Log in

Relevant bibliographies by topics / Intestinal mucosal immune system / Books

To see the other types of publications on this topic, follow the link: Intestinal mucosal immune system.

Books on the topic 'Intestinal mucosal immune system'

Author: Grafiati

Published: 4 June 2021

Last updated: 12 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 23 books for your research on the topic 'Intestinal mucosal immune system.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

Å, Hanson Lars, and Svanborg-Edén Catharina, eds. Mucosal immunobiology: Cellular-molecular interactions in the mucosal immune system. Basel: Karger, 1988.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Whitehead, Richard. Mucosal biopsy of the gastrointestinal tract. 3rd ed. Philadelphia: Saunders, 1985.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Jihad, Hayek, and Federman Micheline, eds. Gastrointestinal mucosal biopsy. New York: Churchill Livingstone, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

Mucosal biopsy of the gastrointestinal tract. 3rd ed. Philadelphia: Saunders, 1985.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

A, Livolsi Virginia, ed. Mucosal biopsy of the gastrointestinal tract. 4th ed. Philadelphia: Saunders, 1990.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Mucosal biopsy of the gastrointestinal tract. 5th ed. Philadelphia: Saunders, 1997.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

1951-, MacDonald Thomas T., ed. Ontogeny of the immune system of the gut. Boca Raton, Fla: CRC Press, 1990.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

(Editor), Richard Blumberg, and Markus F. Neurath (Editor), eds. Immune Mechanisms in Inflammatory Bowel Disease (Advances in Experimental Medicine and Biology). Springer, 2006.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

Blaser, Annika Reintam, and Adam M. Deane. Normal physiology of the gastrointestinal system. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0172.

Full text

Abstract:

The gastrointestinal (GI) system is responsible for digestion and absorption, but also has important endocrine, immune and barrier functions. Additionally, the GI system plays a major role in fluid, electrolyte and acid-base balance. The GI system is regulated by complex myogenic, neural and humoral mechanisms, and, in health, these are affected by the presence of luminal nutrient, thereby modulating function of the GI system. Accordingly, GI function varies depending on whether a person is fasted or in the postprandial state. Adequate fasting and postprandial perfusion, motility and exocrine secretion are required for ‘normal’ functioning. The protective mechanisms of the GI system consist of physical (intact gut mucosa), non-immune (gastric acid, intestinal mucin, bile and peristalsis) and immune (gut-associated lymphoid tissue, GALT) elements. Disruption of GI protection is a putative mechanism underlying the development of multiple-organ dysfunction syndrome. Maintenance of GI function is increasingly recognised as an important factor underlying survival in critical illness.

APA, Harvard, Vancouver, ISO, and other styles

10

Frenkel, Joost, and Hans R. Waterham. Mevalonate Kinase Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0039.

Full text

Abstract:

Mevalonate kinase deficiency (MKD) is an autosomal recessive inborn error of isoprenoid biosynthesis, a pathway yielding sterols and nonsterol isoprenoids.In patients, the enzyme activity of mevalonate kinase is severely reduced due to mutations in the encoding gene, MVK. The substrate, mevalonate, accumulates and is elevated in blood and urine. Shortage of certain downstream products of the pathway, nonsterol isoprenoids, leads to dysregulation of the innate immune system, activation of inflammasomes, and interleukin (IL)-1 mediated inflammation.Symptoms start in early childhood with recurrent attacks of fever, vomiting, diarrhea, headache, sore throat, abdominal pain, arthralgias, painful lymphadenopathy, hepatosplenomegaly, skin rash, and mucosal ulcers. Severely affected patients have additional symptoms, such as intellectual impairment, progressive cerebellar ataxia, and tapetoretinal degeneration. Complications include intestinal obstruction, AA-amyloidosis, hemophagocytosis, and severe infection.Management of MKD is directed at controlling inflammation.

APA, Harvard, Vancouver, ISO, and other styles

11

H, Kiyono, and McGhee Jerry R, eds. Mucosal immunology: Intraepithelial lymphocytes. New York: Raven Press, 1994.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

12

Mucosal Immunology: Intraepithelial Lymphocytes (Advances in Host Defense Mechanisms). Raven Pr, 1994.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

13

Massimo, Conio, ed. Endoscopic mucosal resection. Malden, Mass: Blackwell Pub., 2007.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

14

Repici, Alessandro, Pieter Siersema, and Thierry Ponchon. Endoscopic Mucosal Resection. Blackwell Publishing Limited, 2007.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

15

Derek, Chadwick, and Goode Jamie, eds. Inflammatory bowel disease: Crossroads of microbes, epithelium, and immune systems. Chichester, UK: J. Wiley, 2004.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

16

Hanson, Lars A. Nobel Symposium No 68, Mucosal Immunobiology: Cellular-Molecular Interactions in the Mucosal Immune System (Monographs in Allergy). S Karger Pub, 1988.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

17

Foundation, Novartis. Inflammatory Bowel Disease: Crossroads of Microbes, Epithelium and Immune Systems (Novartis Foundation Symposia). Wiley, 2005.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

18

Moore, Sarah, and Kasipathy Kailasapathy. Cellular Interactions of Probiotic Bacteria with Intestinal and Immune Cells. Nova Science Publishers, Incorporated, 2017.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

19

Rogler, Gerhard. Gastrointestinal system. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0021.

Full text

Abstract:

Rheumatic diseases and diseases of the gastrointestinal (GI) tract are connected in two ways. The extraintestinal manifestations of inflammatory GI diseases such as inflammatory bowel disease affect joints in up to one-third of patients. On the other hand, several rheumatic diseases such as vasculitis or systemic lupus erythematosus (SLE) induce a wide spectrum of gastrointestinal manifestations. The GI tract constitutes a huge area in contact with the environment. It is exposed to billions of food antigens, commensal bacteria, and potential pathogens. Some of those antigens are thought to play a role in the pathogenesis of rheumatic diseases. The intestinal barrier function and the gut immune system are tightly regulated, as on one hand tolerance for food antigens and the resident commensal flora needs to be maintained, and on the other hand pathogens need to be rapidly and effectively eliminated. Non-infectious, chronic inflammatory diseases of the small and large intestine with rheumatic manifestations have been well known for decades. Among the susceptibility genes for Crohn's disease and ulcerative colitis are some that also cause susceptibility to rheumatoid arthritis or SLE, indicating a shared susceptibility and overlapping pathological mechanisms. Subsequently, similar therapeutic principles have successfully been applied in autoimmune GI and rheumatological diseases such as steroids, immunosuppressants, and anti-TNF (tumour necrosis factor) antibodies.

APA, Harvard, Vancouver, ISO, and other styles

20

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Prebiotics and probiotics in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0027.

Full text

Abstract:

Probiotics are live, non-pathogenic commensal microorganisms with beneficial effects on the host organism; they improve and/or maintain intestinal flora balance by suppressing and displacing harmful bacteria. Prebiotics are nondigestible food components that stimulate growth or activity of these beneficial intestinal bacteria. Such microorganisms form an integral part of the intestinal mucosal defence system and are important for the development and maturation of the infant#amp;#x2019;s gastrointestinal tract. Maternal ingestion of probiotics and prebiotics from dietary sources during pregnancy, or by the infant at weaning, may enhance the development and maturation of the neonatal gastrointestinal tract. Probiotic foods may also help control insulin resistance and the development of gestational diabetes.

APA, Harvard, Vancouver, ISO, and other styles

21

Bazzan, Anthony J., and Daniel A. Monti. Diet, Gut, and Brain: A New Horizon. Edited by Anthony J. Bazzan and Daniel A. Monti. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190690557.003.0001.

Full text

Abstract:

There is growing data that dietary factors have profound effects on inflammation, the gut microbiome, intestinal permeability, and the blood–brain barrier; all of which impact brain health and psychological well-being. The Western diet in particular is deleterious for both physical and cognitive/emotional health. This occurs primarily by causing inflammation in the gut and an activation of the immune system along with causing impairment in the integrity of the gut lining. This allows many reactive molecules to enter the general circulation and even cross the blood–brain barrier. Recent research advances elucidate that understanding the harmful physiological effects of certain dietary behaviors is as important as knowing the role of critical nutrients for optimal brain health. This chapter reviews current knowledge regarding diet and nutrition in the context of psychiatric disorders and brain health. Information is reviewed regarding the most appropriate dietary and nutrition approaches to support optimum brain health.

APA, Harvard, Vancouver, ISO, and other styles

22

Keshav, Satish, and Alexandra Kent. Immunology and genetics in gastrointestinal and hepatic medicine. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0196.

Full text

Abstract:

The gut has a pivotal role in immune homeostasis. It is constantly exposed to a wide array of antigens in food, and resident and consumed microorganisms. It is estimated that the number of bacterial cells in the gastrointestinal tract is tenfold greater than the number of cells in the human body. The gut needs to recognize harmful bacteria, and consequently contains the largest number of immune cells in the body. However, it must remain tolerant to commensal bacteria. Bacteria express antigens that stimulate an immunological response via the gut-associated lymphoid tissue (GALT). The GALT includes the appendix, tonsils, Peyer’s patches, and mesenteric lymph nodes. Therefore, the intestinal immune system is finely balanced between tolerance and reactivity. An example of an abnormal response that generally the individual should be tolerant to is gliadin peptides in coeliac disease. An example of excessive tolerance to an otherwise controllable infection is cryptosporidiosis, which causes diarrhoea in patients with HIV infection. The understanding of genetics in disease has progressed rapidly with the introduction of genome-wide association studies. The Welcome Trust Case Control Consortium has performed extensive research on the genetics of many illnesses, including Crohn’s disease, ulcerative colitis, Barrett’s oesophagus, oesophageal adenocarcinoma, and primary biliary cholangitis. Although these studies have increased our understanding of the molecular basis of disease, they have had little impact on clinical management. This may change as studies associate genotype and phenotype. Several gastrointestinal diseases have an etiology based on immunological or genetic aberrations, and these immunological mechanisms and genetic mutations can be utilized for diagnostic purposes. However, there is no genetic or immunological marker that is 100% specific to a disease and, consequently, the markers are used to support clinical, histological, and/or radiological findings.

APA, Harvard, Vancouver, ISO, and other styles

23

Davey, Patrick, and David Sprigings, eds. Diagnosis and Treatment in Internal Medicine. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.001.0001.

Full text

Abstract:

Diagnosis and Treatment in Internal Medicine is a new textbook, written by experts in their field, that provides succinct and authoritative guidance across the breadth of internal medicine. Diagnosis is the bedrock of management, and so how to reach a differential diagnosis of symptoms or presenting problems is a major element of the book. There is also comprehensive coverage of disorders of the body systems, including psychological aspects and palliative care. Chapters are structured so that key information can rapidly be found. Doctors need a broad perspective on health and its promotion, and there are sections addressing nutrition, lifestyle and prevention of disease. Diagnosis and Treatment in Internal Medicine is the ideal reference for doctors early in their careers in hospital medicine or primary care, and senior medical students. Sections of the book: • The approach to the patient • Assessment of symptoms and presenting problems • Cardiovascular disorders • Respiratory disorders • Intensive care medicine • Disorders of the kidney and urinary tract • Diabetes mellitus and endocrine disorders • Gastro-intestinal disorders • Disorders of the liver • Neurological disorders • Disorders of the skin • Disorders of the musculoskeletal system • Haematological disorders • Disorders of the immune system • Infectious diseases • Nutrition and its disorders • Lifestyle and environmental causes of disease • Prevention of disease • Screening for disease

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!