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Books on the topic 'Keratinocyte'

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Author: Grafiati

Published: 4 June 2021

Last updated: 6 July 2024

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1

Irene, Leigh, and Watt Fiona M, eds. Keratinocyte methods. Cambridge: Cambridge University Press, 1994.

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2

Irene, Leigh, Lane E. Birgitte, and Watt Fiona M, eds. The keratinocyte handbook. Cambridge [England]: Cambridge University Press, 1994.

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3

Jemec, Gregor B. E., Lajos Kemeny, and Donald Miech, eds. Non-Surgical Treatment of Keratinocyte Skin Cancer. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-79341-0.

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4

Michel, Darmon, and Blumenberg Miroslav Lj, eds. Molecular biology of the skin: The keratinocyte. San Diego: Academic Press, 1993.

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5

McMullan, Rachel Jane. Regulation of keratinocyte function by Rho Kinase. Birmingham: University of Birmingham, 2002.

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6

Dittlein, Daniela. Influence of keratinocyte derived mediators on CD4+ T cell plasticity. München: Universitätsbibliothek der TU München, 2016.

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7

M, Milstone Leonard, Edelson Richard L, and New York Academy of Sciences., eds. Endocrine, metabolic, and immunologic functions of keratinocytes. New York, N.Y: New York Academy of Sciences, 1988.

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8

Merendino, John Jerome. Keratinocytes produce a PTH-sensitive adenylate cyclase-stimulating factor. [New Haven: s.n.], 1985.

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9

Stacey, Michael William. Studies on cultured skin keratinocytes from normal and basal cell naevus syndrome patients. Birmingham: University of Birmingham, 1989.

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10

1935-, Ōkawara Akira, and McGuire Joseph, eds. The biology of the epidermis: Molecular and functional aspects : proceedings of the Fifth Japan-United States Symposium on the Biology of the Epidermis, Niseko, Hokkaido, 21-25 July 1991. Amsterdam: Elsevier, 1992.

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11

Massimi, Isabella. SspB cysteine protease of Staphylococcus aureus promotes detachment of human keratinocytes and degrades fibronectin and vitronectin. Ottawa: National Library of Canada, 2001.

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12

1957-, Rouabhia Mahmoud, ed. Skin substitute production by tissue engineering. Austin: Landes Bioscience, 1997.

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13

Epidermal cells: Methods and protocols. 2nd ed. Totowa, NJ: Humana Press, 2010.

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14

Gossai, Anala, Dorothea T. Barton, Judy R. Rees, Heather H. Nelson, and Margaret R. Karagas. Keratinocyte Cancers. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0058.

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Keratinocyte cancers (KC) include basal and squamous cell carcinomas that arise from keratinocytes or their precursors. KCs are the most common malignancies in humans. Basal cell carcinoma (BCC) has higher incidence rates, but squamous cell carcinoma (SCC) causes most deaths. Despite increasing incidence rates, the mortality rates have not changed markedly in recent years. The geographic and demographic features of these tumors have remained consistent over the past several decades, with a gradient of increasing incidence rates with proximity to the equator, predominantly affecting fair-skinned populations. Risk increases with age, is higher in men than women, and is associated with artificial as well as natural exposure to UV light. There is emerging evidence that these malignancies, particularly BCCs, may be increasing in younger adults and among women. While basal and squamous cell carcinomas share etiological factors, the relative importance of these factors, pattern of exposure, molecular alterations, and even the factors themselves differ.

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15

Keratinocyte Methods and Keratinocyte Handbook 2 volume set. Cambridge University Press, 1995.

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16

(Editor), Irene M. Leigh, and Fiona M. Watt (Editor), eds. Keratinocyte Methods Canadian binding. Cambridge University Press, 1995.

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17

Nonsurgical Treatment Of Keratinocyte Skin Cancer. Springer, 2009.

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18

Molecular Biology of the Skin: The Keratinocyte. Academic Press, 1993.

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19

(Editor), Michael Darmon, and Miroslav Blumenberg (Editor), eds. Molecular Biology of the Skin: The Keratinocyte. Academic Press, 1993.

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20

Jemec, Gregor, Lajos Kemeny, and Donald Miech. Non-Surgical Treatment of Keratinocyte Skin Cancer. Springer London, Limited, 2009.

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21

Jemec, Gregor, Lajos Kemeny, and Donald Miech. Non-Surgical Treatment of Keratinocyte Skin Cancer. Springer, 2014.

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22

Darmon, Michael, and Miroslav Blumenberg. Molecular Biology of the Skin: The Keratinocyte. Elsevier Science & Technology Books, 2012.

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23

Pasonen-Seppanen, Sanna. REGULATION OF KERATINOCYTE DIFFERENTIATION AND HYALURONAN METABOLISM IN AN ORGANOTYPIC KERATINOCYTE CULTURE [KUOPIO UNIVERSITY PUBLIATIONS D. MEDICAL SCIENCES, 370]. Kuopion Yliopisto Kuopio, Finland, 2005.

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24

Keratinocyte Biology: structure and function in the epidermis [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.95189.

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25

Rouleau, Jessica Leigh. Partial least squares modeling of keratinocyte differentiation through the integration of gene expression profiles and flow cytometry analysis. 2007.

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26

Green, Adèle C., and David C. Whiteman. Ultraviolet Radiation. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0014.

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Ultraviolet (UV) radiation is the principal cause of over 95% of keratinocyte cancers (basal cell carcinomas and squamous cell carcinomas of the skin), the most common cancers in white populations worldwide. UV radiation also causes an estimated 60%–90% of cutaneous melanoma, the cancer affecting the skin’s pigment-producing cells. In addition, UV radiation is the major cause of many eye diseases, including ocular cancers and cataract, the commonest cause of blindness, and is responsible for the underlying changes in skin aging, on which billions of dollars are spent annually in efforts to repair the damage. The sun is the principal source of human exposure to UV radiation. However, artificial sources are encountered in a wide range of industrial and medical settings, and increasingly from commercial tanning facilities. By the late twentieth century, nearly epidemic increases in skin cancer incidence had occurred in white populations, especially in Australia and New Zealand.

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27

Segaert, Siegfried. The Role of 1a,25-Dihydroxyvitamin D3 and the Vitamin D Receptor in the Control of Proliferation and Differentiation of the Epidermal Keratinocyte (Acta Biomedica Lovaniensia 196). Leuven Univ Pr, 1999.

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28

Keratinocytes: Structure, Molecular Mechanisms and Role in Immunity. Nova Science Pub Inc, 2013.

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29

Horch. Cultured Human Keratinocytes and Tissue Engineered Skin Substitutes. THIEME, 2001.

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30

Cultured Human Keratinocytes and Tissue Engineered Skin Substitutes. Thieme Medical Publishers, 2001.

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31

Lechner, Mark Stewart. Mechanisms of epithelial cell transformation by human papillomavirus and simian virus 40. 1993.

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32

Haes, Petra De. Effects of 1,25-dihydroxyvitamin D3 & Analogues on Uvb-irradiated Human Keratinocytes. Leuven Univ Pr, 2004.

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33

Raychaudhury, Amlanav. Structure and expression of a family of human type I epidermal keratin genes located on chromosome 17. 1988.

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34

Vantieghem, Katleen. Photoproduction of Vitamin D3 & Activation into 1a, 25-dihydroxyvitamin D3 in Human Epidermal Keratinocytes, Dermal Fibroblasts & Other Cells. Leuven Univ Pr, 2006.

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35

Kirkham, Bruce. Immunology and cytokine pathways. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0007.

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Psoriatic arthritis immunopathology has become the subject of intense study. These findings show differences to other forms of inflammatory arthritis in key pathways. Increased knowledge of innate immunity and the important role of IL-17/23 biology in both psoriasis and psoriatic arthritis, have led to new theories of immunopathogenesis in both conditions. Direct environmental stimuli could trigger innate immune cells resident in skin, which may then initiate a chronic adaptive immune response. The joint has fewer resident innate immune cells, but new studies show cells producing IL-17 may play key roles in immunopathology. The new information summarized here will provide important hypotheses for investigation of pathogenic pathways. Differences in non-immune cell function may also be critical mediators of response, for example, production of IL-12 or IL-23 by dendritic cells. Keratinocytes in skin and fibroblasts in joints may be critical in mediating cytokine production and effector function.

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36

Stuart, Philip E., Lam C. Tsoi, Caely A. Hambro, and James T. Elder. Genetics of psoriasis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0005.

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Psoriasis is an immune-mediated inflammatory disease (IMID) characterized by skin inflammation, epidermal hyperplasia, increased risk of arthritis, and cardiovascular morbidity. Substantial evidence indicates that psoriasis is driven by abnormal interactions between cells of the innate and adaptive host defence systems, including keratinocytes, dendritic cells, and T-cells, resulting in a dysregulated immune response and markedly increased epidermal proliferation. The precise aetiology of psoriasis remains unknown. Here, we review how innate and adaptive host defence responses are regulated by genetic factors that modulate the overall risk of psoriasis and dictate whether the disease affects the skin and/or the joints. Specifically, we review the epidemiologic basis for considering psoriasis as a genodermatosis, summarize knowledge derived from linkage and association studies of cutaneous psoriasis (PsC) and psoriatic arthritis (PsA), and attempt to relate genetic and immunologic discoveries in a pathogenetic framework that may eventually allow prediction of the development of PsA in psoriatic individuals.

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37

Epidermal Cells: Methods and Protocols (Methods in Molecular Biology). Humana Press, 2004.

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38

Thun, Michael J., Christopher P. Wild, and Graham Colditz. Framework for Understanding Cancer Prevention. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0061.

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The worldwide increase in the number of people affected by cancer and the costs of cancer care has increased the urgency of efforts to translate knowledge about the causes of cancer into effective preventive interventions. A wide range of interventions has proven to be effective for cancer prevention, either by reducing exposure to known causes of human cancer or by disrupting the multistage progression of tumors. Examples of progress include the up to 40% decrease in the age-standardized lung cancer incidence rate among men in high- and middle-income countries due to tobacco control; the 30% decrease in colorectal cancer incidence in the United States from widespread screening and the removal of precursor adenomatous tumors; the decreased prevalence of chronic hepatitis B virus (HBV) infection in East Asia due to neonatal vaccination; and protection against excessive sun exposure and decreased incidence of melanoma and keratinocytic carcinomas in Australia.

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39

Karlsson, Teresa. Studies on Vitamin a Signaling in Psoriasis: A Comparison Between Normal and Lesional Keratinocytes (Comprehensive Summaries of Uppsala Dissertations from the Faculty of Medicine, 1155). Uppsala Universitet, 2002.

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40

Grangsjo, Anders. Epidermal Keratinocytes Studied by X-Ray Microanalysis, With Special Reference to Contact Dermatitis: In Vivo and in Vitro Studies of Functional Changes ... Summaries of Uppsala Dissertations, 884). Uppsala Universitet, 1999.

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