Relevant bibliographies by topics / Indoxyl sulphate

Academic literature on the topic 'Indoxyl sulphate'

Author: Grafiati
Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Indoxyl sulphate"

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Chou, Chia-An, Hwee-Yeong Ng, Wei-Hung Kuo, Ting-Yu Terry Chiou, Sung-Nan Pei, Lung-Chih Li, Yueh-Ting Lee, and Chien-Te Lee. "Rosiglitazone attenuates indoxyl sulphate-induced endothelial dysfunction." Clinical and Experimental Pharmacology and Physiology 42, no. 3 (February 11, 2015): 287–92. http://dx.doi.org/10.1111/1440-1681.12351.

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Mozar, A., L. Louvet, C. Godin, R. Mentaverri, M. Brazier, S. Kamel, and Z. A. Massy. "Indoxyl sulphate inhibits osteoclast differentiation and function." Nephrology Dialysis Transplantation 27, no. 6 (December 1, 2011): 2176–81. http://dx.doi.org/10.1093/ndt/gfr647.

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Valko-Rokytovská, Marcela, Beáta Hubková, Anna Birková, Jana Mašlanková, Marek Stupák, Marianna Zábavníková, Beáta Čižmárová, and Mária Mareková. "Specific Urinary Metabolites in Malignant Melanoma." Medicina 55, no. 5 (May 16, 2019): 145. http://dx.doi.org/10.3390/medicina55050145.

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Background and objectives: Melanin, which has a confirmed role in melanoma cell behaviour, is formed in the process of melanogenesis and is synthesized from tryptophan, L-tyrosine and their metabolites. All these metabolites are easily detectable by chromatography in urine. Materials and Methods: Urine samples of 133 individuals (82 malignant melanoma patients and 51 healthy controls) were analysed by reversed-phase high-performance liquid chromatography (RP-HPLC). The diagnosis of malignant melanoma was confirmed histologically. Results: Chromatograms of melanoma patients showed increased levels of 5,6-dihydroxyindole-2-carboxylic acid, vanilmandelic acid, homovanilic acid, tryptophan, 5-hydroxyindole-3-acetic acid, and indoxyl sulphate compared to healthy controls. Concentration of indoxyl sulphate, homovanilic acid and tryptophan were significantly increased even in the low clinical stage 0 of the disease (indoxyl sulphate, homovanilic acid and tryptophan in patients with clinical stage 0 vs. controls expressed as medium/ interquartile range in µmol/mmol creatinine: 28.37/15.30 vs. 5.00/6.91; 47.97/33.08 vs. 7.33/21.25; and 16.38/15.98 vs. 3.46/6.22, respectively). Conclusions: HPLC detection of metabolites of L-tyrosine and tryptophan in the urine of melanoma patients may play a significant role in diagnostics as well as a therapeutic strategy of melanoma cancer.
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Wu, I. W., K. H. Hsu, C. C. Lee, C. Y. Sun, H. J. Hsu, C. J. Tsai, C. Y. Tzen, Y. C. Wang, C. Y. Lin, and M. S. Wu. "p-Cresyl sulphate and indoxyl sulphate predict progression of chronic kidney disease." Nephrology Dialysis Transplantation 26, no. 3 (September 29, 2010): 938–47. http://dx.doi.org/10.1093/ndt/gfq580.

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Yang, Ke, Cheng Wang, Ling Nie, Xiaohui Zhao, Jun Gu, Xu Guan, Song Wang, et al. "Klotho Protects Against Indoxyl Sulphate-Induced Myocardial Hypertrophy." Journal of the American Society of Nephrology 26, no. 10 (March 24, 2015): 2434–46. http://dx.doi.org/10.1681/asn.2014060543.

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Yousef Selim, Nermien, Hazem Farag Mannaa, Ola Atef Sharaki, Tayseer Zaytoun, Noha Elkholy, and Waleed Arafat. "Highlighting Levels of Indoxyl Sulphate among Critically Ill Patients with Acute Nephrotoxicity; Correlations Between Indoxyl Sulphate Levels and Patients’ Characteristics." Reports of Biochemistry and Molecular Biology 10, no. 2 (August 1, 2021): 266–79. http://dx.doi.org/10.52547/rbmb.10.2.266.

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Ellis, Robert J., David M. Small, David A. Vesey, David W. Johnson, Ross Francis, Luis Vitetta, Glenda C. Gobe, and Christudas Morais. "Indoxyl sulphate and kidney disease: Causes, consequences and interventions." Nephrology 21, no. 3 (January 28, 2016): 170–77. http://dx.doi.org/10.1111/nep.12580.

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Kamiński, Tomasz, Małgorzata Michałowska, and Dariusz Pawlak. "Aryl hydrocarbon receptor (AhR) and its endogenous agonist – indoxyl sulfate in chronic kidney disease." Postępy Higieny i Medycyny Doświadczalnej 71, no. 1 (July 30, 2017): 0. http://dx.doi.org/10.5604/01.3001.0010.3843.

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The indoxyl sulfate (IS, indoxyl sulphate) is the end product of dietary tryptophan degradation by indole pathway and significantly higher serum and tissue concentrations of this compound is observed in patients with impaired renal function. Despite the high albumin binding affinity, the remaining free fraction of IS has a number of biological effects related to the generation of oxidative stress andactivation of signaling pathways related to NF-кB, p53 protein, STAT3, TGF-β and Smad2/3. IS induces the inflammatory process, exerts nephrotoxic activity and is also a factor impairing the cardiovascular system.Its high concentrations are associated with the occurrence of cardiovascular incidents, whose frequency is significantly higher in patients with chronic kidney disease. Evaluation of the mechanisms that underlie the high reactivity of indoxyl sulfate and its biological effects showed that this compound is an agonist of the aryl hydrocarbon receptor (AhR). This receptor plays an important role in maintaining homeostasis Moreover, AhR exerts high transcriptional activity, so ligands of obciążethis receptor may exert different biological effects. The following paper describes the role of indoxyl sulfate as AhR ligand in the context of the excessive accumulation, which appears as one of the symptoms associated with chronic kidney disease.
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Adesso, Simona, Ada Popolo, Giuseppe Bianco, Rosalinda Sorrentino, Aldo Pinto, Giuseppina Autore, and Stefania Marzocco. "The Uremic Toxin Indoxyl Sulphate Enhances Macrophage Response to LPS." PLoS ONE 8, no. 9 (September 30, 2013): e76778. http://dx.doi.org/10.1371/journal.pone.0076778.

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Deguchi, Tsuneo, Mikio Nakamura, Yasuhiro Tsutsumi, Ayaka Suenaga, and Masaki Otagiri. "Pharmacokinetics and tissue distribution of uraemic indoxyl sulphate in rats." Biopharmaceutics & Drug Disposition 24, no. 8 (2003): 345–55. http://dx.doi.org/10.1002/bdd.370.

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Dissertations / Theses on the topic "Indoxyl sulphate"

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ASTORI, EMANUELA. "IN VITRO AND IN VIVO APPROACHES TO STUDY OXIDATIVE STRESS, ANEMIA AND DYSBIOSIS IN CHRONIC KIDNEY DISEASE." Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/818976.

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CKD is diagnosed when there’s a decreased kidney function shown by a GFR less than 60 ml / min (established for a reference man with 1.73 m² body surface area), or markers of kidney damage, or both, of at least 3 months duration. The severity of complications increases in parallel with the GFR decline. We focused on three comorbidities extremely common in CKD patients: oxidative stress and inflammation; anemia and dysbiosis. We investigated these CKD comorbidities both with in vitro and in vivo approaches. More in detail, regarding in vivo studies, we measured oxidative stress biomarkers in a population of ESRD patients before and after the hemodialysis treatment, comparing the results with a population of healthy subjects; we evaluated oxidative stress biomarkers in the plasma of HD patients before, during and after two type of iron treatments (intravenous and sucrosomial iron). Regarding in vitro experiments, we focused on two uremic toxins, urea and indoxyl sulphate, and we evaluated their effects on a human endothelial cell line (Human Microvascular Endothelial Cells 1, HMEC-1).
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Books on the topic "Indoxyl sulphate"

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Covic, Adrian, Mugurel Apetrii, Luminita Voroneanu, and David J. Goldsmith. Vascular calcification. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0120_update_001.

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Vascular calcification (VC) is a common feature of patients with advanced CKD and it could be, at least in part, the cause of increased cardiovascular mortality in these patients. From a morphologic point of view, there are at least two types of pathologic calcium phosphate deposition in the arterial wall—namely, intima calcification (mostly associated with atherosclerotic plaques) and media calcification (associated with stiffening of the vasculature, resulting in significantly adverse cardiovascular outcomes). Although VC was viewed initially as a passive phenomenon, it appears to be a cell-mediated, dynamic, and actively regulated process that closely resembles the formation of normal bone tissue, as discovered recently. VC seems to be the result of the dysregulation of the equilibrium between promoters and inhibitors. The determinants are mostly represented by altered calcium and phosphorus metabolism, secondary hyperparathyroidism, vitamin D excess, high fibroblast growth factor 23, and high levels of indoxyl sulphate or leptin; meanwhile, the inhibitors are vitamin K, fetuin A, matrix G1a protein, osteoprotegerin, and pyrophosphate. A number of non-invasive imaging techniques are available to investigate cardiac and vascular calcification: plain X-rays, to identify macroscopic calcifications of the aorta and peripheral arteries; two-dimensional ultrasound for investigating the calcification of carotid arteries, femoral arteries, and aorta; echocardiography, for assessment of valvular calcification; and, of course, computed tomography technologies, which constitute the gold standard for quantification of coronary artery and aorta calcification. All these methods have a series of advantages and limitations. The treatment/ prevention of VC is currently mostly around calcium-mineral bone disease interventions, and unproven. There are interesting hypotheses around vitamin K, Magnesium, sodium thiosulphate and other potential agents.
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Book chapters on the topic "Indoxyl sulphate"

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Holmar, J., J. Arund, F. Uhlin, R. Tanner, and I. Fridolin. "Quantification of Indoxyl Sulphate in the Spent Dialysate Using Fluorescence Spectra." In IFMBE Proceedings, 45–48. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-21683-1_11.

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Nataatmadja, Melissa, Yeoungjee Cho, Katrina Campbell, and David W. Johnson. "The Roles of Indoxyl Sulphate and p-Cresyl Sulphate in Patients with Chronic Kidney Disease: A Review of Therapeutic Options." In Chronic Kidney Disease - from Pathophysiology to Clinical Improvements. InTech, 2018. http://dx.doi.org/10.5772/intechopen.69325.

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Conference papers on the topic "Indoxyl sulphate"

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Holmar, Jana, Fredrik Uhlin, Rain Ferenets, Kai Lauri, Risto Tanner, Jurgen Arund, Merike Luman, and Ivo Fridolin. "Estimation of removed uremic toxin indoxyl sulphate during hemodialysis by using optical data of the spent dialysate." In 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6611095.

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Sravya, G., Grigory V. Zyryanov, M. Rama Mohan Reddy, V. N. Ratnakaram, and N. Bakthavatchala Reddy. "Meglumine sulphate: An efficient catalyst for the synthesis of bis(indolyl)methanes." In PROCEEDINGS OF INTERNATIONAL CONFERENCE ON RECENT TRENDS IN MECHANICAL AND MATERIALS ENGINEERING: ICRTMME 2019. AIP Publishing, 2020. http://dx.doi.org/10.1063/5.0018097.

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