Relevant bibliographies by topics / Iron in the body

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Iron in the body'

Author: Grafiati
Published: 4 June 2021
Last updated: 30 July 2024

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Journal articles on the topic "Iron in the body"

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Wang, Chia-Yu, and Jodie L. Babitt. "Liver iron sensing and body iron homeostasis." Blood 133, no. 1 (January 3, 2019): 18–29. http://dx.doi.org/10.1182/blood-2018-06-815894.

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Abstract The liver orchestrates systemic iron balance by producing and secreting hepcidin. Known as the iron hormone, hepcidin induces degradation of the iron exporter ferroportin to control iron entry into the bloodstream from dietary sources, iron recycling macrophages, and body stores. Under physiologic conditions, hepcidin production is reduced by iron deficiency and erythropoietic drive to increase the iron supply when needed to support red blood cell production and other essential functions. Conversely, hepcidin production is induced by iron loading and inflammation to prevent the toxicity of iron excess and limit its availability to pathogens. The inability to appropriately regulate hepcidin production in response to these physiologic cues underlies genetic disorders of iron overload and deficiency, including hereditary hemochromatosis and iron-refractory iron deficiency anemia. Moreover, excess hepcidin suppression in the setting of ineffective erythropoiesis contributes to iron-loading anemias such as β-thalassemia, whereas excess hepcidin induction contributes to iron-restricted erythropoiesis and anemia in chronic inflammatory diseases. These diseases have provided key insights into understanding the mechanisms by which the liver senses plasma and tissue iron levels, the iron demand of erythrocyte precursors, and the presence of potential pathogens and, importantly, how these various signals are integrated to appropriately regulate hepcidin production. This review will focus on recent insights into how the liver senses body iron levels and coordinates this with other signals to regulate hepcidin production and systemic iron homeostasis.
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Cook, James D. "Defining optimal body iron." Proceedings of the Nutrition Society 58, no. 2 (May 1999): 489–95. http://dx.doi.org/10.1017/s0029665199000634.

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The major liabilities of Fe lack include defects in psychomotor development in infants, impaired educational performance in schoolchildren, increased perinatal morbidity, and impaired work capacity. Few if any of the relevant investigations have demonstrated these abnormalities in the absence of anaemia. Consequently, adequate Fe nutrition can be defined as a normal haemoglobin concentration. On the other hand, optimal Fe nutrition should be regarded as sufficient body Fe to avoid any limitation in tissue Fe supply, termed Fe-deficient erythropoiesis. A variety of laboratory measurements have been used to identify this milder form of Fe deficiency, including serum ferritin, transferrin saturation, erythrocyte protoporphyrin, mean corpuscular volume, and more recently the concentration of the soluble fragment of transferrin receptor in serum. Recent studies indicate that the serum transferrin receptor is the preferred measurement, because enhanced synthesis of the transferrin receptor represent the initial cellular response to a declining Fe supply. Moreover, unlike other methods, it is not affected by chronic inflammation or infection which are often confused with Fe deficiency. In an otherwise normal healthy population the transferrin receptor: ferritin value provides a useful quantitative index of body Fe over a wide spectrum of Fe status, ranging from Fe repletion to Fe-deficiency anaemia. It is concluded that optimal Fe nutrition is best defined as a normal haemoglobin, serum ferritin and transferrin receptor concentration.
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Cook, James D., Carol H. Flowers, and Barry S. Skikne. "The quantitative assessment of body iron." Blood 101, no. 9 (May 1, 2003): 3359–63. http://dx.doi.org/10.1182/blood-2002-10-3071.

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Current initiatives to reduce the high prevalence of nutritional iron deficiency have highlighted the need for reliable epidemiologic methods to assess iron status. The present report describes a method for estimating body iron based on the ratio of the serum transferrin receptor to serum ferritin. Analysis showed a single normal distribution of body iron stores in US men aged 20 to 65 years (mean ± 1 SD, 9.82 ± 2.82 mg/kg). A single normal distribution was also observed in pregnant Jamaican women (mean ± 1 SD, 0.09 ± 4.48 mg/kg). Distribution analysis in US women aged 20 to 45 years indicated 2 populations; 93% of women had body iron stores averaging 5.5 ± 3.35 mg/kg (mean ± 1 SD), whereas the remaining 7% of women had a mean tissue iron deficit of 3.87 ± 3.23 mg/kg. Calculations of body iron in trials of iron supplementation in Jamaica and iron fortification in Vietnam demonstrated that the method can be used to calculate absorption of the added iron. Quantitative estimates of body iron greatly enhance the evaluation of iron status and the sensitivity of iron intervention trials in populations in which inflammation is uncommon or has been excluded by laboratory screening. The method is useful clinically for monitoring iron status in those who are highly susceptible to iron deficiency.
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Kohgo, Yutaka, Katsuya Ikuta, Takaaki Ohtake, Yoshihiro Torimoto, and Junji Kato. "Body iron metabolism and pathophysiology of iron overload." International Journal of Hematology 88, no. 1 (July 2008): 7–15. http://dx.doi.org/10.1007/s12185-008-0120-5.

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Debnam, Ted, and Kaila Srai. "Hepcidin, body iron and infection." Physiology News, Winter 2005 (January 1, 2006): 38–39. http://dx.doi.org/10.36866/pn.61.38.

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Cook, J. D., and B. S. Skikne. "Intestinal regulation of body iron." Blood Reviews 1, no. 4 (December 1987): 267–72. http://dx.doi.org/10.1016/0268-960x(87)90028-2.

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Sikström, C., L. Beckman, G. Hallmans, and K. Asplund. "Transferrin Types, Iron-Binding Capacity and Body Iron Stores." Human Heredity 43, no. 6 (1993): 337–41. http://dx.doi.org/10.1159/000154156.

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汪, 昕. "Brain Iron Deposition, Body Iron Overload and Cognitive Impairment." Journal of Physiology Studies 01, no. 03 (2013): 16–19. http://dx.doi.org/10.12677/jps.2013.13004.

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Ganz, Tomas, and Elizabeta Nemeth. "Iron imports. IV. Hepcidin and regulation of body iron metabolism." American Journal of Physiology-Gastrointestinal and Liver Physiology 290, no. 2 (February 2006): G199—G203. http://dx.doi.org/10.1152/ajpgi.00412.2005.

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Hepcidin, a small peptide synthesized in the liver, controls extracellular iron by regulating its intestinal absorption, placental transport, recycling by macrophages, and release from stores. Hepcidin inhibits the cellular efflux of iron by binding to and inducing the degradation of ferroportin, the sole iron exporter in iron-transporting cells. In turn, hepcidin synthesis is increased by iron loading and decreased by anemia and hypoxia. Hepcidin is markedly induced during inflammation, trapping iron in macrophages, decreasing plasma iron concentrations, and contributing to the anemia of inflammation. Hepcidin deficiency due to the dysregulation of its synthesis causes most known forms of hemochromatosis.
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Moghadam, Ali Malekshahi, Mahboobeh Mehrabani Natanzi, Mahmoud Djalali, Ahmad Saedisomeolia, Mohammad Hassan Javanbakht, Ali Akbar Saboor-Yaraghi, and Mahnaz Zareei. "Relationship between blood donors' iron status and their age, body mass index and donation frequency." Sao Paulo Medical Journal 131, no. 6 (2013): 377–83. http://dx.doi.org/10.1590/1516-3180.2013.1316554.

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CONTEXT AND OBJECTIVE: Regular blood donation may decrease body iron storage and lead to anemia. The aim here was to evaluate the iron status of Iranian male blood donors and the impact of age, body mass index (BMI) and donation frequency over one year, on iron status indices. DESIGN AND SETTING: Cross-sectional, descriptive and analytical study at Tehran Blood Transfusion Center, Tehran, Iran. METHODS: Between July and September 2011, 117 male blood donors were selected and divided into four groups according to their frequency of blood donation. Thirty male non-donors were also recruited as controls after adjusting for age, weight, height, smoking habits and monthly income. Iron status indices and some criteria such as general health and dietary measurements were determined among all subjects. RESULTS: The values of the iron-related parameters were significantly lower among donors than among non-donors. Only total iron binding capacity (TIBC) was found to be significantly higher among different donor groups than in the controls. A significant positive correlation was observed between age and serum ferritin (SF) only among the donors who had donated once within the preceding year. The iron status indices did not show any significant relationship with BMI among donors or non-donors. CONCLUSION: A donation frequency of more than twice a year had a significant influence on iron-related parameters. Therefore, without annual measurement of these parameters, further phlebotomies may lead to iron deficiency and donor rejection in the future.
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Dissertations / Theses on the topic "Iron in the body"

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Requejo-Roque, Katherinne Isabel. "Iron oxide nanoparticles stable in the human body?" Revista de Química, 2013. http://repositorio.pucp.edu.pe/index/handle/123456789/101184.

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Actualmente, es posible obtener nanopartículas de óxido de hierro solubles en agua y establesen entornos biológicos por medio de la descomposición térmica a altas temperaturas y el intercambiode ligandos. Este método permite un control óptimo de la distribución de tamañopara obtener nanopartículas monodispersas y con superficie apta para funcionalizar, lo cuales fundamental en aplicaciones biológicas.
Currently, it is possible to obtain iron oxide nanoparticles soluble in water with high stability in biological environments through thermal decomposition at high temperatures and ligand exchange. This method of synthesis allows good control of size distribution in order to obtain monodispersed nanoparticles with surfaces suitable for functionalization which is necessary for biological applications.
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Shiono, Yuhta, Hisao Hayashi, Shinnya Wakusawa, Fujiko Sanae, Toshikuni Takikawa, Motoyoshi Yano, Kenntaro Yoshioka, and Hiros Saito. "Body iron stores and Iron restoration rate in Japanese patients with chronic Hepatitis C as measured during therapeutic Iron removal revealed neither Increased body iron stores nor effects of C282y and H63d mutations on iron indices." Nagoya University School of Medicine, 2001. http://hdl.handle.net/2237/5367.

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Hotz, Karin. "Understanding the message of iron isotopes in the human body /." [S.l.] : [s.n.], 2009. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=18376.

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Prasad, Mona Kumar. "The effects of exercise and dietary iron on iron status in 13 months old female rats." Thesis, Virginia Tech, 1988. http://hdl.handle.net/10919/43274.

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The purpose of this study was to investigate the effects of exercise and dietary iron on various components of iron metabolism in 13 month old female rats. Fifty-six 13 month old female Sprague-Dawley rats were randomly assigned to one of five groups of treatment: baseline (BL); 40ppm iron, exercise (40 E); 9ppm iron, exercise (9 E); 40ppm iron, non- exercise (40 NE); 9ppm iron, non-exercise (9 NE). The exercise protocol involved swimming the exercise groups for a period of six weeks, starting with 10 minutes/day until the rats were swimming for 1 hour/day, 5 days/ week, at the sixth week. Results indicated no significant differences in food intake and body weight among the groups. Hematocrit values were similar among groups and did not show significant effects of diet, activity or interaction. Hemoglobin values demonstrated significant effects of activity with values being elevated in the exercise groups (p ≤0.05). No significant effects of diet and interaction on hemoglobin concentrations were observed. Serum iron levels were significantly affected by dietary intake of iron, with levels being lower in the groups consuming the moderately-deficient diet (p≤0.05). Serum iron levels were not significantly affected by activity or interaction of diet and activity. TIBC levels did not demonstrate significant effects of diet, activity or interaction (p<0.05). Tissue weights of liver, spleen, heart, soleus and gastrocnemius muscles were similar among groups. Iron concentrations in the liver and spleen showed significant effects of diet, activity and interaction (p≤0.05). Iron levels were lower in the groups consuming moderately-deficient intakes of dietary iron and were also lower in the exercised animals. Concentrations were significantly lower in the 9 E group than in the 40 E group. Iron concentrations in the gastrocnemius muscle were significantly affected by diet, activity and interaction of diet and activity, and were significantly lower in the exercised animals as compared to their sedentary counterparts. Soleus iron concentrations were similar among groups. The results suggest that there is an iron cost associated with exercise as evidenced by elevated hemoglobin levels and depleted iron stores in the liver, spleen and gastrocnemius muscle. It appears that the body undergoes. physiological adaptions in response to the stress of exercise and therefore prevents anemia by maintaining the iron stores at compromised levels.
Master of Science
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Sinclair, Lisa M. "Effect of iron supplementation on endurance performance in iron deficient trained males and females /." free to MU campus, to others for purchase, 2004. http://wwwlib.umi.com/cr/mo/fullcit?p1422965.

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Kind, Clive Nicholas. "Biochemical aspects of the fate of imferon (iron-dextran) in the body." Thesis, De Montfort University, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.280510.

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Whitnall, Megan. "Iron metabolism, chelation and disease." Thesis, The University of Sydney, 2011. https://hdl.handle.net/2123/28914.

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Organisms depend on iron to survive. This fact is underscored by the critical requirement for iron during DNA synthesis and as a cofactor in proteins involved in respiration and oxygen transport. However, when present in excess of cellular requirements, iron can be toxic, due to its ability to generate reactive oxygen species and induce oxidative stress. The physiological significance of iron renders it a target for the development of iron chelators as therapeutic agents and highlights the potential problems that can occur when iron regulatory pathways are disturbed in disease. The rapid rate of neoplastic cell replication and the involvement of iron in cell cycle progression and DNA synthesis, highlight the potential for using iron chelators for cancer treatment. Chapter 3 of this thesis demonstrates the broad-spectrum in vitro and in vivo anti-tumour activity of the novel iron chelator, di-2-pyridylketone-4,4,-dimethyl-3-thiosemicarbazone (Dp44mT) (Whitnall et.al., Proc Natl Acad Sci USA 2006; 103:14901-6). In vitro results illustrate the potency of Dp44mT over the clinically used chemotherapeutic agent, doxorubicin, and the ability of Dp44mT to overcome multi­drug resistance. The unique ability of Dp44mT to up-regulate the tumour growth and metastasis suppressor, Ndrgl in in vivo experiments, may account for this ligands selective anti-tumour activity (Whitnall et al., 2006). Collectively, these studies demonstrate that iron chelators such as Dp44mT, may be valuable anti-cancer compounds, particularly considering the emergence of multi-drug resistance in tumours. There is no effective treatment for the cardiomyopathy of the most common autosomal recessive ataxia, Friedreich 's ataxia (FA). The identification of potentially toxic mitochondrial iron deposits in FA suggests iron plays a role in its pathogenesis and merits the use of iron chelation therapy for the treatment of FA. Studies in Chapters 4 and 5 used the muscle creatine kinase (MCK) frataxin mutant mouse model that reproduces the classical traits associated with cardiomyopathy in FA, to study the molecular alterations which underlie the pathogenesis of this disease and assess the use of iron chelation therapy (Whitnall et.al., Proc Natl Acad Sci USA 2008; 105:9757-62). Studies specifically in Chapter 4 show that the increased mitochondrial iron in the myocardium of mutants was due to marked transferrin-iron uptake, which was the result of enhanced transferrin receptor 1 (TfRl) expression. 1n contrast to the mitochondrion, cytosolic ferritin expression and the proportion of cytosolic iron were decreased in mutant mice, indicating the cytosol was iron deficient. These studies demonstrate that loss of frataxin alters cardiac iron metabolism due to pronounced changes in iron trafficking away from the cytosol to the mitochondrion. Further work in Chapter 4 showed that the mitochondrial-permeable ligand, pyridoxal isonicotinoyl hydrazone, in combination with the hydrophilic chelator, desferrioxamine, prevented cardiac iron loading and limited cardiac hypertrophy in mutants, but did not lead to overt cardiac iron depletion or toxicity (Whitnall et al., 2008). However, iron chelation did not prevent decreased succinate dehydrogenase expression in the mutants nor loss of cardiac function, indicating that frataxin function must also be replaced in addition to removing the excess mitochondrial iron. In summary, for the first time, studies in this thesis demonstrate that frataxin deficiency markedly alters cellular iron trafficking and that iron chelation limits myocardial hypertrophy in the MCK mutant model of FA. To address the cytosolic iron deficiency in the cardiomyocytes of mutant mice, in Chapter 5, mice were fed a high iron diet aimed at reconstituting the iron deprived cytosolic compartment. From these studies, a significant decrease in cardiac hypertrophy was observed in high iron diet fed mice. Interestingly, while wild-type (WT) mice responded to the high iron diet by decreasing cardiac TfRl expression, no such compensation was observed in high-compared to normal-iron iron diet fed mutants. Similarly, activity of iron regulatory protein 2 (i.e., IRP2 RNA-binding activity) was not decreased in high iron diet fed mutants. These findings demonstrate the mutant heart does not respond to increased iron levels as does the WT animal. An intriguing and important outcome of dietary iron loading investigations, was the marked increase in iron concentration observed in the liver, spleen and kidney of mutant mice that were fed a normal iron diet. The MCK mutant mouse experiences deletion of frataxin in the heart only, and hence, the increase in iron levels observed in frataxin ­intact tissues such as the liver, indicated that the heart is able to influence systemic iron metabolism. Supporting this, changes were observed in iron-metabolism proteins such as hemojuvelin and TfRl not only in the heart, but in the liver. Collectively, these results indicate that frataxin knockout in the heart and the alterations in iron metabolism which lead to cytosolic iron deficiency in the heart, activate a systemic signalling mechanism, most likely to communicate its need for iron within the cytosolic compartment. In the final section of Chapter 5, transmission electron microscopy and magnetic susceptibility measurements were used to assess the molecular composition of accumulated iron in the MCK mutants. These studies showed that the iron accumulating in the mutant heart is not present within ferritin, but in well crystallised anti­ferromagnetic mineral aggregates. In conclusion, the investigations described within this thesis demonstrate the potential for iron chelators to be used for the treatment of cancer and FA. Moreover, they also begin to elucidate the marked alterations in the pathways of iron metabolism that occur on both a cellular and systemic level in FA. ln terms of contributing to our understanding of basic physiological iron homeostasis, they also identify that cardiac iron status is able to markedly influence systemic iron metabolism.
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Nikolaiczuk, Marcia Jane. "Relative availability of iron to rats from beef, soy protein and a beef-soy protein mixture as determined by iron repletion assay." Thesis, University of British Columbia, 1985. http://hdl.handle.net/2429/24874.

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Male weanling Wistar rats were fed a low-iron basal diet for 3 weeks. The iron depleted rats were then divided into 9 groups according to a randomized block design based on body weight. During the repletion period of 2 weeks, one group was fed the low-iron basal diet. The other eight groups received either the basal diet to which was added 5, 10, 15, 20 or 25 mg iron per kg diet as ferrous sulfate or test source diets formulated to provide a total of 15 mg iron per kg diet from either freeze-dried ground beef, textured defatted soy flour product or a 2.3:1 (w/w) mixture of beef and soy product. All diets were isocaloric and isonitrogenous. The relative biological value (RBV) of iron in the test source diet was calculated as the ratio of the amounts of iron from the reference source (ferrous sulfate) and the test source diet required to give the same response in hemoglobin or hematocrit. The RBVs ± 95% confidence limits, calculated on the basis of final hemoglobin levels and hematocrit values, were respectively: freeze-dried ground beef, 56 ± 7 % and 62 ± 7 %; fortified textured defatted soy flour product, 81 ± 10 % and 79 ± 10 %; 2.3:1 (w/w) mixture of freeze-dried ground beef and soy flour product, 65 ± 6 % and 68 ± 6 %. The RBVs obtained for the iron in beef and for that in the soy flour product suggest that the anemic rat might not be a suitable model for normal man when screening such foods for their available iron. In normal man, the absorption of the iron in beef is comparable to that of inorganic reference iron, while that in textured soy flour is about one third.
Land and Food Systems, Faculty of
Graduate
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Mascarenhas, Monica Sousa Dias. "Role of Hfe and hepcidin in the homeostasis of body iron levels." Thesis, University College London (University of London), 2007. http://discovery.ucl.ac.uk/1444809/.

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Recent studies have suggested that hepatic iron stores and the response to anaemia and inflammation are dependent on the mouse strains used and their Hfe status. Hepcidin and iron transporters such as DMT1 and Iregl are regulator molecules important in the pathology and physiology of Iron metabolism. The aim of these studies was to produce Hfe KO mice on a strain known to have higher basal hepatic iron levels and study iron homeostasis in relation to variable dietary iron and inflammation in these mice. The regulation of hepcidin was investigated in Huh7 cells exposed to various stimuli such as iron, inflammation and hypoxia. In addition, Huh7 cells were exposed to conditioned medium obtained from HH1 patient's macrophages. Results obtained from these studies are described and discussed in this thesis. In brief, it was found that the disruption of the Hfe gene causes a preferential accumulation of iron in the caudate lobe of the mouse liver. Contrary to previous findings, iron loaded diet does not have an effect on hepcidin expression in wt SWR mice while in Hfe KO SWR mice it significantly upregulates hepcidin expression. Furthermore, the upregulation of hepcidin by turpentine oil-induced inflammation in SWR mice is Hfe-dependent. Hepcidin expression is down regulated by hypoxia and anaemia and conditioned medium from patients with HH1 macrophages have no effect on hepcidin expression levels. In conclusion, the results show that SWR Hfe KO mice is not an accurate model for the study of HH1, the studies described here, however, contribute to the understanding of how hepatocytes respond to iron status, hypoxia and inflammation and the relevance of the Hfe gene in the regulation of hepcidin expression levels as well as hepatic DMT1 and Iregl expression levels.
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Snape, Susan Dawn. "Studies on the mechanism of intestinal iron absorption with special reference to its intracellular transport." Thesis, Open University, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254664.

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Books on the topic "Iron in the body"

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M, Loehr Thomas, ed. Iron carriers and iron proteins. New York: VCH, 1989.

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Sardi, Bill. The iron time bomb: How iron adversely affects your health. San Dimas, CA: B. Sardi, 2003.

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Moon, Jym. Iron: The most toxic metal. Chico, Calif: George Ohsawa Macrobiotic Foundation, 2008.

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Kim, Ashida. Iron body ninja: The secrets of superior strength. Secaucus, N.J: Carol Pub. Group, 1997.

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1957-, Lauffer Randall Byron, ed. Iron and human disease. Boca Raton: CRC Press, 1992.

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Crawford, Roberta. The iron elephant: What you should know about the danger of excess body iron. Glyndon, Md. (P.O. Box 296, Glyndon, MD 21071-0296): Vida Publishing, 1990.

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Foundation, British Nutrition, ed. Iron: Nutritional and physiological significance : the report of the British Nutrition Foundation's Task Force. London: Published by Chapman & Hall for the British Nutrition Foundation, 1995.

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Anderson, Gregory J., and Gordon McLaren. Iron physiology and pathophysiology in humans. New York: Humana, 2009.

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International Conference on Health Promotion (6th 2001 Atlanta, Ga.). Forging effective strategies to combat iron deficiency: Proceedings of the Sixth Biennial International Conference on Health Promotion held May 7-9, 2001 in Atlanta, GA. Edited by Martorell Reynaldo 1947- and Trowbridge Frederick. Bethesda, MD: American Society for Nutritional Sciences, 2002.

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Kim, Pŏm-yŏng. Maŭm iron: In'gan ŭi maŭm kwa simni ka chŏgyong hanŭn wŏlli. Sŏul-si: Chisik kwa Kamsŏng#, 2017.

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Book chapters on the topic "Iron in the body"

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Worwood, Mark. "Estimation of Body Iron Stores." In Iron Physiology and Pathophysiology in Humans, 499–528. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60327-485-2_25.

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Gupta, Anil. "Iron Metabolism in Human Body." In Nutritional Anemia in Preschool Children, 29–46. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5178-4_4.

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Schenck, J. F., O. M. Mueller, S. P. Souza, and C. L. Dumoulin. "Magnetic Resonance Imaging of Brain Iron Using A4 Tesla Whole-Body Scanner." In Iron Biominerals, 373–85. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3810-3_27.

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Ram, Radha, and Matthew B. Goren. "Iron, Corneal Intraocular Foreign Body of." In Encyclopedia of Ophthalmology, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-35951-4_844-1.

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Garcia-Zalisnak, Debora, and Mark Fernandez. "Rust Ring, Iron Foreign Body Causing." In Encyclopedia of Ophthalmology, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-35951-4_942-2.

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Ram, Radha, and Matthew B. Goren. "Iron, Corneal Intraocular Foreign Body of." In Encyclopedia of Ophthalmology, 974–76. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_844.

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Garcia-Zalisnak, Debora, and Mark M. Fernandez. "Rust Ring, Iron Foreign Body Causing." In Encyclopedia of Ophthalmology, 1567–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_942.

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Gupta, Anil. "Laboratory Estimation of Iron in Body." In Nutritional Anemia in Preschool Children, 119–29. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5178-4_6.

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Gupta, Anil. "Physiological Contemplation of Iron in the Body." In Nutritional Anemia in Preschool Children, 11–27. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5178-4_3.

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Danielsson, Ing-Marie Back. "(UN)Masking Gender — Gold Foil (DIS)Embodiments in Late Iron Age Scandinavia." In Thinking through the Body, 179–99. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0693-5_10.

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Conference papers on the topic "Iron in the body"

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Ogawa, Jun, Toru Kato, Eiji Nakade, Hiroshi Takashima, Tomohiro Matsumoto, and Kazuhiro Sato. "Development of Alloy Cast Iron for Press Die." In International Body Engineering Conference & Exposition. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 1999. http://dx.doi.org/10.4271/1999-01-3215.

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Bonny, Giovanni, Roberto C. Pasianot, Nicolas Castin, Dmitry Terentyev, and Lorenzo Malerba. "Iron-Copper-Nickel Many-Body Potential Consistent With Thermodynamics." In 17th International Conference on Nuclear Engineering. ASMEDC, 2009. http://dx.doi.org/10.1115/icone17-75619.

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The Fe-Cu-Ni ternary alloy is of interest for nuclear applications because Cu and Ni are considered to have major effects on the embrittlement under irradiation of reactor pressure vessel steels. To improve our understanding on this phenomenon, large scale atomistic simulations in this model alloy are desirable. For this purpose we develop a ternary Fe-Cu-Ni many-body potential consistent with thermodynamics is developed for the first time. The potential was validated using molecular static and atomistic kinetic Monte Carlo simulations and a qualitative agreement with experiments was established. In particular, Cu precipitates were found to be enriched by Ni on the precipitate surface. Also, the effects diluting the Fe-Cu alloy by Ni on mean precipitate size and density showed similar trends as observed in experiments; i.e. no effect of Ni on the mean precipitate size and an increase in the maximum precipitate density due to the addition of Ni. In absolute terms, agreement with experiment is poor due to the limited box size used in the simulations, as correspondingly discussed.
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Ahmed, Umbreen, Trevor G. Redgrave, and Phillip S. Oates. "Body Iron Stores Increase Hepatic and Serum Lipids in Rat Nutritional Model." In Annual International Conference on Advanced Research: Physiology. Global Science & Technology Forum (GSTF), 2014. http://dx.doi.org/10.5176/2382-607x_arp14.17.

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Warnke, E. P., W. Steinwarz, W. Sowa, and R. Hu¨ggenberg. "Ductile Cast Iron for Transportation Cask Bodies." In ASME 2003 9th International Conference on Radioactive Waste Management and Environmental Remediation. ASMEDC, 2003. http://dx.doi.org/10.1115/icem2003-4528.

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Casks for transportation and storage are necessary for the handling of spent fuel elements and other radioactive waste. These casks must provide sufficient shielding against nuclear radiation, safe confinement even under severe hypothetical mechanical and thermal accident conditions and assurance of the subcriticality of the content under these conditions. Since more than 20 years ductile cast iron is established as a well qualified and proven material for the fabrication of the cask body of the CASTOR and MOSAIK type casks.
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Stoch, A., and H. Van Den Berg. "Near-Mine Exploration for Iron Ore at Malmberget Using Body-Wave Seismic Interferometry." In NSG2021 27th European Meeting of Environmental and Engineering Geophysics. European Association of Geoscientists & Engineers, 2021. http://dx.doi.org/10.3997/2214-4609.202120027.

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Anand, Aryavart, Jonas Pape, Martin Wille, Klaus Mezger, and Beda Hofmann. "Mn-Cr Chronological Constraints on the Thermal Evolution of IIAB Iron Meteorite Parent Body." In Goldschmidt2020. Geochemical Society, 2020. http://dx.doi.org/10.46427/gold2020.57.

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Sari, Wilin Julian, and Gagus Ketut Sunnardianto. "Atomistic mechanism of hydrogen embrittlement in a screw dislocation body-centered cubic (BCC) iron." In PROCEEDINGS OF THE 1ST CONFERENCE ON QUANTUM SCIENCES AND TECHNOLOGY (CONQUEST 2022). AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0178306.

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Alekseev, A. A., N. A. Pudovkin, V. V. Salautin, and S. D. Klyukin. "Iron metabolism in the body of white rats under the influence of C60 fullerene solutions." In INTERNATIONAL CONFERENCE “SUSTAINABLE DEVELOPMENT: VETERINARY MEDICINE, AGRICULTURE, ENGINEERING AND ECOLOGY” (VMAEE2022). AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0148360.

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Komann, Steffen, Yusuf Kiyak, Frank Wille, Uwe Zerbst, Mike Weber, and Dietmar Klingbeil. "Assessment of Ductile Cast Iron Fracture Mechanics Analysis Within Licensing of German Transport Packages." In ASME 2012 Pressure Vessels and Piping Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/pvp2012-78213.

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In recent years BAM was involved in several licensing procedures of new package designs for the transport of radioactive material, where the cask body was made of Ductile Cast Iron (DCI). According to IAEA regulations type B(U) packages must withstand the defined accident conditions of transport. For the cask material DCI, it is necessary to determine the brittle fracture behaviour. Due to the complex structure of the cask body and the dynamic loading a fracture mechanical assessment in an analytical way is not always possible. Numerical calculations are necessary to determine the fracture mechanical load in the component. At the first step a numerical analysis has to be done to identify the loading state at the cask body. Secondly an analysis of a detail of the cask body is made considering the boundary conditions of the global model. An artificial flaw is considered in this detailed model to calculate the fracture mechanical loading state. The size of the artificial flaw is characterized by the ultrasonic inspection used for the quality assurance of the package. The applicant developed additional analysis tools for calculation of stress intensity factor and/or J-Integral. The paper describes the authority assessment approach for the DCI fracture mechanics analysis.
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Kobelkova, Irina Vitalievna, Margarita Mikhailovna Korosteleva, Dmitry Borisovich Nikityuk, Ksenia Valerievna Vibornaya, Rajabkadi Magomedovich Rajabkadiev, Maria Sergeevna Kobelkova, Muradin Mudalifovich Semenov, and Enver Saidovich Tokayev. "DEVELOPMENT OF A SPECIALIZED FOOD PRODUCT FOR THE PREVENTION OF IRON DEFICIENCY ANEMIA IN JUNIOR ATHLETES." In NEW TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2021. http://dx.doi.org/10.47501/978-5-6044060-1-4.34.

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The prevalence of iron deficiency in female athletes varies significantly, with 31% having a ferritin concentration below 12 ng/ml or a transferrin saturation of less than 16 ng/ml. The medical and biological substantiation of the composition was carried out and a specialized food product was developed for nutrition of female athletes of children and adolescents (12-17 years), that provides the intake an easily digestible of hem form’s iron and vitamins involved in iron metabolism in the body for at least 15 % and no more than 50% of the recommended daily intake.
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Reports on the topic "Iron in the body"

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Ersoy, Daniel, and Ernest Lever. DTPH56-15-T-00006 Characterization and Fitness for Service of Corroded Cast Iron Pipe. Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), December 2018. http://dx.doi.org/10.55274/r0012163.

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The Cast Iron Fitness-For-Service model described in the main body of this report forms an ideal basis for a simulation tool that can display aggregate system performance in a geospatial database. This simulation tool can provide the operator risk-informed geospatial input into their mitigation programs. The tool can also be extended to temporal consideration of future repair/replace programs.
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Kanner, Joseph, Dennis Miller, Ido Bartov, John Kinsella, and Stella Harel. The Effect of Dietary Iron Level on Lipid Peroxidation of Muscle Food. United States Department of Agriculture, January 1995. http://dx.doi.org/10.32747/1995.7604282.bard.

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Biological oxidations are almost exclusively metal ion-promoted reactions and in ths respect iron, being the most abundant, is the commonly involved. The effect of dietary iron levels on pork, turkey and chick muscle lipid peroxidation and various other related compounds were evaluated. Crossbred feeder pigs were fed to market weight on corn-soy rations containing either 62, 131 or 209 ppm iron. After slaughter, the muscles were dissected, cooked and stored at 4°C. Heavily fortifying swine rations with iron (>200 ppm) increase nn-heme iron (NHI), thiobarbituric acid reactive substances (TBARS), and decrease a-tocopherol in cooked stored pork but did not increase warmed-over aroma (WOA). NHI and TBARS were higher in cooked pork from pigs fed high-iron diets. Liver iron correlated with muscle iron. TBARS were strongly related with WOA. The role of dietary vitamin E and ascorbic acid on Fe-induced in vivo lipid peroxidation in swine was also evaluated. Moderate elevation in iron stores had a marked effect on oxidative stress, especially as indicated by liver TBARS. Supplemental vitamin E, and to a lesser extent vitamin C, protect against this oxidative stress. Unsupplementation of Fe in the regular diet of turkeys did not affect body weight, blood hemoglobin level, or iron pool in the liver or muscle. The reason being that it contained "natural" ~120 mg Fe/kg feed, and this amount is high enough to keep constant the pool of iron in the body, liver or muscle tissues. Only Fe-supplementation with high amounts of Fe (500 ppm) significantly increased turkey blood hemoglobin and total iron in the liver, in 1 out of 3 experiments, but only slightly affects iron pool in the muscles. It seems that the liver accumulates very high concentations of iron and significantly regulates iron concentration in skeletal muscles. For this reason, it was very difficult to decrease muscle stability in turkeys through a diet containing high levels of Fe-supplementation. It was shown that the significant increase in the amount of iron (total and "free") in the muscle by injections with Fe-dextran accelerated its lipid peroxidation rate and decreased its a-tocopherol concentration. The level and metabolism of iron in the muscles affects the intensity of in vivo lipid peroxidation. This process was found to ifluence the turnover and accumulation of a-tocopherol in turkey and chick muscles. Treatments which could significantly decrease the amount and metabolism of iron pool in muscle tissues (or other organs) may affect the rate of lipid peroxidation and the turnover of a-tocopherol. Several defense enzymes were determined and found in the turkey muscle, such as superoxide dismutase, catalase, and glutathione peroxidase. Glutathione peroxidase was more active in muscles with a high trend of lipid peroxidation, lmore so in drumsticks than in breast muscles, or muscles with a low a-tocopherol content. The activity of glutathione peroxidase increased several fold in muscle stored at 4°C. Our work demonstrated that it will be much more practical to increase the stability of muscle tissues in swine, turkeys and chickens during storage and processing by increasing the amount of vitamin E in the diet than by withdrawing iron supplementation.
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Dewey, Kathryn, Lydia Bazzano, Teresa Davis, Sharon Donovan, Elsie Taveras, Ronald Kleinman, Darcy Güngör, et al. Iron from Supplements Consumed During Infancy and Toddlerhood and Growth, Size, and Body Composition: A Systematic Review. U.S. Department of Agriculture, Food and Nutrition Service, Center for Nutrition Policy and Promotion, Nutrition Evidence Systematic Review, July 2020. http://dx.doi.org/10.52570/nesr.dgac2020.sr0303.

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Dewey, Kathryn, Lydia Bazzano, Teresa Davis, Sharon Donovan, Elsie Taveras, Ronald Kleinman, Darcy Güngör, et al. Iron from Supplements Consumed During Infancy and Toddlerhood and Growth, Size, and Body Composition: A Systematic Review. U.S. Department of Agriculture, Food and Nutrition Service, Center for Nutrition Policy and Promotion, Nutrition Evidence Systematic Review, July 2020. http://dx.doi.org/10.52570/nesr.dgac2020.sr0303.

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Wills, Ann Elisabet, Aidan P. Thompson, and Sumathy Raman. An Atomistic Introduction to Orientation Relations Between Phases in the Face-centered Cubic to Body-centered Cubic Phase Transition in Iron and Steel. Office of Scientific and Technical Information (OSTI), January 2017. http://dx.doi.org/10.2172/1505395.

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Corriveau, L., and E. G. Potter. Advancing exploration for iron oxide-copper-gold and affiliated deposits in Canada: context, scientific overview, outcomes, and impacts. Natural Resources Canada/CMSS/Information Management, 2024. http://dx.doi.org/10.4095/332495.

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The Geo-mapping for Energy and Minerals (GEM) and Targeted Geoscience Initiative (TGI) programs conducted extensive collaborative research on mineral systems with iron oxide-copper-gold (IOCG) and affiliated deposits in prospective settings of Canada. Regional alteration mapping as well as geochemical and geophysical modelling undertaken under the GEM program documented the evolution of polymetallic metasomatic systems with iron-oxide and alkali-calcic alteration and led to an increased recognition of the mineral potential of poorly explored areas and historic deposits of the Great Bear magmatic zone in the Northwest Territories, thus providing a solid framework for exploration. Early and barren albitite corridors form across the mineral systems and locally host uranium mineralization associated with telescoping of alteration facies by tectonic activity during the metasomatic growth of the systems. Subsequent to albitization, high-temperature Ca-Fe and Ca-K-Fe alteration form iron oxide-apatite (± rare-earth element) mineralization and IOCG variants rich in cobalt and other critical metals, respectively. Systems that further mature to K-Fe alteration form IOCG mineralization and can evolve to mineralized near-surface phyllic alteration and epithermal caps. Transitional facies also host polymetallic skarn mineralization. Rare-earth element enrichments within iron oxide-apatite zones are strongest where remobilization has occurred, particularly along deformation zones. The TGI projects documented the pertinence for a GEM activity in the Great Bear magmatic zone and subsequently synthesized GEM geoscientific data into a system-scale, ore-deposit model, and outlined criteria for mineral resource assessment. This model, and newly developed field-mapping and lithogeochemical tools were shown to be efficient mineral exploration and regional mapping methods in Canada and were also applied to the archetype IOCG deposit, Olympic Dam, and other deposits in the Olympic Cu-Au metallogenic province of Australia. Case examples also include the Romanet Horst in the Trans-Hudson Orogen (second phase of GEM), the Central Mineral Belt in Labrador (TGI), the Wanapitei Lake district in Ontario (private sector exploration results used by TGI), and the Bondy gneiss complex in Quebec (TGI).
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S. Abdellatif, Omar, Ali Behbehani, and Mauricio Landin. Iran COVID-19 Governmental Response. UN Compliance Research Group, February 2021. http://dx.doi.org/10.52008/iran0501.

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The International Health Regulations (2005) are legally binding on 196 States Parties, Including all WHO Member States. The IHR aims to keep the world informed about public health risks, through committing all signatories to cooperate together in combating any future “illness or medical condition, irrespective of origin or source, that presents or could present significant harm to humans.” Under IHR, states agreed to strengthen their public health capacities and notify the WHO of any such illness in their populations. The WHO would be the centralized body for all countries facing a health threat, with the power to declare a “public health emergency of international concern,” issue recommendations, and work with countries to tackle a crisis. Although, with the sudden and rapid spread of COVID-19 in the world, many countries varied in implementing the WHO guidelines and health recommendations. While some countries followed the WHO guidelines, others imposed travel restrictions against the WHO’s recommendations. Some states refused to share their data with the organization. Others banned the export of medical equipment, even in the face of global shortages. The UN Compliance Research group will focus during the current cycle on analyzing the compliance of the WHO member states to the organizations guidelines during the COVID-19 pandemic.
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Champion, Theresa. Studies of Charmless Two-Body, Quasi-Two-Body and Three-Body B Decays. Office of Scientific and Technical Information (OSTI), November 2000. http://dx.doi.org/10.2172/784763.

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Pritchard, Joy, H. R. Whay, and A. Brown. Body condition. Brooke, 2011. http://dx.doi.org/10.46746/gaw.2020.abi.bcs.

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Brown, Ashleigh. Firing: body areas. Brooke, April 2011. http://dx.doi.org/10.46746/gaw.2020.abi.firbdar.

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