek­cija, Acanthamoeba keratitas (AK), Pseudomonas aeru­ginosa keratitas, grybelinis keratitas (FK).
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O’Brien, Rebecca L., Zhifang Yang, Yafei Huang, Josh Loomis, Amie L. Owen, and Willi K. H. Born. "γδ T cells can play a role in provoking an inflammatory attack on the cornea." Journal of Immunology 196, no. 1_Supplement (May 1, 2016): 117.16. http://dx.doi.org/10.4049/jimmunol.196.supp.117.16.

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Abstract Ocular immune privilege results from a complex set of mechanisms that together prevent immunogenic inflammation, but still allow the eye to deal with potentially infectious agents in a localized way that does not compromise vision. Failure of immune privilege can unleash a damaging autoimmune attack on the eye. γδ T cells have been found to be important for these processes in a number of different systems. In mice of the C57BL/10 background (B10), we have previously shown that γδ. T cells help prevent the spontaneous development of keratitis, an inflammation of the cornea that B10.TCRδ −/− mice are highly prone to develop, particularly the females. Paradoxically, B10 mice that can produce γδ T cells but not αβ T cells also develop keratitis at a high rate. We recently found that Vγ4+ cells from the spleens of keratitic B10.TCRβ−/− mice, but not Vγ1+ cells, can adoptively transfer the disease to normally keratitis-resistant B10.TCRβ−/−δ−/− hosts. Immunofluorescence staining of corneal whole mounts from B10.TCRβ−/− mice revealed that Vγ4+ cells infiltrate the keratitic corneas, and show a strong bias to secrete IL-17. In contrast, Vγ1+ cells were more rare in the keratitic corneas and did not produce IL-17. The majority of the γδ T cells in keratitic corneas were Vγ1-negative, Vγ4-negative, and Vγ7-negative, however, and did not produce IL-17. We hypothesize that although some γδ T cell subsets protect against autoimmune attack on the cornea, certain Vγ4+ γδ T cells instead promote keratitis by infiltrating the corneas and secreting IL-17, which attracts and mobilizes neutrophils, resulting in tissue damage.
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ŞİNGAR, Evin, Ayşe BURCU, Ahmet KADERLİ, Züleyha YALNIZ AKKAYA, Selma ÖZBEK UZMAN, and Firdevs ÖRNEK. "Newly Acquired Herpetic Epithelial Keratitis After Penetrating Keratoplasty without Previous History of Herpetic." Turkiye Klinikleri Journal of Ophthalmology 29, no. 4 (2020): 316–23. http://dx.doi.org/10.5336/ophthal.2020-75349.

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Biser, Seth A., Henry D. Perry, Eric D. Donnenfeld, Sima J. Doshi, and Vishnu Chaturvedi. "Arthrographis Keratitis Mimicking Acanthamoeba Keratitis." Cornea 23, no. 3 (April 2004): 314–17. http://dx.doi.org/10.1097/00003226-200404000-00018.

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Sharma, Savitri. "Keratitis." Bioscience Reports 21, no. 4 (August 1, 2001): 419–44. http://dx.doi.org/10.1023/a:1017939725776.

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Corneal inflammation or keratitis is a significant cause of ocular morbidity around the world. Fortunately, the majority of the cases are successfully managed with medical therapy, but the failure of therapy does occur, leading to devastating consequences of either losing the vision or the eye. This review attempts to provide current information on most, though not all, aspects of keratitis. Corneal inflammation may be ulcerative or nonulcerative and may arise because of infectious or noninfectious causes. The nonulcerative corneal inflammation may be confined to the epithelial layer or to the stroma of the cornea or may affect both. For clarity, this section has been divided into nonulcerative superficial keratitis and nonulcerative stromal keratitis. While the former usually includes hypersensitivity responses to microbial toxins and unknown agents, the latter can be either infectious or noninfectious. In the pathogenesis of ulcerative keratitis, microorganisms such as bacteria, fungi, parasites (Acanthamoeba), or viruses play an important role. Approximately, 12.2% of all corneal transplantations are done for active infectious keratitis. Available world literature pertaining to the incidence of microbial keratitis has been provided special place in this review. On the other hand, noninfectious ulcerative keratitis can be related to a variety of systemic or local causes, predominantly of autoimmune origin.
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Lin, Ting-Ting, Rui-Hua Wei, Rui-Bo Yang, Yue Huang, Chen Zhang, Yu-Xian Ning, and Shao-Zhen Zhao. "Fungal Keratitis Associated with Viral Keratitis." Chinese Medical Journal 128, no. 20 (October 2015): 2823–25. http://dx.doi.org/10.4103/0366-6999.167367.

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Froumis, Nicholas A., Bartly J. Mondino, and Ben J. Glasgow. "Acanthamoeba keratitis associated with fungal keratitis." American Journal of Ophthalmology 131, no. 4 (April 2001): 508–9. http://dx.doi.org/10.1016/s0002-9394(00)00827-8.

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Goodall, Karen, Arun Brahma, and Alan Ridgway. "Acanthamoeba keratitis: Masquerading as adenoviral keratitis." Eye 10, no. 5 (September 1996): 643–44. http://dx.doi.org/10.1038/eye.1996.148.

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Mathers, William D. "Coexistent Acanthamoeba Keratitis and Herpetic Keratitis." Archives of Ophthalmology 115, no. 6 (June 1, 1997): 714. http://dx.doi.org/10.1001/archopht.1997.01100150716002.

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Shrestha, Poonam, and Santosh Paudel. "Stromal Keratitis among Herpes Simplex Keratitis Patients in a Tertiary Eye Hospital: A Descriptive Cross-sectional Study." Journal of Nepal Medical Association 60, no. 256 (November 30, 2022): 1008–10. http://dx.doi.org/10.31729/jnma.7906.

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Introduction: The manifestations of herpes simplex virus keratitis range from epithelial keratitis to vision-threatening stromal keratitis. There are limited studies done on our part regarding stromal keratitis. The aim of the study was to find out the prevalence of stromal keratitis among herpes simples keratitis patients in a tertiary eye hospital. Methods: This descriptive cross-sectional study was conducted among patients who presented with herpes simplex virus keratitis in a tertiary eye hospital between 1 January 2020 to 28 February 2022. Ethical clearance was taken from Ethical Review Board (Reference number: 1/2079/80). Data was collected from hospital records which was reviewed and visual acuity at presentation and at one-month follow-up, clinical details on examination were recorded and, diagnosis of the stage of disease given in the case record was noted. Convenience sampling was used. Point estimate and 95% Confidence Interval were calculated. Results: Among 112 Herpes simplex keratitis patients, the prevalence of stromal keratitis was 38 (33.93%) (25.16-42.70, 95% Confidence Interval). Conclusions: The prevalence of stromal keratitis among patients of herpes simplex keratitis was similar to studies conducted in similar settings.
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Dissertations / Theses on the topic "Keratitis"

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Sueke, Henri. "Novel approaches in bacterial keratitis." Thesis, University of Liverpool, 2014. http://livrepository.liverpool.ac.uk/2006499/.

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Bacterial keratitis is a serious ophthalmic disease with significant visual morbidity. The aims of this thesis are to investigate (1) the prevalence and type of bacterial virulence factors in the two major species causing keratitis; Staphylococcal aureus and Pseudomonas aeruginosa and (2) the pharmacodynamics and pharmacokinetics of meropenem, a potentially novel antimicrobial for use in keratitis. The prevalence, genetic diversity and clinical relevance of the lukSF-PV gene, encoding the bacterial toxin Panton Valentine Leukocidin, were investigated in S. aureus, isolated from cases of bacterial keratitis in the UK. Patients with lukSF-PV+ve S. aureus were found to be associated with a trend to worse clinical outcome and more surgical interventions, with an effect unrelated to minimum inhibitory concentrations (MICs). This suggests that lukSF-PV may be an important virulence factor in S. aureus associated keratitis. The genetic characteristics of P. aeruginosa isolates collected from patients with bacterial keratitis were compared between two time periods; 2003-04 and 2009-10 using an Array Tube genotyping system. 71% of keratitis-associated P. aeruginosa isolates clustered together, with no evidence for major variations in the distribution of clone types between the two time periods. The “core keratitis cluster” was found to be related to the P. aeruginosa eccB clonal complex, which is associated with adaptation to survival in environmental water. This suggests that adaptation to environmental water is a key factor in the ability of P. aeruginosa to cause keratitis. Further analysis of clinical data and studies involving additional sets of patients for verification of this hypothesis will provide a clearer picture. Pharmacodynamic properties of commonly used and potentially novel antimicrobials were determined by calculating MICs against isolates from patients with keratitis. Antimicrobial combinations were investigated for synergy or antagonism against isolates of S. aureus and P. aeruginosa using E-Tests. Meropenem was identified as a potentially novel antimicrobial for use in keratitis, offering broad-spectrum cover against both Gram-positive and -negative microorganisms. Meropenem was also shown to show synergistic properties when used in combination with other commonly used antimicrobials. In light of these results, the antimicrobial meropenem was chosen to undergo further pharmacokinetic studies. MTT and Live Dead toxicity assays were performed on human keratocytes and human corneal epithelial cells treated with meropenem. Meropenem was found to have low toxicity against these cells. Corneal penetration of meropenem was assessed in human cadaver corneo-scleral discs mounted onto artificial anterior chambers. The concentration of meropenem in the aqueous estimated by HPLC and disc diffusion bioassay exceeded the MIC90 of E. coli in all 18 corneas tested after the 45 minutes sample point.
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Klocke, Julia. "Spontane bakterielle Keratitis in CD36-/- Knockout- Mäusen." Doctoral thesis, Universitätsbibliothek Leipzig, 2012. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-87190.

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PURPOSE. CD36 is a Class B scavenger receptor that is constitu- tively expressed in the corneal epithelium and has been impli- cated in many homeostatic functions, including the homeosta- sis of the epidermal barrier. The aim of this study is to determine (1) whether CD36 is required for the maintenance of the corneal epithelial barrier to infection, and (2) whether CD36-deficient mice present with an increased susceptibility to bacterial keratitis. METHODS. The corneas of CD36- /- , TSP1- /- , TLR2- /- , and C57BL/6 WT mice were screened via slit lamp microscopy or ex vivo analysis. The epithelial tight junctions and mucin layer were assessed via LC-biotin and Rose Bengal staining, respec- tively. Bacterial quantification was performed on corneal but- tons and GFP-expressing Staphylococcus aureus was used to study bacterial binding. RESULTS. CD36-/- mice develop spontaneous corneal defects that increased in frequency and severity with age. The mild corneal defects were characterized by a disruption in epithelial tight junctions and the mucin layer, an infiltrate of macro- phages, and increased bacterial binding. Bacterial quantifica- tion revealed high levels of Staphylococcus xylosus in the corneas of CD36-/- mice with severe defects, but not in wild-type controls. CONCLUSIONS. CD36 -/- mice develop spontaneous bacterial keratitis independent of TLR2 and TSP1. The authors conclude that CD36 is a critical component of the corneal epithelial barrier, and in the absence of CD36 the barrier breaks down, allowing bacteria to bind to the corneal epithelium and result- ing in spontaneous keratitis. This is the first report of sponta- neous bacterial keratitis in mice. (Invest Ophthalmol Vis Sci. 2011;52:256–263) DOI:10.1167/iovs.10-5566
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Clarke, Daniel William. "The pathogenic cascade of Acanthamoeba Keratitis." Access to abstract only; dissertation is embargoed until after 5/15/2007, 2006. http://www4.utsouthwestern.edu/library/ETD/etdDetails.cfm?etdID=153.

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Otri, Ahmad Muneer. "Infectious keratitis : a molecular and clinical study." Thesis, University of Nottingham, 2013. http://eprints.nottingham.ac.uk/13408/.

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Infectious keratitis is a sight threatening disease which can cause permanent visual loss if not diagnosed and treated at an early stage. It can be caused by different types of microbes which are either commensals or transferred from the environment. To fight against these threats, the ocular surface (OS) has developed innate and adaptive immune mechanisms. Antimicrobial peptides (AMPs) are natural effectors on the OS with actions that range from microbicidal effects to cell signalling. Human beta defensin (hBD) 1-3 and 9, Liver expressed antimicrobial peptide (LEAP) 1 and 2, human cathelicidin (LL37), ribonuclease7 (RNase7) are the main AMPs on the OS. In this work, the pattern of ocular AMPs gene expression in human OS cells treated with Acanthamoeba castellanii, Pseudomonas aeruginosa (P. aeruginosa) and Staphylococcus aureus (S. aureus) was studied and established. This was examined by quantitative real-time PCR (RT-PCR) using the Taqman assay. Among the studied AMPs, hBD3 gene showed the most significant increase in human OS cells infected with Acanthamoeba. LL37 demonstrated the highest level of gene expression in the samples infected with bacteria. In a different study, the gene expression of two AMPs (hBD3 and 9) was studied in OS samples taken from patients with different types of infectious keratitis both during and after the infection. This was compared with the expression in healthy subjects. Impression cytology (IC) was used to obtain samples of OS epithelium from recruited subjects. An optimized method for RNA extraction of IC samples was developed. Corresponding to the results of the in vitro study, hBD3 showed an overall up-regulation in all categories whereas hBD9 was down-regulated. These changes were most significant in patients with acute Acanthamoeba keratitis. The gene expression of both hBD3 and 9 showed a tendency towards returning to the levels found in healthy subjects when healing of the corneal infection was complete. In another study carried out to examine the antimicrobial activity of hBD3 we were surprised to find that we could not replicate this. We were unable to reproduce the previously reported antimicrobial activity of hBD3 but were able to demonstrate that the antimicrobial effect could be attributed to the acidic solvent used in preparing the hBD3 protein. The clinical significance of application of corneal densitometry as measured by the Pentacam system was assessed for the first time in patients with infectious keratitis. We demonstrated that corneal densitometry varied with levels of inflammation and was not confined to the site of infection only. It affected the whole cornea and reverted towards normal values as the inflammation settled when the infection was brought under control. We were able to demonstrate that densitometry can be used as a measure of the corneal response to infection and inflammation and could be used to monitor response to therapy. Finally, separate comprehensive prospective and retrospective studies of the clinical profile of severe infectious keratitis in Nottinghamshire were conducted. These two studies covered a total period of 7 years. The results of both studies were similar. Indeed, OS disease, CL wear and previous ocular surgery were found to be the most common risk factors. Positive results of corneal scraping were obtained only in about 40% of cases. Acanthamoeba, S. aureus, and P. aeruginosa were the most frequent causative organisms. Fortified topical antibiotics were effective in treating most cases. Therapeutic corneal grafting was found to be an effective and safe procedure in refractive infectious keratitis.
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Schubert, Tracey Lee Optometry &amp Vision Science Faculty of Science UNSW. "Increasing the ability of antibiotics to control S. aureus keratitis." Publisher:University of New South Wales. Optometry & Vision Science, 2008. http://handle.unsw.edu.au/1959.4/41516.

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Microbial keratitis is a major cause of avoidable visual impairment worldwide with S. aureus a leading cause of this disease in humans. Recently S. aureus isolated from eye infections have exhibited resistance to many antibiotics with those isolates from more severe infections exhibiting higher rates of antibiotic resistance. New therapies are therefore needed to ensure adequate treatment for these infections. In microbial keratitis the hosts' immune response is responsible for significant disease pathology so development of a therapy which also targets the immune response would be beneficial. The fimbrolides produced by Delisea pulchra are potential candidates as they are both growth inhibitory and immunomodulatory in vitro. The antibiotic susceptibility of clinical S. aureus isolates from ocular infections was determined using the CDS method and similarity of these isolates determined by PFGE and PCR-ribotyping. In addition the effect of fimbrolides on bacterial growth alone or in combination with antibiotics and the immune response to bacterial stimulation in PMNs and HCE cells was determined. Fimbrolides were then topically applied to S. aureus corneal infections and the effect of these compounds on disease progression determined by assessing corneal pathology, bacterial numbers and PMNs recovered from infected corneas. A larger proportion of keratitis isolates exhibited resistance to antibiotics than conjunctivitis isolates. Keratitis isolates were also related within a geographical region. The fimbrolides inhibited bacterial growth and modulated the immune response to bacterial stimulation in vitro. These compounds also exhibited synergy with conventional antimicrobials. In combination with ciprofloxacin the fimbrolides reduced the clinical score and numbers of bacteria recovered from ciprofloxacin-resistant or ciprofloxacin-sensitive S. aureus corneal infections. This thesis has identified that S. aureus isolates which produce the worst disease pathology in the eye are related and also exhibit higher rates of resistance to antibiotics indicating novel therapies to treat these infections are needed. This thesis demonstrated that fimbrolides inhibit S. aureus growth, exhibit synergy with antibiotics and modulate the immune response in vitro. In combination with ciprofloxacin the fimbrolides also improved disease pathology in keratitis, illustrating the potential of fimbrolides to be used as an adjunct therapy in the treatment of S. aureus keratitis.
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Dart, John Kenneth George. "Contact lens related ulcerative keratitis : epidemiology and pathogenesis." Thesis, University of Oxford, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.293396.

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Kozariychuk, N. Ya. "Approaches to recurrent meta-herpetic keratitis therapeutic treatment." Thesis, БДМУ, 2021. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18633.

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Keay, Lisa Jane Optometry &amp Vision Science Faculty of Science UNSW. "Public health impact of contact lens related microbial keratitis." Awarded by:University of New South Wales. School of Optometry and Vision Science, 2006. http://handle.unsw.edu.au/1959.4/26307.

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This thesis describes the impact of contact lens-related microbial keratitis in terms of incidence and severity. Disease outcome is defined by visual outcome, costs to the healthcare system, costs to the individual and duration of disease. A successful 12-month surveillance study was conducted of the populations of Australia and New Zealand to detect all cases of contact lens-related microbial keratitis. A random telephone survey of 32,000 households in Australia and 7,500 in New Zealand accurately determined the level of use of various contact lenses in the community. The impact of new contact lens types: silicone hydrogels and daily disposables were investigated. Increased risk persisted in overnight wear with silicone hydrogel materials. Microbial keratitis associated with silicone hydrogel materials had slightly shorter disease duration however other factors had a stronger influence on severity. Rigid gas permeable and frequent replacement soft lenses when used for daily wear constitute the lowest risk. Cost analysis was developed in a hospital case series of microbial keratitis. This analysis was applied in the surveillance study including cases managed in the private health care sector. Disease duration and associated costs are novel indices of severity for contact lens-related disease. The most dramatic effects on disease severity were seen with the type of organism involved. Keratitis attributed to environmental organisms (Gram-negative bacteria, Acanthamoeba, fungi and Nocardia species) were 10x more likely to cause loss of visual acuity, had longer duration of symptoms and incurred higher costs. Importantly, delays in receiving treatment increased disease duration and associated costs. Greater awareness of the need for specialist healthcare is indicated amongst health care providers and contact lens wearers. The hypothesis that overnight wear in silicone hydrogel lenses would not increase the risk of infection has been disproven. This information is of value to practitioners who are responsible for informing contact lens wearers about the risk of contact lens-related infections and should be weighed against the benefits of continuous wear. The identification of factors which contribute to the outcomes of disease will be used in education campaigns amongst health care providers and contact lens wearers to minimise the impact of disease.
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Williams, David Leonard. "Canine chronic superficial keratitis : histochemical characterisation and clinical management." Thesis, Royal Veterinary College (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.307438.

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Lee, Ming Yang. "Allele-specific siRNA therapy for keratitis-ichthyosis-deafness syndrome." Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10052698/.

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Dominant mutations in the gene GJB2 cause keratitis-ichthyosis-deafness (KID) syndrome, a severe condition affecting the skin, cornea and inner ear. GJB2 encodes the protein connexin-26 (Cx26) which forms hemichannels or gap junction channels allowing the passage of signalling molecules. Approximately 80% of KID syndrome patients carry a c.148G > A (p.D50N) mutation in GJB2, which results in aberrant channel function. We hypothesised that silencing of the mutant allele in patient keratinocytes using allele-specific siRNA could correct the channel function. First, to confirm whether patient keratinocytes with only one wildtype GJB2 allele formed functional channels following allele-specific siRNA treatment, GJB2+/- keratinocytes were generated using CRISPR/Cas9. The scrape-loading dye transfer (SLDT) assay showed no distinguishable difference in gap junction intercellular communication (GJIC) between GJB2+/- and GJB2+/+ cells, suggesting normal GJIC in GJB2+/- keratinocytes. Nineteen siRNAs were designed and tested in HeLa cells expressing wildtype or mutant GJB2-GFP transgene. A lead siRNA, was discovered, which potently inhibited the mutant mRNA and protein without affecting wildtype GJB2 expression. The efficacy of the lead siRNA was assessed using keratinocytes derived from a KID syndrome patient (KID-KC) harbouring heterozygous c.148G>A mutation. These cells displayed pathological features of KID syndrome, with reduced gap junction plaque formation, impaired GJIC and hyperactive hemichannels confirmed by immunostaining, SLDT, patch clamp and neurobiotin uptake assays. Following treatment with the siRNA, selective silencing of mutant GJB2 allele in KID-KCs was confirmed at mRNA and protein levels. Significant improvement of GJIC and reversal of hemichannel activity were detected, with the latter corrected to a level comparable to that recorded in normal keratinocytes. Furthermore, RNA-Seq analysis showed that only six genes in the KID-KC transcriptome were significantly altered by the siRNA treatment, suggesting low-level off-target effects. In conclusion, allele-specific siRNA silencing of pathogenic dominant GJB2 mutation could be a potential therapeutic intervention for KID syndrome.
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Books on the topic "Keratitis"

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Sun, Xuguang. Acanthamoeba Keratitis. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-5212-5.

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Tabery, Helena M. Adenovirus Epithelial Keratitis and Thygeson's Superficial Punctate Keratitis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-21634-3.

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Tandon, Radhika, Anat Galor, Virender Singh Sangwan, and Manotosh Ray, eds. Peripheral Ulcerative Keratitis. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-50404-9.

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Tabery, Helen. Herpes Simplex Virus Epithelial Keratitis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-01012-5.

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Tabery, Helena M. Adenovirus epithelial keratitis and Thygeson's superficial punctate keratitis: In vivo morphology in the human cornea. Heidelberg: Springer, 2012.

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C, Maudgal P., and Missotten Luc, eds. Herpetic eye diseases: Proceedings of the international symposium at the Katholieke Universiteit Leuven, Leuven, Belgium, May 17-19, 1984. Dordrecht: Junk, 1985.

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Tabery, Helena M. Varicella-Zoster Virus Epithelial Keratitis in Herpes Zoster Ophthalmicus. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-14487-5.

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Tabery, Helena M. Herpes simplex virus epithelial keratitis: In vivo morphology in the human cornea. Heidelberg: Springer, 2010.

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Kanski, Jack J. Ocular inflammatory disease. Philadelphia, PA: Elsevier Mosby, 2007.

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Tabery, Helena M. Varicella-Zoster Virus Epithelial Keratitis in Herpes Zoster Ophthalmicus: In Vivo Morphology in the Human Cornea. Berlin, Heidelberg: Springer-Verlag Berlin Heidelberg, 2011.

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Book chapters on the topic "Keratitis"

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Haeussler-Sinangin, Yesim, and Thomas Kohnen. "Keratitis." In Encyclopedia of Ophthalmology, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-642-35951-4_438-3.

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Haeussler-Sinangin, Yesim, and Thomas Kohnen. "Keratitis." In Encyclopedia of Ophthalmology, 992–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_438.

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Domachowske, Joseph, and Manika Suryadevara. "Keratitis." In Clinical Infectious Diseases Study Guide, 21–24. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50873-9_4.

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Gronthoud, Firza Alexander. "Keratitis." In Practical Clinical Microbiology and Infectious Diseases, 248–50. First edition. | Boca Raton : CRC Press, 2020.: CRC Press, 2020. http://dx.doi.org/10.1201/9781315194080-4-35.

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Gooch, Jan W. "Keratitis." In Encyclopedic Dictionary of Polymers, 903. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14072.

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Rai, Mahendra, Avinash P. Ingle, Indarchand Gupta, Pramod Ingle, Priti Paralikar, and Marcelo Luís Occhiutto. "Mycotic Keratitis." In Mycotic Keratitis, 1–17. Boca Raton, FL : CRC Press, Taylor & Francis Group, [2018] | “A: CRC Press, 2019. http://dx.doi.org/10.1201/9780429021473-1.

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Sun, Xuguang. "Epidemiology." In Acanthamoeba Keratitis, 1–3. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-5212-5_1.

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Sun, Xuguang. "Etiology." In Acanthamoeba Keratitis, 5–21. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-5212-5_2.

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Sun, Xuguang. "Pathological Mechanisms and Immunological Reactions." In Acanthamoeba Keratitis, 23–31. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-5212-5_3.

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Sun, Xuguang. "Clinical Manifestations." In Acanthamoeba Keratitis, 33–51. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-5212-5_4.

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Conference papers on the topic "Keratitis"

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Belskaia, K. I., and A. S. Obrubov. "СULTURAL DIAGNOSTIC METHOD OF FUNGAL KERATITIS." In XVII ВСЕРОССИЙСКАЯ ШКОЛА ОФТАЛЬМОЛОГА. ООО Бегемот-М, 2018. http://dx.doi.org/10.30808/978-5-6040782-2018-1-1-99-105.

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Simon, Carole, G. Wolf, M. Walther, K. Winkler, M. Finke, D. Hüttenberger, Markus Bischoff, B. Seitz, J. Cullum, and H. J. Foth. "Photodynamic inactivation of pathogens causing infectious keratitis." In SPIE BiOS, edited by David H. Kessel and Tayyaba Hasan. SPIE, 2014. http://dx.doi.org/10.1117/12.2037704.

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Herzog, Joshua M., and Volker Sick. "Towards Two-Color Fluorescence Imaging for Diagnosis of Microbial Keratitis." In Bio-Optics: Design and Application. Washington, D.C.: Optica Publishing Group, 2023. http://dx.doi.org/10.1364/boda.2023.dtu1a.3.

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Abstract:
Novel methods are needed for diagnosis of microbial keratitis. Two-color fluorescence imaging is proposed and tested on ex vivo porcine eyes. Results show the technique may be feasible but further quantitative characterization is needed.
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Sajeev, Shelda, and Mallika Prem Senthil. "Classifying infective keratitis using a deep learning approach." In ACSW '21: 2021 Australasian Computer Science Week Multiconference. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3437378.3437388.

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Lopes, Camila Martins, Isabela Penido Matosinhos, Wagner Mendes Moura, Rafael Prado Colares, Gustavo Lamego de Barros Costa, and Eduardo José do Rosário e. Souza. "PERIPHERAL ULCERATIVE KERATITIS WITH AUTOIMMUNE FEATURES CASE REPORT." In XL Congresso Brasileiro de Reumatologia. Sociedade Brasileiro de Reumatologia, 2023. http://dx.doi.org/10.47660/cbr.2023.2045.

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Karpinecz, Bianca, Natalie Edwards, and Vesna Zderic. "Ultrasound-Enhanced Drug Delivery for Treatment of Acanthamoeba Keratitis." In 2019 41st Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2019. http://dx.doi.org/10.1109/embc.2019.8856686.

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Vasquez-Perez, Alfonso, Sarah F. Osborne, and Kaveh Vahdani. "P-12 Necrotising blepharoconjunctivitis and keratitis in human monkeypox." In 2023 Proceeding of the XXIII Bowman Club Annual Meeting, 3rd March 2023. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/bmjophth-2023-bcm.12.

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Romano, Davide, Stephenie J. Tiew, and Tariq Mohammad. "P-13 Topical insulin eye drops in infective keratitis." In 2023 Proceeding of the XXIII Bowman Club Annual Meeting, 3rd March 2023. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/bmjophth-2023-bcm.13.

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Guimaraes Vieira Coelho, Izabela, Gustavo Lamego de Barros Costa, Juliana Cabrera Garrido, Corina Quental de Menezes Alvarenga, Filipe Didier Maciel, and Gustavo Braga Hallais França. "PERIPHERAL ULCERATIVE KERATITIS AND RHEUMATOID ARTHRITIS: A CASE REPORT." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17144.

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Jiang, Jiewei, Wei Liu, Jiamin Gong, and Mengjie Pei. "A Two-stage Algorithm for Automatic Diagnosis of Keratitis." In 2022 4th International Conference on Natural Language Processing (ICNLP). IEEE, 2022. http://dx.doi.org/10.1109/icnlp55136.2022.00009.

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