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Relevant bibliographies by topics / Intervertebral disk Infections

Academic literature on the topic 'Intervertebral disk Infections'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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Journal articles on the topic "Intervertebral disk Infections"

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Hovi, I., A. Lamminen, O. Salonen, and R. Raininko. "MR Imaging of the Lower Spine." Acta Radiologica 35, no. 6 (November 1994): 532–40. http://dx.doi.org/10.1177/028418519403500605.

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The findings on MR imaging of 28 patients with spinal infection and 40 patients with spinal malignant disease were compared. Spinal infections involved one to 4 vertebrae, usually (23/28) 2 vertebrae. The posterior elements were involved with certainty in 26/40 patients with malignancy but in none with infection. In the latter group, the posterior elements might have been involved in 3/28. The intervertebral disk between the infected vertebrae was involved in 26/28 patients and 21/28 had a paravertebral mass. Spinal malignancies affected the vertebrae alone in 19 patients and paravertebral extension was found in 21/40 patients. The intervertebral disk was involved only in one patient with malignancy. The differences in the distribution of the MR findings between spinal infection and spinal malignancy were highly significant (p < 0.001). The highest signal intensity of the infectious lesions on T2-weighted images was equal to or higher than that of the cerebrospinal fluid (CSF) in 26/28 patients. In contrast, the signal intensity of the malignant lesions was hypointense as compared to the CSF in 29/40 patients (p < 0.001). MR is a useful method for differentiating between infection and malignancy in the lower spine; T2-weighted images are especially valuable for differentiation.

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Ahsan, Md Kamrul, Md Masud Rana, Zahidul Haq Khan, Naznin Zaman, Md Hamidul Haque, and Abdullah Al Mahmud. "Aggressive discectomy for single level lumbar disk herniation." Bangabandhu Sheikh Mujib Medical University Journal 10, no. 3 (September 3, 2017): 135. http://dx.doi.org/10.3329/bsmmuj.v10i3.32911.

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<p class="Abstract">Aggressive open lumbar discectomy is the most commonly performed surgical procedure for patients with persistent low back and leg pain. In this retrospective study, 1,380 patients were evaluated for long-term results of aggressive discectomy for the single level lumbar disk herniation. Demographic data, surgical data, complications and reherniation rate were collected and clinical outcomes were assessed using visual analogue score (VAS), Oswestry disability index (ODI) and modified Mcnab criteria. The mean follow-up period was 28.8 months. According to the modified Mcnab criteria, the long-term results were excellent in 640 cases, good in 445 cases, fair in 255 cases, and poor in 40 cases. The mean VAS scores for back and radicular pains and ODI at the end of 2 years were 1.1 ± 1.0, 1.5 ± 0.5 and 6.6 ± 3.1% respectively. The complications were foot drop (n=7), dural tear (n=14), superficial wound infection (n=17), discitis (n=37) and reherniation (n=64). The dural tear and superficial wound infections resolved after treatment but 28 discitis patients were treated by conservatively and the remaining 9 underwent surgery. Among reherniation patients, 58 underwent revision discectomy and 4 underwent transforaminal lumbar interbody fusion and stabilization. Aggressive discectomy is an effective treatment of lumbar disk herniation and maintains a lower incidence of reherniation but leads to a collapse of disc height and in long run gives rise to intervertebral instability and accelerates spondylosis.</p>

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Troeltzsch, Matthias, Christof Birkenmaier, Christoph Schwartz, Bogdana Suchorska, Stefan Zausinger, and Alexander Romagna. "Oral Cavity Infection: An Underestimated Source of Pyogenic Spondylodiscitis?" Journal of Neurological Surgery Part A: Central European Neurosurgery 79, no. 03 (December 14, 2017): 218–23. http://dx.doi.org/10.1055/s-0037-1608823.

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Background The incidence of pyogenic spondylodiscitis is increasing; however, the source of infection often remains obscure. We analyzed predisposing factors, pathogens, and outcome of patients undergoing surgical and/or conservative treatment of spondylodiscitis with a focus on the diagnostic work-up including a comprehensive maxillofacial assessment. Patients The analysis of prognostic factors comprised comorbidities, nicotine dependence, symptom duration, and oral cavity peculiarities. After a standardized diagnostic work-up, a detailed examination of the oral cavity was also performed. The outcome analysis included assessment of the patients' clinical status. Results Forty-one patients with pyogenic spondylodiscitis were investigated of whom 24% had undergone spinal surgery within 4 weeks before the infection. A total of 29% of patients were found to have a concomitant bacterial oral cavity disease, and in 22% the definitive source of infection remained unidentified. Among the 12 patients with oral cavity infections, 10 patients had periodontitis; 8, root canal pathologies; 6, periapical lesions, and another 8 patients, caries. In 25% of these patients, typical oral cavity pathogens were found in the intervertebral disk. The prevalence of oral cavity infections was associated with a history of nicotine dependence (p = 0.003). All other analyzed comorbidities did not differ compared with patients without an oral cavity focus. Conclusion Oral cavity infections appear to be a frequent source of pyogenic spondylodiscitis, with smoking its most relevant associated risk factor. In case of an unidentified infection focus, a detailed diagnostic work-up including a mandatory maxillofacial assessment is strongly recommended.

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McCutchen, Thomas M., and Brian G. Cuddy. "Intervertebral Disk Space Infection." Neurosurgery Quarterly 11, no. 3 (September 2001): 209–19. http://dx.doi.org/10.1097/00013414-200109000-00004.

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Amir, J., and P. G. Shockelford. "Kingella kingae intervertebral disk infection." Journal of Clinical Microbiology 29, no. 5 (1991): 1083–86. http://dx.doi.org/10.1128/jcm.29.5.1083-1086.1991.

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Zeiger, Evan H., and Edward J. Zampella. "Intervertebral Disc Infection after Lumbar Chemonucleolysis: Report of a Case." Neurosurgery 18, no. 5 (May 1, 1986): 616–21. http://dx.doi.org/10.1227/00006123-198605000-00017.

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Abstract Intervertebral disc space infection can be a serious and disabling complication of any procedure that affords entry for bacteria into the susceptible disc space. Most disc space infections occur after cervical or lumbar laminectomies. Discitis has been reported after myelography, lumbar puncture, paravertebral injection, and obstetrical epidural anesthesia. A case of septic discitis occurring after intradiscal therapy with chymopapain is presented. Patients who return for evaluation of recurrent spinal pain after chemonucleolysis, especially those with paravertebral muscle spasm, should be evaluated for the possibility of disc space infection by obtaining an erythrocyte sedimentation rate, peripheral white count, differential cell count, and plain roentgenograms. Radionuclide bone scans, although not specific, may provide further objective evidence leading to the diagnosis of an intervertebral disc space infection.

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Deeb, ZL, S. Schimel, RH Daffner, AR Lupetin, FG Hryshko, and JB Blakley. "Intervertebral disk-space infection after chymopapain injection." American Journal of Roentgenology 144, no. 4 (April 1985): 671–74. http://dx.doi.org/10.2214/ajr.144.4.671.

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Fieve, Gérard, Jacques Fays, Jacques Pourrel, and Corinne Bernard. "Intervertebral Disk Space Infection Following Translumbar Aortography." Annals of Vascular Surgery 1, no. 3 (November 1986): 382–84. http://dx.doi.org/10.1016/s0890-5096(06)60139-8.

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Fieve, Gŕard, Jacques Fays, Jacques Pourrel, and Corinne Bernard. "Intervertebral disk space infection following translumbar aortography." Annals of Vascular Surgery 1, no. 3 (November 1986): 382–85. http://dx.doi.org/10.1007/bf02732578.

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Zeiger, H. E., and E. J. Zampella. "Intervertebral disc infection after lumbar chemonucleolysis." Neurosurgery 18, no. 5 (May 1986): 616???21. http://dx.doi.org/10.1097/00006123-198605000-00017.

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Dissertations / Theses on the topic "Intervertebral disk Infections"

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Fraser, Robert D. "Discitis after discography and chemonucleolysis /." Title page, table of contents and abstract only, 1986. http://web4.library.adelaide.edu.au/theses/09MD/09mdf842.pdf.

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Walters, Rebecca. "Lumbar intervertebral disc infection pathology, prevention and treatment /." Click here to access, 2006. http://thesis.library.adelaide.edu.au/public/adt-SUA20061011.164644/index.html.

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Thesis (Ph.D.) -- University of Adelaide, School of Medical Sciences, Discipline of Pathology, 2006.
Includes author's previously published papers. "March 2006" Includes bibliographical references. Also available in a print form.

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Fraser, Robert D. (Robert David). "Discitis after discography and chemonucleolysis." 1986. http://web4.library.adelaide.edu.au/theses/09MD/09mdf842.pdf.

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Walters, Rebecca Mary. "Lumbar intervertebral disc infection : pathology, prevention and treatment." 2006. http://hdl.handle.net/2440/37834.

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Discitis is a potential complication of any open or percutaneous spinal procedure which involves entry into the intervertebral disc. The infection initiates an inflammatory response which leads to endplate rupture. Although there are variations in the severity of symptoms, the main feature of discitis is severe back pain which is not relieved by rest. The infection may spontaneously resolve over time although incapacitating back pain may persist for many months. In some cases serious complications result from the spread of infection to the adjacent vertebral bodies and over time osteomyelitis will develop with resultant bone destruction and collapse. The prognosis for many patients with discitis is poor with continual disabling back pain, prolonged absence from gainful employment and inability to return to daily living activities. Clinical and experimental evidence now supports the prophylactic use of a suitable antibiotic to prevent discitis. In South Australia cephazolin is the antibiotic of choice to prevent or treat discitis due to Staphylococcus spp. While cephazolin has been shown to prevent discitis after inoculation with Staphylococcus spp. it is not universally accepted. Uncertainty exists regarding the ability of the antibiotic to enter the disc, and if it is effective in preventing and treating discitis. This is further complicated by the lack of suitable methods for detecting and measuring the concentration of cephazolin in the disc. An experimental ovine model was used to investigate ( a ) the natural progression of discitis in the growing lumbar spine ; ( b ) a technique to detect and measure the concentration of cephazolin in the disc ; ( c ) the effect of prophylaxis when dose and time of administration of cephazolin was varied ; ( d ) the effect of parenteral cephazolin after discitis was established and ( e ) the influence of health and age of the disc on prophylactic and parenteral treatment with cephazolin. In a clinical study the concentration of cephazolin was measured in degenerate human disc tissue to determine if therapeutic concentrations were achieved. The ovine studies showed that discitis had no significant effect on the development of the growing lumbar spine after one year although infection was associated with reduced disc area and height. Preventing discitis with cephazolin was reasonably successful, regardless of age and health of the disc. Timing of cephazolin administration was crucial to prevent discitis in immature animals. A high - performance liquid chromatography technique was used to measure the concentration of cephazolin in the disc. The greatest concentration of cephazolin in ovine discs was achieved 15 minutes after a bolus dose of intravenous antibiotic was administered, although detectable levels were measured for a further 2 hours. The concentration of cephazolin was not uniform across the disc with greater concentrations in the outer disc compared to the inner disc. Although there were measurable levels of cephazolin in these discs, it was ineffective at treating discitis once established. In the clinical study detectable levels of cephazolin were recovered in human discs for more than 2 hours after administering a 1 - g bolus dose. The concentration of cephazolin peaked in the human discs between 37 and 53 minutes, but in only half of the discs was the concentration of cephazolin considered therapeutic against Staphylococcus aureus. While discitis may spontaneously resolve over time, the infected disc does not recover to its original form. Furthermore, parenteral cephazolin was ineffective at preventing endplate destruction once an intradiscal inoculum was established. While this study proved cephazolin is able to enter the disc and provide reasonable protection against infection, it appears that discitis cannot be completely abolished. The timing of prophylaxis remains a critical factor to achieve therapeutic concentrations of cephazolin in the disc. Due to the serious complications that result from discitis this study supports the use of prophylactic antibiotic administered at an optimal time before the disc is violated during any spinal procedure.
Thesis (Ph.D.)--School of Medical Sciences, 2006.

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Books on the topic "Intervertebral disk Infections"

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Wheaton, Michael, Dustin Nowacek, and Zachary London. Radiculopathy and Plexopathy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0125.

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Disorders of the nerve roots and neural plexi present with pain, numbness, or weakness in the neck, back, or extremities. Although the history and physical examination provide essential diagnostic information, imaging and electrodiagnostic studies may further aid in localizing and characterizing the underlying lesion. Causes of radiculopathy include intervertebral disc herniation, spondylosis, spinal synovial cysts, infection, metastatic disease, hematoma, or infiltrative disease. The brachial and lumbosacral plexi are susceptible to trauma, structural anomalies, neoplastic infiltration, and inflammatory processes. Management of these disorders is directed at treating the underlying cause, alleviating pain, and focused physical rehabilitation.

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Book chapters on the topic "Intervertebral disk Infections"

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Bosmans, Frederik, Johan Van Goethem, and Filip M. Vanhoenacker. "Imaging of Degeneration, Inflammation, Infection, Ossification, and Calcification of the Intervertebral Disk." In The Disc and Degenerative Disc Disease, 19–62. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-03715-4_2.

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Kerwin, Sharon. "Discospondylitis and Related Spinal Infections in the Dog and Cat." In Advances in Intervertebral Disc Disease in Dogs and Cats, 161–67. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118940372.ch20.

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"Inflammatory and Infectious Disorders." In Intervertebral Disk Diseases, edited by Juergen Kraemer. Stuttgart: Georg Thieme Verlag, 2009. http://dx.doi.org/10.1055/b-0034-63545.

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"9 Spine Infections." In Management of Orthopaedic Infections, edited by Antonia F. Chen. New York, NY: Thieme Medical Publishers, Inc., 2021. http://dx.doi.org/10.1055/b-0041-181984.

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Spine infections can have significant morbidity and mortality if not identified and treated appropriately. This category of orthopaedic infections can include infections involving the epidural space (spinal epidural abscesses), vertebral column (vertebral osteomyelitis), or the intervertebral disks (diskitis). These conditions can predispose patients to significant consequences including neurologic compromise, deformity, and pain. Accurate diagnosis of spine infections is based on clinical history and examination, along with appropriate imaging and laboratory tests. Treatment of these infections entails medical management with antibiotic therapy, or, in cases with progressive neurologic deficit or deformity, surgical methods that include open decompression with correction of deformity as indicated.

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Ramrakha, Punit S., Kevin P. Moore, and Amir H. Sam. "Rheumatological emergencies." In Oxford Handbook of Acute Medicine, 659–90. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198797425.003.0011.

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This chapter details rheumatological emergencies, including acute monoarthritis, septic arthritis, crystal arthropathy, polyarthritis, rheumatoid arthritis, seronegative arthritides (spondyloarthropathies), reactive arthritis, ankylosing spondylitis, enteropathic arthritis, infections, vasculitis, systemic lupus erythematosus (SLE), Wegener’s granulomatosus and microscopic polyarteritis nodosa (PAN), cryoglobulinaemia, giant cell (temporal) arteritis, polymyalgia rheumatica (PMR), back pain, prolapsed intervertebral disc, and C₁-esterase inhibitor deficiency (angioneurotic oedema).

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Shikhare, Sumer N., and Wilfred C. G. Peh. "Pyogenic Spondylodiscitis." In Musculoskeletal Imaging Volume 2, edited by Mihra S. Taljanovic and Tyson S. Chadaz, 87–90. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190938178.003.0085.

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Chapter 85 reviews the radiologic features of pyogenic spondylodiscitis and the diagnostic value of different imaging modalities such as conventional radiography, CT, MRI, and bone scintigraphy. Pyogenic spondylodiscitis is an infection of the intervertebral disc and adjacent vertebrae and may involve the paravertebral soft tissue and epidural space. The indolent clinical presentation poses a challenge in early diagnosis and management. Pyogenic spondylodiscitis can be diagnosed based on clinical findings and blood and tissue cultures and is supported by radiological and histopathologic findings. This chapter also reviews the imaging differential diagnoses, including tuberculous spondylodiscitis and malignancies. Imaging plays a crucial role in early diagnosis of disease and in demonstrating the extent of the disease process, as treatment delay significantly reduces the cure rate and increases the complication and morbidity rates.

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