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Books on the topic 'Ionic mechanism'

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1

Went, Charles. Ionic Organic Mechanisms. London: Macmillan Education UK, 1986. http://dx.doi.org/10.1007/978-1-349-07964-3.

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2

Erusalimskii, B. L. Mechanisms of Ionic Polymerization. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-8392-5.

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3

Erusalimskii, B. L. Mechanisms of Ionic Polymerization: Current Problems. Boston, MA: Springer US, 1987.

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4

Erusalimskiĭ, B. L. Mechanisms of ionic polymerization: Current problems. New York: Consultants Bureau, 1986.

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5

Celano, Umberto. Metrology and Physical Mechanisms in New Generation Ionic Devices. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39531-9.

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6

Brown, A. M. Ionic mechanisms of aglycemic axon injury in mammalian central white mater. Philadelphia, Penn: Lippincott Williams & Wilkins, Inc., 2001.

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7

Farley, Joseph. Ionic mechanisms subserving mechanosensory transduction and neural integration in statocyst hair cells of Hermissenda: Final technical report. Princeton, N.J: Dept. of Psychology, Princeton University, 1988.

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8

Nasmith, Patricia Elizabeth. Mechanism and effect of ionic changes during neutrophil activation. 1989.

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9

Mechanisms of Ionic Polymerization. Springer My Copy UK, 1986.

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10

Ionic Organic Mechanisms (Dimensions of Science). Palgrave Macmillan, 1986.

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11

Erusalimskii, B. L. Mechanisms of Ionic Polymerization: Current Problems. Springer, 2012.

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12

Celano, Umberto. Metrology and Physical Mechanisms in New Generation Ionic Devices. Springer, 2018.

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13

Celano, Umberto. Metrology and Physical Mechanisms in New Generation Ionic Devices. Springer, 2016.

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14

Skinner, Frances Kiku. Ionic transport mechanisms in a model of gastric smooth muscle. 1992.

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15

Gukovskaya, A. Mechanisms of Receptor-Mediated Generation of Ionic Signals in Rat Thymocytes and Ehrlich Ascites Tumor Cells. Routledge, 1990.

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16

Advances in Artificial and Biological Membranes: Mechanisms of Ionic Sensitivity, Ion-Sensor Designs and Applications for Ions Measurement. MDPI, 2021. http://dx.doi.org/10.3390/books978-3-0365-1071-2.

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17

Mease, Philip. Neurobiology of pain in osteoarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0013.

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Significant advances in our understanding of the neurobiology of pain in osteoarthritis (OA) have occurred in the last decade and are herein summarized. Pain is the predominant symptom of OA and occurs at multiple levels from non-cartilage peripheral tissues to spinal cord, and brain and back. At each level, nerve function is regulated by complex ionic channels, neuropeptide expression, and cytokine and chemokine activity. Previously considered a non-inflammatory condition, it is now recognized that cell proliferation and inflammatory cytokine production occurs in OA synovium, contributing to peripheral sensitization. Genetic profile influences nociceptive neuropeptide expression and thus, pain perception. Both peripheral and central sensitizing factors, including increased neuropeptide and microglial activity, lead to pain augmentation and persistence. Pain processing in brain centres such as the somatosensory cortex and insula are influenced by affective areas such as the amygdala. Descending receptor pathways through the midbrain to the dorsal horn, such as norepinephrine, serotonin, opioid, and cannabinoid, normally provide pain inhibitory function but this function may be diminished in chronic pain states such as OA, leading to allodynia and hyperalgesia. Functional neuroimaging has contributed to our understanding of the complex interplay of peripheral and central mechanisms. Recent evidence that grey matter volume decrease in chronic pain states may be reversible (e.g. after pain relief post OA hip arthroplasty) illuminates the potential for central neuroplasticity. Greater understanding of the neurobiology of OA pain provides evidence for therapeutic approaches that address peripheral and/or central pain mechanisms and provides a guide for future targeted pain therapeutics.
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