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Relevant bibliographies by topics / Insulin; Diabetes; Hypertension / Dissertations / Theses

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Dissertations / Theses on the topic 'Insulin; Diabetes; Hypertension'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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1

Al-Hussary, Nabeel A. J. "Insulin receptor binding in hypertension and non-insulin dependent diabetes mellitus." Thesis, Aston University, 1986. http://publications.aston.ac.uk/14510/.

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2

Pinkney, Jonathan Henley. "Origins of hypertension in non-insulin dependent diabetes." Thesis, University of London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.286682.

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3

Wiggam, Malcolm Ivan. "Aspects of insulin resistance in essential hypertension and insulin-dependent diabetes mellitus." Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.387971.

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4

Petrie, John Ross. "Serum insulin concentrations, insulin sensitivity, and endothelial function in essential hypertension and non-insulin-dependent diabetes mellitus." Thesis, University of Glasgow, 1997. http://theses.gla.ac.uk/2846/.

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A series of studies is described in which specific and conventional insulin immunoassays, the hyperinsulinaemic clamp technique and forearm venous occlusion plethysmography with local intra-arterial infusions have been used to investigate: the effect of insulin assay specificity on the relationships among serum insulin concentrations, insulin sensitivity, and blood pressure in diabetic and non-diabetic subjects with and without essential hypertension (Chapter 5) the effect of sustained physiological activation of the renin-angiotensin system induced by moderate dietary sodium restriction on in

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5

Courtney, Hamish. "Determinants of insulin action : physiological and therapeutic implications." Thesis, Queen's University Belfast, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343100.

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6

Fryer, Lee George Daniel. "Studies into the role of glucose transporter function in insulin resistance." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265007.

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7

Sanderson, Alison Louise. "Regulation of skeletal muscle metabolism." Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318615.

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8

Hopkinson, Helen Elizabeth. "Betaâ†2-adrenoceptor signalling and the effect of insulin." Thesis, University of Nottingham, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301070.

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9

Wong, Siu-hing, and 王少鑫. "Relationship between resistant hypertension and sodium intake in type 2 diabetes Hong Kong Chinese." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B46373445.

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10

Marinik, Elaina. "Angiotensin II receptor blockade and insulin sensitivity in overweight and obese adults with elevated blood pressure." Diss., Virginia Tech, 2012. http://hdl.handle.net/10919/37369.

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Currently, it is reported that ~65% and 34% of the U.S. population is overweight and obese, respectively. Obesity is a major risk factor for cardiovascular disease. Overweight and obese individuals are also at an increased risk of developing hypertension. Whole-body insulin sensitivity is reduced in obesity, resulting in insulin resistance and increased risk of type 2 diabetes. One possible mechanism contributing to insulin resistance in obesity hypertension is renin-angiotensin system (RAS) overactivation. The RAS exhibits vasocontricting and sodium-retaining properties, yet in vivo and in vi

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11

Woodman, Richard John. "The independent effects of purified EPA and DHA supplementation on cardiovascular risk in treated-hypertensive type 2 diabetic individuals." University of Western Australia. School of Medicine and Pharmacology, 2003. http://theses.library.uwa.edu.au/adt-WU2003.0028.

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[Formulae and special characters can only be approximated here. Please see the pdf version of the Abtract for an accurate reproduction.] Type 2 diabetes at least doubles the risk of cardiovascular disease. This can partly be explained by the increased prevalence of risk factors such as hypertension, dyslipidaemia and obesity. However, the underlying abnormality of insulin resistance and the presence of more recently identified risk factors including endothelial dysfunction, increased inflammation, and increased oxidative stress might also contribute towards the heightened cardiovascular risk

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12

Pöykkö, S. (Seppo). "Ghrelin, metabolic risk factors and carotid artery atherosclerosis." Doctoral thesis, University of Oulu, 2005. http://urn.fi/urn:isbn:9514276566.

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Abstract The increasing prevalence of metabolic syndrome and the consequent cardiovascular diseases, including atherosclerotic diseases and type 2 diabetes, are a cause of public concern worldwide. This development has stimulated an active search for novel risk factors and new candidate genes. The hormones regulating energy balance and the polymorphisms associated with them are of special interest as potential risk factors for metabolic syndrome. Ghrelin is a novel peptide hormone from stomach with strong growth hormone releasing activity. It is also able to modify glucose and insulin metaboli

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13

Mesquita, Maria do Céu Vinhas Sérgio Vilão. "Síndrome metabólica em infetados pelo vírus da imunodeficiência humana." Master's thesis, [s.n.], 2014. http://hdl.handle.net/10284/4841.

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Projeto de Pós-Graduação/Dissertação apresentado à Universidade Fernando Pessoa como parte dos requisitos para obtenção do grau de Mestre em Ciências Farmacêuticas<br>A Síndrome metabólica consiste num conjunto de fatores de risco (obesidade abdominal, hipertriglicemia, diminuição do colesterol de alta densidade, hipertensão e hiperglicemia) que predispõem o indivíduo para doenças do foro cardiovascular. O facto da literatura frequentemente associar os indivíduos infetados com o vírus da imunodeficiência humana a um risco aumentado de hipertensão, hiperlipidemia e diabetes, torna importante i

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14

Beatty, Olivia Louise. "Aspects of insulin resistance, insulin secretion and microalbuminuria in diabetic, hypertensive and non-diabetic subjects." Thesis, Queen's University Belfast, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.484068.

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15

Bouquillon, Jean. "Etude de la pression artérielle au cours de l'évolution du diabète insulino-dépendants : analyse des résultats obtenus par mesure ambulatoire de pression artérielle et FINAPRES." Lille 2, 1995. http://www.theses.fr/1995LIL2M101.

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16

IWANDZA, BRUNO-ALEXIS. "Valeur de la charge tensionnelle nocturne chez le diabetique non insulino-dependant hypertendu traite." Toulouse 3, 1993. http://www.theses.fr/1993TOU31062.

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17

PAGE, JEAN-DANIEL. "Relations entre excretion urinaire d'albumine et hypertrophie ventriculaire gauche dans une population d'hypertendus essentiels et de diabetiques non insulino-dependants hypertendus." Angers, 1990. http://www.theses.fr/1990ANGE1107.

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18

"Studies on erythrocyte ion transport systems in Hong Kong Chinese patients with essential hypertension and non-insulin-dependent diabetes mellitus." Chinese University of Hong Kong, 1993. http://library.cuhk.edu.hk/record=b5887780.

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by Mui Kin Tung.<br>Thesis (M.Phil.)--Chinese University of Hong Kong, 1993.<br>Includes bibliographical references (leaves 97-113).<br>Chapter CHAPTER 1: --- INTRODUCTION --- p.1<br>Chapter CHAPTER 2: --- LITERATURE REVIEW --- p.5<br>Chapter 2.1 --- ION TRANSPORT SYSTEMS IN HUMAN ERYTHROCYTES --- p.6<br>Chapter 2.1.1 --- "Sodium Pump (Na+,K+-ATPase)" --- p.6<br>Chapter 2.1.2 --- Passive Sodium Transport Systems --- p.9<br>Chapter 2.1.2.1 --- Sodium-potassium-chloride cotransport system --- p.9<br>Chapter 2.1.2.2 --- Sodium-lithium Countertransport --- p.13<br>Chapter 2.1.3 --- Ouabain-

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19

Saunders, Allison Jean. "Predictors and trends of adherence to blood pressure self-monitoring among hypertensive type II diabetic patients /." 2007. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&res_dat=xri:pqdiss&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&rft_dat=xri:pqdiss:MR32021.

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Thesis (M.A.)--York University, 2007. Graduate Programme in Psychology.<br>Typescript. Includes bibliographical references (leaves 91-106). Also available on the Internet. MODE OF ACCESS via web browser by entering the following URL: http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&res_dat=xri:pqdiss&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&rft_dat=xri:pqdiss:MR32021

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20

Monteiro, Ana Cláudia do Rosário. "Contribuição do tratamento com fármacos Anti-Hipertensores para a Resistência à Insulina na Diabetes Mellitus." Master's thesis, 2015. http://hdl.handle.net/10437/5994.

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Orientação : Liliana Mendes<br>A Diabetes Mellitus tipo 2 está muitas vezes associada a Hipertensão Arterial ou Pressão Arterial normal alta e está frequentemente presente nos doentes com Síndrome Metabólica. Os fármacos Anti-Hipertensores têm diferentes efeitos a nível metabólico, existindo evidência científica de que o Sistema Renina-Angiotensina-Aldosterona é fundamental para os mecanismos de Resistência à insulina. O objetivo deste trabalho é compreender os mecanismos dos vários Anti- Hipertensores, e de que forma eles contribuem para o aumento da concentração de insulina e para a resistê

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21

"The measurement of insulin resistance in the assessment of drug effects in patients with the metabolic syndrome." 1999. http://library.cuhk.edu.hk/record=b6073202.

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Lee Kwing Chin, Kenneth.<br>Thesis (Ph.D.)--Chinese University of Hong Kong, 1999.<br>Includes bibliographical references (p. 298-357).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Mode of access: World Wide Web.<br>Abstracts in English and Chinese.

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22

Kratochvílová, Simona. "Použití magnetické rezonance spektroskopie při studiu glukózového metabolismu." Doctoral thesis, 2018. http://www.nusl.cz/ntk/nusl-372610.

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Magnetic resonance spectroscopy (MRS) is a noninvasive technique that enables to follow metabolic processes in selected tissues in vivo. Recently the attention has been focused on metabolic mapping in target organs of insulin action to describe the pathophysiology of insulin resistance. The aim of our study was to present the practical application of ³¹P (phosphorus) MRS and ¹H (proton) MRS in metabolic studies of skeletal muscle in insulin resistant subjects and in subjects with impaired fasting glucose. The third study was aimed to evaluate the brain metabolism with ¹H MRS in healthy control

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23

Pena, Dias Jenny. "La contribution du récepteur B1 des kinines dans les complications diabétiques chez le rat traité au glucose, un modèle de résistance à l'insuline." Thèse, 2012. http://hdl.handle.net/1866/7049.

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Les kinines agissent sur deux types de récepteurs couplés aux protéines G, nommés B1 et B2, lesquels jouent un rôle important dans le contrôle cardiovasculaire, la nociception et l’inflammation. Nous considérons l’hypothèse que le récepteur B1 des kinines est induit et contribue aux complications diabétiques, incluant l’hypertension artérielle, les polyneuropathies sensorielles, l’augmentation du stress oxydatif vasculaire, l’inflammation vasculaire et l’obésité chez le rat traité au D-glucose (10% dans l’eau de boisson) pendant 8 ou 12 semaines. Dans ce modèle de résistance à l’insuline, nous

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24

Owen, Meredith Kohr. "Effect of coronary perivascular adipose tissue on vascular smooth muscle function in metabolic syndrome." Thesis, 2013. http://hdl.handle.net/1805/3789.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Obesity increases cardiovascular disease risk and is associated with factors of the “metabolic syndrome” (MetS), a disorder including hypertension, hypercholesterolemia and/or impaired glucose tolerance. Expanding adipose and subsequent inflammation is implicated in vascular dysfunction in MetS. Perivascular adipose tissue (PVAT) surrounds virtually every artery and is capable of releasing factors that influence vascular reactivity, but the effects of PVAT in the coronary circulation are unknown. Accordingly, the goal of this inves

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25

Ghosh, Anindya. "Mécanisme(s) d'action de l'insuline dans la prévention de l'hypertension et la progression de la tubulopathie dans le diabète : rôle de hnRNP F, Nrf2 et Bmf." Thèse, 2018. http://hdl.handle.net/1866/21837.

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