Relevant bibliographies by topics / IgE mediated allergy

Academic literature on the topic 'IgE mediated allergy'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "IgE mediated allergy"

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Białek, Sławomir, and Katarzyna Białek-Gosk. "Modern diagnosis of IgE-mediated allergy – molecular diagnosis of allergies." Diagnostyka Laboratoryjna 52, no. 1 (April 18, 2016): 45–50. http://dx.doi.org/10.5604/01.3001.0008.9630.

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Diagnostic difficulties resulting from the imperfections of natural allergen extracts inspired to use genetic engineering techniques to produce recombinant allergens or obtaining highly purified components (component) allergen. This led to the development of modern diagnostic technique in allergy or molecular diagnostics. The basis for understanding the molecular diagnosis of allergies is to know the properties of allergens. Each allergen is composed of various proteins known. component capable of sensitizing allergen, and each component includes a plurality of epitopes that can be divided into one species-specific epitopes, and the identical amino acid structure of the epitopes derived from different species. Specific epitopes are responsible for primary sensitization, while the epitopes with similar structures are responsible for cross-reactions. Finding sensitization several epitopes is a strong indication of the occurrence of much more dangerous allergic reactions than only one epitope. In addition, molecular diagnosis of allergies allows for personalized diagnosis of allergic patients. It enables the assessment of individual risk of allergic symptoms and allows you to distinguish the original from allergy symptoms caused by cross-reactions. It should be noted, however, that the diagnosis of allergy should be based on a comprehensive evaluation of the results and their confrontation with data from the interview. The mere detection of allergen-specific IgE antibodies, even the method of molecular diagnostics, without the presence of clinical symptoms does not confirm an allergy or illness. Only goes to confirm that the body of such a person is allergic and that the symptoms of this condition may at some point reveal but not necessarily.
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Zhang, PeiAo, Jihui Gao, Huilian Che, Wentong Xue, and Dong Yang. "Molecular Basis of IgE-Mediated Shrimp Allergy and Heat Desensitization." Nutrients 13, no. 10 (September 27, 2021): 3397. http://dx.doi.org/10.3390/nu13103397.

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Crustacean allergy, especially to shrimp, is the most predominant cause of seafood allergy. However, due to the high flexibility of immunoglobulin E (IgE), its three-dimensional structure remains unsolved, and the molecular mechanism of shrimp allergen recognition is unknown. Here a chimeric IgE was built in silico, and its variable region in the light chain was replaced with sequences derived from shrimp tropomyosin (TM)-allergic patients. A variety of allergenic peptides from the Chinese shrimp TM were built, treated with heating, and subjected to IgE binding in silico. Amino acid analysis shows that the amino acid residue conservation in shrimp TM contributes to eliciting an IgE-mediated immune response. In the shrimp-allergic IgE, Glu98 in the light chain and other critical residues that recognize allergens from shrimp are implicated in the molecular basis of IgE-mediated shrimp allergy. Heat treatment could alter the conformations of TM allergenic peptides, impact their intramolecular hydrogen bonding, and subsequently decrease the binding between these peptides and IgE. We found Glu98 as the characteristic amino acid residue in the light chain of IgE to recognize general shrimp-allergic sequences, and heat-induced conformational change generally desensitizes shrimp allergens.
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Cianferoni, Antonella. "Non-IgE Mediated Food Allergy." Current Pediatric Reviews 16, no. 2 (July 1, 2020): 95–105. http://dx.doi.org/10.2174/1573396315666191031103714.

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: Food allergies, defined as an immune response to food proteins, affect as many as 8% of young children and 2% of adults in western countries, and their prevalence appears to be rising like all allergic diseases. In addition to well-recognized urticaria and anaphylaxis triggered by IgE antibody– mediated immune responses, there is an increasing recognition of cell-mediated disorders, such as eosinophilic esophagitis and food protein–induced enterocolitis. Non-IgE-Mediated gastrointestinal food allergies are a heterogeneous group of food allergies in which there is an immune reaction against food but the primary pathogenesis is not a production of IgE and activation of mast cells and basophils. : Those diseases tend to affect mainly the gastrointestinal tract and can present as acute (FPIES) or chronic reaction, such as Eosinophilic Esophagitis (EoE), Food Protein-Induced Allergic Proctocolitis (FPIAP). The role of food allergy in Non-EoE gastrointestinal Eosinophilic disorders (Non- EoE EGID) is poorly understood. : In some diseases like EoE, T cell seems to play a major role in initiating the immunological reaction against food, however, in FPIES and FPIAP, the mechanism of sensitization is not clear. : Diagnosis requires food challenges and/or endoscopies in most of the patients, as there are no validated biomarkers that can be used for monitoring or diagnosis of Non-IgE mediated food allergies. : The treatment of Non-IgE food allergy is dependent on diet (FPIES, and EoE) and/or use of drugs (i.e. steroids, PPI) in EoE and Non-EoE EGID. : Non-IgE mediated food allergies are being being investigated.
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Cianferoni, Antonella. "Non-IgE Mediated Food Allergy." Current Pediatric Reviews 16, no. 2 (May 2020): 95–105. http://dx.doi.org/10.2174/157339631566619103110371.

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Food allergies, defined as an immune response to food proteins, affect as many as 8% of young children and 2% of adults in western countries, and their prevalence appears to be rising like all allergic diseases. In addition to well-recognized urticaria and anaphylaxis triggered by IgE antibody– mediated immune responses, there is an increasing recognition of cell-mediated disorders, such as eosinophilic esophagitis and food protein–induced enterocolitis. Non-IgE-Mediated gastrointestinal food allergies are a heterogeneous group of food allergies in which there is an immune reaction against food but the primary pathogenesis is not a production of IgE and activation of mast cells and basophils. Those diseases tend to affect mainly the gastrointestinal tract and can present as acute (FPIES) or chronic reaction, such as Eosinophilic Esophagitis (EoE), Food Protein-Induced Allergic Proctocolitis (FPIAP). The role of food allergy in Non-EoE gastrointestinal Eosinophilic disorders (Non- EoE EGID) is poorly understood. In some diseases like EoE, T cell seems to play a major role in initiating the immunological reaction against food, however, in FPIES and FPIAP, the mechanism of sensitization is not clear. Diagnosis requires food challenges and/or endoscopies in most of the patients, as there are no validated biomarkers that can be used for monitoring or diagnosis of Non-IgE mediated food allergies. The treatment of Non-IgE food allergy is dependent on diet (FPIES, and EoE) and/or use of drugs (i.e. steroids, PPI) in EoE and Non-EoE EGID. Non-IgE mediated food allergies are being being investigated.
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Wei, Tianhao. "Pathological Mechanisms Underlying IgE-mediated Food Allergy." Highlights in Science, Engineering and Technology 2 (June 22, 2022): 230–34. http://dx.doi.org/10.54097/hset.v2i.578.

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Food allergy is an immune disease triggered by abnormal immune responses against harmless antigens that enter through our gut lumen. There are two major pathways that contribute to the food allergic symptoms: IgE-mediated and non IgE-mediated. Among all food allergy cases that have already been discovered, IgE-mediated mechanisms are responsible for over 80-90% of the cases. The IgE-mediated mechanisms include epithelium damage, T helper 2 cell induction, IgE antibody production, and the final symptoms caused by the effector cells. We also discovered that there may exist a potential relationship between B cell metabolism and the IgE production, which ultimately leads to food allergy. At the same time, since more and more people now enjoy more diverse food sources, issues regarding food allergy are outbursting these years as people’s exposure to different food proteins and antigens rapidly increase. It is shown that the United States government is losing billions dollars annually to cover the lost caused by food allergy. Given the worldwide prevalence of the food allergy and the increasingly unhealthy lifestyles of many people, it is highly crucial for us to understand the fundamental mechanisms underlying the IgE-mediated food allergy, which is the most common and influential pathway that risks millions of lives. Therefore, this Review goes over the basic mechanisms underlying the IgE-mediated food allergy, namely how epithelium damage, T helper 2 cell induction, IgE antibody production, effector cell activation, and B cell metabolism lead to the final symptoms of food allergy.
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Anvari, Sara, Jennifer Miller, Chih-Yin Yeh, and Carla M. Davis. "IgE-Mediated Food Allergy." Clinical Reviews in Allergy & Immunology 57, no. 2 (October 29, 2018): 244–60. http://dx.doi.org/10.1007/s12016-018-8710-3.

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Calhoun, Karen H., and Minka L. Schofield. "IgE-mediated food allergy." Current Opinion in Otolaryngology & Head and Neck Surgery 18, no. 3 (June 2010): 182–86. http://dx.doi.org/10.1097/moo.0b013e328339530e.

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Satyaraj, Ebenezer, Peichuan Sun, and Scott Sherrill. "Fel d1 Blocking Antibodies: A Novel Method to Reduce IgE-Mediated Allergy to Cats." Journal of Immunology Research 2021 (June 19, 2021): 1–7. http://dx.doi.org/10.1155/2021/5545173.

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Fel d1 is an important allergen produced by cats that causes IgE reactions in up to 95% of cat-allergic adults. Immunotherapy to reduce human allergy to cats has demonstrated that people have the capacity to produce allergen-specific neutralizing antibodies that block IgE-mediated allergic responses. We wished to determine if “blocking” antibodies could be used to reduce the IgE binding ability of cat allergens prior to their exposure to humans. Here, we describe the characterization of Fel d1-specific antibodies. We demonstrated the efficacy of a rabbit polyclonal and an allergen-specific chicken IgY to bind to Fel d1 in cat saliva and block Fel d1-IgE binding and IgE-mediated basophil degranulation. Fel d1 blocking antibodies offer a new and exciting approach to the neutralization of cat allergens.
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Shreffler, Wayne G. "Pathophysiology of immunoglobulin E‐mediated food allergy." Journal of Food Allergy 2, no. 1 (September 1, 2020): 7–10. http://dx.doi.org/10.2500/jfa.2020.2.200024.

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The pathophysiology of immunoglobulin E (IgE) mediated food allergy has been understood on a superficial level for several decades. Surveillance by dendritic cells for exogenous antigens leads to a high-affinity IgE response that arms effector cells (sensitization), such that subsequent exposures can trigger a type 1 hypersensitivity recall response. However, merely scratching the surface, whether confronting unmet needs in a clinical setting or probing the basic immunology of allergic immunity, quickly reveals the many unmet fundamental questions that lie there. This review article focused on the following such questions. Why are common allergens common? How does sensitization most often occur? How is IgE maintained over long time periods, even in the apparent absence of exposure? What distinguishes sensitization from clinical allergy? Can we stratify risk (i.e., sensitivity and severity)? What distinguishes the pathophysiology of non‐IgE-mediated allergy when so much of it seems to overlap?
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Pontone, Matteo, Mattia Giovannini, Simona Barni, Francesca Mori, Elisabetta Venturini, Luisa Galli, Claudia Valleriani, et al. "IgE-mediated Anisakis allergy in children." Allergologia et Immunopathologia 51, no. 1 (January 1, 2023): 98–109. http://dx.doi.org/10.15586/aei.v51i1.692.

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Anisakids are nematodes responsible for different clinical patterns in humans. The well-known human-infecting Anisakis species include members of the Anisakis simplex (AS) complex. Humans usually contract anisakiasis through ingestion of raw or undercooked seafood containing Anisakis larvae. Once Anisakis has been ingested, patients may develop disease driven directly by Anisakis larvae and/or by allergic reaction due to this nematode. The capability of inducing allergic reactions depends on the expression of specific antigens by nematodes and host factors. This study aims to resume actual knowledge about AS and Anisakiasis with regard to epidemiology, pathophysiology, clinical presentation, diagnosis, and treatment. Particular attention is paid to Anisakis allergens and their cross-reactivity on available diagnostic methods, and defining a diagnostic pathway for Anisakis allergy. Because only a few data are available in the literature about pediatric population, we focus on this group of patients specifically.
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Dissertations / Theses on the topic "IgE mediated allergy"

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Grönlund, Hans. "Diagnosis and treatment of IgE-mediated allergy : new approaches using recombinant allergens /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-373-6/.

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Palosuo, Kati. "IgE-mediated allergy to dietary gliadin studies on wheat-dependent, exercise-induced anaphylaxis and childhood wheat allergy." Helsinki : University of Helsinki, 2003. http://ethesis.helsinki.fi/julkaisut/laa/kliin/vk/palosuo/.

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Marknell, DeWitt Åsa. "Use of Recombinant Allergens for Component-Resolved Diagnostics (CRD) in IgE-Mediated Allergy." Doctoral thesis, Uppsala University, Department of Medical Biochemistry and Microbiology, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7813.

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Immunoglobulin E (IgE)-mediated allergy occurs when our immune system causes a reaction to otherwise harmless substances (allergens). Allergens are predominantly proteins present in biological materials such as pollens, mites, animal epithelia, moulds and foods.

In vitro tests for specific IgE antibodies usually employ an allergen source extract as an antibody capturing reagent. The proportion of allergenic molecules in these biochemically complex extracts may vary.

Recombinant allergens may be obtained in large quantities with biotechnological techniques. These proteins can be characterized biochemically and immunologically, resulting in tests with minimal batch-to-batch variation. This thesis describes different uses of recombinant allergens in component-resolved diagnostics (CRD).

In CRD, single allergenic proteins are used to establish a sensitization profile of the patient. Two timothy grass (Phleum pratense) pollen allergens, Phl p 11 and Phl p 4, were cloned and expressed as recombinant proteins. They were subsequently characterized and can, for example, be used in a panel for grass pollen CRD.

Single allergens may be useful as diagnostic markers for allergic sensitization. This phenomenon was studied using tropomyosin, a major allergen from the shrimp Penaeus aztecus (Pen a 1). The characteristics of the recombinant and natural proteins were compared. The recombinant tropomyosin was then extensively tested using specific competition for IgE binding against extracts of other crustacean species, house dust mite and cockroach.

In cases when an important allergen is missing or underrepresented in a natural extract, the corresponding recombinant allergen may be added to the extract as a spiking reagent. Previous studies have shown that latex extracts for diagnostic testing may lack the allergen Hev b 5. Recombinant Hev b 5 was expressed from a synthetic gene construct, incorporating several adaptations to enable efficient large scale production of the recombinant protein, to be used as a spiking reagent.

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Marknell, DeWitt Åsa. "Use of recombinant allergens for component-resolved diagnostics (CRD) in IgE-mediated allergy /." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7813.

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Yamani, Amnah. "Dysregulation of Vascular Endothelial Function Modulates Severity of IgE-mediated Anaphylactic Reactions." University of Cincinnati / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1490350649626552.

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Yahya, Mohd Norhakim. "Analysis of the IgE network : inhibition of CD23-mediated IgE upregulation and CD21/C3d interaction." Thesis, University of Oxford, 2011. http://ora.ox.ac.uk/objects/uuid:bc5ff165-2d2c-4e4f-a0e9-5651cacd2ddf.

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Allergic reactions are mainly mediated by the interactions between the IgE and its ligands, amongst them CD23 and CD21 in what is termed the IgE network. CD23 is involved in upregulating IgE expression by forming a trimolecular complex with CD21 and IgE on the B-cell surface, resulting in the specific activation of IgE-positive B cells. CD21 also interacts with C3d and is a bridge between the innate and the immune system. A crystal structure of the interaction has been solved (Szakonyi et al., 2001) but was controversial because it contradicted previous biochemical analyses. The aims of this thesis were to use various biophysical techniques to study the interactions between the molecules in the IgE network and its possible inhibition. Part 1: Characterisation of a phage display-derived peptide that inhibit IgE binding to CD23 A peptide was previously derived using phage display technology and tested for binding ability to CD23 using SPR and ITC. Subsequent NMR experiments were performed to identify the binding site, followed by characterization of its derivatives. Crystallisation of CD23 with the peptide and soaking with its truncated tripeptide, NWP, were also attempted. Part 2: Characterisation of CD23 and its interaction with its ligands X-ray crystallography was undertaken to solve the structure of derCD23 in complex with a phage display-derived peptide (Part1) followed by crystal soaking with a truncated tripeptide, NWP. However, a reproducible, high-resolution wild type derCD23 structure was determined at 1.9 Å. A comparison of the binding behaviour between the monomeric derCD23 and a trimeric CD23 construct was carried out in order to see the effect of oligomerisation upon IgE binding. Using the known interaction map as well as a crystal structure, the possible interacting residues between CD23 and IgE were examined. The characterisation of the CD23/CD21 interaction was continued from previous efforts in order to confirm that the binding epitope of CD23 for CD21 lies within the C-terminus of CD23. Characterisation of the interactions of CD23/IgE/FcεRI was performed to examine these multimolecular interactions and possible regulatory mechanisms in mast cell degranulation. It was shown that CD23 can form multimeric complexes with IgE-Fc that bind to FcεRI with higher apparent affinity than IgE-Fc alone, which may lead to increases in mast cell degranulation. It was also found that the IgE bound on FcεRI still binds to CD23 although with a lower binding capacity, presumably due allosteric changes. The binding of CD23 with a monoclonal antibody IDEC-152 was also characterised using SPR and NMR spectroscopy. It was proposed that IDEC-152 might interfere with the trimerisation site of CD23 thus reducing its affinity for IgE. A thermofluor assay was developed and optimised for potential screening of compounds that bind to derCD23 using a qPCR machine, which may be useful to screen compounds that bind to CD23 as part of future drug discovery project. Crystallisation of the derCD23/CD21 and IgE/triCD23/CD21 complexes was also attempted as part of ongoing crystallisation projects. Part 3: The interaction between C3d and CD21 The interaction between C3d and CD21 is believed to be a bridge between the innate and adaptive immune response, and is thought to be pivotal in the initiation of autoimmune disease. Following from previous studies on this interaction, further characterisations were performed using NMR and ITC to confirm the involved sites on CD21 (SCR1-2) in binding to C3d. Several potential salt bridges have been identified so far, allowing a high-resolution docked structure of the C3d/CD21 complex.
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Gray, Claudia Liesel. "The prevalence and patterns of IgE-mediated food allergy and sensitisation in South African children with atopic dermatitis." Doctoral thesis, University of Cape Town, 2014. http://hdl.handle.net/11427/12874.

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Background: The prevalence of food allergy in South Africa is unknown, but previously thought to be low, particularly in black South Africans. We hypothesised that food allergies would be low in Xhosa patients, even those at increased risk of food allergy such as children with atopic dermatitis (AD). This study aimed to determine the prevalence of, patterns and risk factors for, IgE-mediated food allergy in South African children with moderate to severe AD. It is the first food allergy prevalence study in South Africa to utilise controlled food challenges and component analysis, and is unique for its comparison of food allergy patterns between ethnic groups in the same geographical area. Methodology: This was a prospective, observational study in a paediatric university hospital in Cape Town. Children with moderate to severe AD, aged 6 months to 10 years, were randomly recruited from the dermatology clinic. They were assessed for sensitisation and allergy by questionnaire, skin prick tests (SPT), Immuno Solid Phase Allergen Chip (ISAC) test and incremental food challenges. Sensitised patients were also tested for specific IgE by ImmunoCAP test. Results: One hundred participants (59 black Africans and 41 of mixed race) were enrolled, median age 42 months. There were high overall rates of food sensitisation (66%) and food allergy (40%). Egg (25%) and peanut (24%) were the most common allergies. Black participants had comparable sensitisation (69% vs 61%) but lower allergy rates (34% vs 46%) than mixed race participants. This was especially evident for peanut allergy (15% vs 37%, p=0.01). Early onset AD (< 6 months), severe eczema, and young age < 2 years were significant risk factors for food allergy. The ISAC test was less sensitive than SPT and ImmunoCAP tests. Only 42% of cases of perceived food allergy were confirmed as true food allergy.
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Rolf, Sofia, and Johanna Svensson. "Den dolda funktionsnedsättningen – att leva med IgE-medierad födoämnesallergi : Erfarenheter och uppfattningar av att leva med en folksjukdom." Thesis, Högskolan i Halmstad, Akademin för hälsa och välfärd, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:hh:diva-36067.

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Födoämnesallergi är en dold sjukdom som inte blir synlig förrän individen utvecklar en allergisk reaktion. Därifrån kan det snabbt handla om liv och död. Kroppens fysiska reaktion är väl utforskad och trots det, är hur sjukdomen påverkar patientens livssituation relativt outforskat. Omvårdnadslitteratur inom risker och åtgärder vid allergi nämner ytterst lite om födoämnesallergi. Studiens syfte var att beskriva patienters erfarenheter och uppfattningar av att leva med IgE-medierad födoämnesallergi. Genom en litteraturstudie framstod fyra teman: Rädsla och förlust av säkerhet, Att hantera vardagen, Betydelsen av information vad gäller märkning av livsmedel och Vikten av stöd från omgivningen. Upplevelsen av att inte ha kontroll, en ständig risk för oanade reaktioner, att inte alltid ha förståelse och behöva försvara sina behov, gav en negativ emotionell respons. Påverkan på livssituationen jämfört mellan lätt födoämnesallergi och svår födoämnesallergi var markant. Vägen tillbaka till en fungerande vardag bestod av strategier, förståelse, samt försoning. Ändå fanns hinder i omgivningen kvar. Kunskapen som genererades från studien är viktig för att sjuksköterskan ska kunna vårda en födoämnesallergisk patient utan att hindra läkande och känsla av säkerhet. Samhället behöver möta patientgruppens behov genom anpassning för att livskvalitet ska kunna uppnås.
Food allergy is a hidden disability that does not become visible until the individual develops an allergic reaction. From there on it can quickly become a matter of life or death. The physical response has been well researched and yet how the disease affects the patient’s life situation is relatively unexploded. Nursing literature regarding allergy risks and interventions mention little about food allergy. The aim of the study was therefore to highlight patients’ experiences and perceptions of living with IgE-mediated food allergy. Through a literature study four themes emerged: Fear and loss of security, Managing everyday life, Importance of information regarding labeling of food and The importance of support from others. The experience of not having control, a constant risk of unsuspected reactions, not always having an understanding from others and having to defend their needs gave a negative emotional response. The way back to an ordinary living consisted of strategies, understanding and reconciliation, yet there were still obstacles left in the environment. The knowledge of this study can be used in caring for a food allergic patient without jeopardizing healing process and feeling of security. The society needs to meet the patient group’s needs through adaptation, to achieve quality of life.
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Larsson, Anna-Karin. "Early life cytokines, viral infections and IgE-mediated allergic disease." Doctoral thesis, Stockholm : Wenner-Gren Institute for Experimental Biology, Stockholm university, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-1224.

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Powe, Desmond George. "Do some subjects with idiopathic rhinitis have an allergic inflammatory disease mediated by IgE, in their nasal mucosa?" Thesis, University of Nottingham, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.408604.

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Books on the topic "IgE mediated allergy"

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Puntis, John. Food allergy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0019.

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Food allergy is an immune response to food that can be classified as immunoglobulin (Ig)-E and non-IgE mediated. Milk, egg, peanut, tree nuts, and fish are among the most prevalent causes of food allergy. Mild reactions can include itchy rash, watering eyes, and nasal congestion while a severe reaction results in anaphylaxis. A detailed clinical history is essential when making a diagnosis, and skin prick testing and quantitative measurement of food-specific IgE antibodies can be helpful. Cow milk protein allergy causes a plethora of symptoms and frequently resolves spontaneously over the first 2 years of life; diagnosis is based mainly on clinical history. Food challenges have a pivotal role in the diagnosis of food allergy. Introduction of ‘allergic’ foods at 3–6 months alongside continuing breastfeeding may prevent allergy.
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Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Eosinophilic disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0045.

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Eosinophilic proctocolitis of infancy (dietary protein-induced proctocolitis of infancy) 320Eosinophilic enterocolitis of infancy (dietary protein-induced enterocolitis of infancy) 321Eosinophilic gastroenteropathies in the older child 322Eosinophilic oesophagitis 323Features suggestive of food allergy as a cause of gastrointestinal disease 323This chapter discusses the wide spectrum of eosinophilic (allergic) disorders of the gut. They are generally not IgE mediated. Presentation is with the full spectrum of gastrointestinal symptoms and signs. Outside infancy the disorders may only become apparent on investigation of chronic gut symptoms by endoscopy to exclude oesophagitis, peptic ulceration, enteropathy, or colitis. Important disorders to consider are: ...
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Misbah, Siraj. Suspected anaphylaxis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0075.

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A type I IgE-mediated systemic allergic reaction is characterized by a constellation of symptoms which are due to widespread histamine release and which comprise acute-onset urticaria, angioedema, bronchospasm, and hypotension. While a mild reaction may be limited to localized urticaria and/or angioedema, a full-blown allergic reaction associated with systemic features is best described as anaphylaxis. The term ‘anaphylactoid’, previously used to denote non-IgE-mediated systemic allergic reactions, is no longer recommended for use.
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Book chapters on the topic "IgE mediated allergy"

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Urbanek, R. "IgE-Mediated Allergies." In Food Allergy in Infancy and Childhood, 133–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74357-3_13.

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Sicherer, Scott H. "IgE- and Non-IgE-Mediated Food Allergy." In Eosinophilic Esophagitis, 219–38. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-515-6_16.

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Bischoff, Stephan C., and Gernot Sellge. "The Immunological Basis of IgE-Mediated Reactions." In Food Allergy, 16–30. Chichester, UK: John Wiley & Sons Ltd, 2014. http://dx.doi.org/10.1002/9781118744185.ch2.

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Uzzaman, Ashraf, and Hirsh D. Komarow. "The Immunological Basis of Non-IgE-Mediated Reactions." In Food Allergy, 31–46. Chichester, UK: John Wiley & Sons Ltd, 2014. http://dx.doi.org/10.1002/9781118744185.ch3.

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Fleischer, David M., and Donald Y. M. Leung. "Cutaneous Reactions: Atopic Dermatitis and Other IgE- and Non-IgE-Mediated Skin Reactions." In Food Allergy, 144–57. Chichester, UK: John Wiley & Sons Ltd, 2014. http://dx.doi.org/10.1002/9781118744185.ch11.

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Weidinger, Stephan, J. Ring, and F. M. Köhn. "IgE-Mediated Allergy against Human Seminal Plasma." In Chemical Immunology and Allergy, 2005, 128–38. Basel: KARGER, 2005. http://dx.doi.org/10.1159/000087830.

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Fuchs, T., R. Wahl, and J. Geier. "Immediate-Type Reactions to Rubber Products: IgE Mediated?" In New Trends in Allergy III, 431–36. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-46717-2_57.

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Agostoni, Carlo, and Verduci Elvira. "Growth of Infants with IgE-Mediated Cow’s Milk Allergy." In Handbook of Growth and Growth Monitoring in Health and Disease, 1911–20. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-1795-9_115.

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Khan, Mahejibin, and Nidhi Sori. "Diet-Gut Microbiota-Brain Axis and IgE-Mediated Food Allergy." In Microbiome-Gut-Brain Axis, 153–68. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-1626-6_6.

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Campi, Paolo, Mariangela Manfredi, and Maurizio Severino. "IgE-Mediated Allergy to Pyrazolones, Quinolones and Other Non- &Bg;; -Lactam Antibiotics." In Drug Hypersensitivity, 216–32. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000104202.

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Conference papers on the topic "IgE mediated allergy"

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Shabana, H., and M. Askar. "Non-Ige-Mediated Food Allergy May be the Cause of Chronic Bloody Diarrhea." In ESGE Days 2021. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1724665.

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Irina-Veronica, Costache, and Plesca Doina Anca. "P26 Case report: severe ige-mediated cow’s milk protein allergy in a salmonella carrier." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.114.

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Weinbrand-Goichberg, Jenny, Shira Benor, Menahem Rottem, Nitzan Shacham, Avigdor Mandelberg, Arie Levine, Koby Sade, Shmuel Kivity, and Ilan Dalal. "P9 Long term outcomes following baked milk – containing diet for ige-mediated milk allergy." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.97.

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Fumagalli, Mara, Laura Gianolio, Vania Giacomet, and Gian Vincenzo Zuccotti. "261 Infant hematemesis: a challenging diagnosis, postnatal CMV infection or non IgE-mediated cow’s milk protein allergy? A two case report comparison." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.261.

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Kishida, Tsunao, Yayoi Hiromura, Takemitsu Hama, Jiro Imanishi, Yasuo Hisa, and Osam Mazda. "Interleukin-21 as an Effective Suppressant for IgE-mediated Allergic Hypersensitivity Reactions." In 2007 International Symposium on Micro-NanoMechatronics and Human Science. IEEE, 2007. http://dx.doi.org/10.1109/mhs.2007.4420871.

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Fassakhov, Rustem, and Sergei Boichuk. "Allergen activates eosinophils of atopic bronchial asthma patients via IgE-mediated mechanism." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.2050.

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Milgrom, H., J. Fink, A. Fowler-Taylor, C. Fernandez Vidaurre, M. Blogg, and H. Fox. "Safety of Omalizumab in Children with Inadequately Controlled Moderate–Severe Allergic (IgE-Mediated) Asthma." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2809.

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Gouder, Caroline, Rachelle Asciak, and Stephen Montefort. "The 'real-life' long-term efficacy of omalizumab in severe persistent IgE-mediated allergic asthma in Malta." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa4115.

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Milgrom, H., RL Wasserman, A. Fowler-Taylor, C. Fernandez Vidaurre, M. Blogg, and H. Fox. "Add-On Omalizumab Significantly Reduces Exacerbation Rates in Children with Inadequately Controlled Moderate–Severe Allergic (IgE-Mediated) Asthma." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2767.

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Simons, Anneliese, Karen Regan, Abid Aziz, and Dinesh Saralaya. "Real-life Effectiveness Of Omalizumab In Patients With Severe Persistent Allergic (IgE-Mediated) Asthma At A Single UK Hospital." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1336.

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You might also be interested in the extended bibliographies on the topic 'IgE mediated allergy' for particular source types:
Journal articles Dissertations / Theses
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