Relevant bibliographies by topics / Iron deficiency diseases in children

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Academic literature on the topic 'Iron deficiency diseases in children'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 March 2023

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Journal articles on the topic "Iron deficiency diseases in children"

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de Vizia, Basilio, Vincenzo Poggi, Rodolfo Conenna, Amedeo Fiorillo, and Luigi Scippa. "Iron Absorption and Iron Deficiency in Infants and Children with Gastrointestinal Diseases." Journal of Pediatric Gastroenterology and Nutrition 14, no. 1 (January 1992): 21–26. http://dx.doi.org/10.1097/00005176-199201000-00005.

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Shaw, Julia G., and Jennifer F. Friedman. "Iron Deficiency Anemia: Focus on Infectious Diseases in Lesser Developed Countries." Anemia 2011 (2011): 1–10. http://dx.doi.org/10.1155/2011/260380.

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Iron deficiency anemia is thought to affect the health of more than one billion people worldwide, with the greatest burden of disease experienced in lesser developed countries, particularly women of reproductive age and children. This greater disease burden is due to both nutritional and infectious etiologies. Individuals in lesser developed countries have diets that are much lower in iron, less access to multivitamins for young children and pregnant women, and increased rates of fertility which increase demands for iron through the life course. Infectious diseases, particularly parasitic diseases, also lead to both extracorporeal iron loss and anemia of inflammation, which decreases bioavailability of iron to host tissues. This paper will address the unique etiologies and consequences of both iron deficiency anemia and the alterations in iron absorption and distribution seen in the context of anemia of inflammation. Implications for diagnosis and treatment in this unique context will also be discussed.
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Mohammad, Faisal, M. Sridhar, Madhusudan Samprathi, and Prakash Vemgal. "Thrombocytopenia in severe iron deficiency anaemia: A report of two cases." Tropical Doctor 51, no. 3 (January 10, 2021): 448–50. http://dx.doi.org/10.1177/0049475520983658.

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Iron deficiency, the commonest cause of anaemia in children, is a global public health problem. Worldwide, almost 50% of children <5 years of age are anaemic. Platelet count in iron deficiency anaemia is mostly normal or high; thrombocytopenia is rare. We describe two children with iron deficiency anaemia and severe thrombocytopenia who recovered with iron supplementation alone.
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Abeuova, B. "PO-0351 Iron-deficiency Anaemia And Dental Diseases Among Children." Archives of Disease in Childhood 99, Suppl 2 (October 2014): A360.2—A360. http://dx.doi.org/10.1136/archdischild-2014-307384.999.

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Najafova, V. A. "CHANGES IN SOME FRACTIONS OF CYTOKINES IN CHILDREN WITH IRON DEFICIENCY ANEMIA." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 21, no. 3 (November 16, 2021): 82–87. http://dx.doi.org/10.31718/2077-1096.21.3.82.

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The aim of the study was to investigate the effect of iron deficiency anaemia on certain cytokine fractions (IL-2, INF-γ, TNF-α) in children aged 6 months to 5 years in Azerbaijan. Methods. The study involved 123 children. According to the hematological and biochemical blood parameters of children aged 6 months to 5 years, 95 children (58 boys, 37 girls) were diagnosed as having anaemia of varying degrees: 32 children had mild iron deficiency anaemia, 37 children had moderate iron deficiency anaemia, and 26 children had severe iron deficiency anaemia. Results. The study demonstrates the level of INF-γ is lower in the general group of children with iron deficiency anaemia compared to the control group (2.5±1.2 pg/ml and 3.9±1.6 pg/ml, respectively). In the general group of children with iron deficiency anaemia, the average correlation coefficient (r=+0.7) between INF-γ with haemoglobin and with serum ferritin (r=+0.6) has been shown. Taking into account the weakening of INF-γ in children with iron deficiency anaemia during the study period, we investigated the annual incidence of acute respiratory viral infections among the general group of children with iron deficiency anaemia and the control group (63.2% and 25%, respectively). There were no significant changes in TNF-α value ​​in children of the general group with iron deficiency anaemia compared to the control group (2.7±1.5 pg / ml, 2.7±1.1 pg / ml, respectively). The IL-2 index in the general group was lower than in the control group (1.6±1.0 pg / ml, 2.9±1.6 pg / ml, respectively). Children in the general group with iron deficiency anaemia had an average correlation coefficient (r=+0.65) IL-2 with haemoglobin and a high correlation coefficient with serum ferritin (r=+0.8). The incidence of pneumonia in the group of children with iron deficiency anaemia was 17.9%. Conclusion. The obtained lowered results of İNF-γ and İL-2 in comparison with the control group and an increased incidence of respiratory diseases indicate a possible reduction in cellular immunity.
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Kabakus, N. "Reversal of Iron Deficiency Anemia-induced Peripheral Neuropathy by Iron Treatment in Children with Iron Deficiency Anemia." Journal of Tropical Pediatrics 48, no. 4 (August 1, 2002): 204–9. http://dx.doi.org/10.1093/tropej/48.4.204.

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Samorodnova, Elena A. "Iron deficiency in children: modern aspects of the problem, possibilities of primary prevention: A review." Pediatrics. Consilium Medicum, no. 4 (January 18, 2023): 302–8. http://dx.doi.org/10.26442/26586630.2022.4.201960.

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Iron deficiency (ID) is common in the pediatric population, primarily due to inadequate iron intake from food and a high requirement due to rapid growth. The most significant for pediatric practice are latent iron deficiency and iron deficiency anemia. Hyposiderosis significantly impacts a child's physical and cognitive development and immunological reactivity. It can be an independent condition and complicate the course of several other diseases. The article presents data on the most significant factors contributing to the ID, risk groups, clinical presentation features (sideropenic and anemic syndromes), criteria of laboratory diagnostics of iron deficiency anemia according to the clinical guidelines "Iron deficiency anemia" approved by the Russian Ministry of Health in 2021, and the latent iron deficiency developed by the World Health Organization experts. Also, the algorithm of ID primary prevention, diet therapy approaches, and the use of functional products and dietary supplements to meet the iron requirement of a child's organism are discussed.
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MANTADAKIS, ELPIS. "IRON DEFICIENCY ANEMIA IN CHILDREN RESIDING IN HIGH AND LOW-INCOME COUNTRIES: RISK FACTORS, PREVENTION, DIAGNOSIS AND THERAPY." Mediterranean Journal of Hematology and Infectious Diseases 12, no. 1 (June 28, 2020): e2020041. http://dx.doi.org/10.4084/mjhid.2020.041.

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Iron deficiency and iron deficiency anemia (IDA) affect approximately two billion people worldwide and most of them reside in low- and middle-income countries. In these countries, additional causes of anemia include parasitic infections like malaria, other nutritional deficiencies, chronic diseases, hemoglobinopathies and lead poisoning. Maternal anemia in resource-poor nations is associated with low birth weight, increased perinatal mortality and decreased work productivity. Maintaining a normal iron balance in these settings is challenging, as iron-rich foods with good bioavailability are of animal origin that are expensive and/or available in short supply. Apart from infrequent consumption of meat, inadequate vitamin C intake and diets rich in inhibitors of iron absorption are additional important risk factors for IDA in low-income countries. In-home iron fortification of complementary foods with micronutrient powders has been shown to effectively reduce the risk of iron deficiency and IDA in infants and young children in developing countries but is associated with unfavorable changes in gut flora and induction of intestinal inflammation that may lead to diarrhea and hospitalization. In developed countries, iron deficiency is the only frequent micronutrient deficiency. In the industrialized world, IDA is more common in infants beyond the sixth month of life, in adolescent females with heavy menstrual bleeding, in women of childbearing age and elderly people. Other special at-risk populations for IDA in developed countries are regular blood donors, endurance athletes and vegetarians. Several medicinal ferrous or ferric oral iron products exist, and their use is not apparently associated with harmful effects on the overall incidence of infectious illnesses in sideropenic and/or anemic subjects. Further research is needed to clarify the risks and benefits of supplemental iron for children exposed to parasitic infections in the third world, and for children genetically predisposed to iron overload.
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Stelle, Isabella, Anastasia Z. Kalea, and Dora I. A. Pereira. "Iron deficiency anaemia: experiences and challenges." Proceedings of the Nutrition Society 78, no. 1 (July 10, 2018): 19–26. http://dx.doi.org/10.1017/s0029665118000460.

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Iron deficiency remains the largest nutritional deficiency worldwide and the main cause of anaemia. Severe iron deficiency leads to anaemia known as iron deficiency anaemia (IDA), which affects a total of 1·24 billion people, the majority of whom are children and women from resource-poor countries. In sub-Saharan Africa, iron deficiency is frequently exacerbated by concomitant parasitic and bacterial infections and contributes to over 120 000 maternal deaths a year, while it irreparably limits the cognitive development of children and leads to poor outcomes in pregnancy.Currently available iron compounds are cheap and readily available, but constitute a non-physiological approach to providing iron that leads to significant side effects. Consequently, iron deficiency and IDA remain without an effective treatment, particularly in populations with high burden of infectious diseases. So far, despite considerable investment in the past 25 years in nutrition interventions with iron supplementation and fortification, we have been unable to significantly decrease the burden of this disease in resource-poor countries.If we are to eliminate this condition in the future, it is imperative to look beyond the strategies used until now and we should make an effort to combine community engagement and social science approaches to optimise supplementation and fortification programmes.
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Roth-Walter, Franziska. "Compensating functional iron deficiency in patients with allergies with targeted micronutrition." Allergo Journal International 30, no. 4 (April 20, 2021): 130–34. http://dx.doi.org/10.1007/s40629-021-00171-9.

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SummaryIron deficiency is associated with atopy. Iron deficiency during pregnancy increases the risk of atopic diseases in children, while both allergic children and adults are more likely to have iron deficiency anemia. Immunologically, iron deficiency leads to activation of antigen-presenting cells, promotion of Th2 cells and enables antibody class switch in B cells. In addition, iron deficiency primes mast cells for degranulation, while an increase in their iron content inhibits their degranulation. Many allergens, especially those with lipocalin and lipocalin-like protein structures, are able to bind iron and either deprive or supply this trace element to immune cells. Thus, a local induced iron deficiency will result in immune activation and allergic sensitization. However, lipocalin proteins such as the whey protein β‑lactoglobulin (BLG) can also transport micronutrients into the defense cells (holo-BLG: BLG with micronutrients) and hinder their activation, thereby promoting tolerance and protecting against allergy. Since 2019, several clinical trials have also been conducted in allergic subjects using holo-BLG as a supplementary balanced diet, leading to a reduction in symptom burden. Supplementation with holo-BLG specifically supplied defense cells with micronutrients such as iron and therefore represents a new dietary approach to compensate for functional iron deficiency in allergy sufferers.
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Dissertations / Theses on the topic "Iron deficiency diseases in children"

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Oti-Boateng, Peggy. "Effects of dietary calcium on intestinal non-haem iron absorption during weaning." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09pho881.pdf.

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Corrigenda tipped to title page. Bibliography: leaves 313-353. This study investigated the iron status and dietary intakes in 6-24 month old children in Australia and Ghana and assessed the effects of dietary calcium on intestinal iron absorption. The true prevalence of non-anaemic iron deficiency (NAID) and iron deficiency anaemia (IDA) and dietary intakes in infants and toddlers from a broad socio-economic background were assessed by haematological and biochemical parameters, semi-quantitative diet recall and anthropometric measurements. The high prevalence of iron deficiency and anaemia found in Australian and Ghanaian children can be attributed to the low intake of bioavailable iron in weaning diets which are often ingested with large amounts of calcium. While calcium has been shown to inhibit the absorption of iron, its mechanism of interaction with iron absorption at the intestinal level is not known. The rat was used as an experimental model to investigate the effects of dietary calcium on duodenal iron uptake. The results indicate there is a critical period during weaning when the consumption of high dietary calcium with low iron can retard growth potential. Dietary calcium significantly inhibits non-haem iron absorption at the intracellular level by up-regulating villus enterocyte ferritin concentrations under iron deficiency conditions.
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Adish, Abdulaziz A. "Risk factors and an assessment of control strategies for iron deficiency anemia in children in northern Ethiopia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ36948.pdf.

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Al-Othman, Abdulaziz Mohammad. "Iron intake and iron deficiency in young children." Thesis, University of Edinburgh, 1998. http://hdl.handle.net/1842/26072.

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These studies have sought to assess iron intake in young children (9-36 months), to identify nutritional and other factors that may affect iron intake and iron status. A 4-day weighted food inventory, a semi-quantitative food frequency and social questionnaire and anthropometric measurements were used. Haemoglobin (Hb), mean corpuscular volume (MCV), serum ferritin (SF), mean corpuscular haemoglobin (MCH), mean corpuscular haemoglobin concentration (MCHC) and haematocrit (Hct) were estimated in blood. Studies in Saudi Arabia: 104 healthy children randomly chosen from eight different health centres have been studied either longitudinally (n=55) or cross sectionally (n=49). Twenty four previously diagnosed iron deficient children from three hospitals were studied. Studies in Edinburgh: 62 healthy children aged 9 and 36 months old were studied. They were those whose parents agreed to participate from a larger number chosen randomly from children registered at three health centres in Edinburgh using the Lothian Health Board list. Prevalence of Anaemia in Children at The Royal Hospital for Sick Children (RHSC) and Diet: The prevalence of anaemia over a 2 months period in children whose blood samples were analysed in the Haematology Dept. was calculated. In 59 children whose parents completed a semi-quantitative food frequency and social questionnaire, of those, the iron intake and iron status was studied in detail, 41 with Hb below 11 g/dl, and 18 with normal Hb. Iron intakes less than both the Recommended Nutrient Intake (RNI) and the Lower Recommended Nutrient Intake (LRNI) have been shown to be common in children studied. Breakfast cereals and meat in addition to infant formula are important dietary factors which positively influence iron intake and iron status in this age group who are vulnerable to iron deficiency anaemia. These foods should be strongly recommended to parents for inclusion in the post-weaning diet of children of this age. A food frequency questionnaire can be used to identify children at risk.
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Pacey, Angela. "Iron deficiency and iron deficiency anemia among preschool Inuit children living in Nunavut." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=66931.

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Limited information is available about iron deficiency and iron deficiency anemia (IDA) among preschool-aged Inuit children. A cross-sectional survey was conducted with 388 Inuit children, aged 3 to 5 years, from 16 Nunavut communities. Interviews were conducted on dietary and household characteristics. Height, weight and biomarkers of iron status and Helicobacter pylori (H. pylori) exposure were measured. The prevalence of iron deficiency and IDA was calculated and risk factors were examined. The prevalence of iron deficiency was 19.2%, of IDA was 4.5% and of anemia was 20.3%. Only 0.3% of chil dren had usual iron intakes below the Estimated Average Requirement. H. pylori exposure, food insecurity and household crowding were not associated with iron deficiency or IDA. Three to four year olds were more likely to be iron deficient than 5 year olds. Boys were more likely to be iron deficient than girls.
Peu d'informations sont disponibles sur la carence en fer et l'anémie due à une carence en fer (ACF) chez les Inuits d'âge pré-scolaire. Un sondage transversales a été conduit avec 388 enfants Inuit âgés de 3 à 5 ans, dans 16 communautés du Nunavut. Des interviewers ont conduit des entrevues alimentaires et des questionnaires à propos des caractéristiques des ménages. La taille, le poids, ainsi que des marqueurs biologiques du niveau de fer et de l'exposition à Helicobacter pylori ont été mesurés. La prévalence de la carence en fer et de l'ACF a été calculée et les facteurs de risque ont été examinées. La prévalence de la carence en fer a été 19.2%, de l'ACF a été 4.5% et de l'anémie a été 20.3%. Seulement 0.3% des enfants avaient des apports habituels en fer sous le besoin moyen estimatif. L'exposition à H. pylori, l'insécurité alimentaire et le nombre d'habitants par ménage n'étaient pas associés à une carence en fer ou à de l'ACF. La carence en fer était plus élevée chez les enfants âgés de 3 à 4 ans que chez ceux de 5 ans. La carence en fer était aussi plus élevée chez les garçons que chez les filles.
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Kemmer, Teresa M. "Iron deficiency anemia in refugee children from Burma : a policy proposal /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/6595.

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Newhouse, Ian Joseph. "The effects of prelatent and latent iron deficiency on physical work capacity." Thesis, University of British Columbia, 1987. http://hdl.handle.net/2429/27470.

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In order to examine the effects of prelatent/latent iron deficiency on physical work capacity and selected muscle enzyme activities, forty female subjects were studied before and after eight weeks of supplementation with either oral iron or a matching placebo. Initially, female volunteers engaged in regular endurance running were screened for iron deficiency by blood analysis (serum ferritin and hemoglobin). Forty non-anemic subjects with deficient iron stores underwent physiological and anthropometric tests to obtain a comprehensive profile. The specific physical work capacity tests were alactic and lactacid power on the Wingate cycle ergometer test, lactacid capacity on the anaerobic speed test, anaerobic (ventilatory) threshold using gas exchange variables, V0₂ max. and the max. treadmill velocity during the V0₂ max. test. Muscle biopsy samples pre-, and post- treatment were assayed for citrate synthase and alpha-glycerophosphate dehydrogenase activity. Treatment was oral iron supplementation (320 mg ferrous sulfate = 100 mg elemental iron taken as SLOW-Fe® twice a day) or a matching placebo. The subjects were randomly assigned to one of the treatment groups and a double-blind method of administration of the supplements was used. It was hypothesized that work capacity would be enhanced following oral iron supplementation, possibly due to the repletion of iron containing oxidative enzymes important in energy production. Results could not strongly support this hypothesis with the difference between the two groups on the work capacity and enzyme activity variables being statistically nonsignificant. Serum ferritin values rose from a mean of 12.4+4.5 to 37.7+19.7 ngml⁻¹ for the experimental group and 12.2±4.3 to 17.2±8.9 for the controls; (p=0.0025). Hemoglobin levels remained fairly constant for both treatment groups; 13.4±0.6 to 13.5±0.5 gdl⁻¹ (experimental), and 13.0±0.6 to 13.1+0.5 (control); (p=0.6). Pre to post values on the work capacity variables, experimental vs control respectively were: Alactic power, 8.8 to 8.4 watts-kg⁻¹ body wt. vs 8.4 to 8.2; lactacid capacity, 6.9 to 6.9 watts-kg⁻¹ body wt. vs 7.0 to 6.0; anaerobic speed test, 41.3 to 45.1 seconds vs 43.7 to 44.8; anaerobic threshold, 7.4 to 7.5 mileshour⁻¹ vs 7.2 to 7.2; V0₂ max, 51.3 to 52.7 ml-kg⁻¹ min⁻¹ vs 50.6 to 50.6; max velocity during V0₂ max, 9.8 to 9.8 mileshour⁻¹ vs 9.6 to 9.5. Except for alactic power, the change in work capacity favored the iron treated group. Noting this trend, further study may be warranted. Prelatent/latent iron deficiency appeared not to depress the activities of the two enzymes measured. Cytoplasmic alpha-glycerophosphate dehydrogenase activity rose from 0.066 to 0.085 units for the experimental group (p=.58) vs .058 to .066 for the control group and citrate synthase activity changed from 0.047 to 0.048 (experimental) vs 0.039 to 0.042 (control). It can be concluded that eight weeks of iron supplementation to prelatent/latent iron deficient, physically active females does not significantly enhance work capacity nor the activity of 2 oxidative muscle enzymes (citrate synthase and cytoplasmic alpha-glycerophosphate dehydrogenase). Within the limitations of this study the presence of a serum ferritin below 20 ng-ml⁻¹ does not pose a significant handicap to anaerobic or aerobic capacity.
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Persson, Viveka. "Vitamin A Intake, Status and Improvement Using the Dietary Approach : Studies of Vulnerable Groups in Three Asian Countries." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5106-3/.

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Oppenheimer, Stephen James. "Iron deficiency and susceptibility to infection : a prospective study of the effects of iron deficiency and iron prophylaxis in infants in Papua New Guinea." Thesis, University of Oxford, 1987. http://ora.ox.ac.uk/objects/uuid:1891d054-1564-47f5-b2e0-b6da5f60e996.

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Investigation of the relationship between iron deficiency, iron supplementation and susceptibility to infection, was suggested by the author's initial observations of an association of anaemia with serious bacterial infections in infancy in Papua New Guinea. The bulk of previous longitudinal clinical intervention studies in infancy showed beneficial effects of iron supplementation. However, defects of control and design and recording in these studies and contradictory anecdotal reports left the question unresolved. A prospective, placebo-controlled, randomised, double-blind trial of iron prophylaxis (3ml intramuscular iron dextran = 150mg Fe) to two month old infants was carried out on the North Coast of Papua New Guinea where there is high transmission of malaria. A literature review, pilot studies, protocol, demography, geography and laboratory methods developed are described. Findings indicate that the placebo control group became relatively iron deficient over the first year of life and that the iron dextran group had adequate, although not excessive iron stores and a higher mean haemoglobin; however, the prevalence and effects of malaria recorded in the field were higher in the iron dextran group. Analysis of field and hospital infectious morbidity in the trial indicated a deleterious association with iron dextran for all causes including respiratory infections (the main single reason for admission). Total duration of hospitalisation was significantly increased in the iron dextran group. Analysis of other factors showed (1) a higher admission rate associated with low weight-for-height recorded at the start of the trial; (2) a significant positive correlation between birth haemoglobin and hospital morbidity rates; (3) increased malaria rates in primiparous mothers of the cohort infants who received iron infusion during pregnancy; (4) lower relative risk of malaria associated with iron prophylaxis in individuals with alpha thalassaemia, which was found to be highly prevalent in this region. In conclusion, it is suggested that policies of iron supplementation, total dose iron injection and routine presumptive iron therapy for anaemia which are widely in practice in malaria endemic areas should be closely reviewed.
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Harding, Scott V. "Evaluation of the iron, antioxidant and dietary status of iron supplemented breastfed healthy infants /." Internet access available to MUN users only, 2003. http://collections.mun.ca/u?/theses,160317.

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Kargarnovin, Zahra. "Prevalence, risk factors and results of intervention among anaemic Iranian children in a low socio-economic community in urban Tehran." Thesis, University College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298280.

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Books on the topic "Iron deficiency diseases in children"

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P, Vijayalakshmi, ed. Enhanced bioavailability of iron from mungbeans and its effects on health of schoolchildren. Taiwan: AVRDC-the World Vegetable Center, 2003.

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F, Waters William, and Boy Erick, eds. Fortificación en casa con micronutrientes de los alimentos de los niños y niñas de 6 a 59 meses de edad para combatir la anemia por falta de hierro y otras deficiencias: Una estrategia familiar efectiva al nivel local para disminuir la alta prevalencia de anemia nutricional, usando chis paz de salud. Quito, Ecuador: Aliméntate Ecuador, 2008.

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T, Kothari Monica, and Macro International MEASURE/DHS+ (Programme), eds. Micronutrient update. Calverton, MD: Macro International, 2007.

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O, Earl Robert, Woteki Catherine E, Calloway Doris Howes, Institute of Medicine (U.S.). Committee on the Prevention, Detection, and Management of Iron Deficiency Anemia among U.S. Children and Women of Childbearing Age., and Institute of Medicine (U.S.). Food and Nutrition Board., eds. Iron deficiency anemia: Recommended guidelines for the prevention, detection, and management among U.S. children and women of childbearing age. Washington, DC: National Academy Press, 1993.

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Dincer, Yildiz. Iron deficiency and its complications. Hauppauge, N.Y: Nova Science Publishers, 2011.

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Reddy, M. Ramakrishna. Prevalence of iron deficiency anaemia and malnuitrition in India. Bangalore: Institute for Social and Economic Change, 2004.

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Yehuda, Shlomo. Iron deficiency and overload: From basic biology to clinical medicine. New York, N.Y: Humana Press, 2010.

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Ruel, Marie T. Can food-based strategies help reduce vitamin A and iron deficiencies?: A review of recent evidence. Washington, D.C: International Food Policy Research Institute, 2001.

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Serge, Hercberg, Galan P, and Dupin Henri professeur, eds. Aspects actuels des carences en fer et en folates dans le monde: Colloque international, Paris 11-13 juillet 1989. Paris: Editions de l'Institut national de la santé et de la recherche médicale, 1990.

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Fazlul, Karim, and BRAC (Organization), eds. Reaching sprinkles to millions: Social marketing strategy for combating childhood iron deficiency anemia. Dhaka: BRAC, 2006.

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Book chapters on the topic "Iron deficiency diseases in children"

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Gupta, Anil. "Consequences of Iron Deficiency Anemia." In Nutritional Anemia in Preschool Children, 131–47. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5178-4_7.

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Gupta, Anil. "Aetiology of Iron Deficiency in Children." In Nutritional Anemia in Preschool Children, 47–118. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5178-4_5.

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Schenck, John F. "MRI of Brain Iron and Neurodegenerative Diseases: A Potential Biomarker." In Iron Deficiency and Overload, 223–40. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-462-9_13.

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Mitchell, Deanna, Jessica Foley, and Aarti Kamat. "Nutritional Anemias: Iron Deficiency and Megaloblastic Anemia." In Benign Hematologic Disorders in Children, 15–30. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-49980-8_2.

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Greuter, Thomas, and Stephan R. Vavricka. "Diagnosis and Management of Iron Deficiency in Inflammatory Bowel Disease." In Nutritional Management of Inflammatory Bowel Diseases, 53–64. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26890-3_4.

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Hoffmann, Alexander, Lara Valente de Souza, and Günter Weiss. "Iron Deficiency and Anemia Associated with Infectious and Inflammatory Diseases." In Nutrition and Health, 223–34. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-14521-6_17.

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Yip, Ray. "The Epidemiology of Childhood Iron Deficiency: Evidence for Improving Iron Nutrition among US Children." In Brain, Behaviour, and Iron in the Infant Diet, 27–39. London: Springer London, 1990. http://dx.doi.org/10.1007/978-1-4471-1766-7_3.

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Youdim, M. B. H., K. L. Leenders, and D. Ben-Shachar. "Brain Iron Uptake and Transport in Animal Model of Iron Deficiency, Tardive Dyskinesia and Neurodegenerative Diseases." In Blood—Brain Barrier, 147–56. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-0579-2_12.

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Ghoda, Manoj K. "Case 38: A 3-Year-Old Boy with Recurrent Jaundice and Severe Iron Deficiency Anemia." In Neonatal and Pediatric Liver and Metabolic Diseases, 215–16. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-9231-7_39.

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Julián-Almárcegui, C., A. L. M. Heath, L. Harvey, B. Sarria, and I. Huybrechts. "11. Intake and dietary sources of heme and non-heme iron in children and adolescents: recommendations for preventing iron deficiency." In Human Health Handbooks, 183–202. The Netherlands: Wageningen Academic Publishers, 2016. http://dx.doi.org/10.3920/978-90-8686-822-3_11.

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Conference papers on the topic "Iron deficiency diseases in children"

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Wardani, Endah Kusuma, Nurul Eko Widiyastuti, Lutvia Dwi Rofika, and Wahyu Adri Wirawati. "Factors Affecting Stunting among Children Under Five Years of Age in Banyuwangi, East Java." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.03.80.

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ABSTRACT Background: Stunting, chronic malnutrition, results from the exposure of the fetus and young child to nutritional deficiency and infectious disease. In Indonesia, 30.8% of children were stunted, in which 26.2% was in East Java and 8.1% Banyuwangi Regency. This study aimed to investigate the factors affecting stunting among children under five years of age in Banyuwangi, East Java. Subjects and Method: This was a cross-sectional study conducted at Klatak and Wonosobo Community Health Centers, Central Java. a sample of 60 children under five years of age was selected for this study. The study variables were child’s gender, maternal age at pregnancy, maternal education, maternal work status, iron intake, history of chronic energy deficiency, exclusive breastfeeding, supplementary feeding, and history of infectious disease. The frequency distribution data were reported descriptively. Results: The majority of stunted children under study were male (53.3%). Most of the women were at age 20 to 34 years during pregnancy (58.3%). As many as 73.3% mothers were low educated. Most of the mothers were housewives (85%). 78.3% of women took iron supplement during pregnancy. Most of the children did not have the history of chronic energy deficiency (60%). Most of the children received exclusive breastfeeding (61.7%) and supplementary feeding (65%). Only a few children had the history of infectious disease (6.7%). Conclusion: The characteristics of subjects under study vary with maternal age at pregnancy, maternal education, maternal work status, iron intake, history of chronic energy deficiency, exclusive breastfeeding, supplementary feeding, and history of infectious disease. Keywords: stunting, children under five years of age, factors Correspondence: Endah Kusuma Wardani. Midwifery Program, School of Health Sciences Banyuwangi. Jl. Letkol Istiqlah No. 109, Banyuwangi, East Java, 68422. Email: qsuma89@yahoo.com. Mobile: +6282257193736. DOI: https://doi.org/10.26911/the7thicph.03.80
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Klimov, Leonid, Marina Stoyan, Ekaterina Zavyalova, Irina Zakharova, and Victoria Kuryaninova. "GP187 Anthropometric parameters in children with celiac disease complicated by iron deficiency status." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.248.

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Dudnyk, Veronika, Kateryna Khromykh, and Zhmurchuk Vasyl. "292 Iron deficiency in children with asthma, a predictor of severity and disease control." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.292.

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Segal, Julia, Meghan McCormick, Ram Kalpatthi, and James Zullo. "Hospitalization For Iron Deficiency Anemia In Young Children: A Multicenter Analysis." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.566-a.

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Rutter, Zoe, and Richard Lee-kelland. "608 Prevalence of iron deficiency anaemia in children undergoing autism assessment." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Liverpool, 28–30 June 2022. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2022. http://dx.doi.org/10.1136/archdischild-2022-rcpch.114.

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Danhelovska, Tereza, Hana Kolarova, Jan Langer, Kamila Berankova, Hana Hansikova, Marketa Tesarova, Tomas Honzik, and Jiri Zeman. "GP224 Multisystem mitochondrial diseases in children with maternally inherited complex I deficiency." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.283.

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Nugroho, Fajar Agung, Thomas H. A. Ederveen, Adi Wibowo, Jos Boekhorst, Marien I. de Jonge, and Tom Heskes. "Application of A Causal Discovery Model to Study The Effect of Iron Supplementation in Children with Iron Deficiency Anemia." In 2019 3rd International Conference on Informatics and Computational Sciences (ICICoS). IEEE, 2019. http://dx.doi.org/10.1109/icicos48119.2019.8982503.

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Koh, Magdalena Yvonne, and Aishworiya Ramkumar. "334 Iron and vitamin D deficiency in young children with autism spectrum disorder in singapore." In RCPCH Conference Singapore. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/bmjpo-2021-rcpch.184.

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Sampath-Arutperumselvi, Vidhyashree, Aruna Gorugantu, Sherin Hamza, Mahjouba Ahmid Ahmid, and Alison Kelly. "613 A cross sectional study in children with iron deficiency attending a regional specialist health care service." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 15 June 2021–17 June 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-rcpch.101.

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Turudic, Daniel, Iva Busic, Marko Bilic, and Ernest Bilic. "294 Comparison of mathematical indices used to differentiate between β-thalassemia and iron deficiency anemia in croatian children." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.294.

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Reports on the topic "Iron deficiency diseases in children"

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Vakhlova, I. V., and Yu N. Ibragimova. Electronic educational resource (EOR) "Hematology: Iron deficiency anemia in children". SIB-Expertise, February 2023. http://dx.doi.org/10.12731/er0673.21022023.

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Chen, Yona, Jeffrey Buyer, and Yitzhak Hadar. Microbial Activity in the Rhizosphere in Relation to the Iron Nutrition of Plants. United States Department of Agriculture, October 1993. http://dx.doi.org/10.32747/1993.7613020.bard.

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Iron is the fourth most abundant element in the soil, but since it forms insoluble hydroxides at neutral and basic pH, it often falls short of meeting the basic requirements of plants and microorganisms. Most aerobic and facultative aerobic microorganisms possess a high-affinity Fe transport system in which siderophores are excreted and the consequent Fe complex is taken up via a cognate specific receptor and a transport pathway. The role of the siderophore in Fe uptake by plants and microorganisms was the focus of this study. In this research Rhizopus arrhizus was found to produce a novel siderophore named Rhizoferrin when grown under Fe deficiency. This compound was purified and its chemical structure was elucidated. Fe-Rhizoferrin was found to alleviate Fe deficiency when applied to several plants grown in nutrient solutions. It was concluded that Fe-Rhizoferrin is the most efficient Fe source for plants when compared with other among microbial siderophores known to date and its activity equals that of the most efficient synthetic commercial iron fertilizer-Fe EDDHA. Siderophores produced by several rhizosphere organisms including Rhizopus Pseudomonas were purified. Monoclonal antibodies were produced and used to develop a method for detection of the siderophores produced by plant-growth-promoting microorganisms in barley rhizosphere. The presence of an Fe-ferrichrome uptake in fluorescent Pseudomonas spp. was demonstrated, and its structural requirements were mapped in P. putida with the help of biomimetic ferrichrome analogs. Using competition experiments, it was shown that FOB, Cop B and FC share at least one common determinant in their uptake pathway. Since FC analogs did not affect FOB or Cop-mediated 55Fe uptake, it could be concluded that these siderophores make use of a different receptor(s) than FC. Therefore, recognition of Cop, FOB and FC proceeds through different receptors having different structural requirements. On the other hand, the phytosiderophores mugineic acid (MA and DMA), were utilized indirectly via ligand exchange by P. putida. Receptors from different biological systems seem to differ in their structural requirements for siderophore recognition and uptake. The design of genus- or species-specific drugs, probes or chemicals, along with an understanding of plant-microbe and microbe-microbe relationships as well as developing methods to detect siderophores using monoclonal antibodies are useful for manipulating the composition of the rhizosphere microbial population for better plant growth, Fe-nutrition and protection from diseases.
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