Academic literature on the topic 'Infectious disease'

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Journal articles on the topic "Infectious disease"

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Soda, Kenji, Mitsuhiro Kamakura, and Katsuhiko Kitamura. "Infectious Disease Fight Against Infectious Diseases." Journal of Epidemiology 6, no. 3sup (1996): 61–66. http://dx.doi.org/10.2188/jea.6.3sup_61.

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HIRAI, KANJI. "Correspondence for infectious diseases under infectious diseases '99 "infectious disease new law" . III. Typology of infectious disease Concept of disease and correspondence. 6. New infectious disease Infectious disease caused by unknown pathogen." Nihon Naika Gakkai Zasshi 88, no. 11 (1999): 2196–204. http://dx.doi.org/10.2169/naika.88.2196.

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SAGARA, HIROKO. "Correspondence for infectious diseases under infectious diseases '99 "infectious disease new law". III. Typology of infectious disease Concept of disease and correspondence. 2.2 Class 2 infectious disease." Nihon Naika Gakkai Zasshi 88, no. 11 (1999): 2147–53. http://dx.doi.org/10.2169/naika.88.2147.

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NOMURA, TAKASHI. "Correspondence for infectious diseases under infectious diseases '99 "infectious disease new law" . III. Typology of infectious disease Concept of disease and correspondence. 5. Designated infectious diseases." Nihon Naika Gakkai Zasshi 88, no. 11 (1999): 2189–95. http://dx.doi.org/10.2169/naika.88.2189.

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YANAGIHARA, ITARU. "Correspondence for infectious diseases under infectious diseases '99 "infectious disease new law" . III. Typology of infectious disease Concept of disease and correspondence. 3. Class 3 infectious disease Intestinal hemorrhagic Escherichia coli infectious disease." Nihon Naika Gakkai Zasshi 88, no. 11 (1999): 2154–59. http://dx.doi.org/10.2169/naika.88.2154.

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TAKEMURA, HIROMU. "Correspondence for infectious diseases under infectious diseases '99 "infectious disease new law" . III. Typology of infectious disease Concept of disease and correspondence. 4. Class 4 infectious disease. 2 Vancomycin resistant enterococcus infectious disease." Nihon Naika Gakkai Zasshi 88, no. 11 (1999): 2166–72. http://dx.doi.org/10.2169/naika.88.2166.

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Sanjay Kalra, Suneet Kumar Verma, and Jatin Ahuja. "Infectious disease and diabetes." Journal of the Pakistan Medical Association 73, no. 9 (August 15, 2023): 1914–15. http://dx.doi.org/10.47391/jpma.23-64.

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In this communication, we provide a bird’s eye view of the various ways in which infectious diseases intersect with diabetes. We list the ways in which infectious diseases can influence glucose homoeostasis and diabetes management, and explores how diabetes care is associated with infections and infection management. This is especially important for health care providers in regions with a high burden of infectious disease. Keywords: Bacteria, communicable disease, fungus, protozoa, virus.
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Hart, C. A. "Infectious diseases: meningococcal disease." Western Journal of Medicine 173, no. 2 (August 1, 2000): 125–28. http://dx.doi.org/10.1136/ewjm.173.2.125.

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KASHIWAGI, SEIZABURO. "Infectious diseases 1. Infectious disease. Recent topics." Nihon Naika Gakkai Zasshi 89, no. 3 (2000): 565–69. http://dx.doi.org/10.2169/naika.89.565.

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SAITO, ATSUSHI. "Emerging infectious diseases and insensible bacillus infectious diseases. Emerging infectious diseases. Legionella infectious disease." Nihon Naika Gakkai Zasshi 86, no. 11 (1997): 2039–45. http://dx.doi.org/10.2169/naika.86.2039.

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Dissertations / Theses on the topic "Infectious disease"

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DeMars, Kathleen R., and Nathaniel A. MD Justice. "Pneumonia masking the presentation of incomplete Kawasaki disease." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/asrf/2019/schedule/76.

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Presentation: A 3 month-old male is referred for admission with a 2-day history of fever, having been diagnosed with pneumonia and prescribed a cephalosporin on the previous day. A blood culture obtained at that time is positive for coagulase negative Staphyloccocus. On exam, he is ill-appearing. He has bilateral conjunctivitis that spares the limbus, non-exudative pharyngitis, and a polymorphic truncal rash. There is no appreciable cervical lymphadenopathy or extremity involvement. A chest x-ray demonstrates a round infiltrate of the left upper lobe, and initial labs reveal a white blood count of 17.5, a C-reactive protein (CRP) of 23.9 mg/dL, and a normal comprehensive metabolic panel. His positive blood culture is deemed a contaminant, and antibiotic coverage for community-acquired pneumonia is given with ampicillin. Diagnostic evaluation: On day 5 of illness, his fevers persist despite broadened antibiotic coverage. Further work-up has ruled out viral respiratory pathogens and Epstein-Barr virus as a cause of persistent fevers. Incomplete Kawasaki disease is suspected due to continued fevers, the presence of three clinical criteria, and further increase in his CRP. He lacks other supplemental laboratory criteria, so an echocardiogram is obtained that shows mild dilation of the left anterior descending artery (LAD) of indeterminate significance. A repeat echocardiogram 2 days later reveals progressive dilation of left main coronary artery (LMCA), LAD, and right coronary artery (RCA). Diagnosis: Dilation of the LAD and RCA confirm a diagnosis of incomplete Kawasaki disease. Within 48 hours of treatment with IVIG and high-dose aspirin, the patient is afebrile with resolving symptoms and a declining CRP. He is discharged on the 9th day of illness on low dose aspirin and a cephalosporin to complete an antibiotic course for concurrent pneumonia. Conclusion & Discussion: This case illustrates the importance of maintaining a high index of suspicion for an incomplete presentation of Kawasaki disease, particularly among infants. The American Heart Association’s guidelines were updated in 2017 to improve recognition of incomplete Kawasaki disease, particularly among infants who are more likely to have an incomplete presentation, abnormalities of the coronary arteries, and a delayed diagnosis. The key to this patient’s diagnosis was the presence of a bilateral conjunctivitis that spared the limbus. A bilateral, non-exudative conjunctivitis that spares the limbus has been recognized as a feature suggestive of Kawasaki disease for the better part of four decades; our review of the literature suggests this feature is highly specific to the diagnosis of Kawasaki disease.
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陳奇志 and Qizhi Chen. "Analysis of infectious disease data." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2000. http://hub.hku.hk/bib/B31239985.

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Chen, Qizhi. "Analysis of infectious disease data /." Hong Kong : University of Hong Kong, 2000. http://sunzi.lib.hku.hk/hkuto/record.jsp?B21841627.

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Ashraf, Shamaila. "Studies on infectious bursal disease virus." Connect to resource, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1124124381.

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Thesis (Ph. D.)--Ohio State University, 2005.
Title from first page of PDF file. Document formatted into pages; contains xvi, 216 p.; also includes graphics (some col.). Includes bibliographical references (p. 180-216). Available online via OhioLINK's ETD Center
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Pressley, Meagan E. "Infectious Disease Models for the Zebrafish." Fogler Library, University of Maine, 2004. http://www.library.umaine.edu/theses/pdf/PressleyME2004.pdf.

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Baillie, John Kenneth. "Statistical genetics in infectious disease susceptibility." Thesis, University of Edinburgh, 2013. http://hdl.handle.net/1842/17620.

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Death from infectious disease is common heritable, and in many cases a consequence of the host response, rather than direct effects of the pathogen. Since the host response in sepsis is orchestrated by the transmission of a variety of signals, both intra-cellular and inter-cellular, with which we have at least some capacity to intervene, it follows that it should be possible to prevent death through pharmaceutical modulation of inflammatory cascades. So far, it is not. The best candidate therapy for sepsis, activated protein C, failed to live up to initial promise and was ultimately withdrawn from the market in dismal failure. The premise of the work presented here is that a different approach – to develop an understanding of the host response at a genomic level – may yield more tractable insights, specifically into the problem of host susceptibility to influenza, a heritable cause of death in otherwise healthy people and a significant global threat. Since the sequencing of the human genome, it has become possible to identify genomic loci underlying host susceptibility to disease using genome-wide association studies (GWAS), best exemplified by the Wellcome Trust Case Control Consortium. This new technology creates substantial new challenges. The genetic markers associated with a phenotype are rarely causative, frequently in poorly-understood intergenic regions, and tend to have small effect sizes, such that tens or even hundreds of thousands of subjects must be recruited to have sufficient power to detect them. It is therefore not straightforward to translate these genotype-phenotype associations into useful understanding of the role of genes and gene products in disease pathogenesis. Attempts to overcome these challenges in order to discover genomic loci underlying individual susceptibility to infection form the core of this thesis. Ultimately these efforts converge with the development of a new computational method to detect phenotype-associated loci from genome-wide association studies (GWAS) using co-expression at regulatory regions of the genome.
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Ahmad, Iqra. "Infectious Disease: An agent-based simulation." Digital Commons @ East Tennessee State University, 2013. https://dc.etsu.edu/honors/91.

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One of the primary reasons for studying infectious diseases is to improve control and ultimately eradicate the infection from the population (Keeling 2008). An agent base model was designed to observe the spread of disease and the effect of vaccination using the program known as StarLogo- TNG. Experiments were designed, not having a particular disease in mind, and simulations were run to determine the effects of different variables on the vaccination process of a certain population. Goals included observation of how infections spread and how vaccination plays a role in various situations such as, root of the infection, population size, number of providers for vaccination, and the longevity of vaccination.
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Mekaru, Sumiko Rachel. "Environmental risk factors in infectious diseases: studies in waterborne disease outbreaks, Ebola, and Lyme disease." Thesis, Boston University, 2013. https://hdl.handle.net/2144/11144.

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Thesis (Ph.D.)--Boston University
The resurgence of infectious diseases and global climate change's potential impact on them has refocused public health's attention on the environment's role in infectious disease. The studies in this dissertation utilize the increased availability of satellite image-derived data sets with fine temporal and geographic granularity and the expansion of epidemiologic methods to explore the relationship between the environment and infectious disease in three settings. The first study employed a novel study design and analytic methods to investigate the hypothesis that heavy rainfall is an independent risk factor for waterborne disease outbreaks (WBDOs). We found that a location experiencing a heavy rainfall event had about half the odds of a WBDO two or four weeks later than did a location without a heavy rainfall event. The location-based case-crossover study design utilized in this study may help to expand the research methods available to epidemiologists working in this developing field. The second study employed a location-based case-crossover study design to evaluate standardized differences from historic average of weekly rainfall in locations with a recorded introduction of Ebola into a human. For each 1.0 unit z-score decrease in total rainfall, the odds of an Ebola introduction three weeks later increased by 75%. Given the severity of Ebola outbreaks and the dearth of knowledge about indicators of increased risk, this finding is an important step in advancing our understanding of Ebola ecology. The third study used GIS methods on remote sensing data to estimate the association between peridomestic forest/non-forest interface within 100, 150, 250 meters and Lyme-associated peripheral facial palsy (LAPFP) among pediatric facial palsy patients. After adjustment for sex, age, and socio-economic status, children with the highest level of forest edge in the three radii of analysis had 2.74 (95% CI 1.15, 6.53), 4.58 (1.84, 11.41), and 5.88 (2.11, 16.4) times the odds of LAPFP compared to children with zero forest edge in those radii. This study is the first to examine environmental risk factors for LAPFP. Each of these studies advances the techniques used to investigate environmental risk factors for infectious disease through study design, case definition, data used, or exposure definitions.
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Rudd, Matthew Francis, and mikewood@deakin edu au. "Virulence determinants of infectious bursal disease virus." Deakin University. School of Biological and Chemical Sciences, 2003. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20050825.103742.

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The very virulent (vv) pathotype of infectious bursal disease virus (IBDV) has spread rapidly throughout Europe, Asia, and the Middle East. Although Australia is currently unaffected, there remains the potential for incursion of an exotic isolate. The aim of this study was to identify putative virulence determinants of IBDV to facilitate the development of improved diagnostic assays for detection and characterisation of vvIBDV isolates. Sequencing of Indonesian vvIBDV Tasik94 revealed a unique substitution [ A¨S222] in the hypervariable region (HVR) of viral protein (VP) VP2, which did not appear to impinge on virulence or antigenicity. Phylogenetic analyses indicated that Tasik94 was closely related to Asian and European vvIBDV strains. Extensive alignment of deduced protein sequences across the HVR of VP2 identified residuesI242 I256 and I294 as putative markers of the vv phcnotype. Comparison of the pathology induced by mildly-virulent Australian IBDV 002/73 and Indonesian vvIBDV Tasik94, revealed that histological lesions in the spleen, thymus and bone marrow were restricted to Tasik94-infected birds, suggesting the enhanced pathogenicity of vvIBDV might be attributed to replication in non-bursal lymphoid organs. The biological significance of the VP2 HVR in virulence was assessed using recombinant viruses generated by reverse genetics. Both genomic segments of Australian IBDV 002/73, and recombinant segment A constructs in which the HVR of 002/73 was replaced with the corresponding region of either tissue culture-adapted virus or vvIBDV (Tasik94), were cloned behind T7 RNA polymerase promoter sequences. In vitro transcription/translation of each construct resulted in expression of viral proteins. Co-transfection of synthetic RNA transcripts initiated replication of both tissue culture-adapted parental and recombinant viruses, however attempts to rescue non-adapted viruses in specific-pathogen-free (SPF) chickens were unsuccessful. Nucleotide sequence variation in the HVR of VP2 was exploited for the development of a new diagnostic assay to rapidly detect exotic IBDV isolates, including vvIBDV, using reverse transcription polymerase chain reaction (RT-PCR) amplification and Bmrl restriction enzyme digestion. The assay was capable of differentiating between endemic and exotic IBDV in 96% of 105 isolates sequenced to date.
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Hofmann, Mathias. "Statistical Models for Infectious Disease Surveillance Counts." Diss., lmu, 2007. http://nbn-resolving.de/urn:nbn:de:bvb:19-66012.

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Books on the topic "Infectious disease"

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University, Open, ed. Infectious disease. Milton Keynes: Open University, 2003.

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University, Open, ed. Infectious disease. Milton Keynes: Open University, 2003.

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T, Begg Norman, and Gillespie S. H, eds. Infectious disease. Oxford: Blackwell Science, 1996.

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University, Open, ed. Infectious disease. Milton Keynes: Open University, 2003.

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University, Open, ed. Infectious disease. Milton Keynes: Open University, 2003.

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University, Open, ed. Infectious disease. Milton Keynes: Open University, 2003.

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University, Open, ed. Infectious disease. Milton Keynes: Open University, 2003.

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Hamish, McKenzie, ed. Infectious disease. Chichester: Wiley-Blackwell, 2009.

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University, Open, ed. Infectious disease. Milton Keynes: Open University, 2003.

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University, Open, ed. Infectious disease. Milton Keynes: Open University, 2003.

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Book chapters on the topic "Infectious disease"

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Kanki, Phyllis J. "Infectious Diseases infectious disease , Introduction." In Encyclopedia of Sustainability Science and Technology, 5378–82. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0851-3_927.

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Schneider, Samantha L., and Laurie L. Kohen. "Infectious Disease." In Practical Guide to Dermatology, 257–88. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-18015-7_13.

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Chao Hu, Charles Kung, Heather Dolman, and Patrick McGann. "Infectious Disease." In Surgical Critical Care and Emergency Surgery, 86–94. Chichester, UK: John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781118274231.ch8.

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Ebell, Mark H. "Infectious Disease." In Evidence-Based Diagnosis, 209–31. New York, NY: Springer New York, 2001. http://dx.doi.org/10.1007/978-1-4757-3514-7_10.

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Sarkar, Jusmeen. "Infectious Disease." In Canine and Feline Anesthesia and Co-Existing Disease, 248–63. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2014. http://dx.doi.org/10.1002/9781118834305.ch14.

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Hirsch, Jeffrey G. "Infectious Disease." In Oklahoma Notes, 261–78. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-4010-5_15.

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Pamir, M. Necmettin. "Infectious Disease." In Neurosurgery, 241–47. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-79565-0_7.

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Goltra, Peter S. "Infectious Disease." In Medcin, 509–15. New York, NY: Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4612-2286-6_73.

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Elliott, Peter G. "Infectious Disease." In MRCGP, 184–90. London: Springer London, 1989. http://dx.doi.org/10.1007/978-1-4471-1710-0_15.

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Tsen, Lawrence C., and Errol R. Norwitz. "Infectious Disease." In Anesthetic and Obstetric Management of High-Risk Pregnancy, 381–402. New York, NY: Springer New York, 2004. http://dx.doi.org/10.1007/0-387-21572-7_26.

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Conference papers on the topic "Infectious disease"

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Jang, Beakcheol, Miran Lee, Myeong hwi Kim, Hyun jung Kim, Hoon Yoo, and Jong Wook Kim. "Infectious disease infection index information system." In 2019 IEEE International Conference on Consumer Electronics (ICCE). IEEE, 2019. http://dx.doi.org/10.1109/icce.2019.8661844.

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Vasilevich, F. I., S. A. Shemyakova, and N. V. Esaulova. "VETERINARY AND MEDICAL SIGNIFICANCE OF HORSEFLY (TABANIDAE). REVIEW." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. All-Russian Scientific Research Institute for Fundamental and Applied Parasitology of Animals and Plant – a branch of the Federal State Budget Scientific Institution “Federal Scientific Centre VIEV”, 2023. http://dx.doi.org/10.31016/978-5-6048555-6-0.2023.24.133-137.

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The article provides information on the harmful effect of horseflies (Diptera, Tabanidae) as a midge component and a vector (carrier) transmitting pathogens of infectious and parasitic diseases including zoonosis. Horseflies are harmful to animals and humans in places of their abundance. Horseflies cause significant economic losses to livestock. With an intense attack of horseflies, individual areas of the skin of animals represent a continuous bleeding surface. Horsefly saliva inserted into a wound at the time of the bite is very toxic and allergenic causing a local inflammatory process and general intoxication of the body. The insects are of particular danger as vectors transmitting pathogens of animals and humans. The role of horseflies in the transmission of tularemia in natural foci of this infection has been proven. The sources of horsefly infection are primarily various small mammals including water rats. Horseflies are equally important as carriers of the anthrax pathogen. Causative agents of Coxiella burnetti infection, blackleg, pasteurellosis, and other infections have been isolated from horseflies. Horseflies are involved in the transmission of Trypanosoma evansi in horses and camels, anaplasmosis in cattle, Theileria cervi in reindeer, and equine infectious anemia virus.
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HELIALDO SOUSA DE OLIVEIRA FILHO, FRANCISCO, PRISCILA DOURADO EVANGELISTA, PRISCILA GARCIA CÂMARA CABRAL TAVARES, LUIZ VALÉRIO COSTA VASCONCELOS, MARINA PINTO ROCHA, ANA CAROLINA CAVALCANTE MENDONÇA, ADAH SOPHIA RODRIGUES VIEIRA, et al. "LEPROSY: INFECTIOUS DISEASE MIMICKING RHEUMATIC DISEASES." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileiro de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.1806.

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Panova, I. V., O. E. Chernov, and V. S. Novozhilova. "PERSONS COVID‑19 HAVE SUFFERED CONNECTION OF DISEASE WITH PROFESSION EXAMINATION FEATURES." In The 17th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2023). FSBSI «IRIOH», 2023. http://dx.doi.org/10.31089/978-5-6042929-1-4-2023-1-352-356.

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The causative agent of COVID‑19 infectious disease, determined the structure of occupational morbidity of medical workers in 2020‑2022. The role of COVID‑19 as an infection associated with the provision of medical care with the risk of infection even with a single contact has been established. The main criteria for linking the COVID‑19 disease with the profession of medical workers have been determined: contact (mainly aerosol) with the source of infection (a sick person); terms of the incubation period (from 2 to 14 days); the presence of symptoms of the clinical picture of COVID‑19, characteristic concomitant diseases and complications; determined medical expert outcome: death or severe consequences.
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Efremova, E. A., E. A. Udaltsov, O. M. Bonina, and I. M. Zubareva. "SEASONAL NEMATODIRUS INFECTION IN THE EDILBAEV SHEEP IN THE KULUNDA STEPPE." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. VNIIP – FSC VIEV, 2024. http://dx.doi.org/10.31016/978-5-6050437-8-2.2024.25.130-134.

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The research purpose was to study seasonal characteristics of nematodirosis manifesting in the Edilbaev sheep introduced into the Kulunda Steppe of the Altai Region. Fecal samples collected from spontaneously infected animals were studied by coproovoscopic method per Fülleborn. The Nematodirus infection in sheep in different seasons of the year was characterized in view of incidence rates (prevalence, %) and infection intensity (average number of eggs per gram of feces (egg/g) per one studied and infected animal). A total of 240 fecal samples were examined. It was found that annual dynamics of the epizootic process was typical for Nematodirus. The minimum Nematodirus infection rate in sheep was recorded in July (prevalence = 10.0%) with an increase to 68.8% observed in August and maximum values of 95.6% recorded in October. Similar results were detected for the intensity of infectious process of nematodirosis, namely, the maximum number of eggs in fecal samples per examined and infected animal was recorded in autumn, 193.1 and 235.9 egg/g, accordingly, and the minimum in April, 2.0 and 20.6 egg/g, accordingly. The summer-autumn seasonal pattern of the infectious process of nematodirosis in sheep was due to the maximum contamination of land with helminth eggs during this period.
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Marangoni, Igor Parada. "Hospital morbidity and mortality profile due to pneumonia in Brazil between 2010 and 2020." In II INTERNATIONAL SEVEN MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/homeinternationalanais-080.

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Abstract Pneumonia is a lower respiratory tract infection that affected 489 million people worldwide in the year 2019 according to data from the Global Burden of Diseases (GBD) 2019 study,1,2. The disease was still responsible for over 2.49 million deaths in the year of the survey, surpassing other diseases such as tuberculosis and HIV, making pneumonia the leading infectious cause of death worldwide
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Nugraheni, Arwinda, Ani Margawati, Firdaus Wahyudi, Dea Amarilisa Adespin, and Bambang Hariyana. "Determinant Factors on Stunting Incidence among Children Age 6-24 Months, Pemalang, Central Java: A Case Study." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.03.28.

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ABSTRACT Background: Stunting among children under five can be caused by various factors, including inadequate food intake, characteristics of children, history of infectious diseases, family care pattern, and quality of health services. The dominant cause of stunting is different in each region. This study aimed to determinant the factors on stunting incidence among children age 6-24 months, Pemalang, Central Java. Subjects and Method: This was a case control study conducted in July 2019 in the work area of the Kebondalem Community Health Center in Pemalang, Central Java. A total of 142 stunted children aged 6-24 months were selected for this study. The dependent variable of this study was stunting. The independent variables were nutritional intake, immunization status, hygene, exclusive breastfeeding, parental education, occupation, family income, and a history of infectious diseases. Data were collected using anthropometric measurements and interviews with a questionnaire. Data were analyzed using logistic regression. Results: Mother’s occupation (OR= 0.26; 95% CI= 0.01 to 0.78; p= 0.035;), history of exclusive breastfeeding (OR= 0.07; 95% CI= 0.02 to 0.25; p= 0.001), history of infectious disease (OR= 0.008; 95%CI= 0.02-0.25; p= 0.010), Nutritional intake (OR= 9.44; 95% CI=1.88 to 47.43; p= 0.006), and they were statistically significant. Conclusion: Mother’s occupation, history of exclusive breastfeeding, history of disease infection, and nutritional intake are factors associated with the risk of stunting. Keywords: mother’s occupation, history of exclusive breastfeeding, history of disease infection, and nutritional intake, stunting Correspondence: Arwinda Nugraheni. Faculty of Medicine, Universitas Diponegoro, Yogyakarta, Indonesia. Email: arwindanugraheni@gmail.com DOI: https://doi.org/10.26911/the7thicph.03.28
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Bogdanova, E. N. "POPULATION OF IXODID TICKS IN CITIES OF THE EUROPEAN PART OF THE RUSSIAN FEDERATION AND THEIR EPIDEMIOLOGICAL SIGNIFICANCE." In V International Scientific Conference CONCEPTUAL AND APPLIED ASPECTS OF INVERTEBRATE SCIENTIFIC RESEARCH AND BIOLOGICAL EDUCATION. Tomsk State University Press, 2020. http://dx.doi.org/10.17223/978-5-94621-931-0-2020-64.

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On the territory of Russia there are more than 70 species of ticks of the family Ixodidae. Many of them are vectors of infectious diseases. The most common tick-borne infections are tick-borne encephalitis and Lyme disease. In recent decades, in many countries there has been a process of tick occupation of the territories of settlements, including large cities and megapolices. This leads to an increase in the number of tick attacks on peoples, as well as the cases of tick-borne infections.
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Zubareva, I. M., N. V. Yudina, and E. A. Efremova. "EPIZOOTOLOGICAL CHARACTERISTICS OF ECTOPARASITOSIS OF CARNIVORES IN THE MEGAPOLIS (BY THE EXAMPLE OF NOVOSIBIRSK)." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. All-Russian Scientific Research Institute for Fundamental and Applied Parasitology of Animals and Plant – a branch of the Federal State Budget Scientific Institution “Federal Scientific Centre VIEV”, 2023. http://dx.doi.org/10.31016/978-5-6048555-6-0.2023.24.184-188.

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The most well-known ectoparasites of dogs and cats are fleas and Acariformes, which are also carriers of infectious and invasive diseases. Animals admitted to the veterinary clinic served as the material for the research. The study involved 3346 animals, of which 1596 cats and 1707 dogs. Classical life-time diagnostic methods of ectoparasitosis were used. The results obtained show that among all dermatological patients, the proportion of those infected with ectoparasites was 47.6%. Among ectoparasites, fleas were most often recorded; the prevalence of infection was 4.78%. Aphanipterosis was recorded in all seasons of the year, the maximum invasion of 1.76% was observed in autumn, and the minimum of 1.30% in winter. The second most common disease was otodectosis (IP 1.94%). The heyletiellosis rate was 0.27% (total 9 animals, including 1 cat); and demodicosis, 0.24%. Notoedrosis was only diagnosed in the cats with IP 0.15%, sarcoptosis was recorded in the dogs with IP 0.09%. Ectoparasite infections are recorded at any time of the year, however, the peak of invasion occurs to a greater extent in the summer-autumn months. They can also occur at any age, but are more common in cats and dogs under one year of age.
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Volchkevich, I. G., V. I. Khalaeva, and M. V. Konopatskaya. "PREVALENCE AND DEVELOPMENT OF POTATO DITYLENCHIASIS IN BELARUS." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. VNIIP – FSC VIEV, 2024. http://dx.doi.org/10.31016/978-5-6050437-8-2.2024.25.88-93.

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The stem nematode (Ditylenchus destructor Thorne, 1945) is the causative agent of ditylenchiasis and occupies one of the first places in harmfulness among potato phytohelminth infections and causes significant damage to the crop both in the growing season and storage. The stem nematode is widespread in all areas where potatoes are cultivated and has a quarantine status for many countries. To study ditylenchiasis spread and development in 2019-2023, route surveys of tubers were conducted at typical potato storage houses in Belarus. The results of phytosanitary monitoring of tubers showed an annual increase in their damage by ditylenchiasis. It was determined that the prevalence rate in tubers varied from 0.1 to 3.3% in the preplanting period. We found that the first signs of tuber damage by stem nematode were observed already during the harvesting period. The prevalence rate of ditylenchiasis on potato tubers of new crop when grown in the artificial infectious background can reach 46.7%. The largest number of tubers damaged by the phytohelminth was found in the early Lilea variety, and the smallest number, in the late Vectar variety. The prevalence of the disease increased for the storage period by an average of 1.8, and the development, by 8.8, with the maximum rate of infection development in the first two months of storage.
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Reports on the topic "Infectious disease"

1

Burgess, Lawrence P. Bioterrorism Preparedness for Infectious Disease Proposal. Fort Belvoir, VA: Defense Technical Information Center, January 2006. http://dx.doi.org/10.21236/ada482641.

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Fernandez De Cordoba Farini, Claudia, and Ilan Kelman. Designing infectious disease warnings that work. Edited by Tasha Wibawa. Monash University, March 2022. http://dx.doi.org/10.54377/bd50-dfcb.

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Bailey, Charles L. Infectious Disease Proteome Biomarkers: Final Technical Report. Office of Scientific and Technical Information (OSTI), December 2011. http://dx.doi.org/10.2172/1090082.

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Daughton, Ashlynn Rae, Alina Deshpande, and Eric Nicholas Alexandre Generous. Enhancing Situational Awareness for Infectious Disease Surveillance. Office of Scientific and Technical Information (OSTI), April 2015. http://dx.doi.org/10.2172/1177977.

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Baliga, Priya, and Annette Von Thun. Infectious Disease Border Issues Conference: Meeting Synopsis. Fort Belvoir, VA: Defense Technical Information Center, April 2012. http://dx.doi.org/10.21236/ada558972.

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Burgess, Lawrence P. Bioterrorism Preparedness for Infectious Disease (BTPID) Proposal. Fort Belvoir, VA: Defense Technical Information Center, January 2007. http://dx.doi.org/10.21236/ada479405.

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Allen, Brian M. The Effects of Infectious Disease on Napoleon's Russian Campaign. Fort Belvoir, VA: Defense Technical Information Center, April 1998. http://dx.doi.org/10.21236/ada398046.

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Salerno, Reynolds Mathewson, and William Kessler. Infectious Disease Detection and Control in the Developing World. Office of Scientific and Technical Information (OSTI), October 2014. http://dx.doi.org/10.2172/1163498.

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Gunderson, E. K., C. Garland, and L. L. Hourani. Infectious Disease Rates in the U.S. Navy, 1980-1995. Fort Belvoir, VA: Defense Technical Information Center, October 1999. http://dx.doi.org/10.21236/ada381292.

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Naulls, Stephen, and Clare Lally. Human challenge studies in the study of infectious diseases. Parliamentary Office of Science and Technology, UK Parliament, May 2023. http://dx.doi.org/10.58248/rr01.

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Human challenge studies involve researchers deliberately exposing healthy volunteers to pathogens that cause infectious diseases. This allows researchers to monitor how the body responds to the disease, and to test possible new treatments and vaccines. Human challenge studies have become more well-known for their use in the Covid-19 pandemic, but they have been used for decades to advance our understanding of infectious diseases. Their benefits include the ability to study infections and test treatments in a controlled setting, and during times when there is low prevalence in the general population. However, benefits must be balanced against other considerations, such as health risks to volunteers and other ethical challenges. The Government has recently committed over £13 million of funding to human challenge studies, and international organisations are developing further regulation.
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