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Relevant bibliographies by topics / IMMUNOESCAPE

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Journal articles

Academic literature on the topic 'IMMUNOESCAPE'

Author: Grafiati

Published: 11 March 2023

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Journal articles on the topic "IMMUNOESCAPE"

1

Concha-Benavente, Fernando, Raghvendra M. Srivastava, Soldano Ferrone, and Robert L. Ferris. "EGFR-mediated tumor immunoescape." OncoImmunology 2, no. 12 (2013): e27215. http://dx.doi.org/10.4161/onci.27215.

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2

QUESNEL, BRUNO. "Tumor dormancy and immunoescape." APMIS 116, no. 7-8 (2008): 685–94. http://dx.doi.org/10.1111/j.1600-0463.2008.01163.x.

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3

Mazzolini, Guillermo. "Immunotherapy and immunoescape in colorectal cancer." World Journal of Gastroenterology 13, no. 44 (2007): 5822. http://dx.doi.org/10.3748/wjg.v13.i44.5822.

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4

Van hede, Dorien, Inge Langers, Philippe Delvenne, and Nathalie Jacobs. "Origin and immunoescape of uterine cervical cancer." La Presse Médicale 43, no. 12 (2014): e413-e421. http://dx.doi.org/10.1016/j.lpm.2014.09.005.

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5

Sauleda, Jaume, Francisco Javier Verdú, Sergio Scrimini, Ernest Sala, and Jaume Pons. "Immunoescape the link between emphysema and lung cancer?" Journal of Thoracic Disease 11, S3 (2019): S329—S330. http://dx.doi.org/10.21037/jtd.2018.12.133.

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6

Takasu, Chie, Shoko Yamashita, Yuji Morine, et al. "The role of the immunoescape in colorectal cancer liver metastasis." PLOS ONE 16, no. 11 (2021): e0259940. http://dx.doi.org/10.1371/journal.pone.0259940.

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Abstract:

The expression of programmed death 1 (PD-1) and programmed death-ligand 1 (PD-L1) indicate the efficacy of anti-PD-1/PD-L1 therapy in colorectal cancer (CRC), but are less useful for monitoring the efficacy of therapy of CRC liver metastasis (CRLM). This study investigated the effects of immune molecules on the prognosis of CRLM. We enrolled 71 patients with CRLM who underwent curative resection for CRC. We used immunohistochemistry to analyze the expression of PD-1, PD-L1, indoleamine-pyrrole 2,3-dioxygenase (IDO), and CD163 (a marker of tumor-associated macrophages [TAMs]) in metastatic tumors. The immune molecules PD-1, PD-L1, IDO, and TAMs were expressed in 32.3%, 47.8%, 45.0%, and 47.9% of metastatic CRC samples, respectively. The 5-year overall survival rates associated with immune molecule-positive groups were significantly better than in the negative groups (PD-1: 87.7% vs 53.2%, p = 0.023; PD-L1: 82.4% vs 42.3%, p = 0.007; IDO: 80.7% vs 43.5%, p = 0.007; TAMs: 82.6% vs 48.0%, p = 0.005). Multivariate analysis revealed PD-1 expression (p = 0.032, hazard ratio: 0.19), IDO expression (p = 0.049, hazard ratio: 0.37), and tumor differentiation (p<0.001, hazard ratio: 0.02) as independent prognostic indicators. PD-1 and TAMs in metastases were associated with less aggressive features such as smaller tumors. Furthermore, TAMs positively and significantly correlated with PD-1 expression (p = 0.011), PD-L1 expression (p = 0.024), and tended to correlate with IDO expression (p = 0.078). PD-1, PD-L1, IDO, and TAMs in CRLM were associated with less aggressive features and better prognosis of patients with CRC, indicating adaptive antitumor immunity vs immune tolerance. These molecules may therefore serve as prognostic markers for CRLM.

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7

deCampos-Lima, Pedro-Otavio, Jelena Levitskaya, Teresa Frisan, and Maria G. Masucci. "Strategies of immunoescape in Epstein-Barr virus persistence and pathogenesis." Seminars in Virology 7, no. 1 (1996): 75–82. http://dx.doi.org/10.1006/smvy.1996.0009.

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8

Yaguchi, Tomonori, Hidetoshi Sumimoto, Chie Kudo-Saito, et al. "The mechanisms of cancer immunoescape and development of overcoming strategies." International Journal of Hematology 93, no. 3 (2011): 294–300. http://dx.doi.org/10.1007/s12185-011-0799-6.

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9

Ghiringhelli, François, Mélanie Bruchard, Fanny Chalmin, and Cédric Rébé. "Production of Adenosine by Ectonucleotidases: A Key Factor in Tumor Immunoescape." Journal of Biomedicine and Biotechnology 2012 (2012): 1–9. http://dx.doi.org/10.1155/2012/473712.

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Abstract:

It is now well known that tumor immunosurveillance contributes to the control of cancer growth. Many mechanisms can be used by cancer cells to avoid the antitumor immune response. One such mechanism relies on the capacity of cancer cells or more generally of the tumor microenvironment to generate adenosine, a major molecule involved in antitumor T cell response suppression. Adenosine is generated by the dephosphorylation of extracellular ATP released by dying tumor cells. The conversion of ATP into adenosine is mediated by ectonucleotidase molecules, namely, CD73 and CD39. These molecules are frequently expressed in the tumor bed by a wide range of cells including tumor cells, regulatory T cells, Th17 cells, myeloid cells, and stromal cells. Recent evidence suggests that targeting adenosine by inhibiting ectonucleotidases may restore the resident antitumor immune response or enhance the efficacy of antitumor therapies. This paper will underline the impact of adenosine and ectonucleotidases on the antitumor response.

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10

Wu, Lei, Yanquan Xu, Huakan Zhao та ін. "FcγRIIB potentiates differentiation of myeloid-derived suppressor cells to mediate tumor immunoescape". Theranostics 12, № 2 (2022): 842–58. http://dx.doi.org/10.7150/thno.66575.

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