Books on the topic 'Immunosuppression'

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1

Thomson, Angus W., ed. Therapeutic Immunosuppression. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-010-0765-8.

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2

Eisen, Howard J., ed. Pharmacology of Immunosuppression. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-05118-0.

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3

Cochat, P., J. Traeger, C. Merieux, and M. Derchavane, eds. Immunosuppression under Trial. Dordrecht: Springer Netherlands, 1999. http://dx.doi.org/10.1007/978-94-011-4643-2.

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4

Specter, Steven, Mauro Bendinelli, and Herman Friedman, eds. Virus-Induced Immunosuppression. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5583-0.

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5

Taylor, Charles B. Immunosuppression: New research. New York: Nova Biomedical Books, 2009.

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6

Steven, Specter, Bendinelli Mauro, and Friedman Herman 1931-, eds. Virus-induced immunosuppression. New York: Plenum Press, 1989.

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7

Naor, David, Benjamin Y. Klein, Nora Tarcic, and Jonathan S. Duke-Cohan. Immunosuppression and Human Malignancy. Totowa, NJ: Humana Press, 1990. http://dx.doi.org/10.1007/978-1-4612-4496-7.

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8

David, Naor, ed. Immunosuppression and human malignancy. Clifton, N.J: Humana Press, 1989.

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9

W, Thomson Angus, ed. The Molecular biology of immunosuppression. Chichester: Wiley, 1992.

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10

Gruber, Scott A. Local Immunosuppression of Organ Transplants. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-662-22105-1.

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11

Szekeres-Bartho, Julia. Immunosuppression by progesterone in pregnancy. Boca Raton: CRC Press, 1992.

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12

Gruber, Scott A. Local immunosuppression of organ transplants. New York: Chapman & Hall, 1996.

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13

L, Adorini, ed. Selective immunosuppression: Basic concepts and clinical applications. Basle: Karger, 1994.

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14

Diego, Calif ). International Congress on Immunosuppression (2nd 2001 San. The 2nd International Congress on Immunosuppression: December 6-8, 2001, San Diego Marriott Hotel & Marina, San Diego, California, USA : program and abstracts. Secaucus, N.J: Professional Postgraduate Services/Thomson Healthcare, 2002.

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15

1958-, Hakim Nadey S., ed. Current immunosuppression: An update : proceedings of a round table meeting held in Prague on 6th December 1996. London: Royal Society of Medicine Press, 1997.

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16

Norbert, Gilmore, Wainberg Mark A, Medical Research Council (Canada), Canada. National Advisory Committee on AIDS., and Workshop on Viral Mechanisms of Immunosuppression (1984 : Montréal, Quebec), eds. Viral mechanisms of immunosuppression: Proceedings of a workshop. New York: Liss, 1985.

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17

José, Strauss, and Strauss Louise, eds. Growth, immunosuppression, and renal disorders in neonates and children: Current concepts in diagnosis and management. Coral Gables, Fla: University of Miami Press, 1989.

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18

Bray, Peter William. Allograft microvascular epiphyseal plate transplants with short-term immunosuppression. Ottawa: National Library of Canada, 1999.

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19

J, Klastersky, ed. Infectious complications of cancer. Boston: Kluwer Academic Publishers, 1995.

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20

B, Vogelsang Georgia, and Pavletic Steven, eds. Chronic graft versus host disease: Interdisciplinary management. Cambridge [England] ; New York: Cambridge University Press, 2009.

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21

J, Smialowicz Ralph, and United States. Environmental Protection Agency, eds. Comparative immunosuppression of various glycol ethers orally administered to Fischer 344 rats. [Washington, D.C.?: U.S. Environmental Protection Agency, 1992.

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22

Schapira, David V. The diagnosis and management of the cancer patient with sepsis. New York, NY: Biomedical Information Corp., 1986.

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23

C, Tetta, ed. Immunopharmacology of the renal system. London: Academic Press, 1993.

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24

Kuypers, Dirk R. J., and Maarten Naesens. Immunosuppression. Edited by Jeremy R. Chapman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0281_update_001.

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Combination immunosuppressive therapy produces excellent short-term results after kidney transplantation. Long-term graft survival has improved, but less dramatically. Death with a functioning graft remains the primary cause of graft loss. Dosing of current immunosuppressive therapy balances between careful clinical interpretation of time-driven immunological risk assessments and drug-related toxicity on the one hand, and the use of simple surrogate drug exposure indicators like blood/plasma concentrations on the other. The combined use of calcineurin-inhibitors (CNIs) with mycophenolic acids and corticosteroids has been fine-tuned over the last decade, based on empirically derived observations as well as on the results of large multicentre randomized clinical studies. Corticosteroid withdrawal and avoidance are feasible, at least in patients with a low immunological risk, but CNI-free protocols have had few long-term successes. Some minimization strategies have increased risk of developing acute rejection or (donor-specific) anti-HLA antibodies, with deleterious effects on the graft. Mammalian target of rapamycin inhibitors (mTORi) have shown limited benefit in early CNI replacement regimens and their long-term use as primary drug is hampered by intolerance. In the setting of particular malignant disease occurring after transplantation, such as squamous cell carcinoma of the skin and Kaposi’s sarcoma, mTORi seem promising. Induction agents (anti-interleukin 2 receptor monoclonal antibodies, antithymocyte globulins) effectively diminish the risk of early immunological graft loss in recipients with moderate to high immunological risk but at the price of more infectious or malignant complications. While personalized transplantation medicine is only in its early stages of development, attempts are made to quantitatively measure the clinical degree of immunosuppression, to tailor immunosuppressive therapy more specifically to the patient’s individual profile, and to monitor graft status by use of invasive (e.g. surveillance renal biopsies) and non-invasive biomarkers. These scientific endeavours are a necessity to further optimize the current immunosuppressive therapy which will remain for some time to come.
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25

He, Xuehui, ed. Immunosuppression. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.87403.

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26

He, Xuehui. Immunosuppression. IntechOpen, 2020.

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27

Therapeutic Immunosuppression (Immunology and Medicine, Volume 29). Springer, 2001.

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28

Thomson, A. W. Therapeutic Immunosuppression. Ingramcontent, 2012.

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29

Thomson, A. W. Therapeutic Immunosuppression. Springer, 2012.

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30

Specter, Steven, Herman Friedman, and Mauro Bendinelli. Virus-Induced Immunosuppression. Springer, 2012.

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31

Specter, Steven, Herman Friedman, and Mauro Bendinelli. Virus-Induced Immunosuppression. Springer, 2012.

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32

Wijdicks, Eelco F. M., and Sarah L. Clark. Immunosuppression and Immunotherapy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0010.

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Immune modulation in the neurosciences intensive care unit mostly involves high-dose corticosteroids, plasma exchange, and immunoglobulin. Corticosteroids are frequently used in patients with neurologic complications of cancer. Neurosurgeons typically use corticosteroids after performing a craniectomy to reduce cerebral edema. Corticosteroids are the established initial treatment modality of choice for patients with acute metastatic epidural spinal cord compression. Therapeutic apheresis or immunoglobulin is generally used as supportive therapy in patients with Guillain-Barré syndrome, myasthenia crisis, and autoimmune encephalitis. Immune modulation has been considered essential in autoimmune encephalitis despite lack of controlled clinical trials. In these now better characterized disorders, a combination of corticosteroids, intravenous immune globulin (IVIG), plasma exchange, rituximab, or cyclophosphamide are used. The use of acute immunotherapy and precautionary measures are discussed in this chapter.
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33

Pharmacology of Immunosuppression. Springer International Publishing AG, 2023.

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34

Eisen, Howard J. Pharmacology of Immunosuppression. Springer International Publishing AG, 2022.

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35

Ginns, Leo C. Immunosuppression In Transplantation. Blackwell Publishing, 1999.

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36

Cochat, Pierre. Immunosuppression Under Trial. Springer, 1999.

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37

Virus-Induced Immunosuppression. Island Press, 1989.

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38

Tyagi, Rajeev K., and Prakriti Sharma. Immunosuppression and Immunomodulation. IntechOpen, 2023.

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39

Carmona, Loreto, and Maria Galindo. Perioperative management of immunosuppression. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0093.

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Many rheumatologists are concerned about the complications of maintaining immunosuppression at the time of elective surgery. Complications may be increased risk of infection, or a slow healing of the surgical wound. On the other hand, stopping medication may place the patient at undesirable levels of disease activity. Many rheumatologists actually act on art, stopping the drug perioperatively depending on blood count, drug type and dose, previous postoperative complications, type of surgery, and other factors. The timing of stopping treatment preoperatively and restarting after surgery also significantly differ among rheumatologists. All in all, there is a great variability in clinical practice related to the perioperative use of immunosuppressants. This chapter reviews the evidence on methotrexate, glucocorticoids, and biological therapies related to the perioperative period, and it proposes some recommendations on the management of immunosuppressant drugs in rheumatic diseases. Factors that may be related to complications will also be reviewed.
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40

Carmona, Loreto, and Maria Galindo. Perioperative management of immunosuppression. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0093_update_003.

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Many rheumatologists are concerned about the complications of maintaining immunosuppression at the time of elective surgery. Complications may be increased risk of infection, or a slow healing of the surgical wound. On the other hand, stopping medication may place the patient at undesirable levels of disease activity. Many rheumatologists actually act on art, stopping the drug perioperatively depending on blood count, drug type and dose, previous postoperative complications, type of surgery, and other factors. The timing of stopping treatment preoperatively and restarting after surgery also significantly differ among rheumatologists. All in all, there is a great variability in clinical practice related to the perioperative use of immunosuppressants. This chapter reviews the evidence on methotrexate, glucocorticoids, and biological therapies related to the perioperative period, and it proposes some recommendations on the management of immunosuppressant drugs in rheumatic diseases. Factors that may be related to complications will also be reviewed.
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41

Personalized Immunosuppression in Transplantation. Elsevier, 2016. http://dx.doi.org/10.1016/c2013-0-19247-1.

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42

Immunosuppression and Human Malignancy. Humana Press, 2011.

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43

Naor, David, Benjamin Y. Klein, Nora Tarcic, and Jonathan S. Duke-Cohan. Immunosuppression and Human Malignancy. Humana Press, 2011.

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44

Naor, David, Benjamin Y. Klein, Nora Tarcic, and Jonathan S. Duke-Cohan. Immunosuppression and Human Malignancy. Humana Press, 2012.

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45

Gruber, Scott A. Local Immunosuppression of Organ Transplants. Springer, 2013.

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46

Immunosuppression and Immunomodulation [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.102203.

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47

Szekeres-Bartho, Julia. Immunosuppression by Progesterone in Pregnancy. Taylor & Francis Group, 1992.

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48

Hartung, H. P. Selective Immunosuppression (Neuroscience Intelligence Unit). Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 1996.

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49

Noonan, F. Uv-B Radiation and Immunosuppression. Chapman & Hall, 1996.

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50

Gruber, Scott A. Local Immunosuppression of Organ Transplants. Springer London, Limited, 2013.

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