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Books on the topic 'Immune pathology'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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1

Walker, Pam. The immune system. San Diego, Calif: Lucent Books, 2003.

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2

Walker, Pam. The immune system. San Diego, Calif: Lucent Books, 2003.

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3

Brown, Earl. Basic concepts in pathology: A student's survival guide. New York: McGraw-Hill, Health Professions Division, 1998.

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4

Siegfried, Ansorge, and Langner Jürgen, eds. Cellular peptidases in immune functions and diseases. New York: Plenum Press, 1997.

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5

Olsen, Olger M. Zufall und/oder Determination: Das Phänomen acquired immune deficiency syndrome. Osnabrück: Literarisches Büro, 1985.

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6

Pathology of AIDS: Textbook and atlas of diseases associated with acquired immune deficiency syndrome. Philadelphia: Lippincott, 1989.

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7

A, Roth James, ed. Understanding immunology. St. Louis: Mosby, 1998.

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8

General pathobiology. Norwalk, Conn: Appleton & Lange, 1994.

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9

Birch, Kate. The solution: Homeoprophylaxis, the vaccine alternative : a parent's guide to educating your child's immune system. Bloomington, IN: Balboa Press, 2012.

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10

Evans, Sarah Frances. A study of the immune system and some aspects of the pathology of the osteopetrotic (op/op)rat. Birmingham: University of Birmingham, 1987.

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11

Hale, Mary. The immune support cookbook: Easy, delicious recipes to support your health if you're HIV positive or suffer from CFIDS, cancer or other degenerative diseases. Secaucus, NJ: Carol Pub. Group, 1995.

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12

P, Wormser Gary, Stahl Rosalyn E, and Bottone Edward J, eds. AIDS, acquired immune deficiency syndrome, and other manifestations of HIV infection: Epidemiology, etiology, immunology, clinical manifestations, pathology, control, treatment and prevention. Park Ridge, N.J., U.S.A: Noyes Publications, 1987.

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13

Chistyakova, Guzel, Lyudmila Ustyantseva, Irina Remizova, Vladislav Ryumin, and Svetlana Bychkova. CHILDREN WITH EXTREMELY LOW BODY WEIGHT: CLINICAL CHARACTERISTICS, FUNCTIONAL STATE OF THE IMMUNE SYSTEM, PATHOGENETIC MECHANISMS OF THE FORMATION OF NEONATAL PATHOLOGY. au: AUS PUBLISHERS, 2022. http://dx.doi.org/10.26526/monography_62061e70cc4ed1.46611016.

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The purpose of the monograph, which contains a modern view of the problem of adaptation of children with extremely low body weight, is to provide a wide range of doctors with basic information about the clinical picture, functional activity of innate and adaptive immunity, prognostic criteria of postnatal pathology, based on their own research. The specific features of the immunological reactivity of premature infants of various gestational ages who have developed bronchopulmonary dysplasia (BPD) and retinopathy of newborns (RN) from the moment of birth and after reaching postconceptional age (37-40 weeks) are described separately. The mechanisms of their implementation with the participation of factors of innate and adaptive immunity are considered in detail. Methods for early prediction of BPD and RN with the determination of an integral indicator and an algorithm for the management of premature infants with a high risk of postnatal complications at the stage of early rehabilitation are proposed. The information provided makes it possible to personify the treatment, preventive and rehabilitation measures in premature babies. The monograph is intended for obstetricians-gynecologists, neonatologists, pediatricians, allergists-immunologists, doctors of other specialties, residents, students of the system of continuing medical education. This work was done with financial support from the Ministry of Education and Science, grant of the President of the Russian Federation No. MK-1140.2020.7.
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14

The use of SCID mice in the investigation of human autoimmune disease. Austin: R.G. Landes, 1994.

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15

L, Asachenkov A., ed. Disease dynamics. Boston: Birkhäuser, 1994.

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16

International Subcellular Methodology Forum (9th 1984 Guildford, England). Investigation and exploitation of antibody combining sites. New York: Plenum Press, 1985.

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17

Tang, Patrick Ming-Kuen, Hui Y. Lan, Haiyong Chen, David Nikolic-Paterson, and Ying Tang, eds. Immune Landscape of Kidney Pathology. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-530-2.

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18

Ansorge, Siegfried. Cellular Peptidases in Immune Functions and Diseases. Springer, 2013.

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19

Harrison, Mark. Immune response. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198765875.003.0055.

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This chapter describes the pathology of immune response as it applies to Emergency Medicine, and in particular the Primary FRCEM examination. The chapter outlines the key details of normal and abnormal responses. This chapter is laid out exactly following the RCEM syllabus, to allow easy reference and consolidation of learning.
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20

Brown, Earl. Basic Concepts in Pathology: A Student's Survival Guide. McGraw-Hill Professional Publishing, 1997.

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21

Brown, Earl. Basic Concepts in Pathology: A Student's Survival Guide. McGraw-Hill Professional Publishing, 1997.

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22

Lakatos, Andras, and Gabor Petzold, eds. Primary Glial and Immune Cell Pathology in Neurodegenerative Diseases. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-840-5.

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23

Langner, Jürgen, and Siegfried Ansorge. Cellular Peptidases in Immune Functions and Diseases. Springer London, Limited, 2013.

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24

(Editor), J. S. Abramson, and J. G. Wheeler (Editor), eds. The Neutrophil: The Natural Immune System. Oxford University Press, USA, 1993.

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25

(Editor), J. S. Abramson, and J. G. Wheeler (Editor), eds. The Neutrophil: The Natural Immune System. Oxford University Press, USA, 1993.

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26

Langner, Jürgen, and Siegfried Ansorge. Cellular Peptidases in Immune Functions and Diseases 2. Springer London, Limited, 2006.

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27

Ofek, Itzhak, and Yona Keisari. Biology and Pathology of Innate Immunity Mechanisms. Springer London, Limited, 2006.

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28

Ofek, Itzhak, and Yona Keisari. Biology and Pathology of Innate Immunity Mechanisms. Springer, 2013.

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29

(Editor), Jürgen Langner, and Siegfried Ansorge (Editor), eds. Cellular Peptidases in Immune Functions and Diseases 2 (Advances in Experimental Medicine and Biology). Springer, 2000.

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30

Coe-Clough, Nancy, and James A. Roth. Mosby's Biomedical Science Series: Understanding Immunology (Mosby's Biomedical Science Series). Mosby, 1997.

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31

Feldmann, M., and R. Maini. T-Cell Activation in Health and Disease Disorders of Immune Regulation Infection and Autoimmunity: Papers from an International Meeting in Oxford, U. Academic Press, 1989.

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32

(Editor), Yona Keisari, and Itzhak Ofek (Editor), eds. The Biology and Pathology of Innate Immunity Mechanisms (ADVANCES IN EXPERIMENTAL MEDICINE AND BIOLOGY Volume 479). Springer, 2000.

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33

Gruundmann, E. Reaction Patterns of the Lymph Node Part 2: Reactions Associated With Neoplasia and Immune Deficient States (Current Topics in Pathology). Springer, 1991.

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34

T-cell activation in health and disease: Disorders of immune regulation : infection and autoimmunity : papers from an international meeting in Oxford, UK, in September 1988. London: Academic Press, 1989.

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35

Marc, Feldmann, Maini R. N, Woody James N, and United States. Naval Medical Research and Development Command., eds. T-cell activation in health and disease: Disorders of immune regulation infection and autoimmunity : papers from an international meeting in Oxford, UK, in September 1988. London ; San Diego: Academic Press, 1989.

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36

Dyrberg, Thomas. Role of Viruses and the Immune System in Diabetes Mellitus: Experimental Models. Springer London, Limited, 2013.

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37

Dyrberg, Thomas. Role of Viruses and the Immune System in Diabetes Mellitus: Experimental Models. Springer London, Limited, 2011.

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38

Neuroimmunodegeneration. Georgetown, Tex: R.G. Landes, 1998.

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39

Henderson, Lorna K., Brian J. Nankivell, and Jeremy R. Chapman. Chronic allograft dysfunction. Edited by Jeremy R. Chapman. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0286.

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Despite improvements in short-term renal allograft survival, long-term survival has not appreciably changed. Excepting death with a functioning graft, most late graft loss results from chronic allograft dysfunction. Immune and non-immune-mediated injuries contribute to graft dysfunction over time, ultimately leading to a non-specific and irreversible histological end-point of fibrosis, tubular atrophy, and glomerulosclerosis. Screening and early identification of pathology is crucial to allow timely intervention in order to prevent permanent nephron damage and graft loss. This chapter outlines assessment of renal dysfunction following transplantation, defines the causes of chronic allograft failure, and their pathophysiology, and evaluates current therapeutic strategies used to improve or stabilize chronic allograft dysfunction.
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40

Bielekova, Bibiana, Gary Birnbaum, and Robert P. Lisak, eds. Neuroimmunology. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190050801.001.0001.

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This book provides clinical and supporting scientific background on a diverse group of neurological disorders in an expanding field of neurology, that of neuroimmunology. It includes chapters on multiple sclerosis and related disorders in adults and children, neuromyelitis optica spectrum disorder, Guillain-Barré syndrome, chronic inflammatory deymyelinating polyradiculoneuropathy and variants, immune-mediated disorders of the neuromuscular junction, inflammatory myopathies, paraneoplastic disorders and autoimmune encephalitities, and neurologic manifestations of systemic immune-mediated diseases. In addition there is an introductory chapter dealing with basic of immunology and another on mechanisms of action of therapies used in neuroimmunologic disorders. The clinical chapters cover epidemiology, pathology, pathogenesis, and pathophysiology of the different diseases along with clinical presentation, diagnostic testing, differential diagnosis, and treatment.
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41

Parkes, Joanna E., Simon Rothwell, and Janine A. Lamb. Aetiology and pathogenesis. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0003.

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The aetiology and pathogenesis of idiopathic inflammatory myopathies (IIM) is poorly understood; IIM are thought to result from exposure to environmental factors in genetically susceptible individuals. Both innate and adaptive immune responses are involved in IIM, and there is increasing evidence that non-inflammatory mechanisms play an important role in disease pathology. Several environmental risk factors, including infectious agents, ultraviolet radiation, cigarette smoking, and exposure to statins, have been implicated. Genetic studies have identified the major histocompatibility complex as the most strongly associated region, while recent large scale genome-wide studies have implicated genes that commonly regulate the adaptive immune response, which overlap with other seropositive autoimmune diseases. Integrating data across these various fields should facilitate refined models of disease pathogenesis.
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42

Sherman, Mark E., Melissa A. Troester, Katherine A. Hoadley, and William F. Anderson. Morphological and Molecular Classification of Human Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0003.

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Accurate and reproducible classification of tumors is essential for clinical management, cancer surveillance, and studies of pathogenesis and etiology. Tumor classification has historically been based on the primary anatomic site or organ in which the tumor occurs and on its morphologic and histologic phenotype. While pathologic criteria are useful in predicting the average behavior of a group of tumors, histopathology alone cannot accurately predict the prognosis and treatment response of individual cancers. Traditional measures such as tumor stage and grade do not take into account molecular events that influence tumor aggressiveness or changes in the tumor composition during treatment. This chapter provides a primer on approaches that use pathology and molecular biology to classify and subclassify cancers. It describes the features of carcinomas, sarcomas, and malignant neoplasms of the immune system and blood, as well as various high-throughput genomic platforms that characterize the molecular profile of tumors.
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43

Johnson, Frederick A. Murphy, Margo A. Brinton, Louise T. Chow, Francisco Gonzalez-Scarano, Diane E. Griffin, Kathryn V. Holmes, Julie Overbaugh, and Harriet L. Robinson. Viral Pathogenesis and Immunity. 2nd ed. Lippincott Williams & Wilkins, 2001.

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44

Guo, Yong, and Claudia F. Lucchinetti. Taking a Microscopic Look at Multiple Sclerosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0005.

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The pathology of multiple sclerosis is complex, extends beyond the white matter plaque, and is influenced by stage of demyelinating activity, clinical course, disease duration, and treatment. Technological advances in immunology, molecular biology, and “omic” biology have provided novel insights into the mechanisms for development of white matter plaques, axonal damage, cortical demyelination, and disease progression. Detailed, systematic, and statistically rigorous pathological studies on clinically well-characterized MS cohorts have helped define the heterogeneous pathological substrates of MS and unravel the complex molecular pathogenic mechanisms, with the ultimate goal of identifying targets for therapeutic interventions. It is increasingly clear that the use of human tissues is imperative to improve current diagnostic, prognostic, and therapeutic modalities. Preclinical animal models have been invaluable for discovery of key immune processes, basic disease mechanisms, and candidate immune targeting strategies, but the conclusions have yet be reconciled with the essential features of the human disease.
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45

Harris, Brent T., Galam A. Khan, and Saed Sadeghi. Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0029.

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Although the basic gross and microscopic pathological changes in amyotrophic lateral sclerosis (ALS) have been known for more than 100 years, emerging technology and research into the cellular and molecular changes found in this disease are challenging our understanding about the pathogenesis and pathophysiology. All cell types of the CNS/PNS as well as circulating immune cells have been implicated in the pathology of ALS. Numerous genes, their proteins, and environmental factors have also been associated. However, we still do not understand the specific gene-environmental interactions that bring about and drive this devastating disease in most cases. This short chapter does not address the causal factors and molecular pathogeneses that have been hypothesized and actively researched in the pathology of ALS-as these are discussed in other sections of this text. Here, it shows and discusses the basic pathological changes at the tissue and cellular levels that help to establish the pathological diagnosis of ALS at autopsy.
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46

J, Hoffmann, Janeway Charles A, and Natori Shunji, eds. Phylogenetic perspectives in immunity: The insect host defense. Austin, Tex: R.G. Landes Co, 1994.

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47

Janeway, Charles A. Jr, Jules A. Hoffmann, and Shunji Natori. Phylogenetic Perspectives in Immunity: The Insect Host Defense (Molecular Biology Intelligence Unit). R. G. Landes, 1994.

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48

1941-, Hoffmann J., ed. Phylogenetic perspectives in immunity: The insect host defense. Austin, Tex: R.G. Landes Co., 1994.

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49

Haghighi, Afshin Borhani, and Bernadette Kalman. Other Proven and Putative Autoimmune Disorders of the CNS. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0094.

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Behcet’s Disease (BD) is a multiorgan disorder characterized by oral and genital ulceration, uveitis, and dermatological symptoms. BD is most prevalent in the Mediterranean countries and East Asia, but also occurs in Europe and North America. The etiology remains unknown. Evidence suggests that BD is an autoimmune disorder with complex traits. Neuro-Behcet’s Syndome (NBS) develops in about 5% to 30% of patients with BD and presents with parenchymal or nonparenchymal pathology. The course of NBS is highly variable. Treatment strategies include modulations of the immune response and tissue degeneration, along with symptomatic medications. Main directions of current research include genomic studies, biomarker discovery, and inventive drug- development strategies.
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50

Gordon, Caroline. Systemic lupus erythematosus—clinical features and aetiopathogenesis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0117.

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Systemic lupus erythematosus (SLE or lupus) is a multisystem, autoimmune disease associated with the formation of autoantibodies that form pathological immune complexes and activate a number of inflammatory pathways. The disease is characterized by remissions and relapses (flares) that can present with a variety of clinical manifestations. The symptoms and signs may range from mild features that can be treated easily to organ and even life threatening manifestations requiring potent immunosuppression. This chapter will review the epidemiology and pathology of lupus, then the clinical features including differential diagnosis and investigation of adult patients with SLE. Finally the classification, diagnosis, monitoring and outcome of lupus patients will be discussed.
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