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Books on the topic 'Immune humoral response'

Author: Grafiati

Published: 25 May 2024

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1

M, Snapper Clifford, ed. Cytokine regulation of humoral immunity: Basic and clinical aspects. Chichester: J. Wiley, 1996.

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2

Hassan, Jubril Olugbenga. Some aspects of humoral and cellular immune response to 'Salmonella typhimurium' in chickens. Birmingham: University of Birmingham, 1989.

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3

Kachani, Malika. Humoral immune responses in theileria annulata infection. Uxbridge: Brunel University, 1990.

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4

Manser, Tim, ed. Specialization and Complementation of Humoral Immune Responses to Infection. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-73900-5.

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5

Tim, Manser, ed. Specialization and complementation of humoral immune responses to infection. Berlin: Springer, 2007.

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6

Allen, Stephen John. An epidemiological study of humoral and cell-mediated immune responses to defined plasmodium falciparum vaccine candidate antigens and their role in protection against infection in a rural area of The Gambia. Birmingham: University of Birmingham, 1991.

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7

1938-, Goidl Edmond A., ed. Aging and the immune response: Cellular and humoral aspects. New York: Dekker, 1987.

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8

Goidl, Edmond A. Aging and the Immune Response: Cellular and Humoral Aspects (Immunology Series). Marcel Dekker, 1986.

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9

Wood, Patricia A. Characterization of the humoral immune response to Renibacterium salmonarium in Chinook salmon Oncorhynchus tshawytscha. 1994.

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10

Cytokine Regulation of Humoral Immunity: Basic and Clinical Aspects. Wiley, 1996.

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11

Snapper, Clifford M. Cytokine Regulation of Humoral Immunity: Basic and Clinical Aspects. Wiley & Sons, Incorporated, John, 2008.

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12

Devlin, Hugh, and Rebecca Craven. Immune system. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759782.003.0011.

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The immune system in relation to dentistry is the topic of this chapter. Non-specific body defences are explored. Then follows specific body defences, humoral and cell mediated responses; antibody types and their mechanisms of action and the clinical application in immunization. Inflammation, both acute and chronic, is explored in relation to infections of dental origin and their complications. Problems with the immune system and hypersensitivity follow. Normal oral flora and dental plaque and the body’s response in periodontal inflammation are explored. The final section deals with the implications of what has gone before for infection control in the dental surgery.

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13

S.M. Dibaji, A. Seidavi, Leila Asadpour, F. Sánchez Chopa, V. Laudadio, Elisabetta Casalino, and V. Tufarelli. Effect of Biomin®IMBO on the humoral immune response of broiler chickens. Verlag Eugen Ulmer, 2015. http://dx.doi.org/10.1399/eps.2015.111.

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14

Whitney, Allene M. Effects of fluoride on humoral immunity in Eisenia foetida (Annelida). 1998.

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15

Dill, Thomas O. Effect of Zinc, Copper and Selenium supplementation on the humoral immune response of weaned beef steers. 1991.

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16

Bhole, Malini. Functions of the immune system. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0293.

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This chapter reviews the functions of the immune system, which has evolved to provide a defence mechanism against microbial challenges, and is divided into two main branches, innate and adaptive. In addition, there are physical and chemical barriers, including skin, mucous membrane, mucous secretions, saliva, and various enzymes, and these contribute to the first line of defence against pathogens. The innate immune system provides the initial quick response for rapid recognition and elimination of pathogens, as opposed to the adaptive immune system, which has evolved to provide a more definitive and finely tuned response. The common central feature of both of these systems is the ability to distinguish between self and non-self. The recognition of non-self or ‘foreign’ pathogens and the subsequent immune response is orchestrated by a whole range of cells and soluble (humoral) factors in both innate and adaptive immune systems.

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17

Durán-Avelar, Ma de Jesús. Humoral Immune Response to Salmonella Antigens and Polymorphisms in Receptors for the Fc of IgG in Patients with Ankylosing Spondylitis. INTECH Open Access Publisher, 2012.

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18

Titishov, Nina *. The effects of (-) and (+)-7-hydroxy-delta-6-tetrahydrocannabinol-1, 1-dimethylheptyl on the primary humoral immune response of mice. 1988.

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19

Manser, Tim. Specialization and Complementation of Humoral Immune Responses to Infection. Springer, 2008.

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20

Various and Tim Manser. Specialization and Complementation of Humoral Immune Responses to Infection. Springer, 2010.

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21

Messacar, Kevin, and Mark J. Abzug. Enterovirus and Parechovirus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0003.

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Enteroviruses (EVs) comprise a genus in the Picornaviridae family. They are single-stranded RNA viruses and are common causes of human infection. Polioviruses, the prototypic EVs, were historically responsible for widespread outbreaks of paralytic poliomyelitis; now they are on the verge of global elimination through vaccination. More than 100 serotypes of nonpoliovirus EVs are described and are associated with a wide variety of diseases, ranging from respiratory infections, nonspecific febrile illnesses, herpangina, and hand-foot-and-mouth disease to meningitis, encephalitis, paralytic disease, myocarditis, chronic or disseminated infection in immunocompromised hosts (particularly those with defects in the humoral immune response), and severe disease in neonates. This chapter reviews disease manifestations during pregnancy and in neonates, with an emphasis on clinical presentation, diagnosis, and management. The newly emerging parechoviruses, important causes of central nervous system (CNS) disease, are also reviewed.

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22

Bhole, Malini. Hypersensitivity diseases. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0300.

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Hypersensitivity reactions are aberrant immune responses that are provoked by innocuous extrinsic or self-antigens, are mediated by B-cells or T-cells, and may result in tissue or organ damage. Coombs and Gell classified hypersensitivity reactions into four types, based on the different immune responses: type I, or immediate hypersensitivity; type II, or antibody-mediated (humoral) cytotoxicity; type III, or immune-complex disease; and type IV, or delayed hypersensitivity. This chapter reviews the clinical features, diagnosis, and management of hypersensitivity reactions.

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23

Roxburgh, Campbell S. D., and Donald C. McMillan. Cancer, immunity, and inflammation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199656103.003.0012_update_001.

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The chapter focuses on the role of immunity and inflammation in established cancer. From the evidence reviewed it is clear that immune and inflammatory responses, innate, humoral and adaptive, local and systemic, are intimately linked to the tumour and themselves and impact on cancer survival. It is also possible to identify key mediators that may be targeted in the cancer patient. However, further work is required to elucidate the mechanisms by which these immune and inflammatory responses are activated, maintained, and interact. Therapeutic intervention using non-selective anti-inflammatory agents is widely advocated and likely to become part of routine clinical practice in the near future. Selective therapeutic intervention directed at the immune and inflammatory responses in cancer is in its infancy. Therefore, it would appear that, at least in non-hereditary disease, immune and inflammatory responses are of key, if not of prime, importance in tumour progression and dissemination.

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24

van Assen, Sander, and Marc Bijl. Vaccination in immunocompromised adults. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0094.

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This chapter addresses all important questions regarding vaccination of patients with autoimmune inflammatory rheumatic diseases (AIIRD). First, the incidence of vaccine-preventable infections in these patients is discussed. Pulmonary infections, including influenza and pneumococcal infection, occur more often in AIIRD patients; herpes zoster and human papillomavirus are also more frequent. The efficacy of vaccination for all European registered vaccines is discussed. Treatment with disease-modifying anti-rheumatic drugs (DMARDs) and biologicals (in particular TNFα‎-blocking agents) do not hamper, or only slightly hamper, the immune responses to most vaccines. Rituximab is an exception, severely reducing humoral responses following influenza and pneumococcal vaccination, at least during the first 6 months after administration. Safety of vaccination is an important issue in patients with autoimmune diseases, since increased disease activity of the underlying disease as a result of vaccination is theoretically possible. The available evidence is summarized, suggesting that vaccination is safe in AIIRD patients. Live vaccines, however, are contraindicated in immunosuppressed patients with AIIRD. Finally, the European League Against Rheumatism (EULAR) recommendations are highlighted, summarizing the 'do's' and 'don'ts' of vaccination in adults with AIIRD.

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25

Bunch, Chris. Splenomegaly and other disorders of the spleen. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0035.

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The spleen is a predominantly lymphoid organ, normally about the size of a clenched fist located beneath the diaphragm in the left upper abdomen. It has a dual role as a filter for the circulation, and a primary lymphoid organ in its own right. About three-quarters of its volume is a matrix of capillaries and sinuses (the red pulp), through which blood is able to percolate slowly and come into contact with fixed macrophages, which are able to remove senescent or damaged red cells, or other particulate matter such as bacteria. The lymphoid tissue is organized into scattered follicles (the white pulp), which have a particularly important role in initiating primary humoral immune responses and antibody (IgM) synthesis. The spleen commonly enlarges when either its filtration function is increased—as in haemolysis—or it is stimulated by infection or inflammation. It may also be involved in myeloproliferative and lymphoproliferative neoplasias. This chapter covers hypersplenism, splenectomy, hyposplenism, overwhelming post-splenectomy infection (OPSI), and other infections in hyposplenic patients.

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26

Colombetti, Giovanna, and Neil Harrison. From physiology to experience: Enriching existing conceptions of “arousal” in affective science. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198811930.003.0013.

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This chapter examines the notion of “arousal”, an influential notion in affective science referring to the degree of an individual’s “activation” or “excitement” during an emotional state. It considers this notion specifically in relation to interoception, defined broadly as “sensitivity to stimuli arising inside the organism.” “Physiological arousal” is distinguished from “experienced arousal” and it is argued that both need to be characterized more broadly than commonly done. Physiological arousal cannot be reduced to sympathetic activation, as it involves complex interactions between multiple functionally distinct pathways within sympathetic and parasympathetic divisions of the autonomic nervous system, as well as endocrine and immune systems, and even the gut microbiota. Relatedly, experienced arousal does not reduce to the perception of changes in the body sensed by visceral afferents in response to autonomic nervous system activity but also includes humorally mediated interoceptive pathways, somatic sensations of various kinds, and “background” bodily feelings.

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