Relevant bibliographies by topics / Hypoglycemia

Academic literature on the topic 'Hypoglycemia'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 July 2024

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Journal articles on the topic "Hypoglycemia"

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Abrahamsson, Niclas, Britt Edén Engström, Magnus Sundbom, and F. Anders Karlsson. "Hypoglycemia in everyday life after gastric bypass and duodenal switch." European Journal of Endocrinology 173, no. 1 (July 2015): 91–100. http://dx.doi.org/10.1530/eje-14-0821.

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DesignGastric bypass (GBP) and duodenal switch (DS) in morbid obesity are accompanied by marked metabolic improvements, particularly in glucose control. In recent years, episodes of severe late postprandial hypoglycemia have been increasingly described in GBP patients; data in DS patients are scarce. We recruited three groups of subjects; 15 GBP, 15 DS, and 15 non-operated overweight controls to examine to what extent hypoglycemia occurs in daily life.MethodsContinuous glucose monitoring (CGM) was used during 3 days of normal activity. The glycemic variability was measured by mean amplitude of glycemic excursion and continuous overall net glycemic action. Fasting blood samples were drawn, and the patients kept a food and symptom log throughout the study.ResultsThe GBP group displayed highly variable CGM curves, and 2.9% of their time was spent in hypoglycemia (<3.3 mmol/l, or 60 mg/dl). The DS group had twice as much time in hypoglycemia (5.9%) and displayed CGM curves with little variation as well as lower HbA1c levels (29.3 vs 35.9 mmol/mol,P<0.05). Out of a total of 72 hypoglycemic episodes registered over the 3-day period, 70 (97%) occurred in the postprandial state and only about one-fifth of the hypoglycemic episodes in the GBP and DS groups were accompanied by symptoms. No hypoglycemias were seen in controls during the 3-day period.ConclusionBoth types of bariatric surgery induce marked, but different, changes in glucose balance accompanied by frequent, but mainly unnoticed, hypoglycemic episodes. The impact and mechanism of hypoglycemic unawareness after weight-reduction surgery deserves to be clarified.
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Büyükkaya Besen, Dilek, Hamdiye Arda Sürücü, and Cansu Koşar. "Self-reported frequency, severity of, and awareness of hypoglycemia in type 2 diabetes patients in Turkey." PeerJ 4 (December 13, 2016): e2700. http://dx.doi.org/10.7717/peerj.2700.

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ObjectivesHypoglycemia is a common side effect of insulin therapy in type 1 and type 2 diabetes. Limited data exist on the frequency of hypoglycemic events in type 2 diabetic patients in Turkey. Our study investigated self-reported hypoglycemic events and awareness of hypoglycemia in Turkish patients with type 2 diabetes.MethodsPeople with type 2 diabetes older than 18 years of age were recruited from the two university hospital diabetes clinics. The frequency and severity of hypoglycemia and awareness of hypoglycemia during the preceding year were determinated using questionnaires by the face-to-face interview method.ResultsIn this study of 187 patients with type 2 diabetes, 83.4% had impaired awareness of their hypoglycemia, and 62% reported that they had missed some of the symptoms of hypoglycemia. Of the patients reporting hypoglycemic symptoms and severity level, 84.1% experienced mild hypoglycemia, 60% moderate, and 15.5% severe hypoglycemia in the past year. No significant association was made between hypoglycemia awareness and age, body-mass index (BMI), years of diabetes, dose of insulin, duration of insulin use, number of meals, or amount of snacking. A significant correlation was found between A1c levels and hypoglycemia awareness and severity of hypoglycemia. A significant correlation was found between dose of insulin, amount of snacking, and severity of hypoglycemia. No significant association was made between severity of hypoglycemia and age, BMI, years of diabetes, duration of insulin use, or the number of meals. However, the group with severe hypoglycemia had diabetes longer, and the average daily dose of insulin use was higher than in other groups.ConclusionsAccording to the study results, the percentage of patients with impaired awareness of hypoglycemia is high, and 62% of patients reported that they had missed some of the symptoms of hypoglycemia in type 2 diabetes. In addition, the percentage of severe hypoglycemic events is not low. Impaired awareness of hypoglycemia is a major risk factor for severe hypoglycemic events. Patients should be educated about the danger of hypoglycemia. Education should be improved, and a determined attempt should be made to eradicate the problem.
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Beattie, Sarah. "Hypoglycemia." Nurse Practitioner 48, no. 10 (October 2023): 17–23. http://dx.doi.org/10.1097/01.npr.0000000000000100.

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Abstract: Primary care NPs are central to the management of diabetes mellitus, which carries with it the risk of hypoglycemia. Fully understanding risk factors, prevention strategies, and treatment assist in reducing hypoglycemic events. This article details hypoglycemia, risk factors for hypoglycemia, prevention strategies, and appropriate treatment plans.
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Parilo, Miguel A. "Gatifloxacin-Associated Hypoglycemia." Journal of Pharmacy Technology 18, no. 6 (November 2002): 319–20. http://dx.doi.org/10.1177/875512250201800605.

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Objective: To report a case of prolonged hypoglycemia associated with glyburide and gatifloxacin use. Case Summary: An 82-year-old white woman with diabetes mellitus type 2 and chronic renal insufficiency developed postoperative pneumonia. She had previously been on long-term glyburide therapy. Protracted hypoglycemia after institution of gatifloxacin developed despite discontinuation of oral hypoglycemic therapy. After 2 days of intravenous dextrose, sustained normoglycemia was achieved. Discussion: Hypoglycemic reactions with glyburide and fluoroquinolone antibiotics have been reported, but not with gatifloxacin. Although drug administration error cannot be excluded, no documentation exists to support this. The onset of hypoglycemia soon after administration of gatifloxacin and reports of similar interactions favor the hypothesis that hypoglycemia was induced by a gatifloxacin–glyburide interaction. Conclusions: Fluoroquinolone-associated hypoglycemia has been documented, and an interaction of gatifloxacin and glyburide appears probable. Patients with diabetes should be monitored for the development of resistant hypoglycemia, especially if they are on concomitant oral hypoglycemic medications.
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Singh, Dolat, Hamid Nawaz Ali Memon, Tariq Zaffar Shaikh, and Syed Zulfiquar Ali Shah. "HYPOGLYCEMIA." Professional Medical Journal 22, no. 04 (April 10, 2015): 408–13. http://dx.doi.org/10.29309/tpmj/2015.22.04.1316.

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Objective: To determine the frequency and severity of hypoglycemia in patientswith liver cirrhosis. Study Design: Cross sectional case series study. Period: Six months.Setting: Liaquat University Hospital Hyderabad. Methods: All the patients of liver cirrhosis,of >12 years of age and of either gender were evaluated for hypoglycemia by assessing theglycemic status through random or fasting blood glucose level. The severity of liver cirrhosiswas identified according to the Child-Pugh classification whereas the severity of hypoglycemiawas grouped in mild, moderate and severe categories. The data was entered and saved inSPSS and frequency and percentage was calculated for hypoglycemia in patients with livercirrhosis. The stratification was done for age, gender, hypoglycemia and severity of the diseaseand hypoglycemia. The chi-square test was applied between categorical variables at 95%confidence interval and p -value ≤0.05 was considered as statistically significant. Results:During six months study period, total 100 cirrhotic subjects were studied for hypoglycemia,of which 59% were males and 41% were females. The mean ± SD for age in all (100)cirrhotic patients was 42.33 ± 8.87 while the mean ± SD for age in male cirrhotic patientswas 44.06±11.45 where as in female cirrhotic subjects it was 39.92±12.55 respectively. Thehypoglycemia was observed in 67%, of which 45(67.2%) were males and 22(32.8%) werefemales. The mean random blood glucose level in male and female hypoglycemic cirrhoticpatients was 67.88±8.43 and 65.62±6.75 while the mean fasting blood glucose level in maleand female hypoglycemic cirrhotic patients was 52.93±5.31 and 53.64±8.73 respectively. Outof sixty seven hypoglycemic cirrhotic subjects 45(67%) were males and 22(33%) were females.Of sixty seven, 32(47.8%) had moderate hypoglycemia while 30/67(44.8%) were in Child-Pughclass B (p<0.05). Conclusions: The hypoglycemia was detected in patients with liver cirrhosis,hence frequent blood glucose monitoring is one of the most important way to detect mildhypoglycemia and prevent serious and severe episodes.
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Hekimsoy, Zeliha, Sevinç Biberoǧlu, Abdurrahman Çömlekçi, Oktay Tarhan, Cem Mermut, and Kadir Biberoǧlu. "Trimethoprim/sulfamethoxazole-induced hypoglycemia in a malnourished patient with severe infection." European Journal of Endocrinology 136, no. 3 (March 1997): 304–6. http://dx.doi.org/10.1530/eje.0.1360304.

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Abstract Hypoglycemia resulting from the combination of sulfonylurea and sulfonamides is a recognized drug interaction. Hypoglycemia induced by sulfonamides alone may be encountered less frequently. Because of their structural similarities to sulfonylureas. sulfonamides are liable to facilitate hypoglycemia by increasing insulin release in susceptible individuals. Sulfonamides can potentiate the hypoglycemic effect of sulfonylurea agents when given in combination. We describe a malnourished patient with severe infection who developed hypoglycemia during high-dose trimethoprim/sulfamethoxazole therapy. Elevated C-peptide concentrations during the hypoglycemic episode indicate that hypoglycemia resulted from increased endogenous insulin secretion. As malnourished patients are prone to hypoglycemia, we suggest that they should be monitored carefully if they are on sulfonamide therapy. European Journal of Endocrinology 136 304—306
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Johnson, Jeffrey A., Joanne E. Kappel, and M. Nabi Sharif. "Hypoglycemia Secondary to Trimethoprim/Sulfamethoxazole Administration in a Renal Transplant Patient." Annals of Pharmacotherapy 27, no. 3 (March 1993): 304–6. http://dx.doi.org/10.1177/106002809302700309.

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OBJECTIVE: To report a case of trimethoprim/sulfamethoxazole (TMP/SMX)-induced hypoglycemia in an immunosuppressed renal transplant patient. DATA SOURCES: English-language journal articles and reference texts identified via a MEDLINE search and a bibliographic review of pertinent data sources. DATA SYNTHESIS: Hypoglycemia resulting from the combination of sulfonylureas and sulfonamides is a recognized drug interaction. Hypoglycemia induced by sulfonamides alone may be encountered less frequently. Previously reported cases of TMP/SMX-induced hypoglycemia postulated that the sulfonamide mimics hypoglycemic sulfonylurea agents and stimulates pancreatic islet cells to secrete insulin. We report a case of hypoglycemia following the administration of high-dose TMP/SMX in a renal transplant patient. Elevated C-peptide concentrations following the hypoglycemic episode indicate that hypoglycemia resulted from increased endogenous insulin secretion. CONCLUSIONS: Hypoglycemia has been a rarely encountered result of TMP/SMX use. Patients receiving TMP/SMX, particularly those with impaired renal function and those receiving high doses, should be monitored closely for hypoglycemia.
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Haywood, Samuel C., Adam J. Bree, Erwin C. Puente, Dorit Daphna-Iken, and Simon J. Fisher. "Central but not systemic lipid infusion augments the counterregulatory response to hypoglycemia." American Journal of Physiology-Endocrinology and Metabolism 297, no. 1 (July 2009): E50—E56. http://dx.doi.org/10.1152/ajpendo.90673.2008.

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This study tests the hypothesis that lipids could act as an alternative fuel source in the brain during insulin-induced hypoglycemia. Male Sprague-Dawley rats were subjected to hyperinsulinemic (5 mU·kg−1·min−1) hypoglycemic (∼50 mg/dl) clamps. In protocol 1, intralipid (IL), a fat emulsion, was infused intravenously to prevent the fall in free fatty acid levels that occurs in response to hyperinsulinemic hypoglycemia. Intravenous lipid infusion did not alter the counterregulatory responses to hypoglycemia. To test whether IL could have central effects in mediating the counterregulatory response to hypoglycemia, in protocol 2 the brains of precannulated rats were intracerebroventricularly (icv) infused with IL or artificial cerebrospinal fluid (aCSF) as control. Unexpectedly, the epinephrine and glucagon response to hypoglycemia was significantly augmented with icv IL infusion. To determine whether central IL infusion could restore defective counterregulation, in protocol 3 rats were made recurrently hypoglycemic (RH) for 3 days and on the 4th day underwent hyperinsulinemic hypoglycemic clamps with icv IL or aCSF infusion. RH rats had the expected impaired epinephrine response to hypoglycemia, and icv IL infusion again significantly augmented the epinephrine response in RH rats to normal. With regard to our experimental model of hypoglycemic counterregulation, we conclude that 1) systemic lipid infusion did not alter the counterregulatory response to hypoglycemia, 2) the icv infusion of lipids markedly increased CSF FFA levels and paradoxically augmented the epinephrine and glucagon responses, and 3) the blunted sympathoadrenal response in recurrently hypoglycemic rats was completely normalized with the icv lipid infusion. It is concluded that, in the setting of insulin-induced hypoglycemia, increased brain lipids can enhance the sympathoadrenal response.
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Mete, Türkan, and Mustafa Cesur. "Non-diabetic hypoglycemia." Intercontinental Journal of Internal Medicine 1, no. 4 (November 29, 2023): 94–105. http://dx.doi.org/10.51271/icjim-0021.

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Glucose is the main substrate utilized by the brain, and therefore numerous counterregulatory mechanisms exist to maintain plasma glucose concentration. This makes it rare for hypoglycemia to develop in people who are not taking hypoglycemic drugs, such as insulin or sulfonylureas, for diabetes. The symptoms of hypoglycemia are nonspecific. The presence of Whipple’s triad is necessary for diagnosis. When symptoms occur spontaneously, the patient can be evaluated for hypoglycemia. If this is not possible, then a 72-hour fasting test or a mixed meal tolerance test can be performed to create conditions for symptoms to occur. Non-diabetic hypoglycemia is mainly divided into two main groups: insulin-mediated (hyperinsulinism) and insulin-independent. The main causes of hypoglycemia due to endogenous hyperinsulinism are insulinoma and islet cell hyperplasia (nesidioblastosis), post-bariatric surgery, and autoimmune hypoglycemia with the presence of anti-insulin antibodies. Other important causes of hypoglycemia include hypoglycemic drugs, non-islet cell tumors, hormonal deficiencies (primary adrenal insufficiency, anterior pituitary insufficiency), and critical illnesses (liver/kidney failure). In this article, we provide an overview of the pathogenesis and treatment of hypoglycemia.
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Chakraborty, Abhishek, and Monika Deb. "Hypoglycemia in breastfed neonates: a hospital-based study." International Journal of Contemporary Pediatrics 10, no. 4 (March 27, 2023): 479–83. http://dx.doi.org/10.18203/2349-3291.ijcp20230722.

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Background: Neonates has well-coordinated adaptation system which maintains the blood sugar at certain safe level in extra uterine life. However certain intrauterine risk factors alter this adaptation system leading to hypoglycemia in early post-natal period. Most of the time hypoglycemic episodes are transient but sometimes there may be recurrent or prolonged hypoglycemia leading to permanent insult in brain and neurological deficit in post-natal life. Aims and objective of the study was to find out incidence of hypoglycemia in exclusively breastfeed neonates and the risk factors associated with this in the post-natal ward of a tertiary care centre in North-East India. Methods: This is a prospective study conducted for a period of six month, where 112 exclusively breastfeed neonates who were shifted immediately to post-natal ward were included. Capillary blood sugar was checked at 1, 3, 6, 12, 24, 48 and 72 hours of life. Neonates with capillary blood glucose less than 40 were considered hypoglycemic. All the hypoglycemic babies were extensively evaluated for different intrauterine and post-natal risk factors. Results: Incidence of hypoglycemia was 16% (18 out of 112 babies). Significant numbers (30.5%) of LBW babies had hypoglycemia, where as 6.5% of normal birth weight babies had hypoglycemia. 38.8% of preterm babies had hypoglycemia where as 11.95% of term babies had hypoglycemic episodes. 71.4% (5 out of 7 babies) of neonates born from diabetic mother. All the large for date infants of diabetic mother had hypoglycemia. Conclusions: Our study came to a conclusion that incidence of hypoglycemia is not very uncommon finding in exclusively breastfeed neonates especially those with risk factors. Routine capillary blood glucose screening is utmost important to pick up the babies with hypoglycemia to prevent immediate and long-term complication.
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Dissertations / Theses on the topic "Hypoglycemia"

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Dodd, Will. "Hypoglycemia." Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/etsu-works/8922.

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Schutz, Peter W. "Neuroprotective effects of ketone bodies during hypoglycemia." Thesis, University of British Columbia, 2011. http://hdl.handle.net/2429/34014.

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The ketone body D-3-hydroxybutyrate (3OHB) is an alternative energy substrate for the brain during hypoglycemia. The capacity and limitations of 3OHB to compensate for cerebral glucose depletion in developing brain is insufficiently understood. We studied the effect of 3OHB treatment in a newly developed infant rat model of insulin induced, sustained, and EEG-controlled hypoglycemia. Continuous treatment with 3OHB during hypoglycemia resulted in increased 3OHB plasma levels in hypoglycemic animals and delayed the onset of clinical coma and of EEG burst-suppression (burst-suppression coma). 3OHB treated animals did not survive after resuscitation with glucose, compared to 80% survival of untreated hypoglycemic pups. Cleaved-caspase-3 immunohistochemistry and double labelling studies demonstrated a 20-fold increase of apoptotic mature oligodendrocytes in white matter of 3OHB treated animals, indicating a limited protective effect of 3OHB treatment. In contrast to glucose, D-3-hydroxybutyrate is not an anaplerotic substrate. Anaplerosis plays in important role in cerebral glutamate glutamine metabolism. Combination of D-3-hydroxybutyrate with the anaplerotic substrate propionate could enhance its protective effect during hypoglycemia. We compared the effectiveness of treatment with a single dose D-3-hydroxybutyrate alone or combined with propionate at the time of EEG burst-suppression coma. Both treatments resulted in a reversion of EEG activity from burst suppression to continuity, but only combined treatment resulted in clincal improvement of the comatose state. 3OHB alone largely corrected pathometabolic changes of glutamate metabolism but not of glycolytic and pentose phosphate pathway intermediates or of long chain acylcarnitines. Combined treatment was not associated with biochemical corrections over and above those achieved by 3OHB alone for the metabolites measured. 3OHB treatment has a limited effectiveness on clinical and neuropathology outcome after hypoglycemia in infant rats. The limited effectiveness of 3OHB treatment may be related to its inability to support glycolysis with associated pentose phosphate pathway and anaplerotic activity. Combined treatment with propionate enhances 3OHB’s protective effect during hypoglycemic coma. Future protective treatment should be based on complementary metabolic substrates.
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Eckert, Bodil. "Hypoglycaemia studies on central and peripheral nerve function /." Lund : Dept. of Internal Medicine, University of Lund, 1998. http://catalog.hathitrust.org/api/volumes/oclc/57426099.html.

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Ciraolo, Susan Taylor. "Model of extreme hypoglycemia in the ketotic dog." Case Western Reserve University School of Graduate Studies / OhioLINK, 1994. http://rave.ohiolink.edu/etdc/view?acc_num=case1057250332.

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Minić, Marina. "Investigation of a syndrome of non insulin-dependent hypoglycaemia and overgrowth." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708929.

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Sami, Sumrin Ramiza. "Islet Transplantation in Type I Diabetes Patients with Hypoglycemia." Thesis, Umeå universitet, Farmakologi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-126081.

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Isom, Amanda M. "The Cellular Consequences of Combining Antipsychotic Medications and Hypoglycemia." University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1407407111.

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Clark, DessyeDee M. "Computer-aided hypoglycemia detection in adolescents with insulin-dependent diabetes mellitus /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/7368.

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Kalergis, Maria. "Prevention of noctural hypoglycemia in adults with type 1 diabetes undergoing intensive management." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=19501.

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The objectives of this research were to determine the impact of 4 different bedtime snack compositions on prevention of nocturnal hypoglycemia and to determine whether optimized titration and delivery of bedtime insulin using multiple daily injections of insulin (MDI) or continuous subcutaneous insulin infusion (CSII) could prevent nocturnal hypoglycemia in the absence of bedtime snacks. We also sought to determine whether 3 months of CSII therapy would improve catecholamine response and symptom awareness to experimentally-induced hypoglycemia. The need for and the most appropriate composition of bedtime snacks were dependent on the glycemic level at bedtime. No bedtime snacks were necessary at bedtime glycemic levels > 10 mmol/L. At bedtime glycemic levels between 7-10 mmol/L , a standard snack and cornstarch-containing snack worked best and at bedtime glycemic levels < 7mmol/L, a standard and protein-rich snack were most effective. Despite optimized titration and delivery of bedtime insulin, including the use of CSII, "the gold standard" of nocturnal insulin replacement, the incidence of nocturnal hypoglycemia over 181 nights was 54 episodes per 100 patientnights. However, there was a substantial reduction, by 36% (p=0.17), in the incidence of nocturnal hypoglycemia with the use of bedtime snacks. Therefore bedtime snacks, tailored to the bedtime glycemic level, are recommended for ail adults undergoing intensive management with MDI or CSII. Although, 3 months of CSII therapy did not improve catecholamine response and symptom awareness to experimentally-induced hypoglycemia, it did not deteriorate the responses either. Therefore, CSII therapy is a viable option in intensive management of adults with type 1 diabetes.
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Leelarathna, Lalantha Harendra. "Improving glucose control and reducing the burden of hypoglycaemia : use of novel diabetes technology in type 1 diabetes and critical illness." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648482.

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Books on the topic "Hypoglycemia"

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B, Field James, ed. Hypoglycemia. Philadelphia: Saunders, 1989.

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D, Andreani, Marks Vincent, Lefèbvre P. J. 1934-, and International Symposium on Hypoglycemia (3rd : 1986 : Rome, Italy), eds. Hypoglycemia. New York: Raven Press, 1987.

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1948-, Chow James H., ed. Hypoglycemia for dummies. New York: Wiley Pub., 2003.

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Chow, Cheryl. Hypoglycemia For Dummies. New York: John Wiley & Sons, Ltd., 2007.

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Chow, Cheryl. Hypoglycemia for dummies. 2nd ed. Hoboken, N.J: Wiley, 2007.

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Tenney, Louise. Hypoglycemia, a nutritional approach. Plesant Grove, UT: Woodland Pub., 1996.

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Dulock, Helen L. Hypoglycemia in the newborn. 2nd ed. White Plains, NY: March of Dimes Birth Defects Foundation, 1990.

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M, Ross Harvey, ed. Hypoglycemia: The classic healthcare handbook. New York: Kensington, 1996.

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Gilbert, Manso, and Ingram Colin 1936-, eds. Hypoglycemia and diabetes wellness guide. Lake Charles, La: Body Care Publications, 1995.

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Gyula, Soltész, ed. Hypoglycaemia in infancy and childhood. Edinburgh: Churchill Livingstone, 1985.

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Book chapters on the topic "Hypoglycemia"

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James, Chela, and Khalid Hussain. "Hypoglycemia." In Textbook of Clinical Pediatrics, 3803–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02202-9_389.

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Fanelli, Carmine G., Paola Lucidi, Geremia B. Bolli, and Francesca Porcellati. "Hypoglycemia." In Endocrinology, 617–54. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-44433-8_22.

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Pundyk, Katherine Jane, and Seth Daniel Marks. "Hypoglycemia." In Endocrine Conditions in Pediatrics, 53–55. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-52215-5_8.

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von Dessauer, Bettina, and Derek S. Wheeler. "Hypoglycemia." In Pediatric Critical Care Medicine, 103–7. London: Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6416-6_9.

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Demakis, George J. "Hypoglycemia." In Encyclopedia of Clinical Neuropsychology, 1761–62. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_558.

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Fanelli, Carmine G., Paola Lucidi, Geremia B. Bolli, and Francesca Porcellati. "Hypoglycemia." In Endocrinology, 615–52. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-36694-0_22.

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Lord, Katherine, Diva D. De León, and Charles A. Stanley. "Hypoglycemia." In Pediatric Endocrinology, 701–15. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73782-9_30.

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Fanelli, Carmine G., Paola Lucidi, Geremia B. Bolli, and Francesca Porcellati. "Hypoglycemia." In Endocrinology, 1–38. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-27316-7_22-1.

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Fanelli, Carmine G., Paola Lucidi, Geremia B. Bolli, and Francesca Porcellati. "Hypoglycemia." In Endocrinology, 1–38. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-27316-7_22-2.

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Orbell, Sheina, Havah Schneider, Sabrina Esbitt, Jeffrey S. Gonzalez, Jeffrey S. Gonzalez, Erica Shreck, Abigail Batchelder, et al. "Hypoglycemia." In Encyclopedia of Behavioral Medicine, 1016–17. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_964.

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Conference papers on the topic "Hypoglycemia"

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Aljuaid, Awad. "Hypoglycemia Vehicle Detection System Using Non-Invasive Sensors Applying Both EEG And HRV Real Time Measures: Neuroergonomics Theoretical Design." In 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1001480.

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During COVID 19 pandemic the global needs for online shopping, ride-sharing transportation, and food delivery services have been dramatically increased. The drivers who suffer from diabetes especially low blood sugar level (Hypoglycemia) are more likely at risk than others. Earlier literature has revealed that hypoglycemic issues in patients with diabetes are correlated with significant changes in scalp electroencephalography (EEG); signals amplitude (time domain) or power spectral density (frequency domain). In addition, Haret rate variability HRV which reflects the balance between the sympathetic and parasympathetic nervous system has been proven as one of the indicators of Hypoglycemia. The aim of this paper to propose a conceptual design of a Vehicle detection system using both EEG and HRV measures at the same time in real time feed using non-invasive sensors to reduce the potential of driver’s cognitive dysfunction.
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Barsoum, A., J. Huang, M. Kazemi, J. Price, and I. Chakrabarty. "Hydroxychloroquine Induced Hypoglycemia." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a1364.

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Hirshberg, E., J. Jones, J. Orme, and A. Morris. "Hypoglycemia and Insulin Received." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a1576.

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Varadan, Vijay K., Ashwin K. Whitchurch, and Karunakaran Sarukesi. "Noninvasive biosensor for hypoglycemia." In Micromachining and Microfabrication, edited by Holger Becker and Peter Woias. SPIE, 2003. http://dx.doi.org/10.1117/12.479565.

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Nguyen, H. T., N. Ghevondian, and T. W. Jones. "Real-time detection of nocturnal hypoglycemic episodes using a novel non-invasive hypoglycemia monitor." In 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5335144.

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6

Hematbhai, Satodiya Mohit. "One step versus two step screening for gestational diabetes mellitus." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685382.

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Objective: To compare the incidence, maternal and fetal outcomes of gestational diabetes mellitus using one step vs. two step as a screening procedure. Methodology: A prospective randomized trial involving screening of 1000 pregnant women for gestational diabetes mellitus was conducted. Women were divided in two groups (500 each). Group A comprised of patients screened with two step approach (ACOG recommendation), Group B comprised of women screened by one step method (IADPSG criteria). Women diagnosed with ‘gestational diabetes’ were followed in antenatal clinic and incidence of GDM, maternal and fetal outcome between two groups were analyzed using SPSS. Results: The incidence of GDM was almost double using one step approach versus two step which was 19.2% and 11.8% respectively. Maternal outcomes were comparable in both the groups except the risk of preterm delivery which was 2.5 times more in group A than group B (odds ratio = 2.43 95% CI = 1.01-5.79). Further fetal outcomes were also comparable except neonatal hypoglycemia which was seen in 29.31% in group A vs. 7.4% in group B. In the group B 15 patients (15.8%) patients with GDM (based on FBS ≥92 mg/dl at 1st ANC visit) showed clinical symptoms & blood sugars in hypoglycemic range on MNT requiring resumption of normal diet. Conclusion: The incidence of GDM using IADPSG criteria was almost double versus ACOG criteria. Maternal and fetal outcomes were comparable except in 15.8% women diagnosed as GDM (using FBS ≥92 mg/dl at 1st ANC visit as per IADPSG) suffered from hypoglycemia. A large trial is being proposed before these criteria are adopted.
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Hematbhai, Satodiya Mohit. "Oral Abstract." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685353.

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Objective: To compare the incidence, maternal and fetal outcomes of gestational diabetes mellitus using one step vs. two step as a screening procedure. Methodology: A prospective randomized trial involving screening of 1000 pregnant women for gestational diabetes mellitus was conducted. Women were divided in two groups (500 each). Group A comprised of patients screened with two step approach (ACOG recommendation), Group B comprised of women screened by one step method (IADPSG criteria). Women diagnosed with ‘gestational diabetes’ were followed in antenatal clinic and incidence of GDM, maternal and fetal outcome between two groups were analyzed using SPSS. Results: The incidence of GDM was almost double using one step approach versus two step which was 19.2% and 11.8%respectively. Maternal outcomes were comparable in both the groups except the risk of preterm delivery which was 2.5 times more in group A than group B (odds ratio = 2.43 95% CI = 1.01-5.79). Further fetal outcomes were also comparable except neonatal hypoglycemia which was seen in 29.31% in group A vs. 7.4% in group B. In the group B 15 patients (15.8%) patients with GDM (based on FBS ≥ 92 mg/dl at 1st ANC visit) showed clinical symptoms and blood sugars in hypoglycemic range on MNT requiring resumption of normal diet. Conclusion: The incidence of GDM using IADPSG criteria was almost double versus ACOG criteria. Maternal and fetal outcomes were comparable except in 15.8% women diagnosed as GDM (using FBS ≥ 92 mg/dl at 1st ANC visit as per IADPSG) suffered from hypoglycemia. A large trial is being proposed before these criteria are adopted.
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Leyton, C., G. Glazman-Kuczaj, C. Reisig, and M. Duda. "An Unlikely Cause of Persistent Hypoglycemia." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6783.

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Nuryani, Steve Ling, and H. T. Nguyen. "Ventricular repolarization variability for hypoglycemia detection." In 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6091963.

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Camelo, Clara Gontijo, Cristiane Araújo Martins Moreno, Mariana Cunha Artilheiro, André Macedo Serafim Silva, Alullin Tácio Quadros Monteiro Fonseca, Rodrigo Mendonça de Holanda, Umbertina Conti Reed, and Edmar Zanoteli. "Hypoglycemia in patients with LAMA2-CMD." In SBN Conference 2022. Thieme Revinter Publicações Ltda., 2023. http://dx.doi.org/10.1055/s-0043-1774431.

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Reports on the topic "Hypoglycemia"

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Wang, Yan, Fangxin Jin, Minne Li, Rujiang Li, and Xueli Zhang. The Effect of Liraglutide on Hypoglycemia in Type 1 Diabetes Mellitus: A Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2024. http://dx.doi.org/10.37766/inplasy2024.6.0051.

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Hung, Hsuan-Yu, and Chung-Yu Chen. The impact of Sofosbuvir/Velpatasvir/Voxilaprevir treatment on serum hyperglycemia in HCV infections: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2021. http://dx.doi.org/10.37766/inplasy2021.12.0109.

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Review question / Objective: To assess the possible cause of events, the incidence of grade 3 hyperglycemia after treating Sofosbuvir/Velpatasvir/Voxilaprevir in HCV infections. Condition being studied: Sofosbuvir, velpatasvir, and voxilaprevir (SOF/VEL/VOX) is an effective, safe rescue therapy regimen for patients have previously been treated failure. Initiating Direct-Acting Antiviral (DAA) treatment for HCV infection with diabetes have experienced hypoglycemia, it could improve insulin resistance due to clean HCV. However, some studies shown that SOF/VEL/VOX has Grade 3 hyperglycemia adverse events. This finding contradicts that other DAAs studies. Information sources: Conducting a comprehensive literature search on the pubmed, Cochrane, clinicalkey, Embase, and MEDLINE electronic databases.
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Yang, Yan, Fan Xiumei, Dongqiong Chen, and C. Xie. The influence of hypoglycemia and hyperglycemia on the adverse outcome of COVID-19 combined with diabetes mellitus: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2020. http://dx.doi.org/10.37766/inplasy2020.8.0096.

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Silva, Rodrigo Ribeiro e., Mateus de Miranda Gauza, Julia Opolski Nunes da Silva Opolski, and Maria Eduarda Schramm Guisso. Once-Weekly Insulin Icodec vs Once-Daily Insulin Glargine U100 for Type 2 Diabetes: A Meta-analysis of Phase 2 Randomized Controlled Trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0102.

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Review question / Objective: To compare Once-Weekly Insulin Icodec and Once-Daily Insulin Glargine U100 in patients with Type 2 Diabetes Mellitus using oral hypoglycemic drugs in need of insulin therapy. Condition being studied: Patients with Diabetes Mellitus Type 2 using oral hypoglycemic drugs in need for basal insulin. Eligibility criteria: Inclusion in this meta-analysis was restricted to studies that met all the following criteria: (1) randomized trials; (2) comparing the use once weekly insulin icodec to once daily insulin glargine; (3) enrolling patients with type 1 or type 2 diabetes mellitus; (4) evaluating any of the desired outcomes; (4) articles in written on english language. We excluded studies with (1) no control group; (2) overlapping studies population; clinical trial register entry only; (3) non-human studies and (4) studies reported only as abstracts.
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Li, Yijun, Ying Hu, Kang Chen, Bing Li, Weijun Gu, and Yiming Mu. Comparison of efficacy and safety of three novel hypoglycemic agents in patients with severe diabetic kidney disease. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2021. http://dx.doi.org/10.37766/inplasy2021.12.0106.

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Song, Ruirui, Hongmei Gao, Fang Liu, Jun Chen, and Xiaojing Shi. Effects of new hypoglycemic drugs on patients with heart failure: a systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2023. http://dx.doi.org/10.37766/inplasy2023.9.0031.

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Liu, Boyu, Wenyue Sun, and Qing Zhao. The efficacy of hypoglycemic agents for alzheimer’s disease and mild cognitive Impairment: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2021. http://dx.doi.org/10.37766/inplasy2021.6.0075.

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Liu, Su-tong, Kai-qi Su, Li-hui Zhang, Ming-hao Liu, and Wen-xia Zhao. Hypoglycemic agents for non-alcoholic fatty liver disease with type 2 diabetes mellitus: A protocol for systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2020. http://dx.doi.org/10.37766/inplasy2020.7.0016.

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Wu, Haoyu, Ming Zhang, Bing Ji, Yue Xu, Congjian Jin, and Tianhang Chen. The impact of novel hypoglycemic medications on hormonal and metabolic parameters in patients with polycystic ovary syndrome: a Bayesian network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, January 2024. http://dx.doi.org/10.37766/inplasy2024.1.0001.

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Wang, Yunfang, Jianguo Xu, Jinxing Quan, Ruilan Niu, Zhiwei Hu, and Jing Liu. Safety and efficacy of hypoglycemic agents in patients with type 2 diabetes mellitus (T2DM): protocol for an overview of systematic reviews based on network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2020. http://dx.doi.org/10.37766/inplasy2020.7.0118.

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