Relevant bibliographies by topics / Intracranial hypertension (ICH)

Academic literature on the topic 'Intracranial hypertension (ICH)'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Contents

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Intracranial hypertension (ICH).'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Intracranial hypertension (ICH)"

1

Mrozek, Ségolène, Laurent Lonjaret, Aude Jaffre, Anne-Christine Januel, Nicolas Raposo, Sergio Boetto, Jean-François Albucher, Olivier Fourcade, and Thomas Geeraerts. "Reversible Cerebral Vasoconstriction Syndrome with Intracranial Hypertension: Should Decompressive Craniectomy Be Considered." Case Reports in Neurology 9, no. 1 (January 24, 2017): 6–11. http://dx.doi.org/10.1159/000455090.

Full text
Abstract:
Background: Reversible cerebral vasoconstriction syndrome (RCVS) is a rare cause of intracerebral hemorrhage (ICH) causing intracranial hypertension. Methods: Case report. Results: We report a case of RCVS-related ICH leading to refractory intracranial hypertension. A decompressive craniectomy was performed to control intracranial pressure. We discuss here the management of RCVS with intracranial hypertension. Decompressive craniectomy was preformed to avoid the risky option of high cerebral perfusion pressure management with the risk of bleeding, hemorrhagic complications, and high doses of norepinephrine. Neurological outcome was good. Conclusion: RCVS has a complex pathophysiology and can be very difficult to manage in cases of intracranial hypertension. Decompressive craniectomy should probably be considered.
APA, Harvard, Vancouver, ISO, and other styles
2

Petrayevsky, A. V., K. S. Trishkin, and I. A. Gndoyan. "Idiopathic Intracranial Hypertension: Current Neuroophthalmologic Points." Ophthalmology in Russia 18, no. 4 (December 28, 2021): 791–800. http://dx.doi.org/10.18008/1816-5095-2021-4-791-800.

Full text
Abstract:
Idiopathic intracranial hypertension (ICH) is a neuroophophthalmologic syndrome, the main ophthalmologic symptoms of which are vision loss and bilateral swelling of optic nerve disks (OD). The article provides a review covering various aspects of ICH. Epidemiology: ICH occurs mainly in women aged 20–45 years with body overweight. In this group of patients world incidence is 12–20 per 100 000 of population per year. In the total population it represents 0.5–2 cases per 100 000 of population per year. The eightfold gender predominance of women with ICH is observed. Etiology is not fully known until present time. The reliable connection between obesity in childbearing age women and menstrual cycle disorders as evidences of hormonal changes has been established. The probable mechanisms of increase of ICP are supposed: hyperproduction of and disturbances of its absorption, CSF mechanical pressure on the optic nerve sheath; restriction of venous outflow from the brain due to the pathology of venous sinuses; stimulating effect of abundant vitamin A in diet resulting in the fat tissue formation with the development of obesity; expression of the protein aquaporin, involved in the regulation of body mass and water metabolism in the subarachnoid space. Clinical manifestations. Ophthalmologic symptoms of ICH: transient visual impairment, sustained loss of visual acuity, photopsy, retrobulbar pain, diplopia. Non-ophthalmologic symptoms: headache, throbbing tinnitus, hearing loss, dizziness. Diagnostics. The valuable diagnostic data can be obtained by visual field investigation, ophthalmoscopic examination with revealing of bilateral OD-edema, OD and retinal optical coherent tomography, orbital ultrasound examination, brain MRI with venography. Treatment. Non-drug therapy: weight loss, lowcalorie diet with limited water and salt intake. Drug therapy: long-term oral use of carbonic anhydrase inhibitors (acetazolamide, topiramate). High doses of steroids (methylprednisone) are used for short-term treatment of patients with fulminant disease type before surgery. Surgery: bypass surgery, cerebral venous sinus stenting and fenestrations of the optic nerve sheath: bariatric surgery with reducing of stomach volume.
APA, Harvard, Vancouver, ISO, and other styles
3

Basali, Ayman, Edward J. Mascha, Iain Kalfas, and Armin Schubert. "Relation between Perioperative Hypertension and Intracranial Hemorrhage after Craniotomy." Anesthesiology 93, no. 1 (July 1, 2000): 48–54. http://dx.doi.org/10.1097/00000542-200007000-00012.

Full text
Abstract:
Background Previous data suggest that systemic hypertension (HTN) is a risk factor for postcraniotomy intracranial hemorrhage (ICH). The authors examined the relation between perioperative blood pressure elevation and postoperative ICH using a retrospective case control design. Methods The hospital's database of all patients undergoing craniotomy from 1976 to 1992 was screened. Coagulopathic and unmatchable patients were excluded. There were 69 evaluable patients who developed ICH postoperatively (n = 69). A 2-to-1 matched (by age, date of surgery, pathologic diagnosis, surgical procedure, and surgeon) control group without postoperative ICH was assembled (n = 138). Preoperative, intraoperative, and postoperative blood pressure records (up to 12 h) were examined. Incidence of perioperative HTN (blood pressure > or = 160/90 mmHg) and odds ratios for ICH were determined. Results Of the 11,214 craniotomy patients, 86 (0.77%) suffered ICH, and 69 fulfilled inclusion criteria. The incidence of preoperative HTN was similar in the ICH (34%) and the control (24%) groups. ICH occurred 21 h (median) postoperatively, with an interquartile range of 4-52 h. Sixty-two percent of ICH patients had intraoperative HTN, compared with only 34% of controls (P < 0.001). Sixty-two percent of the ICH patients had prehemorrhage HTN in the initial 12 postoperative hours versus 25% of controls (P < 0.001), with an odds ratio of 4.6 (P < 0.001) for postoperative ICH. Hospital stay (median, 24.5 vs. 11.0 days), and mortality (18.2 vs. 1.6%) were significantly greater in the ICH than in the control groups. Conclusions ICH after craniotomy is associated with severely prolonged hospital stay and mortality. Acute blood pressure elevations occur frequently prior to postcraniotomy ICH. Patients who develop postcraniotomy ICH are more likely to be hypertensive in the intraoperative and early postoperative periods.
APA, Harvard, Vancouver, ISO, and other styles
4

Amarenco, Pierre, Jong S. Kim, Julien Labreuche, Hugo Charles, Maurice Giroud, Philippa C. Lavallée, Byung-Chul Lee, et al. "Intracranial Hemorrhage in the TST Trial." Stroke 53, no. 2 (February 2022): 457–62. http://dx.doi.org/10.1161/strokeaha.121.035846.

Full text
Abstract:
Background and Purpose: Although statins are effective in secondary prevention of ischemic stroke, they are also associated with an increase risk of intracranial hemorrhage (ICH) in certain conditions. In the TST trial (Treat Stroke to Target), we prespecified an exploration of the predictors of incident ICH. Methods: Patients with ischemic stroke in the previous 3 months or transient ischemic attack within the previous 15 days and evidence of cerebrovascular or coronary artery atherosclerosis were randomly assigned in a 1:1 ratio to a target LDL (low-density lipoprotein) cholesterol of <70 mg/dL or 100±10 mg/dL, using statin or ezetimibe. Results: Among 2860 patients enrolled, 31 incident ICH occurred over a median follow-up of 3 years (18 and 13 in the lower and higher target group, 3.21/1000 patient-years [95% CI, 2.38–4.04] and 2.32/1000 patient-years [95% CI, 1.61–3.03], respectively). While there were no baseline predictors of ICH, uncontrolled hypertension (HR, 2.51 [95% CI, 1.01–6.31], P =0.041) and being on anticoagulant (HR, 2.36 [95% CI, 1.00–5.62], P =0.047)] during the trial were significant predictors. On-treatment low LDL cholesterol was not a predictor of ICH. Conclusions: Targeting an LDL cholesterol of <70 mg/dL compared with 100±10 mg/dL in patients with atherosclerotic ischemic stroke nonsignificantly increased the risk of ICH. Incident ICHs were not associated with low LDL cholesterol. Uncontrolled hypertension and anticoagulant therapy were associated with ICH which has important clinical implications. Registration: URL: https://www.clinicaltrials.gov ; Unique identifier: NCT01252875; EUDRACT identifier: 2009-A01280-57.
APA, Harvard, Vancouver, ISO, and other styles
5

Richmond, Therese S. "Intracranial Pressure Monitoring." AACN Advanced Critical Care 4, no. 1 (February 1, 1993): 148–60. http://dx.doi.org/10.4037/15597768-1993-1012.

Full text
Abstract:
Intracranial pressure monitoring (ICP) is a technology that assists critical care nurses in the assessment, planning, intervention, and evaluation of care. The physiologic basis of intracranial hypertension (ICH) and ICP monitoring are reviewed. Types of monitors arc described. Advantages, disadvantages, and complications of fluid-filled versus fiberoptic systems are explored. Priorities in nursing care of the patient with an ICP monitor are examined
APA, Harvard, Vancouver, ISO, and other styles
6

Araújo, Marx, Benjamim Vale, Irapua Ricarte, Lívio de Macêdo, Anderson Rodrigues, and Tomásia Monteiro. "Ultrasonographic Evaluation of the Optic Nerve Sheath in the Diagnosis of Idiopathic Intracranial Hypertension." Arquivos Brasileiros de Neurocirurgia: Brazilian Neurosurgery 38, no. 01 (November 22, 2016): 073–76. http://dx.doi.org/10.1055/s-0036-1594234.

Full text
Abstract:
AbstractIntracranial hypertension (ICH) is a life-threatening condition that can be observed in several diseases. Its clinical presentation is variable, with headache, nausea, vomiting, visual disturbances, papilledema, and alterations in the level of consciousness. The gold standard for the diagnosis of ICH is still the intracranial implantation of invasive devices. Non-invasive techniques, such as ultrasonography of the optic nerve sheath (USONS), have emerged in recent years with promising clinical results. The authors report the case of a patient with progressive headache associated with visual impairment and papilledema, and the eventual diagnosis of idiopathic intracranial hypertension using USONS.
APA, Harvard, Vancouver, ISO, and other styles
7

Carney, Brian J., Erik J. Uhlmann, Maneka Puligandla, Charlene Mantia, Griffin M. Weber, Donna S. Neuberg, and Jeffrey I. Zwicker. "Recurrent Intracranial Hemorrhage and Venous Thromboembolism Following Initial Intracranial Hemorrhage in Patients with Brain Tumors on Anticoagulation." Blood 134, Supplement_1 (November 13, 2019): 2438. http://dx.doi.org/10.1182/blood-2019-126027.

Full text
Abstract:
Introduction Both venous thromboembolism (VTE) and intracranial hemorrhage (ICH) are common potentially life-threatening complications of primary and metastatic brain tumors. Despite emerging evidence regarding the safety of anticoagulation in patients with brain tumors, there is little evidence on appropriate management of VTE following an ICH. Potential management options after an ICH in patients with brain tumors include resumption of full or modified dose anticoagulation or cessation of anticoagulant therapy with or without placement of an inferior vena cava (IVC) filter. We evaluated rates of recurrent VTE and ICH following an initial ICH occurring on anticoagulant therapy. Methods A retrospective cohort study was performed using a hospital-based online medical record database (CQ2) which links ICD-9 and ICD-10 codes with prescription medication records. Cases were identified based on coding for primary brain tumors or brain metastases, after which charts were manually reviewed for a diagnosis of ICH. A blinded review of radiographic imaging was performed, and the initial ICH was categorized as either trace, measurable, or major. Measurable intracranial hemorrhages were those defined as greater than 1 mL in volume and major intracranial hemorrhages were defined as greater than 10 mL in volume, symptomatic, or requiring surgical intervention. The electronic medical record was reviewed to ascertain longitudinal anticoagulation status after the initial ICH. The primary endpoints of the study were recurrent ICH and venous thromboembolism (VTE) within 12 months from the initial ICH. Gray's test was used to compare the cumulative incidence of recurrent ICH and VTE between the groups, with death as a competing risk. Results A total of 79 patients with primary brain tumors or brain metastases and confirmed ICH were included in the study. Fifty-four patients (68.4%) restarted anticoagulation after ICH and 25 patients discontinued anticoagulation entirely. The cohorts were well-matched for tumor diagnosis, age, and comorbidities that portend an increased risk of ICH such as hypertension, chronic kidney disease, and concomitant aspirin use (Table 1). The cumulative incidence of recurrent ICH (95% CI) at one year was 6.1% (1.5 - 15.3) in the restart cohort compared to 4.2% (0.3 - 18.3) in patients who did not restart anticoagulation. Median time from anticoagulation restart to recurrent ICH was 36 days. A total of 16 of 31 patients with major ICH restarted anticoagulation and among these patients two developed subsequent ICH (cumulative incidence 14.5%, 95% CI 2.1 - 38.3). Among the 15 patients with a major ICH who did not restart anticoagulation, the cumulative incidence was 6.7% (0.3 - 27.5). Eleven of 15 patients with measurable ICH restarted anticoagulation and among these patients one subsequently developed ICH (cumulative incidence 0.1%, 95% CI 0.0 - 0.3). No recurrent ICH events were observed in 33 patients with trace initial hemorrhages regardless of restart status. All recurrent ICH events met criteria for classification as a major hemorrhage on the basis of clinical symptoms, and 30-day mortality after recurrent ICH was 100%. The cumulative incidence of recurrent VTE was significantly lower in the restart cohort compared to cohort of patients who did not restart anticoagulation (8.1 vs. 35.3, P=0.003, Figure 1). There were a total of five VTE events in the restart cohort, three deep vein thrombi (DVT) and two pulmonary emboli (PE). Two of the DVT were associated with an IVC filter. There were a total of nine VTE events in patients who did not restart anticoagulation, seven DVT and two PE. Five of the DVT were associated with an IVC filter. The two PE were both submassive events requiring ICU admission. Conclusions Recurrent VTE events are less frequent and less severe in patients who restart anticoagulation following ICH in patients with brain tumors on anticoagulation. Restarting anticoagulation after smaller ICH (trace or measurable) appears safe. However, approximately 1 in 7 patients with major initial ICH who restarted anticoagulation subsequently developed recurrent major ICH that was associated with a very high mortality rate. This raises serious questions as to the safety of restarting therapeutic anticoagulation following major hemorrhage in the setting of brain tumors. Disclosures Neuberg: Pharmacyclics: Research Funding; Madrigal Pharmaceuticals: Equity Ownership; Celgene: Research Funding. Zwicker:Quercegen: Research Funding; Daiichi: Consultancy; Seattle Genetics: Consultancy; Parexel: Consultancy; Incyte: Research Funding; Bayer: Consultancy; Portola: Consultancy.
APA, Harvard, Vancouver, ISO, and other styles
8

de Lima Oliveira, Marcelo, Angela Macedo Salinet, Ricardo de Carvalho Nogueira, Alessandro Rodrigo Belon, Wellingson Silva Paiva, Brasil Chian Ping Jeng, Manoel Jacobsen Teixeira, and Edson Bor-Seng-Shu. "The Effects of Induction and Treatment of Intracranial Hypertension on Cerebral Autoregulation: An Experimental Study." Neurology Research International 2018 (June 25, 2018): 1–8. http://dx.doi.org/10.1155/2018/7053932.

Full text
Abstract:
Background. This study aimed to analyse cerebral autoregulation (CA) during induction and treatment of intracranial hypertension (ICH) in an experimental model. Materials and Methods. Landrace and Duroc piglets were divided into mild and severe ICH groups. Four or seven millilitres of saline solution was infused into paediatric bladder catheter inserted in the parietal lobe (balloon inflation). After 1.5 h, a 3% saline solution was infused via venous catheter, and 30 min later, the bladder catheter balloon was deflated (surgery). The cerebral static autoregulation (sCA) index was evaluated using cerebral blood flow velocities (CBFV) obtained with Doppler ultrasound. Results. Balloon inflation increased ICP in both groups. The severe ICH group showed significantly lower sCA index values (p=0.001, ANOVA) after balloon inflation (ICH induction) and a higher sCA index after saline injection (p=0.02) and after surgery (p=0.04). ICP and the sCA index were inversely correlated (r=−0.68 and p<0.05). CPP and the sCA index were directly correlated (r=0.74 and p<0.05). Conclusion. ICH was associated with local balloon expansion, which triggered CA impairment, particularly in the severe ICH group. Moreover, ICP-reducing treatments were associated with improved CA in subjects with severe ICH.
APA, Harvard, Vancouver, ISO, and other styles
9

Yang, Chunhui, Yiqing Qiu, Jiali Wang, Yina Wu, Xiaowu Hu, and Xi Wu. "Intracranial hemorrhage risk factors of deep brain stimulation for Parkinson’s disease: a 2-year follow-up study." Journal of International Medical Research 48, no. 5 (December 29, 2019): 030006051985674. http://dx.doi.org/10.1177/0300060519856747.

Full text
Abstract:
Objective This study aimed to analyze the risk factors of intracranial hemorrhage (ICH) after deep brain stimulation (DBS) for idiopathic Parkinson’s disease (PD). Methods Patients who received DBS from March 2014 to December 2016 were retrospectively analyzed. The hemorrhage index was derived by combining the hemorrhagic volume and clinical manifestations of ICH. All patients with IHC were followed up for 2 years. Results Computed tomography showed 13 events of ICH in 11 patients (nine cases in the subthalamic nucleus), including eight cases with symptomatic hemorrhage (seven cases in the subthalamic nucleus). Hemorrhage was characterized by intracranial hematoma in the electrode puncture tract. Male sex and hypertension were significant risk factors for ICH. Hemorrhage in the preferred puncture side was significantly higher than that in the non-preferred puncture side. The mean hemorrhage index was 2.23 ± 0.83 in 11 patients, and no permanent neurological impairment was found during the 2-year follow-up. The effect of DBS on motor symptoms was similar in patients with and without ICH. Conclusion Male sex and hypertension are risk factors of ICH after DBS in PD. The risk of hemorrhage on the first puncture site is significantly higher than that on the second puncture site.
APA, Harvard, Vancouver, ISO, and other styles
10

Qdaisat, Aiham, Sai-Ching J. Yeung, Cristhiam H. Rojas Hernandez, Pavani Samudrala, Mona Kamal, Ziyi Li, and Adriana H. Wechsler. "Characteristics and Outcomes of Intracranial Hemorrhage in Cancer Patients Visiting the Emergency Department." Journal of Clinical Medicine 11, no. 3 (January 27, 2022): 643. http://dx.doi.org/10.3390/jcm11030643.

Full text
Abstract:
Intracranial hemorrhage (ICH) is a dreaded complication of both cancer and its treatment. To evaluate the characteristics and clinical outcomes of cancer patients with ICH, we identified all patients with ICH who visited The University of Texas MD Anderson Cancer Center emergency department between 1 September 2006 and 16 February 2016. Clinical and radiologic data were collected and compared. Logistic regression analyses were used to determine the association between clinical variables and various outcomes. During the period studied, 704 confirmed acute ICH cases were identified. In-hospital, 7-day, and 30-day mortality rates were 15.1, 11.4, and 25.6%, respectively. Hypertension was most predictive of intensive care unit admission (OR = 1.52, 95% CI = 1.09–2.12, p = 0.013). Low platelet count was associated with both in-hospital mortality (OR = 0.96, 95% CI = 0.94–0.99, p = 0.008) and 30-day mortality (OR = 0.98, 95% CI = 0.96–1.00, p = 0.016). Radiologic findings, especially herniation and hydrocephalus, were strong predictors of short-term mortality. Among known risk factors of ICH, those most helpful in predicting cancer patient outcomes were hypertension, low platelet count, and the presence of hydrocephalus or herniation. Understanding how the clinical presentation, risk factors, and imaging findings correlate with patient morbidity and mortality is helpful in guiding the diagnostic evaluation and aggressiveness of care for ICH in cancer patients.
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Intracranial hypertension (ICH)"

1

Bednaříková, Jana. "Ošetřovatelská péče u pacienta se zavedeným intrakraniálním čidlem." Master's thesis, 2011. http://www.nusl.cz/ntk/nusl-296844.

Full text
Abstract:
The goal of thesis "nursing care of patients with implemented intracranial sensor" was to analyze nursing care of patients on neurosurgical and critical care units in hospitals of the capital city of Prague. Main objectives were to understand whether there are nursing standards for "Nursing care for patients with established intracranial sensor", how nurses care for patients with increased intracranial pressure, if they have an experience with complications with patients with implemented ICP sensor and finally if they know the main complications that can potentially occur. The theoretical part describes the most common causes of brain damage and associated causes of intracranial hypertension, summarizes the types of sensors for monitoring intracranial pressure and ways of their implementation. It also deals with the specifics of nursing care associated with monitoring of intracranial hypertension. Quantitative research was chosen as methodology for this work. Data for the research was collected through anonymous questionnaire with nursing staff. The result was a finding that none of the cooperating departments have prepared a standard of nursing care for patients with an implemented ICP sensor, and that the majority of interviewed staff were not able to answer correctly if that standard in their...
APA, Harvard, Vancouver, ISO, and other styles
2

Mayer, Martin. "Biomechanické aspekty dynamiky intrakraniálního tlaku při kraniocerebrálním poranění." Doctoral thesis, 2014. http://www.nusl.cz/ntk/nusl-336134.

Full text
Abstract:
Title: Biomechanical aspects of the dynamics of intracranial pressure in traumatic brain injury Author: Ing. Martin Mayer e-mail: mayercz@seznam.cz Department: Department of Anatomy and Biomechanics Supervisor: doc. PaedDr. Karel Jelen, CSc. This PhD thesis "Biomechanical aspects of the dynamics of intracranial pressure in traumatic brain injury" is about the dynamics of intracranial pressure, particularly in relation to the external mechanical action of the patient. Severe head injury is the leading cause of death in patients under 35 years of age. Despite constantly-improving medical and nursing care only one third of patients, after recovery, regained the ability to live independently in the long term. Two-thirds of patients were severely disabled or died. The lifetime cost of such a patient who was not completely cured has been calculated to be $4,000,000. A significant consequence of craniocerebral injuries are secondary brain lesions, which among other means the rise in intracranial pressure (ICP), which can further exacerbate due to intracerebral or extracerebral causes. Therefore, the objective of the treatment is minimizing secondary injury, optimally at the phase of the primary lesion. However, realization of this requirement, about which we can say that is a conditio sine qua non, sometimes leads...
APA, Harvard, Vancouver, ISO, and other styles

Books on the topic "Intracranial hypertension (ICH)"

1

Mathews, Letha, and John Barwise. Refractory Intracranial Hypertension. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0067.

Full text
Abstract:
Intracranial pressure remains constant in adults at 10–15 mmHg under normal conditions with some fluctuations associated with respirations, coughing, sneezing, and so forth. Refractory intracranial hypertension (ICH) is defined by recurrent episodes of intracranial pressure elevation above 20 mmHg for sustained periods (10–15 min) despite medical therapy. The common causes of ICH are traumatic brain injury, brain tumors, subarachnoid hemorrhage, and brain infarction from arterial occlusion, cerebral venous thrombosis, and anoxic encephalopathy. Intracranial infections, abscesses, acute liver encephalopathy, and idiopathic ICH are also recognized causes of ICH. For the purposes of this chapter, the discussion is limited to ICH related to traumatic brain injury.
APA, Harvard, Vancouver, ISO, and other styles
2

Stocchetti, Nino, and Andrew I. R. Maas. Causes and management of intracranial hypertension. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0233.

Full text
Abstract:
Intracranial hypertension may damage the brain in two ways—it causes tissue distortion and herniation, and reduces cerebral perfusion. The many different pathologies that can result in intracranial hypertension include subarachnoid haemorrhage, spontaneous intra-parenchymal haemorrhage, malignant cerebral hemispheric infarction, and acute hydrocephalus. The pathophysiology and specific treatment of intracranial hypertension may be different and depend on aetiology. In patients with subarachnoid haemorrhage a specific focus is on treating secondary hydrocephalus and maintaining adequate cerebral perfusion pressure (CPP). Indications for surgery in patients with intracranial hypertension due to intracerebral haemorrhage (ICH) are not only related to the mass effect, but also to remove the toxic effect of extravasated blood on brain tissue. Decompressive surgery should be considered for patients with a malignant hemispheric infarction, but in order to benefit the patient this surgery should be performed within 48 hours of the onset of the stroke. Hydrocephalus may result from obstruction of cerebrospinal fluid (CSF) flow, from impaired CSF re-absorption and occasionally from overproduction of CSF. Emergency management of acute hydrocephalus can be accomplished by external ventricular drainage of CSF. More definitive treatment may be either by third ventriculostomy or implantation of a CSF shunt diverting CSF to the abdominal cavity (a ventriculoperitoneal shunt) or to the right atrium of the heart (ventriculo-atrial shunt).
APA, Harvard, Vancouver, ISO, and other styles
3

Pirson, Yves, and Dominique Chauveau. Management of intracranial aneurysms. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0310.

Full text
Abstract:
An asymptomatic intracranial aneurysm (ICA) is found by screening in about 8% of patients with autosomal dominant polycystic kidney disease (ADPKD), with a trend to cluster in some families. Though most ICAs will remain asymptomatic, a minority of them may rupture, causing subarachnoid haemorrhage (SAH). Given the grave prognosis of ICA rupture, screening and prophylactic repair of unruptured ICAs have to be considered, with the aim to identify patients with a risk of ICA rupture that exceeds the risk of a prophylactic procedure, surgical or endovascular. Relying on a decision analysis model established in the general population, widespread screening in ADPKD patients is today not recommended. However, the chapter authors advise screening in ADPKD patients with a familial history of ICA or SAH. Additional acceptable indications are high-risk occupations and patient anxiety despite adequate information. Screening is preferably performed by high-resolution, three-dimensional, time-of-flight magnetic resonance imaging. When an asymptomatic ICA is found, a recommendation for whether to intervene depends on its size, site, morphology, patient life expectancy, and general health as well as the experience of the neuroradiologist–neurosurgeon team. Since the risk of new ICAs or enlargement of an existing one is very low in those with small (< 6 mm) ICAs, conservative management is usually recommended. Elimination of tobacco use and aggressive treatment of hypertension are strongly recommended.
APA, Harvard, Vancouver, ISO, and other styles
4

O’Neal, M. Angela. A Lady with a Headache in the Second Trimester. Edited by Angela O’Neal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190609917.003.0014.

Full text
Abstract:
This case illustrates a typical presentation of idiopathic intracranial hypertension (IIH) during pregnancy. The diagnostic criteria, complications, and treatment of the condition during pregnancy are explored. The major complication of IIH is visual loss. The International Headache Society 2013 criteria for idiopathic intracranial hypertension are: that the headache should remit after the CSF pressure is in the normal range, CSF pressure is greater than 250 mm, and the majority of patients have papilledema and other symptoms, which may include visual obscurations, pulsatile tinnitus, double vision, and neck or back pain. IIH treatment includes weight control, high-volume lumbar punctures, and medications.
APA, Harvard, Vancouver, ISO, and other styles
5

Beheiry, Hossam El. Neurophysiology/Neuroprotection. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0027.

Full text
Abstract:
Intracerebral hemorrhage (ICH) is a catastrophic event that may lead to severe disability or death. It is associated with major disruption of intracranial neurophysiology and subsequent neuronal tissue injury. The etiology of ICH may be primary (e.g., hypertensive) or secondary (e.g., traumatic). Treatment options include conservative management or neurosurgical intervention in selected patients. In order to manage these challenging cases, the anesthesiologist and the intensivist should have thorough knowledge pertaining to neurophysiologic concepts of cerebral blood flow, cerebral autoregulation, and neuroprotection principles. Additionally, the specialized team managing the patient with ICH should be cognizant of the most recent evidence-based guidelines recommended by the pertinent associations.
APA, Harvard, Vancouver, ISO, and other styles
6

(Editor), Anthony Marmarou, Ross Bullock (Editor), Cees Avezaat (Editor), Alexander Baethmann (Editor), Donald Becker (Editor), Mario Brock (Editor), Julian Hoff (Editor), Hajime Nagai (Editor), Hans-J. Reulen (Editor), and Graham Teasdale (Editor), eds. Intracranial Pressure and Neuromonitoring in Brain Injury: Proceedings of the Tenth International ICP Symposium, Williamsburg, Virginia, May 25-29, 1997 (Acta Neurochirurgica Supplementum). Springer, 1998.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Whittle, Ian. Raised intracranial pressure, cerebral oedema, and hydrocephalus. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0604.

Full text
Abstract:
The brain is protected by the cranial skeleton. Within the intracranial compartment are also cerebrospinal fluid, CSF, and the blood contained within the brain vessels. These intracranial components are in dynamic equilibrium due to the pulsations of the heart and the respiratory regulated return of venous blood from the brain. Normally the mean arterial blood pressure, systemic venous pressure, and brain volume are regulated to maintain physiological values for intracranial pressure, ICP. There are a range of very common disorders such as stroke, and much less common, such as idiopathic intracranial hypertension, that are associated with major disturbances of intracranial pressure dynamics. In some of these the contribution to pathophysiology is relatively minor whereas in others it may be substantial and be a major contributory factor to morbidity or even death.Intracranial pressure can be disordered because of brain oedema, disturbances in CSF flow, mass lesions, and vascular engorgement of the brain. Each of these may have variable causes and there may be interactions between mechanisms. In this chapter the normal regulation of intracranial pressure is outlined and some common disease states in clinical neurological practice that are characterized by either primary or secondary problems in intracranial pressure dynamics described.
APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Intracranial hypertension (ICH)"

1

Singhi, Sunit. "Acute Intracranial Hypertension." In ICU Protocols, 367–70. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-0902-5_37.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Singhi, Sunit. "Acute Intracranial Hypertension." In ICU Protocols, 709–12. India: Springer India, 2012. http://dx.doi.org/10.1007/978-81-322-0535-7_89.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Muizelaar, J. P. "Induced Arterial Hypertension in the Treatment of High ICP." In Intracranial Pressure VII, 508–10. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73987-3_132.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Piper, I. R., N. M. Dearden, and J. D. Miller. "Can Waveform Analysis of ICP Separate Vascular from Non-Vascular Causes of Intracranial Hypertension?" In Intracranial Pressure VII, 157–63. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73987-3_42.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Robertson, C. S., C. F. Contant, R. K. Narayan, and R. G. Grossman. "Comparison of Methods for ICP Waveform Analysis with Intracranial Hypertension in Head-Injured Patients." In Intracranial Pressure VIII, 348–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77789-9_76.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Albin, M. S., L. Bunegin, and J. Gelineau. "ICP and CBF Reactivity to Isoflurane and Nitrous Oxide During Normocarbia, Hypocarbia and Intracranial Hypertension." In Intracranial Pressure VI, 719–24. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70971-5_137.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Tomei, G., S. M. Gaini, P. Rampini, R. Villani, R. Massei, and L. Beretta. "Some Observations on the Relationship Between Cerebral Pulse Amplitude and ICP in Patients with Intracranial Hypertension." In Intracranial Pressure VI, 286–89. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70971-5_54.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Härtl, Roger, J. Ghajar, H. Hochleuthner, and W. Mauritz. "Hypertonk/Hyperoncotic Saline Reliably Reduces ICP in Severely Head-Injured Patients with Intracranial Hypertension." In Brain Edema X, 126–29. Vienna: Springer Vienna, 1997. http://dx.doi.org/10.1007/978-3-7091-6837-0_39.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Härtl, Roger, T. F. Bardt, K. L. Kiening, A. S. Sarrafzadeh, G. H. Schneider, and A. W. Unterberg. "Mannitol Decreases ICP but Does Not Improve Brain-Tissue pO2 in Severely Head-Injured Patients with Intracranial Hypertension." In Brain Edema X, 40–42. Vienna: Springer Vienna, 1997. http://dx.doi.org/10.1007/978-3-7091-6837-0_12.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Gingrich, Kevin J. "Intracranial Hypertension (ICH)." In Essence of Anesthesia Practice, 217. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4377-1720-4.00191-6.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Intracranial hypertension (ICH)"

1

Pereira, Luiza de Lima, Ana Flávia Silva e. Sousa, Anderson Pedrosa Mota Júnior, Giovanna Martins Romão Rezende, Marcella Ferreira Ribeiro, and Carolina Ferreira Colaço. "Neurological conditions caused by microgravity." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.402.

Full text
Abstract:
Background: Since Space Tourism is closer to reality, a review of the most prevalent neurological pathologies in microgravity is needed. Objective: Review major neurological afflictions in astronauts. Methods: Research into bibliographic reviews at PubMed, using the descriptors “astronauts” and “neurological disorders” Results: Several neurological alterations, such as ataxy, intracranial hypertension (ICH), neuromuscular disorders, ocular disturbances and changes in cognitive functions were assigned to a microgravity environment. Astronauts returning from space presented ICH; being the main pathophysiology hypothesis referred to a change in the liquor dynamics as a result of venous drainage obstruction and hematoencephalic barrier. Also, gravity doesn’t act on the neurovestibular system during space flights. This phenomenon can lead to Space Motion Sickness, situation in which astronauts report balance, coordination and sight disturbances, as well as movement illusions. A subset of this syndrome, called Space Movement Disorder, may occur; which includes symptoms, such as flushing, anorexia, nausea, vomiting, dizziness and malaise. Based on electromagnetic resonance, the occurrence of problems with movement time, balance, spatial working memory and motor coordination after the return of the space crew suggests alterations in cerebellum’s function and structure, which is responsible for the coordination and the fine motor control. Conclusion: Most studies presented disruptions of the neurofunctional homeostasis, for instance, changes in functional connectivity while in rest and alterations of the white and grey matter in sensor motor, somatosensory and cognitive regions of the brain.
APA, Harvard, Vancouver, ISO, and other styles
2

Marques, Grazielle de Oliveira, Gabriel Nogueira Noleto Vasconcelos, Gabriel Rodrigues Gomes da Fonseca, Renato Sarnaglia Proença, Pablo Henrique da Costa Silva, Leonardo Martins Parca, and Ahmad Abdallah Hilal Nasser. "Malignant cerebral infarction (MCI): Review of the benefits of decompression craniectomy (DC)." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.261.

Full text
Abstract:
Background: MCI is caused by occlusion of the middle cerebral artery (MCA) or internal carotid artery. Causing large ischemias, which edema can exert a mass effect, usually between the 2nd and 5th day, resulting in intracranial hypertension, herniation and even death. Objective: Review data related to the benefits of DC, elucidating the procedure, prognosis and indications of the method. Method: Review on MEDLINE and PubMed platforms. The descriptors: “craniectomy” AND “malignant infarction”. Were selected 9 articles dated between 2016 and 2021. Results: MCI has some clinical (Youngs, NIHSS>15, neurological deterioration) and radiological predictors (Impairment> 50% of the ACM territory, midline deviation> 5mm, MRI with DWI> 145 cm3). These patient’s clinical aim is to reduce intracranial pressure (ICP), however, as a consequence of the worse prognosis in clinical therapy, there’s a possibility of intervention by DC, which is a surgical technique that relieves ICP and prevents secondary injuries. It reduces the mortality rate and increases the patient’s survival up 3x compared to clinical management, but at the expense of low quality of life. Patients ≤60 years with loss of consciousness, must have an indication for DC within 48 hours after ictus. The indication should be better evaluated and a thorough discussion with family members. Conclusion: DC minimizes injuries and the risk of herniation. However, despite decreasing mortality, it can lead to complications and poor prognosis, although it isn’t uncommon the indication for the procedure and an approach to palliative care.
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Intracranial hypertension (ICH)' for particular source types:
Journal articles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography