Relevant bibliographies by topics / Immune

Academic literature on the topic 'Immune'

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Published: 4 June 2021
Last updated: 18 May 2024

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Journal articles on the topic "Immune"

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Chattopadhyay, Pratip K., Woodrow E. Lomas, Guo-Jian Gao, Na Li, and Suraj Saksena. "Immune monitoring for immuno-oncology applications." Journal of Immunology 204, no. 1_Supplement (May 1, 2020): 159.29. http://dx.doi.org/10.4049/jimmunol.204.supp.159.29.

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Abstract The immunotherapy revolution has spurred the development of many new drugs and drug regimens for patient treatment. A key challenge is to identify the factors that drive patient toxicities and responses to treatment, with a particularly acute need for predictive biomarkers that can discriminate patients destined to respond and fail treatment. Technologies to interrogate immune cells are now readily available, but important gaps remain in their application, which limit the full realization of the promise of precision oncology. High parameter flow cytometry is a gold-standard but is limited by difficulty of panel design and lack of standardization. We present Color Wheel, a panel design tool, which builds optimized antibody panels based on the user’s instrument. These optimized panels have been manufactured in a dried, ready to use format to drive workflow and assay standardization. Initial results demonstrate excellent concordance between the dried and liquid versions of the high parameter multicolor panel(s). Molecular cytometry represents an exciting new approach to high dimensional immune analysis because it can measure at least 102 proteins and 400 mRNA targets simultaneously per cell. An important application gap for this technology is lack of data indicating the sequencing depth needed for adequate resolution. Here, we present results from a molecular cytometry experiment sequenced deeply, and then bioinformatically sub-sampled at different levels to identify the minimum level of sequencing needed for clear identification of cells, which can serve as a reference guide for users to save time and cost in their experiments. We also present preliminary data generated using dried version of molecular cytometry panel(s).
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Kannan, Rajni, Kathleen Madden, and Stephanie Andrews. "Primer on Immuno-Oncology and Immune Response." Clinical Journal of Oncology Nursing 18, no. 3 (May 27, 2014): 311–17. http://dx.doi.org/10.1188/14.cjon.311-317.

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Holmström, Morten Orebo, Sabrina Cordua, Vibe Skov, Lasse Kjær, Niels Pallisgaard, Christina Ellervik, Hans Carl Hasselbalch, and Mads Hald Andersen. "Evidence of immune elimination, immuno-editing and immune escape in patients with hematological cancer." Cancer Immunology, Immunotherapy 69, no. 2 (January 8, 2020): 315–24. http://dx.doi.org/10.1007/s00262-019-02473-y.

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HK, Singhal. "Suvarnaprashan: An Ayurvedic Immune Booster." Journal of Natural & Ayurvedic Medicine 5, no. 3 (July 13, 2021): 1–4. http://dx.doi.org/10.23880/jonam-16000325.

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The word 'Suvarnaprashan', a logical offshoot of Ayurveda, is made with combination of two words 'Suvarna' and 'Prashan'. The term Suvarna is a common word which refers to the Gold noble metal. Prashan refers to Pra+Ashan which means specially the act of eating or drinking or in taking. Suvarnaprashan has been practiced since a long back to make Vyadhikshamatva i.e. Immunity stronger to prevent infectious diseases as well as maintenance of good physical and mental growth and development of a child. According to WHO, Health is a state of complete physical, mental and social wellbeing and not merely the absence of disease or infirmity. In the wider context, health is a state of total absence of illnesses, diseases, syndromes, infections, abnormal behavior, accidents etc. Vyadhikshamatva i.e. immunity or resistance is the power that protects the body from diseases. It depends on Ojas, Bala, Prakrita Kapha and Balavardhaka Bhava. There has been a significant deterioration in the quality of human health status from generation to generation resulting in decline of immunity. Samskara, Lehana and Rasayana medicines, Suvarnaprashan etc. are recommended in Ayurveda for children for the promotion of health and longevity of life span. According to ancient Ayurved Acharyas like Acharya Kashyap, Sushrut and Vagbhat, Suvarnaprashan strengthens immunity and simultaneously improves digestive functions, intellectual capabilities, activeness and vital power of the body, hence keeps a child healthy. Suvarnaprashan contributes in proper achievement of milestones significantly during growth and development process also.
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Dahal, Tshetiz, and Subarna Rizal. "INNATE IMMUNE CELLSIN IMMUNE TOLERANCE AFTER LIVER TRANSPLANTATION." International Journal of Advanced Research 10, no. 04 (April 30, 2022): 506–15. http://dx.doi.org/10.21474/ijar01/14575.

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Currently, liver transplantation is the most effective treatment for end-stage liver disease. Immuno-suppressive agents are required to be taken after the operations, which have significantly reduced rejection rates and improved the short-term (<1 year) survival rates. However, post-transplant complications related to the immuno-suppressive therapy have led to the development of new protocols aimed at protecting renal function and preventing de novo cancer and dysmetabolic syndrome. Donor specific immune tolerance, which means the mature immune systems of recipients will not attack the grafts under the conditions without any immunosuppression therapies, is considered the optimal state after liver transplantation. There have been studies that have shown that some patients can reach this immune tolerance state after liver transplantation. The intrahepatic immune system is quite different from that in other solid organs, especially the innate immune system. It contains a variety of liver specific cells, such as liver-derived dendritic cells, Kupffer cells, liver sinusoidal endothelial cells, liver-derived natural killer (NK) cells, natural killer T (NKT) cells, and so on. Depending on their specific structures and functions, these intrahepatic innate immune cells play important roles in the development of intrahepatic immune tolerance. In this article, in order to have a deeper understanding of the tolerogenic functions of liver, we summarized the molecular mechanisms of immune tolerance induced by intrahepatic innate immune cells after liver transplantation.
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Shastri, Nilabh, Chansu Park, and Jian Guan. "Immune surveillance of immune surveillance." Molecular Immunology 150 (October 2022): 2. http://dx.doi.org/10.1016/j.molimm.2022.05.018.

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Warsof, Steven L., Kypros H. Nicolaides, and Charles Rodeck. "Immune and Non-immune Hydrops." Clinical Obstetrics and Gynecology 29, no. 3 (September 1986): 533–42. http://dx.doi.org/10.1097/00003081-198609000-00009.

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Yazdanbakhsh, Karina, Hui Zhong, and Weili Bao. "Immune Dysregulation in Immune Thrombocytopenia." Seminars in Hematology 50 (January 2013): S63—S67. http://dx.doi.org/10.1053/j.seminhematol.2013.03.011.

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Davies, Graham E. "Immune mechanisms and immune deficiency." Current Opinion in Pediatrics 2, no. 1 (February 1990): 106–9. http://dx.doi.org/10.1097/00008480-199002000-00020.

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Skhiri, S., J. Anoun, B. H. Imen, A. Mzabi, F. Ben Fredj, M. Karmani, B. Achour, A. Rezgui, and L. Chadia. "Anémie hémolytique auto-immune et maladies auto-immunes." La Revue de Médecine Interne 41 (December 2020): A91. http://dx.doi.org/10.1016/j.revmed.2020.10.146.

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Dissertations / Theses on the topic "Immune"

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Vauleon, Elodie. "Implications des gènes immuns et des cellules immunes dans le glioblastome." Thesis, Rennes 1, 2013. http://www.theses.fr/2013REN1B005/document.

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Introduction : Le glioblastome (GBM) est la tumeur cérébrale primitive la plus fréquente et la plus grave de l’adulte. Des études épidémiologiques ont mis en évidence que les antécédents d’allergie sont un facteur protecteur, soulignant le possible impact de l’immunité sur le GBM. Plusieurs études transcriptomiques ont également mis en évidence des signatures immunes plus ou moins associées à la survie. Matériel et méthodes : Pour clarifier ce lien et déterminer quels gènes immuns étaient les plus impliqués dans le GBM, nous avons étudié l’expression de 791 gènes immuns dans des échantillons de GBM et de cerveaux normaux. Les interactions entre les gènes immuns ont été étudiées par une analyse de co-expression. Nous avons ensuite recherché une association entre les gènes immuns et la survie selon 3 méthodes statistiques, avant d’établir un modèle de risque mathématique validé sur plusieurs jeux de données. Enfin, nous avons étudié les cellules immunes infiltrantes sur des échantillons de gliomes dont 73 GBM par cytométrie de flux. Résultats : Un profil d’expression génique différent significativement entre le cerveau normal et le GBM a été établi de manière robuste, mais pas au sein des GBM. L’analyse de co-expression a mis en évidence 6 modules dont 5 sont enrichis en gènes ayant un lien avec la survie. Cent huit gènes immuns ont une association significative avec la survie et un prédicteur de risque à 6 gènes immuns a permis de distinguer deux groupes de patients en fonction de leur survie, y compris chez les patients dont la tumeur a un promoteur MGMT méthylé et dans le sous-groupe de GBM proneuraux. Enfin, nous avons mis en évidence, dans tous les échantillons de GBM analysés, une infiltration leucocytaire par des cellules macrophagiques/microgliales et parfois par des cellules lymphocytaires ou granulocytaires. L’infiltration de lymphocytes uniquement est associée significativement avec la survie dans notre cohorte. Conclusion : Des gènes, impliqués dans diverses fonctions immunes, sont différentiellement exprimés entre le cerveau normal et le GBM et au sein des GBM. Un prédicteur à 6 gènes robuste a été établi, il sépare les patients en 2 groupes bas et haut risque y compris ceux ayant un bon pronostic. Nous avons enfin mis en évidence dans une série de GBM une infiltration de cellules immunes, dont une infiltration lymphocytaire associée positivement à la survie
Background: Glioblastoma is the most common and lethal primary brain tumor in adults. Epidemiological studies have revealed that a history of allergies is a protective factor, thereby underlining the likely impact of the immune system on GBM. A number of transcriptomic studies have also identified immune signatures more or less associated with patient survival. Methods: In order to clarify and identify which immune-associated (IA) genes were the most involved in GBM, we studied the expression of 791 immune genes in GBM and normal brains samples. Interactions between IA genes were studied through an analysis of co-expression network. We then searched for a link between IA genes and patient survival according to 3 statistical methods, before defining a mathematical risk model based on different data sets. Finally, we studied the infiltrative immune population of 73 GBM by cytometry. Results: A significantly different profile of IA genes expression between healthy brains and GBM was consistently defined, but not among GBM. The analysis of co-expression network revealed 6 modules, 5 of which were enriched by genes associated with patient survival. 108 IA genes have a significant association with patient survival and the 6-IA gene risk predictor allowed us to distinguish two groups of patients according to their survival, including patients whose tumor had a methylated MGMT gene promoter and in the subset of proneural GBM. Finally, in every analyzed GBM sample, we have shown that there was a leukocyte infiltration by macrophages/microglial cells and sometimes by lymphocytes or granulocytes. Only the lymphocytes infiltration was significantly associated with the survival in our group of patients. Conclusion: IA genes that are involved in various immune functions are expressed differentially between healthy brains and GBM and amongst GBM. A robust 6-IA gene risk predictor was defined: it divides patients into two low and high risk groups, including those who have a good prognosis. Finally, we revealed an infiltration of immune cells in a series of GBM, only the lymphocytic infiltration was positively associated with patient survival
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Nerurkar, Louis. "Neuro-immune responses to distal immune stimulus." Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8529/.

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Depression is a major disease burden worldwide and, despite its prevalence and socioeconomic costs, around 30% of patients do not respond to currently available treatments. Inflammation is increasingly associated with, not only depressive illness but also resistance to existing therapies. This highlights the need for investigation of the mechanisms of neuro-inflammation, particularly in the context of peripheral inflammatory stimuli. Specifically, the chemokine molecular family is increasingly associated with human depressive illness, and neuro-inflammation and behavioural change in rodent models, making this an attractive molecular family for study. This thesis describes research aimed at investigating the association of these molecules with human depression and analysis of their role in an animal model of peripherally stimulated neuro-inflammation, the Aldara model of psoriasis-like inflammation. Systematic review and meta-analysis of the human biomarker literature using a random effects, inverse variance model revealed that a number of chemokines (CCL2, CCL3, CCL4, CCL11, CXCL4, CXCL7, CXCL8) are significantly associated with depressive illness in a human population. However this work revealed that there are a number of limitations of the human literature primarily associated with the methodological challenges of studies in human populations and confounding factors. Alongside this work, the Aldara model, which utilises the toll-like receptor 7 (TLR7) ligand imiquimod (IMQ), was investigated as a tool for studying neuroinflammation. Initial time-course investigation revealed that significant chemokine and cytokine transcriptional alterations occur within four hours at the local site of cutaneous treatment, the peripheral tissues and the brain. In addition, protein quantification in the brain confirmed that many of these transcriptional responses are translated to protein. Interestingly, it was shown that the brain response was temporally distinct from that of the peripheral tissues, and that in general brain responses were induced slightly more slowly and persisted for a longer period of time than those in the periphery. Investigation of Iba1+ (microglia/monocytes), GFAP+ (astrocytes) and CD3+ (T-cells) cells within the brain revealed significant changes in the microglial and T-cell populations, which were consistent with microgliosis and T-cell recruitment to the brain parenchyma. Changes in astrocyte populations were more equivocal although there was evidence of astrogliosis. Mechanistic investigations into responses to the Aldara model in inflammatory chemokine receptor (iCCR) KO mice did not reveal significant alterations in chemokine and cytokine transcription or in microglial responses to cutaneous Aldara treatment in the absence of the iCCRs (CCR1, CCR2, CCR3 and CCR5), but there did appear to be evidence of reduced CD3+ T-cell recruitment. In contrast, investigations in type I interferon receptor (IFNAR) KO mice identified a clear role for type I IFN signalling through IFNAR in the induction of chemokine and cytokine gene expression in the brain, and associated changes in Iba1+ microglial and CD3+ T-cell populations in response to cutaneous Aldara treatment. Mass spectrometric analysis of IMQ, the main active ingredient of Aldara, revealed that within four hours it enters both the circulation and the brain. The finding of IMQ within the brain parenchyma suggests that, while it is not an appropriate tool for studying peripheral-central immune crosstalk, it is a useful non-invasive model of TLR7 mediated neuroinflammation. These data provide compelling evidence of a role for chemokines in human depression and in neuro-inflammation, although the precise actions of this family of molecules remain unclear. In addition, building on previous work, the Aldara model appears to be a suitable tool for the study of neuro-inflammation, particularly interferon-driven immune responses, but is less appropriate for studying peripherally driven CNS immune reactions. Further work into the specific role of chemokines and associated cellular populations will hopefully provide additional insight into how CNS immune reactions are co-ordinated.
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Johansson, Susanne. "Compartmentmentalized immuno-sequencing (cI-Seq) : identification of immune complex interactions." Thesis, KTH, Genteknologi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-196872.

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Today, a lot of proteomic research is aimed at discovering disease specific proteins. This requires theavailability of high-throughput, ultra-sensitive protein detection methods. Compartmentalized immunosequencing(cI-Seq) is a proximity-independent immuno-polymerase chain reaction (IPCR) based proteindetection method. Antigen recognition in cI-Seq is mediated by antibody pairs in which one of theantibodies is conjugated to a DNA-probe. The affinity recognition events occur in emulsion droplets inwhich the DNA-probes will be amplified through emulsion PCR (emPCR) and thereafter analyzed usingMassively Parallel Sequencing (MPS). The amplifiable nature of the DNA-probes improves the sensitivityof the detection, while the use of emulsion droplets and MPS increases the multiplex capacity andthroughput. Ultimately, cI-Seq enables analysis and detection even of lowly abundant proteins therebyincreasing the probability of discovering novel disease specific proteins. In this project, conjugation of DNA probes to antibodies was performed through two different approaches;Covalent Conjugation and Conjugation using Biotin and NeutrAvidin. Both of these approaches showedadvantageous and disadvantageous features. However, neither of them succeeded in producing stableconjugates in an efficient and reproducible manner. After conjugation, the DNA-conjugated antibodieswere used in immune complex formation. However, the immune complexes either failed to form or wereformed in an inefficient manner.
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Protheroe, Rachel Elizabeth. "Investigation of immune regulation in human alloreactive immune responses." Thesis, University of Bristol, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.520287.

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Rosa, Gustavo Luis Teixeira Lopes. "Studies of MHV-68 immune evasion and immune control." Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.611892.

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McIntosh, Alistair James. "The role of adipose tissue immune cells in immune responses." Thesis, University of Birmingham, 2018. http://etheses.bham.ac.uk//id/eprint/8113/.

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Recent evidence indicates that immune cells within adipose tissues can drive the formation of ectopic lymphoid structures, known as Fat Associated Lymphoid Clusters (FALC). FALC support B-cell antibody production in response to infection and inflammation. This investigation explores the immune cell composition and role of different adipose tissues both in steady state and during immune responses in mice. Firstly, a detailed analysis of the immune cell composition of peritoneal adipose tissues was performed. To investigate the function and migratory properties of these tissue-resident cells, cytokine and chemokine receptor expression was then assessed. How immune cells in adipose tissues responded to infection was examined using an intestinal helminth infection with the parasite Heligmosomoides polygyrus. Adipose tissues predominantly contained regulatory T cells, invariant natural killer T cells and group 2 Innate Lymphoid Cells (ILC2s). Significant differences were observed in composition, cell surface markers and cytokine production of ILC2s between adipose depots and secondary lymphoid tissues, indicating that tissue specific signals can direct ILC2 responses. Finally, increases were observed in ILC2 and FALC numbers in the mesenteries of WT mice following parasite infection. These data indicate that immune cells within adipose tissues respond to infection and may contribute to immune responses.
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SINGH, REETIKA. "COMPARATIVE ANALYSIS OF DIFFERENT CURCUMIN ANALOGUES TO INHIBIT TLR4 EXPRESSION IN BREAST CANCER- AN IN-SILICO STUDY." Thesis, DELHI TECHNOLOGICAL UNIVERSITY, 2021. http://dspace.dtu.ac.in:8080/jspui/handle/repository/18459.

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Chronic inflammation is closely related to the emergence of a number of cancers, including Breast cancer. Inflammation causes damage to the cell’s DNA which leads to its abnormal growth and formation of tumor mass. One of the most commonly known receptor responsible for inflammatory reactions is Toll-like receptor 4 (TLR4). It is activated majorly by bacterial LPS. Its activation further activates Cyclooxygenase enzyme that catalyzes the conversion of arachidonic acid into prostaglandins that lead to inflammation-like conditions. COX2 has also been correlated to the promotion of tumor growth. It enhances metastasis, neoplasia, lymphangiogenesis, etc., and is also related to poor prognosis in the breast cancer patients. Curcumin derived from turmeric is a proven inhibitor of COX2. In my project I have aimed to analyse and compare the inhibitory properties of other analogues of curcumin that have previously been known to inhibit COX2. The experimental layout began with screening the molecules on the basis of drug-likeness using Lipinski rule of five. The suitable ligand molecules were further subjected to other experiments, i.e., ligand docking and drug potential assessment. After all the experiments, out of the five selected Curcumin analogues, Isoeugenol (extracted from clove) was determined as the best fit molecule. The druglikeliness and drug potential assessment results further validate its use as a potential inhibitor and can further be tested for in-vivo efficacy. This drug can further be used in the 1st line therapy of locally advanced and metastatic breast cancer patients as it will inhibit COX2 that promotes metastasis of cancer cells. Isoeugenol extracted from Eugenia caryophyllus (Cloves) can further be proven as a better COX2 inhibitor than its chemical counterparts, as it is a natural compound and will therefore have significantly less side effects.
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Wassall, Heather Jane Haining. "Dietary influences and immune regulation of neonatal immune responses to allergens." Thesis, University of Aberdeen, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.485812.

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The prevalence of asthma and atopic disease in general, has been increasing in the past forty years, with the UK having one of the highest rates in the world. There have been many theories proposed to explain these increases, but no definitive answer has been found, as yet. Dietary hypotheses suggest that the recent increases in asthma and atopic disease are a consequence of changing dietary lipid intake and lor decreasing dietary antioxidant intake. This hypothesis has been supported by local work, which- has demonstrated that a maternal diet deficient in some antioxidants may influence childhood Th-cell differentiation towards the Th2 phenotype in vitro. Previously, it was thought that Th1 cells regulated Th2 cells. However, it is now thought that Th2 responses may be regulated by other cells, such as Tregulatory cells, (Tr-cells) that have the potential to be involved in the immunopathogenesis ofasthma and atopic disease. . The aims of this project were to characterise -the effects of direct antioxidant and lipid supplementation on umbilical cord blood Th-cells in vitro, and to determine whether the proliferative and cytokine responses of cord blood mononuclear cells, (CBMC) after stimulation with allergens, were mediated through regulatory T-cells. Supplementation with vitamin E, vitamin C or a.-linolenic acid was performed on 135 neonatal cord blood samples. Cell proliferation was quantified 5,6 and 7 days, after stimulation with mitogen, control antigens and allergens, by incorporation of 3H-thymidine into triplicate lOO~1 aliquots drawn from the cultures. Production of the Th1 and Th2 cytokines IFN-y and IL-4, and the regulatory cytokines IL-IO and TGF-13 were measured by a sensitive cellular ELISA adapted for use with CBMC. Flow cytometry was used to identify the phenotype ofTr-cells, by staining for surface antigens and intracellular cytokines. This project has demonstrated that direct supplementation of cord blood Th-cells with vitamin E, vitamin C and a.-linole,nic acid, does influence proliferative and cytokine responses to control stimuli and allergens. Specifically, these nutrients are associated with a general suppression of CBMC proliferative responses to control stimuli and the allergen house dust mite. In contrast, the two vitamins are associated with an increase in CBMC proliferative responses to the allergen, timothy grass pollen. Supplementation is also associated with a general decrease in IFN-y and TGF-13 secretion, with varying effects on IL-IO and IL-4 secretion. This work has also confirmed the presence of functional Tr-cells in neonatal blood at birth. The phenotype of these Tr-cells varies, with cells expressing various combinations and numbers of the three Tr-cell markers; CD25+high, Foxp3 and IL-IO. Allergen stimulation appears to induce Tr-cells in the greatest amounts. In conclusion, this project has demonstrated, for the first time, that direct supplementation of CBMC, with vitamins E and C, and the PUFA, a-linolenic acid, does influence cord blood Th-cell proliferative and cytokine responses to control stimuli and allergens, altering the balance of Th-cell subsets, and suggests that manipulation of maternal diet during pregnancy could modulate neonatal immune responses to allergens and the development of asthma and atopic disease. It has also confirmed the presence of functional Tr-cells in neonatal blood at birth, with variability in numbers between individuals, upon allergen stimulation. The variability in Tr-cell activity associated with allergen stimulation may be linked to the development ofatopic disease in later life.
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Macaulay, R. "The role of immune inhibitory receptors in age-associated immune decline." Thesis, University College London (University of London), 2011. http://discovery.ucl.ac.uk/1317772/.

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The balance between signals delivered by positive and negative costimulatory molecules is crucial to the ultimate fate of cellular immune responses. Manipulation of T cell costimulatory pathways may offer a novel approach for reinvigorating exhausted T cell responses, especially in the context of chronic infections. T cells also display profound exhaustion in old age and this thesis investigates the hypothesis that T cell inhibitory receptor upregulation may define a reversible defect in age onset immune decline. Data presented here illustrate how T cells utilise different inhibitory receptors as they differentiate and that KLRG1 signalling is causative of dysfunctions in highly differentiated CD8+ T cells. The inhibitory receptors KLRG1 and CTLA-4 are revealed to undergo age-associated upregulations on CD8+ T cells but their blockade does not reverse the characteristic hypo-responsiveness of CD8+ T cells amongst old donors. The dysregulated immune response to lifelong chronic cytomegalovirus (CMV) infection is thought to play a major role in driving age related immune dysfunctions. We found that CMV accelerates age-associated telomere attrition amongst CD8+ and CD4+ T cells. CMV infection is also shown to drive CTLA-4, PD-1 and KLRG1 upregulation on both CD4+ and CD8+ T cells. Moreover, the PD-1/Ligand (L) inhibitory pathway defines a reversible proliferative dysfunction in the responses of CMV specific CD8+ T cells. Specifically, the CD45RA re-expressing memory subset exhibits a proliferative deficiency, relative to their central and effector memory counterparts, that is reversible upon PD-L blockade. However, this augmented proliferative response was not accompanied by increased telomerase function, suggesting this does not result in true reversal of exhaustion. In summary, the dysfunctions of highly differentiated and CMV specific CD8+ T cells can be at least partially reversed by perturbation of inhibitory receptor pathways, whose further manipulation may provide a therapeutic modality to combat age-associated immune decline.
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Dehus, Oliver. "Receptor polymorphisms and non-classical immune stimuli in bacterial immune recognition." [S.l. : s.n.], 2008. http://nbn-resolving.de/urn:nbn:de:bsz:352-opus-61639.

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Books on the topic "Immune"

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Phillips, Richard. Immune. Las Vegas, NV: 47 North, 2010.

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Stein-Streilein, Joan, ed. Infection, Immune Homeostasis and Immune Privilege. Basel: Springer Basel, 2012. http://dx.doi.org/10.1007/978-3-0348-0445-5.

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Freitas, Antonio A. de. Tractus immuno-logicus: A brief history of the immune system. Austin, Tex: Landes Bioscience, 2009.

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Freitas, Antonio A. de. Tractus immuno-logicus: A brief history of the immune system. Austin, Tex: Landes Bioscience, 2009.

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Freitas, Antonio A. de. Tractus immuno-logicus: A brief history of the immune system. Austin, Tex: Landes Bioscience, 2009.

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Freitas, Antonio A. de. Tractus immuno-logicus: A brief history of the immune system. Austin, Tex: Landes Bioscience, 2009.

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Immune power: Health and the immune system. London: Optima, 1990.

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The immune. Orlando, FL: LJS & S Pub., 2011.

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Lane, I. William. Immune power. Garden City Park, NY: Avery Pub. Group, 1999.

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Der Immune. Zürich: Diogenes, 1985.

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Book chapters on the topic "Immune"

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Wendt, Julie, Colleen Considine, and Mikhail Kogan. "Immune." In Integrative Geriatric Nutrition, 59–87. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81758-9_4.

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Rose, Randall, and Alan S. Perelson. "Immune Networks and Immune Responses." In Lecture Notes in Biomathematics, 159–72. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-50124-1_9.

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Sedlacek, H. Harald, and Tarik Möröy. "Immune Suppression." In Immune Reactions, 333–43. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79558-9_21.

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Wahn, Volker. "From Immune Substitution to Immuno-modulation." In Antibody Therapy, 15–22. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-68038-5_2.

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Nixon, Brett, Matthew D. Dun, and R. John Aitken. "Proteomic Analysis of Human Spermatozoa." In Immune Infertility, 3–22. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40788-3_1.

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Clarke, Gary N. "ASA in the Female." In Immune Infertility, 161–71. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40788-3_10.

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Hasegawa, Akiko, Minoru Shigeta, and Hiroaki Shibahara. "Sperm Immobilizing Antibody and Its Target Antigen." In Immune Infertility, 173–84. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40788-3_11.

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Francavilla, Felice, and Arcangelo Barbonetti. "Male Autoimmune Infertility." In Immune Infertility, 187–96. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40788-3_12.

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Said, Tamer M., and Ashok Agarwal. "Tests for Sperm Antibodies." In Immune Infertility, 197–207. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40788-3_13.

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Ulcova-Gallova, Zdenka, and Petr Losan. "Impact on Fertility Outcome." In Immune Infertility, 209–21. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40788-3_14.

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Conference papers on the topic "Immune"

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Raza, Ali, and Benito R. Fernandez. "Artificial Immune System for Heterogeneous Mobile Robotic Systems." In ASME 2010 Dynamic Systems and Control Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/dscc2010-4264.

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Artificial immune system draws its inspiration from the biological immune functions mainly those of humans. Recently, newer definitions of biological immune system have appeared and gained significance because of their strong immunological roots e.g. danger theory. This raises the need to look into earlier work on immuno-inspired robotics. Especially, older approach of idiotypic-network must be compared with the newer approach of danger-theory. Authors in this research have successfully applied both the definitions on heterogeneous mobile robotic systems. Idiotypic connections between antibodies have been used as a tool to navigate robots as well as to establish inter-robot communication in an immune network approach. Similarly, co-stimulatory signal concentrations have been used to contextualize the environment, in a danger theory approach, to initiate and regulate the immuno responses. Immune metaphors have been translated into relevant computational models and simulated in search and rescue operation in an obstacle filled arena.
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Lomas, Woodrow E., Guo-Jian Gao, Na Li, Suraj Saksena, and Pratip K. Chattopadhyay. "Abstract 3301: Immune monitoring for immuno-oncology applications." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-3301.

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"Front Matter: Volume 10495." In Biophotonics and Immune Responses XIII, edited by Wei R. Chen. SPIE, 2018. http://dx.doi.org/10.1117/12.2323042.

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Zhou, Benqing, Meng Wang, Feifan Zhou, Jun Song, Junle Qu, and Wei R. Chen. "BSA-modified gold nanorods for combined photothermal therapy and immunotherapy of melanoma." In Biophotonics and Immune Responses XIV, edited by Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2506097.

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Qi, Shuhong, Lisen Lu, and Zhihong Zhang. "ALDH3A2 as a potential molecular marker for nasopharyngeal carcinoma." In Biophotonics and Immune Responses XIV, edited by Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2506121.

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Gallagher, Kyra A., Austin Doughty, Elivia Layton, Sara Zukerman, Benqing Zhou, and Wei R. Chen. "The characterization of graphene-reconstituted low-density lipoproteins and small molecule inhibitor nanoparticles in preparation for use in laser immunotherapy." In Biophotonics and Immune Responses XIV, edited by Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2506714.

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Chen, Wei R., Jingxuan Yang, Min Li, Feifan Zhou, and Meng Wang. "Treatment of pancreatic tumors using combination of laser irradiation and immunological stimulation (Conference Presentation)." In Biophotonics and Immune Responses XIV, edited by Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2506731.

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Wang, Meng, Lu Wang, Benqing Zhou, Feifan Zhou, and Wei Chen. "NIR-triggered photodynamic therapy via antigen-capturing upconversion nanoparticle enhances cancer immunotherapy (Conference Presentation)." In Biophotonics and Immune Responses XIV, edited by Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2507021.

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Liu, Siyu, Feifan Zhou, Yue Zhao, Liwei Liu, Junle Qu, and Wei R. Chen. "Temperature detection by photoacoustic for nanoprobe-mediated photothermal therapy." In Biophotonics and Immune Responses XIV, edited by Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2507030.

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Chen, Haibin, Dezi Li, and Zhifang Li. "Temperature feedback-controlled photothermal treatment based on thermal infrared imager." In Biophotonics and Immune Responses XIV, edited by Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2507579.

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Reports on the topic "Immune"

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Poschet, Jens Fredrich, Amanda Carroll-Portillo, Meiye Wu, Ronald Paul Manginell, Amy Elizabeth Herr, Anthony A. Martino, Thomas D. Perroud, et al. Microscale Immune Studies Laboratory. Office of Scientific and Technical Information (OSTI), January 2009. http://dx.doi.org/10.2172/976947.

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Frame, John D. Lassa Fever Immune Plasma. Fort Belvoir, VA: Defense Technical Information Center, October 1990. http://dx.doi.org/10.21236/ada232145.

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Maier, Steven. Coping and Immune Function. Fort Belvoir, VA: Defense Technical Information Center, May 1991. http://dx.doi.org/10.21236/ada236811.

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Zelikoff, Judith T. Fish Immune Response as Biomarkers. Fort Belvoir, VA: Defense Technical Information Center, March 2001. http://dx.doi.org/10.21236/ada399403.

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Redmond, Sarah Beth, Rachel Tell, Derrick Coble, Carrie Mueller, Dušan Palić, Claire B. Andreasen, and Susan J. Lamont. Genetic Differences in Chicken Splenic Immune Gene Expression in Response to Dietary Immune Modulation. Ames (Iowa): Iowa State University, January 2010. http://dx.doi.org/10.31274/ans_air-180814-166.

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Bowles, Charles A. Immune Alteration Studies in Irradiated Dogs. Fort Belvoir, VA: Defense Technical Information Center, October 1986. http://dx.doi.org/10.21236/ada191079.

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Herrmann, Melissa S., Michael G. Kaiser, and Susan J. Lamont. Rooster’s Genetic Response to Immune Stimulation. Ames (Iowa): Iowa State University, January 2015. http://dx.doi.org/10.31274/ans_air-180814-1314.

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Beury, Daniel W., and Suzanne Ostrand-Rosenberg. Reversing Breast Cancer-Induced Immune Suppression. Fort Belvoir, VA: Defense Technical Information Center, January 2013. http://dx.doi.org/10.21236/ada573503.

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Conejo-Garcia, Jose R. Reprogramming Antitumor Immune Responses with microRNAs. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada585107.

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Conejo-Garcia, Jose R. Reprogramming Antitumor Immune Responses with microRNAs. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada595676.

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