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1

Bories, Jacques, ed. Cerebral Ischaemia. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70943-2.

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2

Dormandy, John A., and Günter Stock, eds. Critical Leg Ischaemia. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75625-2.

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3

A, Myers Kenneth, Nicolaides Andrew N, and Sumner David S, eds. Lower-limb ischaemia. London: Med-Orion, 1997.

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4

K, Widmer L., and Schering Health Care, eds. Critical limb ischaemia. Burgess Hill: Schering Health Care, 1991.

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5

Anderson, Robert H. The heart: Ischaemia. London: Gower, 1993.

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6

A, Grace P., and Mathie Robert T, eds. Ischaemia reperfusion injury. Oxford: Blackwell Science, 1999.

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7

O, Lowe Gordon D., and Schering Health Care, eds. Critical limb ischaemia. Burgess Hill: Schering Health Care, 1993.

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8

Alain, Branchereau, Fagrell Bengt, and Schering Health Care, eds. Critical limb ischaemia. Burgess Hill: Schering Health Care, 1992.

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9

S, Marber Michael, Yellon Derek M, and University College, London. Department of Molecular Pathology., eds. Ischaemia: Preconditioning and adaptation. Oxford: BIOS Scientific Publishers, 1996.

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10

Smith, Thomas C. G., ed. Ischaemia in Head Injury. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80172-3.

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11

Jacques, Bories, and Aymard A, eds. Cerebral ischaemia: A neuroradiological study. Berlin: Springer-Verlag, 1985.

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12

Khaira, Harmeet Singh. Ischaemia-reperfusion in intermittent claudication. Birmingham: University of Birmingham, 1995.

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13

Anderson, Stephen Ian. Studies in skeletal muscle ischaemia. Birmingham: University of Birmingham, 1999.

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14

R, Caplan Louis, ed. Cerebrovascular ischaemia investigation and management. London: Med-Orion, 1996.

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15

Workshop Conference Hoechst-Werk Albert (1987 Frankfurt, Germany). Muscle ischaemia: Functional and metabolic aspects. Edited by Hudlicka O and Okyayuz-Baklouti I. München: C. Wolf, 1988.

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16

A, Chamberlain D., Julian Desmond G, and Sleight Peter, eds. The Management of acute myocardial ischaemia. London: Chapman and Hall Medical, 1990.

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17

Shiner, Ruth Ann. Chronic muscle ischaemia: Local and remote effects. Birmingham: University ofBirmingham, 1994.

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18

A, Dormandy J., and Stock G, eds. Critical leg ischaemia: Its pathophysiology and management. Berlin: Springer-Verlag, 1990.

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19

Buttrum, Stephen Michael. The white blood cell response in ischaemia. Birmingham: University of Birmingham, 1993.

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20

Loddick, Sarah Ann. The role of interleukin-1 in cerebral ischaemia. Manchester: University of Manchester, 1996.

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21

Allen, James. An investigation of myocardial ischaemia using body surface map patterns. [s.l: The Author], 1995.

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22

Wilson, Ian Clark. The role of leucocytes in neonatal myocardial ischaemia-reperfusion injury. Birmingham: University of Birmingham, 1994.

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23

Nelson, Rachael M. Cellular effects of cerebral ischaemia in vitro: Cerebroprotective actions of GABAmimetic agents. Leicester: De Montfort University, 2002.

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24

McGuigan, Kathleen. The effect of walking exercise on patients following lower limb bypass surgery for ischaemia. [S.l: The Author], 2003.

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25

Johns, Lisa. Amino acid transmitter release and the use of neuroprotective agents in models of brain ischaemia. Birmingham: University of Birmingham, 1999.

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26

Johnston, Andrew. An investigation of free radical involvement in cell death in a cell culture model of ischaemia. [s.l.]: typescript, 1999.

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27

Herman, Stanley Leon. Age differences in the susceptibility of the normal rabbit myocardium to injury following ischaemia and reperfusion. Ottawa: National Library of Canada, 1992.

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28

Smith, T. C. G. 1939-, ed. Ischaemia in head injury: 10th European Congress of Neurosurgery, Berlin 1995 ; proceedings of a special symposium. Berlin: Springer, 1996.

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29

Humphreys, William Vowell. An audit of the results of reconstruction and/or amputation in patients with critical lower limb ischaemia. Manchester: University of Manchester, 1994.

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30

Backx, Karianne. An investigation into the effects of cooling, ischaemia and electrical stimulation on physiological tremor measured isotonically and isometrically. Birmingham: University of Birmingham, 1998.

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31

Wilkinson, Caroline Mary. The changes in skin colour due to ischaemia of the lower extremity and its link to surface temperature. Manchester: University of Manchester, 1994.

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32

Ratliff, David. Limb ischaemia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0067.

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Acute limb ischaemia is defined as a decrease in the arterial blood flow, over a period of minutes to days, which threatens the viability of the limb. Presentation is within 2 weeks of the onset of symptoms. Chronic limb ischaemia (presentation >2 weeks after the onset of symptoms) can be divided into critical ischaemia—a potential threat to limb viability, with ischaemic rest pain, ischaemic ulcers, or gangrene—and non-critical ischaemia, which may be symptomatic (typically with claudication) or asymptomatic. This chapter discusses limb ischaemia, including differential diagnosis, context, approach to diagnosis, specific clues to the diagnosis, key diagnostic tests, treatment and therapy, prognosis, and how to handle uncertainty in the diagnosis of the symptom.
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33

Kramer, G., W. Hacke, H. J. Gelmers, and M. Hennerici. Cerebral Ischaemia. Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 1991.

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34

(Editor), K. Thygesen, and J. Kjekshus (Editor), eds. Myocardial Ischaemia. Wiley-Blackwell, 1991.

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35

Sutton. Imaging Ischaemia. Science Press Inc., 2000.

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36

Sutton, Martin St John. Imaging Ischaemia. Science Press, 2000.

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37

Myers, K. A., A. N. Nicolaides, and D. S. Sumner. Lower Limb Ischaemia. Med-Orion Publishing Co Ltd, 1997.

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38

Krieglstein, Josef. Pharmacology of Cerebral Ischaemia. Elsevier, 1986.

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39

Alarcón, Gonzalo, Marian Lazaro, and Antonio Valentín. Migraine, stroke, and cerebral ischaemia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0033.

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This chapter reviews the electroencephalographic changes associated with migraine, stroke and cerebral ischaemia, and their interpretation to aid in their differential diagnosis. The incidence of stroke and cerebral ischaemia is increasing with population aging. They are some of the most common problems faced in emergency medicine, and their diagnosis can be puzzling. This chapter describes and illustrates the patterns seen in such conditions, such as slowing, frontal intermittent delta activity (FIRDA), periodic lateralized epileptiform discharge (PLED), generalized periodic lateralized epileptiform discharge (GPED), and bilateral periodic lateralized epileptiform discharge (BiPED). The chapter reviews their prognostic value in critical care medicine and their differential diagnosis with status epilepticus.
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40

Nicolaides, Andrew N., Wesley S. Moore, Louis R. Caplan, and Edward G. Shifrin. Cerebrovascular Ischaemia: Investigation and Management. Med-Orion Publishing Co Ltd, 1996.

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41

Cerebral Ischaemia: A Neuroradiological Study. Springer, 2011.

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42

Haunso. Coronary Microcirculation During Ischaemia & Reperfusion. Blackwell Science Inc, 1997.

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43

Bories, Jacques. Cerebral Ischaemia: A Neuroradiological Study. Springer London, Limited, 2012.

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44

Jenner, Peter, Richard C. Green, Ronald J. Bradley, Robert Adron Harris, and Alan J. Cross. Neuroprotective Agents and Cerebral Ischaemia. Elsevier Science & Technology Books, 1996.

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45

Haemorrhage, Ischaemia and the Perinatal Brain. Cambridge University Press, 1993.

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46

Fink, Mitchell P. Ischaemia-reperfusion injury in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0308.

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Ischaemia/reperfusion (I/R) injury contributes to the pathogenesis of many common clinical conditions, including stroke, myocardial damage after percutaneous intervention for acute coronary artery occlusion, primary graft dysfunction after solid organ transplantation. The mechanisms that are responsible for I/R injury remain incompletely understood, but damage caused by reactive oxygen species (ROS) and reactive nitrogen species clearly is important. A number of therapeutic approaches, such as administration of ROS scavengers, are effective in animal models of I/R injury, but for the most part, translation of these findings into strategies that can clearly benefit patients has yet to be achieved.
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47

Ischaemia: Preconditioning and Adaptation (Ucl Molecular Pathology). Informa Healthcare, 1996.

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48

Hulme, Sarah. Silent myocardial ischaemia: Impact on clinical practice. 1999.

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49

Dormandy, John A., and Günter Stock. Critical Leg Ischaemia: Its Pathophysiology and Management. Springer London, Limited, 2012.

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50

Critical Leg Ischaemia: Its Pathophysiology and Management. Springer-Verlag Berlin and Heidelberg GmbH & Co. KG, 1990.

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