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Academic literature on the topic 'Islands of Langerhans'

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Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Islands of Langerhans"

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Barakat, Hassan, Khaled Al-Roug, Raya Algonaiman, Sami A. Althwab, Hani A. Alfheeaid, Raghad M. Alhomaid, Mona S. Almujaydil, Taqwa Bushnaq, and Tarek A. Ebeid. "Biological Assessment of Stevioside and Sucralose as Sucrose Substitutes for Diabetics on STZ-Induced Diabetes in Rats." Molecules 28, no. 3 (January 17, 2023): 940. http://dx.doi.org/10.3390/molecules28030940.

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Numerous food organizations have identified excessive calorie consumption and accompanying ailments as significant health risks associated with high sugar consumption. Administering stevioside (ST), sucralose (SU), and the two synergically (SU+ST) affected normal rats’ weight gain. In the current study, SU showed the highest undesired effect. Indeed, administering the three treatments to diabetic rats (DR) did not improve the rats’ weight gain. Although, insulin injection synergically with the treatments improved the weight gain, as recorded after three weeks. The best-improving rate was observed in the ST group. After the administration of ST and ST+SU to the DR, the blood glucose level (GL) was positively affected, with SU having no effects on reducing the GL. A considerable reduction in serum insulin (SIL) was noted in the DR+SU group. On the contrary, ST did not negatively affect the SIL, rather an improvement was recorded. In addition, giving SU did not significantly affect the ALT level in the DR or normal rats (NR). A significant improvement in total bilirubin (TBILI) was observed when insulin was injected with ST or SU in DR groups. Further, triglycerides (TG) after administering ST, SU, or ST+SU to NR had no significant difference compared to the control group (NR). Although, the three treatments markedly but not significantly lowered TG in the DR. For total cholesterol (CHO), both DR and NR had no significant effect after the three treatments. No histopathological alterations were recorded in the NR group. Diffuse and severe atrophy of the islands of Langerhans due to depletion of their cells and mild papillary hyperplasia of the pancreatic ducts were represented by a slightly folded ductal basement membrane and newly formed ductules in STZ-DR. Simultaneous atrophy and absence of the cells of islands of Langerhans besides ductal hyperplasia were evident in DR+SU. Hyperplastic ductal epithelium and atrophic Langerhans cells were seen in DR+SU+In. Degeneration and mild atrophy were observed in the islands of Langerhans structures. There was essentially no noticeable change after utilizing ST. A slight shrinkage of the Langerhans’ islets was detected in DR+ST. In DR+ST+In, no histopathological alterations in the islands of Langerhans were recorded. Congestion in the stromal blood vessels associated with degenerative and necrotic changes in the cells of the islands of Langerhans in DR+SU+ST was observed. In NR+SU, congestion of the blood vessels associated with mild atrophy in the islands of Langerhans and dilatation in stromal blood vessels was noticed. In conclusion, ST is safe, and SU should be taken cautiously, such as mixing with ST and/or taken at a very low concentration to avoid its drastic effect on the human body.
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Aleydaputri, Andarista Diaz, and Nur Kuswanti. "Efek Ekstrak Daun Sawo Manila (Manilkara zapota L.) terhadap Profil Pulau Langerhans dan Berat Badan Mencit Diabetes." LenteraBio : Berkala Ilmiah Biologi 11, no. 1 (November 30, 2021): 122–30. http://dx.doi.org/10.26740/lenterabio.v11n1.p122-130.

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Diabetes melitus adalah penyakit gangguan metabolisme. Penelitian bertujuan mengetahui pengaruh ekstrak daun sawo manila terhadap profil pulau Langerhans dan berat badan mencit diabetes. Studi ini merupakan penelitian ekperimental menggunakan RAL dengan 6 kelompok (kontrol normal, kontrol aloksan, dosis ekstrak 1 (28mg/kg BB), dosis ekstrak 2 (56mg/kg BB), dosis ekstrak 3 (112mg/kg BB) dan glibenclamide), masing-masing dengan 4 ulangan. Mencit diabetes induksi aloksan diberi ekstrak daun sawo dua kali sehari selama 14 hari. Profil pulau Langerhans ditentukan berdasarkan diameternya melalui pengamatan preparat-HE pankreas. Berat badan mencit ditimbang pada hari ke 0, ke 7 dan ke 14. Data dianalisis menggunakan Anova. Hasil analisis menunjukkan ekstrak berpengaruh terhadap diameter p. Langerhans (p<0,05) dan berat badan (p<0,05) dengan perbedaan antar perlakuan. Rerata diameter p. Langerhans setelah 14 hari perlakuan yaitu sebesar 39 µm (KN), 34,5 µm (KA), 38,6 µm (K1), 56,5 µm (K2), 48,3 µm (K3) dan 41,6 µm (KG). Sedangkan berat badan mencit yaitu sebesar 33,25 (KN), 33,75 (KA), 32 (K1), 28,75 (K2), 30,75 (K3) dan 29,50 (KG). Ekstrak daun sawo berpengaruh terhadap profil pulau Langerhans dengan memperbesar diameternya dan terhadap berat badan mencit dengan tidak sejalan dosis. Kata kunci: Berat badan; Daun Sawo; Diabetes; Pulau Langerhans Abstract. Diabetes mellitus is a metabolic disorder disease. The study was to determine effect of sapodilla leaf extract on the profile of the islets of Langerhans and body weight of diabetic mice. This study is experimental study using RAL with 6 groups (normal control, alloxan control, extract dose 1 (28mg/kg BW), extract dose 2 (56mg/kg BW), extract dose 3 (112mg/kg BW) and glibenclamide), respectively. each with 4 replicates. Alloxan induced mice were given sapodilla leaf extract twice a day for 14 days. The profile the islets of Langerhans was determined based on their diameter by observing pancreatic HE-preparations. The BW of the mice was measured on 0, 7 and 14 days. Data were analyzed using Anova. The analysis showed that the extract had an effect on the diameter of the islets Langerhans (p< 0,05) and BW of mice (p< 0,05) with differences between treatments. The mean diameter of the islets of Langerhans after 14 days of treatment was 39 (KN), 34.5 (KA), 38.6 (K1), 56.5 (K2), 48.3 (K3) and 41.6 (KG) µms. While the weight of the mice were 33.25 (KN), 33.75 (KA), 32 (K1), 28.75 (K2), 30.75 (K3) and 29.50 (KG) grs. Sapodilla leaf extract affected the profile of Langerhans islands by increasing their diameter and on the body weight of mice in dose independent manner. Kata kunci: Bodyweight; Sapodilla leaf, Diabetic, Langerhans island
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Raffel, Andreas, Claus F. Eisenberger, Kenko Cupisti, Matthias Schott, Stephan E. Baldus, Imke Hoffmann, Feride Aydin, Wolfram T. Knoefel, and Nikolas H. Stoecklein. "Increased EpCAM expression in malignant insulinoma: potential clinical implications." European Journal of Endocrinology 162, no. 2 (February 2010): 391–98. http://dx.doi.org/10.1530/eje-08-0916.

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ObjectiveEpCAM (CD326) is overexpressed in progenitor cells of endocrine pancreatic islands of Langerhans during fetal development and was suggested to act as a morphoregulatory molecule in pancreatic island ontogeny. We tested whether EpCAM overexpression is reactivated in insulinomas, endocrine tumors arising in the pancreas.Design/methodWe used monoclonal anti-EpCAM antibody Ber-Ep4 for immunohistochemistry on formalin-fixed and paraffin-embedded tumor material. We analyzed 53 insulinomas: 40 benign (disease stage<IIa) and 13 malignant tumors (disease stage IIIb/IV). Disease stage disposition followed new TNM classification of the European Neuroendocrine Tumor Society (ENETS) for foregut neuroendocrine tumors (2006). Additionally, ten insulinoma metastases were analyzed. Clinical follow-up was available for overall survival analysis from 49 patients. The EpCAM expression of the islands of Langerhans was classified as 2+ in healthy pancreatic tissue.ResultsIn 38% of the benign insulinomas (disease stage<IIa), we found strong (3+) EpCAM expression. In contrast, malignant insulinomas (disease stage IIIb/IV) and their metastases exhibited a strong (3+) EpCAM expression with 78 and 80% respectively, significantly more frequent (P<0.01). The malignant tissue was characterized by a significantly lower number of unstained cells and significantly higher number of 3+ stained cells. Quantitative PCR for EpCAM mRNA validated strong EpCAM expression in malignant insulinoma. Kaplan–Meier curves indicated survival disadvantage for EpCAM 3+ insulinomas, but this was not statistically significant (log-rank test).ConclusionThis first EpCAM expression study in benign/malignant insulinomas indicates that strong EpCAM expression could help to identify patients at risk for malignant disease and might be used as a therapeutic target for antibody-based therapies in patients with metastatic insulinoma.
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Jassim Abd, Sarah, and Satar Abood Faris. "Effect of Streptozocin on the Langerhans Islands in the pancreas of birds." Sumer 1 8, CSS 1 (August 15, 2023): 1–8. http://dx.doi.org/10.21931/rb/css/2023.08.01.89.

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In the current study, birds (Columba livia ) were used as a new model to study the effect of streptozotocin on the pancreas gland and the blood glucose level. Three concentrations of 75,65,55 mg/kg were adopted for five consecutive days with one IP dose daily. The experimental animals showed a gradual rise in blood glucose the average glucose in the first week of the experiment was usual for the three groups compared with the control group, while there was a significant change in the blood glucose level in the three groups at the end of the experiment (4th week), where the average glucose in the streptozotocin groups was 55 mg/kg (213.80 ± 12.43) mg/dl and the group 65 mg/kg(282.60 ± 16.78) mg/dl and a group of 75 mg/kg( 371.0 ± 38.39) mg/dl. Keywords: Streptozocin, Langerhans islands, pancreas.
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Da Silva Xavier, Gabriela. "The Cells of the Islets of Langerhans." Journal of Clinical Medicine 7, no. 3 (March 12, 2018): 54. http://dx.doi.org/10.3390/jcm7030054.

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Islets of Langerhans are islands of endocrine cells scattered throughout the pancreas. A number of new studies have pointed to the potential for conversion of non-β islet cells in to insulin-producing β-cells to replenish β-cell mass as a means to treat diabetes. Understanding normal islet cell mass and function is important to help advance such treatment modalities: what should be the target islet/β-cell mass, does islet architecture matter to energy homeostasis, and what may happen if we lose a particular population of islet cells in favour of β-cells? These are all questions to which we will need answers for islet replacement therapy by transdifferentiation of non-β islet cells to be a reality in humans. We know a fair amount about the biology of β-cells but not quite as much about the other islet cell types. Until recently, we have not had a good grasp of islet mass and distribution in the human pancreas. In this review, we will look at current data on islet cells, focussing more on non-β cells, and on human pancreatic islet mass and distribution.
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Hultquist, Gosta T., and Bo Thorell. "CYTOLOGICAL CHANGES DURING THE EMBRYONAL FORMATION OF LANGERHANS' ISLANDS AS REVEALED BY ULTRAVIOLET MICROSCOPY." Acta Pathologica Microbiologica Scandinavica 32, no. 2 (August 18, 2009): 245–50. http://dx.doi.org/10.1111/j.1699-0463.1953.tb00249.x.

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BEEK, CORNELIA, A. J. CH HAEX, and P. J. KOOREMAN. "Two cases of spontaneous hypoglycemia due to a tumor of the islands of Langerhans." Acta Medica Scandinavica 112, no. 2 (April 24, 2009): 164–87. http://dx.doi.org/10.1111/j.0954-6820.1942.tb10011.x.

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TOVAR-HERNÁNDEZ, MARÍA ANA. "On some species of Chone Krøyer, 1856 (Polychaeta: Sabellidae) from world-wide localities." Zootaxa 1518, no. 1 (July 2, 2007): 31–68. http://dx.doi.org/10.11646/zootaxa.1518.1.2.

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The present study deals with the revision of type and non-type material from species of Chone Krøyer, 1856 (Polychaeta: Sabellidae) that have been described from world-wide isolated localities. Chone australiensis Hartmann-Schröder, 1979, C. fauveli McIntosh, 1916, C. kroyerii Sars, 1862, C. letterstedti (Kinberg, 1867), C. murmanica Lukasch, 1910, C. normani McIntosh, 1916, C. paracincta Hartmann-Schröder, 1962, and C. rosea Hartmann-Schröder, 1965, are redescribed. Two additional forms are recognized as independent taxa, referred to as Chone sp. “Aleutian Islands” and Chone sp. “British Virgin Islands”. These species are informally described since few specimens are available; however, they are included here in order to facilitate and encourage further research. Sabella costulata Grube, 1877, is transferred to Chone. Chone suspecta Krøyer, 1856, is synonymized with Chone infundibuliformis Krøyer, 1856; the name infundibuliformis has priority over suspecta. Chone murmanica oculata Annenkova, 1952, deserves specific status. Chone eniwetokensis (Reish, 1968) is returned to the original genus (Euchone). Chone perseyi Zenkewitsch, 1925, and Chone rubrocincta Sars, 1862, are transferred to Euchone. Chone reayi McIntosh, 1916, is transferred to Jasmineira Langerhans, 1880. Differences and similarities with members of Euchone Malmgren, 1866, and close genera are discussed.
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Myint, Myat Su Mon, and Antonio Pinero-Pilona. "LBSAT310 A Rare Disease Engulfing Thyroid And Masquerading As Common Thyroid Disorders." Journal of the Endocrine Society 6, Supplement_1 (November 1, 2022): A747—A748. http://dx.doi.org/10.1210/jendso/bvac150.1542.

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Abstract Introduction – Langerhans cell histiocytosis (LCH) is a rare clonal disorder caused by the CD207+ Langerhans cells derived from the myeloid origin. We present a case of LCH where it mimicked Hashimoto's and Riedel's thyroiditis and completely infiltrated the thyroid gland. Case description – A 54-year-old female was initially diagnosed with hypothyroidism, goiter, and bilateral parotid and submandibular glands enlargement. CT imaging revealed a diffuse goiter which imposed infraglottic airway narrowing and a suspicious left inferior thyroid nodule. Biopsy revealed atypia of undetermined significance. GSC showed a 50% suspicion of malignancy with a negative BRAF. Follow up imaging revealed increased infiltrative masses within the salivary glands, enlarging thyroid gland, and increased infiltration of the superior component of the right thyroid lobe into the submandibular space. A core needle biopsy revealed a background of acute and chronic inflammation without evidence of malignancy. Sjogren's, sarcoidosis and IgG4 diseases were ruled out. She was then referred to us after a myriad of work up. She had a diffusely enlarged goiter with a crusted area of skin over the thyroid gland and significant bilateral parotid swelling. We repeated a thyroid ultrasound which revealed diffusely enlarged and heterogeneous thyroid glands with pseudo-nodules and 3 suspicious nodules greater than 1cm on right and left lobes. She also had prominent reactive lymphadenopathy. Her submandibular glands were hypoechoic, avascular, markedly enlarged and abnormal appearing. She underwent total thyroidectomy. Pathology revealed proliferation of histiocytes, few islands of lymphoid tissue, and eosinophilic micro abscesses without any identifiable thyroid parenchyma in the entire resected specimens. Immunohistochemistry confirmed the diagnosis of Langerhans Cell Histiocytosis with atypical features. BRAF (V600E/V600K) mutation was again not detected. Flow cytometry showed no evidence of neoplastic myeloid blast population. She was referred to an oncologist for further management of LCH. Discussion – Our patient had a remarkably diffuse goiter with a sonographic appearance mimicking Hashimoto's thyroiditis or Riedel's thyroiditis. Initial thyroid FNA failed to reveal LCH. The diagnosis was reached only after total thyroidectomy. Treatment depends on single or multi-system involvement, presence or absence of risk organ involvement, and presence or absence of BRAF mutation. Conclusion In the cases of infiltrated thyroid diseases, Langerhans cell histiocytosis should be on the differential diagnosis, especially if there is concomitant infiltrative process in other organs. The sonographic finding can mimic Hashimoto's or Riedel's thyroiditis which may lead to misdiagnosis and treatment delay. Presentation: Saturday, June 11, 2022 1:00 p.m. - 3:00 p.m.
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Al-Sharoot, Hassaneen Ali. "Anatomical, Histological and Histochemical Architecture of pancreases in Early Hatched Goose (Anser anser)." Kufa Journal For Veterinary Medical Sciences 7, no. 1 (June 30, 2016): 147–53. http://dx.doi.org/10.36326/kjvs/2016/v7i14282.

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The present work includes anatomical, histological and histochemical study of the pancreas in early hatched Goose (Anser anser). The current study was performed on ten early-hatched goose ageing from 5-10 dayes clinically healthy. The animals were anaesthetized by the ether after which they were carefully dissected and examined. Anatomical study shown pancreas it is a vital lobulated organ, pale pinkish in coular, located on the right side of the abdominal cavity between the descending and ascending duodenal loops closely covered by mesentery pancreatic duodenal ligament and Its consist of dorsal, ventral, third and splenic lobe. Histologically parenchyma of gland consisted of exocrine and endocrine parts was located in the meshwork of reticular fibres. Exocrine part was arranged in form of serous tubuloacinar glands that occupied a larger area of pancreas. The islands of langerhans consisted of various shapes and sizes of alpha, beta cell and mixed islets were not observed in the early hatched goose pancreas.
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Dissertations / Theses on the topic "Islands of Langerhans"

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Ar'Rajab, Aamer. "Islet transplantation in the treatment of diabetes number of islets, functional regulation and metabolic control /." Lund : Dept. of Surgery, Lund University, 1991. http://catalog.hathitrust.org/api/volumes/oclc/38187937.html.

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Axcrona, Ulrika Myrsén. "Expression and regulation of neuropeptide Y (NPY) in the Islets of Langerhans." Lund : Dept. of Physiology and Neuroscience, Section for Neuroendocrine Cellbiology, Lund University, 1997. http://books.google.com/books?id=Ew5rAAAAMAAJ.

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Edwin, Nalini. "Quantitative estimation of islet tissue of pancreas in Australian mammals (comparative histological study) /." Title page, contents and summary only, 1986. http://web4.library.adelaide.edu.au/theses/09PH/09phe269.pdf.

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Kulis, Michael D. "Islet neogenesis associated protein-related protein from gene to folded protein /." Available online, Georgia Institute of Technology, 2006, 2006. http://etd.gatech.edu/theses/available/etd-01112006-195113/.

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Thesis (Ph. D.)--Chemistry and Biochemistry, Georgia Institute of Technology, 2006.
Shuker, Suzanne, Committee Chair ; Doyle, Donald, Committee Member ; Orville, Allen, Committee Member ; Barry, Bridgette, Committee Member ; McCarty, Nael, Committee Member.
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Blais, Debbie Lin Marie. "Becoming an islet cell allotransplant recipient." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq21258.pdf.

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Yan, Mengyong. "Interaction of human papillomavirus-like particles with dendritic cells and Langerhans cells : involvement in uptake, activation and cross-presentation /." St. Lucia, Qld, 2003. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17614.pdf.

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Teague, Warwick J. "Mesenchyme-to-epithelial transition in pancreatic organogenesis." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670115.

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Iovino, Giugetta. "The role of lipid peroxidation in pancreatic islet function and destruction in Type 1 diabetes mellitus." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ37131.pdf.

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Wu, Douglas Ching Gee. "Cellular therapeutic strategies for the treatment of Type 1 Diabetes Mellitus." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670111.

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Serra, Navarro Berta. "Implicació de la senyalització dependent de Gsα en l’establiment de la massa cel·lular β." Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/667060.

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La regulación y potenciación de la proliferación de la célula β pancreática es un objetivo principal en la prevención y/o retardo de la diabetes. Su replicación se regula a través de un proceso dinámico, siendo muy elevada en el estado embrionario hasta el nacimiento y disminuyendo gradualmente durante la etapa posnatal, manteniéndose en niveles muy bajos durante la edad adulta. Las vías de señalización que regulan este descenso son poco conocidas. Este estudio pretende establecer el papel de las vías Gsα-dependientes que actúan a través del segundo mensajero AMPc en la regulación de la masa celular β durante la etapa postnatal temprana. Para esta tesis hemos generado ratones deficientes para Gsα en célula β (β-GsαKO). En primer lugar hemos realizado una caracterización fenotípica de este modelo animal tanto a nivel de la homeostasis de la glucosa como a nivel del compartimento β pancreático, mostrando una clara intolerancia la glucosa junto con una reducción en la masa celular β así como en los niveles de proliferación des de una etapa postnatal temprana. Por otro lado el estudio de los determinantes moleculares responsables de las alteraciones observadas en el establecimiento de la masa celular β, pone en manifiesto un defecto claro en la señalización Gs-AMPc/PKA así como en la señalización de la insulina/igf1. El defecto en la vía de la insulina se observa tanto a nivel de ligando como a nivel de receptor con una alteración en la distribución de las isoformas así como en la propia activación de la vía para estimular la proliferación de la célula β en respuesta a insulina. De hecho, hemos visto una relación sinérgica dependiente entre las dos vías de señalización Gs-AMPc/PKA y insulina en la potenciación de la proliferación en respuesta a diferentes mitogenos. Esta tesis revela la importancia de la señalización mediada por AMPc en el establecimiento de la masa de célula β durante la etapa posnatal temprana. Además, demuestra de forma preliminar la existencia de una conexión entre el AMPc y la señalización intracelular mediada por la insulina en la célula β.
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Books on the topic "Islands of Langerhans"

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service), SpringerLink (Online, ed. The Islets of Langerhans. Dordrecht: Springer Science+Business Media B.V., 2010.

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Schock, Roxanne. Understanding insulinomas. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, Clinical Center, 1990.

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K, Federlin, Bretzel R. G, and Hering B. J, eds. Methods in islet transplantation research: International Workshop, Bad Nauheim, June 1989. Stuttgart: G. Thieme, 1990.

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Ellis, Samols, ed. The Endocrine pancreas. New York: Raven Press, 1991.

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Illani, Atwater, Rojas Eduardo 1936-, and Soria Bernat, eds. Biophysics of the pancreatic [beta]-cell. New York: Plenum Press, 1986.

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Illani, Atwater, Rojas Eduardo, and Soria Bernat, eds. Biophysics of the pancreatic (beta)-cell. New York: Plenum Press, 1987.

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1909-, Volk Bruno W., and Arquilla Edward R, eds. The Diabetic pancreas. 2nd ed. New York: Plenum Medical Book Co., 1985.

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Vladimír, Bartoš. Diabetes mellitus a transplantace pankreatu. Praha: Academia, 1990.

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Parker, James N., and Philip M. Parker. The official patient's sourcebook on islet cell carcinoma. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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Kibenge, Molly J. Twinomwe. The relationship between the hypothalamo-pituitary-adrenal axis and the development and persistence of pancreatic islet defects in obese zucker (fa/fa) rats. Charlottetown: University of Prince Edward Island, 1994.

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Book chapters on the topic "Islands of Langerhans"

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Wolff, Günther. "Paul Langerhans — of Islets and Islands." In Diabetes Its Medical and Cultural History, 336–49. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-48364-6_23.

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Conference papers on the topic "Islands of Langerhans"

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Sunaryo, Hadi, Ni Putu Ermi Hikmawanti, and Hesty Awanis Listyaningrum. "Study in Activity Combination of Physalis angulata and Hibiscus sabdariffa in 70% Ethanol Extract to Decrease Blood Sugar Levels and Histopathology of Pancreas Langerhans Island in Alloxan Induced Diabetic Rats." In 1st Muhammadiyah International Conference on Health and Pharmaceutical Development. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0008240401170122.

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