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Relevant bibliographies by topics / IL-10 deficiency

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Journal articles

Academic literature on the topic 'IL-10 deficiency'

Author: Grafiati

Published: 1 April 2023

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Journal articles on the topic "IL-10 deficiency"

1

Asadullah, K., K. Stephanek, M. Leupold, et al. "Relative IL-10 deficiency and effects of IL-10 therapy in psoriasis." Journal of Dermatological Science 16 (March 1998): S30. http://dx.doi.org/10.1016/s0923-1811(98)83173-3.

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2

Wan, Xin, Wen Juan Huang, Wen Chen, et al. "IL-10 Deficiency Increases Renal Ischemia-Reperfusion Injury." Nephron Experimental Nephrology 128, no. 1-2 (2014): 37–45. http://dx.doi.org/10.1159/000366130.

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3

Hennessy, A., H. L. Pilmore, L. A. Simmons, and D. M. Painter. "A Deficiency of Placental IL-10 in Preeclampsia." Journal of Immunology 163, no. 6 (1999): 3491–95. http://dx.doi.org/10.4049/jimmunol.163.6.3491.

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Abstract Accommodation of the fetoplacental unit in human pregnancy requires maternal immune tolerance to this “semiallograft”. Local antiplacental immunity is modified by synthesis of uncommon histocompatibility Ags (e.g., HLA-G), growth factors, and cytokines by the placenta. Placental interleukins have been identified in reproductive tissues, but their roles in adaptive maternal immunity and determining term pregnancy outcomes have not been fully clarified. This study examined the distribution of IL-10 and TNF-α staining in term placentas. Women with proteinuric hypertension (PE, n = 10) we

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4

Zigmond, Ehud, Biana Bernshtein, Gilgi Friedlander, et al. "Macrophage-Restricted Interleukin-10 Receptor Deficiency, but Not IL-10 Deficiency, Causes Severe Spontaneous Colitis." Immunity 40, no. 5 (2014): 720–33. http://dx.doi.org/10.1016/j.immuni.2014.03.012.

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5

Jones, Steven P., Steven D. Trocha, and David J. Lefer. "Cardioprotective actions of endogenous IL-10 are independent of iNOS." American Journal of Physiology-Heart and Circulatory Physiology 281, no. 1 (2001): H48—H52. http://dx.doi.org/10.1152/ajpheart.2001.281.1.h48.

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Myocardial ischemia-reperfusion (I/R) is a well-known stimulus for acute inflammatory responses that promote cell death and impair pump function. Interleukin-10 (IL-10) is an endogenous, potent anti-inflammatory cytokine. Recently, it has been proposed that IL-10 inhibits inducible nitric oxide synthase (iNOS) activity after myocardial I/R and consequently exerts cardioprotective effects. However, whether this actually occurs remains unclear. To test this hypothesis, we utilized iNOS-deficient (−/−), IL-10 −/−, and IL-10/iNOS −/− mice to examine the potential mechanism of IL-10-mediated cardio

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6

Gunnett, Carol A., Donald D. Heistad, Daniel J. Berg, and Frank M. Faraci. "IL-10 deficiency increases superoxide and endothelial dysfunction during inflammation." American Journal of Physiology-Heart and Circulatory Physiology 279, no. 4 (2000): H1555—H1562. http://dx.doi.org/10.1152/ajpheart.2000.279.4.h1555.

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Little is known about the role of interleukin-10 (IL-10), an anti-inflammatory cytokine, in blood vessels. We used IL-10-deficient mice (IL-10 −/−) to examine the hypothesis that IL-10 protects endothelial function after lipopolysaccharide (LPS) treatment. The responses of carotid arteries were studied in vitro 6 h after injection of a relatively low dose of LPS (10 μg ip). In IL-10 −/− mice, the maximum relaxation to ACh (3 μM) was 56 ± 6% (means ± SE) after LPS injection and 84 ± 4% after vehicle injection ( P < 0.05). Thus endothelium-dependent relaxation was impaired in carotid arteries

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7

Wang, B., L. Zhuang, H. Fujisawa, and D. N. Sauder. "Effect of IL-10 deficiency on the migration of epidermal Langerhans cells in IL-10 null mice." Journal of Dermatological Science 16 (March 1998): S90. http://dx.doi.org/10.1016/s0923-1811(98)83537-8.

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8

Zhang, Guag-Xian, Fang Zhou, Bogoljub Ciric, et al. "Ability of LPS to regulate expression of tolerance-related molecules on dendritic cells is blocked by IL-10 deficiency (48.18)." Journal of Immunology 188, no. 1_Supplement (2012): 48.18. http://dx.doi.org/10.4049/jimmunol.188.supp.48.18.

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Abstract Interleukin-10 (IL-10) is an anti-inflammatory cytokine that plays an important role in regulating the local inflammatory immune response, but regulatory mechanisms of this cytokine have not been fully elucidated. Here we demonstrate that IL-10 deficiency renders LPS treatment ineffective in regulating expression of CD40, CD80, CD86, B7-H2 and B7-DC on dendritic cells (DCs) and blocks up-regulation of IL-27. This inability to respond to LPS was found in both IL-10-/- bone marrow-derived and splenic DCs. Compared to wild type DCs, IL-10-/- DCs expressed similar levels of TLR4 and CD14,

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9

Pérez-de Puig, Isabel, Francesc Miró, Angélica Salas-Perdomo, et al. "IL-10 Deficiency Exacerbates the Brain Inflammatory Response to Permanent Ischemia without Preventing Resolution of the Lesion." Journal of Cerebral Blood Flow & Metabolism 33, no. 12 (2013): 1955–66. http://dx.doi.org/10.1038/jcbfm.2013.155.

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Stroke induces inflammation that can aggravate brain damage. This work examines whether interleukin-10 (IL-10) deficiency exacerbates inflammation and worsens the outcome of permanent middle cerebral artery occlusion (pMCAO). Expression of IL-10 and IL-10 receptor (IL-10R) increased after ischemia. From day 4, reactive astrocytes showed strong IL-10R immunoreactivity. Interleukin-10 knockout (IL-10 KO) mice kept in conventional housing showed more mortality after pMCAO than the wild type (WT). This effect was associated with the presence of signs of colitis in the IL-10 KO mice, suggesting tha

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10

Marco, Roberto Di, Ming Xiang, Paola Zaccone, et al. "Concanavalin A-induced Hepatitis in Mice is Prevented by Interleukin (IL)-10 and Exacerbated by Endogenous IL-10 Deficiency." Autoimmunity 31, no. 2 (1999): 75–83. http://dx.doi.org/10.3109/08916939908994050.

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