Books on the topic 'Injury repair'

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1

Lundborg, Göran. Nerve injury and repair. Edinburgh: Churchill Livingstone, 1988.

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2

Knee ligaments: Injury & repair. St. Louis: Mosby, 1993.

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3

Collision sports: Injury and repair. Oxford: Butterworth-Heinemann, 1998.

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4

Schnapp, Lynn M., and Carol Feghali-Bostwick, eds. Acute Lung Injury and Repair. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-46527-2.

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5

M, Gressner A., Heinrich P. C, and Matern S, eds. Cytokines in liver injury and repair. Dordrecht: Kluwer Academic, 2002.

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6

1949-, Lee R. C., Despa Florin, Hamann Kimm Jon, and New York Academy of Sciences., eds. Cell injury: Mechanisms, responses, and repair. New York, N.Y: New York Academy of Sciences, 2005.

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7

1939-, Daniel Dale M., Akeson Wayne H. 1928-, and O'Connor John J. 1934-, eds. Knee ligaments: Structure, function, injury, and repair. New York: Raven Press, 1990.

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8

Brock, Rita Nakashima. Soul repair: Recovering from moral injury after war. Boston: Beacon Press, 2012.

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9

Gabriella, Lettini, ed. Soul repair: Recovering from moral injury after war. Boston: Beacon Press, 2012.

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10

1922-, Chrétien Jacques, and Dusser Daniel 1951-, eds. Environmental impact on the airways: From injury to repair. New York: M. Dekker, 1996.

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11

A, Garner, O'Brien Paul E, and World Congress of Gastroenterology (9th : 1990 : Sydney, N.S.W.), eds. Mechanisms of injury, protection, and repair of the upper gastrointestinal tract. Chichester, West Sussex, England: Wiley, 1991.

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12

name, No. Daniel's knee injuries: Ligament and cartilage structure, function, injury, and repair. 2nd ed. Philadelphia, PA: Lippincott Williams & Wilkins, 2003.

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13

American Academy of Orthopaedic Surgeons. Symposium. Injury and repair of the musculoskeletal soft tissues: Workshop, Savannah, Georgia, June 1987. Park Ridge, Ill: American Academy of Orthopaedic Surgeons, 1988.

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14

Hardy, Robert George. Alterations in cadherin and catenin expression in colonic neoplasia, injury and repair: Regulation of p-cadherin transcription in the colon. Birmingham: University of Birmingham, 2003.

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15

Broken embouchures: An embouchure handbook and repair guide for players suffering from embouchure problems caused by overuse, injury, medical/dental conditions, or damaged mechanics. [United States?]: Oscar's House Press, 2002.

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16

International Conference on the Occasion of the 60th Birthday of T.M. Fliedner (1989 Schloss Reisensburg, Germany). The hemopoietic stem cell: The origin and clinical usefulness of harvested peripheral blood stem cells : repair of stem cells and their kinetic properties in recovery from marrow injury by radiation and chemicals. Ulm: Universitätsverlag Ulm, 1990.

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17

J. Singh, Parminder, and Rohit Kotnis. Injury and repair. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.0004.

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♦ Types of injury—traumatic and overuse♦ Importance of determining the energy involved in an injury♦ The relevance of mechanical load curves♦ Types of fracture healing—how and why they occur♦ Healing in articular cartilage, tendon, ligaments, peripheral nerves, and brain tissues.
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18

Nerve injury and repair. Churchill Livingstone, 1988.

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19

Nerve Injury and Repair. Elsevier, 2004. http://dx.doi.org/10.1016/b978-0-443-06711-2.x5001-x.

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20

R, Bramlage L., Edwards G. B, and Wade J. F, eds. Equine orthopaedic injury and repair. England: Equine Veterinary Journal, 1988.

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21

Spinal Cord Injury (SCI) Repair Strategies. Elsevier, 2020. http://dx.doi.org/10.1016/c2017-0-03009-4.

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22

Rossi, Filippo, and Giuseppe Perale. Spinal Cord Injury (SCI) Repair Strategies. Elsevier Science & Technology, 2019.

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23

Rossi, Filippo, and Giuseppe Perale. Spinal Cord Injury (SCI) Repair Strategies. Elsevier Science & Technology, 2019.

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24

Stevens, Robert D. Introduction: Biological Mechanisms of Injury and Repair. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0026.

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Chapter 26 is an introduction to devoted to biological mechanisms underpinning organ dysfunction and repair in critical illness, and how research has generated fundamental insights into the biology of conditions, such as sepsis and ARDS, and has suggested important new therapeutic paradigms. It also specifically addresses issues surrounding genetic susceptibility, cognitive deficiency, frailty, myocardial ischaemia, muscle wasting, sepsis-associated encephalopathy (SAE), CIP, and structural muscle alterations.
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25

Peripheral Trigeminal Nerve Injury Repair And Regeneration. W.B. Saunders Company, 2011.

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26

Jenner, Peter, Hari Shanker Sharma, and Aruna Sharma. Nanomedicine in Central Nervous System Injury and Repair. Elsevier Science & Technology Books, 2017.

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27

Ruiz, Antonio J. Chavez, and Jose M. Alvarez Mendoza. Soft Tissue: Composition, Mechanisms of Injury and Repair. Nova Science Publishers, Incorporated, 2012.

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28

Watson, Tim. The bioelectric correlates of musculoskeletal injury and repair. 1994.

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29

Jenner, Peter, Hari Shanker Sharma, and Aruna Sharma. Nanomedicine in Central Nervous System Injury and Repair. Elsevier Science & Technology Books, 2017.

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30

Yang, Bin, Cheng Yang, and Ying Wang, eds. Innate Immunity in Kidney Injury, Repair and Fibrosis. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-979-9.

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31

Yang, Bin, Cheng Yang, and Ying Wang, eds. Innate Immunity in Kidney Injury, Repair and Fibrosis. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-979-9.

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32

Nanomedicine in Central Nervous System Injury and Repair. Elsevier, 2017. http://dx.doi.org/10.1016/s0074-7742(17)x0007-x.

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33

Tsai, Ching-Wei, Sanjeev Noel, and Hamid Rabb. Pathophysiology of Acute Kidney Injury, Repair, and Regeneration. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0030.

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Acute kidney injury (AKI), regardless of its aetiology, can elicit persistent or permanent kidney tissue changes that are associated with progression to end-stage renal disease and a greater risk of chronic kidney disease (CKD). In other cases, AKI may result in complete repair and restoration of normal kidney function. The pathophysiological mechanisms of renal injury and repair include vascular, tubular, and inflammatory factors. The initial injury phase is characterized by rarefaction of peritubular vessels and engagement of the immune response via Toll-like receptor binding, activation of macrophages, dendritic cells, natural killer cells, and T and B lymphocytes. During the recovery phase, cell adhesion molecules as well as cytokines and chemokines may be instrumental by directing the migration, differentiation, and proliferation of renal epithelial cells; recent data also suggest a critical role of M2 macrophage and regulatory T cell in the recovery period. Other processes contributing to renal regeneration include renal stem cells and the expression of growth hormones and trophic factors. Subtle deviations in the normal repair process can lead to maladaptive fibrotic kidney disease. Further elucidation of these mechanisms will help discover new therapeutic interventions aimed at limiting the extent of AKI and halting its progression to CKD or ESRD.
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34

(Editor), A. M. Gressner, P. C. Heinrich (Editor), and S. Matern (Editor), eds. Cytokines in Liver Injury and Repair (Falk Symposium). Springer, 2002.

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35

Soul Repair: Recovering from Moral Injury after War. Beacon Press, 2018.

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36

Brock, Rita Nakashima, and Gabriella Lettini. Soul Repair: Recovering from Moral Injury after War. Beacon Press, 2012.

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37

Payne, Russell A., and Elias B. Rizk. Axillary Nerve Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0024.

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Axillary nerve injury has been associated with sports injuries, especially those involving anterior shoulder dislocation. The nerve injury leads to weakness of the deltoid and teres minor muscles, which impairs abduction and external rotation of the arm at the shoulder. Electrodiagnostic studies are helpful for determining extent of reinnervation and recovery after injury. In the absence of clinical or electrodiagnostic signs of recovery 3 to 6 months after injury, it is appropriate to offer surgical exploration. The options for surgical repair include direct nerve repair, nerve grafting, and nerve transfer. In appropriately selected individuals, outcomes are favorable.
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38

Schnapp, Lynn M., and Carol Feghali-Bostwick. Acute Lung Injury and Repair: Scientific Fundamentals and Methods. Humana Press, 2016.

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39

Lundborg, Goran. Nerve Injury and Repair: Regeneration, Reconstruction, and Cortical Remodeling. 2nd ed. Churchill Livingstone, 2005.

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40

Bellusci, Saverio, and Mohammad K. Hajihosseini, eds. FGF10 in Development, Homeostasis, Disease and Repair After Injury. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-828-0.

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41

Schnapp, Lynn M., and Carol Feghali-Bostwick. Acute Lung Injury and Repair: Scientific Fundamentals and Methods. Humana, 2018.

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42

Schnapp, Lynn M., and Carol Feghali-Bostwick. Acute Lung Injury and Repair: Scientific Fundamentals and Methods. Humana Press, 2016.

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43

Hayes, Gary. Traumatic Brain Injury Self-Repair: Relearning How to Remember. Independently Published, 2020.

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44

Chretien, Jacques, and Daniel Dusser. Environmental ImPatt on the Airways: From Injury to Repair. Taylor & Francis Group, 1996.

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45

Hanna, Amgad S., Lisa M. Block, and A. Neil Salyapongse. Emergent Nerve Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0027.

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Injuries to peripheral nerves must be assessed and treated in a thorough and timely manner to achieve optimal outcomes. Physical examination is the cornerstone in diagnosing acute nerve injuries and includes careful inspection as well as precise motor and sensory testing. Nerve conduction studies and electromyography are often more useful in the setting of delayed presentation. Microsurgical repair techniques differ for clean versus ragged lacerations, and resultant nerve gaps will require a conduit or graft to achieve the necessary tension-free closure. The surgeon and patient should be prepared for a lengthy postoperative course and possible complications as the nerve regenerates and function returns.
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46

Socolovsky, Mariano, Rafael Torino, and Leandro Pretto Flores. Facial Nerve Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0026.

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This chapter focuses on the clinical and surgical management of facial nerve palsy that occurs as a consequence of injury during resection of a vestibular schwannoma. If the facial nerve is damaged during cerebellopontine angle (CPA) tumor resection, a first attempt to repair it at the skull base should be made. Because this is commonly infeasible, a nerve transfer—scheduled as an elective procedure after the patient has completely recovered from the resection procedure—is mandatory. Hemihypoglossal, masseter, and cross-facial nerve transfers are the techniques most widely used. The authors’ preferred technique is hemihypoglossal nerve transfer, and the surgical technique is described. By contrast, when the facial nerve is preserved during surgery, but complete facial palsy develops afterward, postoperative rehabilitation should be started and continued for up to 1 year. If, however, facial palsy persists beyond 1 year, then the patient should be offered the option of a nerve transfer.
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47

Lin, Michael, and Erin S. Williams. Open Globe Repair. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0030.

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Penetrating eye injury in the pediatric patient is an emergency that requires careful management by the anesthesiologist to prevent further eye damage by the potential extrusion of vitreous fluid. Additionally, since these injuries are due to trauma, the patients typically are not adequately fasted for surgery. Thus prevention of aspiration of gastric contents must also be considered during the perioperative management of the child with an open globe injury. Depending on the risk of aspiration, the anesthesiologist must determine whether to use succinylcholine, a nondepoloarizing muscle relaxant, or no muscle relaxant at all, prior to laryngoscopy. A delicate balance must exist to achieve a smooth induction of anesthesia, blunted response to airway stimulation during laryngoscopy and intubation, and prevention of aspiration. During the perioperative period, the pediatric anesthesiologist can employ a variety of methods to limit the potential for increased intraocular pressure in the patient with penetrating eye trauma.
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48

Duncan, Kelli A. Estrogen Effects on Traumatic Brain Injury: Mechanisms of Neuroprotection and Repair. Elsevier Science & Technology Books, 2015.

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49

(Editor), Geoffrey J. Bellingan, and Geoffrey J. Laurent (Editor) ;, eds. Acute Lung Injury: From Inflammation to Repair (Biomedical and Health Research). Ios Pr Inc, 2001.

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50

(Editor), Robert A. Pedowitz, John J. O'Connor (Editor), and Wayne H. Akeson (Editor), eds. Daniel's Knee Injuries: Ligament and Cartilage Structure, Function, Injury, and Repair. 2nd ed. Lippincott Williams & Wilkins, 2003.

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