Academic literature on the topic 'Injury'

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Journal articles on the topic "Injury"

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Bisaccia, Michele, Luigi Piscitelli, Giovanni Colleruoli, Giuseppe Rinonapoli, Cristina Vicente, Gabriele Falzarano, Antonio Medici, Luigi Meccariello, Olga Bisaccia, and Auro Caraffa. "EPIDEMIOLOGY OF INJURIES AND DISEASES DUE TO OVERUSE IN RUGBY: OBSERVATIONAL STUDY OF THE PLAYERS OF “CUS PERUGIA RUGBY”." International Journal of Surgery and Medicine 2, no. 3 (2016): 167. http://dx.doi.org/10.5455/ijsm.rugby-injury.

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Cassar-Pullicino, Victor N., and Antonio Leone. "Imaging in paediatric spinal injury." Trauma 19, no. 1_suppl (August 30, 2017): 50–74. http://dx.doi.org/10.1177/1460408617725781.

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Background Paediatric spinal injury is rare and exhibits many unique features. Attending clinicians and radiologists often lack knowledge, expertise and experience in dealing with a potential injury to the paediatric spine. Within the paediatric age range itself there are different age-dependent mechanisms that can injure the paediatric spine. Moreover, the anatomical features and degree of osseous maturity of the developing paediatric spine determine the biomechanical characteristics which promote unique patterns of spinal injury in each paediatric age group. Methods An expert illustrated narrative review of the literature. Results Multiple factors make the imaging interpretation of the injured paediatric spine challenging. Each imaging modality has strengths and weaknesses in depicting spinal anatomy which vary with the type of spinal injury and age of the paediatric patient. Conclusions Attending doctors need to be familiar with the imaging appearances of the normal paediatric spine, its normal variants as well as the imaging features characteristics of paediatric spinal injury seen on radiographs, computed tomography and magnetic resonance imaging.
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NGUTTER, CRYSTAL, and CATHERINE DONNELLY. "Nitrite-Induced Injury of Listeria monocytogenes and the Effect of Selective Versus Nonselective Recovery Procedures on Its Isolation from Frankfurters." Journal of Food Protection 66, no. 12 (December 1, 2003): 2252–57. http://dx.doi.org/10.4315/0362-028x-66.12.2252.

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Sodium nitrite (NaNO2) is used as a curing agent in frankfurters. Although previous studies have documented the bacteriostatic abilities of NaNO2 toward Listeria monocytogenes, few if any studies have been conducted that consider the possibility of sublethal injury to L. monocytogenes by exposure to NaNO2. The goals of this study were to determine whether NaNO2 has the ability to injure L. monocytogenes, to determine whether nitrite injury is reversible, and to compare the recovery of L. monocytogenes from frankfurters containing nitrite with Listeria repair broth (LRB) and University of Vermont modified Listeria enrichment broth (UVM). NaNO2, when used at concentrations of 100 and 200 ppm, was found to injure L. monocytogenes. The injury was completely reversible, or growth of uninjured Listeria occurred in LRB when injury was between 98.5 and 98.7%. However, total recovery was not observed in LRB when injury exceeded 99%. UVM was unable to reverse the effects of nitrite-injured L. monocytogenes. With respect to time, inoculum, and meat type, LRB was found to be consistently superior to UVM at recovering L. monocytogenes from frankfurters. Nitrite injury might be a factor influencing detection and recovery of L. monocytogenes from frankfurters.
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Porterfield, Dunk, Wesley J. Everman, and John W. Wilcut. "Soybean Response to Residual and In-Season Treatments of Trifloxysulfuron." Weed Technology 20, no. 2 (June 2006): 384–88. http://dx.doi.org/10.1614/wt-05-033r.1.

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Experiments were conducted from 1998 to 2000 at Rocky Mount, NC, in weed-free environments to determine soybean tolerance to preplant (PP) applications of trifloxysulfuron and the potential for trifloxysulfuron applied preemergence (PRE) and postemergence (POST) to cotton to injure soybean grown in rotation the following year. Trifloxysulfuron at 3.75 and 7.5 g ai/ha applied PP 2 wk before seeding injured conventional soybean less than 5%, whereas no injury was observed when seeding was delayed 4 or 6 wk after PP treatment. No injury to sulfonylurea-resistant soybean (SR) was observed for any treatment. Soybean yields were not influenced by trifloxysulfuron treatment. Cotton injury was 7% or less with trifloxysulfuron applied PRE or POST at 3.75 and 7.5 g/ha. Trifloxysulfuron at 15 g/ha PRE or POST injured cotton a maximum of 14 to 18%. Trifloxysulfuron did not reduce cotton lint yields regardless of method or rate of application. Both conventional and SR soybean were not injured nor were yields influenced by trifloxysulfuron applied PRE or POST the previous year to cotton.
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Miotke, Sam, Kathleen S. Romanowski, Tina L. Palmieri, David G. Greenhalgh, and Soman Sen. "626 Causes and Injury Patterns of Elderly Burn Patients." Journal of Burn Care & Research 41, Supplement_1 (March 2020): S158—S159. http://dx.doi.org/10.1093/jbcr/iraa024.250.

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Abstract Introduction Older patients are a particularly vulnerable population with respect to burn injuries. The American Burn Association has invested resources in improving burn care and burn prevention within this age group, recognizing the difficulties in both the identification and subsequent modification of risk factors. The National Electronic Injury Surveillance System (NEISS) is maintained by the Consumer Product Safety Commission to surveil for injury trends and is populated with data from a representative group of approximately 100 emergency departments in the United States. This database could serve as a useful tool to evaluate burn injury patterns in elderly patients with an eye toward targeted prevention measures. Methods The NEISS was queried for patients aged 55 years and older with a diagnosis of scald, thermal, chemical, electrical, or unspecified burn injuries for the years 2008 - 2017. Each injury was classified by body part(s) burned, causative product, gender, and age group of patient (55–64, 65–74, 75–84, 85 and older). The ten most commonly burned sites as well as the ten most common causative products were determined for each gender and age group and in aggregate. Weighted estimates from the NEISS data were utilized to calculate injury numbers across the population. Results The most common etiology of burn injuries for all patients aged 55 and older was hot water. Men were most commonly injured by gasoline, a product which did not rank in the top ten for women in any age group. Additionally, within each age group, gasoline was among the top 4 products that was associated with burn injuries in men. Cookware was the second most common cause of burn injury among women aged 55 and older and was the sixth most common cause among men. For women, the combination of cookware and ranges/ovens was among the top 2 products associated with burn injury within each age group. The most commonly injured body part for all patients was the face. This held across most age and gender groupings except for women aged 75 to 84 (upper trunk), and women 85 and older (foot). Men were more likely to injure their hands than women. Conclusions The most common cause of burn injuries in older adults is hot water, and the most common body part affected is the face. Women are more likely to be injured by cookware and ranges/ovens. Men are more likely to be injured secondary to gasoline and are more likely to injure their hands. Applicability of Research to Practice NEISS data could be useful in the development of targeted prevention campaigns, such as focused education about the dangers of hot liquids, cooking, and accelerants.
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Wang, Jing, Mokarram Hossain, Ajitha Thanabalasuriar, Matthias Gunzer, Cynthia Meininger, and Paul Kubes. "Visualizing the function and fate of neutrophils in sterile injury and repair." Science 358, no. 6359 (October 5, 2017): 111–16. http://dx.doi.org/10.1126/science.aam9690.

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Neutrophils have been implicated as harmful cells in a variety of inappropriate inflammatory conditions where they injure the host, leading to the death of the neutrophils and their subsequent phagocytosis by monocytes and macrophages. Here we show that in a fully repairing sterile thermal hepatic injury, neutrophils also penetrate the injury site and perform the critical tasks of dismantling injured vessels and creating channels for new vascular regrowth. Upon completion of these tasks, they neither die at the injury site nor are phagocytosed. Instead, many of these neutrophils reenter the vasculature and have a preprogrammed journey that entails a sojourn in the lungs to up-regulate CXCR4 (C-X-C motif chemokine receptor 4) before entering the bone marrow, where they undergo apoptosis.
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Sevick, Johnathan L., Bryan J. Heard, Ian KY Lo, John A. Randle, Cyril B. Frank, Nigel G. Shrive, and Gail M. Thornton. "Are re-injured ligaments equivalent mechanically to injured ligaments: The role of re-injury severity?" Proceedings of the Institution of Mechanical Engineers, Part H: Journal of Engineering in Medicine 232, no. 7 (July 2018): 665–72. http://dx.doi.org/10.1177/0954411918784088.

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The consequences of ligament re-injury have received limited attention. Although the mechanical properties of injured ligaments improve over time, these properties are never fully recaptured, rendering these injured ligaments susceptible to re-injury. Previous injury is a significant risk factor for recurrent injury, and this re-injury can result in longer absence from activity than the initial injury. A rabbit medial collateral ligament model was used to compare mechanically re-injured right medial collateral ligaments to injured left medial collateral ligaments. Two groups of different re-injury severity were investigated: ‘minor’ re-injury comparing transection re-injured right medial collateral ligaments to transection injured left medial collateral ligaments; ‘major’ re-injury comparing gap re-injured right medial collateral ligaments to transection injured left medial collateral ligaments. Initial injuries for both groups were right medial collateral ligament transections 1 week before re-injury. After 5–6 weeks of healing, mechanical testing was performed to determine (dimensionally) cross-sectional area; (structurally) medial collateral ligament laxity, failure load, and stiffness; and (materially) cyclic creep strain and failure stress. Because we wanted to evaluate whether the mechanical properties of re-injured ligaments were equivalent or, at least, no worse than injured ligaments, we used equivalence/noninferiority testing. This approach evaluates a research hypothesis of equivalence, rather than difference, and determines whether comparisons are ‘statistically equivalent’, ‘noninferior’, or ‘potentially inferior’. Transection re-injured and gap re-injured ligaments were ‘statistically equivalent’ structurally to transection injured ligaments. Transection re-injured ligaments were ‘noninferior’ both materially and dimensionally to transection injured ligaments. Gap re-injured ligaments were ‘potentially inferior’ both materially and dimensionally to transection injured ligaments. Two differences between the re-injuries, which affect healing, may explain the mechanical outcomes: the presence or lack of healing products and the proximity of ligament ends at the time of re-injury. Our findings suggest that (in the short term) there is a severity of re-injury below which there is no additional disadvantage to the healing process, mechanical behaviour, and resulting potential for re-injury.
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Hamm, Robert J., Brian R. Pike, Dianne M. O'Dell, and Bruce G. Lyeth. "Traumatic brain injury enhances the amnesic effect of an NMDA antagonist in rats." Journal of Neurosurgery 81, no. 2 (August 1994): 267–71. http://dx.doi.org/10.3171/jns.1994.81.2.0267.

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✓ The authors have examined the effect of experimental traumatic brain injury on the amnesia produced by the N-methyl-D-aspartate (NMDA) antagonist MK-801. Rats were either subjected to a moderate level of fluid-percussion injury or prepared for injury but not injured (“sham injury”). Nine days following injury or sham injury, the rats were injected either with saline (sham/saline group, nine rats; injured/saline group, nine rats) or with 0.1 mg/kg of MK-801 (sham/MK-801 group, nine rats; injured/MK-801 group, eight rats) 30 minutes before being trained on a passive-avoidance task. Twenty-four hours later, the rats were tested for retention of the passive-avoidance task. Results revealed that the low dose of MK-801 did not significantly affect retention of the passive-avoidance task in the sham-injured group. In injured animals, administration of MK-801 produced a profound amnesia in contrast to the sham-injured animals treated with MK-801 and the injured animals treated with saline. To further investigate this enhanced sensitivity to the amnesic effects of MK-801 exhibited by the injured animals, nine injured and eight sham-injured rats were injected with 0.3 mg/kg of MK-801 15 minutes before injury. Results indicated that the animals treated with MK-801 before injury did not significantly differ from the sham-injured animals in retention of the passive-avoidance task. In addition, test results in the animals treated with MK-801 before injury and reinjected with MK-801 before passive-avoidance testing did not differ from those in untreated injured animals reinjected with saline before passive-avoidance testing. These findings indicate that MK-801 treatment before injury prevented the enhanced sensitivity to MK-801-induced amnesia that follows traumatic brain injury.
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Deng, Kangli, Brian M. Balog, Dan Li Lin, Brett Hanzlicek, Qi-Xiang Song, Hui Zhu, and Margot S. Damaser. "Daily bilateral pudendal nerve electrical stimulation improves recovery from stress urinary incontinence." Interface Focus 9, no. 4 (June 14, 2019): 20190020. http://dx.doi.org/10.1098/rsfs.2019.0020.

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Stress urinary incontinence (SUI) in women is strongly associated with childbirth which injures the pudendal nerve (PN) and the external urethral sphincter (EUS) during delivery. Electrical stimulation (ES) can increase brain-derived neurotrophic factor (BDNF) expression in injured neurons, activate Schwann cells and promote neuroregeneration after nerve injury. The aim of this study was to determine if more frequent ES would increase recovery from SUI in a rat model. Forty female Sprague–Dawley rats underwent either sham injury or pudendal nerve crush (PNC) and vaginal distention (VD) to establish SUI. Immediately after injury, electrodes were implanted at the pudendal nerve bilaterally. Each injured animal underwent sham ES, twice per week ES (2/week), or daily ES of 1 h duration for two weeks. Urethral and nerve function were assessed with leak point pressure (LPP), EUS electromyography and pudendal nerve sensory branch potential (PNSBP) recordings two weeks after injury. LPP was significantly increased after daily ES compared to 2/week ES. EUS neuromuscular junction innervation was decreased after injury with sham ES, but improved after 2/week or daily ES. This study demonstrates that daily bilateral ES to the pudendal nerve can accelerate recovery from SUI. Daily ES improved urethral function more than 2/week ES.
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Samtani, Jayesh B., John B. Masiunas, and James E. Appleby. "White Oak and Northern Red Oak Leaf Injury from Exposure to Chloroacetanilide Herbicides." HortScience 45, no. 4 (April 2010): 696–700. http://dx.doi.org/10.21273/hortsci.45.4.696.

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Previous research by the authors found simulated acetochlor (with atrazine) and s-metolachlor drift to white oak at the leaf unfolding stage caused loss of interveinal tissues (leaf tatters). Reports of leaf tatters in the landscape and nursery settings are more common on white oak (Quercus alba L.) than on northern red oak (Quercus rubra L.). Our objectives were to determine if white and northern red oak differed in susceptibility to chloroacetanilide herbicides, if injury varied between chloroacetanilide herbicides, and if adding atrazine increased leaf injury. Two-year-old seedlings at the leaf unfolding stage were treated with acetochlor, s-metolachlor, and dimethenamid-P alone or combined with atrazine at 1%, 10%, and 25% of the standard field use rate. Within 6 days, all chloroacetanilides at 10% and 25% field use rates, alone or combined with atrazine, caused leaf tatter injury in both species. Acetochlor, s-metolachlor, and dimethenamid-P caused a similar type of leaf injury. Atrazine did not cause loss of leaf tissues or increase injury from chloroacetanilides. At 1% field use rate, only acetochlor, acetochlor + atrazine, and dimethenamid-P caused leaf injury to northern red oaks. The white oaks were not injured by all of the chloroacetanilide treatments at 1% field use rate. The northern red oaks were slightly more susceptible to chloroacetanilides compared with the white oaks. A second study found acetochlor only injured northern red oak when applied at the leaf unfolding stage and only at 25% of field use rate. Acetochlor at 1% field use rate did not injure red oak. Research is needed to explain the greater frequency of leaf tatters on white oaks than on northern red oaks in the landscape and to develop strategies to avoid tree injury.
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Dissertations / Theses on the topic "Injury"

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Carter, Jarrod W. "Compressive cervical spine injury : the effect of injury mechanism on structural injury pattern and neurologic injury potential /." Thesis, Connect to this title online; UW restricted, 2002. http://hdl.handle.net/1773/8010.

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Cates, Darcy Leanne. "Knowledge of Nonsuicidal Self-Injury in Populations That Self-Injure." TopSCHOLAR®, 2010. http://digitalcommons.wku.edu/theses/206.

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Archived data was utilized for the present study which examined knowledge about non-suicidal self-injury, or NSSI, in individuals who engage in various degrees of the behavior and those who do not self-injure. Knowledge about NSSI was measured in three groups of respondents: those with no history of self-injurious behavior (no NSSI group), those with more limited experience with NSSI who reported 1-30 incidences of NSSI (limited NSSI group), and those with an extensive history (extensive NSSI group) who reported over 30 incidences of NSSI. To measure knowledge, participants were asked level of agreement with myths and facts about NSSI using Jeffery and Warm’s (2002) knowledge measure. It was hypothesized that the knowledge base would be higher in individuals with more extensive histories of NSSI. Further, individuals with limited histories of NSSI were predicted to have more knowledge than those who have never self-injured. Additionally, this study also hypothesized that the individual item response will vary; depending on extent of NSSI behavior. Group mean scores on the measure were analyzed for differences using a one-way analysis of covariance (ANCOVA) while controlling for the differing group demographic variables of age, sexual orientation, and education level. Results indicated that individuals who have more extensive histories of NSSI evidenced higher mean scores on the measure when controlling for age, sexual orientation and educational level. Individuals with limited histories of NSSI evidenced lower mean scores, and those with no history of NSSI evidenced the lowest scores. In regard to individual item response, items were correlated with seven levels of NSSI (no NSSI, one incident of NSSI, 2-4 incidences, 5-10 incidences, 11-20 incidences, 21-30 incidences and more than 30 incidences). It was found that accuracy was significantly correlated with degree of self-injurious behaviors, with the exception of one item. This item and three additional items also produced weak correlations with other items on the measure. Each item is discussed with regard to group item performance and possible deletions in order to strengthen the measure. Overall, the results of this investigation supported the reliability and validity of the Jeffery and Warm (2002) knowledge measure for use with individuals who self-injure. Results are discussed in relation to the need for accurate knowledge about NSSI, the importance of refining and strengthen the measure for this use, and additional research directions.
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Canter, Laura. "Looking at athlete's attitudes toward injury : reporting injury /." [St. Lucia, Qld.], 2007. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19828.pdf.

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O'Neill, Daniel Fulham. "Injury contagion: the effect of injury on teammates' performance." Thesis, Boston University, 2005. https://hdl.handle.net/2144/32813.

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Thesis (Ed.D.)--Boston University
PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.
Background: Season-ending injuries, particularly those to the anterior cruciate ligament (ACL), continue at a high-rate in many sports, particularly ski racing. Although many factors are thought to contribute to this injury rate in both genders, no study has looked at possible psychological influences. Hypothesis: There is a form of post-traumatic stress disorder (PTSD) that affects athletes after seeing someone in their own sport sustain a serious injury. The result could be a decrease in performance both on and off the mountain. In the worst-case scenario, this change in tactics could result in injury to themselves, representing an "injury contagion". Study Design: Case-control study; Level of evidence, 3. Methods: Students at four Eastern ski academies were studied for 3 consecutive years (2002-2005). The subjects ranged in age from 13-19 and consisted of 277 men (60.3%) and 182 women (39.7% ). When a subject sustained a season-ending injury, a peer group of that subject was tested for both psychological and performance effects. The results of this testing was compared to previous baseline testing and a similarly constructed control group from an academy without such an injury. Results: There were twelve season-ending injuries sustained over the three-year period. Significant data results were obtained from one aspect of the psychological testing of the peer group. There was a trend toward a possible "injury contagion" i.e. injury to a member of the peer group soon after injury to a teammate. Conclusions: Although the results of this study were inconclusive in establishing the existence of an "injury contagion", there were data to establish a psychological affect on some athletes after injury to one of their teammates. Clinical Relevance: Although a teammate's injury did not measurably effect performance and only showed mild evidence of a possible injury contagion, there were significant psychological affects noted in female subjects. School personnel should be trained in basic counseling techniques specific to this problem.
2031-01-01
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Fishel, Marissa. "Collegiate Student- Athletes Knowledge of Injury and Injury Prevention." University of Cincinnati / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1380613284.

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Akin, Faith W., and Owen D. Murnane. "Vestibular Consequences of Mild Traumatic Brain Injury (Blast Injury)." Digital Commons @ East Tennessee State University, 2008. https://dc.etsu.edu/etsu-works/1940.

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Koenes, Jeanne. "After the injury : what is it like for injured nurses /." ON-CAMPUS Access For University of Minnesota, Twin Cities Click on "Connect to Digital Dissertations", 2001. http://www.lib.umn.edu/articles/proquest.phtml.

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Gillespie, William J. "A comparison of golf swing kinematics among non-injured, rotator cuff injury-repaired, and rotator cuff injury-non-repaired golfers." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ32115.pdf.

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Palmer-Green, Debbie S. "Injury epidemiology and injury prevention in English youth rugby union." Thesis, University of Bath, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.520932.

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Carter-Allison, Samantha Natalie. "Diagnosis threat and injury beliefs after mid traumatic brain injury." Thesis, King's College London (University of London), 2015. https://kclpure.kcl.ac.uk/portal/en/theses/diagnosis-threat-and-injury-beliefs-after-mid-traumatic-brain-injury(c6ba3d52-13d9-46ea-aeee-d34ed2e43943).html.

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Background: Diagnosis threat is a form of stereotype threat, where individuals with a history of mild traumatic brain injury (mTBI) have shown performance decrements on cognitive tasks, owing to negative expectancies around cognitive ability elicited by cues in the environment. This study systematically reviews experimental studies to gauge the presence/absence of an effect of diagnosis threat on neuropsychological task performance in mTBI. It also investigates whether methodological variation and methodological quality contribute to variation in study findings. Method: A systematic search of four online databases (Medline, PyscINFO, SportDISCUS, PsycEXTRA) was conducted to identify diagnosis threat studies that employed an experimental paradigm. Neuropsychological test outcomes were extracted, along with information on inclusion criteria, mTBI diagnostic criteria, participant characteristics and study design. Methodological quality was assessed using modified Scottish Intercollegiate Guidelines Network (SIGN) criteria. Results: A total of nine studies were identified. Evidence for diagnosis threat was found, although there was considerable heterogeneity across study results. The most robust finding was the impact of diagnosis threat on the cognitive domain of attention/working memory. No clear associations between methodological variation, methodological quality and study outcome were noted. Conclusions: The review found evidence for diagnosis threat, although the strength of this effect may be smaller than previously thought. Although there was heterogeneity across elements of study design, there was no obvious relationship between these factors and outcome. However, the substantial variation makes comparison difficult. These issues are similar to findings in other examinations of stereotype threat. Further research is needed to replicate findings and add clarity to the impact of diagnosis threat on both objective and subjective measures, and to further investigate the role of possible moderating variables. A more formal meta-analysis in the area may also be helpful to clarify findings in the research field. Future studies should aim to create established operational definitions and outcomes to improve consistency and comparability between studies.
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Books on the topic "Injury"

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Aird, Catherine. Injury Time. Bath: Chivers, 1996.

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Exall, Gordon. Personal injury. 2nd ed. London: Longman, 1992.

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Yoganandan, Narayan, Alan M. Nahum, and John W. Melvin, eds. Accidental Injury. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1732-7.

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Otte, Andreas. Whiplash Injury. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-28356-7.

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Clark, Robert S. B., and Patrick Kochanek, eds. Brain Injury. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1721-4.

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Nahum, Alan M., and John W. Melvin, eds. Accidental Injury. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4757-2264-2.

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Whitfield, Peter C., Elfyn O. Thomas, Fiona Summers, Maggie Whyte, and Peter J. Hutchinson, eds. Head Injury. Cambridge: Cambridge University Press, 2009. http://dx.doi.org/10.1017/cbo9780511576515.

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Nahum, Alan M., and John W. Melvin, eds. Accidental Injury. New York, NY: Springer New York, 2002. http://dx.doi.org/10.1007/978-0-387-21787-1.

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Li, Guohua, and Susan P. Baker, eds. Injury Research. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-1599-2.

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Inn, No 1. Serjeants', ed. Personal injury. Welwyn Garden City: EMIS Professional Publishing, 2001.

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Book chapters on the topic "Injury"

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Chaubey, Vikas P., Kevin B. Laupland, Christopher B. Colwell, Gina Soriya, Shelden Magder, Jonathan Ball, Jennifer M. DiCocco, et al. "Burn Injury, Inhalation Injury." In Encyclopedia of Intensive Care Medicine, 412–15. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_378.

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Ramirez, Amelie G., Ian M. Thompson, and Leonel Vela. "Injury." In The South Texas Health Status Review, 109–17. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-00233-0_10.

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Citerio, G., C. Giussani, Hugo Sax, Didier Pittet, Xiaoyan Wen, John A. Kellum, Angela M. Mills, et al. "Injury." In Encyclopedia of Intensive Care Medicine, 1248. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1755.

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Robertson, Leon S. "Injury." In Handbook of Prevention, 343–60. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5044-6_15.

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Laing, Gordon S. "Injury." In Accident and Emergency Medicine, 50–68. London: Springer London, 1988. http://dx.doi.org/10.1007/978-1-4471-1608-0_3.

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Laverack, Glenn. "Injury." In A–Z of Public Health, 97–100. London: Macmillan Education UK, 2015. http://dx.doi.org/10.1007/978-1-137-42617-8_39.

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Galli, Nick. "Injury." In Psychosocial Health and Well-Being in High-Level Athletes, 85–100. Abingdon, Oxon; New York, NY: Routledge, 2019.: Routledge, 2019. http://dx.doi.org/10.4324/9781351210942-9.

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Manley, Nathan R., and George O. Maish. "Deceleration Injury: Blunt Aortic Injury." In Clinical Algorithms in General Surgery, 641–43. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-98497-1_157.

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Lee, Yong Seuk. "Combined Injury—Posterolateral Rotational Injury." In Knee Arthroscopy, 169–78. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-8191-5_15.

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Aharonson-Daniel, Limor. "Injury Profiling." In Injury Research, 269–80. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-1599-2_13.

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Conference papers on the topic "Injury"

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Mattson, C., K. Okamura, M. Kiselevach, and B. J. Smith. "Cell Injury Cluster Progression During Ventilator-Induced Injury." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7238.

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Mihalik, Jason P., Avinash Chandran, Jacob R. Powell, Patricia R. Roby, Brian D. Stemper, Alok S. Shah, Steve Rowson, et al. "7.14 Do head injury biomechanics predict injury recovery?" In 6th International Conference on Concussion in Sport. BMJ Publishing Group Ltd and British Association of Sport and Exercise Medicine, 2024. http://dx.doi.org/10.1136/bjsports-2023-concussion.102.

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Nahum, Alan M., and Mark A. Gomez. "Injury Reconstruction: The Biomechanical Analysis of Accidental Injury." In International Congress & Exposition. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 1994. http://dx.doi.org/10.4271/940568.

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Pillai, Nikhil, Abani Patra, and Ehsan Esfahani. "Modeling Post-Impact Injury Propagation in Traumatic Brain Injury." In ASME 2016 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2016. http://dx.doi.org/10.1115/detc2016-60444.

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In this paper, we investigate the effect of mechanical deformation during original impact on the propagation of bleeds during traumatic brain injury (TBI). For this purpose, we have developed a numerical framework that considers Magnetic Resonance Images (MRI) of a rat subjected to TBI modelled using controlled cortical impact (CCI). Using the MRI images of first day of impact a solid model of brain is developed and strains during impact are estimated using the finite element tool LSDyna. It was observed that the actual propagation of blood obtained from day 14 MRI data closely resembles the one developed by solving a time dependent advection equation with advection rates proportional to the strain estimates during impact from LSDyna. This numerical framework holds promise that with proper calibration and validation it can be used to predict the possible propagation of blood post-impact and therefore may be used to inform treatment protocols for such patients.
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Ali, S., N. Chima, and J. E. Parker. "Oxymorphone Related Inhalation Lung Injury - Beyond Vaping Lung Injury." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1876.

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Dolle, Jean-Pierre, Rene Schloss, and Martin L. Yarmush. "Simulating Diffuse Axonal Injury During Traumatic Brain Injury Events." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53414.

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Traumatic Brain Injuries (TBI) affect up to 1.5 million people annually within the United States with as many as 250,000 being hospitalized and 50,000 dying [1]. TBI events occur when the brain experiences a sudden trauma such as a rapid acc/deceleration. These events produce high inertial forces that result in a shearing or elongation of axons (commonly known as Diffuse Axonal Injury [2].
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Moor, Joel. "Interaction between herbicide injury and thrips injury in peanut." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.112022.

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Zaremski, Jason L., Marissa Pazik, Cooper W. Dean, Niran Vijayaraghavan, Nicholas P. Fethiere, Kevin W. Farmer, and MaryBeth Horodyski. "292 Throwing elbow injury prevention: forearm flexor injury association with medial elbow ulnar collateral ligament injury." In IOC World Conference on Prevention of Injury & Illness in Sport 2021. BMJ Publishing Group Ltd and British Association of Sport and Exercise Medicine, 2021. http://dx.doi.org/10.1136/bjsports-2021-ioc.269.

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Yan, Pan. "Cause Investigation of Volleyball Injury and Post-injury Treatment Strategies." In 2016 5th International Conference on Social Science, Education and Humanities Research. Paris, France: Atlantis Press, 2016. http://dx.doi.org/10.2991/ssehr-16.2016.48.

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Nishizawa, E. E., and D. J. Williams. "PERIVASCULAR CAROTID ARTERY INJURY LEAD TO ATHEROSCLEROSIS IN HYPERCHOLESTEROLEMIC RABBITS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643085.

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We have previously shown that endothelial injury results when clotted blood is placed perivascularly around a rabbit carotid artery. This injury was manifested by loss of endothelial cells and adherence of leukocytes and platelets. When similar injury was induced in hypercholesterolemic rabbits, medial lesions as well as intimal proliferation was observed. We have extended these studies and have found that these lesions contain high concentrations of cholesterol (200-300 mg/gm protein compared to 40-80 mg/gm protein in controls) suggesting lipid infiltration. The uniformity of lipid accumulation along the injured tissue segment, which was measured as intimal thickening of serial sections, suggests that these lesions are not focal and that the injury was initiated by the perivascular blood rather than by mechanical injury. The contralateral non-injured vessels showed no lesions (0-2% of the total intima + media area) and normal cholesterol content. When the serum cholesterol level was decreased from about 1200 mg/dl to 450 mg/dl by use of drugs such as Colestid, the intimal proliferation was 8.9% compared to the untreated controls, which were around 36%. Aspirin, which did not lower serum cholesterol, had no effect on intimal proliferation and arterial cholesterol accumulation. These studies suggest that this model might be useful for evaluating antiatherosclerotic drugs.
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Reports on the topic "Injury"

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Terpsma, Ryan, and Chad Hovey. Blunt Impact Brain Injury using Cellular Injury Criterion. Office of Scientific and Technical Information (OSTI), October 2020. http://dx.doi.org/10.2172/1716577.

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CRYOLIFE INC MARIETTA GA. Cellular and Tissue Injury during Nonfreezing Cold Injury and Frostbite. Fort Belvoir, VA: Defense Technical Information Center, September 1992. http://dx.doi.org/10.21236/ada256387.

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Brockbank, K. G. Cellular and Tissue Injury During Nonfreezing Cold Injury and Frostbite. Fort Belvoir, VA: Defense Technical Information Center, April 1993. http://dx.doi.org/10.21236/ada265546.

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Curran, Patrick. Cellular and Tissue Injury During Nonfreezing Cold Injury and Frostbite. Fort Belvoir, VA: Defense Technical Information Center, September 1993. http://dx.doi.org/10.21236/ada270883.

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CRYOLIFE INC MARIETTA GA. Cellular and Tissue Injury During Nonfreezing Cold Injury and Frostbite. Fort Belvoir, VA: Defense Technical Information Center, January 1992. http://dx.doi.org/10.21236/ada244809.

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Taylor, Paul A., Candice Frances Cooper, Andrei A. Vakhtin, and Corey C. Ford. Correlation of Injury Simulation with Clinical Assessment of Traumatic Brain Injury. Office of Scientific and Technical Information (OSTI), June 2018. http://dx.doi.org/10.2172/1529057.

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Taylor, Paul, Candice Cooper, Andrei Vakhtin, and Corey Ford. Correlation of Injury Simulation with Clinical Assessment of Traumatic Brain Injury. Office of Scientific and Technical Information (OSTI), April 2022. http://dx.doi.org/10.2172/1865722.

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Griffing, Bill. Injury reduction at Fermilab. Office of Scientific and Technical Information (OSTI), June 2005. http://dx.doi.org/10.2172/879008.

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Cioffi, William G., David G. Burleson, Basil A. Pruitt, and Jr. Leukocyte Responses to Injury,. Fort Belvoir, VA: Defense Technical Information Center, November 1993. http://dx.doi.org/10.21236/ada277949.

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Stuhmiller, James H. Overuse Injury Assessment Model. Fort Belvoir, VA: Defense Technical Information Center, February 2004. http://dx.doi.org/10.21236/ada423332.

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