Dissertations / Theses on the topic 'Identification of novel genes'
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Travis, Kristina. "Identification of Novel Developmental Genes in Streptomyces Coelicolor." Otterbein University Distinction Theses / OhioLINK, 2005. http://rave.ohiolink.edu/etdc/view?acc_num=otbndist16204640123321.
Full textKotian, Shweta. "Identification of Novel Genes in BRCA1-Regulated Pathways." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1366341897.
Full textArchacki, Stephen R. "MOLECULAR IDENTIFICATION OF NOVEL GENES ASSOCIATED WITH ATHEROSCLEROSIS." Cleveland State University / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=csu1310652996.
Full textHu, Xiao Ping. "Identification and characterisation of novel cellulolytic genes using metagenomics." Thesis, University of the Western Cape, 2010. http://etd.uwc.ac.za/index.php?module=etd&action=viewtitle&id=gen8Srv25Nme4_9293_1308049102.
Full textMetagenomics has been successfully used to discover novel enzymes from uncultured microorganisms in the environment. In this study, metagenomic DNA from a Malawian hot spring soil sample was used to construct a fosmid library. This metagenomic library comprised of more than 10000 clones with an average insert size of 30 kb, representing more than 3.0 x 108 bp of metagenomic DNA (equivalent to approximately 100 bacterial genomes). The library was screened for cellulase activity using a Congo red plate assay to detect zones of carboxymethylcellulose hydrolysis. This yielded 15 positive fosmid clones, of which five were further characterised for activity and thermostability using the 3, 5-dinitrosalicylic assay. Two of the five fosmids (XP008C2 and XP026G5) were selected for DNA pyrosequencing. The full sequence of the XP008C2 (29800bp) fosmid insert is presented in this study and genes thereon were chosen for further study.
Bennetts, Jennifer. "The identification and characterisation of novel genes in development /." [St. Lucia, Qld.], 2006. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19375.pdf.
Full textLooser, Jens. "Identification of two novel CVC domain-containing homeobox genes." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1995. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ45845.pdf.
Full textHaviland, Rachel. "Identification of Novel STAT3 Target Genes Associated with Oncogenesis." Scholar Commons, 2011. http://scholarcommons.usf.edu/etd/3729.
Full textChu, Youngmin. "Identification of Novel Genes Involved in Female Mating Choice." Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1364923180.
Full textVyas, Aditi. "Identification of Novel Stat92E Target Genes in Drosophila Hematopoiesis." Ohio University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1450868635.
Full textGregorio-King, Claudia C., and mikewood@deakin edu au. "The Identification of novel genes differentially expressed in Haemopoietic progenitor cells." Deakin University. School of Health Sciences, 2001. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051111.113037.
Full textSong, Gwon Hwa. "Identification of novel implantation-related genes in the ovine uterus." Texas A&M University, 2003. http://hdl.handle.net/1969.1/5782.
Full textTse, Ka-yu. "Identification of novel methylated genes in patients with endometrial cancers /." View the Table of Contents & Abstract, 2007. http://sunzi.lib.hku.hk/hkuto/record/B38284893.
Full textTse, Ka-yu, and 謝嘉瑜. "Identification of novel methylated genes in patients with endometrial cancers." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B45011473.
Full textMcGregor, Nathaniel Wade. "The identification of novel susceptibility genes involved in anxiety disorders." Thesis, Stellenbosch : Stellenbosch University, 2014. http://hdl.handle.net/10019.1/95859.
Full textENGLISH ABSTRACT: The etiology of anxiety disorders remains incompletely understood. Clear evidence for a genetic component has been proposed; however, there is also an increasing focus on environmental factors and the interaction between these and the genetic components that may mediate (anxiety) disorder pathogenesis. No single gene or genetic component has been explicitly identified as being involved in the development of anxiety disorders. This is most likely due to a number of reasons, which include, for example, the heterogeneity of anxiety disorders, the contribution of environmental factors and methodological limitations (e.g. small sample size) of research studies. Until now, genetic association studies usually focused on one particular psychiatric disorder at a time. However, with the difficulty in identifying susceptibility genes and/or loci in heterogeneous disorders like obsessive-compulsive disorder and other conditions in the anxiety spectrum, it is perhaps timely to consider multivariate genetics and epidemiological studies in a number of disorders sharing a core characteristic – such as anxiety. In addition to genetic underpinnings, a number of environmental variables have also been identified as risk factors for pathological anxiety, including adverse life events like childhood physical and sexual abuse. The hypothesis for this project is that a pre-existing genetic vulnerability (or genetic risk) interacts with the impact of adverse life events to result in the development of one or more anxiety disorder(s). Considering phenotypic overlap amongst the anxiety disorders, it is likely that diverse networks of genes and/ or interacting pathways are responsible for the phenotypic manifestations observed. Sprague Dawley rats exhibiting behaviours indicative of anxiety in the context of environmental stressors (maternal separation and restraint stress) were used as model for the identification of novel susceptibility genes for anxiety disorders in humans. The striatum has previously been implicated as a candidate in the brain architecture of anxiety pathogenicity, and is also a site exhibiting a high degree of synaptic plasticity. The synaptic plasticity pathway was investigated using the dorsal striatum of the rat brain and several genes were identified to be aberrantly expressed in “anxious” rats relative to controls (Mmp9, Bdnf, Ntf4, Egr2, Egr4, Grm2 and Arc). In humans, it was found that the severity of early adversity was significantly and positively associated with the presence of an anxiety disorder in adulthood. When the human homologues of the susceptibility candidate genes that were identified using the animal model were screened in a human cohort of patients with obsessive-compulsive disorder (OCD), panic disorder (PD) or social anxiety disorder (SAD) (relative to controls), five single nucleotide polymorphisms (SNPs) were found to be significantly associated with these conditions. Four of these SNPs were also found to significantly interact with the severity of childhood trauma. Haplotype analysis of variants within the identified susceptibility candidates revealed novel haplotype associations, four of which are located in the MMP9 gene. Notably, this the first study to link these particular mutations in the MMP9 gene with anxiety disorders and this finding is consistent with previous work suggesting that MMP9 is involved in conditions like cardiovascular disease and cancer which have been associated with increased prevalence of anxiety disorders. In conclusion, this project yielded important findings pertaining to the etiology of anxiety disorders. The use of a combined anxiety disorders cohort (OCD, PD and SAD) may suggest that the associations found here may hold true for anxiety disorders in general and not only for a particular clinically delineated condition. Childhood trauma was confirmed as an increased susceptibility risk for anxiety disorders. Also, this research contributed several novel susceptibility genes (MMP9, EGR2, EGR4, NTF4, and ARC), five significant SNP associations, four significant SNP-environment interactions and five haplotype associations (within MMP9 and BDNF) as candidates for anxiety pathogenicity. The identified polymorphisms and haplotypes were demonstrated to be associated with susceptibility to anxiety disorders in a gene-environment correlation and gene-environment interaction.
AFRIKAANSE OPSOMMING: Die oorsake van angssteurings word steeds nie volledig verstaan nie. Daar is duidelike bewyse vir 'n genetiese komponent, maar daar is ook toenemende fokus op omgewingsfaktore en die interaksie tussen hierdie omgewingsfaktore en genetiese komponente by angssteurings. Geen enkele geen of genetiese komponent is al geïdentifiseer as diè wat betrokke is by die ontwikkeling van angssteurings nie. Dit is waarskynlik weens 'n aantal redes, wat byvoorbeeld, die heterogeneïteit van angssteurings, die bydrae van omgewingsfaktore en metodologiese beperkings (bv. klein steekproef) van die navorsingstudies, insluit. Verder het genetiese assosiasiestudies tot nou toe gewoonlik net op een spesifieke psigiatriese versteuring op 'n slag gefokus. Maar, gegewe die uitdaging om vatbaarheidsgene en / of loci in heterogene steurings soos obsessief – kompulsiewe steuring (OKV) en ander toestande op die angsspektrum te identifiseer, is dit tyd om genetiese en kliniese studies in ‘n aantal steurings - met ‘n oorvleuende kern-element soos angs -, gesamentlik te oorweeg. Bykomend tot die genetiese boustene, is ‘n aantal omgewingsveranderlikes soos traumatiese lewenservarings tydens die kinderjare as risikofaktore vir patologiese angs geidentifiseer. Die hipotese vir hierdie projek is dat daar 'n interaksie tussen genetiese kwesbaarheid (of genetiese risiko) en traumatiese lewensevarings is en dat dit tot die ontwikkeling van 'n / veelvoudige angssteuring(s) kan lei. Inaggenome die fenotipiese oorvleueling tussen die angssteurings, is dit waarskynlik dat diverse netwerke van gene en / of interaktiewe geen-paaie vir die manifestasie van hierdie toestande verantwoordelik is. Sprague Dawley-rotte met gedragswyses aanduidend van angs, in die konteks van omgewingstressore (d.i. skeiding van die ma-rot en bedwang-stres [restraint stress]), is as model gebruik vir die identifisering van nuwe vatbaarheidsgene vir angssteurings in mense. Die striatum is voorheen as ‘n kandidaat in die brein-argitektuur van patologiese angs voorgehou, en is ook ‘n plek met ‘n hoë mate van sinaptiese plastisiteit. Die sinaptiese plastisiteit is ondersoek deur te fokus op die dorsale striatum van die rotbrein en daar is verskeie gene gevind wat anders is in “angstige” rotte in vergelyking met kontroles (Mmp9, Bdnf, Ntf4, Egr2, Egr4, Grm2 en Arc). In mense is daar gevind dat die ernstigheidsgraad van vroeë trauma beduidend en positief met die teenwoordigheid van ‘n angssteuring tydens volwassenheid verband hou. Toe die menslike ekwivalente van die vatbaarheidsgene wat met die dieremodel geïdentifiseer is in ‘n mens-kohort met obsessief-kompulsiewe steuring (OKS), panieksteuring (PS) en sosiale angssteuring (SAS) ondersoek is, is gevind dat daar 5 enkele nukleotied polimorfismes (ENPs) is wat met die toestande verband hou. Daar is ook gevind dat vier van hierdie ENPs beduidend verband hou met die ernstigheidsgraad van trauma tydens die kinderjare. Haplotipe analise van variante binne die geïdentifiseerde vatbaarheidsgene het op nuwe haplotipe assosiasies – waarvan 4 op die MMP9-geen geleë is – gedui. Hierdie is dus die eerste studie wat gevind het dat dié spesifieke mutasies van die MMP9-geen met angssteurings verband hou. Hierdie bevinding strook met vorige werk wat daarop dui dat die MMP9-geen by toestande soos kardiovaskulêre siekte en kanker wat ook met verhoogde voorkoms van angssteurings verband hou, betrokke is. Ter afsluiting kan ons sê dat hierdie projek belangrike bevindinge oor die oorsake van angssteurings gemaak het. Die gebruik van ‘n gekombineerde angssteurings-kohort (OKS. PS en SAS) kan moontlik suggereer dat die assosiasies wat ons hier gevind het, waar is vir alle angssteurings en nie net vir ‘n spesifieke afgebakende toestand nie. Traumatiese ervarings tydens die kinderjare is ook bevestig as ‘n risiko vir die ontwikkeling van angssteurings. Hierdie navorsing het ook verskeie nuwe vatbaarheidsgene (MMP9, EGR2, EGR4, NTF4, en ARC), 5 beduidende ENP assosiasies, 4 beduidende ENP-omgewings-interaksies en 5 haplotipe assosiasies (by MMP9 en BDNF) geïdentifiseer as moontlike kandidate wat ‘n rol speel by die ontstaan van patologiese angs. Daar is ook gevind dat die geïdentifiseerde polimorfismes en haplotipes met vatbaarheid vir angssteurings in ‘n geen-omgewing- korrelasie en geen-omgewing- interaksie verband hou. Stellenbosch University http://scholar.sun.ac.za
Coler, Rhea Nadine. "Identification and characterization of novel antigen genes of Mycobacterium tuberculosis /." Thesis, Connect to this title online; UW restricted, 1998. http://hdl.handle.net/1773/9281.
Full textAlmutairi, Mikhlid Hammad. "Identification of novel human cancer-testis-antigen genes in cancers." Thesis, Bangor University, 2014. https://research.bangor.ac.uk/portal/en/theses/identification-of-novel-human-cancertestisantigen-genes-in-cancers(015fda5b-5bd3-42c1-a610-811f0acde19a).html.
Full textCabral, Vitor Hugo. "Identification of novel Candida albicans genes involved in biofilm formation." Paris 7, 2012. http://www.theses.fr/2012PA077224.
Full textCandida albicans is a polymorphic fungus that exists as either a commensal or an opportunistic pathogen, causing mucosal infections and even life-threatening systemic infections. C. Albicans is able to form biofilms (yeast, pseudohyphal and hyphal cells embedded in an extracellular polymeric matrix) that grow on biotic or abiotic surfaces and display specific phenotypic traits (as a high résistance to drugs). Biofilm architecture depends on the yeast-to-hypha transition and surface proteins that mediate hypha-hypha interactions and confer cohesiveness. The main aim of my thesis project will be to identify and characterize additional regulators of biofilm formation that may become targets for preventing or eliminating biofilms. A collection of C albicans over-expression strains, was established and screened using medium throughput biofilm models from which genes whose over-expression alters biofilm formation were identified. Notably, a subset of strains, each overexpressing a gène encoding for glycosylphosphatidylinositol (GPI)-anchored proteins (PGAs), showed increased occupancy of a biofilm formed by a pool of ail thé overexpression strains. We have focused our study on PGA genes that have an effect on biofilm formation and shown that, despite the common higher strain occupancy on a multi-strain biofilm as a result of their overexpression, the mechanisms by which they reach this end differ. This study acts as a proof-of-principle that the ORFeome collection is a useful tool to study biofilm formation, providing insights into the contribution of individual genes as well as complete or partial gene families
Guo, Ling. "Identification of novel SLE susceptibility genes by microarray analysis and candidate gene association study." Oklahoma City : [s.n.], 2008.
Find full textSanhueza, Cubillos Mario Andrés. "Identification of novel genes interacting with DVAP, the causative gene of ALS8 in humans." Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/21080.
Full textGanea, Karin. "Identification and characterisation of novel antidepressant-responsive genes in mouse brain." Diss., lmu, 2009. http://nbn-resolving.de/urn:nbn:de:bvb:19-99940.
Full textSadler, Amanda J. "Identification of novel genes associated with endocrine resistance in breast cancer." Thesis, University of Reading, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.485503.
Full textHorpaopan, Sukanya [Verfasser]. "Identification of Novel Causative Genes for Colorectal Adenomatous Polyposis / Sukanya Horpaopan." Bonn : Universitäts- und Landesbibliothek Bonn, 2015. http://d-nb.info/1077289529/34.
Full textLefebvre, Valerie. "Identification of Novel Parkinson’s Disease Genes Involved in Parkin Mediated Mitophagy." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/30222.
Full textLabermaier, Christiana. "Identification of novel candidate genes involved in individual antidepressant treatment response." Diss., Ludwig-Maximilians-Universität München, 2015. http://nbn-resolving.de/urn:nbn:de:bvb:19-179575.
Full textGroet, Jurgen. "Physical mapping and identification of novel genes in human chromosome 21q11." Thesis, University College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312003.
Full textDavies, Andrea. "Identification of novel p53-dependent genes in etoposide-treated gastrointestinal cells." Thesis, University of Liverpool, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.422990.
Full textSmith, Shavannor Michelle. "Identification and characterization of Rp1 genes with novel phenotypes in maize /." Search for this dissertation online, 2004. http://wwwlib.umi.com/cr/ksu/main.
Full textEve, A. M. J. "Identification and functional analyses of novel genes involved in haemovascular development." Thesis, University College London (University of London), 2016. http://discovery.ucl.ac.uk/1473223/.
Full textLornage, Xavière. "Identification and functional characterization of novel genes implicated in congenital myopathies." Thesis, Strasbourg, 2019. http://www.theses.fr/2019STRAJ067.
Full textCongenital myopathies are severe genetic muscle diseases characterized by a disabling early-onset muscle weakness. In order to identify new genetic causes, we sequenced the exomes of molecularly undiagnosed congenital myopathy patients, and their analysis highlighted two novel myopathy genes. MYPN and ACTN2 encode two structural sarcomeric proteins called myopalladin and alphaactinin-2. To evaluate the impact of the mutations on the protein function and on muscle physiology, molecular and functional analyses were performed in cell and animal models. The MYPN mutations resulted in loss of myopalladin expression, and in mouse muscles, mutated alpha-actinin-2 led to muscle weakness and structural defects similar to those observed in the patient muscles. These results have a direct impact on the disease management of the patients and on genetic counselling, provide a better understanding of the signaling pathways required for muscle physiology, and highlight novel therapeutic targets
Warner, Adam Dennis. "Identification of novel genes affecting body wall muscle in Caenorhabditis elegans." Thesis, University of British Columbia, 2007. http://hdl.handle.net/2429/31429.
Full textMedicine, Faculty of
Medical Genetics, Department of
Graduate
Weraarpachai, Woranontee. "Identification and characterization of novel genes involved in cytochrome c oxidase deficiencies." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=107626.
Full textDans les mitochondries, l'ATP est généré par la phosphorylation oxydative (PHOSOX), un processus qui nécessite cinq complexes enzymatiques multimériques. Le transport des électrons le long des quatre premiers complexes enzymatiques (complexes I-IV) libère l'énergie qui est stockée sous la forme d'un gradient de protons à travers la membrane interne mitochondriale et est ensuite utilisée par l'ATP synthétase (complexe V) pour produire de l'ATP. Le complexe IV ou cytochrome C oxydase (COX) est l'enzyme terminale de la chaîne respiratoire mitochondriale, catalysant l'oxydation du cytochrome c par l'oxygène moléculaire. Il contient 13 sous-unités structurelles chez les mammifères, dont 3 sont codées par l'ADN mitochondriale. Les déficiences en cytochrome C oxydase peuvent être causées par des mutations dans l'ADN mitochondriale ou l'ADN nucléaire. Les carences en COX peuvent être liées à des mutations dans les sous-unités structurelles ou à des mutations dans des facteurs nécessaires à l'assemblage du complexe enzymatique. Dans cette thèse, deux nouveaux gènes mutés ont été identifiés et caractérisés dans deux patients présentant un déficit en COX. Premièrement, nous avons identifié un défaut spécifique dans la synthèse de la sous-unité COX 1 de l'ADN mitochondriale (COX I) dans un pedigree présentant une apparition tardive du syndrome de Leigh et une carence en COX. Nous avons cartographié le défaut génétique au chromosome 17q par la technique de transfert de chromosomes à médiation microcellulaire. Nous avons, par la suite, identifié une mutation homozygote, une insertion d'une base causant l'apparition prématurée d'un codon stop qui entraîne l'arrêt de la synthèse de la protéine CCDC44, renommé TACO1 pour activateur de la traduction de la COX I. TACO1 est membre d'une famille de protéines contenant un domaine conservé à fonction inconnue, nommé DUF28, qui se localise à la matrice mitochondriale. L'expression de l'ADN complémentaire de type sauvage de TACO1 compense le défaut de traduction de COX I. TACO1 est le premier activateur spécifique de la traduction mitochondriale à être identifié chez les mammifères. Il a été observé que l'absence (knock-down) du gène codant l'orthologue de TACO1 chez la levure, le YGR021w, ne perturbait pas la compétence des voies respiratoires, la traduction mitochondriale, ni l'activité de COX. Ceci suggère que TACO1 a évolué et a acquérit une nouvelle fonction dans la traduction mitochondriale chez les mammifères. Deuxièmement, nous avons étudié une famille dans laquelle le sujet présentait une acidose lactique congénitale et dysmorphie associée à un défaut d'assemblage et une diminution de l'activité enzymatique de la COX due à un défaut spécifique dans la traduction de COX I. En utilisant une combinaison de techniques dont le transfert de chromosomes à médiation microcellulaire, la cartographie d'homozygotie et le profilage de transcription, nous avons cartographié le gène défectueux sur le chromosome 12. Nous avons identifié une mutation faux sens à l'état homozygote provoquant un changement d'acide aminé de méthionine en isoleucine dans le gène C12orf62, un gène qui semble restreint à la lignée des vertébrés. L'expression de l'ADN complémentaire de type sauvage de C12orf62 a restauré la synthèse de COX I et le défaut d'assemblage de la COX. C12orf62 est une très petite protéine transmembranaire (6 kDa), non caractérisée, qui se localise aux mitochondries. Les sous-unités COX I, II et IV co-immunoprécipitent avec un épitope marqué de la protéine C12orf62. Les analyses de bleu d'électrophorèse sur gel de polyacrylamide natif (BN-PAGE) en deux dimensions pour les sous-unités nouvellement synthétisées de la COX mitochondriale ont démontré que la COX assemblée est altérée et que le complexe enzymatique naissant est instable dans les fibroblastes du patient atteint. Nous concluons que C12orf62 est nécessaire pour coordonner les étapes précoces de l'assemblage de la COX et de la synthèse de COX I.
Chikkaballi, Anne Gowda Deepak. "Identification of novel Salmonella virulence genes involved in invasion and intracellular survival /." Berlin : Mbv, 2009. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=018885733&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.
Full textMaibaum, Michael Anthony. "The identification of novel susceptibility genes in the lupus prone BXSB mouse." Thesis, Imperial College London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.271233.
Full textMcDade, Donna Marie. "Identification of novel target genes for the plasticity-related transcription factor Zif268." Thesis, University of Glasgow, 2007. http://theses.gla.ac.uk/28/.
Full textGilmore, Paula. "The identification of novel genes involved in chemotherapy resistance in ovarian cancer." Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361248.
Full textCoffer, Paul James. "The identification, cloning and characterisation of novel mammalian protein-serine kinase genes." Thesis, Open University, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.293298.
Full textConnolly, David John. "Identification of novel class 1 genes in the mouse major histocompatibility complex." Thesis, Open University, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.304886.
Full textChikkaballi, Anne Gowda Deepak. "Identification of novel Salmonella virulence genes involved in invasion and intracellular survival." Berlin mbv, 2008. http://d-nb.info/998075302/04.
Full textFarahani, Poupak. "Identification of novel candidate obesity genes in hepatic lipase knockout BSB mice /." For electronic version search Digital dissertations database. Restricted to UC campuses. Access is free to UC campus dissertations, 2003. http://uclibs.org/PID/11984.
Full textWillis, Laura B. (Laura Bethg) 1967. "Identification and characterization of novel Rhizobium meliloti genes involved in carbon metabolism." Thesis, Massachusetts Institute of Technology, 1998. http://hdl.handle.net/1721.1/50353.
Full textWatson, Nicola Sophia Alexandra. "Identification of novel genes involved in Paget's disease by Differential Display PCR." Thesis, University of Aberdeen, 1999. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU535579.
Full textDavies, Benjamin. "Serine proteases expressed in the rodent hippocampus : identification of two novel genes." Thesis, University of Edinburgh, 1997. http://hdl.handle.net/1842/13583.
Full textBabbs, Christian. "Identification and characterisation of novel genes involved in mouse early inner ear development." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.393425.
Full textBesi, Maria. "Identification of novel pathogenicity-related genes in the rice blast fungus, Magnaporthe oryzae." Thesis, University of East Anglia, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539361.
Full textSong, Yang, and 宋揚. "Identification of the novel genes during endochondral ossification in the mandibular condylar cartilage." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43085568.
Full textEthirajan, Lakshmi Priya. "Identification and analysis of novel de-regulated genes/proteins in human carotid atherosclerosis." Thesis, Manchester Metropolitan University, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.440238.
Full textShaw, Lesley E. "The identification of novel genes associated with endocrine resistance in human breast cancer." Thesis, University of Reading, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.408985.
Full textSong, Yang. "Identification of the novel genes during endochondral ossification in the mandibular condylar cartilage." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B43085568.
Full textJoshi, Shreyas. "IDENTIFICATION OF NOVEL SLEEP RELATED GENES FROM LARGE SCALE PHENOTYPING EXPERIMENTS IN MICE." UKnowledge, 2017. http://uknowledge.uky.edu/biology_etds/42.
Full textWong, Frances. "Analysis of genes involved in gonadal development : identification of novel sex determination candidates." Thesis, University of Edinburgh, 2005. http://hdl.handle.net/1842/27706.
Full text