Academic literature on the topic 'Inama'
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Journal articles on the topic "Inama"
Zhichao Huang, Zhichao Huang, Yuxi Sun Zhichao Huang, Yunming Ye Yuxi Sun, Wensheng Gan Yunming Ye, and Wei-Che Chien Wensheng Gan. "INAMA: An Interactive Attentional Model for Node Alignment." 網際網路技術學刊 22, no. 7 (December 2021): 1587–97. http://dx.doi.org/10.53106/160792642021122207012.
Full textSuominen, Kati. "Rules of Origin in International Trade by Stefano Inama Cambridge: Cambridge University Press, 2009." World Trade Review 8, no. 4 (September 17, 2009): 616–18. http://dx.doi.org/10.1017/s1474745609990115.
Full textSilva, Peri. "BOOK REVIEW: "A Review of Rules of Origin in International Trade" Edited by Stefano Inama." Global Journal of Economics 01, no. 01 (June 2012): 1280001. http://dx.doi.org/10.1142/s2251361212800018.
Full textSirhi, Sirilus, Nelly Wedyawati, and Adriana Gandasari. "IPTEKS: KERIPIK BERBAHAN DASAR PELEPAH PISANG DARI DESA SUNGAI UKOI." Jurnal Pengabdian Masyarakat Khatulistiwa 4, no. 2 (November 30, 2021): 90–99. http://dx.doi.org/10.31932/jpmk.v4i2.1291.
Full textGarg, Pallavi, Ranjan K. Nandy, Papiya Chaudhury, Nandini Roy Chowdhury, Keya De, T. Ramamurthy, Shinji Yamasaki, S. K. Bhattacharya, Yoshifumi Takeda, and G. Balakrish Nair. "Emergence of Vibrio cholerae O1 Biotype El Tor Serotype Inaba from the Prevailing O1 Ogawa Serotype Strains in India." Journal of Clinical Microbiology 38, no. 11 (2000): 4249–53. http://dx.doi.org/10.1128/jcm.38.11.4249-4253.2000.
Full textMeeks, Michael D., Rina Saksena, Xingquan Ma, Terri K. Wade, Ronald K. Taylor, Pavol Kováč, and William F. Wade. "Synthetic Fragments of Vibrio cholerae O1 Inaba O-Specific Polysaccharide Bound to a Protein Carrier Are Immunogenic in Mice but Do Not Induce Protective Antibodies." Infection and Immunity 72, no. 7 (July 2004): 4090–101. http://dx.doi.org/10.1128/iai.72.7.4090-4101.2004.
Full textMojašević, Aleksandar, and Branko Radulović. "The importance of spitefulness in the economic analysis of litigation." Zbornik radova Pravnog fakulteta Nis 59, no. 87 (2020): 145–64. http://dx.doi.org/10.5937/zrpfn0-25767.
Full textLuong, Phi, Wayne Lencer, Denis Chang, and Qian Li. "P145 MECHANISMS OF ACTION FOR THE IBD-RISK GENE C1ORF106/INAVA." Inflammatory Bowel Diseases 26, Supplement_1 (January 2020): S30. http://dx.doi.org/10.1093/ibd/zaa010.074.
Full textSh.M. Veliyeva. "İNAM İNTERVALININ KÖMƏYİ İLƏ ŞAGİRDLƏRİN HƏFTƏLİK DƏRSLƏ MƏŞĞUL OLMA SAATININ TƏYİN OLUNMASI." Scientific News of Academy of Physical Education and Sport 3, no. 4 (December 14, 2021): 271–74. http://dx.doi.org/10.28942/ssj.v3i4.437.
Full textPriambodo, Evan Yuri. "Examining the Web Design and Quality of Information to User Satisfaction : The Case of Student at Sekolah Tinggi Ilmu Ekonomi INABA Bandung." International Journal of Business, Technology and Organizational Behavior (IJBTOB) 1, no. 1 (February 22, 2021): 1–6. http://dx.doi.org/10.52218/ijbtob.v1i1.1.
Full textDissertations / Theses on the topic "Inama"
Ferreira, Diogenes Seraphim. "Imunidade inata na asma fatal." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-22092010-115846/.
Full textINTRODUCTION: Airway inflammation in asthma involves innate immune responses. Toll-like receptors (TLRs) and the cytokine thymic stromal lymphopoietin (TSLP) are involved in bronchial inflammation in asthma, but the expression of these proteins in large and small airways of asthmatics has not been investigated. The aims of this study were to analyze the protein expression of TLR2, TLR3, TLR4 and TSLP in large and small airways of asthmatics, to compare their expression in smoking and nonsmoking asthmatics and to investigate if TLR expression in associated with infection by Chlamydophila pneumoniae and Mycoplasma pneumoniae. METHODS: Using immunohistochemistry and image analysis, we investigated the expression of TLR2, TLR3, TLR4 and TSLP in large and small airways of 24 fatal asthma patients (13 nonsmokers and 11 smokers) and 9 nonasthmatic controls. The protein expression was analyzed in four regions of the airways: epithelial, internal, airway smooth muscle and outer layers. C. pneumoniae and M. pneumoniae presence in lung tissue was analyzed by real-time polymerase chain reaction. RESULTS: Fatal asthma patients had increased expression of TLR2 in the epithelial and outer layers of large and small airways, and also higher TLR2 in the muscle layer of small airways. Smoking asthmatics had lower TLR2 in the inner and outer layers of small airways than nonsmoking asthmatics. TSLP was increased in the epithelial and outer layers of large airways. Asthmatics also had greater TLR3 in the outer layer of large airways and greater TLR4 in the outer layer of small airways. C. pneumoniae and M. pneumoniae DNA was not detected in asthmatics or controls. CONCLUSIONS: Innate immunity receptors TLR2, 3 and 4 and innate cytokine TSLP are increased in the airways of fatal asthma patients, and TLRs expression is not associated with the presence of Mycoplasma pneumoniae and Chlamydophila pneumoniae in the lungs. Smoking may reduce TLR2 expression in the small airways of asthmatics. These results suggest that TLR2, 3, 4 and TSLP may contribute to the bronchial inflammation seen in severe exacerbations of asthma and that M. pneumoniae and C. pneumoniae are not involved in fatal asthma exacerbations
Nelson, Mark D. "Integrated network application management (INAM)." Thesis, Monterey, Calif. : Springfield, Va. : Naval Postgraduate School ; Available from National Technical Information Service, 2004. http://library.nps.navy.mil/uhtbin/hyperion/04Dec%5FNelson.pdf.
Full textThesis advisor(s): Alex Bordetsky. Includes bibliographical references (p. 85-86). Also available online.
Bürkle, Lutz. "Elektrooptische Eigenschaften von InAsa-(GaIn)Sb Übergittern." [S.l. : s.n.], 2001. http://nbn-resolving.de/urn:nbn:de:bsz:25-opus-51241.
Full textMarques, Mariana Morato. "Leucotrienos como moduladores da imunidade inata a fungos." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-12112012-092250/.
Full textLeukotrienes (LTs) are lipid mediators derived from arachidonic acid. There is evidence that innate immunity receptors and leukotrienes receptors interact and amplify macrophage effector functions. We investigated if LTs receptors modulate phagocytosis and microbicidal activity mediated by Mannose receptor, Dectin-1 and PTX3 in alveolar macrophages (AMs) and the molecular mechanisms involved. Our results showed that: 1) AMs produce LTs when stimulated with C.albicans, Zy, Zy-PTX3; 2) LTs enhance phagocytosis of C.albicans and Zymosan, but not Zy-PTX3. This is dependent on: recognition via mannose receptor (LTB4) and Dectin-1 (LTD4); integrity of lipid rafts; its action on actin polimerization mechanisms; enhancement of F-actin levels by induction of Cofilin-1 inactivation; activation of LIMKs that regulate Cofilin-1; activation of PKCd and PI3K3) LTs enhance killing of C.albicans by activation of NADPH oxidase. Taken together, our results showed that LTs specifically influence signaling programs of keys PRRs.
Cruz, Meire Karla Miguel. "Receptores da imunidade inata na Leishmaniose visceral canina." PROGRAMA DE P?S-GRADUA??O EM BIOLOGIA PARASIT?RIA, 2017. https://repositorio.ufrn.br/jspui/handle/123456789/24113.
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C?es s?o os reservat?rios prim?rios dos parasitos do g?nero Leishmania. Receptores da imunidade inata fazem a detec??o precoce do parasito e conduzem a imunidade adaptativa espec?fica na tentativa de controlar a infec??o. Entretanto, poucos estudos tem investigado a correla??o entre a express?o de receptores da imunidade inata e a resist?ncia ou susceptibilidade em c?es infectados por Leishmania infantum. O objetivo deste estudo foi correlacionar os achados cl?nicos em c?es naturalmente infectados por L. infantum ? express?o de receptores da imunidade inata (Toll Like Receptors-TLRs e Nod Like Receptors-NLRs). Inicialmente, o soro de 76 c?es foi coletado no Centro de Controle de Zoonoses de Natal, Rio Grande do Norte, Brasil. A positividade dos c?es para L. infantum foi confirmada pela reatividade nos testes de ELISA e DPP?. Os c?es foram clinicamente avaliados e classificados como sintom?ticos (n=19), oligossintom?ticos (n=19), assintom?ticos (n=19) e n?o infectados (n=19). Os c?es naturalmente infectados por L. infantum e controles n?o infectados foram eutanasiados e fragmentos de f?gado foram coletados para quantifica??o da express?o de RNAm de TLRs (TLR1-9), NLRs (NOD1, NOD2, NLRP1 e NLRP3) citocinas (IL1?, IL-6, IL-12, IL-10, TNF?, IFN-?) e iNOS com auxilio da t?cnica de PCR em tempo real. Os resultados demonstram o aumento na express?o da maioria dos receptores do tipo Toll e do tipo Nod nos c?es naturalmente infectados por L. infantum, comparado a animais n?o infectados. Entretanto, c?es sintom?ticos apresentaram maior express?o de TLR1, TLR2, TLR3, TLR4, TLR5, TLR7, TLR8, NLRP1, NLRP3, NOD1 e IL-1? quando comparado a animais assintom?ticos, mostrando significante aumento na transcri??o destas mol?culas com a progress?o da doen?a. Por outro lado, c?es assintom?ticos apresentaram maior express?o de RNAm de citocinas (IFN-?, IL-12) e iNOS quando comparado a animais oligossintom?ticos e sintom?ticos. Este estudo gerou novos conhecimentos envolvendo receptores da imunidade inata (TLRs, NLRs) na leishmaniose visceral canina (LVC), podendo servir de base para o melhor entendimento dos mecanismos de resist?ncia ou susceptibilidade ? infec??o por L. infantum em c?es, bem como dar subs?dio a estrat?gias profil?ticas para o controle da LVC.
Dogs are the primary reservoirs of parasites of the Leishmania genus. Innate immune receptors perform early detection of the parasite and lead to specific adaptive immune response in attempt to infection control. However, few studies have investigated a correlation between the expression of innate immunity receptors and the resistance or susceptibility pattern in dogs naturally infected with Leishmania infantum. The aim of this study was to correlate the clinical status of dogs naturally infected with L. infantum with the mRNA expression levels of innate imune receptors (Toll like receptors-TLRs and Nod Like Receptors-NLRs). Initially, serum of 76 dogs was collected at the Zoonoses Control Center in Natal, Rio Grande do Norte, Brazil. The L. infantum infection in dogs was confirmed by ELISA and DPP? tests. Subsequently, animals were clinially evaluated and classified as asymptomatic (n=19), oligosymptomatic (n=19), symptomatic (n=19) and uninfected (n=19). Dogs naturally infected by L. infantum and uninfected controls were euthanasied and liver samples were collected to quantify mRNA expression of TLRs (TLR1-9), Nod Like receptors-NLRs (NOD1, NOD2, NLRP1, NLRP3), cytokines (IL1?, IL-6, IL-12, IL-10, TNF?, IFN-?) and iNOS using real-time PCR. The results demonstrate the increased expression of almost all TLRs and NLRs in dogs naturally infected by L. infantum compared with uninfected animals. However, symptomatic dogs showed higher expression of TLR1, TLR2, TLR3, TLR4, TLR5, TLR7, TLR8 NLRP1, NLRP3, NOD1 and IL-1? than asymptomatic animals, revealing significant up regulation of transcription with disease progression. On the other hand, asymptomatic dogs presented greater cytokine mRNA expression (IFN-?, IL-12) and iNOS when compared to oligosymptomatic and asymptomatic animals. This study unveil new knowledge involving innate immunity receptors (TLRs, NLRs) and cytokines in canine visceral leishmaniasis and may be used as a basis for better understanding of resistance or susceptibility mechanisms in dogs infected with L. infantum, as well as prophylactic strategies to control canine visceral leishmaniasis.
Reis, Cláudio Araújo dos. "A imunidade inata na insuficiência cardíaca: papel dos monócitos." Master's thesis, Universidade de Aveiro, 2012. http://hdl.handle.net/10773/10087.
Full textOs monócitos desempenham um papel importante na imunidade inata e a ativação desta é mediada pelos recetores Toll-like (TLRs). Os TLRs estão envolvidos no reconhecimento de micro-organismos estranhos pelo sistema imunológico inato, podendo também ser ativados por moléculas endogénas. Os TLR2 e TLR4 têm sido associados à ativação da imunidade inata na insuficiência cardíaca (IC). Os monócitos são populações heterogéneas e compreendem subpopulações com fenótipos distintos, que podem ser identificadas pela diferente expressão dos antigénios CD14 e CD16. Este estudo tem como objetivo clarificar os mecanismos de ativação da imunidade inata em doentes com insuficiência cardíaca crónica (ICC), utilizando a imunofenotipagem celular por citometria de fluxo. Foi colhido sangue periférico a 15 doentes com ICC e a 12 controlos saudáveis. Estudou-se por citometria de fluxo o fenótipo celular dos monócitos (FSC, SSC, TLR2, TLR4, CD4, CD11b, CD14, CD16, CD36, CD45, CD64, HLA-Dr e IREM-2), dos neutrófilos (FSC, SSC, CD10, CD11b, CD13, CD15, CD16 e CD45) e dos linfócitos (CD3, CD4, CD8, CD16, CD19 e CD56). As três subpopulações monocíticas identificadas (CD14+CD16-, CD14+CD16+ e CD14dimCD16+) apresentaram diferenças fenotípicas dos antigénios celulares estudados. A subpopulação CD14+CD16+ foi a que expressou com maior intensidade vários antigénios de ativação e os recetores TLR2 e TLR4. Os doentes com ICC apresentaram um aumento ligeiro da percentagem dos monócitos CD14+CD16+ e uma diminuição dos monócitos CD14+CD16-. A subpopulação CD14dimCD16+ apresentou uma diminuição significativa da sua percentagem com o aumento da gravidade da ICC. Na maioria das subpopulações, o TLR2 e o TLR4 tiveram uma tendência de aumento da sua expressão nos doentes com ICC, do que nos controlos. Os monócitos CD14dimCD16+ apresentaram uma diminuição significativa da expressão do TLR2 nos doentes com ICC, relativamente ao grupo controlo. A expressão diferencial do TLR2 e do TLR4 nos subtipos de monócitos poderá contribuir para o desenvolvimento e progressão da IC. A existência de subpopulações monocíticas com fenótipos e funções distintas poderá fornecer informações valiosas da patogénese da IC.
Monocytes play an important role in innate immunity and these are activated by the Toll-like receptores (TLRs). TLRs are able to recognise foreign microorganisms via the innate immune system and these can also be activated by endogenous molecules. TLR2 and TLR4 have been associated with activation the innate immunity in heart failure (HF). Monocytes are heterogenous populations which are subdivided into distinct phenotypes and can be identified by the different expression of CD14 and CD16 antigens. This study´s main objective is to clarify the activation mechanisms of innate immunity in patients with chronic heart failure (CHF), using the cellular immunophenotyping by flow cytometry. Peripheral blood was taken from 15 patients with CHF and was used also 12 health controls. The cellular phenotypes of the monocytes were studied using flow cytometry (TLR2, TLR4, CD4, CD11b, CD14, CD16, CD36, CD45, CD64, HLA-Dr, IREM-2, FSC and SSC), in neutrophils (CD10, CD11b, CD13, CD15, CD16, CD45, FSC e SSC) and in lymphocytes (CD3, CD4, CD8, CD16, CD19 e CD56). The three monocytic subpopulations identified (CD14+CD16-, CD14+CD16+ e CD14dimCD16+) revealed phenotypic differences of the cellular antigens. The CD14+CD16+ subpopulation expressed greater intensity of several activation antigens as well TLR2 and TLR4 receptors. Patients with CHF showed a slight increase in monocyte percentage CD14+CD16+ and a decrease in CD14+CD16- monocytes. The CD14dimCD16+ subpopulation showed a significant decrease of its percentage with increasing severity of CHF. In most subpopulations, TLR2 and TLR4 had a trend of increased expression in patients with CHF than controls. In patients with CHF, CD14dimCD16+ monocytes showed a significant decrease in expression of TLR2, than the control group. Different expression of TLR2 and TLR4 in the subtypes of monocytes may contribute to the development and progression of HF. The existence of monocytic subpopulations with distinct phenotypes and functions can provide valuable information of the pathogenesis of HF.
Batista, Camila Freitas. "Dinâmica da resposta imune inata do sistema respiratório de bezerros." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/10/10136/tde-04092012-150644/.
Full textThe influences of age in calves\' immune system are described in their first phase of life. The hypothesis that variations occur in the main mechanisms of lung innate response can help to identify periods of greater susceptibility to the respiratory diseases that affect calves in the first stage of their life. With the purpose of minimizing the economic losses associated with respiratory disease in calves, this study aimed to evaluate the innate immune dynamics of the respiratory system of healthy calves in the first three months of life. Nine healthy calves were monitored for three months and eight immunologic evaluations were performed. Bronchoalveolar lavage samples were recovered by bronchoscopy. Then, the alveolar macrophages in samples were identified by protein expression of CD14 and undergone functional evaluation of phagocytosis (SAPI and E.coli) and oxidative burst. Immunoglobulin were also quantified in samples. Data was assessed by one-way ANOVA (unstacked) (parametric) and the Mann-Whitney test nonparametric). Functional alterations in phagocytes CD14 + were observed, and although their relative values were kept throughout the period, higher intensity of phagocytosis in the third week and increased phagocytosis by macrophages CD14 + at 45 days of life was observed. Decreased intensity of phagocytosis was observed after this age. It is concluded that from 45 days of life on, calves began to maintain their immune response, but until 90 days of life they did not achieve the stability to conclude the maturation of local innate response.
Guimarães, Joana Alexandra Carvalho. "Patogénese da artrite idiopática juvenil sistémica. Papel da imunidade inata." Master's thesis, Instituto de Ciências Biomédicas Abel Salazar, 2010. http://hdl.handle.net/10216/62307.
Full textNoleto, Pablo Gomes. "Efeito do metabolismo na resposta imune inata do endométrio bovino." Universidade Federal de Uberlândia, 2016. https://repositorio.ufu.br/handle/123456789/19063.
Full textContaminação bacteriana que normalmente infecta o endométrio bovino no pós-parto prejudica a defesa imunológica quando há estresse metabólico, levando a endometrite e infertilidade. A resposta do endométrio contra bactérias depende da imunidade inata, com o reconhecimento de padrões moleculares associados a patógenos, estimulando a inflamação, caracterizadas por secreção de interleucinas (IL)-1p, IL-6 e IL-8. Entretanto, animais frequentemente falham em eliminar a infecção microbiana quando apresentam reduzidas concentrações de glicose e glutamina, que são fontes de energia para o endométrio no conhecido balanço energético negativo. A contínua inflamação e resposta imune inata contra infecção bacteriana no útero após o parto compromete o bem-estar animal. Neste estudo testou-se a hipótese de que as vias homeostáticas conferidas pela glicose, glutamina e biotina integram o metabolismo energético e a imunidade inata no tecido endometrial bovino. A ausência de glicose reduziu a secreção de IL-1P, IL-6 e IL-8 em culturas de órgãos endometriais desafiadas com a endotoxina lipopolissacarídeo (LPS). Depleção de glutamina também reduziu a atividade inflamatória do endométrio contra LPS. Entretanto, as atividades de glicose e glutamina dependem da presença de glicólise. A vitamina biotina demonstrou ter caráter anti- inflamatório, reduzindo a produção de citocinas pró-inflamatórias no endométrio. Em conclusão, o estresse energético metabólico afetou as respostas inflamatórias à infecções bacterianas no endométrio. Providenciamos dados que demonstram como o balanço energético negativo pode estar ligado a doença uterina no pós-parto.
Bacteria that usually infect the bovine endometrium at postpartum impairs immune defense when exist metabolic stress, leading to endometritis and infertility. The response of the endometrium to bacteria depends on innate immunity, with the recognition of pathogen-associated molecular patterns by stimulating inflammation, characterized by secretion of interleukins (IL) -1p, IL-6 and IL-8. However, animals often fail to eliminate the microbial infection when they have reduced levels of glucose and glutamine, which are sources of energy to the endometrium in the negative energy balance. Continuous inflammation and innate immune response to bacterial infection in the uterus after calving compromise animal welfare. In this study we tested the hypothesis that homeostatic pathways conferred by glucose , glutamine and biotin, link the energy metabolism and innate immunity in the bovine endometrial tissue. The deprivation of glucose reduced the secretion of IL-1P, IL-6 and IL-8 in cultures of endometrial organs challenged with the endotoxin lipopolysaccharide (LPS). Glutamine depletion also reduces the inflammatory activity of the endometrium to LPS. However, glucose and glutamine activities depend on the presence of glycolysis. The vitamin biotin demonstrates anti-inflammatory nature, reducing the production of proinflammatory cytokines in the endometrium. In conclusion, the energy metabolic stress disrupts inflammatory responses to bacterial infections in the endometrium. We provide data on how negative energy balance may be linked to postpartum uterine disease.
Tese (Doutorado)
Santos, Renato de Lima. "Imunidade inata, patogênese e diagnóstico de salmonelose, brucelose e leishmaniose /." Botucatu, 2011. http://hdl.handle.net/11449/122156.
Full textAbstract: This work provides a systematic and critical review of part of the scientific production of its author. It includes three research topics, namely salmonellosis, brucellosis, and leishmaniasis. Each one of these topics is discussed in individual chapters. Most of the data discussed here refers to pathology, pathogenesis, hostpathogen interactions, innate immunity, and diagnosis of these diseases. Instead of being organized as a traditional literature reviews, the chapters actually emphasize the scientific contribution of the author's publications in each of these fields
Books on the topic "Inama"
Nyirahabimana, Solina. Inama ku mikorere y'abafasha mu by'amategeko. Kigali: Umuryango Uharanira Uburenganzira bw'Umwana, Umwari n'Umutegarugori (HAGURUKA), 1999.
Find full textInama nyunguranabitekerezo ku guhesha agaciro Ikinyarwanda (2001 Butare, Rwanda). Inama nyunguranabitekerezo ku guhesha agaciro ikinyarwanda. Butare [Rwanda]: Ikigo cy'Ubushakashatsi mu by'Ubuhanga n'Ikoranabuhanga, Ishami ryiga iby'i Rwanda, 2001.
Find full textInama, Nkuru y'Igihugu y'Ubumwe n'Ubwiyunge (2000 Kigali Rwanda). Inama Nkuru y'Igihugu y'Ubumwe n'Ubwiyunge: Kigali, 18-20 Ukwakira 2000. [Kigali]: Komisiyo y'Igihugu y'Ubumwe n'Ubwiyunge, 2000.
Find full textInama nyungurana-bitekerezo y'imiryango y'abahinzi-borozi ku kibazo cy'ibiribwa bidahagije mu Rwanda (1997 Kabusunzu, Kigali, Rwanda). Inama nyungurana-bitekerezo y'imiryango y'abahinzi-borozi ku kibazo cy'ibiribwa bidahagije mu Rwanda: Yabereye mukigo IWACU ku Kabusunzu, 6-8 ukwakira 1997 : raporo y'inama. Kigali: CCOAIB, 1997.
Find full textLowe, Michele. Inana. New York: Samuel French New York, 2011.
Find full text1981-, Armagnac Juliette, ed. Inawa. Prayssas: Éd. Arphilvolis, 2011.
Find full textKam̆wala, Surajīta Siṅgha. Ināma: Kahāṇīaṃ. Ammritasara: Liṭarecara Hāūsa, 1994.
Find full textUricariu, Doina. Inima axonometrică. București: Universalia, 2002.
Find full text(Japan), Inasa-chō. Inasa chōshi. Shizuoka-ken Inasa-gun Inasa-chō: Inasa-chō, 1991.
Find full textNeagu, Nicolae. Inima: Roman. București: Editura Eminescu, 1989.
Find full textBook chapters on the topic "Inama"
Inaba, Masumi, and Yoshikata Inaba. "Sebaceous Gland Hypothesis of Androgenetic Alopecia (Inaba 1985; Inaba and Inaba 1992a)." In Androgenetic Alopecia, 175–78. Tokyo: Springer Japan, 1996. http://dx.doi.org/10.1007/978-4-431-67038-4_19.
Full textInaba, Masumi, and Yoshikata Inaba. "The Inaba Method." In Human Body Odor, 171–234. Tokyo: Springer Japan, 1992. http://dx.doi.org/10.1007/978-4-431-66908-1_15.
Full textInaba, Masumi, and Yoshikata Inaba. "Trial Treatment of Androgenetic Alopecia with Oxidizing Agents (Inaba and Inaba 1984)." In Androgenetic Alopecia, 221–32. Tokyo: Springer Japan, 1996. http://dx.doi.org/10.1007/978-4-431-67038-4_28.
Full textKönigsberg, Matthew. "Inaka Rōjin Tada no Jijii." In Kindlers Literatur Lexikon (KLL), 1. Stuttgart: J.B. Metzler, 2020. http://dx.doi.org/10.1007/978-3-476-05728-0_2167-1.
Full textNevile, J. W. "A Reply to Dr. Inada." In Post-Keynesian Essays from Down Under Volume I: Essays on Keynes, Harrod and Kalecki, 319–20. London: Palgrave Macmillan UK, 2016. http://dx.doi.org/10.1057/9781137475381_24.
Full textHammitzsch, Horst. "Ryūtei Tanehiko: Nise murasaki inaka genji." In Kindlers Literatur Lexikon (KLL), 1–2. Stuttgart: J.B. Metzler, 2020. http://dx.doi.org/10.1007/978-3-476-05728-0_21182-1.
Full textHammitzsch, Horst, and Matthew Königsberg. "Inaka Rōjin Tada no Jijii: Yūshi hōgen." In Kindlers Literatur Lexikon (KLL), 1–2. Stuttgart: J.B. Metzler, 2020. http://dx.doi.org/10.1007/978-3-476-05728-0_2168-1.
Full textSubramoni, Hari, Albert Mathews Augustine, Mark Arnold, Jonathan Perkins, Xiaoyi Lu, Khaled Hamidouche, and Dhabaleswar K. Panda. "INAM2: InfiniBand Network Analysis and Monitoring with MPI." In Lecture Notes in Computer Science, 300–320. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-41321-1_16.
Full textMalainey, Mary E. "Instrumental Neutron Activation Analysis (INAA or NAA)." In Manuals in Archaeological Method, Theory and Technique, 427–32. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-5704-7_32.
Full textBracher, Johannes. "A New INARMA(1, 1) Model with Poisson Marginals." In Springer Proceedings in Mathematics & Statistics, 323–33. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-28665-1_24.
Full textConference papers on the topic "Inama"
Şıxəliyev, Ramiz. "Müasir şəbəkə təhlükəsizliyi və inam problemləri." In İnformasiya təhlükəsizliyinin aktual multidissiplinar elmi-praktiki problemləri. Institute of Information Technology of Azerbaijan National Academy of Sciences, 2018. http://dx.doi.org/10.25045/ncinfosec.2018.14.
Full textDas, H. A. "APPLICATION OF ANTI-COMPTON COUNTING IN INAA RESULTS." In Proceedings of the 2nd International Summer School. WORLD SCIENTIFIC, 1991. http://dx.doi.org/10.1142/9789814439305_0021.
Full textFarina, F., F. Pino, L. Sneyers, P. Vermaecker, H. Barros, L. Sajo-Bohus, Ma M. Mackowiak de Antczak, et al. "k0-INAA of Archaeological and Industrial Venezuelan Samples." In VII Latin American Symposium on Nuclear Physics and Applications. AIP, 2007. http://dx.doi.org/10.1063/1.2813854.
Full textRibeiro Jr., Iberê S., Frederico A. Genezini, Mitiko Saiki, and Guilherme S. Zahn. "Determination of 140La fission product interference factor for INAA." In XXXVI BRAZILIAN WORKSHOP ON NUCLEAR PHYSICS. AIP Publishing LLC, 2014. http://dx.doi.org/10.1063/1.4901781.
Full textSantos, Daniel Oliveira, CHRISTIAN WALLACE SANTOS MENESES, MELISSA VIEIRA GOMES, and LARISSA EMILY OGANDO DE JESUS SENA. "AS RESPOSTAS DA IMUNIDADE INATA NO PACIENTE COM CANDIDÍASE MUCOCUTÂNEA CRÔNICA." In II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/6752.
Full textRoberts, John W. "The International Nuclear Management Academy." In 2018 26th International Conference on Nuclear Engineering. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/icone26-81124.
Full textSilva, Laila Taniellen Rodrigues Da, Lucas Lopes Barbosa, Milane Da Silva Viana, Jorge Luis De Oliveira Borges, and Athauany Nogueira Dos Santos. "RELAÇÃO DA RESPOSTA IMUNE INATA E ADAPTATIVA À INFECÇÃO DE PARASITOS HELMÍNTICOS." In I Congresso Brasileiro de Parasitologia Humana On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1960.
Full textMARTINS, MARIA YZADORA MOURA, GISELLE FERREIRA DE SOUZA, CAMILA ALVES ROCHA, MARIA BEATRIZ DE CARVALHO SIMPLICIO LEOPOLDINO, and SILVIA FERNANDES RIBEIRO DA SILVA. "O PAPEL DA IMUNIDADE INATA NA INFECÇÃO CONTRA O SARS-COV-2." In II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/9679.
Full textKagawa, Masato, Nihan Karatas, and Michio Okada. "Communication Fundamentals within a Triadic Interaction in a Cooperative Play Mediated by INAMO." In HAI '17: The Fifth International Conference on Human-Agent Interaction. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3125739.3132602.
Full textPinto, Cassiane da Silva Portela, and MARCELLO VIEIRA DOS SANTOS. "RESPOSTA IMUNE NO TRATO URINÁRIO." In II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/5963.
Full textReports on the topic "Inama"
Dulski, P., and J. Luck. Determination of minor and trace elements in four Canadian iron-formation standard samples FeR-1, FeR-2, FeR-3, and FeR-4 by INAA, ICP-MS, and ICP-AES. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1993. http://dx.doi.org/10.4095/193240.
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