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Books on the topic 'Intravenous Immunoglobulin'

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Published: 4 June 2021
Last updated: 21 February 2023

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1

Stephen, Jolles, ed. Intravenous immunoglobulin in dermatology. London: Martin Dunitz, 2003.

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2

Lazarus, Alan H. Immunoglobulin therapy. Bethesda, Md: AABB Press, 2010.

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3

Beratan, Marian E. Intravenous immunoglobulin: Prevention and treatment of disease : January 1986 through April 1990 : 888 citations. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Library of Medicine, Reference Section ; Washington, D.C. : Sold by the Supt. of Docs., U.S. G.P.O., 1990.

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4

International Symposium on IVIG (4th 1996 Interlaken, Switzerland). Advances in intravenous immunoglobulin research and therapy: Proceedings from Interlaken's Fourth International Symposium on IVIG, 11-13 June, 1996. New York: Parthenon Pub. Group, 1996.

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5

International Society of Hematology. Congress. Immunomodulation by intravenous immunoglobulin: The proceedings of a symposium held at the 24th Congress of the International Society of Haematology, London, UK, August 1992, combined with the proceedings of an international symposium held in Mar del Plata, Argentina, October 1992. Carnforth, Lancs, UK: Parthenon Pub. Group, 1993.

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6

1953-, Lee Martin L., and Strand Vibeke, eds. Intravenous immunoglobulins in clinical practice. New York: Dekker, 1997.

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7

Delire, Marcel. Immunoglobulins: Rationale for the clinical use of polyvalent intravenous immunoglobulins. Petersfield, UK: Wrightson Biomedical Pub., 1995.

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8

C, Dalakas Marinos, and Späth Peter C, eds. Intravenous immunoglobulins in the third millennium. Boca Raton: Parthenon Pub. Group, 2004.

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9

Senate, United States Congress. A bill to amend title XVIII of the Social Security Act to improve access of Medicare beneficiaries to intravenous immune globulins. Washington, D.C: U.S. G.P.O., 2008.

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10

United States. Congress. House. A bill to amend title XVIII of the Social Security Act to improve access of Medicare beneficiaries to immune globulins. [Washington, D.C.?]: [United States Government Printing Office], 2007.

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11

Jolles, Stephen. Intravenous Immunoglobulin in Dermatology. Taylor & Francis Group, 2002.

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12

Rewald, E. Immunomodulation by Intravenous Immunoglobulin. Taylor & Francis, 1993.

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13

Intravenous Immunoglobulin: Research and Therapy. Informa Healthcare, 1996.

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14

Intravenous Immunoglobulin in Clinical Practice. 2nd ed. Marcel Dekker Inc, 2006.

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15

L, Yap P., ed. Clinical applications of intravenous immunoglobulin therapy. Edinburgh: Churchill Livingstone, 1992.

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16

Jolles, Stephen. Use of Intravenous Immunoglobulin in Clinical Dermatology. Informa Healthcare, 2002.

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17

C, Dalakas Marinos, ed. The use of intravenous immunoglobulin for neurologic diseases. Hagerstown, MD: Lippincott Williams & Wilkins, 1998.

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18

Perret, B. A., P. Imbach, and G. R. F. Krueger. Long-term Intravenous Immunoglobulin Treatment in Patients with AIDS-related Complex. S Karger Ag, 1990.

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19

Webster, Michelle. Relative efficacy of intravenous immunoglobulin G in ameliorating thrombocytopenia induced by anti-platelet GPIIbIIIa versus GPIbalpha antibodies. 2006.

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20

G, Desai Rajendra, and International Cancer Congress, (15th : 1990 : Hamburg), eds. Recent advances in intravenous immunoglobulin therapy: A symposium held in conjunction with the 15th International Cancer Congress, Hamburg,... 1990. Philadelphia: J.B. Lippincott for the American Cancer Society, 1991.

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21

Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Blood transfusion. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0017.

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Introduction - Using the blood transfusion laboratory - Transfusion of red blood cells - Platelet transfusion - Fresh frozen plasma - Intravenous immunoglobulin - Transfusion transmitted infections - Irradiated blood products - Strategies for reducing blood transfusion in surgery - Maximum surgical blood ordering schedule (MSBOS) - Patients refusing blood transfusion for religious reasons, i.e. Jehovah’s Witnesses
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22

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, Shubha Allard, and Mammit Kaur. Blood transfusion. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0017_update_001.

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Introduction - Using the blood transfusion laboratory - Transfusion of red blood cells - Platelet transfusion - Fresh frozen plasma - Intravenous immunoglobulin - Transfusion transmitted infections - Irradiated blood products - Strategies for reducing blood transfusion in surgery - Maximum surgical blood ordering schedule (MSBOS) - Patients refusing blood transfusion for religious reasons, i.e. Jehovah’s Witnesses
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23

Imbach, Paul. Immunotherapy with Intravenous Immunoglobulins. Elsevier Science & Technology Books, 2012.

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24

Imbach, P. Immunotherapy With Intravenous Immunoglobulins. Edited by P. Imbach. Academic Press, 1991.

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25

Immunotherapy with Intravenous Immunoglobulins. Elsevier, 1991. http://dx.doi.org/10.1016/c2009-0-02690-7.

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26

Wijdicks, Eelco F. M., and Sarah L. Clark. Immunosuppression and Immunotherapy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0010.

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Immune modulation in the neurosciences intensive care unit mostly involves high-dose corticosteroids, plasma exchange, and immunoglobulin. Corticosteroids are frequently used in patients with neurologic complications of cancer. Neurosurgeons typically use corticosteroids after performing a craniectomy to reduce cerebral edema. Corticosteroids are the established initial treatment modality of choice for patients with acute metastatic epidural spinal cord compression. Therapeutic apheresis or immunoglobulin is generally used as supportive therapy in patients with Guillain-Barré syndrome, myasthenia crisis, and autoimmune encephalitis. Immune modulation has been considered essential in autoimmune encephalitis despite lack of controlled clinical trials. In these now better characterized disorders, a combination of corticosteroids, intravenous immune globulin (IVIG), plasma exchange, rituximab, or cyclophosphamide are used. The use of acute immunotherapy and precautionary measures are discussed in this chapter.
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27

Morell, A. Clinical Use of Intravenous Immunoglobulins. Academic Pr, 1986.

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28

Lee, Martin L. Intravenous Immunoglobulins in Clinical Practice. Taylor & Francis Group, 1997.

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29

Lee, Martin L. Intravenous Immunoglobulins in Clinical Practice. Taylor & Francis Group, 1997.

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30

Lee, Martin L., ed. Intravenous Immunoglobulins in Clinical Practice. CRC Press, 1997. http://dx.doi.org/10.1201/9781420001143.

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31

Viral safety of intravenous immunoglobulins. Octapharma, 1997.

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32

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 35-Year-Old Man with Progressive Left-Hand Weakness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0001.

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Multifocal motor neuropathy (MMN) may be mistaken for common entrapment neuropathies, although absence of significant sensory findings is a helpful clue to the diagnosis. Multifocal motor neuropathy may also mimic motor neuron disease. Electrophysiological evidence of conduction block at a nerve site not typically prone to compression is consistent with MMN. A positive anti-GM1 antibody also supports the diagnosis. First-line treatment of MMN is intravenous immunoglobulin (IVIG), and the majority of patients have rapid improvement of their weakness. The clinical features, differential diagnosis, investigations, and treatment options are described in this chapter.
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33

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 48-Year-Old with Progressive Weakness and Pain. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0005.

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Guillain-Barré syndrome may present in several ways, although predominant proximal weakness is a common feature of the disease to recognize. The differential diagnosis may be extensive and can include infection, vasculitis, toxin exposure, and malignancy. A lumbar puncture must be done with minimal delay to evaluate for cerebrospinal fluid (CSF) albuminocytological dissociation, however results may be normal early in the course of the disease. EMG/NCS are helpful to support the diagnosis, and early treatment with intravenous immunoglobulin (IVIG) is essential. This chapter discusses the clinical features and diagnostic considerations of this important condition.
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34

(Editor), Marinos C. Dalakas, and Peter Spath (Editor), eds. Intravenous Immunoglobulins: In The Third Millennium. Informa Healthcare, 2003.

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35

Dalakas, Marinos C., and Peter J. Späth. Intravenous Immunoglobulins in the Third Millennium. Taylor & Francis Group, 2004.

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36

Treatment of Neurological Disorders with Intravenous Immunoglobulins. Informa Healthcare, 2000.

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37

Intravenous Immunoglobulins: Clinical Benefits and Future Prospects. Taylor & Francis, 1994.

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38

(Editor), Martin L. Lee, and David J. Rechtman (Editor), eds. Intravenous Immunoglobulins in Clinical Practice, Second Edition. 2nd ed. Informa Healthcare, 2008.

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39

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 30-Year-Old Male Requiring Management of Progressive Weakness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0006.

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Management of Guillain-Barré syndrome involves several factors. Pulmonary function tests are important to monitor. Telemetry and blood pressure must be monitored because of the significant risk of autonomic dysfunction. Intravenous immunoglobulin is the immunotherapy of choice for this disease, as it is less complicated to administer than plasma exchange. Supportive management issues to address include deep vein thrombosis prophylaxis, bowel/bladder care as ileus and urinary retention may be develop secondary to dysautonomia, physical/occupational/speech therapy consults, nutrition, and pain control. Gabapentin can also be helpful for GBS-related pain. This chapter discusses an approach to treatment that includes the role of respiratory and autonomic monitoring as well as immunotherapy considerations.
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40

Dominioni, U. Intravenous Immunoglobulins Today And Tomorrow (International Congress & Symposium). Royal Society of Medicine Press Ltd, 1992.

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41

Fox, Grenville, Nicholas Hoque, and Timothy Watts. Metabolic problems and jaundice. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198703952.003.0013.

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This chapter covers problems of glycaemic control in neonates (including investigation and management of hypoglycaemia and hyperglycaemia); management of babies of women with diabetes (infant of the diabetic mother); the physiological basis and rational approach to the treatment of metabolic acidosis in the newborn; the presentation, investigation, and management of inborn errors of metabolism presenting in the newborn; and metabolic bone disease (also known as osteopenia or rickets of prematurity). There is an overview of the investigation and treatment of neonatal jaundice, including physiological jaundice, aetiology, and investigation of non-physiological jaundice and aims of treatment of this; with guidelines for the use of phototherapy, exchange transfusion, and intravenous immunoglobulin. The importance of assessment of prolonged jaundice with reference to conjugated hyperbilirubinaemia is also covered in this section.
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42

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. An 80-Year-Old Myasthenia Gravis Patient with Worsening Weakness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0031.

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In myasthenia gravis, weakness and respiratory insufficiency can occur quickly. It is important for the treating physician to recognize this and institute treatment rapidly. Increasing weakness of the neck may herald impending respiratory insufficiency. The single breath count is an easy way ti assess ventilatory function. Because of bulbar weakness and increasing secretions usually bi-level positive pressure airway pressure is used with extreme caution. Intubation with effective management if the airway is preferred. Differentiation of myasthenic crisis from cholinergic crisis is explained; although cholinergic crisis is relatively uncommon. Treatment modalities can include intravenous immunoglobulin, plasma exchange, and corticosteroids. Corticosteroids should be used with caution since they may exacerbate myasthenic symptoms. Treatment with a steroid sparing agent is discussed. A table is presented which lists signs and symptoms that can suggest the need for intubation.
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43

H, Waters A., Webster A. D. B, Sandoz Pharmaceuticals, Medi-Cine Communications International, and Royal Society of Medicine (Great Britain), eds. Intravenous immunoglobulins in immunodeficiency syndromes and idiopathic thrombocytopenic purpura. London: Royal Society of Medicine, 1985.

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44

Donaghy, Michael. Focal peripheral neuropathy. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0487.

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Some causes of focal peripheral nerve damage are self-evident, such as involvement at sites of trauma, tissue necrosis, infiltration by tumour, or damage by radiotherapy. Focal compressive and entrapment neuropathies are particularly valuable to identify in civilian practice, since recovery may follow relief of the compression. Leprosy is a common global cause of focal neuropathy, which involves prominent loss of pain sensation with secondary acromutilation, and requires early antibiotic treatment. Mononeuritis multiplex due to vasculitis requires prompt diagnosis and immunosuppressive treatment to limit the severity and extent of peripheral nerve damage. Various other medical conditions, both inherited and acquired, can present with focal neuropathy rather than polyneuropathy, the most common of which are diabetes mellitus and hereditary liability to pressure palsies. A purely motor focal presentation should raise the question of multifocal motor neuropathy with conduction block, which usually responds well to high-dose intravenous immunoglobulin infusions.
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45

Brealey, David, and Nicholas Hirsch. Diagnosis, assessment, and management of Guillain–Barré syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0246.

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The Guillain–Barré Syndrome describes a spectrum of acute inflammatory polyneuropathies and is the commonest cause of acute flaccid paralysis within the western world. The pathophysiology is complex and poorly understood, but appears to be an immune-mediated destruction of either the myelin sheath and/or the axons, predominantly of motor nerves. The clinical presentation is classically a rapid, ascending, flaccid paralysis, with minimal sensory deficit. This may ascend to involve respiratory or bulbar muscle function. These patients need careful monitoring and, if deteriorating, should be electively intubated and ventilated. Autonomic instability and sensory disturbance, including pain, is common. Treatment of the underlying condition relies upon immunomodulation with either intravenous immunoglobulin or plasma exchange. Supportive care is aimed at maintaining a safe airway, ventilatory support, and managing the complications of autonomic dysfunction and prolonged immobility. Mortality rates range up to 20%, but are significantly better in specialist neuromedical units. Survivors are often left with significant disability.
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46

Plasmapheresis and Intravenous Immunoglobin: Clinical Uses, Potential Complications and Long-Term Health Effects. Nova Science Pub Inc, 2014.

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47

Kiehl, Michael G. Immunomodulation with Immunoglobulins for Autoimmune Diseases and Infections. Edited by Michael G. Kiehl. Thieme Medical Publishers, 2002.

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48

L, Dominioni, and Nydegger U. E, eds. Intravenous immunoglobulins today & tomorrow: Proceedings of a symposium sponsored by Sandoz Pharma AG and the Swiss Red Cross, held in Basle, Switzerland, 1 September 1991. London: Royal Society of Medicine Services, 1992.

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49

Paul, Imbach, ed. Immunotherapy with intravenous immunoglobulins: Proceedings of a conference held in Interlaken 6-9 May 1990. London: Academic Press, 1991.

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50

A, Morell, and Nydegger U. E, eds. Clinical use of intravenous immunoglobulins: Proceedings of a conference held at Interlaken September 15-18, 1985. London: Academic, 1986.

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