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Books on the topic 'Impulsive signal'

Author: Grafiati

Published: 1 April 2023

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1

Ambike, Sachin. Signal detection and filtering in the presence of alpha-Stable impulsive noise. Ottawa: National Library of Canada, 1993.

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2

Gersbacher, Ursula. Aussendienst und Verkauf: Automatische Zielensprache - unbewusste Signale erkennen - positive Impulse senden. München: Heyne, 1991.

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3

J, Evans D., United States. Occupational Safety and Health Administration, and Center for Manufacturing Engineering (U.S.), eds. Response of personal noise dosimeters to continuous and impulse-like signals. Gaithersburg, MD: Manufacturing Engineering Laboratory, National Institute of Standards and Technology, 1991.

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4

Nowożyński, Krzysztof. New methods for parameterization and determination of transfer functions and impulse responses. Warszawa: Institute of Geophysics, Polish Academy of Sciences, 2005.

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5

Duda, Timothy F. Smoothly modulated frequency-bounded impulse signals for tomography: By Timothy F. Duda and James F. Lynch. Woods Hole, Mass: Woods Hole Oceanographic Institution, 1991.

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6

International Conference on Ultrawideband and Ultrashort Impulse Signals (4th 2008 Sevastopolʹ ,Ukraine). 2008 4th International Conference on Ultrawideband and Ultrasho[r]t Impulse Signals: UWBUSIS 2008 : September 15-19, 2008, Sevastopol, Ukraine. [United States]: IEEE, 2008.

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7

UWBUSIS 2006 (2006 Sevastopolʹ, Ukraine). The third international conference, ultrawideband and ultrashort impulse signals: UWBUSIS 2006, September 19-22, 2004, Sevastopol, Ukraine. Piscataway, NJ: IEEE, 2006.

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8

LebensZeichen von Gott - für uns: Analysen und Impulse für eine zeitgemässe Sakramentenpastoral. Berlin: Lit, 2008.

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9

International Workshop on Ultrawideband and Ultrashort Impulse Signals (2nd 2004 Sevastpol, Ukraine). UWBUSIS 2004: 2004 Second International Workshop [on] Ultrawideband and Ultrashort Impulse Signals : September 19-22, 2004, Sevastopol, Ukraine. Piscataway, NJ: IEEE, 2004.

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10

Effect of signal jitter on the spectrum of rotor impulsive noise. Hampton, Va: National Aeronautics and Space Administration, Langley Research Center, 1987.

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11

Bechara, Antoine. Impulse Control Disorders in Neurological Settings. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0126.

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This chapter will argue that impulse control disorders, including addiction, are the product of an imbalance between two separate but interacting neural systems: (1) an impulsive amygdala-striatum–dependent neural system that promotes automatic and habitual behaviors and (2) a reflective prefrontal cortex–dependent neural system for decision making, forecasting the future consequences of a behavior, and inhibitory control. The reflective system controls the impulsive system via several mechanisms. However, this control is not absolute; hyperactivity within the impulsive system can override the reflective system. While most prior research has focused on the impulsive system (especially the ventral striatum and its mesolimbic dopamine projection) in promoting the motivation and drive to seek drugs, or on the reflective system (prefrontal cortex) and its mechanisms for decision making and impulse control, more recent evidence suggests that a largely overlooked structure, namely the insula, plays a key role in maintaining poor impulse control, including addiction. This review highlights the potential functional role the insula plays in addiction. We propose that the insula translates bottom-up, interoceptive signals into what subjectively may be experienced as an urge or craving, which in turn potentiates the activity of the impulsive system and/or weakens or hijacks the goal-driven cognitive resources that are needed for the normal operation of the reflective system.

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12

See Yuen Raymond.* Lee. Machine analysis of impulse radar signals for ice profiling. 1988.

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13

Institute of Electrical and Electronics. Uwbusis 2004: 2004 Second International Workshop [On] Ultrawideband and Ultrashort Impulse Signals: September 19-22, 2004, Sevastopo. Institute of Electrical & Electronics Enginee, 2004.

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14

(Editor), Susan Geideck, and Wolf-Andreas Liebert (Editor), eds. Sinnformeln: Linguistische Und Soziologische Analysen Von Leitbildewrn, Metaphern Und Anderen Kollektiven Orientierungsmustern (Linguistik: Impulse Und Tendenzen). Walter De Gruyter Inc, 2004.

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15

Hogh-Olesen, Henrik. Who Lives Here? Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190927929.003.0007.

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Chapter 6 follows the aesthetic impulse full circle and explores the human need to decorate objects and surroundings, as well as the marking of property and status in the public domain. Furthermore, it looks at phenomena such as folk art, street art, and graffiti. The purpose of the chapter is to show that such extravaganzas too make biological sense and thereby strengthen the argument that aesthetic behavior is natural for humans. Decorations signal personal fitness, ability, care, effort, resources, as well as power, because they ensure social status, for instance to attract more sexual partners. Like our body ornamentation, decoration is immediate communication transmitting key social and evolutionary information to the surroundings.

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16

Hogh-Olesen, Henrik. What a Sexy Tale! Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190927929.003.0004.

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Chapter 3 explores even further back in evolutionary history and examines whether there are traces of the aesthetic impulse in species other than our own. Do other species have a sense of aesthetics? Do they make aesthetic choices, and do they exhibit examples of aesthetic behavior? Among other things, this chapter looks at bird song and crane dance. It follows the fascinating bowerbirds as they create their remarkable and colorful constructions. And it follows the action as experiments are conducted with chimpanzees in the lab. The chapter also discusses honest signals, innate sensibilities, sexual selection, and the evolution of beauty.

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17

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Potassium in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0022.

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Potassium is an important electrolyte involved in transmission of electrical signals for muscle contraction, nerve impulse transmission, and cardiac function. The fetus accumulates potassium throughout gestation, but little is known about maternal potassium balance during pregnancy. Conditions associated with pregnancy, such as severe vomiting or morning sickness, can cause potassium loss. Caffeine increases the renal excretion of potassium, and cases of hypokalaemia in pregnancy have been observed in women with heavy caffeine/cola consumption, resulting in extreme muscle fatigue. To date there is insufficient evidence to suggest that the potassium requirement is increased during pregnancy, although a small increase in intake is needed for lactation. Reduction of cardiovascular risk factors and bone loss may be assisted by increasing potassium intake and/or by dietary sodium reduction.

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18

Gray, Erik. Animals. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198752974.003.0005.

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This chapter focuses on poetry’s frequent use of animals to explore the complexities of love. Animals feature in poems as objects of love, as lovers themselves, or in various other, more figurative, capacities. Although creatures of all kinds populate love poetry, birds are the most ubiquitous. The mating behaviors of birds, at once instinctive and highly patterned, offer a natural parallel to the combination of impulse and predetermined structure that characterizes both love and poetry. And while the same could be said of other animals, birds employ song as a key component of their courtship and so reflect the work of love poetry. A focus on birds and other animals also offers the poet scope to celebrate the role of sexual desire in love. Yet animals, in their mingled familiarity and alienness, ultimately appeal to love poets less as direct models than as signs of erotic uncertainty, queerness, and inconclusiveness.

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19

Tavares, Hermano. Assessment and Treatment of Pathological Gambling. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0091.

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As gambling becomes more popular, more people will be exposed to it; thus, the prevalence of and demand for gambling-related treatments are expected to increase. Pathological gambling (PG) is the most severe level of gambling compromise, characterized by unrestrained gambling to the point of financial and psychosocial harm. Classified among the impulse control disorders, PG resembles other addictive disorders. A host of scales for screening and diagnosing PG are available for both the specialist and the general practitioner. The diagnosis of PG, like that of other addictions, is based upon signs of loss of control over the target behavior (i.e., gambling), dose escalation (increasing amounts wagered to get the same excitement as in previous bets), withdrawal-like symptoms, psychosocial harm, persistent desire, and persistent betting despite the negative consequences. Its treatment requires thorough assessment of psychiatric related conditions, motivational intervention, gambling-focused psychotherapy, relapse prevention, and support for maintenance of treatment gains. Psychopharmacological tools to treat craving and gambling recurrence are an incipient but promising field.

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20

Jacobson, Matthew Frye. The Historian's Eye. University of North Carolina Press, 2019. http://dx.doi.org/10.5149/northcarolina/9781469649665.001.0001.

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Between 2009 and 2013, as the nation contemplated the historic election of Barack Obama and endured the effects of the Great Recession, Matthew Frye Jacobson set out with a camera to explore and document what was discernible to the "historian's eye" during this tumultuous period. Having collected several thousand images, Jacobson began to reflect on their raw, informal immediacy alongside the recognition that they comprised an archive of a moment with unquestionable historical significance. This book presents more than 100 images alongside Jacobson's recollections of their moments of creation and his understanding of how they link past, present, and future. The images reveal diverse expressions of civic engagement that are emblematic of the aspirations, expectations, promises, and failures of this period in American history. Myriad closed businesses and abandoned storefronts stand as public monuments to widespread distress; omnipresent, expectant Obama iconography articulates a wish for new national narratives; flamboyant street theater and wry signage bespeak a common impulse to talk back to power. Framed by an introductory essay, these images reflect the sober grace of a time that seems perilous, but in which “hope” has not ceased to hold meaning.

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21

Rosenmeyer, Patricia A. Talking with the Colossus. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190626310.003.0003.

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Chapter 3 explores the personal relationship visitors thought they had with Memnon. In response to Memnon’s morning cry, visitors used inscriptions to communicate with the articulate yet inanimate statue. Following two main impulses of animation, inscribers either addressed the god as a listener through the rhetorical figure of apostrophe or imagined him as a speaker, calling out to his mother or to them, through prosopopeia. The latter impulse overlaps with the concept of epiphany, where the god makes himself manifest by some sign—usually visual, but in this case aural. This chapter discusses apostrophe, prosopopeia, and epiphany as evidence for visitors’ yearning to commemorate their interactions with Memnon. Inserting themselves into the collective practice of sacred tourism, they nevertheless seek to make the verbal exchange meaningful on a personal level. The inscriptions bear witness to this tension between the communality and the uniqueness of each instance of communication with Memnon.

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22

Compston, Alastair. Multiple sclerosis and other demyelinating diseases. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0871.

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The oligodendrocyte–myelin unit subserves saltatory conduction of the nerve impulse in the healthy central nervous system. At one time, many disease processes were thought exclusively to target the structure and function of myelin. Therefore, they were designated ‘demyelinating diseases’. But recent analyses, based mainly on pathological and imaging studies, (re)emphasize that axons are also directly involved in these disorders during both the acute and chronic phases. Another ambiguity is the extent to which these are inflammatory conditions. Here, distinctions should be made between inflammation, as a generic process, and autoimmunity in which rather a specific set of aetiological and mechanistic conditions pertain. And there are differences between disorders that are driven primarily by immune processes and those in which inflammation occurs in response to pre-existing tissue damage.With these provisos, the pathological processes of demyelination and associated axonal dysfunction often account for episodic neurological symptoms and signs referable to white matter tracts of the brain, optic nerves, or spinal cord when these occur in young people. This is the clinical context in which the possibility of ‘demyelinating disease’ is usually considered by physicians and, increasingly, the informed patient. Neurologists will, with appropriate cautions, also be prepared to diagnose demyelinating disease in older patients presenting with progressive symptoms implicating these same pathways even when there is no suggestive past history. Both in its typical and atypical forms multiple sclerosis remains by far the commonest demyelinating disease. But acute disseminated encephalomyelitis, the leucodystrophies, and central pontine myelinolysis also need to be considered in particular circumstances; and multiple sclerosis itself has a differential diagnosis in which the relapsing-remitting course is mimicked by conditions not associated with direct injury to the axon–glial unit. Since our understanding of the cause, pathogenesis and features of demyelinating disease remains incomplete, classification combines aspects of the aetiology, clinical features, pathology, and laboratory components. Whether the designation ‘multiple sclerosis’ encapsulates one or more conditions is now much debated. We anticipate that a major part of future studies in demyelinating disease will be further to resolve this question of disease heterogeneity leading to a new taxonomy based on mechanisms rather than clinical empiricism. But, for now, the variable ages of onset, unpredictable clinical course, protean clinical manifestations, and non-specific laboratory investigations continue to make demyelinating disease one of the more challenging diagnostic areas in clinical neurology.

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