Relevant bibliographies by topics / Immunotoxicity

Academic literature on the topic 'Immunotoxicity'

Author: Grafiati
Published: 4 June 2021
Last updated: 30 January 2023

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Journal articles on the topic "Immunotoxicity"

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Nakamura, Kazuichi. "Immunotoxicity study." Folia Pharmacologica Japonica 131, no. 3 (2008): 215–19. http://dx.doi.org/10.1254/fpj.131.215.

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Collinge, Mark. "Developmental immunotoxicity." Drug Metabolism and Pharmacokinetics 34, no. 1 (January 2019): S4. http://dx.doi.org/10.1016/j.dmpk.2018.09.019.

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Cunningham, Morven, Marco Iafolla, Yada Kanjanapan, Orlando Cerocchi, Marcus Butler, Lillian L. Siu, Philippe L. Bedard, et al. "Evaluation of liver enzyme elevations and hepatotoxicity in patients treated with checkpoint inhibitor immunotherapy." PLOS ONE 16, no. 6 (June 11, 2021): e0253070. http://dx.doi.org/10.1371/journal.pone.0253070.

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Background and aims Immune checkpoint inhibitors (ICI) are increasingly used in cancer therapy. Elevated liver enzymes frequently occur in patients treated with ICI but evaluation is poorly described. We sought to better understand causes of liver enzyme elevation, investigation and management. Methods Patients treated with anti-PD-1, PDL-1 or CTLA-4 therapy in Phase I/II clinical trials between August 2012 and December 2018 were included. Clinical records of patients with significant liver enzyme elevations were retrospectively reviewed. Results Of 470 ICI-treated patients, liver enzyme elevation occurred in 102 (21.6%), attributed to disease progression (56; 54.9%), other drugs/toxins (7; 6.9%), other causes (22; 21.6%) and ICI immunotoxicity (17; 16.7%; 3.6% of total cohort). Immunotoxicity was associated with higher peak ALT than other causes of enzyme elevation (N = 17; M = 217, 95% CI 145–324 for immunotoxicity, N = 103; M = 74, 95% CI 59–92 for other causes; ratio of means 0.34, 95% CI 0.19–0.60, p = <0.001) and higher ALT:AST ratio (M = 1.27, 95% CI 0.78–2.06 for immunotoxicity, M = 0.69, 95% CI 0.59–0.80 for other causes, ratio of means 0.54, 95% CI 0.36–0.82, p = 0.004). Immunotoxicity was more often seen in patients with prior CPI exposure (41.2% of immunotoxicity vs 15.9% of patients without, p = 0.01), anti-CTLA-4 –containing ICI treatments (29.4% of immunotoxicity vs 6.8% of patients without, p = <0.001) and other organ immunotoxicity (76.5% of immunotoxicity vs 19.2% of patients without, p = <0.001). Cause for enzyme elevation was established in most patients after non-invasive investigation. Liver biopsy was reserved for four patients with atypical treatment response. Conclusions Liver enzyme elevation is common in patients receiving ICI, but often has a cause other than immunotoxicity. A biochemical signature with higher ALT and ALT/AST ratio, a history of prior ICI exposure and other organ immunotoxicities may help to identify patients at a higher likelihood of immunotoxicity. Liver biopsy can be safely deferred in most patients. We propose an approach to diagnostic evaluation in patients with liver enzyme elevations following ICI exposure.
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Wang, Xinge, Na Li, Mei Ma, Yingnan Han, and Kaifeng Rao. "Immunotoxicity in Vitro Assays for Environmental Pollutants under Paradigm Shift in Toxicity Tests." International Journal of Environmental Research and Public Health 20, no. 1 (December 24, 2022): 273. http://dx.doi.org/10.3390/ijerph20010273.

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With the outbreak of COVID-19, increasingly more attention has been paid to the effects of environmental factors on the immune system of organisms, because environmental pollutants may act in synergy with viruses by affecting the immunity of organisms. The immune system is a developing defense system formed by all metazoans in the course of struggling with various internal and external factors, whose damage may lead to increased susceptibility to pathogens and diseases. Due to a greater vulnerability of the immune system, immunotoxicity has the potential to be the early event of other toxic effects, and should be incorporated into environmental risk assessment. However, compared with other toxicity endpoints, e.g., genotoxicity, endocrine toxicity, or developmental toxicity, there are many challenges for the immunotoxicity test of environmental pollutants; this is due to the lack of detailed mechanisms of action and reliable assay methods. In addition, with the strong appeal for animal-free experiments, there has been a significant shift in the toxicity test paradigm, from traditional animal experiments to high-throughput in vitro assays that rely on cell lines. Therefore, there is an urgent need to build high-though put immunotoxicity test methods to screen massive environmental pollutants. This paper reviews the common methods of immunotoxicity assays, including assays for direct immunotoxicity and skin sensitization. Direct immunotoxicity mainly refers to immunosuppression, for which the assays mostly use mixed immune cells or isolated single cells from animals with obvious problems, such as high cost, complex experimental operation, strong variability and so on. Meanwhile, there have been no stable and standard cell lines targeting immune functions developed for high-throughput tests. Compared with direct immunotoxicity, skin sensitizer screening has developed relatively mature in vitro assay methods based on an adverse outcome pathway (AOP), which points out the way forward for the paradigm shift in toxicity tests. According to the experience of skin sensitizer screening, this paper proposes that we also should seek appropriate nodes and establish more complete AOPs for immunosuppression and other immune-mediated diseases. Then, effective in vitro immunotoxicity assay methods can be developed targeting key events, simultaneously coordinating the studies of the chemical immunotoxicity mechanism, and further promoting the paradigm shift in the immunotoxicity test.
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Sharma, R. P. "Evaluation of Pesticide Immunotoxicity." Toxicology and Industrial Health 4, no. 3 (July 1988): 373–80. http://dx.doi.org/10.1177/074823378800400309.

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Immunotoxicologic effects have been reported for a number of pesticides. Since pesticides represent a large range of chemical classes, different types of chemicals may affect the complex immune system by a variety of mechanisms. A preliminary evaluation of pesticides for immunotoxicologic potential can best be incorporated in general subacute and chronic toxicity testing, with additional groups assigned for initial host-sensitivity assays. For chemicals that are possible candidates for immunotoxicity in preliminary assays, a comprehensive immunotoxicity screening has been suggested. Finally, emphasis should be given to mechanistic investigations to objectively assess the immunotoxicity of a new chemical and possible extrapolation to man. Animal models need to be developed for detecting the autoimmunologic potential of pesticides. This paper provides a brief listing of various approaches currently employed in the evaluation of immunotoxicity.
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Chiappelli, Francesco, Michelle A. Kung, Pablo Villanueva, Patricia Lee, Patrick Frost, and Nerissa Prieto. "Immunotoxicity of Cocaethylene." Immunopharmacology and Immunotoxicology 17, no. 2 (January 1995): 399–417. http://dx.doi.org/10.3109/08923979509019759.

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SAKAGUCHI, Takehiro, Sanae SAKAGUCHI, and Yoshiro KUDO. "Immunotoxicity of Beryllium." Nippon Eiseigaku Zasshi (Japanese Journal of Hygiene) 52, no. 4 (1998): 611–17. http://dx.doi.org/10.1265/jjh.52.611.

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Putman, E., J. W. Laan, and H. Loveren. "Assessing immunotoxicity: guidelines." Fundamental and Clinical Pharmacology 17, no. 5 (October 2003): 615–26. http://dx.doi.org/10.1046/j.1472-8206.2003.00181.x.

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Laan, Jan Willem, and Henk Loveren. "Assessing immunotoxicity: guidelines." Fundamental and Clinical Pharmacology 19, no. 3 (June 2005): 329–30. http://dx.doi.org/10.1111/j.1472-8206.2005.00339.x.

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Pfau, Jean C. "Immunotoxicity of asbestos." Current Opinion in Toxicology 10 (August 2018): 1–7. http://dx.doi.org/10.1016/j.cotox.2017.11.005.

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Dissertations / Theses on the topic "Immunotoxicity"

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Karrow, Niel. "Chemical mixture immunotoxicity to rainbow trout." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape9/PQDD_0005/NQ44769.pdf.

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Bartlett, Alison. "QSAR study of immunotoxicity in antibiotics." Thesis, Liverpool John Moores University, 1995. http://researchonline.ljmu.ac.uk/5135/.

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Since their inception the B-Iactam antibiotics have become one of the most important classes of phannaceutical agents, both therapeutically and economically, in modern day usage for the treatment of a wide spectrum of bacterial infections. However, due to the versatility of bacteria many previously treatable species are developing resistance to the antibiotics currently available and so there is ever a need to develop more ~-lactam antibiotics, which are effective and yet safe. A major drawback to the ~-lactams is the degree of immunologically adverse reactions they induce. It was the aim of this study to develop both mechanistic and immunological methods to enable the prediction of a B-lactam's potential to induce an allergic response and to determine if a relationship between these responses and the molecular properties of the ~-lactams was present. In this study a database pertaining to frequency by which 70 p-lactams induce adverse reactions has been compiled and used to produce 27 QSAR models. A highly sensitive assay for the quantitation of cross-reactivity between B-lactams and serum anti-benzylpenicillin antibodies has been developed and used to determine the cross-reactivity potential of 31 ~-lactams and to develop 18 QSAR models. All of the QSARs developed suggest that the shape and electron separation of the ~-lactams are crucial to the development and extent of adverse response or crossreactivity induced by a specific p-lactam antibiotic, new or old. The QSARs developed will enable the design and development of new ~-lactam antibiotics which present a significantly lower risk of inducing immunologically mediated adverse responses when used therapeutically. Two sensitive assays for the quantitative detennination of the cytokines IL2 and IL4 in lymphocyte culture supernatants have been developed, and have been shown to have a potential use in the prediction of the type of immunological response initiated following p-Iactam stimulation of a sensitised individual.
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McDonald, Valerie Alexandra. "Evaluating Immunotoxicity of Quaternary Ammonium Compounds." Thesis, Virginia Tech, 2017. http://hdl.handle.net/10919/79723.

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Alkyl dimethyl benzyl ammonium chloride (ADBAC) and didecyl dimethyl ammonium chloride (DDAC) are common quaternary ammonium compounds used as disinfectants in households, medical, and restaurant settings. They cause occupational skin and respiratory hazards in humans, and developmental and reproductive toxicity in mice. They also cause increased secretions of proinflammatory cytokines in cell lines and vaginal inflammation in porcine models; but have not been evaluated for developmental immunotoxicity. We assessed immunotoxicity in-vitro with J774A.1 murine macrophage cell line by analyzing cytokine production and phagocytosis; and evaluated developmental immunotoxicity in CD-1 mice by analyzing antibody production. Additionally, because of the associations between gut microbiome dysbiosis and immune disease, we monitored changes in the microbiome as a result of ADBAC+DDAC exposure. Production of cytokines TNF-alpha and IL-6 increased at low ADBAC+DDAC concentrations, and IL-10 decreased in the murine macrophages with ADBAC+DDAC exposure. The phagocytic function of macrophages was also severely decreased. ADBAC+DDAC altered the mouse microbiome by decreasing the relative abundance of Bacteroides and increases in Clostridia in F0 and F1 generations. IgG primary and secondary responses were altered in F1 male mice; and IgA and IgM production were decreased in secondary response in F2 male mice. Since ADBAC+DDAC show signs of immunotoxicity in mice, further studies are needed to reassess risk for human exposure as ADBAC+DDAC may be contributing to immune disease.
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Maliji, Ghorban. "Immunotoxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in the rat." Thesis, University of Bristol, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.319022.

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McDonald, Jennifer C. Venables Barney J. "Bacterial challenge in Lumbricus terrestris a terrestrial invertebrate immunotoxicity model /." [Denton, Tex.] : University of North Texas, 2007. http://digital.library.unt.edu/permalink/meta-dc-3640.

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Baken, Kirsten Annika. "Immunotoxicogenomics gene expression profiling as a tool to study immunotoxicity /." [Maastricht] : Maastricht : Universitaire Pers Maastricht ; University Library, Universiteit Maastricht [host], 2007. http://arno.unimaas.nl/show.cgi?fid=9329.

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Rabideau, Christine L. "Pesticide Mixtures Induce Immunotoxicity: Potentiation of Apoptosis and Oxidative Stress." Thesis, Virginia Tech, 2001. http://hdl.handle.net/10919/34547.

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The three insecticides of interest were lindane (an organochlorine), malathion (an organophosphate) and piperonyl butoxide (PBO; a synergist). Based on minimum cytotoxicity (> LC25), the following concentrations were chosen for the pesticide mixture studies: 70μM lindane (Lind), 50μM malathion (Mal) and 55μM PBO. In the AlamarBlue cytotoxicity assay, individual pesticide and mixtures of malathion/PBO (MP) and malathion/lindane (ML) prompted cytotoxicity with varying intensities (Mal 18.8%, Lind 20.4%, PBO 23.5%, ML 53.6% and MP 64.9%). Cytopathological analysis revealed apoptotic features in treated cells and the DNA Ladder Assay confirmed the presence of DNA fragments. The specific mode of cell death was examined via the 7-aminoactinomycin D (7-AAD) Staining Assay. Apoptosis was detected in each treatment (Mal 6.5%, Lind 12.0%, PBO 13.2%, ML 19.3% and MP 23.4%). Furthermore, 7-AAD staining in combination with fluorescent-labeled monoclonal antibodies, PE-CD45RB/220 and FITC-CD90, was performed. B-cells were more susceptible to Mal and PBO treatments than were T-cells. The pro-oxidant activity of the pesticides was monitored via the Dichlorofluorescin Diacetate assay. Exposure to pesticides for 15 minutes increased H2O2 production above the controls, Mal 21.1%; Lind 10.8%; PBO 25.9%; ML 26.8%; MP 37.8%. The activities of antioxidant enzymes, glutathione peroxidase (GSH-Px) and glutathione reductase (GR) were altered by these treatments. GR was significantly reduced for the pesticide mixtures only (control: 51.7; Mal: 48.2; Lind: 50; PBO: 52.3; ML: 40.5; MP: 42 Units/mg). GSH-Px activity was severely reduced for all the pesticide treatments (control: 44.9; Mal: 30.2; Lind: 30.6; PBO: 32.4; ML: 21.1; MP: 21.1 Units/mg). These results indicate that exposure to these pesticide and pesticide mixtures induces apoptosis and oxidative stress.
Master of Science
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Olgun, Selen. "Immunotoxicity of Pesticide Mixtures and the Role of Oxidative Stress." Diss., Virginia Tech, 1998. http://hdl.handle.net/10919/11114.

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The immunotoxic effects of multiple pesticide exposure were evaluated. C57BL/6 mouse thymocytes were exposed to lindane, malathion, and permethrin, either separately or in mixtures of two pesticides, in concentrations ranging from 37.5 uM to 1mM. These exposures caused both apoptotic and necrotic cell death in thymocytes as evaluated by 7-aminoactinomycin-D, Annexin-V/PI, and lactate dehydrogenase release assays. When cells were exposed to lindane+malathion, or lindane+permethrin, a significantly greater-than-additive cytotoxicity was observed. The pesticide exposure caused DNA ladder formation with increased laddering in mixtures. Further, the effect of these pesticides on thymocyte oxidative stress was investigated. Thymocytes treated with any of these pesticides generated superoxide and H2O2. The lindane + malathion caused more-than-additive increase in superoxide production compared to single treatments of these pesticides. However, the effect of the lindane + permethrin was not significantly different from individual components of this mixture. The effects of pesticides on antioxidant enzymes were also investigated and only mixtures were found to have significant effects. Alteration in transcription factor NFkB level was measured as an indicator of oxidative stress in thymocytes following 12 h pesticide exposure, in vitro. Only lindane + malathion was found to increase the protein level. Furthermore, the effects of pesticides and their mixtures on immune functions of mice were studied in vivo. Animals (8-12 week old, male mice) were randomly divided into groups of six and injected intraperitoneally with three different doses (one-half, one-third, one-fourth, or one-eight of LD50) of individual pesticides. Exposure to individual pesticides did not alter the thymus/body or spleen/body weight ratios, thymic or splenic cell counts, or CD4/CD8 or CD45/CD90 ratios. However, anti-sRBC plaque forming cell (PFC) counts were significantly lowered with all treatments. Two other groups of animals were injected with lindane + malathion or lindane + permethrin at one-third of the LD50 of each pesticide. Exposure to pesticide mixtures did not alter the CD4/CD8 or CD45/CD90 ratios. However, the thymus/ and spleen/body weight ratios, thymic and splenic cell counts, and PFC counts were significantly lowered. These data indicate that lindane, malathion, and permethrin are immunotoxic and their mixtures can cause higher toxicity compared to individual exposures. In addition, these data support the hypothesis that oxidative stress were induced in thymocytes by exposure to these pesticides in vitro.
Ph. D.
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McDonald, Jennifer C. "Bacterial Challenge in Lumbricus Terrestris: A Terrestrial Invertebrate Immunotoxicity Model." Thesis, University of North Texas, 2007. https://digital.library.unt.edu/ark:/67531/metadc3640/.

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A bacterial challenge assay was developed utilizing the earthworm, Lumbricus terrestris, in order to assess potential immunotoxic effects from exposure to specific polychlorinated biphenyl congeners. Earthworms were inoculated with Aeromonous hydrophila, establishing a 10-day LD50. In vitro assays for effects of PCBs on phagocytosis agreed with mammalian studies, demonstrating potent suppression of phagocytosis by the non-coplanar PCB congener 138 and no suppression by the coplanar congener 126. However, when the effects of the two PCB congeners were evaluated for suppression of resistance to a whole animal infection challenge assay, coplanar PCB 126 decreased the ability of L. terrestris to withstand infection while non-coplanar PCB 138 did not.
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Mohammadian, Gholamreza. "Immunotoxicity of Chromium Contaminated Soil in the Earthworm, Lumbricus Terrestris." Thesis, University of North Texas, 1993. https://digital.library.unt.edu/ark:/67531/metadc501250/.

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Objective was to assess the toxicity of chromium (Cr) contaminated soil (CS) using the earthworm Lumbricus terrestris. Specific aims were to determine: (1) survival (LC50); .(2) immunotoxicity as indicated by lysozyme activity, coelomocyte counts, secretory (SR) and erythrocyte rosette (ER) formation, and phagocytosis; and (3) compare effects of CS exposure with those of Cr spiked artificial soil (AS) . CS Cr concentration was 8.78 mg/g with 98.2% being Cr^3+ and 1.8% being Cr^6+. Using 14 d AS protocol the LC50 was 6.49% CS: AS mixture. CS concentrations of 0.5, 1.0, 2.5 and 5.0% were sublethal, whereas 6.25, 12.5, 25, 50 and 100% CS were lethal. Sublethal exposure caused no immuno- modulation. Exposure to 50% CS: AS mixture for 5 d caused reduced SR and ER formation. Exposure to AS spiked with 0.27% Cr for 5 d resulted in immunomodulation equivalent to 50% CS: AS mixtures. Results indicated the CS to be acutely toxic.
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Books on the topic "Immunotoxicity"

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Dietert, Rodney R., ed. Immunotoxicity Testing. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-401-2.

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DeWitt, Jamie C., Cheryl E. Rockwell, and Christal C. Bowman, eds. Immunotoxicity Testing. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8549-4.

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Immunotoxicity testing: Methods and protocols. New York, NY: Humana Press, 2010.

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Dayan, A. D., R. F. Hertel, E. Heseltine, G. Kazantzis, E. M. Smith, and M. T. Van der Venne, eds. Immunotoxicity of Metals and Immunotoxicology. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-8443-4.

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D, Dayan Anthony, International Programme on Chemical Safety., Fraunhofer-Institut für Toxikologie und Aerosolforschung (Hannover, Germany), and Medizinische Hochschule Hannover, eds. Immunotoxicity of metals and immunotoxicology. New York: Plenum Press, 1990.

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Dietert, Rodney R., and Robert W. Luebke, eds. Immunotoxicity, Immune Dysfunction, and Chronic Disease. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-812-2.

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Dayan, Anthony D., R. F. Hertel, E. Heseltine, G. Kazantis, and E. B. Smith. Immunotoxicity of Metals and Immunotoxicology. Springer, 2012.

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DeWitt, Jamie C., Cheryl E. Rockwell, and Christal C. Bowman. Immunotoxicity Testing: Methods and Protocols. Humana, 2018.

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Dietert, Rodney R. Immunotoxicity Testing: Methods and Protocols. Humana Press, 2016.

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DeWitt, Jamie C., Cheryl E. Rockwell, and Christal C. Bowman. Immunotoxicity Testing: Methods and Protocols. Springer New York, 2019.

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Book chapters on the topic "Immunotoxicity"

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Nahler, Gerhard. "immunotoxicity." In Dictionary of Pharmaceutical Medicine, 89. Vienna: Springer Vienna, 2009. http://dx.doi.org/10.1007/978-3-211-89836-9_672.

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Hartung, Thomas. "Immunotoxicity." In Methods in Pharmacology and Toxicology, 241–67. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0521-8_11.

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Brousseau, Pauline, and Michel Fournier. "Aquatic Immunotoxicity." In Encyclopedia of Aquatic Ecotoxicology, 79–88. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-5704-2_8.

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Exon, J. H., and L. D. Koller. "Immunotoxicity of Cadmium." In Handbook of Experimental Pharmacology, 339–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70856-5_9.

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Petrarca, Claudia, Rocco Mangifesta, and Luca Di Giampaolo. "Immunotoxicity of Nanoparticles." In Current Topics in Environmental Health and Preventive Medicine, 75–94. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-4735-5_6.

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Salazar, Keith D., and Rosana Schafer. "Introduction to Immunotoxicity." In Molecular and Integrative Toxicology, 3–30. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-812-2_1.

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Taylor, Michael J. "Immunotoxicity of Trichothecene Mycotoxins." In Biodeterioration Research, 85–101. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-9453-3_6.

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Jeong, Tae Cheon. "Metabolism: Role in Immunotoxicity." In Encyclopedia of Immunotoxicology, 599–603. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54596-2_970.

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Keil, Deborah E. "Immunotoxicity of Perfluoroalkylated Compounds." In Toxicological Effects of Perfluoroalkyl and Polyfluoroalkyl Substances, 239–48. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15518-0_10.

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Dietert, Rodney R., and Leigh Ann Burns-Naas. "Developmental Immunotoxicity in Rodents." In Immunotoxicology Strategies for Pharmaceutical Safety Assessment, 271–97. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2008. http://dx.doi.org/10.1002/9780470386385.ch21.

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Conference papers on the topic "Immunotoxicity"

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Niu, Q., Q. Zhang, H. Li, L. Wang, and X. Lu. "1708b The immunotoxicity and neurotoxicity of aluminium." In 32nd Triennial Congress of the International Commission on Occupational Health (ICOH), Dublin, Ireland, 29th April to 4th May 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/oemed-2018-icohabstracts.139.

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Herrmann, Delia, Guido Seitz, Steven W. Warmann, Michael Bonin, Jörg Fuchs, and Sorin Armeanu-Ebinger. "Abstract 3416: Cetuximab promotes immunotoxicity against rhabdomyosarcoma in vitro." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3416.

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Zhernov, Yu V., A. O. Litovkina, I. V. Perminova, and M. R. Khaitov. "Immunotoxicity and allergenic properties of humic acids isolated from peloid." In Fifth International Conference of CIS IHSS on Humic Innovative Technologies «Humic substances and living systems». CLUB PRINT ltd., 2019. http://dx.doi.org/10.36291/hit.2019.zhernov.018.

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Ishihara, Jun, Aslan Mansurov, and Jeffrey Hubbell. "1227 Eliminating the immunotoxicity of interleukin-12 through protease-sensitive masking." In SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.1227.

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Prado Prado, Francisco, Esvieta Tenorio Borroto, and Humberto González-Díaz. "Computational model for multiplex assay of drug immunotoxicity in macrophage - study of the anti-microbial G1 using flow cytometry." In The 17th International Electronic Conference on Synthetic Organic Chemistry. Basel, Switzerland: MDPI, 2013. http://dx.doi.org/10.3390/ecsoc-17-e001.

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Young, Arabella, Vinh Nguyen, Jee Hye Kang, Sadaf Mehdizadeh, Amy Mei, Kathleen C. F. Sheehan, David V. Serreze, Yi-Guang Chen, Robert D. Schreiber, and Jeffrey A. Bluestone. "Abstract PR07: Developing syngeneic NOD tumor models to profile immunotoxicity and antitumor immunity in response to cancer immunotherapies in autoimmune-prone mice." In Abstracts: Fourth CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference: Translating Science into Survival; September 30 - October 3, 2018; New York, NY. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr18-pr07.

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She, Yue, Zhong Wang, Nan Wang, and Yanfei Li. "Notice of Retraction: Effects of Aluminum on Intracellular Calcium Homeostasis of Splenic Lymphocytes in Chickens Cultured In Vitro: Preliminary Study of Aluminum Immunotoxicity in Chickens." In 2011 5th International Conference on Bioinformatics and Biomedical Engineering. IEEE, 2011. http://dx.doi.org/10.1109/icbbe.2011.5781428.

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Reports on the topic "Immunotoxicity"

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Riedel, J. A., and D. R. Mattie. Immunotoxicity of Jet Fuels and Solvents. Fort Belvoir, VA: Defense Technical Information Center, November 2002. http://dx.doi.org/10.21236/ada453158.

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2

Kalinich, John F. Carcinogenicity and Immunotoxicity of Embedded Depleted Uranium and Heavy-Metal Tugsten Alloy in Rodents. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada428281.

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3

Kalinich, John F., ALexandra C. Miller, and David E. McClain. Carcinogenicity and Immunotoxicity of Embedded Depleted Uranium and Heavy-Metal Tungsten Alloy in Rodents. Fort Belvoir, VA: Defense Technical Information Center, October 2006. http://dx.doi.org/10.21236/ada458449.

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4

Carlson, E. A., Y. Li, and J. T. Zelikoff. Inhibition of CYP1A-Mediated Metabolism of Benzo(A)Pyrene (BAP): Effects Upon BAP-Induced Immunotoxicity in Japanese Medaka (Oryzias Latipes). Fort Belvoir, VA: Defense Technical Information Center, March 2001. http://dx.doi.org/10.21236/ada402076.

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5

NTP Immunotoxicity Technical Report on the Dermal Hypersensitivity and Irritancy Studies of 4-Methylcyclohexanemethanol (CASRN 34885-03-5) and Crude 4-Methylcyclohexanemethanol Administered Topically to Female BALB/c Mice. NIEHS, March 2020. http://dx.doi.org/10.22427/ntp-imm-01.

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