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Journal articles on the topic 'Immunity'

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1

DH, Jha. "Immunity and Ayurveda." Journal of Natural & Ayurvedic Medicine 4, no. 2 (April 2, 2020): 1. http://dx.doi.org/10.23880/jonam-16000248.

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2

&NA;. "Immunity lost, immunity regained." Inpharma Weekly &NA;, no. 760 (October 1990): 17. http://dx.doi.org/10.2165/00128413-199007600-00049.

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3

Khudhair, Abdulkareem Salman. "Herd Immunity or Community Immunity." Scholars Journal of Medical Case Reports 08, no. 04 (April 30, 2020): 508–9. http://dx.doi.org/10.36347/sjmcr.2020.v08i04.026.

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4

Cotter, S. C., and R. M. Kilner. "Personal immunity versus social immunity." Behavioral Ecology 21, no. 4 (June 4, 2010): 663–68. http://dx.doi.org/10.1093/beheco/arq070.

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5

Takahashi, Hidemi. "Innate Immunity and Acqired Immunity." Journal of Nippon Medical School 69, no. 5 (2002): 410–14. http://dx.doi.org/10.1272/jnms.69.410.

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6

Lee, Theodore M. "Immunity." Emerging Infectious Diseases 22, no. 4 (April 2016): 766. http://dx.doi.org/10.3201/eid2204.151858.

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7

Wilkinson, Lise. "Immunity." Lancet 365, no. 9469 (April 2005): 1459. http://dx.doi.org/10.1016/s0140-6736(05)66405-7.

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8

Reddy, Karthik, Moritz Schularick, and Vasiliki Skreta. "IMMUNITY." International Economic Review 61, no. 2 (March 31, 2020): 531–64. http://dx.doi.org/10.1111/iere.12433.

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9

Muske, Carol. "Immunity." Missouri Review 9, no. 1 (1985): 20–21. http://dx.doi.org/10.1353/mis.1985.0140.

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10

Lewin, Benjamin. "Immunity." Immunity 1, no. 1 (April 1994): 1. http://dx.doi.org/10.1016/1074-7613(94)90002-7.

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11

Fantry, George T., and Stephen P. James. "Cell-mediated immunity and mucosal immunity." Current Opinion in Gastroenterology 10, no. 4 (July 1994): 365–73. http://dx.doi.org/10.1097/00001574-199407000-00003.

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12

Terry, R. J. "Human immunity to schistosomes: Concomitant immunity?" Parasitology Today 10, no. 10 (January 1994): 377–78. http://dx.doi.org/10.1016/0169-4758(94)90224-0.

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13

Michael, J. Dochniak. "Maladaptive Immunity and Metastasizing Cancer." Cancer Medicine Journal 3, no. 1 (June 30, 2020): 31–34. http://dx.doi.org/10.46619/cmj.2020.3-1017.

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The ability of innate immunity to inhibit metastatic cells is limited, based on Stage IV cancer survival rates. The dysregulation of the immune system through acquired immunity may result in pathological conditions that alter metastatic cells. Immunoglobulin-E (IgE) antibodies developed by the humoral immune system are a significant contributor to maladaptive immunity. Hypersensitivities are maladaptive immune reactions against harmless allergens. Forced allergen-specific immune responses may provide immediate-type allergies that affect the incidence and prevalence of endogenous proteins essential for metastasizing cells. Furthermore, allergies may shift the body’s resource allocation away from metastasizing cells to IgE-primed effector-cell proliferation. Therefore, research efforts need to explore if hyper-allergenic skin creams can be used to starve-out metastatic cells, wherever they are in the body, to determine if maladaptive immunotherapy is a viable treatment for Stage IV cancer.
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14

Kumar, Rajiv. "Routes of Infections, Inflammation, Immunity, Immunity Response and Inflammatory Injury: Elucidation of a Biological Fight." Immunology and Inflammation Diseases Therapy 5, no. 1 (January 13, 2022): 01–04. http://dx.doi.org/10.31579/2637-8876/028.

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Infections, inflammation, immunity, and inflammatory injury are different segments of biological events and link up altogether. Route of infection has no similarity with the cellular signaling pathway of inflammation, even though when inflammation is induced by infection. The organism responds toward infection that is initiated by the pathogen via inflammation, which is a natural way of defense initiated by innate immunity as a safeguard
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15

Zainal Abidin, Anis Siham, Masri Muhamed, Mazidah Nordin, Nor Azizah Abu, Noor Shafina Mohd Noor, Faisal Mohd Fadzli, and Mohammed Fauzi Abdul Rani. "Herd Immunity or Heard Not of Immunity?" Journal of Clinical and Health Sciences 1, no. 2 (December 31, 2016): 4. http://dx.doi.org/10.24191/jchs.v2i1.5857.

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16

McLennan, Ryan. "Does Immunity Granted Really Equal Immunity Received?" Journal of Criminal Law and Criminology (1973-) 91, no. 2 (2001): 469. http://dx.doi.org/10.2307/1144271.

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17

Albright, Joseph F., Julia W. Albright, and Kevin High. "Aging, Immunity, and Infection:Aging, Immunity, and Infection." Clinical Infectious Diseases 38, no. 4 (February 15, 2004): 598–99. http://dx.doi.org/10.1086/381031.

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18

Černý, Jan, and Ilja Stříž. "Adaptive innate immunity or innate adaptive immunity?" Clinical Science 133, no. 14 (July 2019): 1549–65. http://dx.doi.org/10.1042/cs20180548.

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Abstract The innate immunity is frequently accepted as a first line of relatively primitive defense interfering with the pathogen invasion until the mechanisms of ‘privileged’ adaptive immunity with the production of antibodies and activation of cytotoxic lymphocytes ‘steal the show’. Recent advancements on the molecular and cellular levels have shaken the traditional view of adaptive and innate immunity. The innate immune memory or ‘trained immunity’ based on metabolic changes and epigenetic reprogramming is a complementary process insuring adaptation of host defense to previous infections. Innate immune cells are able to recognize large number of pathogen- or danger- associated molecular patterns (PAMPs and DAMPs) to behave in a highly specific manner and regulate adaptive immune responses. Innate lymphoid cells (ILC1, ILC2, ILC3) and NK cells express transcription factors and cytokines related to subsets of T helper cells (Th1, Th2, Th17). On the other hand, T and B lymphocytes exhibit functional properties traditionally attributed to innate immunity such as phagocytosis or production of tissue remodeling growth factors. They are also able to benefit from the information provided by pattern recognition receptors (PRRs), e.g. γδT lymphocytes use T-cell receptor (TCR) in a manner close to PRR recognition. Innate B cells represent another example of limited combinational diversity usage participating in various innate responses. In the view of current knowledge, the traditional black and white classification of immune mechanisms as either innate or an adaptive needs to be adjusted and many shades of gray need to be included.
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19

Yang, Xiaodong. "STATE IMMUNITY OUTSIDE THE STATE IMMUNITY ACT." Cambridge Law Journal 60, no. 1 (March 2001): 1–58. http://dx.doi.org/10.1017/s000819730165061x.

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IS the State Immunity Act 1978 the sole basis for deciding on State immunity? It is and it is not. This seemingly self-contradictory reply is due to the fact that, on the one hand, any proceedings directly or indirectly against a foreign State must be brought under the 1978 Act while, on the other, certain provisions of that Act might paradoxically render the Act itself inapplicable and therefore entail recourse to rules outside the Act for settling the issue of State immunity. This is amply illustrated by the decision of the House of Lords in Holland v. Lampen-Wolfe [2000] 1 W.L.R. 1573, which involved a claim for defamation brought by a US university professor teaching international relations at a US military base in England as part of an education programme provided by her university under a commercial agreement with the US Government. The claim was brought against the education services officer at the base, who had written a memorandum listing serious complaints about the plaintiff’s performance and questioning her professional competence. The US Government claimed immunity on the defendant’s behalf.
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20

Benbaji, Yitzhak, and Susanne Burri. "Civilian Immunity Without the Doctrine of Double Effect." Utilitas 32, no. 1 (September 9, 2019): 50–69. http://dx.doi.org/10.1017/s0953820819000335.

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AbstractCivilian Immunity (‘Immunity’) is the legal and moral protection that civilians enjoy against the effects of hostilities under the laws of armed conflict and according to the ethics of killing in war. Immunity specifies different permissibility conditions for directly targeting civilians on the one hand, and for harming civilians incidentally on the other hand. Immunity is standardly defended by appeal to the Doctrine of Double Effect (DDE). We show that Immunity's prohibitive stance towards targeting civilians directly, and its more permissive stance towards harming them incidentally, can be defended without appealing to the DDE if agents suffer from overconfidence. Overconfidence is a cognitive bias that affects agents who are required to make decisions in the presence of significant uncertainty.
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21

Kurzon, Dennis. "From immunity to immunity. From immunity to silence: The case of Gilad Sharon." Semiotica 2017, no. 216 (May 24, 2017): 265–79. http://dx.doi.org/10.1515/sem-2015-0091.

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AbstractIn 2003, Gilad Sharon, the younger son of the late Ariel Sharon, then Israeli Prime Minister, was being investigated by the police for his part in what is known as the Cyril Kern affair, which involved not only illegal donations to Ariel Sharon’s campaign fund to become leader of the Likud Party in 1999 but also a long money trail around the world. Large sums of money were being paid to Gilad Sharon as fees for vague consulting services. Since Ariel Sharon was the prime minister and a member of the Knesset, he had immunity from search unless it was withdrawn by the Israeli parliament. Gilad Sharon lived on the same ranch as his father, taking full advantage of his father’s immunity. The police refrained from issuing a search warrant of the Prime Minister’s private home, but demanded documents from Sharon relating to the money trail. When questioned by the police, he claimed the right of silence, which – so it was asserted by Sharon and his lawyers – covered documents he may have in his possession in his house. Moreover, Sharon claimed that the documents may not incriminate him, but someone else (his father). A warrant was issued by the magistrate’s court requiring Sharon to submit the documents. This was overturned by the district court, but finally in December 2003 the Israeli Supreme Court ordered Sharon to submit the documents to the magistrate’s court in order for the proceedings to continue. In the paper, the court case will be examined through the semantics of the phraseimmunity fromfollowing the concept of the semiotic square (e. g., Greimas 1988). It will be shown that the narrative of the case may be built up from one immunity leading to another, and then to a third. The neutral termnot liable to + not immune frommay lead to silence.
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22

Musa, Sanjin. "On immunity: an inoculation, Eula Biss." Central European Journal of Paediatrics 13, no. 1 (March 15, 2017): 81–83. http://dx.doi.org/10.5457/p2005-114.175.

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23

RMV, Rao. "Immunity in Medically Important Parasitic Infections." Virology & Immunology Journal 5, no. 1 (January 12, 2021): 1–8. http://dx.doi.org/10.23880/vij-16000267.

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Immunity is the rule. It is often incomplete and takes many years to develop and fade away quickly. Human life is a battlefield in which we are like soldiers attacked from all sides by bacteria, viruses, fungi and parasites. Our body is bestowed with a defense mechanism in the form of an immune system It has long been recognized that infections with parasites, such as intestinal worms, are often accompanied by blood eosinophilia, and this is due to an immunological process. Conditions in which blood eosinophilia is common include intestinal infections with Ancylostoma duodenale, Ascaris lumbricoides, Trichuristrichiura, various forms of Wuchereria bancroft, Brugiamalayi, loaloa, Dracunculus medinensis, mite infection of the lungs (including at least some cases of tropical eosinophilia);and hydrated disease is due to Echinococcus granulosus. Eosinophilia, in large numbers invades tissues in which antigen-antibody reaction has taken place. They appear to be attracted by some product of the antigen-antibody reaction and it has been shown that if tissues from sensitized guinea -pigs mixed with antigen in vitro, or tissues from guinea-pigs which have died from anaphylaxis, are transferred to the peritoneal cavity of normal guinea-pigs, the recipient develops very marked eosinophilia within 24 hours. The active agent has not been infected, but it is probably not histamine. The eosinophils of rodents are very actively phagocytic, and ingest cellular debris, mast cell granules, etc, but it is not certain whether this is true of eosinophils from other species, nor it is known what functions the eosinophils serve in these reactions. A multitude of defensive mechanisms are involved in parasitic infection. A humoral response develops when parasites invade blood stream (Malaria, Trypanosoma), whereas cell-mediated immunity is elicited by parasites that grow within the tissues (Eg: Cutaneous leishmaniasis). In protozoal infections, IgG, and IgM are produced. In Addition, IgA also produced in intestinal infection. With helminthic infections, IgG, IgM and IgE antibodies are produced.
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24

Alderton, Gemma. "Antiviral immunity." Science 371, no. 6528 (January 28, 2021): 477.10–479. http://dx.doi.org/10.1126/science.371.6528.477-j.

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25

Ashby, Ben, and Alex Best. "Herd immunity." Current Biology 31, no. 4 (February 2021): R174—R177. http://dx.doi.org/10.1016/j.cub.2021.01.006.

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26

Alderton, Gemma. "Aging immunity." Science 369, no. 6501 (July 16, 2020): 264.11–266. http://dx.doi.org/10.1126/science.369.6501.264-k.

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27

Maria Navarro, Jose, and Mercedes Perez-Ruiz. "Antiviral Immunity." Current Immunology Reviews 7, no. 1 (February 1, 2011): 19–24. http://dx.doi.org/10.2174/157339511794474244.

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28

Strange, Carolyn. "Rethinking Immunity." BioScience 45, no. 10 (November 1995): 663–68. http://dx.doi.org/10.2307/1312669.

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29

Soumya, M., and R. Deepa. "Herd immunity." International Journal of Nursing Education and Research 9, no. 1 (2021): 125–27. http://dx.doi.org/10.5958/2454-2660.2021.00032.6.

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30

Ash, Caroline. "Relative immunity." Science 372, no. 6541 (April 29, 2021): 477.5–478. http://dx.doi.org/10.1126/science.372.6541.477-e.

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31

Joon, Sneha. "Sansad Immunity." International Journal for Research in Applied Science and Engineering Technology 10, no. 10 (October 31, 2022): 406–7. http://dx.doi.org/10.22214/ijraset.2022.47013.

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Abstract: Immunity means a privilege enjoyed by a certain class of individuals which others might not have a access to. In reference to the parliament1 privilege refers to the powers and immunities given to each house of the parliament and its members collectively to enjoy the rights. These privileges are an exception to common law and allow the members to enact their duties without the fear of being threatened or punished, and even without any obstacle. The privileges of the parliament are equivalent to the immunity of the crown2 . Like the crown can administer without the help or interference of the parliament or the judges. Even both the houses of union supervise the privileges without the involvement of the judges. This paper talks about these parliamentary privileges in detail and to what extent the members can exercise them.
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32

Seppa, Nathan. "Revised Immunity." Science News 161, no. 22 (June 1, 2002): 339. http://dx.doi.org/10.2307/4013319.

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33

Hill, Colin, Lorraine Draper, R. Ross, and Paul Cotter. "Lantibiotic Immunity." Current Protein & Peptide Science 9, no. 1 (February 1, 2008): 39–49. http://dx.doi.org/10.2174/138920308783565750.

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34

Karp, Richard D. "Cockroach Immunity." Science 270, no. 5233 (October 6, 1995): 17. http://dx.doi.org/10.1126/science.270.5233.17.a.

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35

Cohen, Jon. "Waning immunity." Science 364, no. 6437 (April 19, 2019): 224–27. http://dx.doi.org/10.1126/science.364.6437.224.

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36

Mayer, Lloyd. "Mucosal Immunity." Pediatrics 111, Supplement_3 (June 1, 2003): 1595–600. http://dx.doi.org/10.1542/peds.111.s3.1595.

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Food allergy is the manifestation of an abnormal immune response to antigen delivered by the oral route. Normal mucosal immune responses are generally associated with suppression of immunity. A normal mucosal immune response relies heavily on a number of factors: strong physical barriers, luminal digestion of potential antigens, selective antigen sampling sites, and unique T-cell subpopulations that effect suppression. In the newborn, several of these pathways are not matured, allowing for sensitization rather than suppression. With age, the mucosa associated lymphoid tissue matures, and in most individuals this allows for generation of the normal suppressed tone of the mucosa associated lymphoid tissue. As a consequence, food allergies are largely outgrown. This article deals with the normal facets of mucosal immune responses and postulates how the different processes may be defective in food-allergic patients.
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37

Davis, Daniel M. "Engineering immunity." New Scientist 250, no. 3341 (July 2021): 40–43. http://dx.doi.org/10.1016/s0262-4079(21)01155-6.

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38

Crotty, Shane. "Hybrid immunity." Science 372, no. 6549 (June 24, 2021): 1392–93. http://dx.doi.org/10.1126/science.abj2258.

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39

Kaur, Bani Preet, and Elizabeth Secord. "Innate Immunity." Immunology and Allergy Clinics of North America 41, no. 4 (November 2021): 535–41. http://dx.doi.org/10.1016/j.iac.2021.07.003.

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40

van Oss, Carel J. "Mucosal Immunity." Immunological Investigations 14, no. 3 (January 1985): 279. http://dx.doi.org/10.3109/08820138509076154.

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41

NAGURA, HIROSHI. "Mucosal immunity." Nihon Naika Gakkai Zasshi 84, no. 4 (1995): 632–39. http://dx.doi.org/10.2169/naika.84.632.

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42

Hou, Shuguo, Yifei Yang, Daoji Wu, and Chao Zhang. "Plant immunity." Plant Signaling & Behavior 6, no. 6 (June 2011): 794–99. http://dx.doi.org/10.4161/psb.6.6.15143.

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43

Miller, Julie Ann. "Mouth Immunity." Science News 128, no. 14 (October 5, 1985): 221. http://dx.doi.org/10.2307/3970272.

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44

Ely, John Hart. "Interclausal Immunity." Virginia Law Review 87, no. 6 (October 2001): 1185. http://dx.doi.org/10.2307/1073951.

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45

Lamberty, Mireille, Daniel Zachary, René Lanot, Christian Bordereau, Alain Robert, Jules A. Hoffmann, and Philippe Bulet. "Insect Immunity." Journal of Biological Chemistry 276, no. 6 (October 26, 2000): 4085–92. http://dx.doi.org/10.1074/jbc.m002998200.

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46

Dwinell, Michael B., and Martin F. Kagnoff. "Mucosal immunity." Current Opinion in Gastroenterology 15, no. 1 (January 1999): 33. http://dx.doi.org/10.1097/00001574-199901000-00007.

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47

Mayer, Lloyd. "Mucosal immunity." Immunological Reviews 206, no. 1 (August 2005): 5. http://dx.doi.org/10.1111/j.0105-2896.2005.00296.x.

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48

Medzhitov, Ruslan, and Charles Janeway. "Innate Immunity." New England Journal of Medicine 343, no. 5 (August 3, 2000): 338–44. http://dx.doi.org/10.1056/nejm200008033430506.

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49

Rennie, John. "Balanced Immunity." Scientific American 268, no. 5 (May 1993): 22–24. http://dx.doi.org/10.1038/scientificamerican0593-22.

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50

Mathison, John. "Innate Immunity." Journal of Pediatric Gastroenterology and Nutrition 40, Supplement 1 (April 2005): S13—S15. http://dx.doi.org/10.1097/00005176-200504001-00008.

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