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Journal articles on the topic 'Immunological aspects'

Author: Grafiati
Published: 4 June 2021
Last updated: 31 July 2024

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1

Fauchère, Jean-Louis, and Leif P. Andersen. "Immunological aspects." Current Opinion in Gastroenterology 11 (1995): 21–24. http://dx.doi.org/10.1097/00001574-199501001-00005.

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2

Engstrand, Lars, and J. Dominique de Korwin. "Immunological aspects." Current Opinion in Gastroenterology 12 (January 1996): 21–23. http://dx.doi.org/10.1097/00001574-199601001-00005.

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3

Adebajo, Adewale O., and David A. Isenberg. "Immunological aspects." Baillière's Clinical Rheumatology 9, no. 1 (February 1995): 215–29. http://dx.doi.org/10.1016/s0950-3579(05)80157-8.

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4

Heyworth, Martin F. "Immunological aspects ofGiardiainfections." Parasite 21 (2014): 55. http://dx.doi.org/10.1051/parasite/2014056.

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5

Overeem, Sebastiaan, John Logan Black, and Gert Jan Lammers. "Narcolepsy: Immunological aspects." Sleep Medicine Reviews 12, no. 2 (April 2008): 95–107. http://dx.doi.org/10.1016/j.smrv.2007.07.010.

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6

Grogan, Laura F., Josephine E. Humphries, Jacques Robert, Chantal M. Lanctôt, Catherine J. Nock, David A. Newell, and Hamish I. McCallum. "Immunological Aspects of Chytridiomycosis." Journal of Fungi 6, no. 4 (October 19, 2020): 234. http://dx.doi.org/10.3390/jof6040234.

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Amphibians are currently the most threatened vertebrate class, with the disease chytridiomycosis being a major contributor to their global declines. Chytridiomycosis is a frequently fatal skin disease caused by the fungal pathogens Batrachochytrium dendrobatidis (Bd) and Batrachochytrium salamandrivorans (Bsal). The severity and extent of the impact of the infection caused by these pathogens across modern Amphibia are unprecedented in the history of vertebrate infectious diseases. The immune system of amphibians is thought to be largely similar to that of other jawed vertebrates, such as mammals. However, amphibian hosts are both ectothermic and water-dependent, which are characteristics favouring fungal proliferation. Although amphibians possess robust constitutive host defences, Bd/Bsal replicate within host cells once these defences have been breached. Intracellular fungal localisation may contribute to evasion of the induced innate immune response. Increasing evidence suggests that once the innate defences are surpassed, fungal virulence factors suppress the targeted adaptive immune responses whilst promoting an ineffectual inflammatory cascade, resulting in immunopathology and systemic metabolic disruption. Thus, although infections are contained within the integument, crucial homeostatic processes become compromised, leading to mortality. In this paper, we present an integrated synthesis of amphibian post-metamorphic immunological responses and the corresponding outcomes of infection with Bd, focusing on recent developments within the field and highlighting future directions.
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7

Chuhlovina, M. L., and E. A. Bichun. "IMMUNOLOGICAL ASPECTS OF NEUROSYPHILIS." Russian Journal of Infection and Immunity 5, no. 2 (June 28, 2015): 131–36. http://dx.doi.org/10.15789/2220-7619-2015-2-131-136.

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8

Bender, T. "Immunological aspects of balneology." Boletin Sociedad Española Hidrologia Medica 33, S1 (2018): 67. http://dx.doi.org/10.23853/bsehm.2018.0587.

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9

Teplen’kiy, M. P., M. V. Chepeleva, and E. I. Kuznetsova. "PERTHES DISEASE: IMMUNOLOGICAL ASPECTS." Russian Clinical Laboratory Diagnostics 65, no. 4 (April 15, 2020): 239–43. http://dx.doi.org/10.18821/0869-2084-2020-65-4-239-243.

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Considering a stage of the pathological process patients (boys at the age of 8-12 years) were divided into two (2) groups. Group I included 14 patients with the fragmentation stage (Perthes disease Stage II). Group 2 included 15 children with Perthes disease Stage III (the stage of re-ossification). Perthes disease regardless of the stage of the disease was characterized by the increase in oxygen-dependent and lysosomal phagocytic activity of neutrophils, the increase in the number of early extracellular traps, as well as by increased concentrations of pro-inflammatory cytokines (IL-1β and TNFa), IgE, decreased concentrations of IL-18. The fragmentation stage was characterized by moderate activation of cellular immunity with a prevailing increase in the number of T-lymphocytes with early activation markers (CD25). At the re-ossification stage the predominance of T-lymphocytes was observed with late activation markers (HLADR), being accompanied by moderate activation of humoral immunity (increased concentrations of class A and G serum immunoglobulins). The obtained data can be used as additional criteria for clarifying Perthes disease stage, predicting osteonecrosis development when making decision of the feasibility of performing reconstructive surgeries on the joint.
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10

Scheithauer, Heike, Claus Belka, Kirsten Lauber, and Udo S. Gaipl. "Immunological aspects of radiotherapy." Radiation Oncology 9, no. 1 (2014): 185. http://dx.doi.org/10.1186/1748-717x-9-185.

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11

Sethi, D., and E. R. Maher. "Immunological Aspects of Glomerulonephritis." Journal of the Royal Society of Medicine 80, no. 3 (March 1987): 189–91. http://dx.doi.org/10.1177/014107688708000322.

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12

Woollard, Kevin J. "Immunological aspects of atherosclerosis." Clinical Science 125, no. 5 (May 7, 2013): 221–35. http://dx.doi.org/10.1042/cs20120576.

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Cardiovascular disease is the leading cause of death in several countries. The underlying process is atherosclerosis, a slowly progressing chronic disorder that can lead to intravascular thrombosis. There is overwhelming evidence for the underlying importance of our immune system in atherosclerosis. Monocytes, which comprise part of the innate immune system, can be recruited to inflamed endothelium and this recruitment has been shown to be proportional to the extent of atherosclerotic disease. Monocytes undergo migration into the vasculature, they differentiate into macrophage phenotypes, which are highly phagocytic and can scavenge modified lipids, leading to foam cell formation and development of the lipid-rich atheroma core. This increased influx leads to a highly inflammatory environment and along with other immune cells can increase the risk in the development of the unstable atherosclerotic plaque phenotype. The present review provides an overview and description of the immunological aspect of innate and adaptive immune cell subsets in atherosclerosis, by defining their interaction with the vascular environment, modified lipids and other cellular exchanges. There is a particular focus on monocytes and macrophages, but shorter descriptions of dendritic cells, lymphocyte populations, neutrophils, mast cells and platelets are also included.
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13

Latinne, D., Y. Vandeput, M. De Bruyere, F. Bottazzo, G. Sokal, and J. Crabbe. "Addison's disease: immunological aspects*." Tissue Antigens 30, no. 1 (December 11, 2008): 23–24. http://dx.doi.org/10.1111/j.1399-0039.1987.tb01591.x.

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14

De Sanctis, G., A. Frezzolini, S. Cadoni, R. Perricone, V. Bottari, and L. Fontana. "Raynaud's Phenomenon: Immunological Aspects." International Journal of Immunopathology and Pharmacology 9, no. 2 (September 1996): 33. http://dx.doi.org/10.1177/039463209600900216.

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15

Vinatier, D. "Immunological aspects of endometriosis." Human Reproduction Update 2, no. 5 (September 1, 1996): 371–284. http://dx.doi.org/10.1093/humupd/2.5.371.

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16

MOUNTFORD, A. P. "Immunological aspects of schistosomiasis." Parasite Immunology 27, no. 7-8 (July 2005): 243–46. http://dx.doi.org/10.1111/j.1365-3024.2005.00798.x.

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17

de Rie, M. "Immunological aspects of psoriasis." Netherlands Journal of Medicine 53, no. 3 (September 1998): 143–44. http://dx.doi.org/10.1016/s0300-2977(98)00090-4.

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18

Zarczuk, Radosław, Dariusz Łukasik, Marian Jędrych, and Kinga K. Borowicz. "Immunological aspects of epilepsy." Pharmacological Reports 62, no. 4 (July 2010): 592–607. http://dx.doi.org/10.1016/s1734-1140(10)70317-0.

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19

Corrigan, Chris J. "Immunological Aspects of Asthma." Clinical Immunotherapeutics 1, no. 1 (January 1994): 31–42. http://dx.doi.org/10.1007/bf03258489.

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20

Bárány, Peter, and Ingela Fehrman-Ekholm. "Immunological Aspects of Haemodialysis." Clinical Immunotherapeutics 1, no. 6 (June 1994): 469–80. http://dx.doi.org/10.1007/bf03259039.

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21

Roubey, Robert A. S. "Antiphospholipid antibodies: immunological aspects." Clinical Immunology 112, no. 2 (August 2004): 127–28. http://dx.doi.org/10.1016/j.clim.2004.02.010.

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22

Čolić, Miodrag, Sergej Tomić, and Marina Bekić. "Immunological aspects of nanocellulose." Immunology Letters 222 (June 2020): 80–89. http://dx.doi.org/10.1016/j.imlet.2020.04.004.

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23

Pradalier, A., and J. M. Launay. "Immunological aspects of migraine." Biomedicine & Pharmacotherapy 50, no. 2 (January 1996): 64–70. http://dx.doi.org/10.1016/0753-3322(96)84715-9.

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24

Carnevale, Daniela, and Giuseppe Lembo. "Immunological Aspects of Hypertension." High Blood Pressure & Cardiovascular Prevention 23, no. 2 (April 14, 2016): 91–95. http://dx.doi.org/10.1007/s40292-016-0141-8.

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25

Dmowski, W. P. "Immunological aspects of endometriosis." International Journal of Gynecology & Obstetrics 50 (September 1995): S3—S10. http://dx.doi.org/10.1016/0020-7292(95)02508-a.

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26

Garrido-Urbani, S., M. Meguenani, F. Montecucco, and B. A. Imhof. "Immunological aspects of atherosclerosis." Seminars in Immunopathology 36, no. 1 (November 9, 2013): 73–91. http://dx.doi.org/10.1007/s00281-013-0402-8.

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27

Matt, K., K. Reimand, Ü. Kadastik, K. Metskula, and R. Uibo. "Immunological aspects of infertility." International Journal of Gynecology & Obstetrics 70 (2000): B136. http://dx.doi.org/10.1016/s0020-7292(00)83144-6.

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28

Yip, Leona, James McCluskey, and Rodney Sinclair. "Immunological aspects of pregnancy." Clinics in Dermatology 24, no. 2 (March 2006): 84–87. http://dx.doi.org/10.1016/j.clindermatol.2005.10.022.

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29

Ringvold, Amund. "EXFOLIATION SYNDROME IMMUNOLOGICAL ASPECTS." Acta Ophthalmologica 66, S184 (May 28, 2009): 35–43. http://dx.doi.org/10.1111/j.1755-3768.1988.tb02626.x.

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30

Aarli, Johan A. "Immunological aspects of epilepsy." Brain and Development 15, no. 1 (January 1993): 41–49. http://dx.doi.org/10.1016/0387-7604(93)90005-s.

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31

Schirrmacher, V. "Immunological aspects in oncology." Journal of Cancer Research and Clinical Oncology 115, no. 5 (September 1989): 491–93. http://dx.doi.org/10.1007/bf00393347.

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32

Tsirogianni, Afrodite K., Niki Maria Moutsopoulos, and Haralampos M. Moutsopoulos. "Wound healing: Immunological aspects." Injury 37, no. 1 (April 2006): S5—S12. http://dx.doi.org/10.1016/j.injury.2006.02.035.

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33

DeGraba, Thomas J. "Immunological Aspects of Acute Stroke." BioDrugs 13, no. 1 (January 2000): 1–8. http://dx.doi.org/10.2165/00063030-200013010-00001.

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34

Garzetti, Giuseppe G., Maurizio Cignitti, Floriana Marchegiani, Andrea L. Tranquilli, and Carlo Romanini. "Immunological Aspects of Pregnancy Hypertension." Clinical and Experimental Hypertension. Part B: Hypertension in Pregnancy 7, no. 1-2 (January 1988): 113–19. http://dx.doi.org/10.3109/10641958809023508.

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35

Timasheva, Ya R. "IMMUNOLOGICAL ASPECTS OF ESSENTIAL HYPERTENSION." Medical Immunology (Russia) 21, no. 3 (July 13, 2019): 407–18. http://dx.doi.org/10.15789/1563-0625-2019-3-407-418.

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According to modern concept of the etiopathogenesis of essential hypertension, immune cells play an important role in its development. Mediators produced by immunocompetent cells participate in the initiation and maintenance of chronic systemic inflammation and promote the development of vascular remodeling which is an important part of the pathogenesis of the disease and target organ damage. The immune mechanisms underlying blood pressure elevation include the activation of innate and adaptive immune cells. Endothelial damage triggers an inflammatory cascade, causing migration of the immune cells to the inflammatory site, mediated by chemokines and adhesion molecules. Macrophage infiltration of perivascular tissue contributes to impaired vasodilation and damage to target organs due to the production of active forms of oxygen. Angiotensin II also causes T cell infiltration of perivascular adipose tissue and adventitia and an increased production of tumor necrosis factor alpha and interferon gamma. In addition, T lymphocytes express the mineralocorticoid receptor involved in the development of systemic hypertension. An important role in the progression of hypertension belongs to interleukin-17, which is involved in blood pressure elevation and vascular remodeling. The review also contains data on the effect of gut microbiota on the regulation of blood pressure and the development of hypertension.
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36

Nickerson, S. C. "Immunological Aspects of Mammary Involution." Journal of Dairy Science 72, no. 6 (June 1989): 1665–78. http://dx.doi.org/10.3168/jds.s0022-0302(89)79278-x.

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37

Czerwaty, Katarzyna, Katarzyna Piszczatowska, Jacek Brzost, Nils Ludwig, Mirosław J. Szczepański, and Karolina Dżaman. "Immunological Aspects of Chronic Rhinosinusitis." Diagnostics 12, no. 10 (September 29, 2022): 2361. http://dx.doi.org/10.3390/diagnostics12102361.

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Chronic rhinosinusitis (CRS) is related to persistent inflammation with a dysfunctional relationship between environmental agents and the host immune system. Disturbances in the functioning of the sinus mucosa lead to common clinical symptoms. The major processes involved in the pathogenesis of CRS include airway epithelial dysfunctions that are influenced by external and host-derived factors which activate multiple immunological mechanisms. The molecular bases for CRS remain unclear, although some factors commonly correspond to the disease: bacterial, fungal and viral infections, comorbidity diseases, genetic dysfunctions, and immunodeficiency. Additionally, air pollution leads increased severity of symptoms. CRS is a heterogeneous group of sinus diseases with different clinical courses and response to treatment. Immunological pathways vary depending on the endotype or genotype of the patient. The recent knowledge expansion into mechanisms underlying the pathogenesis of CRS is leading to a steadily increasing significance of precision medicine in the treatment of CRS. The purpose of this review is to summarize the current state of knowledge regarding the immunological aspects of CRS, which are essential for ensuring more effective treatment strategies.
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38

Belova, O. V., T. I. Arefieva, and S. N. Moskvina. "Immunological aspects of Parkinson's disease." Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 120, no. 2 (2020): 110. http://dx.doi.org/10.17116/jnevro2020120021110.

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39

Vikulov, G. Kh. "Immunological aspects of herpesvirus infections." Klinicheskaya dermatologiya i venerologiya 14, no. 5 (2015): 104. http://dx.doi.org/10.17116/klinderma2015145104-114.

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40

Martins-Filho, Agrimaldo, Millena Prata Jammal, Eddie Fernando Candido Murta, and Rosekeila Simões Nomelini. "Immunological aspects of ovarian malignancy." Clinical and Experimental Obstetrics & Gynecology 49, no. 2 (February 8, 2022): 035. http://dx.doi.org/10.31083/j.ceog4902035.

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41

Sarno, Euzenir Nunes. "The immunological aspects of leprosy." Memórias do Instituto Oswaldo Cruz 82, suppl 2 (1987): 159–62. http://dx.doi.org/10.1590/s0074-02761987000600024.

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42

Kumar, Vijay, and Asha Kumar. "Immunological aspects of corneal transplant." Immunological Investigations 43, no. 8 (October 8, 2014): 888–901. http://dx.doi.org/10.3109/08820139.2014.910024.

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43

Kwak, Yi-Sub, Chul-Woo Kim, and Young-Ho Paik. "Immunological Aspects of Contemporary Exercise." Journal of Life Science 17, no. 8 (August 30, 2007): 1166–71. http://dx.doi.org/10.5352/jls.2007.17.8.1166.

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44

Kay, A. B. "Immunological Aspects of Chronic Asthma." Allergy and Asthma Proceedings 8, no. 5 (September 1, 1987): 297–300. http://dx.doi.org/10.2500/108854187779023541.

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45

Stoll, Guido, Sebastian Jander, Mario Siebler, and Michael Schroeter. "Immunological Aspects of Ischaemic Stroke." CNS Drugs 14, no. 3 (September 2000): 213–28. http://dx.doi.org/10.2165/00023210-200014030-00004.

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46

Gergert, V. Ja, M. M. Averbakh, and A. E. Ergeshov. "Immunological aspects of tuberculosis pathogenesis." Terapevticheskii arkhiv 91, no. 11 (November 15, 2019): 90–97. http://dx.doi.org/10.26442/00403660.2019.11.000262.

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The morphological aspects of TB pathogenesis are well described in the publications. Much is also known about the main stages of development and formation of specific adaptive immunity. However, from our point of view, not enough attention is being paid to the involvement of the immune system in the pathogenesis of clinically relevant TB abnormalities, as well as various forms of the disease. Nevertheless, there is no doubt that the variety of clinical manifestations of any disease associated with the penetration of a foreign agent into the body, and Mycobacterium tuberculosis (MTB) in particular, is due to the collective interaction of the infectious agent and the individual response of the macroorganism to this infectious agent. The mosaic of such interactions usually imposes its own adjustments on the development of different forms of the process, its speed and direction, as well as the outcomes. Certainly, the response of a macroorganism to MTB is an integral part of pathogenesis and consists of many general components including the responses associated with the mechanisms of natural and acquired immunity. Intensity of these reactions depends on the characteristics of an agent (MTB) and a macroorganism. For the development of TB disease, massiveness of TB infection, dose and duration of MTB exposure to the human body, as well as virulence of MTB and the level of body's protection during the exposure play a very important role. TB pathogenesis is somewhat different in primary MTB infection and re - infection. With primary infection, 88-90% of individuals do not have clinical manifestations, and only the tuberculin skin test conversion signals the onset of infection. In some cases, without any use of anti-TB drugs limited abnormalities may result in spontaneous cure with the minimal residual changes in the lungs, intrathoracic lymph nodes and tissues of other organs, often in the form of calcifications and limited areas of fibrosis in more advanced cases. Only 10-12% of newly infected individuals develop TB with severe clinical manifestations requiring TB therapy. The absence of clinical manifestations of primary TB infection can be explained by a high level of natural resistance of the human body to tuberculosis, and sometimes can be an effect of acquired protection due to BCG vaccination. This review attempts to discuss the role of immune mechanisms in the pathogenesis both at the beginning of disease development, and in the process of its various manifestations. Issues of genetically determined resistance or susceptibility to TB are not being covered in detail in this manuscript.
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47

Brand, A. "Immunological aspects of blood transfusions." Blood Reviews 14, no. 3 (September 2000): 130–44. http://dx.doi.org/10.1054/blre.2000.0131.

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48

Mueller-Loebnitz, Christoph, Helmut Ostermann, Anke Franzke, Juergen Loeffler, Lutz Uharek, Max Topp, and Hermann Einsele. "Immunological Aspects ofCandidaandAspergillusSystemic Fungal Infections." Interdisciplinary Perspectives on Infectious Diseases 2013 (2013): 1–7. http://dx.doi.org/10.1155/2013/102934.

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Patients with allogeneic stem cell transplantation (SCT) have a high risk of invasive fungal infections (IFIs) even after neutrophil regeneration. Immunological aspects might play a very important role in the IFI development in these patients. Some data are available supporting the identification of high-risk patients with IFI for example patients receiving stem cells from TLR4 haplotype S4 positive donors. Key defense mechanisms against IFI include the activation of neutrophils, the phagocytosis of germinating conidia by dendritic cells, and the fight of the cells of the innate immunity such as monocytes and natural killer cells against germlings and hyphae. Furthermore, immunosuppressive drugs interact with immune effector cells influencing the specific fungal immune defense and antimycotic drugs might interact with immune response. Based on the current knowledge on immunological mechanism inAspergillus fumigatus, the first approaches of an immunotherapy using human T cells are in development. This might be an option for the future of aspergillosis patients having a poor prognosis with conventional treatment.
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49

Cerny, Andreas, and Francis V. Chisari. "Immunological Aspects of HCV Infection." Intervirology 37, no. 2 (1994): 119–25. http://dx.doi.org/10.1159/000150366.

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50

Trifonova, N. S., E. V. Zhukova, L. S. Aleksandrov, A. V. Simonova, A. I. Ishchenko, N. I. Borisova, and A. P. Nikonov. "Immunological aspects of allogeneic pregnancy." Voprosy ginekologii, akušerstva i perinatologii 15, no. 5 (2016): 59–66. http://dx.doi.org/10.20953/1726-1678-2016-5-59-66.

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