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Books on the topic 'Jaws Muscles'

Author: Grafiati

Published: 4 June 2021

Last updated: 21 February 2023

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1

Ziermann, Janine M., Raul E. Diaz Jr, and Rui Diogo, eds. Heads, Jaws, and Muscles. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-93560-7.

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2

Fujimori, Hiroshi. Shikkari kande imasuka: Ago no hone to kinʾniku. Tōkyō: Kaiseisha, 1994.

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3

Schieferstein, Heinrich. Experimentelle Analyse des menschlichen Kausystems. München: Herbert Utz, 2003.

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4

Clinical anatomy of the masticatory apparatus peripharyngeal spaces. Stuttgart: G. Thieme Verlag, 1995.

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5

1939-, Morimoto Toshifumi, Matsuya Tokuzo, and Takada Kenji, eds. Brain and oral functions: Oral motor function an dysfunction : selected papers from the Osaka International Oral Physiology Symposium on Brain and Oral Function, Osaka, 3-5 September 1994. Amsterdam: Elsevier, 1995.

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6

Sectakof, Pavel A. Effects of functional appliances on functional activities of jaw muscles in Macaca Fascicularis. [Toronto: Faculty of Dentistry, University of Toronto], 1990.

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7

Bourque, Paul J. Effects of functional appliances on jaw muscle activity in macaca fascicularis. [Toronto: s.n.], 1987.

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8

Sectakof, Pavel A. The effects of functional appliances on functional activities of jaw muscles in Macaca fascicularis. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1992.

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9

John, Verity Jayne. The time course of naloxone-induced recurrence of mustard oil-evoked jaw muscle electromyographic activity. Ottawa: National Library of Canada, 1998.

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10

Tsai, Chih-Mong. Central neural pathways involved in craniofacial nociceptive reflex responses evoked in jaw muscles by mustard oil injection into the temporomandibular joint region. [Toronto: University of Toronto, Faculty of Dentistry], 1997.

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11

Cai, Bonnie Bao Yan. Sex-related differences in the suppressive effects of peripheral morphine but not GABA on reflex jaw muscle activity evoked by glutamate application to the TMJ region in rats. Ottawa: National Library of Canada, 2001.

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12

Heads, Jaws, and Muscles: Evolution, Development, Anatomical Diversity and Function. Springer International Publishing AG, 2019.

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13

Iven, Klineberg, Sessle Barry J. 1941-, International Union of Physiological Sciences. International Commission on Oral Physiology., International Association for Dental Research. Neuroscience Group., Australian Society of Prosthodontists, and International Symposium on "Oro-Facial Pain and Neuromuscular Dysfunction: Mechanisms and Clinical Correlates" (1983 : Sydney, N.S.W.), eds. Oro-facial pain and neuromuscular dysfunction: Mechanisms and clinical correlates. Oxford: Pergamon Press, 1985.

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14

(Editor), Toshifumi Morimoto, Tokuzo Matsuya (Editor), and Kenji Takada (Editor), eds. Brain and Oral Functions. Oral Motor Function and Dysfunction. Elsevier Science Pub Co, 1995.

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15

Devlin, Hugh, and Rebecca Craven. Temporomandibular joint and surrounding musculature. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759782.003.0002.

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The temporomandibular joint in relation to dentistry is the topic of this chapter. The first part of the chapter describes the muscle environment of the mandible and its movement in function. The treatment of temporomandibular disorders is followed by a discussion of occlusion and its clinical assessment. The final sections of the chapter describe the jaw reflexes, their physiology, and muscle function.

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16

Grandhe, Radhika P., Matthew Valeriano, and Dmitri Souza. Mechanical Chronic Jaw Pain. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0003.

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Mechanical jaw pain and temporomandibular joint (TMJ) disorders are the most common causes of nondental orofacial pain. The pain can originate from the joint structures or from the muscles of mastication. Diagnosis is based predominantly on the clinical history and exam findings, but imaging is indicated in certain circumstances. Secondary causes of chronic jaw pain must be sought out and meticulously ruled out. Patients presenting with TMJ pain have a high prevalence of fibromyalgia and other chronic pain conditions. Multidisciplinary treatment involving medications, minimizing parafunctional habits, oral splints, physical therapy, psychotherapy, and injections forms the cornerstone of management of this complex condition. Surgery is indicated in select conditions, such as ankylosis of the joint or tumors.

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17

Adams, Leverett A. Memoir on the Phylogeny of the Jaw Muscles in Recent and Fossil Vertebrates. Creative Media Partners, LLC, 2018.

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18

Zeller, Ranee. How To : Kinesiology? Relieve Jaw Pain and Speech Difficulties: Kinesiology Muscle Monitoring. Independently Published, 2019.

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19

Donaghy, Michael. The clinical approach. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0030.

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This chapter describes the appropriate clinical approach to take when presented with a patient reporting a neurological symptom. Just under 10 per cent of the population consult their general practitioner about a neurological symptom each year in the United Kingdom. About 10 per cent of these are referred for a specialist opinion, usually to a neurologist. Nine conditions account for roughly 75 per cent of general neurological referrals and are diagnosed initially on purely clinical grounds, with the other 25 per cent representing the full range of other, potentially very rare, neurological disorders.This chapter underlines the importance of a thorough and informative history to achieve successful diagnosis. Crucial facets for a good history include information on the time course of symptom development, whether symptoms are negative or positive, previous neurological history (both personal and familial), as well as other potentially contributory general medical disorders. The general neurological examination is also described, as are specific examination manoeuvres that may be added to the general neurological examination in specific clinical circumstances.Reflexes play an important role in diagnostic neurology because they reflect the integrity of, or alterations in, the neural structures responsible for their arc. Loss of a reflex may be due to interruption of the afferent path by a lesion involving the first sensory neurone in the peripheral nerves, plexuses, spinal nerves, or dorsal roots, by damage to the central paths of the arc in the brainstem or spinal cord, by lesions of the lower motor neurone at any point between the anterior horn cells and the muscles, of the muscles themselves, or by the neural depression produced by neural shock. In clinical practice, the most useful and oft-elicited reflexes are the tendon reflexes of the limbs, the jaw jerk, the plantar response, the superficial abdominal reflexes, the pupil-light response, and in infants, the Moro reflex. The place of these particular reflexes in the routine neurological examination is outlined, and the elicitation and significance of these reflexes and of a wide variety of others which are used occasionally are described.Examinations that allow localization lesions that are responsible for muscle weaknesses and the assessment of somatosensory abnormalities are described, as are neurological disorders that result in identifiable gait disorders. The clinical signs and examinations relevant to autonomic disorders are also discussed.Intensive care may be required for patients critically ill either as a result of primary neurological disease, or in those in whom a neurological disorder is a component of, or secondary to, a general medical disorder. Indications for admission to neurological intensive care have been defined (Howard et al. 2003): impaired consciousness, bulbar muscle failure, severe ventilatory respiratory failure, uncontrolled seizures, severely raised intracranial pressure, some monitoring and interventional treatments, and unforeseen general medical complications. Naturally specific treatments indicated for the particular diagnosis should be instituted along with general intensive care measures.Finally, the discussion of diagnoses of chronic or terminal conditions with patients is discussed, with particular focus on the best way to present the diagnosis to the patient.

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20

Thakore, Nimish J., and Erik P. Pioro. Clinical Presentations, Diagnostic Criteria, and Lab Testing. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0023.

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Amyotrophic lateral sclerosis (ALS) is the protypical motor neuron disease, which is characterized by the simultaneous presence of upper motor neuron (UMN) and lower motor neuron (LMN) signs in the same extremity or in the cranial-bulbar region. UMN signs at spinal levels include spasticity, slowness of motor activation, hyperactive deep tendon reflexes and extensor plantar responses, whereas UMN signs at the cranial level include spastic dysarthia (slow, labored, nasal); slowness of tongue movements, and hyperactive jaw, gag, and facial reflexes. LMN signs at the spinal level include muscle atrophy, fasciculations, and weakness and LMN signs at the cranial level include tongue atrophy and weakness, facial weakness, tongue and facial fasciculations, palatal weakness, weak cough, and dysphonia. ALA is fatal in 2 to 4 years, and the only medication known to prolong tracheostomy-free survival

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